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Journal articles on the topic 'H1 O157'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Rump, Lydia V., Narjol Gonzalez-Escalona, Wenting Ju, et al. "Genomic Diversity and Virulence Profiles of Historical Escherichia coli O157 Strains Isolated from Clinical and Environmental Sources." Applied and Environmental Microbiology 81, no. 2 (2014): 569–77. http://dx.doi.org/10.1128/aem.02616-14.

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ABSTRACTEscherichia coliO157:H7 is, to date, the majorE. coliserotype causing food-borne human disease worldwide. Strains of O157 with other H antigens also have been recovered. We analyzed a collection of historic O157 strains (n= 400) isolated in the late 1980s to early 1990s in the United States. Strains were predominantly serotype O157:H7 (55%), and various O157:non-H7 (41%) serotypes were not previously reported regarding their pathogenic potential. Although lacking Shiga toxin (stx) andeaegenes, serotypes O157:H1, O157:H2, O157:H11, O157:H42, and O157:H43 carried several virulence factor

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2

A., R. Rabia, N. Wambura P., I. Kimera S., H. Mdegela R., Mzula A., and A. Khamis F. "Phenotypic Characterisation of Escherichia coli Isolates from Fish, Diarrheic and Healthy Children in Zanzibar, Tanzania." International Journal of TROPICAL DISEASE & Health 24, no. 3 (2017): 1–11. https://doi.org/10.9734/IJTDH/2017/34262.

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<strong>Aims: </strong>This study was carried out with the objective of investigating <em>E. coli</em> virulence factors, antibiotic sensitivity, presence of extended-spectrum-β-lactamase [ESBL] and serotype H1 O157 in <em>E. coli</em> from fish foods in comparison with those from healthy and diarrheic children in Zanzibar. <strong>Study Design:</strong> Repeated cross sectional design was used to collect samples from fish, vendors and fish consumers through the seasons. Cross sectional design was used to collect children faecal samples from Mnazimmoja referral hospital. <strong>Place and Dura

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3

Chen, Chen, Patrick Bales, Julia Greenfield, Ryan D. Heselpoth, Daniel C. Nelson, and Osnat Herzberg. "Crystal Structure of ORF210 from E. coli O157:H1 Phage CBA120 (TSP1), a Putative Tailspike Protein." PLoS ONE 9, no. 3 (2014): e93156. http://dx.doi.org/10.1371/journal.pone.0093156.

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4

Chen, Y. C., Y. S. Lai, D. J. H. Shyu, et al. "C-Terminal Part of Glutamate-Ammonia-Ligase Adenyltransferase Gene Identified by RAPD-HRM with 3H Primer for E. Coli Screening." Folia Biologica 65, no. 2 (2019): 88–100. http://dx.doi.org/10.14712/fb2019065020088.

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A single random oligonucleotide 3H primer has been previously applied in random-amplified- polymorphic-DNA (RAPD)-PCR to distinguish stocked bacteria E. coli within a cocktail mixture also containing Enterococcus faecalis, Bifidobacterium longum and Ruminococcus gnavus. In this study, we demonstrate that a 702 base pair (bp) gene fragment can be amplified as a unique pattern by RAPD-PCR using a 3H primer in human faeces containing E. coli. This unique 702 bp amplicon contained a 687 bp gene fragment identified as the C-terminal region of the glutamate-ammonia-ligase adenyltransferase (glnE) ge

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5

Prats, Guillem, Ferran Navarro, Beatriz Mirelis, et al. "Escherichia coli Serotype O15:K52:H1 as a Uropathogenic Clone." Journal of Clinical Microbiology 38, no. 1 (2000): 201–9. http://dx.doi.org/10.1128/jcm.38.1.201-209.2000.

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ABSTRACT To clarify the clinical and bacteriological correlates of urinary-tract infection (UTI) due to Escherichia coli O15:K52:H1, during a 1-year surveillance period we prospectively screened all 1,871 significant E. coli urine isolates at the Hospital de la Santa Creu i Sant Pau, Barcelona, Spain, for this serotype and assessed the epidemiological features of community-acquired UTI due to E. coli O15:K52:H1 versus other E. coli serotypes. We also compared the 25 O15:K52:H1 UTI isolates from the present study with 22 O15:K52:H1 isolates from other, diverse geographic locales and with 23 sta

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6

Riley, P. A., E. J. Threlfall, T. Cheasty, K. G. Wooldridge, P. H. Williams, and I. Phillips. "Occurrence of FImeplasmids in multiply antimicrobial-resistantEscherichia coliisolated from urinary tract infection." Epidemiology and Infection 110, no. 3 (1993): 459–68. http://dx.doi.org/10.1017/s0950268800050883.

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SUMMARYPlasmids belonging to the FImeincompatibility group were found in seven different serogroups of multiply antimicrobial-resistantEscherichia coliisolated from patients with urinary tract infection (UTI) and living in south-east London. Although widespread inSalmonellaspp., FImeplasmids have only previously been described inE. coliin a strain of serogroup O15 K52 H1 responsible for an extensive and protracted outbreak of invasive community-acquired infection in south-east London in 1986. Our findings suggest either a wider background occurrence of FImeplasmids inE. coliassociated with UTI

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7

Olesen, B., F. Scheutz, M. Menard, et al. "Three-Decade Epidemiological Analysis of Escherichia coli O15:K52:H1." Journal of Clinical Microbiology 47, no. 6 (2009): 1857–62. http://dx.doi.org/10.1128/jcm.00230-09.

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8

Dalmau, D., F. Navarro, B. Mirelis, J. Blanco, J. Garau, and G. Prats. "Escherichia coli bacteraemia. Serotype O15:K52:H1 as a urinary pathogen." Journal of Hospital Infection 34, no. 3 (1996): 233–34. http://dx.doi.org/10.1016/s0195-6701(96)90073-0.

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9

Moreira dos Santos Carmo, Andréia, Fernanda Miranda Lima, Flávia De Paulo Daurelio, et al. "Frequência laboratorial e resistência antimicrobiana de enteropatógenos bacterianos isolados em crianças na primeira infância na região do ABC Paulista, 2015 - 2016." BEPA. Boletim Epidemiológico Paulista 16, no. 182 (2019): 1–9. http://dx.doi.org/10.57148/bepa.2019.v.16.37691.

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O objetivo deste estudo foi avaliar o perfil dos enteropatógenos bacterianos isolados emcrianças menores de 5 anos durante casos de diarreia em instituições de 4 municípios doEstado de São Paulo, durante 2015 e 2016. A coleta das fezes foi realizada em 107 crianças,78 (72,9%) crianças com diarreia e 29 (27,1%) crianças sem diarreia. A metodologia foicoprocultura, identificação bacteriana e teste de sensibilidade aos antimicrobianos. Quarentae seis das 107 (43%) amostras clínicas apresentaram crescimento de enteropatógenos.Amostras de Escherichia coli enteropatogênicas (EPEC), Escherichia coli

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10

Johnson, James R., Megan Menard, Brian Johnston, Michael A. Kuskowski, Kim Nichol, and George G. Zhanel. "Epidemic Clonal Groups of Escherichia coli as a Cause of Antimicrobial-Resistant Urinary Tract Infections in Canada, 2002 to 2004." Antimicrobial Agents and Chemotherapy 53, no. 7 (2009): 2733–39. http://dx.doi.org/10.1128/aac.00297-09.

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ABSTRACT The extent to which clonal spread contributes to emerging antimicrobial resistance in Escherichia coli is incompletely defined. To address this question within a recent, nationally representative strain collection, three established drug-resistant E. coli clonal groups (i.e., clonal group A, E. coli O15:K52:H1, and sequence type 131 [ST131]) were sought among 199 E. coli urine isolates recovered from across Canada from 2002 to 2004, with stratification by resistance to trimethoprim-sulfamethoxazole (TS) and fluoroquinolones (FQs). The isolates' clonal backgrounds, virulence genotypes,

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11

Koga, Maika, Yumi Nogami, Asami Nakano, et al. "An Outbreak of Food Poisoning Caused by Enteroaggregative <i>Escherichia coli</i> O15 : H1." Japanese Journal of Food Microbiology 38, no. 4 (2021): 153–59. http://dx.doi.org/10.5803/jsfm.38.153.

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12

Johnson, J. R., K. Owens, M. Sabate, and G. Prats. "Rapid and Specific Detection of the O15:K52:H1 Clonal Group of Escherichia coli by Gene-Specific PCR." Journal of Clinical Microbiology 42, no. 8 (2004): 3841–43. http://dx.doi.org/10.1128/jcm.42.8.3841-3843.2004.

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13

Vianello, Marco Aurelio, Brenda Cardoso, Danny Fuentes-Castillo, et al. "International high-risk clone of fluoroquinolone-resistant Escherichia coli O15:H1-D-ST393 in remote communities of Brazilian Amazon." Infection, Genetics and Evolution 91 (July 2021): 104808. http://dx.doi.org/10.1016/j.meegid.2021.104808.

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14

Johnson, J. R., A. L. Stell, T. T. O'Bryan, et al. "Global Molecular Epidemiology of the O15:K52:H1 Extraintestinal Pathogenic Escherichia coli Clonal Group: Evidence of Distribution beyond Europe." Journal of Clinical Microbiology 40, no. 6 (2002): 1913–23. http://dx.doi.org/10.1128/jcm.40.6.1913-1923.2002.

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15

Platell, Joanne L., Rowland N. Cobbold, James R. Johnson, Connie R. Clabots, and Darren J. Trott. "Fluoroquinolone-resistant extraintestinal Escherichia coli clinical isolates representing the O15:K52:H1 clonal group from humans and dogs in Australia." Comparative Immunology, Microbiology and Infectious Diseases 35, no. 4 (2012): 319–24. http://dx.doi.org/10.1016/j.cimid.2012.02.002.

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16

Cortés, Pilar, Vanessa Blanc, Azucena Mora, et al. "Isolation and Characterization of Potentially Pathogenic Antimicrobial-Resistant Escherichia coli Strains from Chicken and Pig Farms in Spain." Applied and Environmental Microbiology 76, no. 9 (2010): 2799–805. http://dx.doi.org/10.1128/aem.02421-09.

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ABSTRACT To ascertain whether on animal farms there reside extended-spectrum β-lactamase (ESBL) and plasmidic class C β-lactamase-producing Escherichia coli isolates potentially pathogenic for humans, phylogenetic analyses, pulsed-field gel electrophoresis (PFGE) typing, serotyping, and virulence genotyping were performed for 86 isolates from poultry (57 isolates) and pig (29 isolates) farms. E. coli isolates from poultry farms carried genes encoding enzymes of the CTX-M-9 group as well as CMY-2, whereas those from pig farms mainly carried genes encoding CTX-M-1 enzymes. Poultry and pig isolat

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17

Cagnacci, S., L. Gualco, E. Debbia, G. C. Schito, and A. Marchese. "European Emergence of Ciprofloxacin-Resistant Escherichia coli Clonal Groups O25:H4-ST 131 and O15:K52:H1 Causing Community-Acquired Uncomplicated Cystitis." Journal of Clinical Microbiology 46, no. 8 (2008): 2605–12. http://dx.doi.org/10.1128/jcm.00640-08.

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18

Mostafavi, Seyyed Khalil Shokouhi, Shahin Najar‐Peerayeh, Ashraf Mohabbati Mobarez, and Mehdi Kardoust Parizi. "Characterization of uropathogenic E. coli O25b‐B2‐ST131, O15:K52:H1, and CGA: Neutrophils apoptosis, serum bactericidal assay, biofilm formation, and virulence typing." Journal of Cellular Physiology 234, no. 10 (2019): 18272–82. http://dx.doi.org/10.1002/jcp.28459.

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19

Sabharwal, Harshana, Christoph Cichon, Tobias A. Ölschläger, Ulrich Sonnenborn, and M. Alexander Schmidt. "Interleukin-8, CXCL1, and MicroRNA miR-146a Responses to Probiotic Escherichia coli Nissle 1917 and Enteropathogenic E. coli in Human Intestinal Epithelial T84 and Monocytic THP-1 Cells after Apical or Basolateral Infection." Infection and Immunity 84, no. 9 (2016): 2482–92. http://dx.doi.org/10.1128/iai.00402-16.

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Bacterium-host interactions in the gut proceed via directly contacted epithelial cells, the host's immune system, and a plethora of bacterial factors. Here we characterized and compared exemplary cytokine and microRNA (miRNA) responses of human epithelial and THP-1 cells toward the prototype enteropathogenicEscherichia coli(EPEC) strain E2348/69 (O127:H6) and the probiotic strainEscherichia coliNissle 1917 (EcN) (O6:K5:H1). Human T84 and THP-1 cells were used as cell culture-based model systems for epithelial and monocytic cells. Polarized T84 monolayers were infected apically or basolaterally

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20

Blanco, J., A. Mora, R. Mamani, et al. "National survey of Escherichia coli causing extraintestinal infections reveals the spread of drug-resistant clonal groups O25b:H4-B2-ST131, O15:H1-D-ST393 and CGA-D-ST69 with high virulence gene content in Spain." Journal of Antimicrobial Chemotherapy 66, no. 9 (2011): 2011–21. http://dx.doi.org/10.1093/jac/dkr235.

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21

Mora, Azucena, Miguel Blanco, Cecilia López, et al. "Emergence of clonal groups O1:HNM-D-ST59, O15:H1-D-ST393, O20:H34/HNM-D-ST354, O25b:H4-B2-ST131 and ONT:H21,42-B1-ST101 among CTX-M-14-producing Escherichia coli clinical isolates in Galicia, northwest Spain." International Journal of Antimicrobial Agents 37, no. 1 (2011): 16–21. http://dx.doi.org/10.1016/j.ijantimicag.2010.09.012.

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22

Elsyaed, Mohamed Sabry Abd Elraheam, and Mary Mounir. "Virulence Factors and Antimicrobial Resistance Patterns of Non-O157 Shiga Toxin-producing Escherichia coli Isolated from Different Sources at Sadat City." Microbiology Research Journal International, May 1, 2020, 64–73. http://dx.doi.org/10.9734/mrji/2020/v30i330205.

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Aims: A great concern directed to non-O157 Shiga toxin-producing Escherichia coli (STEC) serotypes due to their public health importance. Detecting the existence, antimicrobial profiles, and virulence repertoire of different STEC serotypes from animals essential for human food are important. Study Design: This study aimed to investigate the presence of STEC in different hosts, the distribution pattern of stx1, stx2, eaeA, and hlyA genes encoding Shiga toxins 1 and 2, intimin, and enterohemolysin, respectively, and the antimicrobial resistance of the detected serotypes. Results: A t

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23

Alexander, Gill, Dussault Forest, McMahon Tanis, et al. "Characterization of Atypical Shiga Toxin Gene Sequences and Description of Stx2j, a New Subtype." Cournal of Clinical Microbiology 60, no. 3 (2022). https://doi.org/10.5281/zenodo.6637837.

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Shiga toxin (Stx) is the definitive virulence factor of Shiga toxin-producing Escherichia coli (STEC). Stx variants are currently organized into a taxonomic system of three Stx1 (a, c, and d) and seven Stx2 (a, b, c, d, e, f, and g) subtypes. In this study, seven STEC isolates from food and clinical samples possessing stx2 sequences that do not fit current Shiga toxin taxonomy were identified. Genome assemblies of the STEC strains were created from Oxford Nanopore and Illumina sequence data. The presence of atypical stx2 sequences was confirmed by Sanger sequencing, as were Stx2 expression and

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24

Alexander, Gill, Dussault Forest, McMahon Tanis, et al. "Characterization of Atypical Shiga Toxin Gene Sequences and Description of Stx2j, a New Subtype." Journal of Clinical Microbiology, March 17, 2022. https://doi.org/10.5281/zenodo.6364420.

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Shiga toxin (Stx) is the definitive virulence factor of Shiga toxin-producing Escherichia coli (STEC). Stx variants are currently organized into a taxonomic system of three Stx1 (a, c, and d) and seven Stx2 (a, b, c, d, e, f, and g) subtypes. In this study, seven STEC isolates from food and clinical samples possessing stx2 sequences that do not fit current Shiga toxin taxonomy were identified. Genome assemblies of the STEC strains were created from Oxford Nanopore and Illumina sequence data. The presence of atypical stx2 sequences was confirmed by Sanger sequencing, as were Stx2 expression and

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25

Patil, Sandip, Liu Pai, Hongyu Chen, et al. "Genetic landscape of ESBL producing international clone ST410 of Escherichia coli from pediatric infections in Shenzhen, China." Frontiers in Cellular and Infection Microbiology 14 (September 11, 2024). http://dx.doi.org/10.3389/fcimb.2024.1403234.

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BackgroundThe emergence of ESBLs producing cephalosporin-resistant Escherichia coli isolates poses a threat to public health. This study aims to decipher the genetic landscape and gain insights into ESBL-producing E. coli strains belonging to the high-risk clone ST410 from pediatric patients.Methods29 E. coli ST410 isolates were collected from young children and subjected to antimicrobial susceptibility testing, Whole-genome sequencing (WGS), serotype analysis, MLST, ESBL genes, virulence genes, and plasmid profiling.ResultsAntimicrobial susceptibility testing demonstrated a high level of resi

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