Relevant bibliographies by topics / Gutkind, lee

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Academic literature on the topic 'Gutkind, lee'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 July 2025

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Journal articles on the topic "Gutkind, lee"

1

Jeffrey J. Williams. "True Stories: An Interview with Lee Gutkind." symplokē 18, no. 1-2 (2010): 349. http://dx.doi.org/10.5250/symploke.18.1-2.0349.

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2

Hamilton, Amy T. "Hurricanes and Carnivals: Essays by Chicanos, Pochos, Pachucos, Mexicanos, and Expatriates ed. by Lee Gutkind." Western American Literature 43, no. 3 (2008): 335–36. http://dx.doi.org/10.1353/wal.2008.0070.

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3

Cullingham, James. "Hurricanes and Carnivals: Essays by Chicanos, Pochos, Pachucos, Mexicanos, and Expatriates - Edited by Gutkind, Lee (ed.)." Bulletin of Latin American Research 28, no. 4 (2009): 573–74. http://dx.doi.org/10.1111/j.1470-9856.2009.00320_11.x.

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4

Trager, Jonathan D. K. "Book Review One Children's Place: A profile of pediatric medicine By Lee Gutkind. 273 pp. New York, Grove Weidenfeld, 1990. $19.95." New England Journal of Medicine 325, no. 8 (1991): 590–91. http://dx.doi.org/10.1056/nejm199108223250824.

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5

Jeong, Woo-Jin, Jiyeong Kim, Hye-Yeon Lee, Zhiyong Wang, Walt Amornphimoltham, and Silvio Gutkind. "Abstract 4951: Proteasome inhibitor carfilzomib induces apoptosis via downregulation of AKT pathway in HPV negative HNSCC." Cancer Research 83, no. 7_Supplement (2023): 4951. http://dx.doi.org/10.1158/1538-7445.am2023-4951.

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Abstract Background Head and neck squamous cell carcinoma (HNSCC) presents in heterogenous sites, varied etiology and risk factors, and has diverse genetic background which confer dissimilar response to treatments, including chemotherapy. Here we tested the hypothesis that investigation of HPV presence in a HNSCC cell line would identify pathway contributing to oncogenesis of other HNSCC. We sought to identify pathways that differentially affect HNSCC according to the genetic background, therefore identifying targets and suitable candidate for treatment. We used genetic and pharmacological app
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6

Pagadala, Meghana, Victoria Wu, Eva Pérez-Guijarro, et al. "Abstract 3825: Germline modifiers of the tumor immune microenvironment reveal drivers of immunotherapy response." Cancer Research 82, no. 12_Supplement (2022): 3825. http://dx.doi.org/10.1158/1538-7445.am2022-3825.

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Abstract With the continued promise of immunotherapy as an avenue for treating cancer, understanding how host genetics contributes to the tumor immune microenvironment (TIME) is essential to tailoring cancer risk screening and treatment strategies. Using genotypes from over 8,000 European individuals in The Cancer Genome Atlas and 137 heritable tumor immune phenotype components (IP components), we identified and investigated 532 TIME-SNPs. Focusing on 77 variants that were relevant to cancer risk, survival, or treatment response, we explored their potential to reveal novel targets for immunoth
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7

Gutkind, J. Silvio. "Abstract IA020: Targeting mTOR signaling in oral premalignant lesions: From bench to clinic and back." Cancer Prevention Research 15, no. 12_Supplement_2 (2022): IA020. http://dx.doi.org/10.1158/1940-6215.tacpad22-ia020.

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Abstract Despite encouraging recent results from novel treatment options, such as immunotherapy, for head and neck squamous cell carcinoma (HNSCC), limited progress has been made in improving outcomes for most patients. Prevention and early detection are key to improving the prognosis of HNSCC. Our team has focused on decoding the oncogenic signaling circuitries driving HNSCC initiation and progression, aimed at identifying novel druggable targets to treat and prevent this aggressive malignancy. These efforts led to the early discovery that persistent activation of PI3K/mTOR signaling circuitr
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8

Yung, Bryan S., Victoria H. Wu, Farhoud Faraji та ін. "Abstract 2868: A chemogenetic approach reveals a GPCR-Gαs-PKA signaling axis promoting T cell dysfunction and cancer immunotherapy failure". Cancer Research 83, № 7_Supplement (2023): 2868. http://dx.doi.org/10.1158/1538-7445.am2023-2868.

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Abstract Recent advances in immune checkpoint blockade (ICB) inhibiting programmed death-1 (PD-1) and cytotoxic T-lymphocyte-associated protein (CTLA-4) have revolutionized the standard of care for cancer treatment. However, the limited response rates to ICB across multiple cancer types suggest that new approaches and targets are clearly needed to fully elucidate the underlying biology of dysfunctional and exhausted CD8 T cells in cancer in order to achieve durable responses (cure). G protein-coupled receptors (GPCRs) are the most intensively studied drug targets, primarily due to their drugga
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9

Saddawi-Konefka, Robert, Riyam Al Msari, Cynthia Tang, et al. "Abstract 3764: Mapping the tumor-sentinel node immune migratome demonstrates a key role for CCR7+ dendritic cells in the successful response to immunoradiotherapy." Cancer Research 85, no. 8_Supplement_1 (2025): 3764. https://doi.org/10.1158/1538-7445.am2025-3764.

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Abstract Immune checkpoint inhibition has demonstrated no benefit when combined with chemoradiotherapy for locally advanced HNSCC, raising the possibility that standard therapies compromise the response to immunotherapy. We previously demonstrated that ablating tumor draining lymphatics abolishes the response to immune-oncology therapy. Specifically, we defined that the response to immunotherapy is coordinated by the cDC1 population in tumor-draining lymph nodes. These findings support the premise that successful tumor responses to immuno-oncology therapies is predicated upon intact and functi
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10

Saddawi-Konefka, Robert, Riyam Al Msari, Shiqi Tang, et al. "Abstract P004: Mapping the tumor-sentinel node immune migratome demonstrates a key role for CCR7+ dendritic cells in the successful response to immunoradiotherapy." Clinical Cancer Research 31, no. 2_Supplement (2025): P004. https://doi.org/10.1158/1557-3265.targetedtherap-p004.

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Abstract Immune checkpoint inhibition has demonstrated no benefit when combined with chemoradiotherapy for locally advanced HNSCC, raising the possibility that standard therapies compromise the response to immunotherapy. To address this hypothesis, we previously demonstrated that ablating tumor draining lymphatics abolishes the response to immune-oncology therapy. Specifically, we defined that the response to immunotherapy is coordinated by interferon type-I signaling through the cDC1 population in tumor-draining lymph nodes. These findings support the premise that successful tumor responses t
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Books on the topic "Gutkind, lee"

1

Sam, Gutkind, ed. Truckin' with Sam: A father and son, the Mick and the Dyl, rockin' and rollin', on the road. Excelsior Editions, State University of New York Press, 2010.

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2

Gutkind, Lee. Forever Fat: Essays by the Godfather. Bison Books, 2010.

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