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Journal articles on the topic 'H. pylori'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

. "NSAID, gebruikNSAID-gebruik en H. pylori, tetracycline H. pylori, naproxen H. pylori, metronidazol H. pylori, bismutoxideH. Pylori-eradicatie." Medisch-Farmaceutische Mededelingen 38, no. 4 (2000): 72. http://dx.doi.org/10.1007/bf03057514.

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2

Assumpção, Paulo Pimentel de, Robert M. Genta, Maria Constanza Camargo, Jessica Manoelli Costa da Silva, Manuel Ricardo Amieva, and Massimo Rugge. "The Landscape of Helicobacter pylori-related Gastric Carcinogenesis." Journal of Gastrointestinal and Liver Diseases 33, no. 4 (2024): 524–34. https://doi.org/10.15403/jgld-5959.

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The relationship between Helicobacter pylori (H. pylori) and humans remains a complex enigma. While other factors contribute to gastric cancer (GC), their impact pales in comparison to the central role of H. pylori. Various cofactors, such as dietary carcinogens and Epstein-Barr virus infection, can lead to GC independently of H. pylori. However, it is likely the combination of mechanisms, especially those driven by H. pylori, that represents the primary force behind GC development. Identifying individuals at high risk of developing H. pylori-related GC or detecting the disease in its earliest

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3

Sakaguchi, Takuki, Takaaki Sugihara, Ken Ohnita, et al. "Pyloric Incompetence Associated with Helicobactor pylori Infection and Correlated to the Severity of Atrophic Gastritis." Diagnostics 12, no. 3 (2022): 572. http://dx.doi.org/10.3390/diagnostics12030572.

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Duodenogastric reflux (DGR) causes bile reflux gastritis (BRG) and may develop into gastric cancer. DGR is classified as primary in non-operated stomachs or secondary to surgical intervention. Primary DGR and Helicobacter pylori (H. pylori) infection are reportedly related. However, the mechanism is not fully understood. This study aimed to elucidate the relationship between H. pylori infection and pyloric incompetence in a non-operated stomach. A total of 502 non-operated participants who underwent an upper intestinal endoscopy were prospectively enrolled. Endoscopic findings (EAC, endoscopic

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4

Garcés-Duran, R., S. Kindt, K. Kotilea, et al. "Belgian consensus for Helicobacter pylori management 2023." Acta Gastro Enterologica Belgica 86, no. 1 (2023): 74–91. http://dx.doi.org/10.51821/86.1.11327.

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Helicobacter pylori (H. pylori) infection causes chronic gastritis, peptic ulcers and gastric cancer. Although H. pylori prevalence is decreasing worldwide, regional variations exist in Europe, with the lowest infection prevalence in Northern Europe, and the highest in Eastern and Southern Europe (1). Changes in the treatment recommendations and the increasing available evidence have justified the implementation of new recommendations since last Belgian consensus in 1998 (2). Several non-H. pylori Helicobacter species (NH.PYLORI-H), colonizing the stomach of domestic animals, also have the abi

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Öztekin, Merve, Birsen Yılmaz, Duygu Ağagündüz, and Raffaele Capasso. "Overview of Helicobacter pylori Infection: Clinical Features, Treatment, and Nutritional Aspects." Diseases 9, no. 4 (2021): 66. http://dx.doi.org/10.3390/diseases9040066.

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Helicobacter pylori (H. pylori) is a 0.5–1 µm wide, 2–4 µm long, short helical, S-shaped Gram-negative microorganism. It is mostly found in the pyloric region of the stomach and causes chronic gastric infection. It is estimated that these bacteria infect more than half of the world’s population. The mode of transmission and infection of H. pylori is still not known exactly, but the faecal–oral and oral–oral routes via water or food consumption are thought to be a very common cause. In the last three decades, research interest has increased regarding the pathogenicity, microbial activity, genet

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Blasiak, Janusz, Jan Chojnacki, and Cezary Chojnacki. "Inflammation, oxidative stress, DNA damage response and epigenetic modifications interact behind the beneficial actions of melatonin on H. pylori-mediated gastric disorders." Melatonin Research 6, no. 2 (2023): 135–47. http://dx.doi.org/10.32794/mr112500145.

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Helicobacter pylori (H. pylori) infection is associated with several disorders of the gastrointestinal tract, including gastric cancer. Studies of ours and others suggest that H. pylori infection may affect melatonin synthesis in the gastric epithelial cells. On the other hand, melatonin ameliorates gastric disorders as shown in clinical trials and experimental studies. Moreover, melatonin not only suppresses the DNA-damaging reaction of diet-related mutagens that can initiate carcinogenesis in gastric mucosa, but also the oxidative DNA damage evoked by reactive oxygen and nitrogen species pro

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7

Asonuma, Sho, Akira Imatani, Naoki Asano, et al. "Helicobacter pylori induces gastric mucosal intestinal metaplasia through the inhibition of interleukin-4-mediated HMG box protein Sox2 expression." American Journal of Physiology-Gastrointestinal and Liver Physiology 297, no. 2 (2009): G312—G322. http://dx.doi.org/10.1152/ajpgi.00518.2007.

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Helicobacter pylori is a major cause of the transdifferentiation into intestinal metaplasia that may develop gastric cancer. However, the molecular pathogenesis of this transdifferentiation is poorly understood. A SRY-related HMG box protein Sox2 is an essential transcription factor of organ development in brain, lung, and stomach. Our aim of this study was to investigate the mechanism responsible for regulation of Sox2 in host Th1-dominant response to H. pylori. Sox2 protein was immunohistochemically expressed in both human oxyntic and pyloric glands with H. pylori infection, but not in intes

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Dr., Tarak Banik, Prasenjit Kumar Bar Dr., and Saikat Mandal Dr. "Helicobacter pylori chronic gastritis: correlation between endoscopic findings and histopathology with special reference to updated sydney system." International Journal of Medical Research and Review 8, no. 1 (2020): 7–13. https://doi.org/10.17511/ijmrr.2020.i01.02.

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Aims of the study: Helicobacter pylori (H. pylori) induces inflammatory changes in the gastric mucosa variably correlated to different endoscopic and histologic features. The present prospective study aimed to correlate different endoscopic findings with histomorphological changes and the presence of H. pylori in the gastro-duodenal mucosa, in samples of dyspeptic patients. Methods: 60 dyspeptic patients of 18 years to 60 years of age were selected from outpatient department and screened with gastro-duodenoscopy and biopsy. The presence of H. pylori was determined by urease test on fresh biops

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Imam, Alam Imam Bakhsh Irshad Ahmad Zakir Hussain Sanjay Kumar. "Association Of Helicobacter Pylori With Perforated Peptic Ulcer In Pakistani Population An Institution Based Study." Multicultural Education 8, no. 3 (2022): 130. https://doi.org/10.5281/zenodo.6344724.

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<strong><em>Background: </em></strong><em>Most patients with chronic peptic ulcer disease have Helicobacter pylori (H. pylori) infection. In the past, immediate acid-reduction surgery has been strongly advocated for perforated peptic ulcers because of the high incidence of ulcer relapse after simple closure. Simple over sewing procedures either by an open or laparoscopic approach together with H. pylori eradication appears to supersede definitive ulcer surgery.</em> <strong><em>Objective:</em></strong><em> To determine the frequency of H pylori in patients with perforated duodenal ulcer.</em>

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Takatsuna, Masafumi, Rie Azumi, Takeshi Mizusawa, et al. "A case of Helicobacter pylori-negative early gastric adenocarcinoma with gastrointestinal phenotype." Endoscopy International Open 09, no. 06 (2021): E863—E866. http://dx.doi.org/10.1055/a-1396-3854.

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AbstractA 40-year-old man with slightly depressed (0-IIc) type gastric cancer of the pyloric anterior gastric area underwent pre-operative screening for tetralogy of Fallot and endoscopic submucosal dissection (ESD) and was tested for Helicobacter pylori antigens and antibodies. Both tests were negative. He did not have a history of eradication. Pathological diagnosis of ESD showed a well-differentiated adenocarcinoma. The tumor was CD10-positive, MUC5AC-negative, and MUC6-confocal positive; it showed differentiation with gastrointestinal phenotype. Moreover, the tumor cells were lysozyme-posi

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Kosai, Nik Ritza, Hardip Singh Gendeh, Abdul Rashid Norfaezan, Jamin Razman, Paul Anthony Sutton, and Srijit Das. "Prolapsing Gastric Polyp Causing Intermittent Gastric Outlet Obstruction." International Surgery 100, no. 6 (2015): 1148–52. http://dx.doi.org/10.9738/intsurg-d-14-00205.1.

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Gastric polyps are often an incidental finding on upper gastrointestinal endoscopy, with an incidence up to 5%. The majority of gastric polyps are asymptomatic, occurring secondary to inflammation. Prior reviews discussed Helicobacter pylori (H pylori)–associated singular gastric polyposis; however, we present a rare and unusual case of recurrent multiple benign gastric polyposis post H pylori eradication resulting in intermittent gastric outlet obstruction. A 70-year-old independent male, Chinese in ethnicity, with a background of diabetes mellitus, hypertension, and a simple renal cyst prese

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Fiorini, Giulia, Ilaria Maria Saracino, Angelo Zullo, et al. "Antibiotic Resistance and Therapy for H. pylori Infection in Immigrant Patients Treated in Italy." Journal of Clinical Medicine 9, no. 5 (2020): 1299. http://dx.doi.org/10.3390/jcm9051299.

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Background: Helicobacter pylori (H. pylori) infection is the leading cause of both peptic ulcers and gastric tumors, including low-grade MALT-lymphoma and adenocarcinoma. Although it is decreasing in developed countries, H. pylori prevalence remains high in developing areas, mainly due to low socio-economic levels, and the potential consumption of contaminated water. Moreover, a different pattern of primary antibiotic resistance is expected in their H. pylori isolates, potentially affecting the efficacy of standard eradication therapies. Indeed, a previous study showed the eradication rate fol

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Di Ciaula, Agostino, Giuseppe Scaccianoce, Marino Venerito, et al. "Eradication Rates in Italian Subjects Heterogeneously Managed for Helicobacter pylori Infection. Time to Abandon Empiric Treatments in Southern Europe." Journal of Gastrointestinal and Liver Diseases 26, no. 2 (2017): 129–37. http://dx.doi.org/10.15403/jgld.2014.1121.262.itl.

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Background & Aims: H. pylori eradication is strongly affected by various factors, including the ongoing antibiotic resistance. We describe a “real life” scenario in patients managed for H. pylori-related conditions, living in a southern Italian region (Apulia), an area with clarithromycin resistance >15%.Methods: 2,224 subjects were studied in two tertiary referral centers in Apulia. Analyses included: reason for referral, H. pylori infection rates (13C-urea breath test – UBT or upper endoscopy), and eradication rates following distinct regimens previously prescribed or prospectively pr

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German, Serafima V., A. V. Modestova, I. E. Zykova, I. P. Bobrovnitsky, and M. Yu Yakovlev. "ON THE POSSIBLE PARTICULARITY OF HELICOBACTER PYLORI INFECTION TO GALLSTONE DISEASE." Hygiene and sanitation 97, no. 11 (2018): 1076–79. http://dx.doi.org/10.18821/0016-9900-2018-97-11-1076-79.

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Introduction. Up to now, it has not been established whether Helicobacter pylori, the most common bacterial pathogen of human, is involved in cholelithiasis. Material and Methods. Based on the analysis of prophylactic medical examination of working people in the Moscow region, the determination the pyloric Helicobacter infection and assessment of the virulence of bacteria there were studied the associations of H. pylori infection and gallstones. The infection was detected by the presence of serum specific antibodies of IgG class, the virulence of the strain H. pylori - by the presence of total

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Hassan, Md Ali, MA Ahad, MH Rahman, MSH Bhuiyan, and MAH Khan. "Association between cytotoxin producing H. pylori and gastric carcinoma." Bangladesh Medical Journal Khulna 49, no. 1-2 (2017): 13–17. http://dx.doi.org/10.3329/bmjk.v49i1-2.31819.

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Background: Enormous studies have been conducted worldwide regarding CagA+ status of H. pylori in gastric carcinoma Objective: No study relating CagA+ status and gastric carcinoma has been carried out in our country yet. This study has been designed to see the association between CagA+ H. pylorl strain and gastric carcinomaMethods: For this purpose, a total number of 80 (eighty) patients were selected. Of the 80 (eighty) patients, 40 (forty) were selected as cases (malignant) and the remainder 40 (forty) were selected as controls (non malignant). H. pylori was detected by applying non invasive

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Jaafar, Salim Shamkhi, Ehab Y. Jabber, Amer Abdul Husain Alwish, Raheem Tuama Obayes Al- Mammori та Hasanain Khaleel Shareef. "THE IMMUNOLOGICAL ROLE OF ANTI-H. PYLORI ANTIBODIES LEVEL, INF-Γ, AND TNF-Α IN A NUMBER OF PATIENTS WITH A TUMOR IN THE DIGESTIVE SYSTEM IN BABYLON GOVERNORATE". International Journal Multidisciplinary (IJMI) 1, № 3 (2024): 168–76. https://doi.org/10.61796/ijmi.v1i3.184.

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General Background: Helicobacter pylori is a well-known bacterium responsible for chronic infections, significantly contributing to gastric and mucosa-associated lymphoid tissue cancers, as well as peptic ulcer disease. Specific Background: The link between H. pylori infection and gastrointestinal (GIT) tumors remains under-explored, particularly concerning immunological parameters. Knowledge Gap: The role of immune markers, such as anti-H. pylori IgG antibodies, TNF-α, and IFN-γ, in distinguishing between malignant and benign GIT tumors is unclear. Aims: This study aimed to assess the relatio

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I.M., Saracino, Pavoni M., Zullo A., et al. "Antibiotic Resistance and Therapy Outcome in H. pylori Eradication Failure Patients." Antibiotics 9, no. 3 (2020): 121. http://dx.doi.org/10.3390/antibiotics9030121.

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Helicobacter pylori (H. pylori) eradication fails in a definite amount of patients despite one or more therapeutic attempts. Curing these patients is progressively more difficult, due to development of antibiotic resistance. Current guidelines suggest testing antibiotic susceptibility in H. pylori isolates following two therapeutic attempts. Aim: to evaluate the development of antibiotic resistance, MIC values trends and therapeutic outcomes in patients who failed at least one H. pylori eradication therapy. Methods: consecutive patients, referred to perform upper gastrointestinal endoscopy (UG

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Coconnier, Marie-Helene, Vanessa Lievin, Elisabeth Hemery, and Alain L. Servin. "Antagonistic Activity againstHelicobacter Infection In Vitro and In Vivo by the HumanLactobacillus acidophilus Strain LB." Applied and Environmental Microbiology 64, no. 11 (1998): 4573–80. http://dx.doi.org/10.1128/aem.64.11.4573-4580.1998.

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ABSTRACT The purpose of the present study was to examine the activity of the human Lactobacillus acidophilus strain LB, which secretes an antibacterial substance(s) against Helicobacter pyloriin vitro and in vivo. The spent culture supernatant (SCS) of the strain LB (LB-SCS) dramatically decreased the viability of H. pylori in vitro independent of pH and lactic acid levels. Adhesion of H. pylori to the cultured human mucosecreting HT29-MTX cells decreased in parallel with the viability of H. pylori. In conventional mice, oral treatment with the LB-SCS protected against infection with Helicobac

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Adewumi, F. A., J. O. Ipinlaye, C. O. Esan, and A. O. Oluyege. "Rapid detection of <i>Helicobacter pylori</i> infection among participants presenting with dyspepsia in a primary health care facility in Ado-Ekiti, Ekiti State." World Journal of Applied Science & Technology 16, no. 1 (2025): 98–102. https://doi.org/10.4314/wojast.v16i1.98.

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Helicobacter pylori (H. pylori) is a Gram-negative bacterium capable of colonizing the pyloric region of the stomach, and induces chronic gastric infection. It is closely related to gastrointestinal diseases such as gastritis, gastric ulcer, and gastric cancer. Notably, this bacterium has achieved pandemic magnitudes, infecting over half of the global population. This study sought to investigate the prevalence of Helicobacter pylori infection among patients presenting with dyspepsia at a primary healthcare facility in Ado-Ekiti, Ekiti State, utilizing non-invasive diagnostic procedures. Two hu

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Haruna, Sani Ado. "Prevalence and Risk Factors of Helicobacter Pylori Infection among Patients with Peptic Ulcer Disease Attendeing Muhammad Abdullahi Wase Teaching Hospital." Scholars Academic Journal of Biosciences 11, no. 12 (2023): 467–73. http://dx.doi.org/10.36347/sajb.2023.v11i12.008.

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Duodenal (DU) and gastric (GU) ulcers are both included in the category of peptic ulcer disease (PUD). The first section of the duodenum or the stomach's pre-pyloric region (antrum) are the most common sites for duodenal ulcers. Most cases of gastric ulcers occur in the stomach's lesser curvature, which connects to the body's antrum (angularis). The purpose of the study is to ascertain the prevalence and risk factors of Helicobacter pylori infections in Muhammad Abdullahi Wase Teaching Hospital patients suffering from peptic ulcer disease. Patients with PUD who had an H. pylori infection test

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Chicherin, I. Yu, I. P. Pogorelskiy, E. P. Kolevatykh, I. A. Lundovskikh, and M. R. Shabalina. "First experience with the metaprebiotic Stimbifid plus used for eradication of Helicobacter pylori in patients with gastric ulcer." Infekcionnye bolezni 19, no. 3 (2021): 92–103. http://dx.doi.org/10.20953/1729-9225-2021-3-92-103.

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Objective. To analyze the relationships between probiotic microorganisms and H. pylori KM-11 (RifR) and evaluate their effect on the structural organization of the pathogen and natural colonization resistance of the stomach both independently and with the metaprebiotic Stimbifid plus using bacteriological methods and electron microscopy. Our findings can be potentially used for effective eradication of H. pylori KM-11 (RifR) and treatment of gastric ulcer in volunteers during metaprebiotic therapy. Materials and methods. The following strains of microorganisms were used in this study: Lactobac

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Krishnamurthy, Partha, Mary Parlow, Jason B. Zitzer, et al. "Helicobacter pylori Containing Only Cytoplasmic Urease Is Susceptible to Acid." Infection and Immunity 66, no. 11 (1998): 5060–66. http://dx.doi.org/10.1128/iai.66.11.5060-5066.1998.

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ABSTRACT Helicobacter pylori, an important etiologic agent in a variety of gastroduodenal diseases, produces large amounts of urease as an essential colonization factor. We have demonstrated previously that urease is located within the cytoplasm and on the surface of H. pylori both in vivo and in stationary-phase culture. The purpose of the present study was to assess the relative contributions of cytoplasmic and surface-localized urease to the ability of H. pylori to survive exposure to acid in the presence of urea. Toward this end, we compared the acid resistance in vitro of H. pylori cells

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Nisreen Jawad Kadhim and Saeed Hameed Lafta. "Helicobacter pylori: Challenges and Advances in Diagnosis, Treatment, and Eradication." Academic International Journal of Medical Update 2, no. 1 (2024): 43–52. https://doi.org/10.59675/u216.

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A common and extensively distributed "Helicobacter pylori" still poses a serious risk to public health. "H. pylori" infections can cause various stomach problems, such as inflammation, gastroduodenal ulcers, and cancers; therefore, it is essential to discover and treat them early to stop their progression. To reduce or perhaps completely eradicate "H. pylori," people must be informed of the bacteria and the health risks it poses. Raising the bar for the present diagnostic, eradication, and alternate treatment approaches is therefore essential. Furthermore, a succinct review of conventional and

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Saler, Tayyibe, Şakir Özgür Keşkek, Sibel Kırk, Süleyman Ahbab, and Gülay Ortoğlu. "H. pyloriMay Not Be Associated with Iron Deficiency Anemia in Patients with Normal Gastrointestinal Tract Endoscopy Results." Advances in Hematology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/375915.

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Background. The aim of this study was to investigate the association between iron deficiency anemia andH. pyloriin patients with normal gastrointestinal tract endoscopy results.Materials and Methods. A total of 117 male patients with normal gastrointestinal tract endoscopy results were included in this retrospective study. The study and control groups included 69 and 48 patients with and without iron deficiency anemia, respectively. The prevalence ofH. pylori, the number of RBCs, and the levels of HGB, HTC, MCV, iron, and ferritin were calculated and compared.Results. There was no statisticall

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Lee, Woo-Kon, Keiji Ogura, John T. Loh, Timothy L. Cover, and Douglas E. Berg. "Quantitative Effect of luxS Gene Inactivation on the Fitness of Helicobacter pylori." Applied and Environmental Microbiology 72, no. 10 (2006): 6615–22. http://dx.doi.org/10.1128/aem.01291-06.

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ABSTRACT Furanone metabolites called AI-2 (autoinducer 2), used by some bacterial species for signaling and cell density-regulated changes in gene expression, are made while regenerating S-adenosyl methionine (SAM) after its use as a methyl donor. The luxS-encoded enzyme, in particular, participates in this activated methyl cycle by generating both a pentanedione, which is transformed chemically into these AI-2 compounds, and homocysteine, a precursor of methionine and SAM. Helicobacter pylori seems to contain the genes for this activated methyl cycle, including luxS, but not genes for AI-2 up

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Sanaz, Mehrabani* Mohammad Reza Esmaeili-dooki Hassan Tadayoni Mohammad Pornasrollah. "RELATIONSHIP BETWEEN HELICOBACTER PYLORI INFECTION AND UPPER GASTRO-INTESTINAL BLEEDING IN NORTHERN IRAN CHILDREN." Indo American Journal of Pharmaceutical Sciences 04, no. 08 (2017): 2583–90. https://doi.org/10.5281/zenodo.853282.

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Background: There are controversies regarding relationship between Helicobacter pylori (H. pylori) and upper gastro-intestinal bleeding (UGIB) in children. The goal of this study was to assess the relationship between H. pylori infection and UGIB in Northern Iranian children. Material and method: One hundred children who had UGIB indicated for upper gastrointestinal endoscopy and 100 children without UGIBwho were candidate for upper endoscopy because of chronic abdominal pain considered as their controls. After stabling vital sign, nasogastric tube inserted and washing was done then within 24

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Akcakavak, Gokhan, Mehmet Tuzcu, Nevin Tuzcu, Zeynep Celik, Aysenur Tural, and Osman Dagar. "Investigation with Real-Time PCR and Histopathology on the presence of H. felis, H. heilmannii and H. pylori in dogs." Revista Científica de la Facultad de Ciencias Veterinarias XXXIII, no. 1 (2023): 1–7. http://dx.doi.org/10.52973/rcfcv-e33214.

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Helicobacter species such as H. heilmannii, H. pylori, H. felis, H. bizzozeronii and H. salomonis have been identified in cats and dogs, and research suggesting that these species may be zoonotic agents and has been studied intensified in recent years. The aim of this study was to reveal the presence, comparing the histopathological findings and Real-time PCR results of H. felis, H. heilmannii, and H. pylori in the stomach and liver tissues taken during the necropsies of owned, stray or shelter dogs. The material of the study consisted of stomach and liver tissues taken from 35 dogs that died

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Malik, G. M., M. Mubarik, and S. A. Kadla. "Helicobacter pylori Infection in Endoscopic Biopsy Specimens of Gastric Antrum: Laboratory Diagnosis and Comparative Efficacy of Three Diagnostic Tests." Diagnostic and Therapeutic Endoscopy 6, no. 1 (1999): 25–29. http://dx.doi.org/10.1155/dte.6.25.

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Aims and objectives The present study was undertaken to compare the diagnostic yield of three available test procedures for detecting Helicobacter pylori (H. pylori) infection in endoscopic biopsies.Methods H. pylori infection was sought in 150 patients referred for upper gastrointestinal (GI) endoscopy. Multiple (about six) biopsy specimens were taken from pyloric antrum in each patient. Two biopsy specimens were subjected to one minute endoscopy room test – OMERT (a modified form of urease test), two were sent for histopathological analysis, where multiple sections were subjected to Giemsa s

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&NA;. "Treating H. pylori." Inpharma Weekly &NA;, no. 1125 (1998): 16. http://dx.doi.org/10.2165/00128413-199811250-00029.

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&NA;. "H. pylori update." Inpharma Weekly &NA;, no. 933 (1994): 15. http://dx.doi.org/10.2165/00128413-199409330-00037.

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&NA;. "H. pylori update." Inpharma Weekly &NA;, no. 946 (1994): 5. http://dx.doi.org/10.2165/00128413-199409460-00006.

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Delaney, Brendan, and Paul Moayyedi. "Eradicating H pylori." BMJ 328, no. 7453 (2004): 1388–89. http://dx.doi.org/10.1136/bmj.328.7453.1388.

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&NA;. "Eradicating H. pylori." Inpharma Weekly &NA;, no. 816 (1991): 11. http://dx.doi.org/10.2165/00128413-199108160-00032.

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Desai, Tusar, Ahmed I. Edhi, and Seifeldin Hakim. "Eradicating H. pylori." American Journal of Gastroenterology 114, no. 12 (2019): 1827–28. http://dx.doi.org/10.14309/ajg.0000000000000424.

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Labenz, Joachim. "H. pylori bändigen." Der Hausarzt 49, no. 4 (2012): 49–52. http://dx.doi.org/10.1007/s15200-012-0201-7.

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Fennerty, MB. "Is the only good H. pylori a dead H. pylori?" Gastroenterology 111, no. 6 (1996): 1773–74. http://dx.doi.org/10.1016/s0016-5085(96)70047-1.

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Siregar, Gontar Alamsyah, Sahat Salim, and Ricky Rivalino Sitepu. "Comparison of IL-6, IL-8 Concentrations in H. pylori- and non-H. pylori-associated Gastritis." Indonesian Biomedical Journal 6, no. 3 (2014): 163. http://dx.doi.org/10.18585/inabj.v6i3.29.

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BACKGROUND: Helicobacter pylori is a non-invasive microorganism causing intense gastric mucosal inflammatory and immune reaction. The gastric mucosal levels of the proinflammatory cytokines Interleukin 6 (IL-6) and IL-8 have been reported to be increased in H. pylori infection, but the serum levels in H. pylori infection is still controversial. The purpose of this study was to investigate the serum levels of IL-6 and IL-8 in H. pylori infection.METHODS: A cross sectional study was done on eighty consecutive gastritis patients admitted to endoscopy units at Adam Malik General Hospital and Perma

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Iwai, Komei, Tetsuji Azuma, Takatoshi Yonenaga, et al. "Association between dental caries and Helicobacter pylori infection in Japanese adults: A cross-sectional study." PLOS ONE 17, no. 7 (2022): e0271459. http://dx.doi.org/10.1371/journal.pone.0271459.

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Helicobacter pylori (H. pylori) is widely known as a cause of gastric disorders. Presence of H. pylori in dental pulp has been reported. Dental caries may influence the presence or absence of systemic H. pylori infection by serving as a source of H. pylori. In this cross-sectional study, we examined whether H. pylori infection in blood were associated with dental caries in Japanese adults. The participants were 752 individuals (513 males and 239 females, mean age 53.8 years) who underwent both H. pylori testing (H. pylori antibody test and pepsinogen test) and dental checkups at the Asahi Univ

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Shafrir, Asher, Michal Shauly-Aharonov, Lior H. Katz, Ora Paltiel, Yishai Pickman, and Zvi Ackerman. "The Association between Serum Vitamin D Levels and Helicobacter pylori Presence and Eradication." Nutrients 13, no. 1 (2021): 278. http://dx.doi.org/10.3390/nu13010278.

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Background: The success of Helicobacter pylori (H. pylori) eradication depends on several host and treatment factors. Serum vitamin D levels may be associated with H. pylori infection and eradication rates. We investigated the association between vitamin D and H. pylori infection and eradication, using a large electronic database based on medical records from a population-based health maintenance organization. Methods: Data regarding adults who underwent H. pylori testing and had vitamin D measurements within one month of H. pylori testing were collected. H. pylori infection was ascertained us

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Manish, Kumar Bhaskar. "A Hospital Based Study to Assess the Role of Helicobacter Pylori Infection in the Pathogenesis of Minimal Hepatic Encephalopathy and Effect of its Eradication." International Journal of Toxicological and Pharmacological Research 13, no. 11 (2023): 334–38. https://doi.org/10.5281/zenodo.11005176.

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<strong>Aim: </strong>The aim of the study was to find the role of Helicobacter pylori infection in the pathogenesis of minimal hepatic encephalopathy and effect of its eradication. <strong>Methods: </strong>A prospective study was conducted in the Department of Gastroenterology for the period of 2 years. 200 patients were included in the study. In patients of suspected cirrhosis of liver ultrasound and endoscopic examination were performed. <strong>Results: </strong>Hepatitis B virus infection was the most common (35%) cause of cirrhosis. In the study group, most pati

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Tongtawee, Taweesak, Wareeporn Wattanawongdon, and Theeraya Simawaranon. "Effects of periodontal therapy on eradication and recurrence of Helicobacter pylori infection after successful treatment." Journal of International Medical Research 47, no. 2 (2019): 875–83. http://dx.doi.org/10.1177/0300060518816158.

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Objectives This study aimed to evaluate the effects of periodontal therapy on the efficacy of Helicobacter pylori eradication and on the recurrence of infection after eradication. Methods We conducted a prospective randomized trial on 698 gastric H. pylori-infected patients, of whom 347 received gastric H. pylori treatment alone and 342 received gastric H. pylori treatment plus periodontal therapy. The presence of H. pylori and associated virulence genes were detected by real-time polymerase chain reaction. Results After eradication of gastric H. pylori infection, the recurrence of gastric H.

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Dr., Zain Nayyer Dr. Kashif Abdullah Dr. Mariam Rafiq. "A COMPARARTIVE STUDY TO KNOW THE PREVALENCE OF HELICOBACTER PYLORI INFECTION AMONG MALES AND FEMALES." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 10 (2018): 10573–76. https://doi.org/10.5281/zenodo.1468985.

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<strong><em>Objective:</em></strong><em> To compare the prevalence of H. pylori infection in men and women.</em> <strong><em>Study design:</em></strong><em> A comparative study. </em> <strong><em>Place and Duration of the Study:</em></strong><em> In the Gastroenterology Department, Jinnah Hospital, Lahore for one year duration from September 2016 to Seotember2017.</em> <strong><em>Methods:</em></strong><em> The study included serum samples, stool samples and gastric biopsies and 100 patients with H. pylori infection including males and females.</em> <strong><em>Results:</em></strong><em> Of th

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Callixte, Yadufashije, Yvette Uwitonze Ange, Manizabayo Yvonne, and Habyarimana Thierry. "Detection of Helicobacter pylori among gastritis patients attending Nemba district hospital." African Journal of Biological Sciences 1, no. 4 (2019): 68–75. https://doi.org/10.33472/AFJBS.1.4.2019.68-75.

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<strong>Background:</strong> <em>Helicobacter pylori</em> (<em>H. pylori</em>) is a small, spiral-shaped bacterium that lives in the surface of the stomach and duodenum. More than half of the world’s population is colonized with <em>H. pylori </em>in the gastric mucosa which is the major cause of chronic gastritis, peptic ulcer and also the most important etiological factor responsible for the duodenal and gastric ulcer and has an important role in the pathogenesis leading to gastric cancer. <strong>Objectives:</strong> The objectives of this study was to determine the prevalence of <em>

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Syam, Ari F. "Current situation of Helicobacter pylori infection in Indonesia." Medical Journal of Indonesia 25, no. 4 (2017): 263–6. http://dx.doi.org/10.13181/mji.v25i4.1408.

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Data epidemiologi infeksi Helicobacter pylori (H. pylori) terus berubah dalam beberapa dekade terakhir. Indonesia dilaporkan memiliki prevalensi infeksi H. pylori yang rendah dibandingkan dengan negara lain di Asia. Beberapa penelitian di Indonesia melaporkan bahwa sanitasi yang buruk, usia, agama, etnis merupakan faktor risiko untuk infeksi H. pylori. Dibandingkan dengan tes diagnostik lainnya, tes urine merupakan tes yang dapat diandalkan untuk mendeteksi H. pylori di Indonesia karena tes tersebut bersifat non-invasif dengan harga yang cukup terjangkau dan memiliki akurasi yang tinggi. Meski

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Chen, Yang. "The overview of Clarithromycin treatment for Helicobacter pylori infection." Theoretical and Natural Science 46, no. 1 (2024): 142–48. http://dx.doi.org/10.54254/2753-8818/46/20240604.

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Helicobacter pylori (H. pylori) is a Gram-negative bacillus that causes infections worldwide. Helicobacter pylori's drug resistance and infectivity are vital factors causing Helicobacter pylori infection. The virulence factors of Helicobacter pylori include acid escape virulence factors and epithelial cell colonizing factors, which can directly or indirectly cause chronic gastritis or gastric ulcer. As the resistance of Helicobacter pylori increases, a combination of multiple antibiotics is now the recommended therapy, and clarithromycin is one of them. The production process of clarithromycin

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Elfira, Vanda, Dwi Prasetyo, Dzulfikar DLH, and Kusnandi Rusmil. "Perbedaan Status Gizi dan Perawakan Pendek pada Anak Sakit Perut Berulang dengan Helicobacter Pylori Positif dan Negatif." Sari Pediatri 20, no. 5 (2019): 303. http://dx.doi.org/10.14238/sp20.5.2019.303-8.

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Latar belakang. Sakit perut berulang (SPB) merupakan keluhan yang paling sering pada anak. Infeksi Helicobacter pylori (H. pylori) saat ini merupakan salah satu penyebab organik terbanyak pada anak SPB. Infeksi H. pylori dapat menyebabkan malnutrisi dan perawakan pendek, tetapi hal ini masih kontroversial.Tujuan. Mengetahui perbedaan status gizi dan perawakan pendek antara anak SPB dengan infeksi H. pylori positif dan negatif.Metode Penelitian potong lintang analitik dilakukan pada anak SMP dan SMA di Bandung yang mengalami SPB. Infeksi H. pylori berdasarkan pemeriksaan serologis menggunakan k

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Lemke, Laura B., Zhongming Ge, Mark T. Whary, et al. "Concurrent Helicobacter bilis Infection in C57BL/6 Mice Attenuates Proinflammatory H. pylori-Induced Gastric Pathology." Infection and Immunity 77, no. 5 (2009): 2147–58. http://dx.doi.org/10.1128/iai.01395-08.

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ABSTRACT Because coinfections can alter helicobacter gastritis, we investigated whether enterohepatic Helicobacter bilis modulates Helicobacter pylori gastritis in C57BL/6 mice. Thirty mice per group were sham dosed, H. bilis or H. pylori infected, or H. bilis infected followed in 2 weeks by H. pylori and then evaluated at 6 and 11 months postinfection (mpi) for gastritis and premalignant lesions. Compared to H. pylori-infected mice, H. bilis/H. pylori-infected mice at 6 and 11 mpi had less severe gastritis, atrophy, mucous metaplasia and hyperplasia (P < 0.01) and, additionally, at 11 mpi,

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Hiengrach, Pratsanee, Wimonrat Panpetch, Ariya Chindamporn, and Asada Leelahavanichkul. "Helicobacter pylori, Protected from Antibiotics and Stresses Inside Candida albicans Vacuoles, Cause Gastritis in Mice." International Journal of Molecular Sciences 23, no. 15 (2022): 8568. http://dx.doi.org/10.3390/ijms23158568.

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Due to (i) the simultaneous presence of Helicobacter pylori (ulcer-induced bacteria) and Candida albicans in the stomach and (ii) the possibility of prokaryotic–eukaryotic endosymbiosis (intravacuolar H. pylori in the yeast cells) under stresses, we tested this symbiosis in vitro and in vivo. To that end, intravacuolar H. pylori were induced by the co-incubation of C. albicans with H. pylori under several stresses (acidic pH, non-H. pylori-enrichment media, and aerobic environments); the results were detectable by direct microscopy (wet mount) and real-time polymerase chain reaction (PCR). Ind

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Ahmed, Suha Hussein, and Saad Abdul Kareem Mohammed. "Helicobacter pylori infection in pregnant women and it’s correlation with the alterations of some trace elements levels in the serum at Maternity Teaching Hospital in Erbil City." Al Mustansiriyah Journal of Pharmaceutical Sciences 18, no. 2 (2018): 41–50. http://dx.doi.org/10.32947/ajps.v18i2.474.

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Helicobacter pylori (H. pylori) is a spiral-shaped pathogenic bacterium found on the human gastric mucosa, Warren and Marshall isolated H pylori for the first time in 1982. It is one of the most common worldwide human infections [1].H. pylori play a vital role in the development of chronic gastritis, gastric ulcer, duodenal ulcer, gastric adenocarcinoma, and gastric mucosa-associated lymphoid tissue lymphoma [2]. The current study included (120) pregnant women, (50) positive anti -H. Pylori Ig and (70) negative anti- H. Pylori Ig with pregnancy for first, second and third semester at mean age/

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*Dr., Bilal Ahmed Baig *Dr. Allah Yar Khan *Dr. Muhammad Maaz Ali. "FREQUENCY OF HELICOBACTER PYLORI INFECTION IN PATIENTS OF DYSPEPSIA PRESENTED IN THE GASTROENTEROLOGY DEPARTMENT OF HOLY FAMILY HOSPITAL RAWALPINDI." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 11 (2018): 12825–29. https://doi.org/10.5281/zenodo.1493049.

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<strong><em>Objective:</em></strong><em> To determine the frequency of H. pylori detection in dyspeptic patients and its association with gastro-duodenal pathologies confirmed on histology.</em> <strong><em>Study Design: </em></strong><em>A Retrospective Study.</em> <strong><em>Place and Duration: </em></strong><em>In the Gastroenterology Department of Holy Family Hospital, Rawalpindi for one year duration from June 2017 to June 2018.</em> <strong><em>Methods:</em></strong><em> Upper GI endoscopy was performed in 100 adult cases with sequential dyspepsia. For histology, biopsy was taken from A

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