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1

Coe, John Clark. Gladys Grafton Eames: A memoir : with a lexicon from Gladys. J.C. Coe, 2000.

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2

Johnson, Carole D. Lithology and fracture characterization from drilling investigations in the Mirror Lake area, Grafton County, New Hampshire. U.S. Dept. of the Interior, U.S. Geological Survey, 1998.

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3

Johnson, Carole D. Lithology and fracture characterization from drilling investigations in the Mirror Lake Area, Grafton County, New Hampshire. U.S. Department of the Interior, U.S. Geological Survey, Toxic Substance Hydrology Program, 1998.

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4

District, United States Army Corps of Engineers Chicago. Charts of the Illinois waterway: From Mississippi River at Grafton, Illinois to Lake Michigan at Chicago and Calumet harbors. U.S. Army Engineer District, Corps of Engineers, 1987.

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5

Rosenfeld, Richard. The Gardener's Guide to Propagation: Step-by-step instructions for creating plants for free, from propagating seeds and cuttings to dividing, layering and grafting. Lorenz Books, 2011.

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6

Chaloner, E. Combined vascular and orthopaedic injuries. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.012009.

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♦ Early diagnosis of an arterial injury is critical in reducing the risk of limb loss♦ Don’t assume that missing pulses are due to arterial ‘spasm’♦ Don’t assume that presence of distal pulses rules out a proximal vascular injury – arterial intimal tears can occlude the vessel many hours after the initial injury♦ After an arterial repair has been completed there is still a risk of subsequent compartment syndrome from reperfusion♦ Arterial shunts can procure some time for skeletal fixation prior to definitive arterial repair or grafting.
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7

Pisapia, Jared M., Zarina S. Ali, Gregory G. Heuer, and Eric L. Zager. Adult Upper Trunk Brachial Plexus Injury. Edited by Meghan E. Lark, Nasa Fujihara, and Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0022.

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This chapter takes a case-based approach to the diagnosis and management of adult brachial plexus injury involving the upper trunk. The clinical presentation and differential diagnosis associated with this injury pattern are reviewed, as well as the findings of electrodiagnostic and imaging studies. Preoperative considerations include the timing from initial injury and the difference between pre- and postganglionic injury. Options for nerve reconstruction include nerve grafting, nerve transfer, or a combination of both. The options are compared, and a detailed description of each surgical procedure is provided, along with related complications, alternative repair strategies, and outcomes.
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8

Masrani, Abdulrahman, and Bulent Arslan. In Vivo Fenestration During Endovascular Aneurysm Repair. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0008.

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Abdominal aortic aneurysms have been managed endovascularly during the past 10–15 years. The main limitations in the ability to treat patients endovascularly are anatomical constraints. The most important factors are aortic neck and iliofemoral access anatomy. This chapter describes a technique to overcome a short neck with a renal artery originating from the aneurysm that does not allow enough proximal landing zone for stent grafting. Several techniques have been developed to overcome this obstacle, including custom-made grafts with fenestrations, back table fenestration, and parallel graft placement. This chapter discusses the in vivo graft fenestration technique to preserve the renal artery lumen during the endovascular repair of an abdominal aortic aneurysm.
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9

Calder, Peter. Chronic long bone osteomyelitis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.011001.

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Pathological features of chronic osteomyelitis♦ Necrotic bone♦ Compromised soft tissues with reduction in vascularity♦ Ineffective host response♦ Sequestrum formation♦ New bone formation from viable periosteum and endosteum♦ Formation of involucrum:Treatment principles in chronic osteomyelitis♦ Surgical debridement – remove all devitalized necrotic tissue♦ Dead space management:• Soft tissue defect – avoid healing by secondary intention. Consider local and free flaps• Bone defects – small structural with autologous bone graft, consider Papineau ‘open bone grafting’ where free tissue transfer is not an option, distraction osteogenesis with bifocal and bone transport for large defects including fibula transfer♦ Bone stability – movement needs to be eliminated♦ Antibiotic therapy – based on culture and sensitivity, local administration with PMMA beads or collagen sponge, Lautenbach procedure in resistant cases.
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10

Taggart, David, and Yasir Abu-Omar. Heart surgery. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0098.

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Cardiac surgery is still a relatively young specialty, having been developed only in the latter half of the twentieth century with the introduction of extracorporeal circulation or ‘cardiopulmonary bypass’ (CPB). This initiated the era of open heart surgery, initially allowing the repair of congenital heart defects, then valve replacements, coronary artery bypass grafting (CABG), and, finally, heart transplantation. Over the last two decades, improvements in medical, anaesthetic, and surgical management of patients, allied to refinements in extracorporeal perfusion technology, have resulted in a decreasing mortality and morbidity from heart surgery despite the advanced age and significant comorbidity of many patients. Today, heart surgery continues to improve the prognosis and quality of lives of patients around the world. Surgical techniques and technologies continue to evolve and recent years have witnessed the emergence of, amongst others, the use of long-lasting conduits for CABG procedures, beating-heart (‘off-pump’) surgery, the use of minimally invasive and robotic techniques, and long-term mechanical circulatory support.
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11

Thiele, Holger, and Uwe Zeymer. Cardiogenic shock in patients with acute coronary syndromes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0049.

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Cardiogenic shock complicating an acute coronary syndrome is observed in up to 10% of patients and is associated with high mortality still approaching 50%. The extent of ischaemic myocardium has a profound impact on the initial, in-hospital, and post-discharge management and prognosis of the cardiogenic shock patient. Careful risk assessment for each patient, based on clinical criteria, is mandatory, to decide appropriately regarding revascularization by primary percutaneous coronary intervention or coronary artery bypass grafting, drug treatment by inotropes and vasopressors, mechanical left ventricular support, additional intensive care treatment, triage among alternative hospital care levels, and allocation of clinical resources. This chapter will outline the underlying causes and diagnostic criteria, pathophysiology, and treatment of cardiogenic shock complicating acute coronary syndromes, including mechanical complications and shock from right heart failure. There will be a major focus on potential therapeutic issues from an interventional cardiologist’s and an intensive care physician’s perspective on the advancement of new therapeutical arsenals, both mechanical percutaneous circulatory support and pharmacological support. Since studying the cardiogenic shock population in randomized trials remains challenging, this chapter will also touch upon the specific challenges encountered in previous clinical trials and the implications for future perspectives in cardiogenic shock.
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12

Thiele, Holger, and Uwe Zeymer. Cardiogenic shock in patients with acute coronary syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0049_update_001.

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Cardiogenic shock complicating an acute coronary syndrome is observed in up to 10% of patients and is associated with high mortality still approaching 50%. The extent of ischaemic myocardium has a profound impact on the initial, in-hospital, and post-discharge management and prognosis of the cardiogenic shock patient. Careful risk assessment for each patient, based on clinical criteria, is mandatory, to decide appropriately regarding revascularization by primary percutaneous coronary intervention or coronary artery bypass grafting, drug treatment by inotropes and vasopressors, mechanical left ventricular support, additional intensive care treatment, triage among alternative hospital care levels, and allocation of clinical resources. This chapter will outline the underlying causes and diagnostic criteria, pathophysiology, and treatment of cardiogenic shock complicating acute coronary syndromes, including mechanical complications and shock from right heart failure. There will be a major focus on potential therapeutic issues from an interventional cardiologist’s and an intensive care physician’s perspective on the advancement of new therapeutical arsenals, both mechanical percutaneous circulatory support and pharmacological support. Since studying the cardiogenic shock population in randomized trials remains challenging, this chapter will also touch upon the specific challenges encountered in previous clinical trials and the implications for future perspectives in cardiogenic shock.
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13

Thiele, Holger, and Uwe Zeymer. Cardiogenic shock in patients with acute coronary syndromes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0049_update_002.

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Cardiogenic shock complicating an acute coronary syndrome is observed in up to 10% of patients and is associated with high mortality still approaching 50%. The extent of ischaemic myocardium has a profound impact on the initial, in-hospital, and post-discharge management and prognosis of the cardiogenic shock patient. Careful risk assessment for each patient, based on clinical criteria, is mandatory, to decide appropriately regarding revascularization by primary percutaneous coronary intervention or coronary artery bypass grafting, drug treatment by inotropes and vasopressors, mechanical left ventricular support, additional intensive care treatment, triage among alternative hospital care levels, and allocation of clinical resources. This chapter will outline the underlying causes and diagnostic criteria, pathophysiology, and treatment of cardiogenic shock complicating acute coronary syndromes, including mechanical complications and shock from right heart failure. There will be a major focus on potential therapeutic issues from an interventional cardiologist’s and an intensive care physician’s perspective on the advancement of new therapeutical arsenals, both mechanical percutaneous circulatory support and pharmacological support. Since studying the cardiogenic shock population in randomized trials remains challenging, this chapter will also touch upon the specific challenges encountered in previous clinical trials and the implications for future perspectives in cardiogenic shock.
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14

Thiele, Holger, and Uwe Zeymer. Cardiogenic shock in patients with acute coronary syndromes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0049_update_003.

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Cardiogenic shock complicating an acute coronary syndrome is observed in up to 10% of patients and is associated with high mortality still approaching 50%. The extent of ischaemic myocardium has a profound impact on the initial, in-hospital, and post-discharge management and prognosis of the cardiogenic shock patient. Careful risk assessment for each patient, based on clinical criteria, is mandatory, to decide appropriately regarding revascularization by primary percutaneous coronary intervention or coronary artery bypass grafting, drug treatment by inotropes and vasopressors, mechanical left ventricular support, additional intensive care treatment, triage among alternative hospital care levels, and allocation of clinical resources. This chapter will outline the underlying causes and diagnostic criteria, pathophysiology, and treatment of cardiogenic shock complicating acute coronary syndromes, including mechanical complications and shock from right heart failure. There will be a major focus on potential therapeutic issues from an interventional cardiologist’s and an intensive care physician’s perspective on the advancement of new therapeutical arsenals, both mechanical percutaneous circulatory support and pharmacological support. Since studying the cardiogenic shock population in randomized trials remains challenging, this chapter will also touch upon the specific challenges encountered in previous clinical trials and the implications for future perspectives in cardiogenic shock.
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15

Fabbri, Andrea, Giorgio Bartolini, Maurizio Lambardi, and Stan Kailis. Olive Propagation Manual. CSIRO Publishing, 2004. http://dx.doi.org/10.1071/9780643091016.

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This practical manual is an authoritative guide to olive propagation, providing extended information on seed germination, rooting of cuttings, grafting and micropropagation. The authors describe each topic in detail and discuss the relative advantages and disadvantages of each procedure.
 The Olive Propagation Manual has been developed to take into account the future demand for olive oil, which is expected to increase to three million tonnes annually over the next 10 years. Such volumes will require active farming programs and olive trees for new orchards and the replacement of olive trees in existing orchards. As the olive industry moves from traditional manual methods to mechanised operations, planting stock will need to be developed to meet future challenges. Varietal selection will need to be directed to clones that are early bearing, disease resistant, able to be mechanically harvested, and produce quality fruit and oil. Each of these issues are addressed throughout this book.
 The Olive Propagation Manual explores historical perspectives, traditional methods and state-of-the-art olive propagation including theoretical explanations and all practical aspects.
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16

Dilsizian, Vasken, Ines Valenta, and Thomas H. Schindler. Myocardial Viability Assessment. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0021.

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Heart failure may be a consequence of ischemic or non-ischemic cardiomyopathy. Etiologies for LV systolic dysfunction in ischemic cardiomyopathy include; 1) transmural scar, 2) nontransmural scar, 3) repetitive myocardial stunning, 4) hibernating myocardium, and 5) remodeled myocardium. The LV remodeling process, which is activated by the renin-angiotensin system (RAS), stimulates toxic catecholamine actions and matrix metalloproteinases, resulting in maladaptive cellular and molecular alterations5, with a final pathway to interstitial fibrosis. These responses to LV dysfunction and interstitial fibrosis lead to progressive worsening of LV function. Established treatment options for ischemic cardiomyopathy include medical therapy, revascularization, and cardiac transplantation. While there has been continuous progress in the medical treatment of heart failure with beta-blockers, angiotensin-converting enzyme (ACE) inhibition, angiotensin II type 1 receptor (AT1R) blockers, and aldosterone to beneficially influence morbidity and mortality, the 5-years mortality rate for heart failure patients remains as high as 50%. Revascularization procedures include percutaneous transluminal coronary artery interventions (PCI) including angioplasty and endovascular stent placement and coronary artery bypass grafting (CABG). Whereas patents with heart failure due to non-coronary etiologies may best benefit from medical therapy or heart transplantation, coronary revascularization has the potential to improve ventricular function, symptoms, and long term survival, in patients with heart failure symptoms due to CAD and ischemic cardiomyopathy.
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17

Steven, Gilbar, and Stewart Dean, eds. Tales of Santa Barbara: From native storytellers to Sue Grafton. J. Daniel, 1994.

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18

Gilbar, Steven. Tales of Santa Barbara: From Native Storytellers to Sue Grafton. John Daniel & Company Books, 1994.

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19

Schirmer, Uwe, and Andreas Koster. Anaesthesia for cardiac surgery. Edited by Philip M. Hopkins. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0056.

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Cardiac anaesthesia continues to develop as a specialized discipline within the wide field of clinical anaesthesia. A comprehensive knowledge of cardiovascular physiology and its improved monitoring with modern invasive and non-invasive devices is the basis for the pharmacological treatment of complex cardiovascular disorders. Excellent skills in intraoperative transoesophageal echocardiography have become essential. Rapid developments in cardiopulmonary bypass techniques and surgical devices have resulted in the speedy introduction of new surgical techniques which anaesthesia has to embrace. The developments in the field of (left) ventricular assist devices are expansive. By changing the paradigm of the indication of implantation from ‘bridging to heart transplantation’ to ‘destination therapy’, particularly in the large group of elderly patients with end-stage heart failure, these complex operations are no longer restricted to the small group of heart centres performing heart transplantation. This chapter provides a comprehensive review of modern cardiac anaesthesia in the contemporary world of quickly evolving cardiac surgery. The basics of anaesthesia management for the ‘cardiac’ patient are described and principles of extracorporeal circulation as well as diagnostic and treatment strategies of disturbances of the haemostatic system are highlighted. Pharmacological strategies to treat left- and right-heart failure and strategies for temporary mechanical support are outlined. Further areas of focus are the anaesthetic implications of modern less or minimally invasive procedures such as off-pump coronary artery bypass grafting and minimally invasive valve implantation/surgery and anaesthesia for implantation of ventricular assist devices and heart transplantation.
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20

Pierce, Frederick Clifton. History of Grafton, Worcester County, Massachusetts, from its Early Settlement by the Indians in 1647 to the Present Time, 1879 (A Heritage classic). Heritage Books, 2007.

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21

Benedict, William Addison. History of the town of Sutton, Massachusetts, from 1704 to 1876: Including Grafton until 1735 ; Millbury until 1813 ; and parts of Northbridge, Upton and Auburn (A Heritage classic). Heritage Books, Inc, 2000.

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22

N, Barton Harlan, and Geological Survey (U.S.), eds. Analytical results and sample locality map of heavy-mineral-concentrate and rock samples from the Mount Grafton Wilderness study area (NV-040-169), White Pine and Lincoln counties, Nevada. U.S. Dept. of the Interior, Geological Survey, 1986.

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23

Analytical results and sample locality map of heavy-mineral-concentrate and rock samples from the Mount Grafton Wilderness study area (NV-040-169), White Pine and Lincoln counties, Nevada. U.S. Dept. of the Interior, Geological Survey, 1986.

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24

N, Barton Harlan, and Geological Survey (U.S.), eds. Analytical results and sample locality map of heavy-mineral-concentrate and rock samples from the Mount Grafton Wilderness study area (NV-040-169), White Pine and Lincoln counties, Nevada. U.S. Dept. of the Interior, Geological Survey, 1986.

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25

Dead Again boxed set: Dead Aim - Johansen; D is for Deadbeat - Grafton; Dead Right - Robinson; The Dead of Jericho - Baxter; Dead Simple - James; Dead Cert - Francis; Back from the Dead - Petit; Mourn Not Your Dead - Crombie. Pan Books, 2007.

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26

The proceedings of a convention of delegates, from the states of Massachusetts, Connecticut and Rhode-Island, the counties of Cheshire and Grafton, in the state of New-Hampshire, and the county of Windham, in the state of Vermont: Convened at Hartford, in the state of Connecticut, December 15th, 1814. Printed and published by Wells and Lilly, 1985.

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