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Journal articles on the topic 'GRIM'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 April 2022

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1

Higashi, Tatsuichiro, Yuko Iwasaki, Yasuo Ohnishi, and Sueharu Horinouchi. "A-Factor and Phosphate Depletion Signals Are Transmitted to the Grixazone Biosynthesis Genes via the Pathway-Specific Transcriptional Activator GriR." Journal of Bacteriology 189, no. 9 (2007): 3515–24. http://dx.doi.org/10.1128/jb.00055-07.

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ABSTRACT Grixazone (GX), which is a diffusible yellow pigment containing a phenoxazinone chromophore, is one of the secondary metabolites under the control of A-factor (2-isocapryloyl-3R-hydroxymethyl-γ-butyrolactone) in Streptomyces griseus. GX production is also induced by phosphate starvation. The whole biosynthesis gene cluster for GX was cloned and characterized. The gene cluster consisting of 13 genes contained six transcriptional units, griT, griSR, griR, griAB, griCDEFG, and griJIH. During cultivation in a phosphate-depleted medium, the six promoters were activated in the order (i) griR, (ii) griC and griJ, and (iii) griT, griS, and griA. Disruption of griR, which encodes a SARP family transcriptional regulator, abolished the transcriptional activation of all other genes in the cluster. In addition, ectopic expression of griR from a constitutively active promoter resulted in GX overproduction even in the absence of AdpA, a key transcriptional activator in the A-factor regulatory cascade, and in the presence of phosphate at a high concentration. GriR monomers bound direct repeat sequences in the griC and griJ promoters in a cooperative manner. Therefore, the early active genes (griCDEFG and griJIH), all of which, except for griG (which encodes a transporter-like protein), encode the GX biosynthesis enzymes, were directly activated by GriR. The transcription of griR was greatly reduced in the presence of phosphate at a high concentration and was hardly detected in the absence of AdpA. These findings showed that both A-factor and phosphate depletion signals were required for griR transcription and both signals were transmitted to the GX biosynthesis genes solely via the griR promoter.

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2

Lehane, Mike. "Grim death." Nursing Standard 11, no. 7 (1996): 18. http://dx.doi.org/10.7748/ns.11.7.18.s38.

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3

Cook, Rosemary. "Grim reality." Nursing Standard 12, no. 52 (1998): 24. http://dx.doi.org/10.7748/ns.12.52.24.s41.

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4

Nutt, David. "Grim repercussions." New Scientist 226, no. 3025 (2015): 24–25. http://dx.doi.org/10.1016/s0262-4079(15)30560-1.

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5

Wallich, Paul. "Grim Statistics." Scientific American 269, no. 5 (1993): 14. http://dx.doi.org/10.1038/scientificamerican1193-14a.

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6

Unferth, Deb Olin. "Grim Innovation." American Book Review 32, no. 4 (2011): 16. http://dx.doi.org/10.1353/abr.2011.0092.

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7

Svoboda, Terese. "Grim Sleeper." Baffler 22 (March 2013): 54–55. http://dx.doi.org/10.1162/bflr_a_00139.

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8

Kruse, G. "Grim prospects." Manufacturing Engineer 81, no. 6 (2002): 260–61. http://dx.doi.org/10.1049/me:20020604.

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9

Yam, Philip. "Grim Expectations." Scientific American 264, no. 3 (1991): 33–34. http://dx.doi.org/10.1038/scientificamerican0391-33.

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10

Buchan, James. "Grim determination." Nursing Standard 3, no. 49 (1989): 22. http://dx.doi.org/10.7748/ns.3.49.22.s33.

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11

Lenci, Edoardo, Luca Cantini, Federica Pecci, et al. "The Gustave Roussy Immune (GRIm)-Score Variation Is an Early-on-Treatment Biomarker of Outcome in Advanced Non-Small Cell Lung Cancer (NSCLC) Patients Treated with First-Line Pembrolizumab." Journal of Clinical Medicine 10, no. 5 (2021): 1005. http://dx.doi.org/10.3390/jcm10051005.

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Background: The Gustave Roussy Immune (GRIm)-Score takes into account neutrophil-to-lymphocyte ratio (NLR), serum albumin concentration and lactate dehydrogenase (LDH) and its prognostic value has been investigated in patients treated with immune check-point inhibitors (ICIs). To further assess the prognostic and predictive value of baseline GRIm-Score (GRImT0) in advanced non-small cell lung cancer (aNSCLC) patients, we separately investigated two cohorts of patients treated with first-line pembrolizumab or chemotherapy. We also investigated whether GRIm-Score at 45 days since treatment initiation (GRImT1) and GRIm-Score difference between the two timepoints may better predict clinical outcomes (GRImΔ = GRImT0 − GRImT1). Methods: We retrospectively evaluated 222 aNSCLC patients: 135 treated with pembrolizumab and 87 treated with chemotherapy as the first-line regimen. NLR, serum albumin and LDH concentrations were assessed at T0 and at T1. According to the GRIm-Score, patients were assigned 1 point if they had NLR > 6, LDH > upper limit normal or albumin < 3.5 g/dL. Patients with a GRIm-Score < 2 were considered as having a low Score. Results: In both cohorts, no difference in terms of overall survival (OS) between patients with low and high GRImT0 was found. Otherwise, median OS and progression free survival (PFS) of the low GRImT1 group were significantly longer than those of the high GRImT1 group in pembrolizumab-treated patients, but not in the CHT cohort (pembrolizumab cohort: low vs. high; median OS not reached vs. 9.2 months, p = 0.004; median PFS 10.8 vs. 2.3 months, p = 0.002). Patients receiving pembrolizumab with stable/positive GRImΔ had better OS (median OS not reached vs. 12.0 months, p < 0.001), PFS (median PFS 20.6 vs. 2.6 months, p < 0.001) and objective response rate (58.2% vs. 7.6%, p = 0.003) compared to patients with negative GRImΔ. Conclusion: Our data shown that GRImT1 and GRImΔ are more reliable peripheral blood biomarkers of outcome compared to GRImT0 in aNSCLC patients treated with pembrolizumab and might represent useful biomarkers to drive clinical decisions in this setting.

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12

Reigber, C., G. Balmino, H. Müller, W. Bosch, and B. Moynot. "GRIM gravity model improvement using LAGEOS (GRIM3-L1)." Journal of Geophysical Research 90, B11 (1985): 9285. http://dx.doi.org/10.1029/jb090ib11p09285.

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13

McMahon, Brendan. "Grim fairy tales." Nursing Standard 13, no. 29 (1999): 24. http://dx.doi.org/10.7748/ns.13.29.24.s38.

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14

Kazez, Jean. "The Grim Reader." Philosophers' Magazine, no. 62 (2013): 112–13. http://dx.doi.org/10.5840/tpm201362105.

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15

Dibb, Paul, and Peter Prince. "Indonesia’s grim outlook." Orbis 45, no. 4 (2001): 621–36. http://dx.doi.org/10.1016/s0030-4387(01)00100-4.

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16

Ray, L. B. "The Grim RIPper." Science Signaling 2, no. 80 (2009): ec246-ec246. http://dx.doi.org/10.1126/scisignal.280ec246.

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17

Cohen, Eliot A. "Lincoln's Grim Realism." Survival 51, no. 5 (2009): 155–62. http://dx.doi.org/10.1080/00396330903309915.

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18

Harris, Peter J. "The grim REAPper." Manufacturing Engineer 70, no. 7 (1991): 10. http://dx.doi.org/10.1049/me:19910149.

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19

Brown, Nicholas J. L., and James A. J. Heathers. "The GRIM Test." Social Psychological and Personality Science 8, no. 4 (2016): 363–69. http://dx.doi.org/10.1177/1948550616673876.

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We present a simple mathematical technique that we call granularity-related inconsistency of means (GRIM) for verifying the summary statistics of research reports in psychology. This technique evaluates whether the reported means of integer data such as Likert-type scales are consistent with the given sample size and number of items. We tested this technique with a sample of 260 recent empirical articles in leading journals. Of the articles that we could test with the GRIM technique ( N = 71), around half ( N = 36) appeared to contain at least one inconsistent mean, and more than 20% ( N = 16) contained multiple such inconsistencies. We requested the data sets corresponding to 21 of these articles, receiving positive responses in 9 cases. We confirmed the presence of at least one reporting error in all cases, with three articles requiring extensive corrections. The implications for the reliability and replicability of empirical psychology are discussed.

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20

Pancake, Breece D'J. "A Grim Wind." Appalachian Heritage 40, no. 3 (2012): 30–34. http://dx.doi.org/10.1353/aph.2012.0086.

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21

DIGGINS, KRISTENE. "A Grim Prognosis." Journal of Christian Nursing 22, no. 3 (2005): 36. http://dx.doi.org/10.1097/01.cnj.0000262312.92275.cf.

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22

Lawley, Paul. ""The Grim Journey"." Samuel Beckett Today / Aujourd'hui 11, no. 1 (2018): 253–66. http://dx.doi.org/10.1163/18757405-01101032.

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23

Smithers, Gregory D. "A Grim Present." American Book Review 37, no. 3 (2016): 11. http://dx.doi.org/10.1353/abr.2016.0035.

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24

Dutil, Patrice A. "Federalism's grim reapers?" Canadian Public Administration/Administration publique du Canada 43, no. 4 (2000): 479–83. http://dx.doi.org/10.1111/j.1754-7121.2000.tb01156.x.

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25

Yang, Yang, Yanyan Sun, Laiyang Cheng, et al. "GRIM-19, a gene associated with retinoid-interferon-induced mortality, affects endometrial receptivity and embryo implantation." Reproduction, Fertility and Development 29, no. 7 (2017): 1447. http://dx.doi.org/10.1071/rd16104.

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GRIM-19 is associated with apoptosis, abnormal proliferation, immune tolerance and malignant transformation, and it also plays an important role in early embryonic development. Although the homologous deletion of GRIM-19 causes embryonic lethality in mice, the precise role of GRIM-19 in embryo implantation has not been elucidated. Here we show that GRIM-19 plays an important role in endometrial receptivity and embryo implantation. Day 1 to Day 6 pregnant mouse uteri were collected. Immunohistochemistry studies revealed the presence of GRIM-19 on the luminal epithelium and glandular epithelium throughout the implantation period in pregnant mice. The protein and mRNA levels of GRIM-19 were markedly decreased on Day 4 of pregnancy in pregnant mice, but there was no change in GRIM-19 levels in a group of pseudopregnant mice. Overexpression of GRIM-19 decreased the adhesion rate of RL95–2–BeWo co-cultured spheroids and increased apoptosis. Furthermore, STAT3 and IL-11 mRNA and protein levels were reduced by overexpressing GRIM-19, but protein and mRNA levels of TNF-α were increased. These findings indicate the involvement of GRIM-19 in the embryo implantation process by regulating adhesion, apoptosis and immune tolerance.

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26

Cheng, Yong, Hong-yan Zhang, Ying Zhou, Feng Tao, and Yong-hua Yu. "Decreased expression of GRIM-19 and its association with high-risk HPV infection in cervical squamous intraepithelial neoplasias and cancer." Clinical & Investigative Medicine 37, no. 2 (2014): 77. http://dx.doi.org/10.25011/cim.v37i2.21089.

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Purpose: GRIM-19 has been shown to be down-regulated in cervical cancers. This study investigated the expression of GRIM-19 in cervical intra-epithelial neoplasias and its association with high-risk human papillomavirus (HR-HPV) infection. Methods: The expression of GRIM-19 was assessed in cervical exfoliated cells and cervical intra-epithelial neoplasia tissues by immunohistochemistry, and the level of GRIM-19 was also evaluated by Western blotting using cervical exfoliated cells. HR-HPV infection of cervical exfoliated cells was detected by HC II. Results: GRIM-19 is predominantly expressed in the cytoplasm of the middle layer of normal cervical epithelial cells, whereas the surface layer cells of the normal cervix showed no GRIM-19 expression. The expression of GRIM-19 gradually decreased from atypical squamous cells of undetermined significance (ASCUS), low-grade squamous intraepithelial lesion (LSIL) and high-grade squamous intraepithelial lesion (HSIL) to squamous cell carcinoma (SCC); a pattern which was also observed in cervical intra-epithelial neoplasias tissues. The reduced expression of GRIM-19 was correlated with HR-HPV infection. Conclusion: GRIM-19 may regulate the differentiation of normal cervical tissue, and a decrease in GRIM-19 may be the result of HR-HPV infection, which in turn leads to the malignant transformation of the cells.

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27

Fitzpatrick, Mike. "A grim future foretold." British Journal of General Practice 57, no. 544 (2007): 927. http://dx.doi.org/10.3399/096016407782318035.

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28

Dwyer, D. E., R. Howard, J. Downie, and A. L. Cunningham. "The “Grim Reaper” campaign." Medical Journal of Australia 149, no. 1 (1988): 49–50. http://dx.doi.org/10.5694/j.1326-5377.1988.tb120487.x.

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29

Rosser, B. Simon. "Reaping the grim results." Medical Journal of Australia 148, no. 7 (1988): 368. http://dx.doi.org/10.5694/j.1326-5377.1988.tb133742.x.

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30

Nainggolan, Lisa. "A grim global picture." Cancer Nursing Practice 2, no. 4 (2003): 5. http://dx.doi.org/10.7748/cnp.2.4.5.s6.

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31

Mejia, R. "HUMAN RIGHTS: Grim Statistics." Science 313, no. 5785 (2006): 288–90. http://dx.doi.org/10.1126/science.313.5785.288.

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32

Hoth, J. Jason. "A Grim Picture, Indeed*." Critical Care Medicine 41, no. 3 (2013): 923–24. http://dx.doi.org/10.1097/ccm.0b013e31827c0631.

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33

Jackson, David, Christopher Bowers, Nathalie Phillips, Nesha Rampersad, and Sarah Leibowitz. "Denying the Grim Reaper." Journal of the American Academy of Physician Assistants 30, no. 12 (2017): 1. http://dx.doi.org/10.1097/01.jaa.0000526985.26267.d4.

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34

Rozzini, Renzo. "The COVID Grim Reaper." Journal of the American Medical Directors Association 21, no. 7 (2020): 994. http://dx.doi.org/10.1016/j.jamda.2020.05.001.

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35

Huang, Guochang, Hao Lu, Aijun Hao, et al. "GRIM-19, a Cell Death Regulatory Protein, Is Essential for Assembly and Function of Mitochondrial Complex I." Molecular and Cellular Biology 24, no. 19 (2004): 8447–56. http://dx.doi.org/10.1128/mcb.24.19.8447-8456.2004.

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ABSTRACT Mitochondria play essential roles in cellular energy production via the oxidative phosphorylation system (OXPHOS) consisting of five multiprotein complexes and also in the initiation of apoptosis. NADH:ubiquinone oxidoreductase (complex I) is the largest complex that catalyzes the first step of electron transfer in the OXPHOS system. GRIM-19 was originally identified as a nuclear protein with apoptotic nature in interferon (IFN)- and all-trans-retinoic acid (RA)-induced tumor cells. To reveal its biological role, we generated mice deficient in GRIM-19 by gene targeting. Homologous deletion of GRIM-19 causes embryonic lethality at embryonic day 9.5. GRIM-19−/− blastocysts show retarded growth in vitro and, strikingly, display abnormal mitochondrial structure, morphology, and cellular distribution. We reexamined the cellular localization of GRIM-19 in various cell types and found its primary localization in the mitochondria. Furthermore, GRIM-19 is detected in the native form of mitochondrial complex I. Finally, we show that elimination of GRIM-19 destroys the assembly and electron transfer activity of complex I and also influences the other complexes in the mitochondrial respiratory chain. Our result demonstrates that GRIM-19, a gene product with a specific role in IFN-RA-induced cell death, is a functional component of mitochondrial complex I and is essential for early embryonic development.

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36

Vucic, Domagoj, William J. Kaiser, and Lois K. Miller. "Inhibitor of Apoptosis Proteins Physically Interact with and Block Apoptosis Induced by Drosophila Proteins HID and GRIM." Molecular and Cellular Biology 18, no. 6 (1998): 3300–3309. http://dx.doi.org/10.1128/mcb.18.6.3300.

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ABSTRACT Reaper (RPR), HID, and GRIM activate apoptosis in cells programmed to die during Drosophila development. We have previously shown that transient overexpression of RPR in the lepidopteran SF-21 cell line induces apoptosis and that members of the inhibitor of apoptosis (IAP) family of antiapoptotic proteins can inhibit RPR-induced apoptosis and physically interact with RPR through their BIR motifs (D. Vucic, W. J. Kaiser, A. J. Harvey, and L. K. Miller, Proc. Natl. Acad. Sci. USA 94:10183–10188, 1997). In this study, we found that transient overexpression of HID and GRIM also induced apoptosis in the SF-21 cell line. Baculovirus and Drosophila IAPs blocked HID- and GRIM-induced apoptosis and also physically interacted with them through the BIR motifs of the IAPs. The region of sequence similarity shared by RPR, HID, and GRIM, the N-terminal 14 amino acids of each protein, was required for the induction of apoptosis by HID and its binding to IAPs. When stably overexpressed by fusion to an unrelated, nonapoptotic polypeptide, the N-terminal 37 amino acids of HID and GRIM were sufficient to induce apoptosis and confer IAP binding activity. However, GRIM was more complex than HID since the C-terminal 124 amino acids of GRIM retained apoptosis-inducing and IAP binding activity, suggesting the presence of two independent apoptotic motifs within GRIM. Coexpression of IAPs with HID stabilized HID levels and resulted in the accumulation of HID in punctate perinuclear locations which coincided with IAP localization. The physical interaction of IAPs with RPR, HID, and GRIM provides a common molecular mechanism for IAP inhibition of theseDrosophila proapoptotic proteins.

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37

KARLSTRÖM, PETTER, TERESA CERRATTO-PARGMAN, HENRIK LINDSTRÖM, and OLA KNUTSSON. "Tool mediation in Focus on Form activities: case studies in a grammar-exploring environment." ReCALL 19, no. 1 (2007): 39–56. http://dx.doi.org/10.1017/s0958344007000419.

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We present two case studies of two different pedagogical tasks in a Computer Assisted Language Learning environment called Grim. The main design principle in Grim is to support ‘Focus on Form’ in second language pedagogy. Grim contains several language technology-based features for exploring linguistic forms (static, rule-based and statistical), intended to be used while writing. Our question is, in what ways does Grim support Focus on Form in actual classroom use. We have explored this question within sociocultural theory, emphasizing tool mediation and how tools shape the learner’s activity. The first case concerns a text-reconstruction exercise in which students worked in a pair within the Grim environment. The second case was conducted with another group of students, who engaged in collaborative revision of texts, written in advance by one of the students, in student pairs. In both studies, students were instructed and encouraged to use the different features of Grim. Data was collected by recording dialogue during the sessions with Grim. Our results show how learners put the features of Grim into use in their writing tasks. In some instances, the program was used creatively, in combination with external tools such as the users’ own dictionaries, knowledge of other languages, or teachers. In other instances, we note that Grim was used for error correction, rather than as a language resource. The learners’ activities are thus transformed by their use of the program, from the tasks of revision and text-reconstruction into error correction. The application shapes the activity, in conjunction with the pedagogical tasks. We argue for studying the activities of students with CALL tools, in order to find out in detail how tasks and technology concur in use and what view on language and pedagogy they mediate.

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38

Rai, Rajesh, and Bal Krishan Shrivastva. "Effect of grass on soil reinforcement and shear strength." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 3 (2012): 127–30. http://dx.doi.org/10.1680/grim.10.00001.

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39

Dong, Phan Huy, Kimitoshi Hayano, Hidetoshi Takahashi, and Yoriko Morikawa. "Mechanical characteristics of lean-mixed cement-treated granular soil." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 3 (2012): 131–46. http://dx.doi.org/10.1680/grim.10.00004.

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40

Kumar, Santhosh, Benny Mathews Abraham, Asuri Sridharan, and Babu T. Jose. "Improvement of shear strength of loose sandy soils by grouting." Proceedings of the Institution of Civil Engineers - Ground Improvement 166, no. 1 (2013): 3–8. http://dx.doi.org/10.1680/grim.10.00015.

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41

McCarthy, Michael J., Laszlo J. Csetenyi, Anisha Sachdeva, and Ravindra K. Dhir. "Fly ash influences on sulfate-heave in lime-stabilised soils." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 3 (2012): 147–58. http://dx.doi.org/10.1680/grim.10.00016.

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42

Juneja, Ashish, and Bashir Ahmed Mir. "Behaviour of clay reinforced by sand compaction pile with smear." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 2 (2012): 111–24. http://dx.doi.org/10.1680/grim.10.00020.

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43

Mitrani, Helen, Gopal S. P. Madabhushi, and Felipe A. Villalobos. "Discussion: Cementation liquefaction remediation for existing buildings." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 3 (2012): 183–84. http://dx.doi.org/10.1680/grim.10.00028.

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44

Kitazume, Masaki. "Ground improvements in Haneda/Tokyo International Airport expansion project." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 2 (2012): 77–86. http://dx.doi.org/10.1680/grim.10.00031.

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45

Indraratna, Buddhima, Cholachat Rujikiatkamjorn, Richard Kelly, and Henk Buys. "Soft soil foundation improved by vacuum and surcharge loading." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 2 (2012): 87–96. http://dx.doi.org/10.1680/grim.10.00032.

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46

Raju, Venu R., and Jonathan Daramalinggam. "Ground improvement: principles and applications in Asia." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 2 (2012): 65–76. http://dx.doi.org/10.1680/grim.10.00033.

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47

Mitrani, Helen, and S. P. Gopal Madabhushi. "Geomembrane containment walls for liquefaction remediation." Proceedings of the Institution of Civil Engineers - Ground Improvement 166, no. 1 (2013): 9–20. http://dx.doi.org/10.1680/grim.10.00035.

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48

Murty, Varudu Ramana, and Padavala Hari Krishna. "Piled footings – an alternative foundation system in expansive soils." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 3 (2012): 159–66. http://dx.doi.org/10.1680/grim.10.00039.

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49

Halabian, Amir M., Ibrahim Naeemifar, and S. Hamid Hashemolhosseini. "Numerical analysis of vertically loaded rammed aggregate piers and pier groups." Proceedings of the Institution of Civil Engineers - Ground Improvement 165, no. 3 (2012): 167–81. http://dx.doi.org/10.1680/grim.10.00041.

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50

Becker, Timothy S., Joseph E. Dove, and Patricia M. Dove. "Experimental creep behaviour and modelling of silicified sand." Proceedings of the Institution of Civil Engineers - Ground Improvement 166, no. 2 (2013): 115–24. http://dx.doi.org/10.1680/grim.11.00001.

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