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Books on the topic 'Haemodynamics'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Crispian, Oates, ed. Cardiovascular haemodynamics and Doppler waveforms explained. Greenwich Medical Media, 2001.

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2

Cold, Georg E., and Niels Juul, eds. Monitoring of Cerebral and Spinal Haemodynamics During Neurosurgery. Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-77873-8.

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3

Gwynn, Brian Rodney. The influence of arteriovenous fistulae on in-situ vein graft haemodynamics. University of Birmingham, 1987.

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4

Lingman, Göran. Human fetal haemodynamics: Ultrasonic assessment in normal pregnancy and in fetal cardiac arrhythmia. [s.n.], 1985.

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5

Huang, Chunlong. A study of the action of hormones and drugs on regional haemodynamics of the rat kidney. University of Birmingham, 1994.

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6

Räsänen, Juha. Fetal heart and haemodynamics in risk pregnancies and their changes after maternal vasoactive agents: An ultrasonic study. [University of Oulu], 1992.

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7

T, Mathie Robert, and Griffith Tudor M, eds. The haemodynamic effects of nitric oxide. Imperial College Press, 1999.

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8

Mäkivirta, Aki. Use of the median filter in haemodynamic monitoring. Technical Research Centre of Finland, 1992.

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9

Dillon, A. Haemodynamic profiles and the critically ill patient: A practical guide. Edited by Coombs M. A and Lyon J. BIOS Scientific Publishers, 1997.

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10

Franceschi, Claude. Conservative haemodynamic cure of incompetent and varicose veins in ambulatory patients. Éditions de l'Armançon, 1993.

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11

Pesola, Maija. The haemodynamic effects induced by endurance training and nandrolone in the rat. University of Oulu, 1988.

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12

Partanen, Juhani. Cardiovascular responses induced by haemodynamic interventions and inotropics: A series of noninvasive studies. University Central Hospital, 1989.

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13

Fry, J. E. Effect of smoking on arterial stiffness and haemodynamic parameters in type 1 diabetes mellitus. University of Surrey Roehampton, 2004.

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14

Alfred Benzon Symposium (37th 1993 Royal Danish Academy of Sciences and Letters). Brain lesions in the newborn: Hypoxic and haemodynamic pathogenesis : Alfred Benzon Symposium 37 : proceedings of a symposium held at the Royal Society of Sciences and Letters, 15-19 August 1993. Edited by Greisen Gorm, Larsen J[0]rgen Falck, Lou Hans C, and Alfred Benzon Foundation. Munksgaard, 1994.

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15

Oates, Crispian. Cardiovascular Haemodynamics and Doppler Waveforms Explained. University of Cambridge ESOL Examinations, 2008.

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16

Cardiovascular Haemodynamics and Doppler Waveforms Explained. Greenwich Medical Media, 2001.

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17

Badano, Luigi P., and Denisa Muraru. Assessment of right heart function and haemodynamics. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0011.

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Assessment of right ventricular (RV) size, function, and haemodynamics has been challenging because of its unique cavity geometry. Conventional two-dimensional assessment of RV function is often qualitative. Doppler methods involving tricuspid inflow and pulmonary artery flow velocities, which are influenced by changes in pre- and afterload conditions, may not provide robust prognostic information for clinical decision making. Recent advances in echocardiographic assessment of the RV include tissue Doppler imaging, speckle-tracking imaging, and volumetric three-dimensional imaging, but they ne

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18

Juul, Niels, Georg E. Cold, M. Rasmussen, et al. Monitoring of Cerebral and Spinal Haemodynamics during Neurosurgery. Springer, 2010.

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19

Juul, Niels, Georg E. Cold, M. Rasmussen, A. Tankisi, and H. Bundgaard. Monitoring of Cerebral and Spinal Haemodynamics During Neurosurgery. Springer London, Limited, 2008.

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20

Monitoring Of Cerebral And Spinal Haemodynamics During Neurosurgery. Springer, 2008.

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21

Clarke, Howard Martyn. The haemodynamics and viability of skin and muscle flaps. 1985.

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22

Page, Clive, M. J. Parnham, and Jacques Bruinvels. Haemodynamics and Immune Defense: Discoveries in Pharmacology Volume 3. Elsevier Science & Technology Books, 2023.

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23

Parnham, M. J. J., Clive P. Page, and Jacques Bruinvels. Haemodynamics and Immune Defense: Discoveries in Pharmacology Volume 3. Elsevier Science & Technology Books, 2023.

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24

McCormick, Peter Aiden. The effect of food and drugs on portal haemodynamics in portal hypertension. 1991.

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25

Thorne, Sara, and Paul Clift, eds. Cardiac catheterization. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199228188.003.0004.

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Introduction 42Indications for catheterization 44Precatheterization care 46Calculations 48The purpose of cardiac catheterization in this patient group is to gain information about complex anatomy and haemodynamics, especially with respect to PA pressure and vascular resistance. In order to gain complete angiographic and haemodynamic information, studies are best performed in specialist units. In recent years, catheterization has been increasingly combined with percutaneous interventional procedures, reducing the need for further cardiac surgery in some individuals. ...

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26

Goltsov, Alexey, Viktor V. Sidorov, Sergei G. Sokolovski, and Edik Rafailov, eds. Advanced Non-invasive Photonic Methods for Functional Monitoring of Haemodynamics and Vasomotor Regulation in Health and Diseases. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-760-7.

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27

Vieillard-Baron, Antoine. Right ventricular function in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0135.

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Under normal conditions, the right ventricle (RV) virtually acts as a passive conduit. In critically-ill patients many situations induce uncoupling between the right ventricle and pulmonary circulation, leading to RV systolic dysfunction, then failure. Mechanical ventilation has a major impact by decreasing RV preload, but also significantly increasing RV afterload. RV function should thus always be interpreted and re-evaluated in the light of respiratory mechanics and ventilator settings. RV systolic function is key to the patient’s haemodynamic profile and must be monitored to achieve optima

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28

Archer, Nick, and Nicky Manning. Neurodevelopment and fetal cardiac disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198766520.003.0026.

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This chapter explores neurodevelopment and fetal cardiac disease, including an introduction and discussion on haemodynamics, specific cardiac lesions, methods of assessment, prevention or limitation, and counselling.

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29

Dyer, Robert A., Michelle J. Arcache, and Eldrid Langesaeter. The aetiology and management of hypotension during spinal anaesthesia for caesarean delivery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0023.

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The management of hypotension during spinal anaesthesia for caesarean delivery remains a challenge for anaesthesiologists. Close control of maternal haemodynamics is of great importance for maternal and fetal safety, as well as maternal comfort. Haemodynamic responses to spinal anaesthesia are influenced by aortocaval compression, the baricity and dose of local anaesthetic and opioid employed, the rational use of fluids, and the goal-directed use of vasopressors. The most common response to spinal anaesthesia is hypotension and an increased heart rate, which reflects a decreased systemic vascu

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30

Ebm, Claudia, and Andrew Rhodes. Post-operative fluid and circulatory management in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0363.

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Fluid and circulatory management is an integral part of the peri-operative care of critically-ill patients. Precisely estimating the volumetric needs of post-operative patients remains difficult. While the majority of patients tolerate intra-operative fluid loss easily, patients with reduced physiological reserve present more of a challenge. Targeting specific physiological goals and optimizing haemodynamics with fluids and inotropes, means outcomes of these patients can be improved. This approach is often referred as goal-directed therapy (GDT). ‘Individualized goal-directed therapy’ can vary

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31

Thorne, Sara, and Sarah Bowater. Cardiac catheterization. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759959.003.0005.

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The purpose of cardiac catheterization in patients with congenital heart disease is to gain information about complex anatomy and haemodynamics, especially with respect to pulmonary artery pressure and vascular resistance. This chapter outlines indications for cardiac catheterization, precatheterization care, normal values and calculations, and catheter interventions in ACHD.

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32

Sidhu, Kulraj S., Mfonobong Essiet, and Maxime Cannesson. Cardiac and vascular physiology in anaesthetic practice. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0001.

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This chapter discusses key components of cardiovascular physiology applicable to clinical practice in the field of anaesthesiology. From theory development to ground-breaking innovations, the history of cardiac and vascular anatomy, as well as physiology, is presented. Utilizing knowledge of structure and function, parameters created have allowed adequate patient clinical assessment and guided interventions. A review of concepts reveals the impact of multiple physiological variables on a patient’s haemodynamic state and the need for more accurate and efficient measurements. In particular, it i

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33

Colebourn, Claire, and Jim Newton. The pericardium. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757160.003.0006.

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This chapter looks in detail at the anatomy and physiology of the pericardium and the implications of pericardial fluid on haemodynamics in the critically unwell patient. Specific focus is given to making the diagnosis of tamponade in the spontaneously ventilating patient, compared to patients who are intubated and mechanically ventilated.

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34

(Editor), X. Y. Xu, and M. W. Collins (Editor), eds. Haemodynamics of Arterial Organs : Comparison of Computational Predictions with In Vitro and In Vivo Data (Advances in Computational Bioengineering Vol 1). WIT Press (UK), 1999.

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35

Fromy, Bérengère Michèle. Experimental and statistical analyses of the effects of a uniform positive pressure applied to the lower limb in humans on vascular haemodynamics. 1997.

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36

Schairer, John R., and Steven J. Keteyian. Pathophysiology and causes of pericardial tamponade. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0166.

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Pericardial disease leading to pericardial effusion (PEF) is a common clinical disorder. The most common causes are viral infections, metastatic cancer, renal disease, and bleeding disorders. PEF that accumulates slowly can become quite large before haemodynamic embarrassment occurs, while PEF that accumulates rapidly from trauma or aortic dissection can be small,yet cause haemodynamic embarrassment. As the PEF increases in size, the pressure in the pericardial space increases, leading to a decrease in atrial and ventricular chamber sizes, and limiting filling of the chambers. Ultimately, card

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37

Lameire, Norbert. Prevention of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0225_update_001.

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This chapter describes the most important non-pharmacologic interventions in the prevention of acute kidney injury. Specific for bypass surgery is the choice between on- versus off-pump surgery in coronary artery bypass grafting. Other interventions include optimization and maintenance of oxygen delivery and of cardiovascular haemodynamics; careful selection of fluid therapy, particularly in septic shock and the postoperative period; possible application of preoperative remote ischaemic preconditioning; maintaining euglycaemia, and application of lung-protective artificial ventilation.

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38

Kallmeyer, Andrea, José Luis Zamorano, G. Locorotondo, Madalina Garbi, José Juan Gómez de Diego, and Miguel Ángel García Fernández. Non-invasive haemodynamic assessment. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0005.

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The diagnostic power of two-dimensional (2D) echocardiography resides not only in its capability of providing anatomical information and of studying myocardial contractile function, but also in the possibility of performing a non-invasive haemodynamic assessment. Such non-invasive haemodynamic assessment is the subject of this chapter.2D echocardiography, colour flow imaging, and Doppler modality make this haemodynamic assessment possible, by studying the following parameters: ◆ Blood flow velocities. ◆ Transvalvular pressure gradients. ◆ Valvular areas. ◆ Stroke volume, regurgitant volume, an

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39

Haemodynamic Monitoring And Manipulation. M&K; Update Ltd, 2009.

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40

Griffith, Tudor M. Haemodynamic Effects of Nitric Oxide. Imperial College Press, 1999.

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41

Lancellotti, Patrizio, and Bernard Cosyns. The Standard Transthoracic Echo Examination. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0002.

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Functional imaging by modern echocardiography offers a variety of methods to assess regional and global myocardial function beyond classic dimension, volume and ejection fraction measurements. This chapter shows how various modalities of Doppler echocardiography can be used for assessment of valves, haemodynamics, and coronary flow reserve. It also provides information on myocardial function can be extracted from echo images using a tissue Doppler or speckle tracking approach. 3Dechocardiography provides real-time 3D images of the heart in motion. Various types of examination and quantificatio

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42

Mathie, Robert T., and Tudor M. Griffith. The Haemodynamic Effects of Nitric Oxide. PUBLISHED BY IMPERIAL COLLEGE PRESS AND DISTRIBUTED BY WORLD SCIENTIFIC PUBLISHING CO., 1999. http://dx.doi.org/10.1142/p068.

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43

(Editor), Robert T. Mathie, and Tudor M. Griffith (Editor), eds. The Haemodynamic Effects of Nitric Oxide. Imperial College Press, 1999.

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44

Alderson, Helen, Constantina Chrysochou, James Ritchie, and Philip A. Kalra. Ischaemic nephropathy. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0212.

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Ischaemic nephropathy describes loss of renal function or renal parenchyma due to stenosis or occlusion of the renal artery or its branches. In the Western world, this is usually the result of atherosclerotic renovascular disease, but other aetiologies include arteritis, embolic disease, dissection, and fibromuscular disease.Chronic kidney disease is the most common manifestation of ischaemic nephropathy, but hypertension, flash pulmonary oedema, sensitivity to angiotensin blockade, and sensitivity of glomerular filtration rate to blood pressure reduction are all possible manifestations of occ

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45

Knight, Simon R., and Rutger J. Ploeg. Immediate post-transplant care and surgical complications. Edited by Jeremy R. Chapman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0280_update_001.

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Care of the post-transplant kidney patient is complex and requires multidisciplinary team working. Careful attention is paid to haemodynamics, fluid balance, microbiology, drug prescription, and patient instruction. Delays in, or reduction of, graft function should be investigated and treated immediately to ensure long-term graft survival. Because complications do occur, they must be recognized early and dealt with promptly. The nature of the transplant operation and the need for immunosuppression mean that the complications differ from those of ordinary general surgical patients, and so requi

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46

Gasser, T. Christian. Physical processes in the vessel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0003.

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Evolution has developed a complex cardiovascular system, the analysis of which involves many physical disciplines. Specifically, cardiovascular function critically depends on the proper interaction between blood and the vessel wall, such that haemodynamics-based biomechanical factors are a common denominator of cardiovascular pathologies. This chapter reviews biomechanics-related physical processes in the vessel. Specifically, mechanical load transition mechanisms in blood and the vessel wall, blood-wall interaction phenomena, as well as simple analytical solutions to Newton’s second law of me

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47

Waldmann, Carl, Neil Soni, and Andrew Rhodes. Renal disorders. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199229581.003.0019.

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Prevention of acute renal failure 312Diagnosis of acute renal failure 314Acute kidney injury (AKI) often complicates the course of critical illness and was previously considered as a marker rather than a cause of adverse outcomes, it is independently associated with an increase in both morbidity and mortality. The major causes of AKI in the ICU include hypoperfusion, sepsis and direct nephrotoxicity, with the common aetiology believed to be a change in intrarenal haemodynamics with resultant acute tubular dysfunction and oxidant stress. Treatment of established acute renal failure in the ICU e

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48

Nešković, Aleksandar N., and Andreas Hagendorff. Echocardiography in the emergency room. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0026.

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Echocardiography can provide rapid and accurate assessment of cardiac morphology and haemodynamics under stressful conditions in the emergency room (ER). Using this information, critical decisions regarding management of cardiovascular emergencies and the critically ill are made. To avoid potentially catastrophic errors with medicolegal consequences, adequate education and experience in echocardiography and cardiology are required and teamwork is encouraged. In addition, emergency cases must be well documented and this documentation stored and retrievable. Transthoracic echocardiography is the

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49

Dhaun, Neeraj, and David J. Webb. Endothelins and their antagonists in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0114_update_001.

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The endothelins (ETs) are a family of related peptides of which ET-1 is the most powerful endogenous vasoconstrictor and the predominant isoform in the cardiovascular and renal systems. The ET system has been widely implicated in both cardiovascular disease and chronic kidney disease (CKD). ET-1 contributes to the pathogenesis and maintenance of hypertension and arterial stiffness, as well endothelial dysfunction and atherosclerosis. By reversal of these effects, ET antagonists, particularly those that block ETA receptors, may reduce cardiovascular risk. In CKD patients, antagonism of the ET s

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50

van den Bosch, Annemien E., Luigi P. Badano, and Julia Grapsa. Right ventricle and pulmonary arterial pressure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0023.

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Right ventricular (RV) performance plays an important role in the morbidity and mortality of patients with left ventricular dysfunction, congenital heart disease, and pulmonary hypertension. Assessment of RV size, function, and haemodynamics has been challenging because of its complex geometry. Conventional two-dimensional echocardiography is the modality of choice for assessment of RV function in clinical practice. Recent developments in echocardiography have provided several new techniques for assessment of RV dimensions and function, include tissue Doppler imaging, speckle-tracking imaging,

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