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Journal articles on the topic 'Handl'

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Published: 4 June 2021

Last updated: 2 February 2022

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1

Barros-Gutiérrez, Camilo Ernesto, Edwin Silva-Monsalve, and Sandra Gualtero-Trujillo. "Síndrome HaNDL." MedUNAB 17, no. 3 (February 6, 2015): 190–92. http://dx.doi.org/10.29375/01237047.1959.

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Introducción: El Síndrome HaNDL (Headache and neurologic deficits with cerebroespinal fluid lymphocytosis) por sus siglas en inglés, es una patología que cursa con cefalea, focalización neurológica y linfocitosis en el líquido cefalorraquídeo. Objetivo: este artículo busca presentar un caso de Síndrome de HaNDL, puesto que esta condición nosológica implica un reto diagnóstico. Resultados y conclusiones: Se presenta el caso de un paciente con diagnóstico de síndrome de HaNDL los hallazgos al examen físico y del líquido cefalorraquídeo. [Barros-Gutiérrez CE, Silva-Monsalve E, Gualtero-Trujillo S. Síndrome HaNDL. MedUNAB 2015; 17(3): 190-192].

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2

Handl, Günther. "CHAIR: Günther Handl." Proceedings of the ASIL Annual Meeting 83 (1989): 62. http://dx.doi.org/10.1017/s0272503700075054.

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Fernandez, Nicholas, Ruwani Wijeyekoon, Anna Richardson, and Matthew Jones. "HaNDL with care." Acute Medicine Journal 14, no. 3 (July 1, 2015): 119–21. http://dx.doi.org/10.52964/amja.0446.

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Headache with neurological deficit and cerebrospinal fluid (CSF) lymphocytosis (HaNDL) syndrome is an important diagnosis to consider in patients presenting with the relevant features to acute medicine. Investigations should aim to exclude more serious differential diagnoses such as infectious, inflammatory and neoplastic causes prior to making a formal diagnosis of HaNDL. Increased awareness and early consideration of HaNDL would help to avoid unnecessary prolonged courses of antimicrobial therapy and invasive investigations such as cerebral angiography.

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4

García-Gómez, Francisco Javier, Pablo Antonio de la Riva-Pérez, Dolores Páramo, Guillermo Navarro-Mascarell, Luminita Dinca-Avarvarei, and María de la Cinta Calvo-Morón. "Diagnosis of HaNDL syndrome." Revista Española de Medicina Nuclear e Imagen Molecular (English Edition) 38, no. 5 (September 2019): 332–34. http://dx.doi.org/10.1016/j.remnie.2018.11.003.

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5

Handl, Gunther. "Remarks By Gunther Handl." Proceedings of the ASIL Annual Meeting 105 (2011): 427–30. http://dx.doi.org/10.5305/procannmeetasil.105.0427.

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Handl, Günther. "Remarks by Günther Handl." Proceedings of the ASIL Annual Meeting 79 (1985): 318–22. http://dx.doi.org/10.1017/s0272503700016827.

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Handl, Gunther F. "CHAIR: Gunther F. Handl." Proceedings of the ASIL Annual Meeting 82 (1988): 371. http://dx.doi.org/10.1017/s0272503700073663.

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8

Handl, Günther. "Remarks by Günther Handl." Proceedings of the ASIL Annual Meeting 83 (1989): 62–66. http://dx.doi.org/10.1017/s0272503700075066.

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9

Parasram, Melvin, Ashwin Malhotra, Andrea S. Yoo, and Saad A. Mir. "HaNDL Syndrome Presenting with Thunderclap Headache." Case Reports in Neurological Medicine 2021 (May 31, 2021): 1–3. http://dx.doi.org/10.1155/2021/9925004.

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Introduction. Transient headache and neurologic deficits with cerebrospinal fluid lymphocytosis (HaNDL) is defined as a secondary, nonvascular headache disorder characterized by the findings described in its name. Patients with HaNDL syndrome typically present with gradual onset migrainous headaches of moderate to severe intensity with transient neurological symptoms. Case Report. We discuss a patient who presented with thunderclap headache, recent transient neurologic deficits, and was ultimately diagnosed with HaNDL after an extensive neurologic evaluation. Conclusion. Thunderclap headache has very rarely been described in patients with HaNDL. After excluding emergent and secondary causes, HaNDL should be considered in patients with thunderclap-quality headaches, particularly when there is a history of transient neurological symptoms.

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DEMİR, Serkan, Asım ÜLÇAY, Mehmet Güney ŞENOL, and Ergenekon KARAGÖZ. "A Case Report Diagnosed as HaNDL Syndrome." Turkiye Klinikleri Journal of Case Reports 23, no. 3 (2015): 251–53. http://dx.doi.org/10.5336/caserep.2013-37960.

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11

Wang, Wendy, Heather G. Mack, Richard Stawell, Owen White, and John Waterston. "HaNDL with bilateral central venous occlusions." BMJ Neurology Open 2, no. 1 (June 2020): e000043. http://dx.doi.org/10.1136/bmjno-2020-000043.

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BackgroundThe syndrome of Headache and focal Neurological Deficits with cerebrospinal fluid Lymphocytosis (HaNDL) is considered a rare, idiopathic and self-limited condition.MethodsWe present a patient with HaNDL who had unique findings of florid optic disc haemorrhages from bilateral central retinal vein occlusions.ResultsOur patient made a full recovery with conservative management.ConclusionIt is important for medical attendants to recognise that HaNDL syndrome can be associated with neuro-ophthalmic complications.

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McKay, Sarah E., and Melanie R. F. Greenway. "A Case Report: The CSF Characteristics of HaNDL Syndrome." Journal of Doctoral Nursing Practice 13, no. 2 (July 1, 2020): 103–7. http://dx.doi.org/10.1891/jdnp-d-19-00045.

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BackgroundHeadache and Neurologic Deficits with cerebrospinal fluid Lymphocytosis (HaNDL) is a rare and underdiagnosed syndrome that mimics recurrent stroke, migraine with aura, and encephalitis.ObjectiveDescribe the presentation, clinical characteristics, and cerebrospinal fluid (CSF) findings in a patient with HaNDL and provide insight into the importance of the recognition of this syndrome.MethodsThe authors describe a unique case of (HaNDL) during which the patient underwent three lumbar punctures over 26 days.ResultsThe authors demonstrate the natural course of CSF characteristics of a patient with HaNDL, with rising and falling lymphocyte counts. Additionally, the authors provide an example of the clinical presentation of HaNDL, with episodic attacks over the course of 1 month of migraine headache, hemibody paresthesias, hemibody weakness, and encephalopathy.ConclusionsHaNDL is a headache syndrome mimicking viral encephalitis, migraine with aura, and recurrent cerebral ischemic events. While HaNDL is a diagnosis of exclusion, the syndrome's association with characteristic clinical and laboratory findings are important to recognize. Furthermore, a monophasic pattern of CSF lymphocytosis in HaNDL may be observed.Implications for NursingIncreased recognition of this syndrome may help prevent unnecessary tests and treatments when patients present with recurrent episodes.

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Sisman, Aysel Büsra, Muhammet Duran Bayar, Sema İçöz, Vuslat Yilmaz, Murat Kürtüncü, Erdem Tüzün, Mustafa Ertaş, and Birgül Baştan. "A Case of HaNDL with Low Cerebrospinal Fluid Level of Neurofilament Light Chain." Case Reports in Neurology 12, no. 3 (October 2, 2020): 334–38. http://dx.doi.org/10.1159/000508944.

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Diagnosis of the syndrome of headache and neurological deficits with cerebrospinal fluid (CSF) lymphocytosis (HaNDL) is based on clinical features, and no diagnostic biomarkers are available. We present a case presenting with characteristic features of HaNDL and an MRI lesion in the splenium of corpus callosum. CSF neurofilament light chain (NFL) levels were assessed in this patient together with 7 additional HaNDL patients, 18 multiple sclerosis (MS) patients, and 15 primary headache patients. Both HaNDL and primary headache patients showed significantly lower NFL levels than MS patients. Our results suggest that increased CSF levels of NFL and neuroaxonal loss are not characteristic features of HaNDL. Neurological disorders mimicking HaNDL often present with increased levels of NFL, and thus CSF measurement of NFL might be useful in differential diagnosis of HaNDL.

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García-Gómez, F. J., P. A. de la Riva-Pérez, D. Páramo, G. Navarro-Mascarell, L. Dinca-Avarvarei, and M. C. Calvo-Morón. "Diagnóstico de síndrome de HaNDL." Revista Española de Medicina Nuclear e Imagen Molecular 38, no. 5 (September 2019): 332–34. http://dx.doi.org/10.1016/j.remn.2018.11.001.

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Kaplan, Y., S. Kamisli, O. Kamisli, and H. Celik. "HaNDL syndrome presenting during pregnancy." Journal of the Neurological Sciences 333 (October 2013): e517-e518. http://dx.doi.org/10.1016/j.jns.2013.07.1827.

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Silva, Filipa, Rita Ferraz, Nelia Neves, Paulo Andrade, Claudia Carvalho, and Antonio Sarmento. "How to HaNDL This Case?" Journal of Medical Cases 7, no. 3 (2016): 84–86. http://dx.doi.org/10.14740/jmc2419w.

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Handl, Gunther F. "Remarks by Gunther F. Handl." Proceedings of the ASIL Annual Meeting 82 (1988): 371–73. http://dx.doi.org/10.1017/s0272503700073675.

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18

Barragán, Nuria García, Marta Guillán, Beatriz Zarza, Cristina González-Gordaliza, Alicia Felipe Mimbrera, María Alonso Leciñana, and Jaime Masjuan. "Focal CT hypoperfusion in HaNDL." Journal of Neurology 259, no. 8 (February 9, 2012): 1755–57. http://dx.doi.org/10.1007/s00415-012-6450-5.

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Adib-Samii, Poneh, Simon Little, Angela Vincent, and Niranjanan Nirmalananthan. "Case report: Headache and neurological deficits with CSF lymphocytosis (HaNDL) associated with P/Q type voltage-gated calcium channel antibodies (CACNA1A)." Cephalalgia 40, no. 9 (April 10, 2020): 1003–7. http://dx.doi.org/10.1177/0333102420916746.

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Background Headache and Neurological Deficits with cerebrospinal fluid (CSF) Lymphocytosis (HaNDL) is an increasingly recognised syndrome but the aetiology remains unclear. HaNDL has striking clinical features similar to Familial Hemiplegic Migraine (FHM), commonly related to gene mutations encoding the P/Q-type voltage-gated calcium channel (VGCC). Case report We report a case of HaNDL associated with high P/Q-type voltage-gated calcium channel antibodies. Extensive investigations excluded alternative diagnoses and CSF lymphocytosis resolved within 3 months. The case was complicated by raised intracranial pressure resulting in an enlarged blind spot, papilloedema and bilateral lateral rectus palsies. Conclusion This novel association of P/Q-type voltage-gated calcium channel antibodies with HaNDL has implications for the pathology of HaNDL and spectrum of voltage-gated calcium channel-antibody disorders. We compare the clinical features of FHM and HaNDL and the potential pathological role of these antibodies. This case also highlights that raised intracranial pressure is a common feature of HaNDL, rarely resulting in serious complications.

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20

Finke, Carsten, Annerose Mengel, Harald Prüss, Winfried Stöcker, Andreas Meisel, and Klemens Ruprecht. "Anti-NMDAR encephalitis mimicking HaNDL syndrome." Cephalalgia 34, no. 12 (March 11, 2014): 1012–14. http://dx.doi.org/10.1177/0333102414526070.

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Background Anti-NMDA receptor encephalitis typically manifests as severe multistage neuropsychiatric syndrome. However, milder or incomplete forms of the disorder have been recognised. Here, we report on a patient with anti-NMDA receptor encephalitis with a clinical phenotype mimicking the syndrome of headache with neurological deficits and cerebrospinal fluid (CSF) lymphocytosis (HaNDL). Case A 67-year-old man presented with recurrent stereotyped episodes of hemianopia, aphasia and right hemiparesis accompanied by throbbing headaches as well as confusion and agitation. CSF analysis showed lymphocytic pleocytosis. Additional analysis revealed NMDA receptor IgG antibodies in the patient’s CSF. Following immunotherapy, no further episodes occurred and NMDAR antibodies became undetectable. No NMDAR or other neuronal antibodies were detected in archived serum and CSF samples of 12 HaNDL patients fulfilling the current diagnostic criteria. Conclusions While anti-NMDAR encephalitis can manifest with a HaNDL-like clinical picture, HaNDL syndrome itself does not appear to be mediated by anti-NMDAR antibodies.

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Kürtüncü, Murat, Dilaver Kaya, Luigi Zuliani, Ece Erdağ, Sema İçöz, Elif Uğurel, Filiz Çavuş, et al. "CACNA1H antibodies associated with headache with neurological deficits and cerebrospinal fluid lymphocytosis (HaNDL)." Cephalalgia 33, no. 2 (October 30, 2012): 123–29. http://dx.doi.org/10.1177/0333102412463494.

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Background Patients with the syndrome of headache with neurological deficits and lymphocytosis (HaNDL) typically present with recurrent and temporary attacks of neurological symptoms and cerebrospinal fluid lymphocytosis. Aim and methods To identify potential HaNDL‐associated antibodies directed against neuronal surface and/or synapse antigens, sera of four HaNDL patients and controls were screened with indirect immunohistochemistry, immunofluorescence, cell-based assay, radioimmunoassay, protein macroarray and enzyme-linked immunosorbent assay (ELISA). Results Although HaNDL sera did not yield antibodies to any of the well-characterized neuronal surface or synapse antigens, protein macroarray and ELISA studies showed high-titer antibodies to a subunit of the T-type voltage-gated calcium channel (VGCC), CACNA1H, in sera of two HaNDL patients. Conclusion Our results support the notion that ion channel autoimmunity might at least partially contribute to HaNDL pathogenesis and occurrence of neurological symptoms.

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Gómez Alonso, Juan, Delicias Muñoz García, and María Rodríguez Rodríguez. "Síndrome HaNDL y encefalopatía de Hashimoto." Revista de Neurología 46, no. 04 (2008): 255. http://dx.doi.org/10.33588/rn.4604.2007655.

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Quintas, Sonia, Rocío López Ruiz, Santiago Trillo, Ana Beatriz Gago-Veiga, Gustavo Zapata-Wainberg, Julio Dotor García-Soto, Álvaro Ximénez-Carrillo, and Jose Vivancos. "Clinical, imaging and electroencephalographic characterization of three cases of HaNDL syndrome." Cephalalgia 38, no. 7 (October 3, 2017): 1402–6. http://dx.doi.org/10.1177/0333102417735846.

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Introduction The syndrome of transient headache and neurological deficits with cerebrospinal fluid lymphocytosis (HaNDL) may mimic stroke when patients present with acute/subacute focal neurological deficits. It would be helpful to identify investigations that assist the neurologist in differentiating between HaNDL and stroke. Case reports We describe three cases that proved to be HaNDL, but were initially considered to be strokes. Hypoperfusion was noted in the CT perfusion (CTP) studies in all three cases, which extended beyond any single cerebral arterial supply. The CTP findings suggested a stroke mimic, and there was no improvement on thrombolysis. MRI failed to show any abnormalities in diffusion and EEGs showed non-epileptiform changes. Lumbar punctures demonstrated a lymphocytic pleocytosis. Conclusion The diagnosis of HaNDL is based on clinical and CSF criteria, but neuroimaging, including CT perfusion, can be helpful in differentiating the clinical syndrome from stroke.

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Nelson, Sarah. "Confusional State in HaNDL Syndrome: Case Report and Literature Review." Case Reports in Neurological Medicine 2013 (2013): 1–4. http://dx.doi.org/10.1155/2013/317685.

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The syndrome of transient headache and neurologic deficits with cerebrospinal fluid lymphocytosis (HaNDL syndrome) is a self-limited condition. Confusional states are uncommonly reported as a clinical manifestation of this syndrome. Here, I report a 76-year-old female who presented with headache, confusion, and agitation with a mild CSF lymphocytosis. Other workup to determine the cause of her altered mental status was otherwise negative. The literature available in the English language on HaNDL syndrome is reviewed, including its history, pathophysiology, possible associations with migraine and stroke, and previously reported cases of confusional states in this syndrome. While HaNDL syndrome has been a described entity since the 1980s, its pathophysiology has yet to be clearly defined.

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Stelten, Bianca ML, Jeroen Venhovens, Lieven BJ van der Velden, Jan Meulstee, and Wim IM Verhagen. "Syndrome of transient headache and neurological deficits with cerebrospinal fluid lymphocytosis (HaNDL): A case report with serial electroencephalography (EEG) recordings.. Is there an association with human herpes virus type 7 (HHV-7) infection?" Cephalalgia 36, no. 13 (July 20, 2016): 1296–301. http://dx.doi.org/10.1177/0333102415618616.

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Introduction The syndrome of transient headache and neurological deficits with cerebrospinal fluid lymphocytosis (HaNDL) is a diagnosis made by exclusion. In the literature, different etiological explanations are proposed for HaNDL, including an immune-mediated reaction after a viral infection. Case description We present a case of a 23-year-old woman with several episodes of transient headache, neurological deficits and cerebrospinal fluid lymphocytosis. All diagnostic criteria for the HaNDL syndrome were fulfilled; however, additional cerebrospinal fluid analysis showed a positive polymerase chain reaction (PCR) for human herpes virus type 7 (HHV-7). Discussion The possible role of a (prodromal) viral infection in the etiology of HaNDL is discussed. Also the role of electroencephalography (EEG) recordings is discussed. Serial EEG recordings showed generalized slowing, frontal intermittent rhythmic delta activity (FIRDA) and symmetric triphasic frontal waves with a dilation lag.

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Parissis, Dimitris, Panagiotis Ioannidis, George Balamoutsos, and Dimitris Karacostas. "Confusional State in the Syndrome of HaNDL." Headache: The Journal of Head and Face Pain 51, no. 8 (March 31, 2011): 1285–88. http://dx.doi.org/10.1111/j.1526-4610.2011.01884.x.

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Armstrong-Javors, Amy, and Kalpathy Krishnamoorthy. "HaNDL Syndrome: Case Report and Literature Review." Journal of Child Neurology 34, no. 3 (December 5, 2018): 161–67. http://dx.doi.org/10.1177/0883073818811546.

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Headache and Neurologic Deficits with cerebrospinal fluid Lymphocytosis (HaNDL) syndrome is a rare stroke mimicker characterized by moderate to severe headache temporally associated with transient neurologic deficits, typically hemiparesis, hemisensory disturbance, and/or aphasia. Cerebrospinal fluid studies reveal a lymphocytosis and elevated protein. Episodes recur over a period no longer than 3 months. Here we describe the case of a 16-year-old boy who presented with 3 episodes of self-resolving neurologic deficits, papilledema on fundoscopic examination, and leptomeningeal enhancement on magnetic resonance imaging (MRI). We additionally review the 30 previously reported pediatric cases of HaNDL syndrome, with a focus on possible etiologic and pathophysiologic mechanisms of disease. The reported case and literature review highlight the benign episodic nature of this likely underrecognized syndrome as well as the higher than expected frequency of abnormal neuroimaging findings.

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Martín Balbuena, Sebastián, and Fco Javier Arpa Gutiérrez. "Síndrome HaNDL y encefalopatía de Hashimoto. Réplica." Revista de Neurología 48, no. 02 (2009): 112. http://dx.doi.org/10.33588/rn.4802.2008545.

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Fernandes, Linford, and Jeremy Cosgrove. "A Case of HaNDL Presenting With Papilledema." Headache: The Journal of Head and Face Pain 60, no. 6 (March 13, 2020): 1196–97. http://dx.doi.org/10.1111/head.13787.

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Thomas, Merlin C., Robert Walker, and Alan Wright. "HaNDL SYNDROME AFTER "BENIGN" OKT3-INDUCED MENINGITIS." Transplantation 67, no. 10 (May 1999): 1384–85. http://dx.doi.org/10.1097/00007890-199905270-00019.

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Apetse, Kossivi, Ludovic Breynaert, Chloe Butaud, Albert Beschet, Karine Blanc-Lasserre, Loic Ribouillard, and Victor Chan. "Transient Headache and Neurological Deficits with Cerebrospinal Fluid Lymphocytosis Associated with IgM Antibodies to the Epstein-Barr Virus Viral Capsid Antigen." Case Reports in Neurological Medicine 2013 (2013): 1–3. http://dx.doi.org/10.1155/2013/975709.

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Some authors have suggested that the syndrome of transient headache and neurological deficits with cerebrospinal fluid lymphocytosis (HaNDL) results from an immunological response directed against a viral agent. Here we report a case of HaNDL in an immunocompetent 19-year-old male that could support this hypothesis.

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Ashhurst, Jasmine F., and Rami Haddad. "085 Syndrome of transient headache and neurologic deficits with cerebral fluid lymphocytosis (HANDL) as a mimic for transient ischaemic attack (TIA)." Journal of Neurology, Neurosurgery & Psychiatry 90, e7 (July 2019): A27.2—A27. http://dx.doi.org/10.1136/jnnp-2019-anzan.73.

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IntroductionHaNDL is a rare neurological disorder of unknown aetiology that is characterised by headache, neurological deficit and pleocytosis in the cerebrospinal fluid (CSF). It is a benign condition that has spontaneous resolution of symptoms within months.A 50 year old female presented to Emergency with an acute focal neurological deficit of right sided weakness, dysphasia and dysarthria lasting less than one hour. Over the preceding months she had a new onset of headache. She was worked up for a likely diagnosis of TIA.MethodsCase report.ResultsInitial CT Brain(angiography) showed no stroke or other identifiable cause of symptoms.MRI brain showed excessive nonspecific T2 hyperintensities, requiring further investigation for possible vasculitis. MR angiography was normal and there was no evidence of stroke.Viral PCR’s were negative.Lumbar puncture (LP) showed pleocytosis (leucocytes 309×10E6/L), raised intracranial pressure and high protein, raising suspicion for HaNDL. This normalised on subsequent outpatient LP, along with symptoms.ConclusionsA diagnosis of HaNDL is made as a diagnosis of exclusion, though should be considered as a differential diagnosis for various presentations in which transient acute focal neurology is a presenting complaint.Due to relatively few reported cases of HaNDL, it is possible that HaNDL is being underdiagnosed due to variability in patient presentation and lack of understanding of the syndrome.As in this case, when a patient presents with transient acute focal neurology in the absence of headache as a prominent presenting symptom, it is reasonable to consider HaNDL as a differential diagnosis.

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Erdag, Ece, Erdem Tuzun, Murat Kurtuncu, and Burcak Vural. "Antibodies to DNA repair proteins in HaNDL patients." Journal of Neuroimmunology 275, no. 1-2 (October 2014): 59. http://dx.doi.org/10.1016/j.jneuroim.2014.08.155.

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BIÇAKCI, Şebnem, Behice KURTARAN, Mehmet Fahrettin OVER, and Yunus Kenan BIÇAKCI. "ICHD-II’ya göre HaNDL Sendromunun Yorumları Yeterli mi?" Nöro Psikiyatri Arşivi 51, no. 2 (June 5, 2014): 178–80. http://dx.doi.org/10.4274/npa.y6858.

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Gabaldón Albero, Alba, David Bernad Mechó, Marta Alemany Albert, María Desamparados Villalba Pérez, and Miguel Tomás Vila. "Síndrome HaNDL en una niña de 12 años." Revista de Neurología 69, no. 08 (2019): 332. http://dx.doi.org/10.33588/rn.6908.2019205.

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Frediani, Fabio, and Gennaro Bussone. "Confusional state as first symptom of HaNDL syndrome." Neurological Sciences 36, S1 (May 2015): 71–74. http://dx.doi.org/10.1007/s10072-015-2194-1.

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Rathore, Geetanjali S. "Generalized Convulsions- a rare Presentation of HaNDL Syndrome." American Journal of Biomedical Science & Research 10, no. 2 (September 2, 2020): 100–102. http://dx.doi.org/10.34297/ajbsr.2020.08.001484.

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Hazeena, Philo, Naga Karthik Vanukuri, and Shankar V. "Rare mimicker of transient ischaemic attacks: the syndrome of transient headache and neurological deficits with cerebrospinal luid lymphocytosis." International Journal of Research in Medical Sciences 7, no. 6 (May 29, 2019): 2442. http://dx.doi.org/10.18203/2320-6012.ijrms20192545.

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The syndrome of transient headache and neurologic deficits associated with cerebrospinal fluid lymphocytosis (HaNDL) is a benign and self limiting disorder characterized by 1 or more episodes of severe headache and transient neurologic deficits with lymphocytic pleocytosis in the cerebrospinal fluid. We report a case of a 30-year-old male who presented with four episodes of headache followed by hemi paresis and hemisensory loss in a week the patient was completely asymptomatic in between each episode of headache with neurological deficits. Persistent serial imaging to visualize the brain were normal with evidence of cerebrospinal fluid lymphocytosis, which lead to the diagnosis of HaNDL. We need to advocate a high degree of suspicion for HaNDL in the background of strong clinical history and findings, when imaging is normal.

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Vieira, José Pedro, Maria João Brito, and Isabel Lopes de Carvalho. "Borrelia lusitaniae Infection Mimicking Headache, Neurologic Deficits, and Cerebrospinal Fluid Lymphocytosis." Journal of Child Neurology 34, no. 12 (June 28, 2019): 748–50. http://dx.doi.org/10.1177/0883073819858263.

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Headache with neurologic deficits and cerebrospinal fluid lymphocytosis (HaNDL) is a rare headache syndrome included in the Classification of Headache of the International Headache Society as a “headache attributed to non-infectious inflammatory intracranial disease.” We report one 15-year-old patient with clinical history and cerebrospinal fluid findings compatible with the diagnosis of HaNDL in whom Borrelia lusitaniae was identified in cerebrospinal fluid by polymerase chain reaction.

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Yilmaz, A., H. Kaleagasi, O. Dogu, E. Kara, and A. Ozge. "Abnormal MRI in a patient with ‘headache with neurological deficits and CSF lymphocytosis (HaNDL)’." Cephalalgia 30, no. 5 (August 1, 2009): 615–19. http://dx.doi.org/10.1111/j.1468-2982.2009.01950.x.

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A 27-year-old woman was admitted to the Emergency Department with right upper-extremity numbness and mild weakness followed by a bifrontal throbbing headache for 30 min, which was similar to a headache lasting for 12 h that had occurred 3 days ago. Laboratory tests were unremarkable except for cerebrospinal fluid (CSF) lymphocytic pleocytosis. On the following day, a headache episode with left hemiparesis and hemihypoaesthesia, left hemifield visio-spatial inattention, anosagnosia and confusion recurred. The headache was diagnosed as headache and neurological deficits with cerebrospinal fluid lymphocytosis (HaNDL) syndrome according to the criteria of the second edition of the International Classification of Headache Disorders. Simultaneously performed magnetic resonance imaging (MRI) revealed swelling of the grey matter, CSF enhancement in the sulci of the right temporal and occipital regions and hypoperfusion of the same brain regions. During the following 10 days two more similar episodes recurred and during the ensuing 12 months the patient remained headache free. Neuroimaging findings of the HaNDL syndrome are always thought as virtually normal. MRI abnormalities in our patient have not been reported in HaNDL syndrome previously, although they have been reported in hemiplegic migraine patients before. The findings in our case suggest that hemiplegic migraine and HaNDL syndrome may share a common pathophysiological pathway resulting in similar imaging findings and neurological symptoms.

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Burke, Matthew J., Michael J. Lamb, Marika Hohol, and Christine Lay. "Unique CT Perfusion Imaging in a Case of HaNDL: New Insight into HaNDL Pathophysiology and Vasomotor Principles of Cortical Spreading Depression." Headache: The Journal of Head and Face Pain 57, no. 1 (October 24, 2016): 129–34. http://dx.doi.org/10.1111/head.12968.

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42

Gorai, Debobroto, Frank Niclason, and Michael Dreyer. "406: HaNDL syndrome – A revisit with 3 case reports." Journal of Clinical Neuroscience 15, no. 3 (March 2008): 343. http://dx.doi.org/10.1016/j.jocn.2007.07.019.

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43

Vallet, Anne-Evelyne, Virginie Desestret, Florence Tahon, Tae-Hee Cho, and Norbert Nighoghossian. "Acute Perfusion MR Imaging in a HaNDL-Like Syndrome." Cerebrovascular Diseases 29, no. 1 (2010): 98–100. http://dx.doi.org/10.1159/000259619.

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44

Kotan, Dilcan, Saadet Sayan, and Semra Alacam Koksal. "A rare neurologic deficiency in HaNDL syndrome: cranial neuropathy." Archives of Medical Science - Atherosclerotic Diseases 1 (2016): 49–52. http://dx.doi.org/10.5114/amsad.2016.60799.

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45

Bondesan, Aldino, Alberto Carton, and Vittoria Laterza. "Leo Handl and the Ice City (Marmolada Glacier, Italy)." Rendiconti Online della Società Geologica Italiana 36 (September 2015): 31–34. http://dx.doi.org/10.3301/rol.2015.137.

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46

Martínez-Velasco, Elena, Patricia Mulero, Johanna Barón, Mariam Amer, and Angel L. Guerrero. "Confusional state in HaNDL syndrome: an uncommon clinical manifestation." Neurological Sciences 37, no. 3 (December 12, 2015): 483–85. http://dx.doi.org/10.1007/s10072-015-2403-y.

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47

Gendre, T., C. Giron, S. Evrard, B. Lapergue, P. Graveleau, and F. Bourdain. "Lymphome primitif des séreuses méningé révélé par un syndrome HaNDL." Pratique Neurologique - FMC 5, no. 3 (September 2014): 219–22. http://dx.doi.org/10.1016/j.praneu.2014.06.010.

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48

Skorobogatykh, K., Y. Azimova, and G. Tabeeva. "Syndrome of headache with neurologic deficits and CSF lymphocytosis (HaNDL)." Journal of Headache and Pain 1, Suppl 1 (2013): P163. http://dx.doi.org/10.1186/1129-2377-1-s1-p163.

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49

Kulesh, A. A., N. L. Starikova, V. E. Drobakha, L. Yu Brokhin, and A. S. Chubarov. "The syndrome of transient headache and neurologic deficits with cerebrospinal fluid lymphocytosis (HaNDL): a description of three patients." Neurology, Neuropsychiatry, Psychosomatics 12, no. 4 (August 27, 2020): 65–72. http://dx.doi.org/10.14412/2074-2711-2020-4-65-72.

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Abstract:

Three clinical cases of the syndrome of transient headache and neurologic deficits with cerebrospinal fluid lymphocytosis (HaNDL) are first described in Russian literature. The patients were young (aged 30–35 years), had symptoms characteristic of the prodromal period of infections. In all the patients, the disease started with intense headache, followed by focal neurological symptoms: aphasia and hemihypesthesia in all cases and confusion with psychomotor agitation in two cases. All the three patients showed spontaneous recovery within 2–3 days. Perfusion computed tomography, magnetic resonance imaging, and electroencephalography are compared in one of the cases. The differential diagnosis of HaNDL with acute cerebrovascular accident, herpetic encephalitis, epilepsy, and migraine is discussed.

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50

Sýkora, Pavel. "Notes on the style and spiritual categorisation of Moralia by Jacobus Handl Gallus." Musicological Annual 44, no. 2 (December 1, 2008): 29–38. http://dx.doi.org/10.4312/mz.44.2.29-38.

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Abstract:

Toward the end of his life, Jacobus Handl-Gallus (1550-1591) wrote two books of madrigals: Harmoniae morales and Moralia. The uniting element of both cycles is, except for the moralizing contents, Latin language, which is not usual for madrigal style.

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