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Books on the topic 'Growth hormone – Adverse effects'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 February 2022

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1

Wambera, Katherine Martina. Potential memory enhancing effects of peripheral growth hormone administration. Ottawa: National Library of Canada, 1992.

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2

Lindgren, Ann Christin. Prader-Willi syndrome: Diagnosis and effects of growth hormone treatment. Stockholm: Repro Print AB, 1998.

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3

Hormone toxicity in the newborn. Berlin: Springer-Verlag, 1990.

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4

Toxic bodies: Hormone disruptors and the legacy of DES. New Haven [Conn.]: Yale University Press, 2010.

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5

The greatest experiment ever performed on women: Exploding the estrogen myth. New York: Seven Stories Press, 2009.

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6

Nerve growth factor and pain. New York: Nova Science Publishers, 2010.

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7

Julie, Taguchi, and Formby Bent, eds. Sex, lies, and menopause: The shocking truth about hormone replacement therapy. New York: Morrow, 2003.

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8

Mahmud, Iffat. Water, sanitation, hygiene, and nutrition in Bangladesh: Can building toilets affect children's growth? Washington, DC: International Bank for Reconstruction and Development/The World Bank, 2015.

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9

Meletis, Chris D. The male menopause controversy: Arguments, affects, and treatments of andropause. Westport, Conn: Praeger Publishers, 2009.

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10

The greatest experiment ever performed on women: Exploding the estrogen myth. New York: Hyperion, 2003.

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11

Seaman, Barbara. The greatest experiment ever performed on women: Exploding the estrogen myth. New York: Hyperion, 2003.

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12

Humans, IARC Working Group on the Evaluation of Carcinogenic Risks to. Hormonal contraception and post-menopausal hormonal therapy. Lyon, France: IARC, 1999.

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13

Cosgrove, Christine. Normal at Any Cost. New York: Penguin USA, Inc., 2009.

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14

1948-, Cohen Susan, ed. Normal at any cost: Tall girls, short boys, and the medical industry's quest to manipulate height. New York: Jeremy P. Tarcher/Penguin, 2009.

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15

Harmey, Judith H. VEGF and cancer. Georgetown, Tex: Landes Bioscience/Eurekah.com, 2004.

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16

Th, Smit Sibinga C., Das P. C, and Fratantoni Joseph C, eds. Alternative approaches to human blood resources in clinical practice: Proceedings of the Twenty-Second International Symposium on Blood Transfusion, Groningen 1997. Dordrecht: Kluwer Academic, 1998.

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17

Advanced Course on Steroid Enzymes and Cancer (9th 2008 Erice, Italy). Steroid enzymes and cancer. Malden, MA: Wiley-Blackwell, 2009.

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18

Organization, World Health, International Agency for Research on Cancer, and ebrary Inc, eds. Combined estrogen-progestogen contraceptives and combined estrogen-progestogen menopausal therapy. Lyon, France: International Agency for Research on Cancer, 2007.

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19

International Symposium on the Effects of Therapy on the Biology and Kinetics of the Surviving Tumor (1989 Vancouver, B.C.). Effects of therapy on biology and kinetics of the residual tumor: Proceedings of an International Symposium on the Effects of Therapy on the Biology and Kinetics of the Surviving Tumor, held in Vancouver, British Columbia, Canada, February 15-18, 1989. Edited by Ragaz J. 1945-. New York: Wiley-Liss, 1990.

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20

Nitrous oxide emissions from rice fields: Past, present, and future. Hauppauge, NY: Nova Science Publishers, 2009.

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21

Novel Aspects of Pthrp Physiopathology. Nova Science Pub Inc, 2007.

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22

C Diaz, Eva, Celeste C Finnerty, and David N. Herndon. Severe Burn Injuries and Their Long-Term Implications. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0016.

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Burn injury is notable for the degree and duration of pathophysiological alterations that it induces. Burn triggers profound changes in metabolism, immune function, and endocrine function, leading to a host of negative effects, including catabolism of muscle and bone and insulin resistance. These changes may persist or evolve for years after the injury has occurred, delaying recovery. This chapter discusses all of these consequences of burn injury, along with other adverse outcomes, specifically growth delay in children and hypertrophic scarring. Particular attention is placed on what is known about the mechanisms underlying each of these pathological changes and, in some cases, current practice in their management. A description is also provided of some of the pharmacologic (i.e. oxandrolone and recombinant human growth hormone) and non-pharmacologic (i.e. exercise therapy) approaches that hold promise in the treatment of burn injury and its consequences.

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23

(Editor), Andrea Giustina, Alberto Angeli (Editor), Ernesto Canalis (Editor), and Filippo Manelli (Editor), eds. Glucocorticoid-Induced Osteoporosis (Frontiers of Hormone Research). S. Karger Publishers (USA), 2002.

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24

(Editor), Anders Juul, and Jens O. L. Jørgensen (Editor), eds. Growth Hormone in Adults: Physiological and Clinical Aspects. Cambridge University Press, 1996.

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25

Anders, Juul, and Jørgensen Jens O. L, eds. Growth hormone in adults: Physiological and clinical aspects. Cambridge [England]: Cambridge University Press, 1996.

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26

Woodhouse, Linda June. Effects of recombinant human growth hormone (rhGH) on impairment and functional capacity in adults with growth hormone deficiency (GHD). 2005.

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27

Sherer, Todd Thomas. Changes in thyroid hormone receptor MRNA expression and the effects of thyroid hormone in the developing rat cerebellum. 1991.

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28

Erythropoietin: Human Production, Potential Uses and Adverse Effects. Nova Science Pub Inc, 2013.

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29

Effects of acute bouts of exercise on growth hormone secretion in females. 1989.

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30

Effects of acute bouts of exercise on growth hormone secretion in females. 1986.

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31

J, Wald Nicholas, Baron John, and Great Britain. Dept. of Health., eds. Smoking and hormone-related disorders. Oxford: Oxford University Press, 1990.

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32

Eiholzer, Urs. Prader-Willi Syndrome: Effects of Human Growth Hormone Treatment (Endocrine Development, Vol. 3). S Karger Pub, 2001.

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33

1928-, Mann Ronald D., ed. Hormone replacement therapy and breast cancer risk. Carnforth, Lancs, UK: Parthenon Pub. Group, 1992.

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34

Cardiovascular Disease and Steroid Hormone Contraception. World Health Organization, 1998.

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35

I.B. Helva*, M. Aksit, and S. Yalcin. Effects of monochromatic light on growth performance, welfare and hormone levels in broiler chickens. Verlag Eugen Ulmer, 2019. http://dx.doi.org/10.1399/eps.2019.273.

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36

Lutwin, Becker, ed. Hormone-related malignant tumors. Berlin: Springer-Verlag, 1990.

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37

Langston, Nancy. Toxic Bodies: Hormone Disruptors and the Legacy of DES. Yale University Press, 2010.

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38

Toxic Bodies: Hormone Disruptors and the Legacy of Des. Yale University Press, 2011.

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39

Langston, Nancy. Toxic Bodies: Hormone Disruptors and the Legacy of Des. Yale University Press, 2010.

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40

G, Crosignani P., and International Symposium on Women's Health in Menopause (1993 : Milan, Italy), eds. Women's health in menopause: Behaviour, cancer, cardiovascular disease, hormone replacement therapy. Dordrecht: Kluwer Academic Publishers, 1994.

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41

Seaman, Barbara. The Greatest Experiment Ever Performed on Women: Exploding the Estrogen Myth. Hyperion, 2003.

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42

R, Genazzani Andrea, and International Menopause Society, eds. Hormone replacement therapy and cancer: The currrent status of research and practice. New York: Parthenon Pub. Group, 2002.

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43

Genazzani, Andrea R. Hormone Replacement Therapy and Cancer: The Current Status of Research and Practice. Taylor & Francis Group, 2020.

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44

Genazzani, Andrea R. Hormone Replacement Therapy and Cancer: The Current Status of Research and Practice. Taylor & Francis Group, 2020.

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45

Fawcett, Erin Eileen. The skeletal effects of a growth hormone-derived peptide (AOD9604) in the aged rat model of postmenopausal osteoporosis. 2004.

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46

Feifel, David *. Central injections of growth hormone-releasing factor in the rat effects feeding differentially across light and dark photoperiods. 1988.

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47

1958-, Parhar Ishwar S., ed. Gonadotropin-releasing hormone: Molecules and receptors. Amsterdam: Elsevier, 2002.

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48

Lopez-Arvizu, Carmen, Carmel Bogle, and Harolyn M. E. Belcher. Neurobiology of Fetal Alcohol Spectrum Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0179.

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Prenatal exposure to ethanol can result in a wide range of clinical presentations that are grouped under the term “Fetal Alcohol Spectrum Disorders” (FASD). The direct cellular teratogenic effects of ethanol on fetal neurodevelopment include damage to cell survival, proliferation, and migration mechanisms. Dysregulation of neurotransmission and alteration of genetic transcription have also been implicated in the neurotoxic effects of prenatal ethanol exposure. These deleterious events lead to brain volume reduction, corpus callosum dysgenesis, cerebellar, and other neuroanatomical anomalies that have been observed in individuals with FASD. Beyond direct ethanol-induced insults, the impact that ethanol has on maternal nutrition, metabolism, hormonal regulation, and placental physiology also adversely effects fetal development. The complex interactions between numerous neurobiological and psychosocial mechanisms that hinder optimal fetal neurodevelopment are reflected by the heterogeneous clinical presentation of FASD, including impaired growth, dysmorphic facial features, and cognitive and behavioral disorders.

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49

D. Shao, Y. Hu, Q. Wang, Y. Shen, X. Zhao, H.B. Tong*, and S.R. Shi*. The Growth hormone receptor is involved in the effects of dietary microencapsulated sodium butyrate on growth performance and intestinal morphology of yellow-feathered broilers. Verlag Eugen Ulmer, 2016. http://dx.doi.org/10.1399/eps.2016.167.

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50

Piero, Fioretti, ed. Postmenopausal hormonal therapy: Benefits and risks. New York: Raven Press, 1987.

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