Relevant bibliographies by topics / Hashimoto's

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Hashimoto's'

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Published: 24 April 2022

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Journal articles on the topic "Hashimoto's"

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Pakdaman, Michael Navid, Michael P. Hier, Martin J. Black, Michael Tamilia, and Richard J. Payne. "Micropapillary Thyroid Carcinoma and Hashimoto's Thyroiditis." Otolaryngology–Head and Neck Surgery 139, no. 2_suppl (August 2008): P40. http://dx.doi.org/10.1016/j.otohns.2008.05.132.

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Objective An increase in papillary thyroid carcinoma in patients with Hashimoto's thyroiditis has been described. We have yet to find literature discussing a relationship with micropapillary thyroid carcinoma(MPC). Our objective is (1) to assess the relationship between Papillary Microcarcinoma and Hashimoto's Thyroiditis in thyroids without malignant disease, (2) compare effect of the presence of Hashimoto's on prognostic factors in patients. Methods Pathology reports were reviewed for consecutive patients undergoing total thyroidectomy over a 6-year period from 2002 to 2007. Patients with benign pathologies were included. Patients with MPC and no other malignancy were also included. This lead to a cohort of 314 patients. Statistical significance was calculated using Fisher's exact test and a chi-squared analysis. Results Out of the 314 patients, 98 were positive for Hashimoto's thyroiditis on biopsy of the removed thyroid. 58% of the patients with Hashimoto's thyroiditis had an incidental MPC, as compared to 42% of patients where Hashimoto's was not found. Additionally, in Hashimoto patients, 61% of the MPCs were multifocal versus 43% multifocal in the non-Hashimoto group (p-value <0.05 in each comparison). Bilaterality of MPC was also increased in patients with Hashimoto's (22% vs 39%). Conclusions Patients with Hashimoto's thyroiditis in this study had an incidence of MPC of 58%, which is significantly greater than the 42% in those without. Hashimoto's was also associated with an increased risk of bilaterality and multifocality. Studies are needed to further corroborate these findings.
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SHIBATA, MASAKI. "Why is Toru Hashimoto Called ‘a Japanese version of Trump’ or ‘Hitler’? A Linguistic Examination of Hashimoto's Attack on His Opponents." Japanese Journal of Political Science 19, no. 1 (January 17, 2018): 23–40. http://dx.doi.org/10.1017/s1468109917000202.

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AbstractThe former Osaka Mayor Toru Hashimoto is the focus of much public attention in Japan. He is a polarizing figure who is both liked and disliked in equal measures, and his political character, including his argumentative approach, has been widely discussed by the Japanese and international media, as well as within academic research. For example, theJapan Timeslabelled his speech style as ‘a Japanese version of Donald Trump and the former Tokyo mayor, Shintaro Ishihara, said Hashimoto's speech is like ‘Hitler's’. This study examines the differences between Hashimoto's rhetoric and that of other Japanese politicians. Of the various tactics involved in the argumentative strategy of Hashimoto, this study specifically focuses on his attacking of opponents, which is one of the common and important tactics used in political debate. This study analyses the three linguistic approaches used in political debate: the policies or character of the target; the evaluative meanings of attack, and negotiation used to reduce the assertiveness of attack. The data were drawn from a televised political debate, in which Hashimoto was required to debate with three opponents who opposed his points of view. This study demonstrates that the target and the evaluative meanings behind Hashimoto's attacks were not remarkably different from the other politicians. However, Hashimoto did not use negotiation and and he was more assertive in presenting criticisms while the other politicians’ criticisms were mediated by negotiation.
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Atia, Ahmed, Rihan Alathream, and Abdulwahab Al-Deib. "Incidence of Hashimoto Thyroiditis Among Libyans: A Retrospective Epidemiological Study." Journal of Medical Research and Innovation 5, no. 1 (March 6, 2021): e000251. http://dx.doi.org/10.32892/jmri.251.

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Background and aims. Hashimoto's disease is an autoimmune disorder in which the body produces antibodies that attack the thyroid gland, leading to chronic inflammation, destruction of the gland, and hypothyroidism. This study aimed to assess the epidemiology of this disease among Libyan patients. Methods. A cross-sectional retrospective study conducted from June 2012 to April 2020 in order to examine the anti TPO level among Libyan population. Data was collected from eastern and western part of Libya, and were analyzed from available sample for 244 apparently patients with thyroid disorders collected from different private clinic’s laboratories. The analysis for serum anti-TPO was done by electrochemiluminescence protein binding assay (ECLIA) using Roche diagnostics and Cobas e411 analyzer. Results. The current results showed that females predominate the study, and most of them were in the age group of (>40) years old. About 49.18% of these cases were suffering from Hashimoto's disease (High ATPO level). The mean value of anti-TPO status among females was (0.5±2) nmol/L, while among males it was (0.45±3) nmol/L. Significantly, more women (81.66%) had Anti- TPO Above (34 IU/ml), compared to (18.33%) of male participants. Conclusion. Hashimoto disease is common among patients with thyroid dysfunction especially females. Our findings suggest that different interventional strategies are needed to reduce the chances of developing Hashimoto’s and its associated negative health outcomes in Libya.
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Hapsari, Siti Nurul, and Sidarti Soehita. "Hyperthyroid Phase of Hashimoto's Thyroiditis." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 26, no. 1 (November 22, 2019): 123. http://dx.doi.org/10.24293/ijcpml.v26i1.1779.

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Hashimoto thyroiditis (chronic autoimmune thyroiditis) is the most common cause of hypothyroidism in iodine-sufficient areas of the world. This condition, however, can sometimes show hyperthyroidism. A 39-year-old femalewas admitted to hospital due to shortness of breath and tremor four hours before hospitalization. There were nausea, chestpain, cold chills, and palpitation. She was diagnosed with Hashimoto's thyroiditis and routinely received tyrosol,propranolol, and dexamethasone. Physical examination showed cervical mass, afebrile, blood pressure of 130/70 mmHg,pulse rate of 110 beats/minute and respiratory rate of 20 breaths/minute. Laboratory examinations showed WBC 7.53 x 109/L, Hb 11.0 g/dL and platelet count of 168 x 109/L. Chest X-Ray: negative for infiltrates. Several laboratory testswere performed, abnormal results were as follows: FT4 level of 2.96 ng/dL (increased), TSH level of 0.003 µIU/mL(decreased), anti-TPO (antithyroid microsomal antibody) level of 306 IU/ml (increased), and IgE level of 213.6 IU/mL(increased). Peripheral blood smear, coagulation test, serum electrolytes, liver function tests, renal function tests, urinalysis,CEA and Ca 125 were within normal limits. Thyroid ultrasound was performed and showed a benign lesion. Fine needleaspiration biopsy showed lymphocytic Hashimoto's thyroiditis. Echocardiography showed hyperthyroid heart disease. Dueto an increase of anti-TPO and FT4 levels, a decrease of TSH levels and lymphocytic thyroiditis from FNAB, this patient wasdiagnosed with a hyperthyroid phase of Hashimoto's thyroiditis. Thyroid function tests and thyroid antibody tests must bemonitored to distinguish between the hyperthyroid and hypothyroid phase of Hashimoto thyroiditis.
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Kandror, V. I., I. V. Kryukova, S. I. Krainova, N. A. Mkrtumova, Yu M. Keda, N. Yu Sviridenko, E. N. Bazarova, and N. V. Latkina. "Antithyroid antibodies and autoimmune diseases of the thyroid." Problems of Endocrinology 43, no. 3 (September 18, 2019): 25–30. http://dx.doi.org/10.14341/probl10392.

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The significance and mechanisms of action of different antithyroid antibodies in diffuse toxic goiter (Graves' disease) and chronic lymphocytic thyroiditis (Hashimoto’s disease) arc analyzed. Antibodies immediately decreasing the level of cAMP in isolated thyrocytes were revealed in the sera of adolescents with juvenile struma, often resulting from lymphocytic thyroiditis. Complement-fixing cytotoxic antibodies are heterogeneous in patients with Graves' diseases and Hashimoto's thyroiditis. Thyrocytes from the tissue of diffuse toxic goiter are resistant to the cytolytic effect of such antibodies from patients with Graves' diseases but not from patients with Hashimoto's thyroiditis. The causes and mechanisms of development of resistance of thyrocytes from diffuse toxic goiter to antibody-dependent complement-mediated cytotoxicity of sera from patients with Graves' disease and the possibility of using this phenomenon as a differential diagnostic test are discussed.
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Ramalho, Joana, and Mauricio Castillo. "Hashimoto's encephalopathy." Radiology Case Reports 6, no. 1 (2011): 445. http://dx.doi.org/10.2484/rcr.v6i1.445.

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KONISHI, JUNJI. "Hashimoto's disease." Practica Oto-Rhino-Laryngologica 79, no. 1 (1986): 106–10. http://dx.doi.org/10.5631/jibirin.79.106.

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Aquino, Renata Telles Rudge de, and Eduardo Genaro Mutarelli. "Hashimoto's encephalopathy." Arquivos de Neuro-Psiquiatria 67, no. 3a (September 2009): 724–25. http://dx.doi.org/10.1590/s0004-282x2009000400034.

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ZIMMERMAN, ROBERT S. "Hashimoto's Thyroiditis." Annals of Internal Medicine 104, no. 3 (March 1, 1986): 355. http://dx.doi.org/10.7326/0003-4819-104-3-355.

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Sue, C. M., V. Fung, J. P. Halpern, S. C. Boyages, and C. Yiannikas. "Hashimoto's encephalopathy." Journal of Clinical Neuroscience 4, no. 1 (January 1997): 74–77. http://dx.doi.org/10.1016/s0967-5868(97)90018-7.

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Dissertations / Theses on the topic "Hashimoto's"

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Quintino-Moro, Alessandra 1973. "Infertilidade em mulheres com Doença de Graves e Tireoidite de Hashimoto : Infertility among women with Graves's disease or Hashimoto's thyroiditis." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308429.

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Orientadores: Arlete Maria dos Santos Fernandes, Denise Engelbrecht Zantut Wittmann
Dissertação (Mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: A disfunção autoimune, representada pelo hipertireoidismo da Doença de Graves (DG) e pelo hipotireoidismo da Tireoidite de Hashimoto (TH), ocorre de forma mais frequente em mulheres e interfere no mecanismo da reprodução, em especial no processo ovulatório. Existe o senso comum de que as disfunções tireoideanas diminuem o potencial de fertilidade das mulheres; entretanto, não existem estudos sobre a prevalência da infertilidade nesses grupos. Objetivos: Determinar a prevalência de infertilidade em mulheres com TH e DG e as possíveis variáveis associadas. Sujeitos e métodos: Foi um estudo de corte transversal. Mulheres com TH (n=66) e idade entre 18 e 60 anos e DG (n=193) com idade entre 18 e 50 anos, em seguimento no Ambulatório de Tireoidopatias do HC/UNICAMP, no período de agosto de 2010 a dezembro de 2011, foram entrevistadas com respeito às variáveis ginecoobstétricas: história de períodos de infertilidade, alterações do ciclo menstrual, história obstétrica e antecedentes familiares. Infertilidade foi definida como ausência de gravidez após período de exposição ?12 meses. Após a entrevista, seus prontuários foram revisados para determinar as características da doença: idade ao diagnóstico, tempo do diagnóstico, antecedentes de outras doenças autoimunes associadas, exames laboratoriais e de imagem. O estudo obteve aprovação do Comitê de Ética em Pesquisa da FCM/UNICAMP. Os critérios de inclusão foram: ao menos um ano de convívio com parceiro masculino e concordância em participar da pesquisa. Os dados foram anotados em ficha de coleta e posteriormente digitados em banco de dados elaborado para o estudo. Após a consistência do banco, foi realizada análise descritiva das variáveis gineco-obstétricas e características da doença, e foram aplicados os testes quiquadrado ou exato de Fisher, teste de Mann-Whitney e teste de Wilcoxon. Posteriormente, foi realizada a análise univariada com o cálculo de Odds Ratio bruto e respectivo intervalo de confiança (IC) de 95%, seguida pela regressão logística múltipla incluindo todas as variáveis, buscando aquelas significativamente associadas à infertilidade. O grau de significância estatística foi de 5%. Resultados: A prevalência de infertilidade foi de 52,3% e 47,0% nas mulheres, respectivamente com DG e TH. Na DG, as perdas gestacionais tiveram prevalência de 18,8% e 21,7% em mulheres com e sem infertilidade, enquanto na TH as perdas gestacionais ocorreram em 22,6% e 20,6% nos mesmos grupos. A média do número de gestações foi menor após o diagnóstico de DG e TH nas mulheres com idade ?35 anos. No mesmo grupo de idade, a média do número de gestações antes do diagnóstico foi de 1,68 (DP ±1,41) para DG e 1,48 (DP ±1,31) para TH. As alterações de ciclo menstrual na DG ocorreram em 47,5% e 35,9% das mulheres, respectivamente com e sem infertilidade, e na TH as taxas foram de 64,5% e 34,3% nos mesmos grupos. A única variável associada à infertilidade na TH foi o tempo de doença menor que seis anos. Não houve variável associada nas mulheres com DG. Conclusões: a prevalência de infertilidade foi alta e, no grupo de mulheres com idade ?35 anos, houve diminuição na média de gestações, mostrando o comprometimento da fertilidade das mulheres com DG e TH. Mulheres com TH com menos tempo de doença foram as mais afetadas pela infertilidade
Abstract: The autoimmune dysfunction, hyperthyroidism represented by the Graves disease (GD) and by hypothyroidism of Hashimoto's thyroiditis (TH), occurs more often in women and interferes in the mechanism of reproduction, especially in the ovulatory process. There is common sense that thyroid dysfunction decreases the fertility potential of women, however, there are no studies on the prevalence of infertility in these groups. Objectives: To determine the prevalence of infertility in women with TH and DG and possible associated variables. Subjects and Methods: This was a cross-sectional study. Women with TH (n = 66) aged between 18 and 60 years and DG (n = 193) aged between 18 and 50 years was followed at the Endocrinology Division, Departament of Clinical Medicine, School of Medical Sciences, University of Campinas (UNICAMP), from August 2010 to December 2011 were interviewed with respect to gynecological and obstetric variables: history of periods of infertility, menstrual abnormalities, obstetric history and family history. Infertility was defined as 12 months of unprotected sexual intercourse without conception. After the interview, their medical records were reviewed to determine the characteristics of the disease: age at diagnosis, time since diagnosis, history of other autoimmune diseases associated, laboratory tests and imaging tests. The study was approved by the Research Ethics Committee of the FCM/UNICAMP. Inclusion criteria were: at least a year of living with a male partner, and agreed to participate. Data were recorded on collection and subsequently entered into a Summary - xvii database designed for the study. After the database consistency, descriptive analysis was performed gynecological and obstetric variables and disease characteristics, and applied the chi-square or Fisher's exact test, Mann-Whitney and Wilcoxon test. Subsequently, univariate analysis was performed to calculate crude odds ratios and confidence intervals (CI) of 95%, followed by multiple logistic regressions including all variables significantly associated with those seeking infertility. The level of statistical significance was 5%. Results: The prevalence of infertility was 52.3% and 47.0% in women, with DG and TH respectively. In DG, the miscarriages had a prevalence of 18.8% and 21.7% in women with and without infertility, while in the TH pregnancy loss occurred in 22.6% and 20.6% in the same groups. The mean number of pregnancies was lower after the diagnosis of GD and HT in women aged ? 35 years. In the same age group, the mean number of pregnancies before diagnosis was 1.68 (SD ± 1.41) for DG and 1.48 (SD ± 1.31) for TH. Changes in the menstrual cycle in DG occurred in 47.5% and 35.9% of women, respectively with and without infertility, and TH rates were 64.5% and 34.3% in the same groups. The only variable associated with infertility in TH disease duration was less than six years. There was no associated variable in women with GD. Conclusions: The prevalence of infertility was high and, in the group of women aged ? 35 years, there was a decrease in average pregnancies, showing impairment of fertility in women with GD and HT. Women with TH with shorter disease were most affected by infertility
Mestrado
Fisiopatologia Ginecológica
Mestra em Ciências da Saúde
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Kashanian, Gholam Reza Moshtaghi. "Role of bioactive peptides in autoimmune thyroid disease." Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318115.

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Ewins, David Laurence. "Characterisation of autoantigenic epitopes on thyroid peroxidase recognised by antibodies and T lymphocytes in autoimmune thryroid disease." Thesis, University of Southampton, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240770.

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Wu, Zhonglin. "Molecular analysis of HLA class II genes : from Hashimoto's thyroiditis to thyrocytes." Thesis, Queen Mary, University of London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265188.

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Clemens, Cody Marshall. "Stories To Tell: Examining Experiences And Identities Of Individuals With Hashimoto's Thyroiditis." Bowling Green State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1585771697321886.

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Botelho, Ilka Mara Borges 1979. "Prevalência de insuficiência de vitamina D em pacientes com tireoidite de Hashimoto e sua relação com autoimunidade tireoideana = Prevalence of vitamin D insufficiency in patients with Hashimoto's thyroiditis and its relationship with thyroid autoimmunity." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308788.

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Orientadores: Denise Engelbrecht Zantut Wittmann, Sarah Monte Alegre
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: Introdução: Vitamina D tem sido apontada como importante regulador da resposta imune. Estudos tem demonstrado haver relação entre insuficiência de vitamina D e presença de doenças autoimunes como Tireoidite de Hashimoto (TH). É possível que o processo autoimune na TH seja inibido em diferentes estágios pela vitamina D em sua forma ativa. Nossos objetivos foram estudar a prevalência de insuficiência de vitamina D e a relação de suas concentrações séricas com marcadores de função e autoimunidade tireoideana. Material e Métodos: Amostras de sangue foram coletadas de 54 pacientes com TH e 54 indivíduos saudáveis sem diagnóstico de TH com idade entre 18 e 75 anos. Foram realizadas dosagens séricas de vitamina D (25OHD), TSH, T4 livre, cálcio, fósforo, paratormônio (PTH), anticorpos anti-tireoperoxidase (AcTPO), anti-tireoglobulina (AcTG) e anti-receptor de TSH (TRAb). Volume tireoideano foi estimado por ultrassonografia. Foram coletados dados demográficos, de peso, altura, índice de massa corporal (IMC) e tempo de diagnóstico. Pacientes e indivíduos do grupo de controle foram pareados por idade e sexo. O nível de significância estatística adotado foi 5%. Resultados: Prevalência de insuficiência de vitamina D foi encontrada em 68.5% dos pacientes e em 38.9% dos indivíduos do grupo de controle (p =0,002). Houve uma correlação positiva entre níveis de AcTPO e maior volumetireoideano nos pacientes (r = 0,319; p= 0.019). Não houve correlação entre concentração de vitamina D, TSH, T4livre,TRAb, AcTGe volume tireoideano. Conclusões: Demonstramosmaior prevalência deinsuficiência de vitamina Dem pacientescom tireoidite de Hashimotoem relaçãoa indivíduos de um grupo controlesaudável, não havendo correlaçãocom o estado hormnal tireoideanooumarcadores séricos deautoimunidadeda tireóide.Por sua vez, maior volume da tireóidese associou a maior grau de infiltração inflamatóriaautoimune,refletido pelacorrelaçãocom maiores concentrações AcTPO
Abstract: Introduction: Vitamin D has been pointed out as an important immune response regulator. Studies have shown a relationship between vitamin D insufficiency and the presence of autoimmune diseases such as Hashimoto's Thyroiditis (HT). It's possible that the autoimmune process in HT is inhibited in its different stages by vitamin D on its active form .Our aims were to study the prevalence of vitamin D insufficiency and relationship of the serum concentrations with thyroid function and autoimmunity markers. Material and Methods: Blood samples were collected from 54 patients with HT and 54 healthy individuals without a diagnosis of HT, aged 18 to 75 years. We conducted serum 25OH vitamin D, TSH, free T4, calcium, phosphorus, PTH, TPOAb, TgAb and TRAb. Thyroid volume was estimated by ultrasound. Data on demographic, weight, height, body mass index and time since diagnosis were collected. Patients and control subjects were matched by sexand age. The significance level for statistical analysis was 5%. Results: Prevalence of vitamin D insufficiency was found in 68.5% of patients and in 38.9% of subjects in the control group (p= 0.002). There was a positive correlation between TPOAb and volume in patients (p= 0.019). There was no correlation between vitamin D concentration and thyroid volume, TRAb, TgAb, TSH or free T4. Conclusions: We demonstrated a higher prevalence of vitamin D insufficiency in patients with Hashimoto's thyroiditis compared to individuals of a healthy control group, no correlation with thyroid state hormonal or serum markers of thyroid autoimmunity. In turn, greater thyroid volume was associated with a higher degree of autoimmune inflammatory infiltration, reflected by the correlation with higher concentrations AcTPO
Mestrado
Clinica Medica
Mestra em Ciências
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Arda, Saadet [Verfasser]. "Psychische Störungen bei Autoimmunthyreoiditis (Hashimoto) / Saadet Arda." Ulm : Universität Ulm. Medizinische Fakultät, 2014. http://d-nb.info/1053381468/34.

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陳結清 and Kit-ching Amanda Chan. "Detection of RET/PTC translocation in hashimoto thyroiditis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40738413.

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Chan, Kit-ching Amanda. "Detection of RET/PTC translocation in hashimoto thyroiditis." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B40738413.

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DELIGNE, DI MARCANTONIO DI MARTINO AUTILIA. "Mecanismes de l'hypothyroidie au cours de la thyroidite de hashimoto." Lille 2, 1993. http://www.theses.fr/1993LIL2M170.

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Books on the topic "Hashimoto's"

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Kharrazian, Datis. Why do I still have thyroid symptoms?: When my lab tests are normal : a revolutionary breakthrough in understanding Hashimoto's disease and hypothyroidism. Carlsbad, CA, USA: Elephant Press LP, 2010.

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Why do I still have thyroid symptoms? when my lab tests are normal: A revolutionary breakthrough in understanding hashimoto's disease and hypothyroidism. 3rd ed. Carlsbad, CA: Elephant Printing LLC, 2012.

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Iinkai, Sagamihara-shi Kyōiku. Hashimoto iseki: Hashimoto site 1981-1986. Sagamihara-shi: Sagamihara-shi Kyōiku Iinkai, 1987.

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Hashimoto gekijō. Tōkyō: Chūō Kōron Shinsha, 2012.

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Iwami, Takao. Jitsuroku Hashimoto Ryūtarō. Tōkyō: Asahi Sonorama, 1995.

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Nakane, Yukie. Hashimoto Sanai jiseki. Fukui-shi: Fukui Shiritsu Kyōdo Rekishi Hakubutsukan, 1987.

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Hashimoto Shirō ronbunshū. Tōkyō: Kadokawa Shoten, 1986.

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Hashimoto, Meiji. Hashimoto Meiji kaikoten. [Tokyo]: Nihon Keizai Shinbunsha, 1992.

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Hashimoto, Yoshimi. Yoshimi Hashimoto: Neue Skulpturen. St. Wendel: Museum St. Wendel, 1994.

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Hashimoto gyōkaku no kenshō. Tōkyō: Gyōsei, 2006.

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Book chapters on the topic "Hashimoto's"

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, Luiz Marco, Dror Robinson, Eitan Melamed, Alexander K. C. Leung, et al. "Hashimoto's Disease." In Encyclopedia of Molecular Mechanisms of Disease, 776. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8669.

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, Luiz Marco, Dror Robinson, Eitan Melamed, Alexander K. C. Leung, et al. "Hashimoto's Thyroiditis." In Encyclopedia of Molecular Mechanisms of Disease, 775–76. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_747.

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Radetti, Giorgio. "Clinical Aspects of Hashimoto's Thyroiditis." In Paediatric Thyroidology, 158–70. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000363162.

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Wiersinga, Wilmar M. "Hashimoto’s Thyroiditis." In Endocrinology, 205–47. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-45013-1_7.

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Wiersinga, Wilmar M. "Hashimoto’s Thyroiditis." In Endocrinology, 1–44. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29195-6_7-1.

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Yoneda, Makoto, Akiko Matsunaga, and Masamichi Ikawa. "Hashimoto’s Encephalopathy." In Neuroimmunological Diseases, 235–44. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55594-0_15.

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Halenka, Milan, and Zdeněk Fryšák. "Hashimoto’s Thyroiditis." In Atlas of Thyroid Ultrasonography, 17–39. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53759-7_3.

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Chew, Sheena, and Nagagopal Venna. "Hashimoto’s Encephalopathy." In Neurorheumatology, 169–78. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-16928-2_17.

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Coca, Andrés, and Carlos Suárez Nieto. "Hashimoto’s Thyroiditis." In Otorhinolaryngology, Head and Neck Surgery, 587. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68940-9_63.

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Sakr, Mahmoud F. "Hashimoto’s Disease." In Thyroid Disease, 71–132. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-48775-1_2.

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Conference papers on the topic "Hashimoto's"

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Endres, D. "The role of Hashimoto's encephalopathy in psychiatry." In Abstracts of the 30th Symposium of the AGNP. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1606422.

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Xue, Shuai, Peisong Wang, Zhe Han, Chen Guang, Jia Liu, and Li Zhang. "Analysis of Ultrasonic Characteristics of Hashimoto's Thyroiditis Benign Nodules and Its Relationship with Serum TSH." In 2015 7th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2015. http://dx.doi.org/10.1109/itme.2015.16.

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Moskva, K., O. Kikhtyak, and L. Lapovets. "CHANGES OF GUT MICROBIOTA UNDER THE INFLUENCE OF METFORMIN, PIOGLITAZONE, AND LEVOTHYROXINE IN OVERWEIGHT PATIENTS WITH TYPE 2 DIABETES MELLITUS AND HYPOTHYROIDISM CAUSED BY HASHIMOTO'S THYROIDITIS." In GRUNDLAGEN DER MODERNEN WISSENSCHAFTLICHEN FORSCHUNG. European Scientific Platform, 2021. http://dx.doi.org/10.36074/logos-10.09.2021.68.

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Delfim, William de Souza, Nayara Christina de Lima Curti, Marília Pires de Souza e. Silva, Lorena Dias Araújo, Indianara Keila Pastorio, Francine de Paula Roberto Domingos, Sayuri Aparecida Hirayama, Rafael de Almeida, Raquel de Mattos Filgueiras, and Rafael Batista João. "The diagnostic challenge of Hashimoto’s Encephalopathy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.579.

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Introduction: The diagnosis of Hashimoto Encephalopathy (HE) is generally considered in patients with a wide range of neurological symptoms, accompanied by normal or nonspecific findings on magnetic resonance imaging and CSF, normal thyroid function or mild hypothyroidism, increased serum levels of thyroid peroxidase antibodies, and clinical response to steroids. Case report: We attended a 76-year-old patient, brought by lowering the level of consciousness 3 days ago, insidiously. Neurological exam: did not obey commands, and only said incomprehensible sounds. Myoclonus in upper limbs and random multidirectional movements of the eyes with horizontal nystagmus, rapid phase to the left. Cranial tomography had only signs of microangiopathy. Electroencephalogram: diffuse slow waves, with no signs of status epilepticus. Laboratory tests: there were increased protein in CSF (107mg/dl) and Anti-TPO serum (>1000 U/ml) levels, without other specific alterations. After these results, therapy with Methylprednisolone 1g/day for 5 days, and Levothyroxine, were chosen. There was a gradual improvement in the neurological condition from the 3rd day of treatment. Conclusion: immediate recognition of Hashimoto encephalopathy is important. Although the pathogenesis is unknown, the disorder is treatable. This entity should always be remembered for the proper direction of therapeutic approaches, thus enabling better outcomes to the patient.
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Da Nóbrega, Citânia Cordeiro. "TIREOIDITE DE HASHIMOTO: ASPECTOS IMUNOLÓGICOS E PATOGÊNICOS." In I Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1011.

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Introdução: A tireoide é uma glândula responsável pela produção e regulação hormonal, essenciais para o crescimento, reprodução e regulação do metabolismo. As doenças autoimunes que acometem essa glândula constituem 30%, destacando-se a Tireoidite de Hashimoto (TH). A TH é uma doença caracterizada pela presença de autoanticorpos que destroem os tecidos tireoidianos. A suscetibilidade do desenvolvimento da TH está associado a fatores genéticos e ambientais, porém sua patogênese não é totalmente esclarecida. Objetivo: Analisar a cerca da imunopatogênese da TH por meio das publicações científicas. Método: Trata-se de uma revisão bibliográfica, realizada no mês de março nas bases de dados PubMed, LILACS e SciELO, por meio da combinação dos descritores e seus correspondentes em inglês, utilizando o operador booleando AND. Os critérios de inclusão adotados foram: abordar a temática da pesquisa, texto completo disponível online, recorte temporal dos últimos cinco anos. Como critérios de exclusão: publicação que não fossem artigos. Resultado: Estudos mostraram que, tanto fatores ambientais quanto genéticos têm papel importante na expressão da doença. A TH caracteriza-se por vários graus de disfunção tireoidiana, anticorpos circulantes - antitireoglobulina e antiperoxidase tireoidiana - contra antígenos tireoidianos e infiltração da glândula por células mononucleares e fibrose. Os mecanismo de iniciação e progressão da destruição celular na TH são: alterações decorrentes de um processo infeccioso; participação ativa das células tireoidianas na apresentação de antígenos; atuação da imunidade humoral através de células B e produção de auto-anticorpos que além de induzirem citotoxicidade complemento mediada contra as células tireoidianas, participam da destruição tecidual via citotoxicidade anticorpo dependente; a imunidade celular via células T help e citotóxicas; e os mecanismos de apoptose celular. Conclusão: O estudo evidenciou que a patogênese da TH ainda não está bem esclarecida, porém o tireócito parece ter uma participação mais ampla do que o previsto anteriormente. Os níveis aumentados de linfócitos Th17 patogênicos e células Th22 no sangue periférico de pacientes com TH apresentam correlações significativas entre os níveis dessas células, a atividade da doença, a duração da doença e com o grau de hipoecogenicidade ultrassonográfica da tireoide. No entanto, observa-se uma necessidade de investigação mais profunda acerca dessa temática.
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Buyuksireci, D. E., D. Tecer, B. Bolayir, M. E. E. Yon, M. Akturk, and F. Gogus. "FRI0564 Ultrasonographic evaluation of shoulder tendons in patients with hashimoto’s disease." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.4807.

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Bokulić, Ema, and Nikola Šoštar. "Ideomotor apraxia and dysphasia as a clinical manifestation of Hashimoto’s encephalopathy - case report." In NEURI 2015, 5th Student Congress of Neuroscience. Gyrus JournalStudent Society for Neuroscience, School of Medicine, University of Zagreb, 2015. http://dx.doi.org/10.17486/gyr.3.2206.

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Jordan, B., î. Uer, T. Buchholz, A. Spens, and S. Zierz. "Muskuläre Beteiligung und Fatigue bei Patienten mit Hashimoto Thyreoiditis." In 24. Kongress des Medizinisch-Wissenschaftlichen Beirates der Deutschen Gesellschaft für Muskelkranke (DGM) e.V. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1685031.

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Pessoa, Ingrid Lacerda, Rafaela Oliveira Tavares, Andressa Clemente Mesquita, João Pedro de F. J. F. de Mendonça, Maryana Beltrão de Carvalho, Raquel Fontana, Pedro Teles de Mendonça Neto, and Mariana Aparecida Brunossi Moura Proença. "DERRAME PERICÁRDICO COMO PRIMEIRA MANIFESTAÇÃO DA TIREOIDITE DE HASHIMOTO." In 4º Congresso Internacional Sabará de Saúde Infantil. São Paulo: Editora Blucher, 2020. http://dx.doi.org/10.5151/cissi2020-25.

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Koshiishi, Masato, Hitoshi Seto, Shigeaki Tanaka, and Ryoji Obata. "Applicability of Formula of the Irradiation Assisted Stress Corrosion Cracking Rates for Neutron-Irradiated Type 316L Stainless Steels to Various Types of Stainless Steels Under Boiling Water Reactor Condition." In ASME 2020 Pressure Vessels & Piping Conference. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/pvp2020-21332.

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Abstract The Hashimoto-Koshiishi model is the predictive mechanistic model for crack growth rates (CGRs) of stress corrosion cracking (SCC). The simplified formula of the Hashimoto-Koshiishi model was developed for the calculation of CGRs of irradiated Type 316L stainless steels (SSs). This study evaluated the applicability of the developed formula to various types of stainless steels by analyzing experimental data of CGRs. The developed formula was determined to be applicable for Type 304L SSs and 316NG SSs as well as Type 316L SSs. On the other hand, the formula was unsuitable for irradiated Type 304 SSs because the mechanical properties and the Cr concentrations at the grain boundaries of Type 304 SSs were different from these quantities for Type 316L SSs.
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Reports on the topic "Hashimoto's"

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Wen, Yi, Changda Li, Chunxue Zang, Chanyuan Zhou, and Tianshu Gao. A protocol for systematic review and bayesian network meta-analysis of Integrated traditional Chinese and Western medicine therapies for Hashimoto Thyroiditis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0106.

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