Relevant bibliographies by topics / Hashimoto thyroiditis

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Hashimoto thyroiditis'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 June 2025

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Journal articles on the topic "Hashimoto thyroiditis"

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Karki, Prakriti, Parikshit Prasai, Vivek Acharya Chetri, Arun Gautam, and Robin Maskey. "Autoimmune Hemolytic Anemia with Autoimmune Hypothyroidism: A Case Report." Journal of Nepal Medical Association 61, no. 263 (2023): 614–16. http://dx.doi.org/10.31729/jnma.8217.

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Autoimmune hemolytic anaemia is a relatively rare disorder caused by autoantibodies directed against self-red blood cells. Though autoimmune thyroid disease is associated with other autoimmune diseases, only a few cases of Hashimoto's thyroiditis with autoimmune hemolytic anaemia have been reported. We present a case of a 22-year-old woman, a known case of Hashimoto’s thyroiditis whose serum demonstrated antibodies against red blood cells. Blood investigations were done which showed findings suggestive of hemolytic anemia. She was managed with blood transfusion, thyroxine and steroids.Our study may guide physicians toward possible hemolytic anaemia while treating Hashimoto thyroiditis.
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Ahmed, Rania, Safa Al-Shaikh, and Mohammed Akhtar. "Hashimoto Thyroiditis." Advances In Anatomic Pathology 19, no. 3 (2012): 181–86. http://dx.doi.org/10.1097/pap.0b013e3182534868.

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Lichtenstein, A. O., and A. A. Agafonov. "Hashimoto type thyroiditis." Kazan medical journal 43, no. 5 (2021): 55–56. http://dx.doi.org/10.17816/kazmj87888.

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Thyroiditis (struma limfomatosa - Hashimoto's disease) is a two-sided diffuse dense goiter, intimately fused with the trachea. Fusion with other organs and tissues is not observed. At the same time, the thyroid gland is often slightly enlarged and can cause compression of the trachea, which leads to hoarseness. The disease usually affects older women over 40 years of age (Babcock). On the cut, the tissue of the thyroid gland is dense, whitish, with significant lymphoid infiltration, proliferation of connective tissue, atrophy of the glandular tissue and degeneration of the epithelium. Struma Hashimoto was considered a disease of the Anglo-Saxon countries, where about 200 observations are described. However, in recent years, the domestic literature has been significantly enriched with reports of chronic lymphoid thyroiditis.
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Kasprzak, Amelia, Aleksandra Mańkowska, Monika Truchta, et al. "Vitamin D supplementation in patients with Hashimoto’s disease." Journal of Education, Health and Sport 53 (January 18, 2024): 125–36. http://dx.doi.org/10.12775/jehs.2024.53.009.

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Introduction : Hashimoto’s thyroiditis (HT) is the most common autoimmune disease and the leading cause of hypothyroidism in the world. The condition results in damage to the thyroid gland, brought about by the infiltration of lymphocytes. For the majority of individuals with Hashimoto's thyroiditis, a lifelong requirement for levothyroxine substitution is required. The potential contribution of diet and supplements to the management of HT is frequently overlooked. Low vitamin D levels are said to play a significant role in occurrence and severity of autoimmune thyroiditis. Currently, there is a continuing discussion regarding the optimal plasma concentration of 25-hydroxyvitamin D necessary for preventing or treating autoimmune diseases.
 
 Aim of the study : The purpose of this literature review is to evaluate the influence of vitamin D supplementation on the course of Hashimoto’s thyroiditis in the light of most up to date research.
 
 Materials and methods. This article is a review of publications obtained from the PubMed database, published between 2017-2023, based on the keywords "Hashimoto thyroiditis" "vitamin D" and "autoimmune thyroid disease".
 
 Conclusions. The correlation of vitamin D supplementation and Hashimoto’s disease still remains unclear due to conflicting results from numerous studies. Further research is necessary to accurately determine the effect of vitamin D supplementation on Hashimoto’s thyroiditis.
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Thomsen, Hauke, Xinjun Li, Kristina Sundquist, Jan Sundquist, Asta Försti, and Kari Hemminki. "Familial risks between Graves disease and Hashimoto thyroiditis and other autoimmune diseases in the population of Sweden." Journal of translational autoimmunity 3, no. 1 (2020): 1–5. https://doi.org/10.1016/j.jtauto.2020.100058.

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  Genetic and family studies have indicated that Graves disease and Hashimoto thyroiditis have a heritable component which appears to be shared to some extend also with some other autoimmune diseases (AIDs). In the present nation-wide study we describe familial risk for Graves disease and Hashimoto thyroiditis identified from the Swedish Hospital Discharge Register (years 1964 through 2012) and the Outpatient Register (2001 through 2012). Family relationships were obtained from the Multigeneration Register and cancers from the Cancer Registry. Familial standardized incidence ratios (SIRs) were calculated for 29,005 offspring with Graves disease and for 25,607 offspring with Hashimoto thyroiditis depending on any of 43 AIDs in parents or siblings. The concordant familial risks for Graves disease and Hashimoto thyroiditis were 3.85 and 4.75, higher for men than for women. The familial risks were very high (11.35, Graves and 22.06, Hashimoto) when both a parent and a sibling were affected. Spousal familial risks were higher for Hashimoto thyroiditis (1.98/1.93) than for Graves disease (1.48/1.50). For Graves disease, 24 discordant AIDs showed a significant association; for Hashimoto thyroiditis, 20 discordant associations were significant. All significant discordant associations were positive for the two thyroid AIDs, with the exception of Hashimoto thyroiditis with Reiter disease. Overall 8 associations were significant only for Graves disease and 6 Hashimoto thyroiditis. The overall high concordant familial risks for Graves disease and Hashimoto thyroiditis suggest a strong genetic contribution to the familial risk. Significant familial associations among more than half of the 43 AIDs attest to the extensive polyautoimmunity among thyroid AIDs.
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Pakdaman, Michael Navid, Michael P. Hier, Martin J. Black, Michael Tamilia, and Richard J. Payne. "Micropapillary Thyroid Carcinoma and Hashimoto's Thyroiditis." Otolaryngology–Head and Neck Surgery 139, no. 2_suppl (2008): P40. http://dx.doi.org/10.1016/j.otohns.2008.05.132.

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Objective An increase in papillary thyroid carcinoma in patients with Hashimoto's thyroiditis has been described. We have yet to find literature discussing a relationship with micropapillary thyroid carcinoma(MPC). Our objective is (1) to assess the relationship between Papillary Microcarcinoma and Hashimoto's Thyroiditis in thyroids without malignant disease, (2) compare effect of the presence of Hashimoto's on prognostic factors in patients. Methods Pathology reports were reviewed for consecutive patients undergoing total thyroidectomy over a 6-year period from 2002 to 2007. Patients with benign pathologies were included. Patients with MPC and no other malignancy were also included. This lead to a cohort of 314 patients. Statistical significance was calculated using Fisher's exact test and a chi-squared analysis. Results Out of the 314 patients, 98 were positive for Hashimoto's thyroiditis on biopsy of the removed thyroid. 58% of the patients with Hashimoto's thyroiditis had an incidental MPC, as compared to 42% of patients where Hashimoto's was not found. Additionally, in Hashimoto patients, 61% of the MPCs were multifocal versus 43% multifocal in the non-Hashimoto group (p-value <0.05 in each comparison). Bilaterality of MPC was also increased in patients with Hashimoto's (22% vs 39%). Conclusions Patients with Hashimoto's thyroiditis in this study had an incidence of MPC of 58%, which is significantly greater than the 42% in those without. Hashimoto's was also associated with an increased risk of bilaterality and multifocality. Studies are needed to further corroborate these findings.
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Thompson, Lester D. R. "Chronic Lymphocytic Thyroiditis (Hashimoto Thyroiditis)." Ear, Nose & Throat Journal 93, no. 4-5 (2014): 152–53. http://dx.doi.org/10.1177/0145561314093004-508.

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Hapsari, Siti Nurul, and Sidarti Soehita. "HASHIMOTO’S THYROIDITIS HYPERTHYROID STAGE." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 26, no. 1 (2019): 123. http://dx.doi.org/10.24293/ijcpml.v26i1.1423.

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Preliminary: Hashimoto thyroiditis (chronic autoimmune thyroiditis) is the most common cause of hypothyroidism in iodine- sufficient areas of the world, but can sometimes show hyperthyroidism. Case: A 39-year-old female was referred due to shortness of breath and tremor, four hours before hospitalization. There was nausea, chest pain, cold chills and palpitation. She was diagnosed with Hashimoto’s thyroiditis and routinely received tyrosol, propanolol and dexamethasone. Physical examination: cervical mass, afebrile, blood pressure 130/70 mmHg, pulse rate 110 x/minute and respiratory rate 20 x /minute. Laboratory examinations showed WBC 7.53 x 109/L, Hb 11.0 g/dL and platelet count 168 x 109/L. Chest X-Ray: negative for infiltrates. Several laboratory tests were performed, abnormal results were as follows: FT4 2.96 ng/dL (increased), TSH 0.003 uIU/mL (decreased), anti-TPO (antithyroid microsomal antibody) 306 IU/ml (increased), Ig E 213.6 IU/mL (increased). Peripheral blood smear, coagulation test, serum electrolytes, liver function tests, renal function tests, urinalysis, CEA and Ca 125 were within normal limits. A thyroid ultrasound resulted in a benign lesion. Fine Needle Aspiration Biopsy concluded in lymphocytic Hashimoto’s thyroiditis. Echocardiography showed hyperthyroid heart disease. Discussion: Due to an increase in anti-TPO and FT4, a decrease in TSH and lymphocytic thyroiditis from FNAB, this patient was diagnosed with Hashimoto’s Thyroiditis Hyperthyroid Stage. Conclusion: Thyroid function tests and thyroid antibody tests must be monitored to decide whether it is hyperthyroid or hypothyroid stage of Hashimoto thyroiditis.
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Sobolevskaia, Polina A., Boris V. Andreev, Anton N. Gvozdetckii, et al. "The association of neuropsychiatric disorders and endocrine parameters in hashimoto thyroiditis." Pediatrician (St. Petersburg) 11, no. 4 (2020): 55–68. http://dx.doi.org/10.17816/ped11455-68.

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Hashimoto thyroiditis is the most common thyroid disease. This form of pathology has a diverse clinical picture, including neuropsychiatric disorders. There are frequent cases of comorbidity of autoimmune thyroiditis and psychiatric forms of pathology, along with such a nosological entity as Hashimotos encephalopathy (aka: Steroid-responsive encephalopathy of autoimmune thyroiditis), characterized by an increased level of antithyroid autoantibodies and various mental disorders, with still unclear pathogenesis. The question arises, how to regard patients with psychiatric disorders and Hashimoto thyroiditis either as patients having autoimmune thyroiditis, comorbid with psychiatric forms of pathology, or as patients with Hashimotos encephalopathy? We studied groups of patients with autoimmune thyroiditis free from any psychiatric disorders, autoimmune thyroiditis comorbid with psychiatric forms of pathology, and a group of healthy donors similar as regards to their age and sex. We also studied medical history, clinical manifestations of the disease, instrumental data and the serum levels of thyrotropin, thyroid hormones, various antithyroid autoantibodies, and prolactin. We analyzed the correlation of laboratory and instrumental parameters and clinical data in all groups of patients. Therewas a significant relationship (p 0,05) between various psychiatric symptoms and a decreased level of free thyroxine, an increased level of thyroid stimulating hormone (TSH), an increased level of prolactin and an increased volume of a thyroid gland. Asignificant relationship (p 0,05) was also found between various symptoms of hypothyroidism and a decreased level of free triiodothyronine (FT3), an increased level of antibodies to thyroglobulin (anti-TG Ab), and an increased level of antibodies to thyroid peroxidase (anti-TPO Ab).
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Moghimi, Mansour, Fateme Sadat Shamsadini Ezabadi, Seyed Mohammadreza Mortazavi Zadeh, and Saead Hossein Khalilzade. "Clinico-pathological Features and Survival Time of Papillary Thyroid Carcinoma in Patients With and Without Hashimoto’s Thyroiditis: A Cross-sectional Study." Crescent Journal of Medical and Biological Sciences 10, no. 4 (2022): 188–92. http://dx.doi.org/10.34172/cjmb.2023.45.

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Objectives: Researchers have reported different results regarding the association between Hashimoto’s disease and papillary thyroid carcinoma (PTC). Some believe that the coexistence of these diseases can lead to fewer tumor invasion and recurrence rates. This study evaluated the clinico-pathological features and survival time of PTC in patients with and without Hashimoto’s thyroiditis. Materials and Methods: In this cross-sectional study, medical records of 251 participants who underwent total or subtotal thyroidectomy due to PTC from 2012 to 2019 were reviewed. The clinico-pathological features of participants, such as age, gender, tumor stage, tumor size, lymph node involvement, metastasis, capsular invasion, single or multi-focal tumor status, and survival time were recorded from their medical records and pathology report and compared in two groups with and without Hashimoto’s thyroiditis. Results: From 251 participants, 92 (36.6%) had Hashimoto’s thyroiditis, whereas 159 (63.4%) did not show any signs of this disease. Fifteen participants in the Hashimoto group and 46 in the non-Hashimoto group had a recurrence. Although there were no significant differences between the two groups in the term of recurrence rate (P = 0.08), the mean survival time was significantly difference between the two groups (69.03 and 58.78, respectively; P = 0.038) Conclusions: Results of the study revealed that Hashimoto’s thyroiditis could increase the survival time of patients with PTC.
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Dissertations / Theses on the topic "Hashimoto thyroiditis"

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陳結清 and Kit-ching Amanda Chan. "Detection of RET/PTC translocation in hashimoto thyroiditis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40738413.

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Chan, Kit-ching Amanda. "Detection of RET/PTC translocation in hashimoto thyroiditis." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B40738413.

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Quintino-Moro, Alessandra 1973. "Infertilidade em mulheres com Doença de Graves e Tireoidite de Hashimoto : Infertility among women with Graves's disease or Hashimoto's thyroiditis." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308429.

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Orientadores: Arlete Maria dos Santos Fernandes, Denise Engelbrecht Zantut Wittmann<br>Dissertação (Mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2019-01-09T11:51:06Z (GMT). No. of bitstreams: 1 Moro_AlessandraQuintino_M.pdf: 1160579 bytes, checksum: da4d51faeb6041f68c9c786482aef230 (MD5) Previous issue date: 2013<br>Resumo: A disfunção autoimune, representada pelo hipertireoidismo da Doença de Graves (DG) e pelo hipotireoidismo da Tireoidite de Hashimoto (TH), ocorre de forma mais frequente em mulheres e interfere no mecanismo da reprodução, em especial no processo ovulatório. Existe o senso comum de que as disfunções tireoideanas diminuem o potencial de fertilidade das mulheres; entretanto, não existem estudos sobre a prevalência da infertilidade nesses grupos. Objetivos: Determinar a prevalência de infertilidade em mulheres com TH e DG e as possíveis variáveis associadas. Sujeitos e métodos: Foi um estudo de corte transversal. Mulheres com TH (n=66) e idade entre 18 e 60 anos e DG (n=193) com idade entre 18 e 50 anos, em seguimento no Ambulatório de Tireoidopatias do HC/UNICAMP, no período de agosto de 2010 a dezembro de 2011, foram entrevistadas com respeito às variáveis ginecoobstétricas: história de períodos de infertilidade, alterações do ciclo menstrual, história obstétrica e antecedentes familiares. Infertilidade foi definida como ausência de gravidez após período de exposição ?12 meses. Após a entrevista, seus prontuários foram revisados para determinar as características da doença: idade ao diagnóstico, tempo do diagnóstico, antecedentes de outras doenças autoimunes associadas, exames laboratoriais e de imagem. O estudo obteve aprovação do Comitê de Ética em Pesquisa da FCM/UNICAMP. Os critérios de inclusão foram: ao menos um ano de convívio com parceiro masculino e concordância em participar da pesquisa. Os dados foram anotados em ficha de coleta e posteriormente digitados em banco de dados elaborado para o estudo. Após a consistência do banco, foi realizada análise descritiva das variáveis gineco-obstétricas e características da doença, e foram aplicados os testes quiquadrado ou exato de Fisher, teste de Mann-Whitney e teste de Wilcoxon. Posteriormente, foi realizada a análise univariada com o cálculo de Odds Ratio bruto e respectivo intervalo de confiança (IC) de 95%, seguida pela regressão logística múltipla incluindo todas as variáveis, buscando aquelas significativamente associadas à infertilidade. O grau de significância estatística foi de 5%. Resultados: A prevalência de infertilidade foi de 52,3% e 47,0% nas mulheres, respectivamente com DG e TH. Na DG, as perdas gestacionais tiveram prevalência de 18,8% e 21,7% em mulheres com e sem infertilidade, enquanto na TH as perdas gestacionais ocorreram em 22,6% e 20,6% nos mesmos grupos. A média do número de gestações foi menor após o diagnóstico de DG e TH nas mulheres com idade ?35 anos. No mesmo grupo de idade, a média do número de gestações antes do diagnóstico foi de 1,68 (DP ±1,41) para DG e 1,48 (DP ±1,31) para TH. As alterações de ciclo menstrual na DG ocorreram em 47,5% e 35,9% das mulheres, respectivamente com e sem infertilidade, e na TH as taxas foram de 64,5% e 34,3% nos mesmos grupos. A única variável associada à infertilidade na TH foi o tempo de doença menor que seis anos. Não houve variável associada nas mulheres com DG. Conclusões: a prevalência de infertilidade foi alta e, no grupo de mulheres com idade ?35 anos, houve diminuição na média de gestações, mostrando o comprometimento da fertilidade das mulheres com DG e TH. Mulheres com TH com menos tempo de doença foram as mais afetadas pela infertilidade<br>Abstract: The autoimmune dysfunction, hyperthyroidism represented by the Graves disease (GD) and by hypothyroidism of Hashimoto's thyroiditis (TH), occurs more often in women and interferes in the mechanism of reproduction, especially in the ovulatory process. There is common sense that thyroid dysfunction decreases the fertility potential of women, however, there are no studies on the prevalence of infertility in these groups. Objectives: To determine the prevalence of infertility in women with TH and DG and possible associated variables. Subjects and Methods: This was a cross-sectional study. Women with TH (n = 66) aged between 18 and 60 years and DG (n = 193) aged between 18 and 50 years was followed at the Endocrinology Division, Departament of Clinical Medicine, School of Medical Sciences, University of Campinas (UNICAMP), from August 2010 to December 2011 were interviewed with respect to gynecological and obstetric variables: history of periods of infertility, menstrual abnormalities, obstetric history and family history. Infertility was defined as 12 months of unprotected sexual intercourse without conception. After the interview, their medical records were reviewed to determine the characteristics of the disease: age at diagnosis, time since diagnosis, history of other autoimmune diseases associated, laboratory tests and imaging tests. The study was approved by the Research Ethics Committee of the FCM/UNICAMP. Inclusion criteria were: at least a year of living with a male partner, and agreed to participate. Data were recorded on collection and subsequently entered into a Summary - xvii database designed for the study. After the database consistency, descriptive analysis was performed gynecological and obstetric variables and disease characteristics, and applied the chi-square or Fisher's exact test, Mann-Whitney and Wilcoxon test. Subsequently, univariate analysis was performed to calculate crude odds ratios and confidence intervals (CI) of 95%, followed by multiple logistic regressions including all variables significantly associated with those seeking infertility. The level of statistical significance was 5%. Results: The prevalence of infertility was 52.3% and 47.0% in women, with DG and TH respectively. In DG, the miscarriages had a prevalence of 18.8% and 21.7% in women with and without infertility, while in the TH pregnancy loss occurred in 22.6% and 20.6% in the same groups. The mean number of pregnancies was lower after the diagnosis of GD and HT in women aged ? 35 years. In the same age group, the mean number of pregnancies before diagnosis was 1.68 (SD ± 1.41) for DG and 1.48 (SD ± 1.31) for TH. Changes in the menstrual cycle in DG occurred in 47.5% and 35.9% of women, respectively with and without infertility, and TH rates were 64.5% and 34.3% in the same groups. The only variable associated with infertility in TH disease duration was less than six years. There was no associated variable in women with GD. Conclusions: The prevalence of infertility was high and, in the group of women aged ? 35 years, there was a decrease in average pregnancies, showing impairment of fertility in women with GD and HT. Women with TH with shorter disease were most affected by infertility<br>Mestrado<br>Fisiopatologia Ginecológica<br>Mestra em Ciências da Saúde
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Noal, Cristiano Bicca. "ATIVIDADE DE ENZIMAS QUE DEGRADAM NUCLEOTÍDEOS E NUCLEOSÍDEO DE ADENINA EM PLAQUETAS DE PACIENTES COM TIREOIDITE DE HASHIMOTO EM TRATAMENTO COM LEVOTIROXINA SÓDICA." Universidade Federal de Santa Maria, 2012. http://repositorio.ufsm.br/handle/1/5976.

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Hashimoto Thyroiditis (HT) or chronic lymphocytic thyroiditis is an autoimmune disease that causes the destruction of the thyroid gland by inflammatory infiltrates and consequently loss of function. This disease is spread worldwide and predominantly affects females. The consequences of alterations in its activity range from cretinism to the vascular changes. It is known that the enzymes ectonucleoside diphosphohydrolase triphosphate (E-NTPDase, EC 3.6.1.5, CD39), ecto-5'nucleotidase (EC 3.1.3.5, CD73) and adenosine deaminase (ADA, EC 3.5.4.4) are involved in regulating the immune response and thrombotic events, since they regulate extracellular levels of adenine nucleotides and nucleoside. Since HT patients have an autoimmune response and present microvascular changes, the objective of this study was to evaluate the influence of purinergic signaling in the regulation of microvascular dysfunction triggered by the disease determining the activity of the ectoenzymes involved in the metabolism of ATP in platelets from patients with HT in treatment with levothyroxine. Samples were collected from patients with HT treated with levothyroxine and from a control group. In this study we determined the activity of the enzymes E-NTPDase, ecto-5'-nucleotidase and E-ADA; detected the expression of the enzyme in platelets and E-NTPDase; and measured hormone levels of TSH and fT4 in patients with HT treated with levothyroxine and control group. Results obtained in the enzyme activity showed that patients with HT in hormone replacement with levothyroxine presented no significant changes when compared with the control group. In conclusion, levothyroxine used in patients with HT may reverse the effects of hypothyroidism when used regularly in these patients, while maintaining the enzyme activity in basal levels.<br>A Tireoidite de Hashimoto (TH) ou tireoidite linfocítica crônica é uma doença autoimune que causa a destruição da glândula tireóide por meio de infiltrados inflamatórios e consequente perda da função. Esta patologia encontra-se difundida mundialmente e acomete prevalentemente o sexo feminino. As consequências das alterações de sua atividade vão desde cretinismo à alterações vasculares. Sabe-se que as enzimas ectonucleosídeo trifosfato difosfo-hidrolase (E-NTPDase; E.C. 3.6.1.5; CD39), ecto-5 nucleotidase (E.C.3.1.3.5; CD73) e adenosina desaminase (ADA; E.C.3.5.4.4) participam tanto da regulação da resposta imune quanto de eventos trombóticos, uma vez que regulam os níveis extracelulares dos nucleotídeos e nucleosídeo da adenina. Visto que, pacientes portadores da TH possuem uma resposta autoimune e também apresentam alterações microvasculares, o objetivo deste estudo foi avaliar a influência da sinalização purinérgica na regulação das disfunções microvasculares desencadeadas pela doença, através da determinação da atividade de ectoenzimas envolvidas no metabolismo do ATP em plaquetas de pacientes com TH em tratamento com levotiroxina. Foram coletadas amostras de pacientes com TH em tratamento com levotiroxina e um grupo controle. Neste estudo determinamos a atividade das enzimas E-NTPDase, ecto-5 -nucleotidase e E-ADA, verificamos a expressão da enzima E-NTPDase em plaquetas e dosamos os níveis hormonais de TSH e fT4 em pacientes com TH em tratamento com levotiroxina, bem como do grupo controle. Os resultados obtidos na atividade das enzimas e na concentração dos nucleotídeos e nucleosídeo da adenina, não demonstraram alterações significativas quando comparamos os pacientes com TH em reposição hormonal com levotiroxina, com o grupo controle. Em conclusão, sugere-se que a levotiroxina usada em pacientes com TH pode reverter os efeitos do hipotireoidismo quando usada regularmente por estes pacientes, mantendo as atividades das enzimas em níveis basais.
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Botelho, Ilka Mara Borges 1979. "Prevalência de insuficiência de vitamina D em pacientes com tireoidite de Hashimoto e sua relação com autoimunidade tireoideana = Prevalence of vitamin D insufficiency in patients with Hashimoto's thyroiditis and its relationship with thyroid autoimmunity." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308788.

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Orientadores: Denise Engelbrecht Zantut Wittmann, Sarah Monte Alegre<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-28T02:28:01Z (GMT). No. of bitstreams: 1 Botelho_IlkaMaraBorges_M.pdf: 1340793 bytes, checksum: 6ef5a41b67a7eddca52fc55dea2f96d4 (MD5) Previous issue date: 2014<br>Resumo: Introdução: Vitamina D tem sido apontada como importante regulador da resposta imune. Estudos tem demonstrado haver relação entre insuficiência de vitamina D e presença de doenças autoimunes como Tireoidite de Hashimoto (TH). É possível que o processo autoimune na TH seja inibido em diferentes estágios pela vitamina D em sua forma ativa. Nossos objetivos foram estudar a prevalência de insuficiência de vitamina D e a relação de suas concentrações séricas com marcadores de função e autoimunidade tireoideana. Material e Métodos: Amostras de sangue foram coletadas de 54 pacientes com TH e 54 indivíduos saudáveis sem diagnóstico de TH com idade entre 18 e 75 anos. Foram realizadas dosagens séricas de vitamina D (25OHD), TSH, T4 livre, cálcio, fósforo, paratormônio (PTH), anticorpos anti-tireoperoxidase (AcTPO), anti-tireoglobulina (AcTG) e anti-receptor de TSH (TRAb). Volume tireoideano foi estimado por ultrassonografia. Foram coletados dados demográficos, de peso, altura, índice de massa corporal (IMC) e tempo de diagnóstico. Pacientes e indivíduos do grupo de controle foram pareados por idade e sexo. O nível de significância estatística adotado foi 5%. Resultados: Prevalência de insuficiência de vitamina D foi encontrada em 68.5% dos pacientes e em 38.9% dos indivíduos do grupo de controle (p =0,002). Houve uma correlação positiva entre níveis de AcTPO e maior volumetireoideano nos pacientes (r = 0,319; p= 0.019). Não houve correlação entre concentração de vitamina D, TSH, T4livre,TRAb, AcTGe volume tireoideano. Conclusões: Demonstramosmaior prevalência deinsuficiência de vitamina Dem pacientescom tireoidite de Hashimotoem relaçãoa indivíduos de um grupo controlesaudável, não havendo correlaçãocom o estado hormnal tireoideanooumarcadores séricos deautoimunidadeda tireóide.Por sua vez, maior volume da tireóidese associou a maior grau de infiltração inflamatóriaautoimune,refletido pelacorrelaçãocom maiores concentrações AcTPO<br>Abstract: Introduction: Vitamin D has been pointed out as an important immune response regulator. Studies have shown a relationship between vitamin D insufficiency and the presence of autoimmune diseases such as Hashimoto's Thyroiditis (HT). It's possible that the autoimmune process in HT is inhibited in its different stages by vitamin D on its active form .Our aims were to study the prevalence of vitamin D insufficiency and relationship of the serum concentrations with thyroid function and autoimmunity markers. Material and Methods: Blood samples were collected from 54 patients with HT and 54 healthy individuals without a diagnosis of HT, aged 18 to 75 years. We conducted serum 25OH vitamin D, TSH, free T4, calcium, phosphorus, PTH, TPOAb, TgAb and TRAb. Thyroid volume was estimated by ultrasound. Data on demographic, weight, height, body mass index and time since diagnosis were collected. Patients and control subjects were matched by sexand age. The significance level for statistical analysis was 5%. Results: Prevalence of vitamin D insufficiency was found in 68.5% of patients and in 38.9% of subjects in the control group (p= 0.002). There was a positive correlation between TPOAb and volume in patients (p= 0.019). There was no correlation between vitamin D concentration and thyroid volume, TRAb, TgAb, TSH or free T4. Conclusions: We demonstrated a higher prevalence of vitamin D insufficiency in patients with Hashimoto's thyroiditis compared to individuals of a healthy control group, no correlation with thyroid state hormonal or serum markers of thyroid autoimmunity. In turn, greater thyroid volume was associated with a higher degree of autoimmune inflammatory infiltration, reflected by the correlation with higher concentrations AcTPO<br>Mestrado<br>Clinica Medica<br>Mestra em Ciências
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Sheremet, M. I. "Surgical treatment of Hashimoto's thyroiditis." Thesis, БДМУ, 2021. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18639.

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Alami, Harchali Asmae. "Détection par immunephelemetrie sur supports microparticulaires d'autoanticorps anti-thyroïde de spécificité épisodique définie : mise au point de la méthode et applications." Nancy 1, 1994. http://www.theses.fr/1994NAN10286.

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Ce travail a pour objectif d'adapter à la détection et à la caractérisation d'autoanticorps, la technique d'immunonephelemetrie sur supports microparticulaires. Le but est d'affiner le pouvoir discriminant de cette technique jusqu'à l'identification de la spécificité épitopique des anticorps décelés. Un premier groupe d'applications porte sur la détection des autoanticorps anti-thyroglobuline humaine. Un test de détection de ces autoanticorps anti-domaine antigénique deux de la molécule de thyroglobuline, a été développé. Il est basé sur l'aptitude des autoanticorps à inhiber les systèmes agglutinants thyroglobuline-anticorps monoclonaux anti-thyroglobuline ont ainsi été détectés et quantifiés chez tous les patients atteints de thyroïdite d'Hashimoto et dans plus du trois-quarts des patients présentant une maladie de basedow non traitée. Les résultats obtenus se comparent favorablement à ceux de l'immunofluorescence indirecte et de l'hemagglutination passive. Le système mis au point peut aussi se prêter au dosage de la thyroglobuline sérique par inhibition. La détection des autoanticorps anti-thyroperoxydase humaine est calquée dans son principe sur le protocole mis au point pour les anticorps anti-thyroglobuline. Ce test confirme la polyclonalité des autoanticorps anti-thyroperoxydase et leur réactivité préférentielle envers les épitopes localisés sur les domaines antigéniques a et b de la molécule de thyroperoxydase, épitopes impliques dans les maladies thyroïdiennes auto-immunes. Ces mêmes spécificités sont observées chez les patients atteints de thyroïdite d'Hashimoto et de maladie de basedow. Les données immunonephelemetriques sont en accord avec les résultats des autres moyens d'investigation et avec les autres éléments des dossiers cliniques
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MAERENS, BEATRICE. "Vitiligo et thyroidite lymphocytaire juvenile." Dijon, 1994. http://www.theses.fr/1994DIJOM012.

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PATRIZI, MICHELA. "Ipotiroidismo subclinico: le ragioni per uno screening universale." Doctoral thesis, Università degli Studi di Roma "Tor Vergata", 2010. http://hdl.handle.net/2108/1225.

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Le malattie della tiroide sono, insieme al diabete mellito, le endocrinopatie più frequenti in gravidanza (0.2-0.5%) e nel puerperio (5-10%). L’incidenza delle disfunzioni tiroidee clinicamente evidenti nelle donne in gravidanza è del 1-2% ma le forme subcliniche sono probabilmente più comuni. La tiroidite di Hashimoto rappresenta, sicuramente, uno dei fattori eziopatogenetici più importanti di ipotiroidismo. Si parla di ipotiroidismo subclinico quando ad un TSH aumentato si associano valori normali di FT4. Nel primo studio,condotto tra il 2006 e i 2008, sono state esaminate 40 donne con storia di aborti ricorrenti. Tutte le pazienti sono state sottoposte a screening tiroideo. 12 pazienti con valori di TSH >2.5 mUI/ml sono state sottoposte a terapia con L- thyroxina. Nel gruppo che ha ricevuto il trattamento sostitutivo il 67% ha presentato una gravidanza a termine e l’8,3% gravidanza pre-termine (34 settimane). Nel secondo studio sono state sottoposte a screening per valutare la prevalenza di ipotiroidismo in una piccola popolazione 50 donne gravide, nate a Civita Castellana, vicino a Viterbo, zona con alta incidenza di tiroidine autoimmune (S. di Hashimoto). Di queste, soltanto 6 (12%) hanno mostrato livelli di TSH superiori a 2.5 mUI/ml. Tali gravidanze, in donne sottoposte a terapia, sono giunte a termine. Le disfunzioni tiroidee sono spesso associate a complicanze durante la gravidanza come ipertensione, parto prematuro, basso peso alla nascita, distacco di placenta e morte fetale. La relazione tra ipotiroidismo subclinico ed esito della gravidanza non sono stati bene studiati. I programmi di screening possono individuare i soggetti con ipotiroidismo subclinico o quali dei rischi che si sviluppino patologie tiroidee, ma i costi, i rischi e i benefici dello screening dei disordini tiroidei che compaiono prima o durante la gravidanza devono essere considerati. Lo screening delle patologie tiroidee in gravidanza è raccomandato soltanto nelle donne considerate ad alto rischio sulla base della storia personale o familiare di patologie tiroidee o storia di altre patologie autoimmunitarie L’american College of Obstetrician and Gynecologist (ACOG) ha espresso parere negativo nei confronti dello screening di routine per la malattia tiroidea subclinica, poiché non c’è nessuna evidenza che l’individuazione ed il trattamento delle donne in stato di gravidanza con ipotiroidismo subclinico, possa migliorare gli outcome materni e fetali. La prevalenza di tiroiditi autoimmuni non è abbastanza elevata da giustificare lo screening su tutta la popolazione. Al contrario, in piccole popolazioni in cui la prevalenza delle tiroiditi autoimmuni è alta, dobbiamo proporre lo screening. Rilevanti studi hanno mostrato una associazione tra autoimmunità tiroidea nella gravidanza precoce e successivo aborto. In queste pazienti, lo screening nella fase precoce della gravidanza è probabilmente ciò che di meglio possiamo offrire.<br>Thyroid diseases are, together with diabetes, the most frequent endocrinal diseases in pregnancy (0.2-0.5%) and postpartum (5-10%). The incidence of overt thyroid dysfunction in pregnant women is around 1-2% but unrecognized subclinical forms of hypothyroidism are probably even more prevalent. Hashimoto’s thyroiditis seems to be the most important cause of hypothyroidism. Mild or subclinical hypothyroidism is characterized by normal serum free thyroxine (FT4) concentrations with elevated serum thyroid-stimulating hormon (TSH) concentrations. In the first study a sample of 40 women, whit history of recurrent miscarriages occurred between 2006 and 2008, was recruited. All women experienced thyroid screening. Twelve patients with TSH values at or above 2.5 mUI/ml were treated with thyroid replacement therapy. In the thyroid supplementation group, 8 out of 12 pregnancies (67%) ended in live birth, 1 out of 12 (8,33%) ended in pre-term live birth (34 weeks). In the second observation we undertook a thyroid screening study to evaluate the prevalence of subclinical thyroid disease in a small population of 50 pregnant patients who were born in Civita Castellana, near Viterbo,. These patients exhibited high incidence of chronic autoimmune (Hashimoto’s) thyroiditis. Of these, 6 (12%) showed a serum TSH level higher than 2.5 mUI/ml. These pregnancies treated with L-thyroxina resulted in live births. Even though subjects with unsuspected hypothyroidism or those at risk of developing thyroid disease can be detected by means of screening programs, costs, risks and benefits associated to screening before and during pregnancy must be considered at all. Screening for thyroid disfunction in pregnancy is highly recommended just in case women are considered ‘high risk patients’ due to personal or family history of thyroid disease as well as due to other autoimmune responses. Routinary screening has not been recommended by the American College of Obstetrician and Gynecologist (ACOG) because a clear relationship between prefetal treatment and successful birth outcome was not supported by unambiguous observations Clinical thyroid dysfunction has been associated with pregnancy complications such as hypertension, preterm birth, low birth weight, placental abruption, and fetal death. The relationship between subclinical hypothyroidism and pregnancy outcomes has not been investigated in a great detail. For example, the prevalence of autoimmune thyroiditis is usually not high enough to justify a full screening of the entire population; on the contrary when small populations with high prevalence of autoimmune thyroiditis are considered, a screening operation is fully recommended. Relevant studies have shown a connection between of thyroid autoimmunity in early pregnancy and subsequent miscarriage. In these patients screening in early pregnancy is most likely to be the best strategy to be accomplished.
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Sebastianes, Fernando Moreno. "Alterações da PET-FDG na avaliação pré-operatória de pacientes com nódulos tireoidianos e correlação com marcadores imuno-histoquímicos." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-22072011-124925/.

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INTRODUÇÃO: Cerca de 80% dos nódulos de tireóide com citologia indeterminada são benignos. É possível que a tomografia por emissão de pósitrons (PET) com 2-[18F]- fluoro-2-desoxi-D-glicose (FDG) ajude a identificar quais dessas lesões são malignas. A captação de FDG depende da expressão de GLUTs (transportadores de glicose transmembrana) e hexoquinases. Captação difusa de FDG no leito tireoidiano tem ainda sido associada à tireoidite autoimune crônica (TAC), embora haja evidências de que pacientes com parênquima tireoidiano aparentemente sadio possam ter essa alteração. OBJETIVOS: (1) Determinar a sensibilidade e especificidade da PET-FDG no diagnóstico pré-operatório de malignidade dos nódulos tireoidianos, especialmente daqueles com resultados citológicos indeterminados, e avaliar esse desempenho no subgrupo de pacientes com TAC. (2) Determinar a correlação entre o achado de captação difusa tireoidiana da FDG à PET com o diagnóstico histopatológico de TAC e compará-la às concentrações plasmáticas dos anticorpos antitireoidianos. (3) Determinar a correlação entre a expressão dos marcadores imuno-histoquímicos GLUT 1, GLUT 3, GLUT 12, hexoquinase 2 e hexoquinase 3 com o diagnóstico histopatológico dos nódulos e com a captação de FDG apresentada pelos mesmos. MÉTODOS: 56 pacientes com nódulo de tireóide (42 com citologia indeterminada, 10 compatíveis com carcinoma papilífero e 4 com citologia benigna) realizaram PET-FDG e foram submetidos à tireoidectomia. Os resultados da PET-FDG foram comparados com o diagnóstico histopatológico do nódulo, com o infiltrado linfocitário no parênquima tireoidiano, com o diagnóstico de TAC e com os resultados do estudo imuno-histoquímico de micromatriz tecidual de tecido correspondente ao nódulo tireoidiano puncionado e ao tecido tireoidiano não nodular. RESULTADOS: 1) Todos os 21 pacientes com câncer de tireóide (11 com citologia indeterminada) apresentaram captação focal de FDG na tireóide. 2) Captação focal de FDG correlacionou-se com maior risco de malignidade (p<0,001). 3) Dos 31 pacientes com nódulos benignos com citologia indeterminada, 12 não tinham captação focal de FDG (especificidade de 39%). 4) Captação difusa no leito tireoidiano à PET-FDG foi associada à presença de TAC no exame histopatológico (p=0,019). Porém, 5 pacientes, sem sinais de infiltrado linfocitário, apresentaram captação difusa à PET-FDG. 5) Imunoexpressão dos anticorpos contra GLUTs 1, 3 e 12 e hexoquinases 2 e 3 nas células epiteliais dos nódulos não esteve positivamente associada com captação de FDG e com malignidade. 6) Não houve associação entre captação difusa de FDG no leito tireoidiano e imunoexpressão desses marcadores no parênquima tireoidiano não nodular. CONCLUSÕES: 1) A PET-FDG tem alta sensibilidade para diagnóstico de lesões malignas de tireóide, com especificidade de 39% para nódulos com citologia indeterminada. 2) Captação difusa de FDG no leito tireoidiano está associada à presença de TAC, mas pode ocorrer em pacientes sem infiltrado linfocitário. 3) A imuno-histoquímica para os anticorpos contra GLUTs 1, 3 e 12 e hexoquinases 2 e 3 não contribui para a diferenciação de nódulos malignos dos benignos e não se associa com a captação de FDG pelos nódulos<br>INTRODUCTION: Around 80% of thyroid nodules with indeterminate cytological result are benign. Positron emission tomography (PET) with 2-[18F]-fluoro-2-deoxy- D-glucose (FDG) might help to identify malignant thyroid lesions. FDG uptake depends on GLUTs expression (transmembranous glucose transporters) and on hexokinases. Although diffuse FDG thyroid uptake has been associated with chronic autoimmune thyroiditis (CAT), there is some evidence that patients with apparently normal thyroid parenchyma can display this pattern of FDG uptake. OBJECTIVES: (1) To assess the sensitivity and specificity of FDG PET in identifying thyroid malignancy in the preoperative evaluation of thyroid nodules and evaluate these results in the subgroup of patients with indeterminate cytological results and in the subgroup with CAT. (2) To compare the finding of diffuse FDG thyroid uptake with the histopathologic diagnosis of CAT and with the serum levels of antithyroid antibodies. (3) To evaluate the immunoexpression by thyroid nodules of GLUT 1, GLUT 3, GLUT 12, hexokinase 2 and hexokinase 3 and to compare it with the histopathologic diagnosis of these nodules and with FDG uptake. METHODS: 56 patients with thyroid nodules (42 with indeterminate cytological result, 10 with papillary carcinoma and 4 with benign cytological result) underwent FDG PET and, subsequently, thyroidectomy. FDG PET results were compared with the histopathologic diagnosis of the nodule, lymphocytic infiltrate of thyroid parenchyma, CAT diagnosis and immunohistochemical results of tissue microarray of the tissue from the thyroid nodule and the normal thyroid parenchyma. RESULTS: 1) All the 21 patients with thyroid cancer (11 with indeterminate cytological result) had focal thyroid FDG uptake. 2) Focal FDG uptake was associated with malignancy (p<0.001). 3) From 31 patients with benign nodules whose cytological result was indeterminate, 12 did not display focal FDG uptake (specificity of 39%). 4) Diffuse FDG uptake in thyroid bed was associated with CAT in the histopathologic exam (p=0.019). However, 5 patients without lymphocytic infiltrate had diffuse FDG uptake. 5) Immunoexpression of GLUTs 1, 3 and 12 and hexokinases 2 and 3 by epithelial cells of thyroid nodules was not positively associated with FDG uptake and malignancy. 6) There was no association between diffuse FDG uptake in thyroid bed and immunoexpression of these markers in the normal thyroid tissue. CONCLUSIONS: 1) FDG PET has high sensitivity to the diagnosis of malignancy in thyroid bed, with a specificity of 39% for thyroid nodules with indeterminate cytological result. 2) Diffuse FDG uptake in the thyroid bed is associated with CAT, but can be found in patients without lymphocytic infiltrate. 3) Immunohystochemistry against GLUTs 1, 3 and 12 and hexokinases 2 and 3 does not add in the differentiation of malignant and benign thyroid nodules and is not associated with FDG uptake by these nodules
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Books on the topic "Hashimoto thyroiditis"

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International Hashimoto Symposium (1992 Fukuoka-shi, Japan). 80 years of Hashimoto disease: Proceedings of the International Hashimoto Symposium, 2-5 December, 1992, Fukuoka, Japan, International Satellite Meeting, 6-7 December, 1992, Kyoto, Japan : and memorial lecture on the International Hashimoto Symposium, 2 December, 1992, Fukuoka, Japan. Edited by Nagataki S, Mori Toru 1934-, Torizuka Kanji 1926-, and International Satellite Meeting on Thyrotropin Receptor Antibodies (1992 : Kyoto, Japan). Excerpta Medica, 1993.

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International Hashimoto Symposium (1992 Fukuoka-shi, Japan). 80 years of Hashimoto disease: Proceedings of the International Hashimoto Symposium, 2-5 December, 1992, Fukuoka, Japan, International Satellite Meeting, 6-7 December, 1992, Kyoto, Japan, and Memorial Lecture on the International Hashimoto Symposium, 2 December, 1992, Fukuoka, Japan. Edited by Mori Toru 1934-, Nagataki S, Torizuka Kanji 1926-, and International Satellite Meeting on Thyrotropin Receptor Antibodies (1992 : Kyoto, Japan). Elsevier Science Publishers, 1993.

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Kharrazian, Datis. Why do I still have thyroid symptoms?: When my lab tests are normal : a revolutionary breakthrough in understanding Hashimoto's disease and hypothyroidism. Elephant Press LP, 2010.

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GRANT, Richard. Hashimoto Thyroiditis: Useful Information Regarding Hashimoto Thyroiditis Symptoms, Causes, Treatment and Precautions. Independently Published, 2022.

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Dutch, Michael Michael. Beginners Guide to Hashimoto Thyroiditis. Independently Published, 2021.

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Nagataki, S., and Toru Mori. 80 Years of Hashimoto Disease: Proceedings of the : International Hashimoto Symposium, 2-5 December, 1992, Fukuoka, Japan; International Satellite M (International Congress). Elsevier Science & Technology, 1993.

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Hashimoto Thyroiditis for Beginners: An Easy Guide to Hahsimoto's Thyroiditis, Symptoms, Causes, Treatment and Precautions. Independently Published, 2022.

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Peters, Doctor Mabel. Hashimoto Thyroiditis: Understanding the Cause of Hashimoto's Thyroiditis and How to Cure Your Thyroid Problem to Live a Healthy and More Fulfilling Life. Independently Published, 2019.

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MASTEJ, Morris. Hashimoto's Thyroiditis: The Complete Nutritional Guide to Treating Hypothyroidism with Hashimoto Diet Meal Plan and Cookbook. Independently Published, 2022.

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WILLIAMS, Betty. Hashimoto Vegetarian Cookbook: Everything You Need to Know about Hashimoto Thyroiditis Diseases with Healthy Recipes to Boost the Immune System. Independently Published, 2022.

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Book chapters on the topic "Hashimoto thyroiditis"

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Rocchi, Roberto, Noel R. Rose, and Patrizio Caturegli. "Hashimoto Thyroiditis." In Diagnostic Criteria in Autoimmune Diseases. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_41.

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Basolo, Fulvio, Agnese Proietti, and Clara Ugolini. "Hashimoto Thyroiditis." In Encyclopedia of Pathology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-28845-1_5062-1.

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Adeniran, Adebowale J., and David Chhieng. "Hashimoto Thyroiditis." In Common Diagnostic Pitfalls in Thyroid Cytopathology. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-31602-4_5.

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Basolo, Fulvio, Agnese Proietti, and Clara Ugolini. "Hashimoto Thyroiditis." In Endocrine Pathology. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-62345-6_5062.

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Antonini, Simone, Maria Francesca Birtolo, Andrea Lania, and Elena Vittoria Longhi. "Hashimoto Thyroiditis." In Managing Psychosexual Consequences in Chronic Diseases. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-31307-3_9.

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Cochand-Priollet, Beatrix. "Hashimoto Thyroiditis, Cytological Findings." In Encyclopedia of Pathology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-33286-4_891.

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Schmidt, K. P., Eva Deckart, R. Pilz, and E. Strehlau. "Combination of Hashimoto- and de Quervain thyroiditis." In New Aspects in Thyroid Diseases, edited by H. F. Deckart and E. Strehlau. De Gruyter, 1992. http://dx.doi.org/10.1515/9783110874051-027.

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Cuan-Baltazar, Y., and E. Soto-Vega. "Microorganisms in Pathogenesis and Management of Hashimoto Thyroiditis." In Role of Microorganisms in Pathogenesis and Management of Autoimmune Diseases. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-1946-6_15.

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Russell, Marika D., and Janet Chiang. "Diagnosis and Management of Thyroiditis: Hashimoto, de Quervain, Riedel." In Endocrine Surgery Comprehensive Board Exam Guide. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-84737-1_4.

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Kohila, S., and G. Sankara Malliga. "Texture Analysis on Thyroid Ultrasound Images for the Classification of Hashimoto Thyroiditis." In Computer Aided Intervention and Diagnostics in Clinical and Medical Images. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-04061-1_28.

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Conference papers on the topic "Hashimoto thyroiditis"

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Santhoshkumar, Sundar, Uma Ramasamy, Romany F. Mansuour, and E. Ramaraj. "A Review on Statistical Importance and Biomarkers Identification in Hashimoto Thyroiditis Disease." In 2021 11th International Conference on Cloud Computing, Data Science & Engineering (Confluence). IEEE, 2021. http://dx.doi.org/10.1109/confluence51648.2021.9377133.

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Geisler, Antje, and Christoph Becker. "Hashimoto&apos;s thyroiditis – Risk factor for the development of thyroid carcinoma." In 95th Annual Meeting German Society of Oto-Rhino-Laryngology, Head and Neck Surgery e. V., Bonn. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1784615.

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Chantziantoniou, Nikolaos. "Coexisting Papillary Thyroid Carcinoma and Hashimoto Thyroiditis in Thyroid Fna, with Associated Genomic Predisposition." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2016. http://dx.doi.org/10.5339/qfarc.2016.hbpp2392.

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Leite, Luíza Maria Guimarães de Souza, MAURO CASTRO DE ALBUQUERQUE FILHO, THAINÁ DE FÁTIMA MOURA COSTA, and EDENILSON CAVALCANTE SANTOS. "SELÊNIO E ESTRESSE OXIDATIVO EM PACIENTES COM TIREOIDITES AUTOIMUNES: UMA REVISÃO." In II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/6749.

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Introdução: O selênio atua nos sistemas de metabolização dos hormônios tireoidianos e como cofator de enzimas que promovem o equilíbrio oxidativo e inflamatório no tecido tireoidiano. A falta desse oligoelemento pode acarretar o desbalanço desse processos e acarretar aumento do estresse oxidativo celular. A tireoidite de Hashimoto (TH) é uma doença autoimune caracterizada por danos aos tecidos tireoidianos mediados pela ação de autoanticorpos específicos. Estudos prévios verificaram que a suplementação com selênio pode reduzir o estresse oxidativo nas células tireoidianas. Objetivo: Neste estudo, busca-se revisar na literatura acerca da suplementação de selênio e os impactos em parâmetros relativos ao estresse oxidativo e as repercussões em pacientes com ou sem tireoidite autoimune. Material e métodos: Foi realizada pesquisa nas plataformas PubMed e BVS, tendo sido utilizados os termos de busca “autoimmune thyroiditis”; “oxidative stress” e “selenium”, com a seleção de 28 estudos. Com a exclusão dos estudos duplicados, foram incluídos 4 estudos para a realização desta revisão. Resultados: Verificou-se que a atividade das enzimas antioxidantes não apresentou diferença significativa entre os pacientes com tireoidites autoimunes e o grupo saudável, mas evidenciou que o grupo com a doença apresentou atividade reduzida das enzimas superóxido dismutase e da glutationa peroxidase em um dos estudos. A suplementação de selenometionina demonstrou correlação positiva entre a concentração de malondialdeído e o anticorpo antitireoglobulina, não tendo sido observada correlação entre a atividade da superóxido dismutase e a presença dos anticorpos anti-tireoperoxidase e antitireoglobulina. O estudo que associou selenometionina ao inositol demonstrou promover menor grau de estresse oxidativo, na comparação à selenometionina isolada e ao placebo. Conclusão: Com esta revisão conclui-se que, embora o selênio seja um elemento fundamental na redução do estresse oxidativo e na regulação da função tireoidiana, não existem evidências de que a concentração sérica ou suplementação desse oligoelemento promova substancial impacto na redução estresse oxidativo repercutindo na diminuição da concentração sérica de dos autoanticorpos envolvidos na tireoidite de Hashimoto.
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Athanassiou, P., L. Athanassiou, M. Mavroudi, P. Tsakiridis, N. Koukosias, and I. Kostoglou-Athanassiou. "PO.4.89 Hashimoto’s thyroiditis in systemic lupus erythematosus patients." In 13th European Lupus Meeting, Stockholm (October 5–8, 2022). Lupus Foundation of America, 2022. http://dx.doi.org/10.1136/lupus-2022-elm2022.115.

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Adil Mansoor, AL-Husnah. "Role of Interleukin-35 In The Pathogenesis of Hashimoto’s Thyroiditis Disease." In X INTERNATIONAL CONGRESS OF PURE AND APPLIED TECHNOLOGICAL SCIENCES. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress10-1.

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The immune system has the ability to distinguish between self and non-self. Also, Regulatory T-cells are a portion of Tcells that are in charge of preserving regulation of the immune system and immune tolerance through active immunerepression. Immune tolerance is very important to prevent the immune system from interacting against itself and thus to avoid the development of autoimmune diseasesInterleukin-35 (IL-35), which is a member of the interleukin-12 (IL12) cytokine family, is an anti-inflammatory immune response that plays the important function in the obstruction of the development of autoimmune diseases in different tests autoimmunities. When loss of self-tolerance occurs becuses many genetic and environmental factors, this causes the absence of balanced anti-inflammatory immune responses, which reduces the in vivo suppressive capacity of Treg, which is taken to be a regular T-cell-linked autoimmune disease., especially Hashimoto’s thyroiditis (HT). Methods: The current study included 90 samples. 60 hypothyroid HT patients (Group 1) and 30 healthy controls (Group 2) were enrolled in the study. Blood samples were collected and separated into serum from the two groups to be used to measure the functions of the thyroid gland by measuring the levels of thyroid hormone and thyroid stimulating hormone, measuring the level of anti-thyroid auto-antibodies, and determining the standard of serum Interloukin-35 through ELISA. Results: The results of the current study Through statistical analysis using the program SPSS version 22, it was shown that serum IL-35 levels were inversely Correlation used Pearson test with TSH, TPO-Ab, and TG-Ab in patients HT (-.292 (P≥0.01), -.245 (P≥0.02), -.269 (P≥0.01). While serum IL-35 levels were directly correlated with TSH, TPO-Ab, and TGAb in healthy controls (.151 (P≥0.01),.192 (P≥0.01),.097 (P≥0.01). The results of the current study showed a highly significant difference in TSH and FT4, respectively (P≥0.01), between the control and hypothyroid groups. Hypothyroidism is more common in females than males, and the incidence of hypothyroidism steadily increases with advancing age. Thyroid hormone concentrations show a significant change in patients with hormonal hypothyroidism compared with the control group. Conclusion: Our findings indicate an effective role for interleukin 35 in the reverse association with auto-antibodies and the development of autoimmune diseases, especially Hashmoto's disease
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Paliwal, Shikha, Pinaki Adak, and Ruchi Kant. "Prevalence of Hashimoto’s thyroiditis among type 1 diabetes mellitus in Jaipur district." In RECENT ADVANCES IN SCIENCES, ENGINEERING, INFORMATION TECHNOLOGY & MANAGEMENT. AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0154424.

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Corlan, Ana-Silvia, Diogen Babuc, Flavia Costi, and Darian Onchis. "Prediction and Classification Models for Hashimoto’s Thyroiditis Risk Using Clinical and Paraclinical Data." In 2023 25th International Symposium on Symbolic and Numeric Algorithms for Scientific Computing (SYNASC). IEEE, 2023. http://dx.doi.org/10.1109/synasc61333.2023.00043.

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Xue, Shuai, Peisong Wang, Zhe Han, Chen Guang, Jia Liu, and Li Zhang. "Analysis of Ultrasonic Characteristics of Hashimoto's Thyroiditis Benign Nodules and Its Relationship with Serum TSH." In 2015 7th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2015. http://dx.doi.org/10.1109/itme.2015.16.

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Allig, Alex Resende, and Nilson Penha Silva. "Selenium supplementation in subclinical Hashimoto’s Thyroiditis: a randomized clinical trial’s systematic review of the preventive clinical and therapeutic impacts." In XXVI Semana Científica do Curso de Medicina da UFU. Even3, 2024. http://dx.doi.org/10.29327/xxvi-semana-cientifica-do-curso-de-medicina-da-ufu-379451.759562.

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Reports on the topic "Hashimoto thyroiditis"

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Wen, Yi, Changda Li, Chunxue Zang, Chanyuan Zhou, and Tianshu Gao. A protocol for systematic review and bayesian network meta-analysis of Integrated traditional Chinese and Western medicine therapies for Hashimoto Thyroiditis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.3.0106.

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Vargas-Uricoechea, Hernando, Alejandro Castellanos-Pinedo, Karen Urrego-Noguera, María V. Pinzón-Fernández, Ivonne A. Meza-Cabrera, and Hernando Vargas-Sierra. A Scoping Review on Prevalence of Hashimoto's Thyroiditis and Possible Associated Factors. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2025. https://doi.org/10.37766/inplasy2025.3.0049.

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Vargas-Uricoechea, Hernando, Alejandro Castellanos-Pinedo, Ivonne A. Meza-Cabrera, María V. Pinzón-Fernández, Karen Urrego-Noguera, and Hernando Vargas-Sierra. Iodine Intake From Universal Salt Iodization Programs and Hashimoto's thyroiditis. A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2025. https://doi.org/10.37766/inplasy2025.4.0074.

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zhang, heng, Zhelong Liu, and Yang yang. Clinical Efficacy of Selenium Supplementation in Patients with Hashimoto's Thyroiditis: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.10.0085.

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