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Relevant bibliographies by topics / GP90 lymphocyte homing

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Journal articles

Academic literature on the topic 'GP90 lymphocyte homing'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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Journal articles on the topic "GP90 lymphocyte homing"

1

Pals, S. T., F. Hogervorst, G. D. Keizer, T. Thepen, E. Horst, and C. C. Figdor. "Identification of a widely distributed 90-kDa glycoprotein that is homologous to the Hermes-1 human lymphocyte homing receptor." Journal of Immunology 143, no. 3 (1989): 851–57. http://dx.doi.org/10.4049/jimmunol.143.3.851.

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Abstract Homing of recirculating lymphocytes from the blood into the lymphoid tissues is mediated by 90-kDa homing receptors on the lymphocyte cell surface, allowing selective binding to specialized endothelium lining high endothelial venules. This study describes two novel mAb, NKI-P1 and NKI-P2, directed against functional epitopes of a human lymphocyte homing receptor, gp90. Biochemical studies demonstrated that these antibodies recognize a 90-kDa glycoprotein which is similar to the Ag recognized by the mAb Hermes-1. This notion was confirmed by immunohistochemical studies showing identica

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2

Jung, T. M., and M. O. Dailey. "Rapid modulation of homing receptors (gp90MEL-14) induced by activators of protein kinase C. Receptor shedding due to accelerated proteolytic cleavage at the cell surface." Journal of Immunology 144, no. 8 (1990): 3130–36. http://dx.doi.org/10.4049/jimmunol.144.8.3130.

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Abstract Lymphocyte entry into lymph nodes and Peyer's patches is initiated by the adhesion of the lymphocytes to specialized postcapillary high endothelial venules (HEV). The binding of lymphocytes to lymph node HEV is mediated by the cell surface receptor gp90MEL-14 (gp90). Previous work has shown that gp90 is down-regulated over a period of days after mitogenic or mixed lymphocyte reaction stimulation of T lymphocytes. In our study, it is shown that stimulation of lymphocytes with activators of protein kinase C (PKC), such as PMA or 1-oleoyl 2-acetyl-glycerol, results in the nearly complete

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Mobley, J. L., and M. O. Dailey. "Regulation of adhesion molecule expression by CD8 T cells in vivo. I. Differential regulation of gp90MEL-14 (LECAM-1), Pgp-1, LFA-1, and VLA-4 alpha during the differentiation of cytotoxic T lymphocytes induced by allografts." Journal of Immunology 148, no. 8 (1992): 2348–56. http://dx.doi.org/10.4049/jimmunol.148.8.2348.

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Abstract A variety of adhesion molecules regulate the traffic and tissue localization of lymphocytes in vivo by mediating their binding to vascular endothelial cells. The homing receptor gp90MEL-14 (gp90), also known as LECAM-1 or L-selectin, mediates the adhesion of lymphocytes to specialized high endothelial venules in lymph nodes (LN) and is the primary molecule regulating lymphocyte recirculation and homing to LN, whereas other adhesion molecules have a major role in the localization of lymphocytes in inflammatory sites. We used four-color flow cytometric analysis to examine the regulation

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4

Hamann, A., D. Jablonski-Westrich, A. Duijvestijn, et al. "Evidence for an accessory role of LFA-1 in lymphocyte-high endothelium interaction during homing." Journal of Immunology 140, no. 3 (1988): 693–99. http://dx.doi.org/10.4049/jimmunol.140.3.693.

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Abstract In a variety of lymphocyte interactions, lymphocyte function-associated antigen-1 (LFA-1) plays an important role as an accessory mechanism mediating cell adhesion. We tested the possibility that LFA-1 could also be involved in the specific binding of lymphocytes to high endothelial venules (HEV) during homing. Antibodies against LFA-1 but not against various other cell surface molecules (except the putative gp90 homing receptor defined by the MEL-14 antibody) were found to inhibit in vitro adherence of lymphocytes to HEV in frozen sections of lymph nodes. Binding of T cell lines to H

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5

Mountz, J. D., W. C. Gause, F. D. Finkelman, and A. D. Steinberg. "Prevention of lymphadenopathy in MRL-lpr/lpr mice by blocking peripheral lymph node homing with Mel-14 in vivo." Journal of Immunology 140, no. 9 (1988): 2943–49. http://dx.doi.org/10.4049/jimmunol.140.9.2943.

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Abstract MRL-lpr/lpr mice develop massive lymphadenopathy and autoimmunity. There is evidence that both migration and local proliferation contribute to the accumulation of Ly-2-, L3T4-, 6B2+ T cells in the peripheral lymph node (PLN). Mel-14 is an antibody which binds to the lymphocyte lymph node homing receptor (gp90Mel-14) and can block migration of lymphocytes to the PLN. Treatment of mice from birth to 11 wk of age with Mel-14 and another rat IgG2a mAb, 6B2, resulted in reduction (10- to 20-fold) in lymphadenopathy. Mel-14, but not 6B2, preferentially reduced the percentages of Thy-1+, 6B2

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Brown, T. A., T. Bouchard, T. St John, E. Wayner, and W. G. Carter. "Human keratinocytes express a new CD44 core protein (CD44E) as a heparan-sulfate intrinsic membrane proteoglycan with additional exons." Journal of Cell Biology 113, no. 1 (1991): 207–21. http://dx.doi.org/10.1083/jcb.113.1.207.

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We previously identified a 90-kD (GP90), collagen-binding, membrane glycoprotein, termed extracellular matrix receptor III (ECMR III), that is homologous to the lymphocyte homing receptor and CD44 antigen (Gallatin, W. M., E. A. Wayner, P. A. Hoffman, T. St. John, E. C. Butcher, and W. G. Carter. 1989. Proc. Natl. Acad. Sci. USA. 86:4654-4658). CD44 is abundantly expressed in many epithelial tissues, and is localized predominantly to filopodia in cultured keratinocytes. Here we establish CD44 as a polymorphic family of related membrane proteoglycans and glycoproteins possessing extensive diver

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Strbo, Natasa, Laura Padula, Eva Fisher, et al. "Secreted heat shock protein gp96-Ig vaccine induces malaria specific intrahepatic CD8 T cell responses." Journal of Immunology 204, no. 1_Supplement (2020): 168.2. http://dx.doi.org/10.4049/jimmunol.204.supp.168.2.

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Abstract A highly-efficacious and durable malaria vaccine is an essential tool for global malaria eradication. One promising strategy to develop such a vaccine is to induce robust CD8+ T cell-mediated protective immunity against malaria liver stage parasites. We developed and tested a novel malaria vaccine platform based on the secreted form of the heat shock protein gp96 immunoglobulin, (gp96-Ig) to induce malaria antigen-specific liver resident memory CD8+ T cells. Gp96-Ig has ideal properties as an antigen carrier and vaccine adjuvant to activate antigen presenting cells (APCs) and effectiv

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8

Ayoubi, Riham, Walaa Alshafie, Peter S. McPherson, and Carl Laflamme. "Antibody Characterization Report for CD44 antigen." April 30, 2021. https://doi.org/10.5281/zenodo.4730966.

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