Academic literature on the topic 'GSTPi'

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Journal articles on the topic "GSTPi"

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Paret, Aurelie, Delphine Rolland, Vincent Ribrag, Bertrand Coiffier, and Catherine Thieblemont. "Inhibition of GSTpi Activity Increases the Sensitivity of MCL Cell Lines to Cisplatin, Cytarabin and Bortezomib, but Not Doxorubicin." Blood 106, no. 11 (2005): 2806. http://dx.doi.org/10.1182/blood.v106.11.2806.2806.

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Abstract Preliminary data suggest drug resistance plays a major role in mantle cell lymphoma (MCL) and is linked to the aberrant expression of molecules such as Glutathion S-Transferase pi (GST pi) that catalyzes the nuclear conjugation of a broad variety of reactive electrophiles to the glutathion. We investigated in vitro the effect of the inhibition of GSTpi activity on the chemosensitivity of MCL cell lines by inhibiting the nuclear transport of GSTpi with Agaricus bisporus leptine (ABL). Methods. Four MCL cell lines (Granta, NCEB, REC and UPN1) were analyzed for GSTP1 transcript expressio
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Veropotvelyan, P. N., I. S. Tsehmistrenko, N. P. Veropotvelyan, N. S. Rusak, and M. S. Pivnev. "The importance and significance of gene polymorphisms in preeclampsia." HEALTH OF WOMAN, no. 8(114) (October 30, 2016): 45–49. http://dx.doi.org/10.15574/hw.2016.114.45.

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Was to conduct a systematic review of data on the relationship between polymorphisms genes of detoxification system and development of preeclampsia (РЕ). Рresents the main genes of detoxification system (GSTPI, GSTМI, GSTТI, GРХI, ЕРНХI, SOD-2, SOD-3, CYPIAL, MTHЕR, MTR) and their functions. Of interest is the possibility of calculating the individual risk of PE based on the results about the presence of a combination of different polymorphisms in the genotype of the female. Question about early diagnosis of РЕ remains controversial and not fully understood. It is necessary to conduct further
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Yang, Yang, Fangyuan Yin, Qiyun Hang, et al. "Regulation of Endothelial Permeability by Glutathione S-Transferase Pi Against Actin Polymerization." Cellular Physiology and Biochemistry 45, no. 1 (2018): 406–18. http://dx.doi.org/10.1159/000486918.

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Background/Aims: Inflammation-induced injury of the endothelial barrier occurs in several pathological conditions, including atherosclerosis, ischemia, and sepsis. Endothelial cytoskeleton rearrangement is an important pathological mechanism by which inflammatory stimulation triggers an increase of vascular endothelial permeability. However, the mechanism maintaining endothelial cell barrier function against inflammatory stress is not fully understood. Glutathione S-transferase pi (GSTpi) exists in various types of cells and protects them against different stresses. In our previous study, GSTp
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Ruzzo, Annamaria, Francesco Graziano, Kazuyuki Kawakami, et al. "Pharmacogenetic Profiling and Clinical Outcome of Patients With Advanced Gastric Cancer Treated With Palliative Chemotherapy." Journal of Clinical Oncology 24, no. 12 (2006): 1883–91. http://dx.doi.org/10.1200/jco.2005.04.8322.

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Purpose To investigate whether polymorphisms with putative influence on fluorouracil/cisplatin activity are associated with clinical outcomes of patients with advanced gastric cancer (AGC). Patients and Methods Peripheral blood samples from 175 prospectively enrolled AGC patients treated with fluorouracil/cisplatin palliative chemotherapy were used for genotyping 13 polymorphisms in nine genes (TS, MTHFR, XPD, ERCC1, XRCC1, XRCC3, GSTPI, GSTTI, GSTMI). Genotypes were correlated to response and survival. Results The overall response rate was 41%, the median progression-free survival (PFS) was 2
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Zhang, Guowei, Xing Zeng, Caixia Li, et al. "Inhibition of Urinary Bladder Carcinogenesis by Aqueous Extract of Sclerotia of Polyporus umbellatus Fries and Polyporus Polysaccharide." American Journal of Chinese Medicine 39, no. 01 (2011): 135–44. http://dx.doi.org/10.1142/s0192415x11008701.

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The study aimed to evaluate inhibition effect of sclerotia of Polyporus umbellatus Fries aqueous extract (SPUE) and polyporus polysaccharide (PPS) on bladder cancer, then to measure their effect on mRNA expression of glutathione S-transferase π (GSTPi) and NAD(P)H:quinone oxidoreductase 1 (NQO1) in female Fischer-344 rats model. The model rats were induced by N-butyl-N-(4-hydroxybutyl)-nitrosamine (BBN) for a period of 8 weeks and saccharin for 12 weeks. SPUE (50 mg/kg, 250 mg/kg, 500 mg/kg) and PPS (28 mg/kg) were orally administrated to the model rats during the whole study. Compared to the
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Gerardi, Daniel G., Mirela Tinucci-Costa, Ana Carolina T. Silveira, and Juliana V. Moro. "Expression of P-glycoprotein, multidrug resistance-associated protein, glutathione-S-transferase pi and p53 in canine transmissible venereal tumor." Pesquisa Veterinária Brasileira 34, no. 1 (2014): 71–78. http://dx.doi.org/10.1590/s0100-736x2014000100012.

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The overexpression of proteins P-glycoprotein (P-gp), multidrug resistance-associated protein (MRP1), mutant p53, and the enzyme glutathione-S-transferase (GSTpi) are related to resistance to chemotherapy in neoplasms. This study evaluated the expression of these markers by immunohistochemistry in two groups of canine TVT, without history of prior chemotherapy (TVT1, n=9) and in TVTs presented unsatisfactory clinical response to vincristine sulfate (TVT2, n=5). The percentage of specimens positively stained for P-gp, MRP1, GSTpi and p53 were, respectively 88.8%, 0%, 44.5% and 22.2% in TVT1 and
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SUNDBERG, Kathrin, Bengt JERNSTRÖM, and Stellan SWEDMARK. "Studies on the differential inhibition of glutathione conjugate formation of (+)-anti-benzo[a]pyrene 7,8-dihydrodiol 9,10-epoxide and 1-chloro-2,4-dinitrobenzene in V79 Chinese hamster cells." Biochemical Journal 349, no. 3 (2000): 693–96. http://dx.doi.org/10.1042/bj3490693.

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V79 Chinese hamster cells have previously been shown to lack the capacity to detoxify the mutagenic and carcinogenic compound (+)-anti-benzo[a]pyrene 7,8-dihydrodiol 9,10-epoxide [(+)-anti-BPDE] by Pi class glutathione transferase (GSTPi)-catalysed conjugation with GSH, although these cells contain such an enzyme [Romert, Dock, Jenssen and Jernström (1989) Carcinogenesis 10, 1701–1707; Swedmark, Romert, Morgenstern and Jenssen (1992) Carcinogenesis 13, 1719–1723; Swedmark and Jenssen (1994) Gene 139, 251–256]. Previous findings also indicate that these results do not depend on an inactive GSTP
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Singal, R., L. Navarro, E. Gordian, et al. "Aberrant promoter methylation in serum of prostate cancer patients and controls." Journal of Clinical Oncology 27, no. 15_suppl (2009): e16046-e16046. http://dx.doi.org/10.1200/jco.2009.27.15_suppl.e16046.

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e16046 Background: The current screening method for prostate cancer (PC), which is based on prostate specific antigen (PSA) level lacks specificity and sensitivity. Previous studies have reported that increased levels of free circulating DNA (fcDNA) and hypermethylation of genes are associated with several cancers. In this study we evaluated the use of fcDNA level and aberrant promoter methylation of 4 genes in serum as biomarkers for PC. Methods: 89 PC patients, 59 with prostatitis (PT) and 104 with benign prostatic hyperplasia (BPH) were enrolled. Serum DNA was analyzed for methylation of GS
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Mittal, Disha, Largee Biswas, and Anita Kamra Verma. "Redox resetting of cisplatin-resistant ovarian cancer cells by cisplatin-encapsulated nanostructured lipid carriers." Nanomedicine 16, no. 12 (2021): 979–95. http://dx.doi.org/10.2217/nnm-2020-0400.

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Aim: To sensitize cisplatin (Cis)-resistant ovarian cancer cells toward Cis using Cis-loaded nanostructured lipid carriers (CisNLCs). Materials & methods: CisNLCs were synthesized and characterized using dynamic light scattering, Fourier transform IR and x-ray diffraction (XRD). Sensitivity of PA-1 and CaOV3 cells to Cis and its biotoxicity were assessed. Further, expression of the Cis-resistance markers GSTPi and ATP7B, and apoptotic markers Bax, Bcl2 and Cas9 were quantified by real-time PCR. Results: The size of synthesized CisNLCs was approximately 179.3 ± 2.32 nm and surface charge wa
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Raposo, Catarina, Ana Karolina de Santana Nunes, Rayana Leal de Almeida Luna, Shyrlene Meiry da Rocha Araújo, Maria Alice da Cruz-Höfling, and Christina Alves Peixoto. "Sildenafil (Viagra) Protective Effects on Neuroinflammation: The Role of iNOS/NO System in an Inflammatory Demyelination Model." Mediators of Inflammation 2013 (2013): 1–11. http://dx.doi.org/10.1155/2013/321460.

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We recently demonstrated that sildenafil reduces the expression of cytokines, COX-2, and GFAP in a demyelinating model induced in wild-type (WT) mice. Herein, the understandings of the neuroprotective effect of sildenafil and the mediation of iNOS/NO system on inflammatory demyelination induced by cuprizone were investigated. The cerebella of iNOS−/−mice were examined after four weeks of treatment with cuprizone alone or combined with sildenafil. Cuprizone increased GFAP, Iba-1, TNF-α, COX-2, IL-1β, and IFN-γexpression, decreased expression of glutathione S-transferase pi (GSTpi), and damaged
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Dissertations / Theses on the topic "GSTPi"

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Schroer, Kathy T. "The role of glutathione S-transferase Pi (GSTPi) in asthma." University of Cincinnati / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1267721309.

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Afzaal, Nadeem Mohammed. "Gene Expression of Anti-Oxidant Genes (GCL, GPx AND GSTpi) in Zebrafish Exposed to Chemicals That Alter Thyroid Hormone." Thesis, Southern Illinois University at Edwardsville, 2013. http://pqdtopen.proquest.com/#viewpdf?dispub=1545317.

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<p> Gene expression of the biotransformation enzymes, glutathione S-transferase (GST) glutathione peroxidase (GPx) and glutathione synthesizing enzyme glutamyl-cysteine-ligase (GCL), was investigated in zebrafish (Dani orerio) exposed to arsenate, perchlorate, a mixture of these two chemicals, and thyroxine (T4). The exposure persisted for 30 days for perchlorate, 7 days for arsenate and 7 days for thryroxin. To test the mixture toxicity of arsenate and perchlorate, arsenate was added for 7 days in addition to 30 days exposure to perchlorate. Arsenate is known to induce oxidative stress, and G
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Alkhaeir, Sawsaneh. "Etude comparative de nouvelles approches thérapeutiques dans le lymphome à cellules du Manteau : utilisation des inhibiteurs de mTOR kinase et BTK." Thesis, Université Paris-Saclay (ComUE), 2016. http://www.theses.fr/2016SACLS432/document.

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La voie PI3Kinase/AKT/mTOR, est une cible thérapeutique du temsirolimus, un inhibiteur de mTORC1. Dans le but d'obtenir une inhibition plus importante de cette voie j’utilise dans ce projet deux nouvelles molécules :- le NVP-BEZ 235 (BEZ) qui inhibe à la fois mTORC1 et la PI3kinase- l'AZD8055 (AZD), un inhibiteur des complexes mTORC1 et mTORC2. En utilisant différentes lignées de LCM, j’ai démontré que l'effet de ces nouveaux inhibiteurs sur la survie cellulaire est plus important que celui du temsirolimus. Cela est probablement dû à l'inhibition de la phosphorylation de l'AKT et la 4EBP. La d
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Gerardi, Daniel Guimarães [UNESP]. "Reatividade de tecidos neoplásicos caninos à proteína associada à resistência a múltiplas drogas-1 (MRP1), à glutationa-s-transferase pi (GSTpi) e à proteína p53." Universidade Estadual Paulista (UNESP), 2008. http://hdl.handle.net/11449/101267.

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Made available in DSpace on 2014-06-11T19:31:11Z (GMT). No. of bitstreams: 0 Previous issue date: 2008-12-18Bitstream added on 2014-06-13T19:20:17Z : No. of bitstreams: 1 gerardi_dg_dr_jabo.pdf: 906206 bytes, checksum: 29e6a477f11cbcccee68cc2a21495f1a (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>Tendo em vista a expressão da proteína associada à resistência a múltiplas drogas (MRP1), da enzima glutationa-S-transferase pi (GSTpi) e da proteína p53 com o desenvolvimento da resistência a múltiplas drogas (RMD) nas células neoplásicas, objetivou-se neste estudo avali
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Gerardi, Daniel Guimarães. "Reatividade de tecidos neoplásicos caninos à proteína associada à resistência a múltiplas drogas-1 (MRP1), à glutationa-s-transferase pi (GSTpi) e à proteína p53 /." Jaboticabal : [s.n.], 2008. http://hdl.handle.net/11449/101267.

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Orientadora: Mirela Tinucci Costa<br>Banca: Renée Laufer Amorim<br>Banca: Carlos Roberto Daleck<br>Banca: Felipe Augusto Ruiz Sueiro<br>Banca: Noeme Sousa Rocha<br>Resumo: Tendo em vista a expressão da proteína associada à resistência a múltiplas drogas (MRP1), da enzima glutationa-S-transferase pi (GSTpi) e da proteína p53 com o desenvolvimento da resistência a múltiplas drogas (RMD) nas células neoplásicas, objetivou-se neste estudo avaliar a expressão desses três marcadores, pela imunoistoquímica, em 68 espécimes de neoplasias caninas, incluindo tumores venéreos transmissíveis (TVTC), masto
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SILVA, Rafaela Bezerra da. "Desenvolvimento computacional de um inibidor das proteínas midkina e glutationa s-transferase." Universidade Federal de Campina Grande, 2015. http://dspace.sti.ufcg.edu.br:8080/jspui/handle/riufcg/1256.

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Submitted by Rosana Amâncio (rosana.amancio@ufcg.edu.br) on 2018-07-26T13:36:43Z No. of bitstreams: 1 RAFAELA BEZERRA DA SILVA - DISSERTAÇÃO PPGCNBio 2015..pdf: 1983101 bytes, checksum: b3bac7399ef1511132df3b9a76b369e7 (MD5)<br>Made available in DSpace on 2018-07-26T13:36:43Z (GMT). No. of bitstreams: 1 RAFAELA BEZERRA DA SILVA - DISSERTAÇÃO PPGCNBio 2015..pdf: 1983101 bytes, checksum: b3bac7399ef1511132df3b9a76b369e7 (MD5) Previous issue date: 2015-07-23<br>CNPq<br>O desenho racional de fármacos tem sido largamente utilizado para o desenvolvimento de medicamentos mais eficazes no tratamento
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Lengert, André van Helvoort. "Análise da metilação dos genes GSTP1 e MGMT e de polimorfismos dos genes GSTP1, GSTT1, GSTM1, CYP1A1 e MGMT como marcadores moleculares do câncer de bexiga." Universidade Estadual de Londrina. Centro de Ciências Biológicas. Programa de Pós-Graduação em Genética e Biologia Molecular, 2012. http://www.bibliotecadigital.uel.br/document/?code=vtls000175468.

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O câncer de bexiga é o segundo tipo de tumor mais frequente encontrado no sistema urinário. No Brasil, segundo os dados mais recentes do Instituto Nacional de Câncer (INCA), a estimativa para o ano de 2012 é de 8.900 novos casos. O diagnóstico precoce, a estimativa de risco e de progressão deste tumor possuem um impacto significativo no prognóstico da doença. Embora existam alguns marcadores moleculares para este tumor, a acurácia obtida ainda não é a ideal. Estudos que correlacionam diferentes polimorfismos, metilação do DNA e níveis de expressão gênica para o câncer de bexiga são escassos na
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Brockmann, Anette [Verfasser]. "GSTP1-1 and thiazolide-induced apoptosis in colon carcinoma cells / Anette Brockmann." Konstanz : Bibliothek der Universität Konstanz, 2015. http://d-nb.info/1088797318/34.

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Biagini, Myers Jocelyn Marie. "Effects of Environmental Tobacco Smoke and CYP2A6 and GSTP1 Exposure on Childhood Wheeze." University of Cincinnati / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1212161341.

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Hemmingsen, Anja. "The expression and functional significance of glutiathione S-transferase P1 (GSTP1) polymorphism in the lung." Thesis, Keele University, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288498.

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Books on the topic "GSTPi"

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Indonesia. Delegasi ke Pertemuan Global System of Trade Preferences Tingkat Menteri, 1986, Brasilia, Brazil. Laporan Delegasi R.I. ke Pertemuan Global System of Trade Preferences (GSTP) Tingkat Menteri, Brasilia, 19-23 Mei 1986. Perutusan Tetap, Rebublik Indonesia pada P.B.B., 1986.

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Hans, Linnemann, ed. South-South trade preferences: The GSTP and trade in manufactures. Sage, 1992.

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Linnemann, Hans. South - South Trade Preferences: The GSTP and Trade in Manufactures (Indo-Dutch Studies on Development Alternatives series). Sage Publications Pvt. Ltd, 1992.

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Tremblay, Steve. A comparison of gene expression pattern in OSCC between young and older individuals, with emphasis on GSTP1 and the Fanconi anemia genes. 2006.

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Book chapters on the topic "GSTPi"

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Brabender, J., R. Metzger, P. M. Schneider, et al. "Die Bedeutung der Glutathion S-Transferase PI (GSTPI) mRNA Expression bei der Pathogenese und Progression des Barrett-Ösophagus." In Deutsche Gesellschaft für Chirurgie. Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-55715-6_214.

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Henrique, Rui, and Carmen Jerónimo. "GSTP1 Hypermethylation for Prostate Cancer Detection." In Prostate Cancer Screening. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-281-0_19.

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Lee, Wen-Hsiang, Pratibha Joshi, and Rong Wen. "Glutathione S-Transferase Pi Isoform (GSTP1) Expression in Murine Retina Increases with Developmental Maturity." In Retinal Degenerative Diseases. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-3209-8_4.

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Hudec, Robert E. "The Structure of South-South Trade Preferences in the 1988 GSTP Agreement: Learning to say MFMFN." In Developing Countries and the Global Trading System. Palgrave Macmillan UK, 1989. http://dx.doi.org/10.1007/978-1-349-20417-5_11.

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Akbas, E., H. Mutluhan-Senli, N. Eras-Erdogan, T. Colak, Ö. Türkmenoglu, and S. Kul. "The Investigation of Relationship between the Poly-Morphism in Exon 5 of Glutathione S-Transferase P1 (Gstp1) Gene and Breast Cancer." In Biodefence. Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-94-007-0217-2_15.

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Xavier Santos, João, Célia Rasga, and Astrid Moura Vicente. "Exposure to Xenobiotics and Gene-Environment Interactions in Autism Spectrum Disorder: A Systematic Review." In Autism Spectrum Disorder - Profile, Heterogeneity, Neurobiology and Intervention. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95758.

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Heritability estimates indicate that genetic susceptibility does not fully explain Autism Spectrum Disorder (ASD) risk variance, and that environmental factors may play a role in this disease. To explore the impact of the environment in ASD etiology, we performed a systematic review of the literature on xenobiotics implicated in the disease, and their interactions with gene variants. We compiled 72 studies reporting associations between ASD and xenobiotic exposure, including air pollutants, persistent and non-persistent organic pollutants, heavy metals, pesticides, pharmaceutical drugs and nutrients. Additionally, 9 studies reported that interactions between some of these chemicals (eg. NO2, particulate matter, manganese, folic acid and vitamin D) and genetic risk factors (eg. variants in the CYP2R1, GSTM1, GSTP1, MET, MTHFR and VDR genes) modulate ASD risk. The chemicals highlighted in this review induce neuropathological mechanisms previously implicated in ASD, including oxidative stress and hypoxia, dysregulation of signaling pathways and endocrine disruption. Exposure to xenobiotics may be harmful during critical windows of neurodevelopment, particularly for individuals with variants in genes involved in xenobiotic metabolization or in widespread signaling pathways. We emphasize the importance of leveraging multilevel data collections and integrative approaches grounded on artificial intelligence to address gene–environment interactions and understand ASD etiology, towards prevention and treatment strategies.
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Paixao, BMC, DS Lima, F. Saggioro, AC Moreira, HR Machado, and M. Castro. "A Subset of Non Functioning Pituitary Adenoma with Immune Positivity for Anterior Pituitary Glycoprotein Hormones Presents under Expression of DLK1 and miR-143 and Overexpression of GSTP1 on Its Molecular Pathogenesis." In The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part1.p5.p1-234.

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Conference papers on the topic "GSTPi"

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Kleih, Markus, Simon Heine, Michael Dengler, et al. "Abstract 3732: Combined targeting of NOXA and GSTpi effectively kills mantle cell lymphoma cells." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-3732.

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Vidal, Igor D., Stephanie Glavaris, Tracy Jones, et al. "Abstract 5303: GSTP1 positive prostate cancer." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-5303.

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Li, Jiexing, Rimma V. Nehme, and Jeffrey Naughton. "GSLPI: A Cost-Based Query Progress Indicator." In 2012 IEEE International Conference on Data Engineering (ICDE 2012). IEEE, 2012. http://dx.doi.org/10.1109/icde.2012.74.

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Park, M., J. Yoon, J. Lee, M. Choi, H. Youn, and S. Jung. "GSTP1 promoter hypermethylation during breast cancer development." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-5042.

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Antoun, Gamil R., R. McLendon, H. Friedman, A. Friedman, D. Bigner, and F. Ali-Osman. "Abstract 1177: The mutational status of the p53 tumor suppressor gene is a determinant of GSTP1 expression and mediates GSTP1-dependent drug resistance in human glioblastoma." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1177.

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Miltenburg, NC, M. Opdam, M. Winter, et al. "Abstract P1-15-05: GSTP1 polymorphism is associated with chemotherapy induced neuropathy." In Abstracts: Thirty-Fifth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 4‐8, 2012; San Antonio, TX. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/0008-5472.sabcs12-p1-15-05.

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Okamura, Tatsunori, and Francis Ali-osman. "Abstract 703: GSTP1 inhibition of HSP27 phosphorylation contributes to glioma drug resistance." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-703.

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Chang, Minsun, Xiyuan Liu, and Eun Young Park. "Abstract LB-389: miR133a-3p sensitizes breast cancer cells by targeting GSTP1." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-lb-389.

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Singh, Rahul Raj, and Katie M. Reindl. "Abstract 764: GSTP1 knockdown and inhibition impairs pancreatic ductal adenocarcinoma (PDAC) growth." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-764.

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Dworski, Ryszard T., and Aimee Hoskins. "GSTP1 Polymorphism Modulates Allergen-Induced Airway Inflammation In Human Atopic Asthmatics In Vivo." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6185.

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Reports on the topic "GSTPi"

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Dudley, A., M. M. Peden-Adams, J. E. Daly, and D. E. Keil. JP-8 Jet Fuel Induces CYP2B1, CYP2BE1, and GSTPI but not CYP1A1 in Murine Liver. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada402064.

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