Relevant bibliographies by topics / Hamsoch

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  2. Dissertations / Theses
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Academic literature on the topic 'Hamsoch'

Author: Grafiati
Published: 26 July 2024

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Journal articles on the topic "Hamsoch"

1

You, Qi, Ziming Liu, Jun Zhang, Mengjie Shen, Yuwan Li, Ying Jin, and Yi Liu. "Human Amniotic Mesenchymal Stem Cell Sheets Encapsulating Cartilage Particles Facilitate Repair of Rabbit Osteochondral Defects." American Journal of Sports Medicine 48, no. 3 (January 15, 2020): 599–611. http://dx.doi.org/10.1177/0363546519897912.

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Background: Human amniotic mesenchymal stem cells (hAMSCs) are being widely applied in various fields. Therefore, hAMSCs represent a promising candidate to facilitate cartilage regeneration. Nonetheless, no studies have investigated the application of hAMSC sheets to repair cartilage defects in vivo. Purpose: To evaluate hAMSC sheets encapsulating cartilage particles to promote repair of rabbit osteochondral defects. Study Design: Controlled laboratory study. Methods: hAMSC sheets were constructed with passage 3 hAMSCs. The phenotypic and structural characteristics of hAMSC sheets were evaluated by flow cytometry and scanning electron microscopy, respectively. The potential for chondrogenic differentiation of hAMSC sheets was assessed by cartilage-specific marker staining, immunohistochemistry, and mRNA and protein expression (SOX9, COLII, and ACAN). Osteochondral defects (diameter, 3.5 mm; depth, 3 mm) were created in the left patellar grooves of 20 New Zealand White rabbits (female or male). The defects were treated with hAMSC sheet/cartilage particles (n = 5), cartilage particles (n = 5), hAMSC sheets (n = 5), or fibrin glue (n = 5). Macroscopic and histological evaluations of the regenerated tissue were conducted after 3 months. The survival time and differentiation of transplanted hAMSCs in the defect area were evaluated by immunofluorescence. Results: hAMSC sheets had a multilayered structure, with cells stacked layer by layer. Importantly, hAMSC sheets highly expressed phenotypic markers of mesenchymal stem cells. Cartilage-specific marker staining and immunohistochemistry were positive, and mRNA and protein expression was higher in the chondrogenically induced hAMSC sheet group than in the hAMSC sheet group ( P < .05). hAMSC sheet/cartilage particles formed a large amount of hyaline-like cartilage in the defect area. In addition, macroscopic and histological scores were significantly higher than those in the other groups. Integration with surrounding normal cartilage and subchondral bone regeneration in the hAMSC sheet/cartilage particles group were better when compared with the other groups. A large number of human nuclear-specific antigen-positive cells were observed in the defect area of hAMSC sheet/cartilage particles and hAMSC sheet groups. Moreover, some positive cells expressed SOX9. Conclusion: hAMSC sheets encapsulating cartilage particles facilitate osteochondral defect repair. Clinical Relevance: Delivery of cells in the form of a cell sheet in conjunction with cartilage particles provides a novel approach for cell-based cartilage regeneration.
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Hur, Junseok W., Min-Sik Kim, Se-Yeon Oh, Ho-Young Kang, Jingi Bae, Hokeun Kim, Hangyeore Lee, Sang-Won Lee, and Dong-Hyuk Park. "Label-Free Quantitative Proteome Profiling of Cerebrospinal Fluid from a Rat Stroke Model with Stem Cell Therapy." Cell Transplantation 30 (January 1, 2021): 096368972110234. http://dx.doi.org/10.1177/09636897211023474.

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Human adipose-derived mesenchymal stem cells (hAMSCs) are capable of immunomodulation and regeneration after neural injury. For these reasons, hAMSCs have been investigated as a promising stem cell candidate for stroke treatment. However, noninvasive experiments studying the effects of grafted stem cells in the host brain have not yet been reported. Cerebrospinal fluid (CSF), which can be collected without sacrificing the subject, is involved in physiological control of the brain and reflects the pathophysiology of various neurological disorders of the central nervous system (CNS). Following stem cell transplantation in a stroke model, quantitative analysis of CSF proteome changes can potentially reveal the therapeutic effect of stem cells on the host CNS. We examined hAMSC-secreted proteins obtained from serum-free culture medium by liquid chromatography-tandem mass spectrometry (LC-MS/MS), which identified several extracellular matrix proteins, supporting the well-known active paracrine function of hAMSCs. Subsequently, we performed label-free quantitative proteomic analysis on CSF samples from rat stroke models intravenously injected with hAMSC (experimental) or phosphate buffered saline (control). In total, 524 proteins were identified; among them, 125 and 91 proteins were increased and decreased with hAMSC treatment, respectively. Furthermore, gene set enrichment analysis revealed three proteins, 14-3-3 theta, MAG, and neurocan, that showed significant increases in the hAMSC-treated model; these proteins are core members of neurotrophin signaling, nerve growth factor (NGF) signaling, and glycosaminoglycan metabolism, respectively. Subsequent histological and neurologic function experiments validated proliferative neurogenesis in the hAMSC-treated stroke model. We conclude that (i) intravenous injection of hAMSCs can induce neurologic recovery in a rat stroke model and (ii) CSF may reflect the therapeutic effect of hAMSCs. Additionally, proteins as 14-3-3 theta, MAG, and neurocan could be considered as potential CSF biomarkers of neuroregeneration. These CSF proteome profiling results would be utilized as valuable resource in further stroke studies.
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Yin, Lu, Zhi-xuan Zhou, Ming Shen, Ning Chen, Fei Jiang, and Shou-Lin Wang. "The Human Amniotic Mesenchymal Stem Cells (hAMSCs) Improve the Implant Osseointegration and Bone Regeneration in Maxillary Sinus Floor Elevation in Rabbits." Stem Cells International 2019 (December 11, 2019): 1–10. http://dx.doi.org/10.1155/2019/9845497.

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Insufficient bone height in the posterior maxilla is a challenging problem in dental implantation. Bio-Oss, though routinely used in maxillary sinus floor elevation (MSFE), is not osteoinductive. Human amniotic mesenchymal cells (hAMSCs) isolated from placental tissues have potential for multidifferentiation and immunomodulatory properties and can be easily obtained without the need for invasive procedures and without ethical concerns. This is the first study to use hAMSCs to improve implant osseointegration and bone regeneration after MSFE. Human AMSCs were loaded into a fibrin gel and injected into rabbit MSFE models. The rabbits were assigned to four groups (n=3 per group), i.e., the control group, the hAMSC group, the Bio-Oss group, and the hAMSC/Bio-Oss group. The animals were sacrificed at postsurgery for four and twelve weeks and evaluated by histology and immunohistochemistry. Bone volume, bone volume/tissue volume, bone-to-implant contact ratio, and vessel-like structures in the hAMSC/Bio-Oss group were significantly better than those in other groups in the peri-implant and augmented areas. Immunofluorescence staining showed that alkaline phosphatase (ALP) activities of two hAMSC groups were higher than those of the other two groups. Sequential fluorescent labeling was performed in all of the 12-week groups. Observations showed that hAMSCs accelerated mineralized deposition rates on implant surfaces and in bone-augmented areas. These data demonstrated that hAMSCs could enhance implant osseointegration and bone regeneration after MSFE and might be used to optimize dental implantation in the future.
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Miceli, Vitale, Alessandro Bertani, Cinzia Maria Chinnici, Matteo Bulati, Mariangela Pampalone, Giandomenico Amico, Claudia Carcione, Eva Schmelzer, Jörg C. Gerlach, and Pier Giulio Conaldi. "Conditioned Medium from Human Amnion-Derived Mesenchymal Stromal/Stem Cells Attenuating the Effects of Cold Ischemia-Reperfusion Injury in an In Vitro Model Using Human Alveolar Epithelial Cells." International Journal of Molecular Sciences 22, no. 2 (January 6, 2021): 510. http://dx.doi.org/10.3390/ijms22020510.

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The clinical results of lung transplantation (LTx) are still less favorable than other solid organ transplants in both the early and long term. The fragility of the lungs limits the procurement rate and can favor the occurrence of ischemia-reperfusion injury (IRI). Ex vivo lung perfusion (EVLP) with Steen SolutionTM (SS) aims to address problems, and the implementation of EVLP to alleviate the activation of IRI-mediated processes has been achieved using mesenchymal stromal/stem cell (MSC)-based treatments. In this study, we investigated the paracrine effects of human amnion-derived MSCs (hAMSCs) in an in vitro model of lung IRI that includes cold ischemia and normothermic EVLP. We found that SS enriched by a hAMSC-conditioned medium (hAMSC-CM) preserved the viability and delayed the apoptosis of alveolar epithelial cells (A549) through the downregulation of inflammatory factors and the upregulation of antiapoptotic factors. These effects were more evident using the CM of 3D hAMSC cultures, which contained an increased amount of immunosuppressive and growth factors compared to both 2D cultures and encapsulated-hAMSCs. To conclude, we demonstrated an in vitro model of lung IRI and provided evidence that a hAMSC-CM attenuated IRI effects by improving the efficacy of EVLP, leading to strategies for a potential implementation of this technique.
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Jiang, Fei, Jie Ma, Yi Liang, Yuming Niu, Ning Chen, and Ming Shen. "Amniotic Mesenchymal Stem Cells Can Enhance Angiogenic Capacity via MMPsIn VitroandIn Vivo." BioMed Research International 2015 (2015): 1–15. http://dx.doi.org/10.1155/2015/324014.

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The aim of this study was to evaluate the angiogenic capacity and proteolytic mechanism of coculture using human amniotic mesenchymal stem cells (hAMSCs) with human umbilical vein endothelial cells (HUVECs)in vivoandin vitroby comparing to those of coculture using bone marrow mesenchymal stem cells with HUVEC. For thein vivoexperiment, cells (HUVEC-monoculture, HUVEC-hAMSC coculture, and HUVEC-BMMSC coculture) were seeded in fibrin gels and injected subcutaneously in nude mice. The samples were collected on days 7 and 14 and histologically analyzed by H&E and CD31 staining. CD31-positive staining percentage and vessel-like structure (VLS) density were evaluated as quantitative parameters for angiogenesis. The increases of CD31-positive staining area and VLS density in both HUVEC-hAMSC group and HUVEC-BMMSC group were found between two time points, while obvious decline of those was observed in HUVEC-only group. For thein vitroexperiment, we utilized the same 3D culture model to investigate the proteolytic mechanism related to capillary formation. Intensive vascular networks formed by HUVECs were associated with hAMSCs or BMMSCs and related to MMP2 and MMP9. In conclusion, hAMSCs shared similar capacity and proteolytic mechanism with BMMSCs on neovascularization.
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Sandonà, Martina, Federica Esposito, Anna Cargnoni, Antonietta Silini, Pietro Romele, Ornella Parolini, and Valentina Saccone. "Amniotic Membrane-Derived Stromal Cells Release Extracellular Vesicles That Favor Regeneration of Dystrophic Skeletal Muscles." International Journal of Molecular Sciences 24, no. 15 (August 5, 2023): 12457. http://dx.doi.org/10.3390/ijms241512457.

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Duchenne muscular dystrophy (DMD) is a muscle disease caused by mutations in the dystrophin gene characterized by myofiber fragility and progressive muscle degeneration. The genetic defect results in a reduced number of self-renewing muscle stem cells (MuSCs) and an impairment of their activation and differentiation, which lead to the exhaustion of skeletal muscle regeneration potential and muscle replacement by fibrotic and fatty tissue. In this study, we focused on an unexplored strategy to improve MuSC function and to preserve their niche based on the regenerative properties of mesenchymal stromal cells from the amniotic membrane (hAMSCs), that are multipotent cells recognized to have a role in tissue repair in different disease models. We demonstrate that the hAMSC secretome (CM hAMSC) and extracellular vesicles (EVs) isolated thereof directly stimulate the in vitro proliferation and differentiation of human myoblasts and mouse MuSC from dystrophic muscles. Furthermore, we demonstrate that hAMSC secreted factors modulate the muscle stem cell niche in dystrophic–mdx-mice. Interestingly, local injection of EV hAMSC in mdx muscles correlated with an increase in the number of activated Pax7+/Ki67+ MuSCs and in new fiber formation. EV hAMSCs also significantly reduced muscle collagen deposition, thus counteracting fibrosis and MuSCs exhaustion, two hallmarks of DMD. Herein for the first time we demonstrate that CM hAMSC and EVs derived thereof promote muscle regeneration by supporting proliferation and differentiation of resident muscle stem cells. These results pave the way for the development of a novel treatment to counteract DMD progression by reducing fibrosis and enhancing myogenesis in dystrophic muscles.
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Sun, Shoujia, Quan Zhang, Man Li, Pan Gao, Kuan Huang, Rajluxmee Beejadhursing, Wei Jiang, Ting Lei, Mingxin Zhu, and Kai Shu. "GDNF Promotes Survival and Therapeutic Efficacy of Human Adipose-Derived Mesenchymal Stem Cells in a Mouse Model of Parkinson’s Disease." Cell Transplantation 29 (January 1, 2020): 096368972090851. http://dx.doi.org/10.1177/0963689720908512.

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Mesenchymal stem cell (MSC)-based regenerative therapy is regarded as a promising strategy for the treatment of Parkinson’s disease (PD). However, MSC components may exhibit poor intracranial survivability, particularly in the later stages following cell transplantation, limiting their potential curative effect and also clinical applications. Glial cell line-derived neurotrophic factor (GDNF), which encompasses a variety of transforming growth factor beta super family members, has been reported to enhance motor function and exert neuroprotective effects. However, no previous studies have investigated the effects of GDNF on human primary adipose-derived MSCs (hAMSCs), despite its potential for enhancing stem cell survival and promoting therapeutic efficacy in the treatment of PD. In the present study, we proposed a novel approach for enhancing the proliferative capacity and improving the efficacy of hAMSC treatment. Pre-exposure of engineered hAMSCs to GDNF enhanced the proliferation and differentiation of these stem cells in vitro. In addition, in 6-hydroxydopamine-lesioned mice, a common PD model, intracranial injection of hAMSCs-GDNF was associated with greater performance on behavioral tests, larger graft volumes 5 weeks after transplantation, and higher levels of Nestin, glial fibrillary acidic protein, and Tuj-1 differentiation than those treated with hAMSCs-Vector. Following transplantation of hAMSCs-GDNF into the striatum of lesioned models, we observed significant increases in tyrosine hydroxylase- and NeuN-positive staining. These findings highlight the therapeutic potential of hAMSCs-GDNF for patients with PD, as well as an efficient method for promoting therapeutic efficacy of these delivery vehicles.
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8

Fu, Qingjie, Shunsuke Ohnishi, and Naoya Sakamoto. "Conditioned Medium from Human Amnion-Derived Mesenchymal Stem Cells Regulates Activation of Primary Hepatic Stellate Cells." Stem Cells International 2018 (October 8, 2018): 1–11. http://dx.doi.org/10.1155/2018/4898152.

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Mesenchymal stem cells (MSCs), or multipotent mesenchymal stromal cells, are present in almost all organs and tissues, including the amnion. Human amnion-derived mesenchymal stem cell (hAMSC) transplantation has been reported to ameliorate liver fibrosis in animal models. However, the mechanism for the prevention of liver fibrosis is poorly understood. In this study, we investigated the effects, and underlying mechanisms, of a conditioned medium obtained from hAMSC cultures (hAMSC-CM) on a primary culture of rat hepatic stellate cells (HSCs). We observed that in routine culture, hAMSC-CM in HSCs significantly inhibited the expression of alpha-smooth muscle actin (α-SMA), an activation marker of HSCs, and the production of collagen type 1 (COL1), a dominant component of the extracellular matrix (ECM) in the culture medium. In addition, hAMSC-CM upregulated the expression of ECM degradation-related genes, such as metalloproteinase- (Mmp-) 2, Mmp-9, Mmp-13, and tissue inhibitor of metalloproteinase- (Timp-) 1; however, it did not affect the expression of collagen type 1α1 (Col1a1). These regulatory effects on HSCs were concentration-dependent. A cell proliferation assay indicated that hAMSC-CM significantly suppressed HSC proliferation and downregulated the expression of cyclin B (Ccnb), a proliferation-related gene. Transforming growth factor-beta (TGF-β) treatment further activated HSCs and hAMSC-CM significantly inhibited the upregulation of α-Sma and Col1a1 induced by TGF-β. These findings demonstrated that hAMSC-CM can modulate HSC function via secretory factors and provide a plausible explanation for the protective role of hAMSCs in liver fibrosis.
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Fioretti, Daniela, Mario Ledda, Sandra Iurescia, Raffaella Carletti, Cira Di Gioia, Maria Grazia Lolli, Rodolfo Marchese, Antonella Lisi, and Monica Rinaldi. "Severely Damaged Freeze-Injured Skeletal Muscle Reveals Functional Impairment, Inadequate Repair, and Opportunity for Human Stem Cell Application." Biomedicines 12, no. 1 (December 21, 2023): 30. http://dx.doi.org/10.3390/biomedicines12010030.

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Background: The regeneration of severe traumatic muscle injuries is an unsolved medical need that is relevant for civilian and military medicine. In this work, we produced a critically sized nonhealing muscle defect in a mouse model to investigate muscle degeneration/healing phases. Materials and methods: We caused a freeze injury (FI) in the biceps femoris of C57BL/6N mice. From day 1 to day 25 post-injury, we conducted histological/morphometric examinations, an analysis of the expression of genes involved in inflammation/regeneration, and an in vivo functional evaluation. Results: We found that FI activates cytosolic DNA sensing and inflammatory responses. Persistent macrophage infiltration, the prolonged expression of eMHC, the presence of centrally nucleated myofibers, and the presence of PAX7+ satellite cells at late time points and with chronic physical impairment indicated inadequate repair. By looking at stem-cell-based therapeutic protocols of muscle repair, we investigated the crosstalk between M1-biased macrophages and human amniotic mesenchymal stem cells (hAMSCs) in vitro. We demonstrated their reciprocal paracrine effects where hAMSCs induced a shift of M1 macrophages into an anti-inflammatory phenotype, and M1 macrophages promoted an increase in the expression of hAMSC immunomodulatory factors. Conclusions: Our findings support the rationale for the future use of our injury model to exploit the full potential of in vivo hAMSC transplantation following severe traumatic injuries.
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Kamadjaja, David. "The Osteogenic Capacity of HumanAmniotic Membrane Mesenchymal Stem Cell (hAMSC) and Potential for Application in Maxillofacial Bone Reconstruction in Vitro Study." Journal of Stem Cell Research and Tissue Engineering 4, no. 1 (August 26, 2020): 17. http://dx.doi.org/10.20473/jscrte.v4i1.21590.

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Amniotic membrane of human placenta is a source of abundant mesenchymal stem cell (hAMSC) which makes it a potential source of allogeneic multipotent cell for bone healing. However, much has to be explored about its isolation procedure and the osteogenic differentiation potential. The aims of this study are to establish the procurement procedure of human amniotic membrane, the isolation and culture of hAMSC, the MSC phenotypic characterization, and the in vitro osteogenic differentiation of hAMSC. Results of the study are as follows. The quality of human amniotic membrane would be best if procured from Caesarean operation under highly aseptic condition to avoid fungal and bacterial contamination on the culture. Isolation procedure using modified Soncini protocol yielded large amount of MSC with high proliferative capacity in culture medium. Characterization of hAMSC showed that the majority of the target cells exhibited specific MSC markers (CD10S and CD90) with a small number of these cells expressing CD45the marker of hematopoeitic cells. The in vitro osteogenic differentiation of hAMSC followed by Alizarin Red staining showed that osteoblastic differentiation was detected in a significantly high number of cells. This study concludes that hAMSCs isolated from human amniotic membrane have the capacity for in vitro osteogenesis which makes them be one of the potential allogeneic stem cells for application in maxillofacial bone reconstruction.
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Dissertations / Theses on the topic "Hamsoch"

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Hamscha, Susanne [Verfasser]. "The fiction of America : performing the cultural imaginary in American literature and culture / Susanne Hamscha." Berlin : Freie Universität Berlin, 2012. http://d-nb.info/1029851212/34.

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Nahas, Elizabeth Leila. "Physical processes controlling circulation and frontal zones in Shark Bay, Western Australia." University of Western Australia. Centre for Water Research, 2005. http://theses.library.uwa.edu.au/adt-WU2005.0011.

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Shark Bay is a large inverse estuary, located in Western Australia. It has a number of unique habitats that support important species. The dynamics of circulation in Shark Bay have an influence on the species that inhabit the region, on small, local scales as well as on large Bay-wide scales. Numerical modeling and field data were used to examine small-scale dynamics in relation to an important recreational fish, pink snapper (Pagrus auratus). Icthyoplankton surveys collected and recorded egg density in regions where snapper are found. A barotropic three-dimensional hydrodynamic model was coupled with a two-dimensional Lagrangian particle-tracking program to simulate the passive transport of eggs through regions where spawning is known to occur. Circulation modeling results indicated residual flows on small scales that served to retain the eggs in the region where they were originally spawned. Results corroborate genetic work on adult snapper, which found no evidence intermixing of populations in Shark Bay. The numerical model was then further refined to run in a baroclinic mode. Simulations of salinity and temperature gradients were used to recreate frontal systems in Shark Bay. Frontal regions divide the Bay into a northern and a southern section as well as separate it from the ocean. Application of an analytical method for calculating front locations was consistent with the observed results and indicated that the primary forces determining frontal locations in the Bay are tides and gravitational circulation. Winds are a secondary influence, and solar heating is minimal in influence
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Books on the topic "Hamsoch"

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Khānī, Aḥmad. Hamsah. al-Riyāḍ: A.ʻA.al-R. al-Khānī, 1999.

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ʻĀṣī, Ibrāhīm. Hamsah fī udhun Ḥawwāʼ. 4th ed. [Cairo]: Dār al-Salām, 1986.

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Sibai, Yousuf al. Hamsah ghabirah, aqwamin al-zaman. Cairo: Maktaba-e-Misr, 1994.

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Mutawallī, Aḥmad ʻAbd Allāh. al- Hamsah al-ṣākhibah: Majmūʻah qiṣaṣīyah. [Cairo]: al-Majlis al-Aʻlá lil-Thaqāfah, 1991.

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Hamsah fī ādhān al-rijāl: Riwāyah. al-Dār al-Bayḍāʼ: Afrīqiyā al-Sharq, 2012.

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Intihāk al-muwaẓẓaf al-ʻāmm: Ḥurmat al-maskan wa-ʻuqūbtuh : dirāsah taʼṣīlīyah muqāranah. al-Rīyāḍ: Turkī ibn ʻĪd al-Sharāfī, 2014.

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Avisar, Tzvi T. Sharon, ḥamesh shanim Ḳadimah =: Sharon, five years forward = Sharun, baʻad hamsah sanawat. Yerushalayim: Tsevi Tsiḳi Aviśar, 2011.

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Yu, Yusupov É, Sulaĭmonova Fozila, Akademiya nauk Uzbekskoĭ SSR, and Kh. S. Suleĭmonov Institute of manuscripts., eds. Nizomiĭ "Khamsa" siga ishlangan rasmlar =: Miniatyurȳ k "Khamse" Nizami = Miniatures illuminations of Nisami's "Hamshah". Toshkent: Ŭzbekiston SSR "Fan" nashriëti, 1985.

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I︠U︡, I︠U︡supov Ė., Sulaĭmonova Fozila, Ŭzbekiston SSR fanlar akademii︠a︡si, and Ḣ.S. Sulaĭmonov nomidagi qŭlëzmalar instituti., eds. Nizomiĭ "Khamsa" siga ishlangan rasmlar =: Miniati︠u︡ri k "Khamse" Nizami = Miniatures illuminations of Nizami's "Hamsah". Toshkent: Fan, 1985.

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Khani, Ahmad. Hamsah. A. al-Khani, 1999.

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Book chapters on the topic "Hamsoch"

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Nurulla-Khojaeva, Nargis. "‘Imitated’ or genuine? The value of resilience in Sufi-hamsoya." In Resilient Communities of Central Eurasia, 75–90. London: Routledge, 2023. http://dx.doi.org/10.4324/9781003299998-5.

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Guang, L., G. Plosila, J. Isoaho, and H. Tenhunen. "HAMSoC." In Embedded Multi-Core Systems, 135–64. CRC Press, 2011. http://dx.doi.org/10.1201/b11421-7.

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"HAMSoC: A Monitoring-Centric Design Approach for Adaptive Parallel Computing." In Autonomic Networking-on-Chip, 161–90. CRC Press, 2018. http://dx.doi.org/10.1201/9781315217826-13.

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"Working, Managing, and Leading in Turbulent Times: Ned Hamson, Association for Quality and Participation." In AFTER ATLANTIS: Working, Managing, and Leading in Turbulent Times, 18–41. Routledge, 2012. http://dx.doi.org/10.4324/9780080498904-7.

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"Requisite for Future Success: Discontinuous Improvement. Ned Hamson, AQP and Robert Holder, Greymatter Production." In AFTER ATLANTIS: Working, Managing, and Leading in Turbulent Times, 178–86. Routledge, 2012. http://dx.doi.org/10.4324/9780080498904-15.

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"Fuller, L, ‘Consideration and form’ (1941) 41 Columbia LR 799. Fuller, L and Perdue, W, ‘The reliance interest in contract damages’ (1936–37) 46 Yale LJ 52. Furmston, M, ‘Return to Dunlop v Selfridge?’ (1960) 23 MLR 373. Gardner, S, ‘Trashing with Trollope: a deconstruction of postal rules in contract’ (1992) 2 OJLS 170. Goff, R, and Jones, G, The Law of Restitution, 4th edn, 1993, London: Sweet & Maxwell. Gower, L, ‘Exemption clauses – contractual and tortious liability’ (1954) 17 MLR 155. Hamson, C, ‘The reform of consideration’ (1938) 54 LQR 233. Harris, D, Ogus, A and Phillips, J, ‘Contact remedies and the consumer surplus’ (1979) 95 LQR 581. Harrison, Good Faith in Sales, 1997, London: Sweet & Maxwell. Howarth, W, ‘The meaning of objectivity in contract’ (1984) 100 LQR 265. Jacobs, J, ‘The battle of the forms: standard term contracts in comparative perspective’ (1985) 34 ICLQ 297. Kessler, ‘Contracts of adhesion: some thoughts about freedom of contract’ (1943) Columbia LR 629. Law Commission, First Report on Exemption Clauses in Contracts, Law Com No 24, 1969, London: HMSO. Law Commission, Firm Offers, 1975, Working Paper No 60, London: HMSO. Law Commission, Second Report on Exemption Clauses in Contracts, Law Com No 69, 1975, London: HMSO. Law Commission, Law of Contract: The Parol Evidence Rule, Law Com No 154, Cmnd 9700, 1986, London: HMSO. Law Commission, Privity of Contract: Contracts For The Benefit of Third Parties, Law Com No 242, Cm 3329, 1996, London: HMSO." In Sourcebook on Contract Law, 809. Routledge-Cavendish, 1995. http://dx.doi.org/10.4324/9781843141518-323.

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Conference papers on the topic "Hamsoch"

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Joshi, Dev, Nathaniel Frissell, William Liles, Juha Vierinen, and Ethan S. Miller. "Early Results from the Ionospheric Sounding Mode Using Chirp Ionosondes of Opportunity for the HamSCI Personal Space Weather Station." In 2021 XXXIVth General Assembly and Scientific Symposium of the International Union of Radio Science (URSI GASS). IEEE, 2021. http://dx.doi.org/10.23919/ursigass51995.2021.9560441.

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Soares, Carlos Guedes, Ralf Weisse, Juan Carlos Carretero, and Enrique Alvarez. "A 40 Year Hindcast of Wind, Sea Level and Waves in European Waters." In ASME 2002 21st International Conference on Offshore Mechanics and Arctic Engineering. ASMEDC, 2002. http://dx.doi.org/10.1115/omae2002-28604.

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The paper describes the initial efforts in a project whose objective is to obtain a 40-year hindcast of wind, sea level and wave climatology for European waters. The 40-year global atmospheric re-analysis carried out by the National Centre for Environmental Prediction, Washington, USA (NCEP) and the National Centre for Atmospheric Research, Boulder, Colorado, USA (NCAR) will be used as forcing of limited area atmospheric models. The fine grid atmospheric fields will be used to force state-of-the-art wave models (WAM) and sea level models (HAMSOM and TELEMAC) in regional areas around Europe so as to produce climatic information on waves, sea levels, and currents in a very large extend of the European waters, including the Mediterranean, North East Atlantic and North Sea. The available satellite data, including wind, wave and sea-level data, will be collected and will be used to be compared with the hindcast results, so as to yield uncertainty measures related to the data. Statistical analysis of the produced atmospheric, sea level and wave hindcast and remote sensed data will be performed in order to provide information about the climatological trends in the European Waters and Coastal Seas.
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