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Journal articles on the topic 'Granulomatous response'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

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1

Holle, Julia U., Christin Dubrau, Karen Herlyn, et al. "Rituximab for refractory granulomatosis with polyangiitis (Wegener's granulomatosis): comparison of efficacy in granulomatous versus vasculitic manifestations." Annals of the Rheumatic Diseases 71, no. 3 (2011): 327–33. http://dx.doi.org/10.1136/ard.2011.153601.

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ObjectiveFirst, to investigate the overall efficacy and safety of rituximab (RTX) in refractory granulomatosis with polyangiitis (GPA) in a tertiary referral centre. Second, to compare the efficacy of RTX in granulomatous and vasculitic manifestations in GPA.Patients and methodsThis study comprised a retrospective, standardised data collection from all patients who received RTX for refractory Wegener's granulomatosis from 2002 to 2010. Patients were assessed by a standardised interdisciplinary diagnostic procedure (including ear, nose and throat and ophthalmology assessment, MRI, immunodiagnos

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2

Sheffield, E. A. "The granulomatous inflammatory response." Journal of Pathology 160, no. 1 (1990): 1–2. http://dx.doi.org/10.1002/path.1711600102.

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3

Granton, John T., Dean W. Chamberlain, and Robert H. Hyland. "Bronchocentric Granulomatosis: Clinical Course Illustrated by Serial Computerized Tomography of the Chest." Canadian Respiratory Journal 1, no. 4 (1994): 261–64. http://dx.doi.org/10.1155/1994/840952.

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Bronchocentric granulomatosis is a pathological process characterized by granulomatous inflammation in conducting airways. A case of bronchoccntric granulomatosis of unknown etiology in a previously well 24-year-old female is reported. The variable and often rluctuating course of this entity and its response to corticosteroids is demonstrated by serial computerized tomography nr the chest. The pathology, etiology and therapy ofbronchoccntric granulomatosis is reviewed.

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4

Starshinova, A. A., I. V. Kudryavtsev, A. A. Rubinstein, et al. "Autoimmune disorders in patients with granulomatosis diseases after COVID-19: T- and B-cells subsets function." Russian Journal of Infection and Immunity 14, no. 2 (2024): 251–66. http://dx.doi.org/10.15789/2220-7619-eou-16874.

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Sarcoidosis and tuberculosis are both granulomatous diseases that have many similarities, making the differential diagnosis of sarcoidosis and tuberculosis difficult, as well as leading to inappropriate treatment selection of both diseases. Autoimmune inflammation (AI) is one of the processes identified tuberculosis and sarcoidosis. Current evidences about the risk and clinical outcomes of COVID-19 infection in patient with sarcoidosis and M. tuberculosis co-infection are still not well understood. SARS-CoV-2 has direct damage to the epithelial cells of the respiratory system, and in-directly

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5

Gudmundsson, Gunnar, Martha M. Monick, and Gary W. Hunninghake. "Viral Infection Modulates Expression of Hypersensitivity Pneumonitis." Journal of Immunology 162, no. 12 (1999): 7397–401. http://dx.doi.org/10.4049/jimmunol.162.12.7397.

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Abstract Hypersensitivity pneumonitis (HP) is a granulomatous, inflammatory lung disease caused by inhalation of organic Ags, most commonly thermophilic actinomycetes that cause farmer’s lung disease. The early response to Ag is an increase in neutrophils in the lung, whereas the late response is a typical Th1-type granulomatous disease. Many patients who develop disease report a recent viral respiratory infection. These studies were undertaken to determine whether viruses can augment the inflammatory responses in HP. C57BL/6 mice were exposed to the thermophilic bacteria Saccharopolyspora rec

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Yamashita, T., and D. L. Boros. "IL-4 influences IL-2 production and granulomatous inflammation in murine schistosomiasis mansoni." Journal of Immunology 149, no. 11 (1992): 3659–64. http://dx.doi.org/10.4049/jimmunol.149.11.3659.

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Abstract In previous studies the dynamics of IL-2 production by splenic cells of Schistosoma mansoni infected mice was correlated with the intensity of hepatic granulomatous inflammation. To extend those observations, the present studies examined the role of IL-4 on the immune responsiveness of infected mice. The dynamics of IL-4 production by soluble egg Ag-stimulated splenic cells was similar to that of IL-2: minimal levels at the pre-oviposition or early worm egg deposition stages (4 to 6 wk) peak production coincident with maximal granulomatous response (8 wk) followed by a concurrent decl

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Husnan, Mujiburrahman, Yamsun Muhammad, Agung Wijayana Kamal, and Muhammad Fiqham. "Response to Adjuvant Therapy of Granulomatous Lymphadenitis: Evidence from High Burden Country." International Journal Of Medical Science And Clinical Research Studies 02, no. 12 (2022): 1458–62. https://doi.org/10.5281/zenodo.7417382.

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<strong>Introduction: </strong>A granulomatous lymphadenitis is a form of inflammation of the lymph nodes that are often found. Until now, effective treatment of granulomatous lymphadenitis is still inconsistent, namely between surgical intervention, antibiotics, or adjuvant therapy.   <strong>Objective: </strong>This study aims to determine the response to adjuvant therapy in patients with granulomatous lymphadenitis at RSUD Prof. Dr. Margono Soekarjo in 2019 – 2020.   <strong>Methods:</strong> The research was a descriptive observational study with a retrospect

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8

Fagbemi, B. O., N. Ø. Christensen, and O. O. Dipeolu. "Effects of Trypanosoma brucei and Babesia microti infections on the primary granulomatous reaction to Schistosoma eggs in mice." Laboratory Animals 21, no. 2 (1987): 121–24. http://dx.doi.org/10.1177/002367728702100207.

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Mouse infection with the blood protozoa Trypanosoma brucei suppressed significantly the frequency and intensity of the primary granulomatous inflammatory response to eggs of the blood flukes Schistosoma mansoni and S. bovis injected into the pulmonary microvasculature. In addition, the dynamics of the cellular infiltrate of the egg granuloma were strongly affected. It is suggested that the modulation of the granulomatous response is a result of impairment of the cell-mediated immunological responsiveness induced by T. brucei. Infection with Babesia microti did not induce similar effects on the

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9

Joseph, A. L., and D. L. Boros. "Tumor necrosis factor plays a role in Schistosoma mansoni egg-induced granulomatous inflammation." Journal of Immunology 151, no. 10 (1993): 5461–71. http://dx.doi.org/10.4049/jimmunol.151.10.5461.

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Abstract In murine schistosomiasis mansoni several proinflammatory lymphokines participate in the circumoval granulomatous inflammatory response. At the acute stage of the infection lymphokine secretion is maximal and coincides with the strong granulomatous reaction. With chronicity, Ag-elicited lymphokine production diminishes and the granulomatous inflammation is downmodulated. In this study the role of TNF in the granulomatous response was investigated. Macrophages isolated from vigorous liver granulomas of acute infection mice on LPS stimulation produced significantly more TNF than their c

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Alguacil-Garcia, G. F., J. Moreno-Requena, M. Martinez-Albadalejo, H. Hallal-Hachem, B. Gonzalez-Pina, and M. de Paco-Moya. "Idiopathic granulomatous vasculitis: response to immunosuppressive therapy." Journal of Clinical Pathology 48, no. 6 (1995): 579–82. http://dx.doi.org/10.1136/jcp.48.6.579.

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11

Jinkala, SreeRekha, Elancheran Muthalagan, and BhawanaAshok Badhe. "Granulomatous response in intracranial germinomas: Diagnostic problems." International Journal of Applied and Basic Medical Research 8, no. 1 (2018): 51. http://dx.doi.org/10.4103/ijabmr.ijabmr_157_17.

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12

Coyne, J. D., and N. Y. Haboubi. "Invasive carcinoma with a granulomatous stromal response." Histopathology 25, no. 4 (1994): 397–397. http://dx.doi.org/10.1111/j.1365-2559.1994.tb01366.x.

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13

Kunkel, S. L., R. M. Strieter, N. Lukacs, and S. W. Chensue. "Initiation and maintenance of the granulomatous response." Chest 103, no. 2 (1993): 135S—137. http://dx.doi.org/10.1378/chest.103.2.135s.

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Kunkel, Steven L., Robert M. Strieter, Nicholas Lukacs, and Stephen W. Chensue. "Initiation and Maintenance of the Granulomatous Response." Chest 103, no. 2 (1993): 135S—137S. http://dx.doi.org/10.1378/chest.103.2_supplement.135s.

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15

Braley-Mullen, H., G. C. Sharp, and M. Kyriakos. "Differential requirement for autoantibody-producing B cells for induction of lymphocytic versus granulomatous experimental autoimmune thyroiditis." Journal of Immunology 152, no. 1 (1994): 307–14. http://dx.doi.org/10.4049/jimmunol.152.1.307.

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Abstract Mouse thyroglobulin (MTg)-sensitized spleen cells activated in vitro with MTG transfer experimental autoimmune thyroiditis (EAT) in which the thyroid cellular infiltrate consists primarily of mononuclear cells (lymphocytic EAT). Addition of anti-IL2R antibody to cultures with MTg leads to activation of cells that induce granulomatous EAT, accompanied by high serum anti-MTg autoantibody responses, in recipient mice. CD4+ T cells are required to induce both forms of EAT; whether B cells and/or autoantibodies produced by MTg-sensitized B cells also contribute to disease severity or the t

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16

Coelho, Paulo Marcos Z., Pedro Raso, Rômulo Teixeira de Mello, and Nivaldo H. Toppa. "Schistosoma mansoni in mice: modulation of granulomatous response after reinfection and chemotherapeutic treatment." Revista da Sociedade Brasileira de Medicina Tropical 27, no. 3 (1994): 119–25. http://dx.doi.org/10.1590/s0037-86821994000300001.

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Mice previously infected with Schistosoma mansoni, and cured by specific treatment (400mg/kg oxamniquine, p. o.) in the chronic phase of the disease, were reinfected 20 days after treatment to assess their capacityfor modulation ofthe granulomatous response. Histopathologic examination of the animals ' liver, at 60 days after reinfection, evidenced the presence of typical granulomas of the chronic phase in most animals. This infer that the capacity for modulation of the granulomatous response had been maintained, thus preventing a new acute phase of the disease. Conversely, a group of previous

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17

Haley, Patrick J. "Mechanisms of Granulomatous Lung Disease from Inhaled Beryllium: The Role of Antigenicity in Granuloma Formation." Toxicologic Pathology 19, no. 4_part_1 (1991): 514–25. http://dx.doi.org/10.1177/0192623391019004-117.

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Granulomatous lung disease is a debilitating and sometimes fatal condition encountered in humans, for which the cellular and molecular mechanisms are poorly understood. Two patterns of granulomatous lung disease are recognized; foreign-body reactions and immune-mediated granulomas. Beryllium inhalation by humans results, in a small number of exposed individuals, in a chronic, granulomatous, immune-mediated pulmonary disease (chronic beryllium lung disease, CBD). Animal models used to study CBD have demonstrated significant species differences in the pathologic response to beryllium. While rats

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18

Bonek, Krzysztof, Eliza Brożek-Mądry, Jakub Wroński, et al. "Combination Treatment of Locoregionally Aggressive Granulomatosis with Polyangiitis and Cranial Base Infiltration." Brain Sciences 13, no. 8 (2023): 1140. http://dx.doi.org/10.3390/brainsci13081140.

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Objectives: To present a personalized approach in three cases of treatment-resistant, locoregionally aggressive forms of cANCA-positive granulomatosis with polyangiitis (GPA) and skull base involvement. Methods: Three patients with GPA and skull base involvement were described alongside a critical review of the current literature. Results: All presented patients suffered from GPA with an inflammatory tumor at the skull base, alongside cerebellopontine angle involvement, cranial nerve palsies, cerebellar disorders, concomitant hearing loss, and severe otalgia. Symptoms were associated with prog

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19

Egyan, S. P., and V. N. Gaponova. "Ophthalmoform of granulomatous meningoencephalomyelitis in dogs." Legal regulation in veterinary medicine, no. 4 (January 6, 2023): 96–99. http://dx.doi.org/10.52419/issn2782-6252.2022.4.96.

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Granulomatous meningoencephalomyelitis (GME) is an inflammatory disease of the central nervous system in animals characterized by focal or disseminated granulomatous lesions of the brain and spinal cord. The local form can affect the intersection of the optic nerves. Inflammation of this site leads to the ophthalmoform of granulomatous encephalomyelitis, which is characterized by the progressive occurrence of blindness due to optic neuritis, may be accompanied by uveitis. The aim of the work is to study the effect of vaccination on the development of the ophthalmoform of granulomatous meningoe

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20

Auger, Manon, Ann T. Moriarty, Rodolfo Laucirica, et al. "Granulomatous Inflammation—An Underestimated Cause of False-Positive Diagnoses in Lung Fine-Needle Aspirates: Observations From the College of American Pathologists Nongynecologic Cytopathology Interlaboratory Comparison Program." Archives of Pathology & Laboratory Medicine 134, no. 12 (2010): 1793–96. http://dx.doi.org/10.5858/2009-0491-cpr2.1.

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Abstract Context—The false-positive rate for fine-needle aspirates of the lung has been cited as less than 1% for granulomatous inflammation, comprising one of the known causes of false-positive diagnoses. Objective—To determine the rate of false-positive diagnoses of granulomatous inflammation for lung fine-needle aspirates by assessing the false-positive response rate in the context of the College of American Pathologists Nongynecologic Cytopathology Interlaboratory Comparison Program. Design—We performed a retrospective review of 1092 participant responses for lung fine-needle aspirate chal

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Takaki, Kevin K., Francisco J. Roca, Gabriele Schramm, et al. "Tumor Necrosis Factor and Schistosoma mansoni egg antigen omega-1 shape distinct aspects of the early egg-induced granulomatous response." PLOS Neglected Tropical Diseases 15, no. 1 (2021): e0008814. http://dx.doi.org/10.1371/journal.pntd.0008814.

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Infections by schistosomes result in granulomatous lesions around parasite eggs entrapped within the host tissues. The host and parasite determinants of the Schistosoma mansoni egg-induced granulomatous response are areas of active investigation. Some studies in mice implicate Tumor Necrosis Factor (TNF) produced in response to the infection whereas others fail to find a role for it. In addition, in the mouse model, the S. mansoni secreted egg antigen omega-1 is found to induce granulomas but the underlying mechanism remains unknown. We have recently developed the zebrafish larva as a model to

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Trifunovic, Gordana, Goran Plavec, Ilija Tomic, Lidija Popovic, and Dusan Stefanovic. "Allergic granulomatous angiitis." Vojnosanitetski pregled 61, no. 3 (2004): 321–25. http://dx.doi.org/10.2298/vsp0403321t.

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Allergic granulomatous angiitis (AGA) - Churg-Strauss syndrome, is a rare autoimmune disease characterized by three distinct clinical phases prodromal, eosinophilic, and vasculitic, and most of respiratory symptoms and signs begin in the first two phases of the disease. Two female patients of different age, who fulfilled the diagnostic criteria for AGA, and were in different phases and with the different duration of the disease are presented. The first patient (24 years of age) was admitted to the hospital due to aggravation of asthma, heart failure, and polyneuropathy. The second one (45 year

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Akter, Sonia, Karina Rahman, Md Mahfuzul Momen, and Deb Prosad Paul. "A Study on Outcome of Wide Local Excision in Chronic Granulomatous Mastitis." Journal of Enam Medical College 10, no. 3 (2022): 174–78. http://dx.doi.org/10.3329/jemc.v10i3.59359.

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Background: Granulomatous mastitis can be divided into idiopathic granulomatous mastitis and granulomatous mastitis occurring as a rare secondary complication of a great variety of other conditions such as tuberculosis and other infections, sarcoidosis and granulomatosis with polyangitis. Idiopathic granulomatous mastitis (IGM) is an uncommon benign chronic inflammatory disease which can clinically and radiographically mimic abscess or breast cancer. Definitive diagnosis was made by histopathology and exclusion of an identifying etiology. Optimal treatment has not been yet established. Objecti

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P, Nitheash, and Malini P. "The Granulomatous Lesions of Oral Tissue - A Brief Review." International Journal of Research and Review 10, no. 1 (2023): 436–43. http://dx.doi.org/10.52403/ijrr.20230150.

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Granulomatous inflammation represents a unique form of the chronic inflammatory response. Their occurrence in oral soft & hard tissues is an uncommon but represents a diagnostic dilemma because of the wide variety of possible etiologic factors. Recognition is important because of the limited number of possible conditions that causes it and for specific treatment and outcome of the disease. They often have systemic manifestations that affect organs throughout the body and share similar histogenesis. Therefore, an extensive clinical, microscopic and laboratory evaluation is required to ident

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Mathew, R. C., and D. L. Boros. "Regulation of granulomatous inflammation in murine schistosomiasis. III. Recruitment of antigen-specific I-J+ T suppressor cells of the granulomatous response by I-J+ soluble suppressor factor." Journal of Immunology 136, no. 3 (1986): 1093–99. http://dx.doi.org/10.4049/jimmunol.136.3.1093.

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Abstract Down-modulation of the schistosome egg-induced granulomatous response involves various interacting subsets of T suppressor (TS) lymphocytes. In the present study the inductive phase of the process of modulation was analyzed. A soluble, I-J+ granuloma TS cell recruiting factor (Gr-TSRF) derived from spleen cells of chronically infected mice is described. This factor eluted from immunoabsorbent columns coupled with anti-I-Jk alloantisera induced the recruitment and expansion of antigen-specific I-J+ TS cells from a TS precursor cell population in the spleens of acutely infected mice. Th

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Guidry, Tera V., Robert L. Hunter, and Jeffrey K. Actor. "CD3+ cells transfer the hypersensitive granulomatous response to mycobacterial glycolipid trehalose 6,6′-dimycolate in mice." Microbiology 152, no. 12 (2006): 3765–75. http://dx.doi.org/10.1099/mic.0.29290-0.

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The granulomatous response is the characteristic histological feature of Mycobacterium tuberculosis infection that is essential for organism containment. Trehalose 6,6-dimycolate (TDM), a cell-wall glycolipid present on most mycobacterial species, has been implicated in the pathogenesis of M. tuberculosis infection. TDM has potent immunoregulatory and inflammatory properties, and can be used to model granulomatous reactions that mimic, in part, pathology caused during active infection. This study examined the hypersensitive granulomatous response, focusing on cellular responses specific to TDM

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27

Junek, Mats, Lynn Fussner, and Michael Walsh. "Clinician Views and Clinic-Pathologic Correlations Concerning the Prevalence and Impact of Granulomas on Diagnosis, Management, and Outcomes of ANCA-Associated Vasculitis: An International Survey." Journal of Rheumatology 52, Suppl 2 (2025): 76.2–76. https://doi.org/10.3899/jrheum.2025-0314.66.

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ObjectivesGranulomatous inflammation occurs in ANCA-associated vasculitis (AAV), however, which manifestations are attributable to granulomas are unclear. We sought to understand clinician beliefs concerning which manifestations of AAV are attributable to granulomas and if the presence of granulomas informs diagnostic, prognostic and treatment decisions.MethodsWe conducted an international survey of physicians who care for individuals with AAV with the following domains: clinical experience; the extent to which the presence of granulomatous manifestations impact diagnosis, prognosis, and treat

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Strieter, Robert, Stephen Chensue, Theodore Standiford, Joseph Lynch, Mark Rolfe, and Steven Kunkel. "Host Response in Granulomatous Inflammation of the Lung." Seminars in Respiratory and Critical Care Medicine 13, no. 03 (1992): 149–57. http://dx.doi.org/10.1055/s-2007-1006267.

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29

FLANNERY, MICHAEL T., EVA QUIROZ, L. SHANE GRUNDY, and STEPHEN BRANTLEY. "Pneumocystis carinii Pneumonia With an Atypical Granulomatous Response." Southern Medical Journal 89, no. 4 (1996): 409–10. http://dx.doi.org/10.1097/00007611-199604000-00011.

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30

Ish-Shalom, Z., I. Misselevich, D. G. Mendes, and J. H. Boss. "Foreign Body Granulomatous Response to Particulate Bony Debris." Veterinary and Comparative Orthopaedics and Traumatology 09, no. 03 (1996): 119–25. http://dx.doi.org/10.1055/s-0038-1632516.

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SummaryArchival material was retrieved in order to histologically assess the body’s response to micron-sized, necrotic bony particles. Specimens were obtained from (1) human bone grafts in a subcutaneous or muscular pouch of athymic nude mice, (2) a massive bone allograft replacing a patient’s humerus, (3) rabbits’ healing tibial cortical bone defects littered with finely dispersed bony particles, (4) periprosthetic tissues of patient’s aseptically loosened artificial joints and (5) interfacial membranes of intramedullary nails used for fixation of patients’ fractured long bones. Necrotic bony

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Anderson, Lyndal, Peter Russell, Chris Halloway, and Patricia Bannatyne. "Uterine plexiform leiomyomatosis with an intrinsic granulomatous response." Pathology 38, no. 2 (2006): 179–81. http://dx.doi.org/10.1080/00313020600562011.

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COYNE, J., and N. Y. HABOUBI. "Micro-invasive breast carcinoma with granulomatous stromal response." Histopathology 20, no. 2 (1992): 184–85. http://dx.doi.org/10.1111/j.1365-2559.1992.tb00953.x.

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33

Oberman, Harold A. "Invasive Carcinoma of the Breast with Granulomatous Response." American Journal of Clinical Pathology 88, no. 6 (1987): 718–21. http://dx.doi.org/10.1093/ajcp/88.6.718.

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Denisova, O. A., N. V. Baranovskaya, G. E. Chernogoryuk, and T. P. Kalacheva. "Sarcoidosis as granulomatous response to environmental geochemical factors." Prospect and protection of mineral resources, no. 1 (2023): 21–25. http://dx.doi.org/10.53085/0034-026x_2023_01_21.

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Alba, Marco, J. Jennette, and Ronald Falk. "Pathogenesis of ANCA-Associated Pulmonary Vasculitis." Seminars in Respiratory and Critical Care Medicine 39, no. 04 (2018): 413–24. http://dx.doi.org/10.1055/s-0038-1673386.

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AbstractAntineutrophil cytoplasmic antibodies (ANCAs) are autoantibodies specific for antigens located in the cytoplasmic granules of neutrophils and lysosomes of monocytes. ANCAs are associated with a spectrum of necrotizing vasculitis that includes granulomatosis with polyangiitis, microscopic polyangiitis, and eosinophilic granulomatosis with polyangiitis. Pulmonary vasculitis and related extravascular inflammation and fibrosis are frequent components of ANCA vasculitis. In this review, we detail the factors that have been associated with the origin of the ANCA autoimmune response and summa

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Cywińska, A., K. Czumińska, and A. Schollenberger. "Granulomatous inflammation duringHeligmosomoides polygyrusprimary infections in FVB mice." Journal of Helminthology 78, no. 1 (2004): 17–24. http://dx.doi.org/10.1079/joh2003205.

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AbstractHost responses to primary infections withHeligmosomoides polygyruswere studied in fast responding FVB mice (H-2q). Pathological changes in the intestinal mucosa, mesenteric lymph nodes and spleen were examined. Features of the fast response were typical: low effectiveness of infection and limiting of parasite survival and egg production, with worm expulsion occurring about 60 days post-infection. The intestinal inflammatory response involved infiltration by different cells into the intestinal mucosa and granulomata formation. As is typical for intestinal nematode infection enteropathy,

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Janssen, Ulf, Shirin Naderi, and Kerstin Amann. "Idiopathic granulomatous interstitial nephritis and isolated renal sarcoidosis: Two diagnoses of exclusion." SAGE Open Medicine 9 (January 2021): 205031212110384. http://dx.doi.org/10.1177/20503121211038470.

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Granulomatous interstitial nephritis is a rare finding in renal biopsy caused by drugs, infections, and inflammatory or autoimmune diseases. Idiopathic cases account for 18% of granulomatous interstitial nephritis in native kidneys. Sarcoidosis and drugs are the most common causes of granulomatous interstitial nephritis in Western countries, while in India tuberculosis prevails. Few cases of renal sarcoidosis without extrarenal involvement, that is, isolated renal sarcoidosis, have been reported. The diagnostic criteria of isolated renal sarcoidosis remain, however, unclear. Extrarenal sarcoid

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Hattori, Keita, Yuri Teramachi, Yoshinori Kobayashi, et al. "A Case of Effective Mepolizumab Induction Therapy for Severe Eosinophilic Granulomatosis with Polyangiitis Diagnosed by Eosinophilic Cholecystitis and Interstitial Nephritis." Case Reports in Rheumatology 2021 (June 19, 2021): 1–5. http://dx.doi.org/10.1155/2021/6678893.

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A 66-year-old man with a history of bronchial asthma and sinusitis was admitted with cholecystitis and peripheral neuropathy. The histopathological findings of the gallbladder revealed necrotic vasculitis and granulomatous inflammation with marked eosinophilic infiltration. Kidney biopsy also showed marked eosinophilic infiltration in the tubulointerstitial area and eosinophilic tubulitis. He was diagnosed with eosinophilic granulomatosis with polyangiitis (EGPA) and treated with corticosteroids. However, he showed no response. Therefore, he was administered mepolizumab 300 mg, which resulted

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Arellano, Kari, James C. Mosley, and Donald C. Moore. "Case Report of Ipilimumab-Induced Diffuse, Nonnecrotizing Granulomatous Lymphadenitis and Granulomatous Vasculitis." Journal of Pharmacy Practice 31, no. 2 (2017): 227–29. http://dx.doi.org/10.1177/0897190017699762.

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Ipilimumab is indicated for the treatment of melanoma in both the metastatic and adjuvant setting. Ipilimumab inhibits cytotoxic T-lymphocyte antigen 4, leading to the augmentation of T-cell activity and an antitumor immune system response. The side effect profile of ipilimumab consists of autoimmune-like events such as dermatitis, colitis, and thyroiditis. These immune-related adverse events can be serious, often resulting in the need for systemic immunosuppression with corticosteroids. We present a case of diffuse, nonnecrotizing granulomatous lymphadenitis and granulomatous vasculitis in a

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Victor, Hugo Solis-Reyna, Valeria Espinosa-Rosales Alicia, Josimar Ucan-Gamboa Dioney, and Alfonso Vidales-Lopez Raul. "Resection of aRuptured Epidermoid Cyst with Limberg Flap: Case Report." International Journal of Medical Science and Clinical Research Studies 04, no. 10 (2024): 1866–70. https://doi.org/10.5281/zenodo.13939344.

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Epidermoid cysts, also known as epidermal cysts, keratin cysts, epithelial cysts, or sebaceous cysts (although this term is no longer widely used), are benign lesions frequently seen in clinical practice. These cysts arise from epidermal cells trapped beneath the skin's surface. The most common complication is cyst rupture, which can lead to significant inflammation and even a granulomatous response. This article presents the clinical case of a male patient with a ruptured epidermoid cyst that triggered a granulomatous reaction, treated with surgical excision and reconstruction using a Limberg

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Ponce, Lorena Narváez, Judith Carrió, Enrique R. Soriano, Carlos D. Santos, Patricia M. Imamura, and Luis J. Catoggio. "Diabetes Insípida como forma de presentación de Granulomatosis de Wegener." Revista de la Facultad de Ciencias Médicas de Córdoba 66, no. 1 (2009): 31–35. http://dx.doi.org/10.31053/1853.0605.v66.n1.23538.

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Wegener´s granulomatosis is a granulomatous necrotizing vasculitis which predominantly affects the respiratory tract, kidney, and less frequently other organs such as the nervous system. The latter may occur in up to 54% of cases and when it does it is more frequently of the peripheral nerves. We present a 19 year old woman who commenced her disease with involvement of respiratory sinuses, lungs and kidney and who developed central insipid diabetes (CID) at onset. The CID persisted in spite of adequate response of the other organs and systems with immunosuppresor treatment. The development of

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Breitschwerdt, Edward B., Kasandra R. Blann, Martha E. Stebbins, et al. "Clinicopathological Abnormalities and Treatment Response in 24 Dogs Seroreactive to Bartonella vinsonii (berkhoffii) Antigens." Journal of the American Animal Hospital Association 40, no. 2 (2004): 92–101. http://dx.doi.org/10.5326/0400092.

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Bartonella vinsonii (B. vinsonii) subspecies berkhoffii is a recently recognized cause of endocarditis, myocarditis, and granulomatous disease in dogs. In an effort to elucidate other potential disease manifestations, the case records of 24 dogs that were seroreactive to B. vinsonii (berkhoffii) antigens were studied retrospectively. Diagnoses included immune-mediated hemolytic anemia, neutrophilic or granulomatous meningoencephalitis, neutrophilic polyarthritis, cutaneous vasculitis, and uveitis. Repeated B. vinsonii (berkhoffii) antibody titers became negative after treatment. This study ind

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Legallet, Claire, Kelley Thieman Mankin, Kathy Spaulding, and Joanne Mansell. "Granulomatous Inflammatory Response to a Microchip Implanted in a Dog for Eight Years." Journal of the American Animal Hospital Association 53, no. 4 (2017): 227–29. http://dx.doi.org/10.5326/jaaha-ms-6418.

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ABSTRACT An 8 yr old neutered male springer spaniel dog was referred to Texas A&M University, College of Veterinary Medicine for a large, firm, fixed mass, located in the dorsal cervical tissue. The dog was otherwise healthy and had undergone microchip implantation approximately 8 yr prior. Radiographs, ultrasound, and microchip scanner confirmed the presence of a microchip within the mass. The microchip and associated mass were surgically excised, and histopathologic examination revealed granulomatous inflammation surrounding a cracked microchip. This case represents the first report of a

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Aubin, Guillaume Ghislain, Da Silva Grâce Ada, Yoshinobu Eishi, et al. "Immune discrepancies during in vitro granuloma formation in response to Cutibacterium (formerly Propionibacterium ) acnes infection." Anaerobe 48 (December 1, 2017): 172–76. https://doi.org/10.1016/j.anaerobe.2017.08.014.

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Cutibacterium (formerly Propionibacterium) acnes is involved in chronic/low-grade pathologies such as sarcoidosis or prosthetic joint infection (PJI). In these diseases, granulomatous structures are frequently observed. In this study, we induced a physiological granulomatous reaction in response to different wellcharacterized clinical C. acnes isolates in order to investigate the cellular process during granuloma formation. Three C. acnes isolates selected according to their origin (PJI, sarcoidosis and acne) were typed by MLST. All C. acnes isolates generated granulomatous structures in our e

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BASIKA, TATIANA, NATALIA MUÑOZ, CECILIA CASARAVILLA, et al. "Phagocyte-specific S100 proteins in the local response to theEchinococcus granulosuslarva." Parasitology 139, no. 2 (2012): 271–83. http://dx.doi.org/10.1017/s003118201100179x.

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SUMMARYInfection by larvalEchinococcus granulosusis usually characterized by tight inflammatory control. However, various degrees of chronic granulomatous inflammation are also observed, reaching a high point in infection of cattle by the most prevalent parasite strain worldwide, which is not well adapted to this host species. In this context, epithelioid and multinucleated giant macrophages surround the parasite, and the secreted products of these cells often associate with the larval wall. The phagocyte-specific S100 proteins, S100A8, S100A9 and S100A12, are important non-conventionally secr

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Bharadwaj, S., J. T. Anim, F. Ebrahim, and A. Aldahham. "Granulomatous Inflammatory Response in a Case of Typhoid Fever." Medical Principles and Practice 18, no. 3 (2009): 239–41. http://dx.doi.org/10.1159/000204357.

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Baig, Abdul Mannan. "Granulomatous amoebic encephalitis: ghost response of an immunocompromised host?" Journal of Medical Microbiology 63, no. 12 (2014): 1763–66. http://dx.doi.org/10.1099/jmm.0.081315-0.

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Kamal, Natasha, Bryan F. Curtin, Anna Strongin, et al. "Mo1889 - The Response to Vedolizumab in Chronic Granulomatous Disease." Gastroenterology 154, no. 6 (2018): S—840. http://dx.doi.org/10.1016/s0016-5085(18)32853-1.

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Blennerhassett, J. B. "Acute, chronic and granulomatous inflammation the coordinated host response." Pathology 23 (1991): 11. http://dx.doi.org/10.1016/s0031-3025(16)36193-1.

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Takenoshita, Hideo, and Toshiyuki Yamamoto. "Granulomatous isotopic response possibly to herpes zoster in childhood." Journal of Dermatology 41, no. 7 (2014): 651–52. http://dx.doi.org/10.1111/1346-8138.12519.

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