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Relevant bibliographies by topics / Granulomatous reaction

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Academic literature on the topic 'Granulomatous reaction'

Author: Grafiati

Published: 19 February 2023

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Journal articles on the topic "Granulomatous reaction"

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Wood, Andrew, Stuart A. Hamilton, William A. Wallace, and Asok Biswas. "Necrobiotic Granulomatous Tattoo Reaction." American Journal of Dermatopathology 36, no. 8 (August 2014): e152-e155. http://dx.doi.org/10.1097/dad.0000000000000032.

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NISHIBAYASHI, Hiroki, Yuji UEMATSU, Tomoaki TERADA, and Toru ITAKURA. "Pineal Germinoma With Granulomatous Reaction." Neurologia medico-chirurgica 45, no. 8 (2005): 415–17. http://dx.doi.org/10.2176/nmc.45.415.

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Hartel, Paul H., Konstantin Shilo, Mary Klassen-Fischer, Ronald C. Neafie, Irem H. Ozbudak, Jeffrey R. Galvin, and Teri J. Franks. "Granulomatous Reaction to Pneumocystis jirovecii." American Journal of Surgical Pathology 34, no. 5 (May 2010): 730–34. http://dx.doi.org/10.1097/pas.0b013e3181d9f16a.

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GONZÁLEZ-VELA, M. CARMEN, SUSANA ARMESTO, MARCOS A. GONZÁLEZ-LÓPEZ, J. HÉCTOR FERNÁNDEZ-LLACA, and J. FERNANDO VAL-BERNAL. "Perioral Granulomatous Reaction to Dermalive." Dermatologic Surgery 34, no. 7 (July 2008): 986–88. http://dx.doi.org/10.1097/00042728-200807000-00024.

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Gokdemir, Gonca, Asli Küçükünal, and Damlanur Sakiz. "Cutaneous Granulomatous Reaction from Mesotherapy." Dermatologic Surgery 35, no. 2 (February 2009): 291–93. http://dx.doi.org/10.1111/j.1524-4725.2008.01053.x.

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GONZLEZ-VELA, M. CARMEN, SUSANA ARMESTO, MARCOS A. GONZLEZ-LPEZ, J. HCTOR FERNNDEZ-LLACA, and J. FERNANDO VAL-BERNAL. "Perioral Granulomatous Reaction to Dermalive." Dermatologic Surgery 34, no. 7 (July 2008): 986–88. http://dx.doi.org/10.1111/j.1524-4725.2008.34194.x.

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Carlos, Giuliana, Rachael Anforth, Shaun Chou, and Pablo Fernandez-Peñas. "Dabrafenib-associated necrobiotic granulomatous reaction." Australasian Journal of Dermatology 55, no. 4 (November 2014): 306–8. http://dx.doi.org/10.1111/ajd.12226.

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Demaree, Elizabeth, David Cleaver, Lloyd Cleaver, Nathan Cleaver, and Jonathan Cleaver. "16622 Granulomatous reaction to microneedling." Journal of the American Academy of Dermatology 83, no. 6 (December 2020): AB67. http://dx.doi.org/10.1016/j.jaad.2020.06.357.

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Gulniflioglu, Gulen, Mehtat Unlu, Nurullah Yuceer, and Erdener Ozer. "Childhood intracranial germinoma with granulomatous reaction." Turkish Journal of Pathology 27, no. 1 (2011): 87. http://dx.doi.org/10.5146/tjpath.2010.01054.

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Trindade, Maria Ângela Bianconcini, H. Brandt, R. Teixeira, M. N. Sotto, and R. N. Fleury. "Leprosy with necrosis in granulomatous reaction." Brazilian Journal of Infectious Diseases 12, no. 5 (October 2008): 460. http://dx.doi.org/10.1590/s1413-86702008000500022.

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Dissertations / Theses on the topic "Granulomatous reaction"

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ALBA, CATHERINE. "Reaction cutanee granulomateuse apres la mise en place d'un pacemaker." Toulouse 3, 1993. http://www.theses.fr/1993TOU31070.

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Lewis, Colleen Jenna. "The Role of Nitric Oxide in Immune Responses to T cell-stimulating Polysaccharide Antigens; Implications for Chronic Granulomatous Disease." Case Western Reserve University School of Graduate Studies / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=case1274987560.

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Tavares, Marcos Soares. "Estudo caso-controle da região HLA de pacientes com Granulomatose com poliangeíte." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/5/5150/tde-01032017-134802/.

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Os alelos HLA-DPB1*04 e HLA-DRB1*15 estão fortemente associados à Granulomatose com poliangeíte (GPA). Neste estudo, analisamos se os pacientes brasileiros com diagnóstico de GPA apresentam uma base genética na região HLA. Conduzimos um estudo caso-controle, em que analisamos os alelos da região HLA classe I e II em 55 pacientes com diagnóstico de GPA, atendidos no ambulatório de Vasculites Pulmonares do Hospital das Clínicas da Faculdade de Medicina da Universidade de São Paulo, e comparamos com os resultados de 110 controles saudáveis. Comparamos também quatro diferentes apresentações clínicas da GPA e a positividade do anticorpo anticitoplasma de neutrófilos (ANCA) com os alelos da região HLA classe I e II. Foi também construída uma árvore de decisões, usando o algoritmo de CART, para a verificação da associação entre os alelos HLA e GPA. Como resultados, observamos que a GPA esteve fortemente associada à presença dos alelos DPB1*04 e DRB1*15 (p = 0,007, odds ratio [OR]: 2,9, 95% intervalo de confiança [IC]: 1,09-3,8; p = 0,006, OR: 2,87, 95% IC: 1,44-4,75, respectivamente) e não à presença do alelo DRB1*04. O alelo DRB1*13 esteve associado com proteção contra GPA (p = 0,042, OR: 0,42, 95% CI: 0,21-0,99). O alelo DPB1*04 esteve significativamente associado a GPA e ANCA-C positivo (OR: 5,47) e à presença de insuficiência renal aguda (p = 0,01037). Concluímos que houve uma interdependência significativa entre os alelos DPB1*0401, DPB1*0402, DRB1*13, C*2 e GPA. Na população estudada, quando o alelo DPB1*04 esteve presente em homozigose, o risco de GPA foi de 81%. Quando o alelo DPB1*0401 esteve ausente ou em heterozigose com o DPB1*0402, como o outro alelo, ou DPB1*0402 esteve em homozigose, o risco da GPA foi de 52,9%. No caso de ausência dos alelos DPB1*0401, DPB1*0402 e DRB1*13, a presença do alelo C*2 aumentou o risco da GPA para 62,5%. Finalmente, na ausência do alelo DPB1*0401 e DPB1*0402 e na presença do alelo DRB1*13, o risco de GPA diminuiu para 0%
The alleles HLA-DPB1*04 and HLA-DRB1*15 are strongly associated with granulomatosis with polyangiitis (GPA). In this study, we examined whether Brazilian patients with GPA had an HLA region genetic background. We conducted a case-control study, in which we analysed alleles of HLA region class I and II from 55 patients with GPA (at the Pulmonary Vasculitis Clinic of the University of São Paulo) and compared the results with those from 110 healthy controls. Comparisons were also performed for 4 different clinical presentations of GPA and anti-neutrophil cytoplasmic antibody (ANCA) positivity and the HLA class I and II region alleles. A tree model decision analysis was conducted using CART algorithm. Our results showed that GPA was strongly associated with alleles DPB1*04 and DRB1*15 (p = 0.007, odds ratio [OR]: 2.9, 95% confidence interval [CI]: 1.09-3.8; p = 0.006, OR: 2.87, 95% CI: 1.44-4.75, respectively) and not with the allele DRB1*04. DRB1*13 allele was associated with protection against GPA (p = 0.042, OR: 0.42, 95% CI: 0.21-0.99). DPB1*04 was significantly associated with GPA plus positive C-ANCA (OR: 5.47) and acute renal failure (p = 0.01037). We concluded that there was a significant interdependence among alleles and GPA. In our population, when allele DPB1*04 was presented in homozygous, the risk of GPA was 81%. When DPB1*0401 allele was absent or heterozygous with DPB1*0402 as the other allele, or DPB1*0402 was homozygous, the risk of disease was 52.9%. If DPB1*0401, DPB1*0402, and DRB1*13 were absent, the presence of C*2 increased the risk of GPA to 62.5%. Finally, in the absence of DPB1*0401 and DPB1*0402 and the presence of DRB1*13, the risk of GPA decreased to 0%

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Lakhoo, Deepna Govind. "Determining the validity of the mycobacterium polymerase chain reaction assay in histological samples showing granulomatous inflammation with a negative ziehl-neelsen stain." Thesis, 2016. http://hdl.handle.net/10539/21411.

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A research report submitted to the Faculty of Health Sciences, University of the Witwatersrand, in partial fulfilment for the Degree of Master of Medicine In the branch of Anatomical Pathology Johannesburg 2015
Background: Mycobacterium tuberculosis (Mtb) poses a major global health problem. According to the World Health Organization, South Africa is a country with one of the highest reported incidence rates of this disease. Key to overcoming this preventable and treatable disease lies in establishing a reliable and rapid diagnostic approach. Aims and Objectives: This study aims to investigate the validity of the mycobacterium polymerase chain reaction (PCR) assay applied to formalin-fixed, paraffin-embedded tissue in which the histology showed granulomatous inflammation with no demonstrable acid-fast bacilli. Methods: A retrospective, cross sectional and non-interventional study was conducted on 121 histopathology cases showing granulomatous inflammation with a negative Ziehl-Neelsen (ZN) stain. The mycobacterium PCR results obtained in these cases were compared against the results of mycobacterium culture obtained from a specimen derived from the same or related site as the biopsy. Results: The mean age of the study population was 35.3 years and the study cohort included 63 males and 58 females. The sensitivity of nested mycobacterium PCR (detecting the 133 base pair product of the heat shock protein 65 kilo Dalton gene), was 64.1% and the specificity was 68.2%. The positive and negative predictive values were 49% and 80% respectively. Twenty six of the 121 cases studied had a false positive result (21.5%). CONCLUSION: There are many factors that may influence the result of a PCR assay and the interpretation thereof. Some of these factors include the inability of the test to distinguish between live and dead bacilli, the high risk of carry over contamination, and the paucibacillary nature of certain samples with an unequal distribution of the few bacilli that may be present. Although the sensitivity and specificity of mycobacterium PCR on paucibacillary, formalin-fixed, paraffin embedded tissue is suboptimal, the interpretation of these results must be performed in conjunction with the overall clinical presentation of the patient.
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(9820148), Luke Moertel. "Microarray analysis of the Schistosoma japonicum transcriptome." Thesis, 2006. https://figshare.com/articles/thesis/Microarray_analysis_of_the_Schistosoma_japonicum_transcriptome/13423745.

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Schistosomiasis, a disease of humans caused by helminth parasites of the genus Schistosoma, kills 200 to 500 thousand people annually, endangering over 600 million people world-wide with 200 million people infected in 2003 [1, 2]. Three species of schistosome are primarily responsible for human infections, namely, Schistosoma haematobium, endemic to Africa, India, and the Middle East, S. mansoni, endemic to Africa / South America, and S. japonicum endemic to China and the Philippines [3]. The major pathological effects of schistosomiasis result from the deposition of parasite ova in human tissues and the subsequent intense granulomatous response induced by these eggs. There is a high priority to provide an effective sub-unit vaccine against these schistosome flukes, using proteins encoded by cDNAs expressed by the parasites at critical phases of their development. One technique that may expedite this gene identification is the use of microarrays for expression analysis. A 22,575 feature custom oligonucleotide DNA microarray designed from public domain databases of schistosome ESTs (Expressed Sequence Tags) was used to explore differential gene expression between the Philippine (SJP) and Chinese (SJC) strains of S. japonicum, and between males and females. It was found that 593, 664 and 426 probes were differentially expressed between the two geographical strains when mix sexed adults, male worms and female worms were compared respectively. Additionally, the study revealed that 1,163 male- and 1,016 female-associated probes were differentially expressed in SJP whereas 1,047 male- and 897 female-associated probes were differentially expressed in SJC [4]. Further to this, a detailed real time PCR expression study was used to explore the differential expression of eight genes of interest throughout the SJC life cycle, which showed that several of the genes were down-regulated in different life cycle stages. The study has greatly expanded previously published data of strain and gender-associated differential expression in S. japonicum. Further, the new data will provide a stepping stone for understanding the complexities of the biology, sexual differentiation, maturation, and development of human schistosomes, signaling new approaches for identifying novel intervention and diagnostic targets against schistosomiasis [4].

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Books on the topic "Granulomatous reaction"

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Heyns, Chris. Tuberculosis and parasitic infestations involving the urogenital system. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0006.

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Urogenital tuberculosis is caused by Mycobacterium tuberculosis, which evokes a granulomatous tissue reaction leading to caseous necrosis, fibrosis, and eventual calcification. It most commonly presents as cystitis with sterile pyuria but can show many other symptoms and signs requiring a high index of suspicion to make the diagnosis. Schistosomiasis (Bilharzia) affecting the urinary tract is caused by the flatworm Schistosoma haematobium. Humans are infested by contact with fresh water harbouring the intermediate snail host. Echinococcosis (hydatid disease), is caused by the tapeworm Echinococcus granulosis or multilocularis. Human infection results from close contact with the parasite host (usually dogs and sheep). Filariasis, caused by the roundworm Wuchereria bancrofti, is transmitted by mosquito bite

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Adam, Sheila, Sue Osborne, and John Welch. The immune system and the immunocompromised patient. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199696260.003.0013.

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This chapter discusses the physiology of the immune system and its related disorders in the critically ill patient. The causes, complications, and management of the immunocompromised patient is discussed at length, detailing the specific nursing management. It also discusses the management of autoimmune disease such as Wegener's granulomatosis and rheumatoid arthritis, and hypersensitivity reactions such as drug allergy and anaphylactic reaction. Cancer is explained in detail, including its complications, specific therapies such as stem cell transplant, and the role of the cancer patient within critical care.

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Book chapters on the topic "Granulomatous reaction"

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Dancygier, Henryk, and Peter Schirmacher. "Granulomatous Reaction." In Clinical Hepatology, 247–49. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-93842-2_27.

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Adams, D. O., and T. A. Hamilton. "The Activated Macrophage and Granulomatous Inflammation." In Cell Kinetics of the Inflammatory Reaction, 151–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73855-5_7.

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Rambaldi, Pier Francesco. "Follow-Up of Pulmonary Cancer After Surgery: Granulomatous Reaction on Scar." In Whole-Body FDG PET Imaging in Oncology, 293–96. Milano: Springer Milan, 2013. http://dx.doi.org/10.1007/978-88-470-5295-6_67.

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Mcdowell, Elizabeth M., and Theodore F. Beals. "Granulomatous reactions." In Biopsy Pathology of the Bronchi, 192–220. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4899-3398-0_6.

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Long, Kathleen A. "Granulomatous Drug Reactions." In Cutaneous Drug Eruptions, 389–94. London: Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-6729-7_36.

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Caplan, Avrom, Karolyn A. Wanat, Joseph C. English, and Misha Rosenbach. "Reactive Granulomatous Dermatitis (Interstitial Granulomatous Dermatitis, Palisaded Neutrophilic and Granulomatous Dermatitis, and Variants)." In New and Emerging Entities in Dermatology and Dermatopathology, 423–45. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-80027-7_30.

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Vaiphei, Kim. "Uncommon Types of Gastritis and Gastropathies Including Anti-parietal Cell, GVHD, Reactive and Granulomatous Gastritis." In Interpretation of Endoscopic Biopsy - Gastritis, Gastropathies and Beyond, 201–18. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-6026-9_19.

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Saffiotti, Umberto. "Lung Cancer Induction by Silica in Rats, but not in Mice and Hamsters: Species Differences in Epithelial and Granulomatous Reactions." In Environmental Hygiene II, 235–38. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-46712-7_54.

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Johnston, Ronald B. "Granulomatous Reaction Pattern." In Weedon's Skin Pathology Essentials, 133–61. Elsevier, 2012. http://dx.doi.org/10.1016/b978-0-7020-3574-6.50007-6.

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Johnston, Ronald B. "Granulomatous Reaction Pattern." In Weedon's Skin Pathology Essentials, 134–62. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-7020-6830-0.50007-4.

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Conference papers on the topic "Granulomatous reaction"

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Vieira, Daniella Serafin Couto, Bráulio Leal Fernandes, Érica Elaine Traebert Simezo, Amanda Amaro Pereira, and Mara Scheffer. "STANDARDIZATION OF THE LABORATORY RESEARCH PROTOCOL AND ANATOMOPATHOLOGICAL DIAGNOSIS OF BREAST GRANULOMATOUS INJURIES." In Scientifc papers of XXIII Brazilian Breast Congress - 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s1020.

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Introduction: Granulomatous breast lesions are multifactorial conditions, with clinical, mammographic, and ultrasound findings like those observed in cases of breast carcinoma. Histological evaluation can present key characteristics to define the lesion pattern. Although this entity is rarely reported in the literature, it is associated with inflammatory conditions such as Ductal Ectasia and foreign body reaction. However, it can also be associated with agents such as bacteria, fungi, and parasites. Furthermore, idiopathic causes and exclusion diagnosis, such as sarcoidosis, can be included in the etiology of the process. Objectives: To establish a correlation between the anatomopathological diagnosis and the laboratory investigation by culture for breast granulomatous lesions diagnosis, with validation of the sample’s analysis protocol. Methods: Samples were selected from 17 women treated at the Mastology Service of a Public Hospital in Brazil, with a history and physical examination that raised suspicion of breast granulomatous lesion and they had previous clinical indication of breast core biopsy. The collection of samples was guided by ultrasonography (USG). In turn, they were stored in a blood culture flask (BD BACTECTM) to perform culture tests by automation (VITEK2), bacterioscopy and fungi exams, and acid resistant bacillus (ARB) tests at the Laboratory of Clinical Analyses. Simultaneously, core biopsy samples, fixed in 10% buffered formaldehyde, were sent to a Laboratory of Pathological Anatomy, for a morphological evaluation and research of ARB, fungi, and other bacteria, using the Ziehl-Nielsen, Grocott, PAS and GRAM histochemical methods. Results: Of the 17 samples, 11 had a chronic inflammatory response pattern with a non-lobulocentric granulomatous reaction component and one of them had a lymphocytic mastitis pattern. The five patients with morphological pattern of lobulocentric granulomatous mastitis presented positive culture, four for Corynebacterium kroppenstedtil, and one for Staphylococcus hominis. Sensitivity, Specificity, Positive Predictive Value and Negative Predictive Value in this sample was 100%. Conclusion: The clinical and radiological aspects can bring difficulties that obscure the diagnostic and etiological interpretation of granulomatous lesions. Thus, the morphological details observed in the anatomical pathological examination and the use of the laboratory investigation protocol with standardization of histochemical reactions associated with the use of tools for microbiological diagnosis show increased sensitivity and specificity for the detection of specific etiologic agents in granulomatous mastitis.

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almogairen, sultan, and Ahmed Alhumaidi. "AB0110 RITUXIMAB INDUCED GRANULOMATOUS HEPATITIS WITH A SARCOIDOSIS LIKE REACTION: A BLINDED TRIAL IN MICE." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.2356.

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Fraga, Vanessa de Miranda, Luciano Gonçalves do Nascimento Júnior, Davi Rodrigues de Sousa, Eduardo Pimentel Carneiro Braga, Alex John Sinema Alvarez, and Isabella Wanderley de Queiroga Evangelista. "Arteritic bilateral anterior ischemic optical neuropathy: case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.310.

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Introduction: Arteritic anterior ischemic optic neuropathy (AAION) is infarction in anterior segment of optic nerve in which there is thrombotic occlusion of short posterior ciliary arteries due to granulomatous inflammation of giant cell arteritis (ACG). Case report: Male patient, 66 years old, complaining of loss of vision in left eye (LE) for 28 days, and in right eye (RE) for 8 days, accompanied by left hemicranial headache, pulsatile, irradiation from frontal to cervical region, of strong intensity, prevalent at night. Report of pain on chewing, chills, night sweats, neck pain and headache for 2 months. On examination, there was no light perception and bilateral non-reactive pupils. At simple retinography, LE with pale optical disc (OD), attenuation of retinal vascularization, atrophic and whitish areas; RE with OD with undefined borders, peripapillary hemorrhages and areas of retinal pallor. At fluorescent retinography; LE without arterial filling; CBC, discrete anisocytosis, thrombocytosis and microcytosis; ESR, 71mm/h in first hour; CRP, 113.6 mg/L. At, doppler USG of temporal arteries, significant increase in thickness of the myointimal layer, causing hemodynamic repercussions, with monophasic flow. Conclusion: Meeting 4 of 5 criteria of American College of Rheumatology, diagnosis of AAION was made. The condition was already irreversible at the time of examination.

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