Relevant bibliographies by topics / H9c2 cell differentiation

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  1. Journal articles
  2. Dissertations / Theses
  3. Conference papers

Academic literature on the topic 'H9c2 cell differentiation'

Author: Grafiati
Published: 4 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "H9c2 cell differentiation"

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Kankeu, Cynthia, Kylie Clarke, Delphi Van Haver, et al. "Quantitative proteomics and systems analysis of cultured H9C2 cardiomyoblasts during differentiation over time supports a ‘function follows form’ model of differentiation." Molecular Omics 14, no. 3 (2018): 181–96. http://dx.doi.org/10.1039/c8mo00036k.

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Sucharov, Carmen C., Stephen Langer, Michael Bristow, and Leslie Leinwand. "Shuttling of HDAC5 in H9C2 cells regulates YY1 function through CaMKIV/PKD and PP2A." American Journal of Physiology-Cell Physiology 291, no. 5 (2006): C1029—C1037. http://dx.doi.org/10.1152/ajpcell.00059.2006.

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YY1 is a transcription factor that can activate or repress transcription of a variety of genes and is involved in several developmental processes. YY1 is a repressor of transcription in differentiated H9C2 cells and in neonatal cardiac myocytes but an activator of transcription in undifferentiated H9C2 cells. We now present a detailed analysis of the functional domains of YY1 when it is acting as a repressor or an activator and identify the mechanism whereby its function is regulated in the differentiation of H9C2 cells. We show that histone deacetylase 5 (HDAC5) is localized to the cytoplasm
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Lama, Stefania, Vincenzo Monda, Maria Rosaria Rizzo, et al. "Cardioprotective Effects of Taurisolo® in Cardiomyoblast H9c2 Cells under High-Glucose and Trimethylamine N-Oxide Treatment via De Novo Sphingolipid Synthesis." Oxidative Medicine and Cellular Longevity 2020 (November 12, 2020): 1–11. http://dx.doi.org/10.1155/2020/2961406.

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In addition to high plasma glucose, increased levels of trimethylamine N-oxide (TMAO) have been found in obese subjects, where are considered as a novel risk factor for cardiovascular diseases. The present study aimed to investigate the effect of a novel nutraceutical formulation based on grape polyphenols (registered as Taurisolo®) in counteracting TMAO- and high glucose (HG)-induced cytotoxicity in cardiomyoblast H9c2 cells. Cell damage was induced with HG (HG-H9c2) and HG+TMAO (THG-H9c2); both experimental cell models were, thus, incubated for 72 h in the presence or absence of Taurisolo®.
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Ashrafi, Elham, Milica Radisic, and Janet A. W. Elliott. "Systematic cryopreservation study of cardiac myoblasts in suspension." PLOS ONE 19, no. 3 (2024): e0295131. http://dx.doi.org/10.1371/journal.pone.0295131.

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H9c2 myoblasts are a cell line derived from embryonic rat heart tissue and demonstrate the ability to differentiate to cardiac myotubes upon reduction of the serum concentration (from 10% to 1%) and addition of all-trans retinoic acid in the growth medium. H9c2 cells are increasingly being used as an easy-to-culture proxy for some functions of cardiomyocytes. The cryobiology of cardiac cells including H9c2 myoblasts has not been studied as extensively as that of some cell types. Consequently, it is important to characterize the cryobiological response and systematically develop well-optimized
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Zevolis, Evangelos, Anastassios Philippou, Athanasios Moustogiannis, Antonios Chatzigeorgiou, and Michael Koutsilieris. "The Effects of Mechanical Loading Variations on the Hypertrophic, Anti-Apoptotic, and Anti-Inflammatory Responses of Differentiated Cardiomyocyte-like H9C2 Cells." Cells 11, no. 3 (2022): 473. http://dx.doi.org/10.3390/cells11030473.

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Cardiomyocytes possess the ability to respond to mechanical stimuli by adapting their biological functions. This study investigated cellular and molecular events in cardiomyocyte-like H9C2 cells during differentiation as well as the signalling and gene expression responses of the differentiated cells under various mechanical stretching protocols in vitro. Immunofluorescence was used to monitor MyHC expression and structural changes during cardiomyoblast differentiation. Moreover, alterations in the expression of cardiac-specific markers, cell cycle regulatory factors, MRFs, hypertrophic, apopt
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Chen, Lena, Catherine S. W. Choi, Juan C. Sanchez-Arias, Laura T. Arbour, and Leigh Anne Swayne. "Ankyrin-B p.S646F undergoes increased proteasome degradation and reduces cell viability in the H9c2 rat ventricular cardiomyoblast cell line." Biochemistry and Cell Biology 98, no. 2 (2020): 299–306. http://dx.doi.org/10.1139/bcb-2019-0082.

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Ankyrin-B (AnkB) is scaffolding protein that anchors integral membrane proteins to the cardiomyocyte cytoskeleton. We recently identified an AnkB variant, AnkB p.S646F (ANK2 c.1937 C>T) associated with a phenotype ranging from predisposition for cardiac arrhythmia to cardiomyopathy. AnkB p.S646F exhibited reduced expression levels in the H9c2 rat ventricular-derived cardiomyoblast cell line relative to wildtype AnkB. Here, we demonstrate that AnkB is regulated by proteasomal degradation and proteasome inhibition rescues AnkB p.S646F expression levels in H9c2 cells, although this effect is n
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Pagano, M., S. Naviglio, A. Spina, et al. "Differentiation of H9c2 cardiomyoblasts: The role of adenylate cyclase system." Journal of Cellular Physiology 198, no. 3 (2004): 408–16. http://dx.doi.org/10.1002/jcp.10420.

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Giacomo, Viviana di, Monica Rapino, Silvia Sancilio та ін. "PKC-δ signalling pathway is involved in H9c2 cells differentiation". Differentiation 80, № 4-5 (2010): 204–12. http://dx.doi.org/10.1016/j.diff.2010.06.002.

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Sumi, Daigo, Kazusa Abe, and Seiichiro Himeno. "Arsenite retards the cardiac differentiation of rat cardiac myoblast H9c2 cells." Biochemical and Biophysical Research Communications 436, no. 2 (2013): 175–79. http://dx.doi.org/10.1016/j.bbrc.2013.05.069.

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Hu, Wei-Syun, Wei-Yu Liao, Chin-Hsien Chang, and Tung-Sheng Chen. "Paracrine IGF-1 Activates SOD2 Expression and Regulates ROS/p53 Axis in the Treatment of Cardiac Damage in D-Galactose-Induced Aging Rats after Receiving Mesenchymal Stem Cells." Journal of Clinical Medicine 11, no. 15 (2022): 4419. http://dx.doi.org/10.3390/jcm11154419.

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Aging is one of the causative agents associated with heart failure. Cell-based therapies show potential in the treatment of cardiac aging due to the characteristics of stem cells, including differentiation and the paracrine effect. This study aimed to investigate in detail the mechanism related to biomolecules released from mesenchymal stem cells in the treatment of cardiac aging. In vitro and in vivo models were designed to explore the above hypothesis. Experimental results from the in vitro model indicated that the elevation of oxidative stress, the expression of aging marker p53, and the su
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Dissertations / Theses on the topic "H9c2 cell differentiation"

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Loiselle, Julie Jennifer. "Analysis of RBM5 and RBM10 expression throughout H9C2 skeletal and cardiac muscle cell differentiation." Thesis, Laurentian University of Sudbury, 2013. https://zone.biblio.laurentian.ca/dspace/handle/10219/2032.

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RNA Binding Motif (RBM) domain proteins RBM5 and RBM10 have been shown to influence apoptosis, cell cycle arrest and splicing in transformed cells. In this study, RBM5 and RBM10 were examined in non-transformed cells in order to gain a wider range of knowledge regarding their function. Expression of Rbm5 and Rbm10, as well as select splice variants, was examined at the mRNA and protein level throughout H9c2 skeletal and cardiac myoblast differentiation. Results suggest that Rbm5 and Rbm10 may (a) be involved in regulating cell cycle arrest and apoptosis during skeletal myoblast differentiation
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Humphrey, Peter Saah. "Signal transduction mechanisms for stem cell differentation into cardiomyocytes." Thesis, University of Hertfordshire, 2009. http://hdl.handle.net/2299/3760.

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Cardiovascular diseases are among the leading causes of death worldwide and particularly in the developed World. The search for new therapeutic approaches for improving the functions of the damaged heart is therefore a critical endeavour. Myocardial infarction, which can lead to heart failure, is associated with irreversible loss of functional cardiomyocytes. The loss of cardiomyocytes poses a major difficulty for treating the damaged heart since terminally differentiated cardiomyocytes have very limited regeneration potential. Currently, the only effective treatment for severe heart failure i
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Oliveira, Vilma Marisa Arrojado Soares Sardão. "H9C2 myoblasts as a tool to study doxorubicin-induced cardiomyopathy : mechanisms of cell death and relevanceof cell differentiation." Doctoral thesis, 2008. http://hdl.handle.net/10316/9633.

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Conference papers on the topic "H9c2 cell differentiation"

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Anene-Nzelu, Chukwuemeka G., Deepak Choudhury, Huipeng Li, et al. "Gratings on a Dish: A Scalable Cell Alignment Substrate on Optical Media." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14109.

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Cell alignment by underlying topographical cues has been shown to affect important biological processes such as differentiation and functional maturation in vitro. However, the routine use of cell culture substrates with micro/nano-topographies is currently hampered by the high cost and specialized facilities required to produce these substrates. Here we present commercially available optical media as substrates for aligning cells in culture. These optical media, including CD-R, DVD-R and optical grating, allow different cell types to attach, align and grow on them. This cytoskeletal reorganiz
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