Journal articles: 'Hafnia alvei'

1

Klapholz, Ari, Klaus-Dieter Lessnau, Benson Huang, Wilfredo Talavera, and John F. Boyle. "Hafnia alvei." Chest 105, no. 4 (1994): 1098–100. http://dx.doi.org/10.1378/chest.105.4.1098.

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Janda, J. Michael, and Sharon L. Abbott. "The Genus Hafnia: from Soup to Nuts." Clinical Microbiology Reviews 19, no. 1 (2006): 12–28. http://dx.doi.org/10.1128/cmr.19.1.12-28.2006.

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SUMMARY The genus Hafnia, a member of the family Enterobacteriaceae, consists of gram-negative bacteria that are occasionally implicated in both intestinal and extraintestinal infections in humans. Despite the fact that the genus currently contains only a single species (H. alvei), more extensive phylogenetic depth (two or more species) is apparent based upon DNA relatedness and 16S rRNA gene sequencing studies. Hafnia causes a variety of systemic infections, including septicemia and pneumonia; however, its role as a gastrointestinal pathogen is controversial. Many of the data supporting a role for hafniae as enteric pathogens were incorrectly attributed to this genus rather than to the actual pathogen, Escherichia albertii. There are numerous gaps in our understanding of this genus, including ecologic habitats and population genetics, disease-producing role in animals, phenetic and genetic methods useful in distinguishing genomospecies within the H. alvei complex, and bona fide pathogenicity factors.

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Rodríguez, Eugenio, José Antonio Prieto, Jordi Bugés, and M. a. Teresa Bastida. "Empiema por Hafnia alvei." Medicina Clínica 114, no. 3 (2000): 119. http://dx.doi.org/10.1016/s0025-7753(00)71214-0.

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Ramos-Vivas, José. "Microbiología de Hafnia alvei." Enfermedades Infecciosas y Microbiología Clínica 38 (January 2020): 1–6. http://dx.doi.org/10.1016/j.eimc.2020.02.001.

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Huys, Geert, Margo Cnockaert, Sharon L. Abbott, J. Michael Janda, and Peter Vandamme. "Hafnia paralvei sp. nov., formerly known as Hafnia alvei hybridization group 2." International Journal of Systematic and Evolutionary Microbiology 60, no. 8 (2010): 1725–28. http://dx.doi.org/10.1099/ijs.0.018606-0.

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It has been shown previously, based largely on DNA–DNA hybridizations and partial 16S rRNA gene sequencing, that Hafnia alvei is genotypically heterogeneous and consists of at least two DNA hybridization groups (HGs). In the present study, the taxonomic status of H. alvei HGs 1 and 2 was reassessed. A panel of 24 reference strains and isolates previously assigned to one of the two HGs in H. alvei was subjected to (GTG)5-PCR fingerprinting; this resulted in the delineation of two (GTG)5-PCR clusters in perfect accordance with the respective HG designations. Based on full 16S rRNA gene sequencing of a selection of reference strains, H. alvei HGs 1 and 2 showed internal sequence similarities of 99.8 and 99.5 %, respectively. Between the two groups, sequence similarities ranged from 98.8 to 99.1 %. Mean DNA–DNA hybridization values of 74.7–99.9 % were obtained within each of the two HGs, whereas cross-hybridizations between members of H. alvei HG 1 (including ATCC 13337T) and HG 2 revealed only 32.7–48.7 % DNA–DNA hybridization. Previously published and new phenotypic data revealed that a combination of malonate assimilation and β-glucosidase activity enabled correct assignment of Hafnia isolates to one of the two HGs. Collectively, taxonomic data from this study confirm that H. alvei comprises at least two taxa at the species level, of which HG 1 corresponds to H. alvei sensu stricto because it includes the type strain ATCC 13337T. Strains formerly classified as members of H. alvei HG 2 represent a novel species, for which the name Hafnia paralvei sp. nov. is proposed; ATCC 29927T (=CDC 4510-73T =LMG 24706T), the former reference strain of H. alvei HG 2, is designated the type strain.

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Basani, Laxman, and Roja Aepala. "Hafnia alvei causing late onset sepsis in neonates: report of two cases and review of literature." International Journal of Contemporary Pediatrics 5, no. 2 (2018): 673. http://dx.doi.org/10.18203/2349-3291.ijcp20180580.

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Hafnia alvei, a Gram negative motile bacillus that belongs to Enterobacteriaceae family is rarely associated with infection in pediatric patients and is exceptionally rare in the neonatal period. H. alvei is ubiquitous in the environment, causing infections in debilitated and immuno-compromised patients with few cases being reported in neonates. We report two cases of late onset sepsis in term neonates caused by H. alvei that were successfully treated in our unit. To the best of our knowledge, infection due to H. alvei has not been reported in neonates from India. Hafnia alvei causes infection rarely in neonates. Because it can cause nosocomial outbreaks, awareness regarding this uncommon pathogen and initiation of appropriate antibiotic therapy improves the outcome and prevents mortality.

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Fazal, B. A., J. E. Justman, G. S. Turett, and E. E. Telzak. "Community-Acquired Hafnia alvei Infection." Clinical Infectious Diseases 24, no. 3 (1997): 527–28. http://dx.doi.org/10.1093/clinids/24.3.527.

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8

Thomson, K. S., C. C. Sanders, and J. A. Washington. "Ceftazidime resistance in Hafnia alvei." Antimicrobial Agents and Chemotherapy 37, no. 6 (1993): 1375–76. http://dx.doi.org/10.1128/aac.37.6.1375.

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9

Agustin, E. T., and B. A. Cunha. "Buttock Abscess Due to Hafnia alvei." Clinical Infectious Diseases 20, no. 5 (1995): 1426. http://dx.doi.org/10.1093/clinids/20.5.1426.

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10

Joseph W., B., E. A. Dominguez, and L. C. Preheim. "Hafnia alvei Infection After Liver Transplantation." Clinical Infectious Diseases 24, no. 6 (1997): 1263–64. http://dx.doi.org/10.1093/clinids/24.6.1263.

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11

Fernández Peláez, Juan M., Manuel Vives Soto, Herminia Marqueño Ortega, and Ignacio Goig Abarca. "Peritonitis bacteriana espontánea por Hafnia alvei." Medicina Clínica 116, no. 11 (2001): 437. http://dx.doi.org/10.1016/s0025-7753(01)71857-x.

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12

Savini, Vincenzo, Chiara Catavitello, Marzia Talia, et al. "Isolation of colistin-resistant Hafnia alvei." Journal of Medical Microbiology 58, no. 2 (2009): 278–80. http://dx.doi.org/10.1099/jmm.0.001321-0.

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13

Ramos, A., and D. Dámaso. "Extraintestinal Infection due to Hafnia alvei." European Journal of Clinical Microbiology & Infectious Diseases 19, no. 9 (2000): 708–10. http://dx.doi.org/10.1007/s100960000356.

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14

Savini, Vincenzo, Angela Valentina Argentieri, Roberta Marrollo, et al. "The Italian Hafnia alvei strain LMG 27376 is Hafnia paralvei." Veterinary Microbiology 167, no. 3-4 (2013): 742–43. http://dx.doi.org/10.1016/j.vetmic.2013.07.026.

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15

Gu, Zhengquan, Yongqin Liu, Liang Shen, et al. "Hafnia psychrotolerans sp. nov., isolated from lake water." International Journal of Systematic and Evolutionary Microbiology 65, Pt_3 (2015): 971–74. http://dx.doi.org/10.1099/ijs.0.000049.

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A psychrotolerant, Gram-stain-negative, motile, aerobic, peritrichous bacterium, strain DJC1-1T, was isolated from Lake Dajiaco, Tibetan Plateau, China. The strain was negative for citrate utilization, lipase activity and α-glucosidase, but positive for the Voges–Proskauer reaction and N-acetyl-β-glucosaminidase. 16S rRNA gene sequence analysis indicated that Hafnia paralvei ATCC 29927T, Hafnia alvei ATCC 13337T, Serratia grimesii DSM 30063T and Serratia plymuthica DSM 4540T were the closest relatives of strain DJC1-1T, with similarities of 97.76, 96.80, 97.71 and 97.58 %, respectively. The DNA G+C content of strain DJC1-1T was 53.9 mol%. The predominant fatty acids were C16 : 0 and C17 : 0 cyclo. Based on these characteristics, strain DJC1-1T can be assigned to the genus Hafnia . In DNA–DNA hybridization tests, strain DJC1-1T shared 50.6, 35.1, 36.5 and 18.1 % DNA–DNA relatedness with the type strains of H. paralvei , H. alvei , S. grimesii and S. plymuthica , respectively. The growth temperature ranged from 0 to 40 °C, with optimum growth at 15 °C. Physiological and biochemical tests differentiated strain DJC1-1T from the type strains of recognized species of the genus Hafnia . Therefore, strain DJC1-1T is identified as representing a novel species of the genus Hafnia, for which the name Hafnia psychrotolerans sp. nov. is proposed. The type strain is DJC1-1T ( = JCM 30077T = CGMCC1.12806T).

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16

Rodríguez, L. A., J. Vivas, C. S. Gallardo, F. Acosta, L. Barbeyto, and F. Real. "Identification of Hafnia alvei with the MicroScan WalkAway System." Journal of Clinical Microbiology 37, no. 12 (1999): 4186–88. http://dx.doi.org/10.1128/jcm.37.12.4186-4188.1999.

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Hafnia alvei is a gram-negative facultatively anaerobic bacillus that belongs to the family Enterobacteriaceae. This organism is a causative agent of intestinal disorders and is found in different environments. H. alvei has received increased clinical attention as a cause of different infections in humans. This study was performed to compare the MicroScan WalkAway automated identification system in conjunction with the new MicroScan Combo Negative type 1S panels with conventional biochemical methods for identification of 21 H. alvei strains. The MicroScan WalkAway system was found capable of correctly identifying 20 of the 21 strains tested.

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17

Lugowski, C., T. Niedziela, W. Jachymek, et al. "Structural and serological characterization of Hafnia alvei lipopolysaccharide core region." Acta Biochimica Polonica 42, no. 1 (1995): 51–54. http://dx.doi.org/10.18388/abp.1995_4667.

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The structures and serological activities of core oligosaccharide of Hafnia alvei strains have been investigated. Methylation analysis, NMR spectroscopy and various specific degradation procedures were the principal methods used. It is concluded that, core hexasaccharides are identical in the lipopolysaccharides tested and are built of two glucose, three heptose and one 2-keto-3-deoxyoctulosonic acid residues. The antiserum raised against the ATCC13337 oligosaccharide core-tetanus toxoid conjugate cross-reacted strongly with all lipopolysaccharides used as antigens in ELISA test, suggesting that this core region is the common structure in the Hafnia genus.

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18

Wang, Yanan, Xue Li, Gongliang Zhang, Jingran Bi, and Hongman Hou. "Transcriptome Reveals Regulation of Quorum Sensing of Hafnia alvei H4 on the Coculture System of Hafnia alvei H4 and Pseudomonas fluorescens ATCC13525." Foods 13, no. 2 (2024): 336. http://dx.doi.org/10.3390/foods13020336.

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In the food industry, foodborne spoilage bacteria often live in mixed species and attach to each other, leading to changes in spoilage characteristics. Quorum sensing (QS) has been reported to be a regulating mechanism for food spoiling by certain kinds of bacteria. Here, the contents of biofilm, extracellular polysaccharides, and biogenic amines in the coculture system of Hafnia alvei H4 and Pseudomonas fluorescens ATCC13525 were significantly reduced when the QS element of H. alvei H4 was deleted, confirming that QS of H. alvei H4 is involved in the dual-species interactions. Then, transcriptomics was used to explore the regulatory mechanism at the mRNA molecular level. The deletion of the QS element decreased the transcript levels of genes related to chemotaxis, flagellar assembly, and the two-component system pathway of H. alvei H4 in the coculture system. Furthermore, a total of 732 DEGs of P. fluorescens ATCC13525 were regulated in the dual species, which were primarily concerned with biofilm formation, ATP-binding cassette transporters, and amino acid metabolism. Taken together, the absence of the QS element of H. alvei H4 weakened the mutual cooperation of the two bacteria in the coculture system, making it a good target for managing infection with H. alvei and P. fluorescens.

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19

Girlich, Delphine, Thierry Naas, Samuel Bellais, Laurent Poirel, Amal Karim, and Patrice Nordmann. "Heterogeneity of AmpC Cephalosporinases ofHafnia alvei Clinical Isolates Expressing Inducible or Constitutive Ceftazidime Resistance Phenotypes." Antimicrobial Agents and Chemotherapy 44, no. 11 (2000): 3220–23. http://dx.doi.org/10.1128/aac.44.11.3220-3223.2000.

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ABSTRACT Ten unrelated Hafnia alvei clinical isolates were grouped according to either their low-level and inducible cephalosporinase production or their high-level and constitutive cephalosporinase production phenotype. Their AmpC sequences shared 85 to 100% amino acid identity. The immediate genetic environment ofampC genes was conserved in H. alvei isolates but was different from that found in other ampC-possessing enterobacterial species.

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Bailon Gaona, MC, M. Moreno Barrueco, F. Berdugo Hurtado, and A. Barrientos Delgado. "Dysbacteriosis by hafnia alvei: a purpose of a case." Revista Andaluza de Patología Digestiva 46, no. 3 (2023): 159–60. http://dx.doi.org/10.37352/2023463.6.

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Resumen Hafnia alvei, una enterobacteria que forma parte de la microbiota intestinal humana, raras veces crece en coprocultivo y ocasionalmente se considera patógena. Sin embargo, en pacientes con enfermedades crónicas o largos tratamientos con antibiótico, este microorganismo puede generar infecciones invasoras intestinales y extraintestinales a través de un mecanismo de virulencia desconocido. En estos casos, es necesario realizar tratamiento con antibioterapia, siendo de elección el empleo de quinolonas.

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El-Abasy, Moshira A., F. F. El-Khayat, A. A. El-Gohary, M. H. Awaad, and Amany, M. Taha. "SOME STUDIES ON HAFNIA ALVEI IN CHICKENS." Kafrelsheikh Veterinary Medical Journal 9, no. 2 (2011): 45–58. http://dx.doi.org/10.21608/kvmj.2011.113538.

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22

Westblom, T. U., and T. W. Milligan. "Acute Bacterial Gastroenteritis Caused by Hafnia alvei." Clinical Infectious Diseases 14, no. 6 (1992): 1271–72. http://dx.doi.org/10.1093/clinids/14.6.1271.

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23

EL-GOHARY, A., and AMAL YOUSEIF. "HAFNIA ALVEI INFECTION IN BROILER BREEDER CHICKENS." Veterinary Medical Journal (Giza) 50, no. 1 (2002): 171–90. http://dx.doi.org/10.21608/vmjg.2002.369223.

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Erre, Gian Luca, Enrico Colombo, Stefano Bibbò, and Maria Pina Dore. "Reactive arthritis secondary to Hafnia alvei enterocolitis." BMJ Case Reports 12, no. 5 (2019): e228513. http://dx.doi.org/10.1136/bcr-2018-228513.

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Diagnosis of postenteritic reactive arthritis (ReA) is a challenge and might have a broad range of differential diagnoses. A 50-year-old man was referred to our attention because of persistent inflammatory low back pain and asymmetric oligoarthritis. The clinical history was positive for diarrhoea in the previous 3 months. Inflammatory bowel disease, Whipple and celiac diseases were carefully excluded. In addition, serology, stool cultures, biopsies from the upper gastrointestinal tract yielded negative results for infections. A presumptive diagnosis of ReA was done and a non-steroidal anti-inflammatory drug trial prescribed. Persistence of symptoms prompted us for a second look of the colon. Biopsy collected from the terminal ileum were cultured and surprisingly colonies of Hafnia alvei, a rod-shaped Enterobacteriaceae, were detected. Treatment with ciprofloxacin leads to fast symptoms resolution. Although enterocolitis from H. alvei has been rarely reported, the culture of intestinal specimens might be recommended in the work-up of patients with suspected postenteritic ReA.

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Moriuchi, Hiroyuki, and Michele Trucksis. "Urinary Tract Infection Caused by Hafnia alvei." Infectious Diseases in Clinical Practice 5, no. 8 (1996): 517. http://dx.doi.org/10.1097/00019048-199611000-00016.

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26

Newmark, Jeffrey J., William N. Hobbs, and Bruce E. Wilson. "Reactive arthritis associated with Hafnia Alvei entiritis." Arthritis & Rheumatism 37, no. 6 (1994): 960. http://dx.doi.org/10.1002/art.1780370628.

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Lukasiewicz, Jolanta, Tomasz Niedziela, Wojciech Jachymek, Lennart Kenne, and Czeslaw Lugowski. "Two Kdo-Heptose Regions Identified in Hafnia alvei 32 Lipopolysaccharide: the Complete Core Structure and Serological Screening of Different Hafnia O Serotypes." Journal of Bacteriology 191, no. 2 (2008): 533–44. http://dx.doi.org/10.1128/jb.00891-08.

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ABSTRACT Hafnia alvei, a gram-negative bacterium, is an opportunistic pathogen associated with mixed hospital infections, bacteremia, septicemia, and respiratory diseases. Various 3-deoxy-d-manno-oct-2-ulosonic acid (Kdo)-containing fragments different from known structures of core oligosaccharides were previously found among fractions obtained by mild acid hydrolysis of some H. alvei lipopolysaccharides (LPSs). However, the positions of these segments in the LPS structure were not known. Analysis of de-N,O-acylated LPS by nuclear magnetic resonance spectroscopy and mass spectrometry allowed the determination of the location of a Kdo-containing trisaccharide in the structure of H. alvei PCM 32 LPS. It was established that the trisaccharide {l-α-d-Hepp-(1→4)-[α-d-Galp6OAc-(1→7)]-α-Kdop-(2→} is an integral part of the outer-core oligosaccharide of H. alvei 32 LPS. The very labile ketosidic linkage between →4,7)-α-Kdop and →2)-Glcp in the core oligosaccharide was identified. Screening for this Kdo-containing trisaccharide was performed on the group of 37 O serotypes of H. alvei LPSs using monospecific antibodies recognizing the structure. It was established that this trisaccharide is a characteristic component of the outer-core oligosaccharides of H. alvei 2, 32, 600, 1192, 1206, and 1211 LPSs. The weaker cross-reactions with LPSs of strains 974, 1188, 1198, 1204, and 1214 suggest the presence of similar structures in these LPSs, as well. Thus, we have identified new examples of endotoxins among those elucidated so far. This type of core oligosaccharide deviates from the classical scheme by the presence of the structural Kdo-containing motif in the outer-core region.

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Savini, Vincenzo, Erminia Di Bartolomeo, Chiara Catavitello, et al. "Graft versus host disease-related Hafnia alvei colonization and probable infection." Journal of Medical Microbiology 57, no. 9 (2008): 1167–69. http://dx.doi.org/10.1099/jmm.0.2008/001164-0.

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We describe the case of a graft versus host disease (GvHD) patient, in whom Hafnia alvei was cultured as a single organism, and at high bacterial counts from stool samples, from the onset of the disease until its resolution. This case is a further example of the contentious role of this species in causing human intestinal disease. Furthermore, it focuses on enteric damage by GvHD as a risk factor for acquiring H. alvei colonization, and probably infection.

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Meah, Ibnat, and David R. Singleton. "A novel Hafnia-specific bacteriophage is capable of transduction of ampicillin resistance between bacterial species." Journal of the Pennsylvania Academy of Science 95, no. 2 (2021): 88–103. http://dx.doi.org/10.5325/jpennacadscie.95.2.0088.

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Abstract A bacteriophage isolated for its ability to infect the Gram-negative and ampicillin-sensitive bacterium Hafnia alvei was also able to infect a strain of ampicillin-resistant Klebsiella pneumoniae. An apparatus was designed to allow cultures of the two bacterial species to be separated by a 0.2-μm filter. A phage-infected culture of K. pneumoniae was grown next to an uninfected H. alvei culture. Following incubation, samples from the H. alvei culture were plated on nonselective media (nutrient agar), and plated colonies were then replica plated to ampicillin-containing media. Several ampicillin-resistant colonies of H. alvei were identified. Of the potential methods for horizontal gene transfer (transformation, transduction, and conjugation), only viral transduction of antibiotic resistance is supported.

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Zhang, Xuan, Hai Chi, Di Peng, et al. "Integrated Metagenomic and LC–MS/MS Analysis Reveals the Biogenic Amine-Producing Strains of Two Typical Chinese Traditional Fish Products: Fermented Mandarin Fish (Siniperca chuatsi) and Semi-Dried Yellow Croaker (Larimichthys crocea)." Foods 14, no. 6 (2025): 1016. https://doi.org/10.3390/foods14061016.

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Two typical fish products—fermented mandarin fish and semi-dried yellow croaker—are associated with biogenic amines (BAs), which are harmful to human health. The objective of this study was to investigate the bacterial ecology of the two fish products and to determine their capacity for producing BAs. Putrescine and cadaverine were major BAs detected in the fish products. Concentrations of BAs were significantly corrected with microbial count (p < 0.05). BA-producing isolates (33) in the two fish products were all multiple BA producers. Several of them, including Lactobacillus sakei, Bacillus cereus and Hafnia alvei isolated from fermented mandarin fish, as well as Shewanella baltica, Aeromonas veronii, and Photobacterium phosphoreum isolated from semi-dried yellow croaker, showed remarkable BA-producing capacity. Hafnia alvei produced the greatest abundance of putrescine, cadaverine, tyramine and 2-phenylethylamine. Lactobacillus sakei mainly produced tryptamine and putrescine. Photobacterium phosphoreum showed the strongest histamine-producing capacity.

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Severiche-Bueno, Diego Fernando, María Teresa Vargas-Cuervo, Luis Medina-Lee, et al. "Hafnia alvei pneumonia: from bees to human beings." GERMS 11, no. 2 (2021): 306–9. http://dx.doi.org/10.18683/germs.2021.1265.

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Casanova-Román, Manuel, Antonio Sanchez-Porto, and Manuel Casanova-Bellido. "Late-onset neonatal sepsis due to hafnia alvei." Scandinavian Journal of Infectious Diseases 36, no. 1 (2004): 70–71. http://dx.doi.org/10.1080/00365540310017375.

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Ruiz-Moreno, J. Ma, J. L. Alió, and F. De La Hoz. "Delayed-Onset Postoperative Endophthalmitis Caused by Hafnia Alvei." European Journal of Ophthalmology 11, no. 2 (2001): 189–92. http://dx.doi.org/10.1177/112067210101100217.

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34

Reina, J., J. Hervas, and N. Borrell. "Acute Gastroenteritis Caused by Hafnia alvei in Children." Clinical Infectious Diseases 16, no. 3 (1993): 443. http://dx.doi.org/10.1093/clind/16.3.443.

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35

Real, F., A. Fernandez, F. Acosta, et al. "Septicemia Associated with Hafnia alvei in Laying Hens." Avian Diseases 41, no. 3 (1997): 741. http://dx.doi.org/10.2307/1592170.

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36

Litrenta, Jody, and Matthew Oetgen. "Hafnia alvei : A new pathogen in open fractures." Trauma Case Reports 8 (April 2017): 41–45. http://dx.doi.org/10.1016/j.tcr.2017.01.019.

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37

UGOWSKI, C. "Lipopolysaccharide core region of Hafnia alvei: Serological characterization." FEMS Immunology and Medical Microbiology 10, no. 2 (1995): 119–24. http://dx.doi.org/10.1016/0928-8244(94)00065-2.

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38

Ramos-Vivas, José, Olga Tapia, María Elexpuru-Zabaleta, et al. "The Molecular Weaponry Produced by the Bacterium Hafnia alvei in Foods." Molecules 27, no. 17 (2022): 5585. http://dx.doi.org/10.3390/molecules27175585.

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Hafnia alvei is receiving increasing attention from both a medical and veterinary point of view, but the diversity of molecules it produces has made the interest in this bacterium extend to the field of probiotics, the microbiota, and above all, to its presence and action on consumer foods. The production of Acyl Homoserine Lactones (AHLs), a type of quorum-sensing (QS) signaling molecule, is the most often-studied chemical signaling molecule in Gram-negative bacteria. H. alvei can use this communication mechanism to promote the expression of certain enzymatic activities in fermented foods, where this bacterium is frequently present. H. alvei also produces a series of molecules involved in the modification of the organoleptic properties of different products, especially cheeses, where it shares space with other microorganisms. Although some strains of this species are implicated in infections in humans, many produce antibacterial compounds, such as bacteriocins, that inhibit the growth of true pathogens, so the characterization of these molecules could be very interesting from the point of view of clinical medicine and the food industry. Lastly, in some cases, H. alvei is responsible for the production of biogenic amines or other compounds of special interest in food health. In this article, we will review the most interesting molecules that produce the H. alvei strains and will discuss some of their properties, both from the point of view of their biological activity on other microorganisms and the properties of different food matrices in which this bacterium usually thrives.

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39

Beier, Horst, Lothar F. Fecker, and Jochen Berlin. "Lysine Decarboxylase from Hafnia alvei: Purification, Molecular Data and Preparation of Polyclonal Antibodies." Zeitschrift für Naturforschung C 42, no. 11-12 (1987): 1307–12. http://dx.doi.org/10.1515/znc-1987-11-1226.

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The purification and molecular properties of lysine decarboxylase from Hafnia alvei and the preparation of polyvalent antibodies specific for this enzyme are described. The enzyme was purified within two HPLC steps on a TSK G 4000 SW and a MonoQ column to homogeneity. The subunit of the enzyme has a molecular weight of approximately 80.000 d. Under “native” conditions it seems to form aggregates up to ten subunits. Lysine decarboxylase from H. alvei contains one mol pyridoxal phosphate per mol subunit. Antibodies against the lysine decarboxylase were purified by affinity chromatography.

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Silva, Reginara Teixeira da, Joyce Bitencourt Athayde Lopes, Kleydejany Lima Lemos de Oliveira, José Carlos Ribeiro Júnior, and Vanerli Beloti. "Perfil de sensibilidade a antimicrobianos de bactérias patogênicas humanas isoladas de leite cru." Revista do Instituto de Laticínios Cândido Tostes 74, no. 3 (2019): 185–94. http://dx.doi.org/10.14295/2238-6416.v74i3.743.

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A presença de bactérias no leite cru, associada ao possível uso indiscriminado de antimicrobianos no gado leiteiro, pode influenciar no surgimento de bactérias resistentes. O perfil de sensibilidade a antimicrobianos de seis cepas bacterianas patogênicas, isoladas de leite cru do estado do Maranhão (Acinetobacter septicus, Hafnia alvei, Lactococcus garvieae, Staphylococcus epidermidis, Pantoea spp. e Klebsiella spp.), foi avaliado neste estudo por meio do teste de antibiograma por difusão de disco em Ágar Müeller Hinton, e para a interpretação das categorias de sensibilidade foram utilizados os critérios previamente estabelecidos. O antimicrobiano com menor eficiência foi a lincomicina, onde 83,33 % das cepas apresentaram resistência. A gentamicina e a tetraciclina apresentaram 100% de eficiência frente os isolados deste estudo. Klebsiella spp. destacara-se por apresentar resistência >50% aos antimicrobianos testados. No entanto, observou-se multirresistência em 50% dos isolados: Hafnia alvei, Pantoea spp. e Klebsiella spp. Os resultados verificados demonstram a multirresistência de patógenos de grande importância para a saúde pública, e o leite cru como uma possível fonte de veiculação desses micro-organismos multirresistentes.

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41

Yap, D. Y. H., S. K. P. Lau, S. Lamb, et al. "An Unusual Organism for PD-Related Peritonitis: Hafnia Alvei." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 30, no. 2 (2010): 254–55. http://dx.doi.org/10.3747/pdi.2009.00090.

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Teshima, Chisato, Shigeharu Kudo, Yoshimi Ohtani, and Asako Saito. "Kidney Pathology from the Bacterium Hafnia alvei: Experimental Evidence." Transactions of the American Fisheries Society 121, no. 5 (1992): 599–607. http://dx.doi.org/10.1577/1548-8659(1992)121<0599:kpftbh>2.3.co;2.

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Murphy, Adrian D., Frank J. Conroy, and Padraic J. Regan. "Re: Hafnia alvei infection of the deep palmar space." Journal of Hand Surgery (European Volume) 33, no. 4 (2008): 534. http://dx.doi.org/10.1177/1753193408089524.

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CARAVALHO, JOSEPH, VICTOR M. McMILLAN, ROBERT B. ELLIS, and ARTURO BETANCOURT. "Endogenous Endophthalmitis Due to Salmonella arizonae and Hafnia alvei." Southern Medical Journal 83, no. 3 (1990): 325–27. http://dx.doi.org/10.1097/00007611-199003000-00017.

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Savini, Vincenzo, Alessandro Santarelli, Ennio Polilli, et al. "Hafnia alvei from the farm to the delivery room." Veterinary Microbiology 163, no. 1-2 (2013): 202–3. http://dx.doi.org/10.1016/j.vetmic.2012.12.002.

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Michalski, Mateusz, Anna St. Swierzko, Jolanta Lukasiewicz, et al. "Ficolin-3 activity towards the opportunistic pathogen, Hafnia alvei." Immunobiology 220, no. 1 (2015): 117–23. http://dx.doi.org/10.1016/j.imbio.2014.08.012.

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Ratnam, S. "Etiologic role of Hafnia alvei in human diarrheal illness." Infection and Immunity 59, no. 12 (1991): 4744–45. http://dx.doi.org/10.1128/iai.59.12.4744-4745.1991.

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Albert, M. J., K. Alam, M. Islam, et al. "Hafnia alvei, a probable cause of diarrhea in humans." Infection and Immunity 59, no. 4 (1991): 1507–13. http://dx.doi.org/10.1128/iai.59.4.1507-1513.1991.

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49

Okada, Shoko, and David M. Gordon. "Genetic and Ecological Structure of Hafnia alvei in Australia." Systematic and Applied Microbiology 26, no. 4 (2003): 585–94. http://dx.doi.org/10.1078/072320203770865891.

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Yoon, M. Y., K. A. Thayercook, A. J. Berdis, W. E. Karsten, K. D. Schnackerz, and P. F. Cook. "Acid-Base Chemical Mechanism of Aspartase from Hafnia alvei." Archives of Biochemistry and Biophysics 320, no. 1 (1995): 115–22. http://dx.doi.org/10.1006/abbi.1995.1348.

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Journal articles on the topic 'Hafnia alvei'

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