Relevant bibliographies by topics / Hashimoto thyroiditis

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Academic literature on the topic 'Hashimoto thyroiditis'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 April 2022

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Journal articles on the topic "Hashimoto thyroiditis"

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Ahmed, Rania, Safa Al-Shaikh, and Mohammed Akhtar. "Hashimoto Thyroiditis." Advances In Anatomic Pathology 19, no. 3 (May 2012): 181–86. http://dx.doi.org/10.1097/pap.0b013e3182534868.

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Lichtenstein, A. O., and A. A. Agafonov. "Hashimoto type thyroiditis." Kazan medical journal 43, no. 5 (November 16, 2021): 55–56. http://dx.doi.org/10.17816/kazmj87888.

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Thyroiditis (struma limfomatosa - Hashimoto's disease) is a two-sided diffuse dense goiter, intimately fused with the trachea. Fusion with other organs and tissues is not observed. At the same time, the thyroid gland is often slightly enlarged and can cause compression of the trachea, which leads to hoarseness. The disease usually affects older women over 40 years of age (Babcock). On the cut, the tissue of the thyroid gland is dense, whitish, with significant lymphoid infiltration, proliferation of connective tissue, atrophy of the glandular tissue and degeneration of the epithelium. Struma Hashimoto was considered a disease of the Anglo-Saxon countries, where about 200 observations are described. However, in recent years, the domestic literature has been significantly enriched with reports of chronic lymphoid thyroiditis.
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Thompson, Lester D. R. "Chronic Lymphocytic Thyroiditis (Hashimoto Thyroiditis)." Ear, Nose & Throat Journal 93, no. 4-5 (April 2014): 152–53. http://dx.doi.org/10.1177/0145561314093004-508.

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Pakdaman, Michael Navid, Michael P. Hier, Martin J. Black, Michael Tamilia, and Richard J. Payne. "Micropapillary Thyroid Carcinoma and Hashimoto's Thyroiditis." Otolaryngology–Head and Neck Surgery 139, no. 2_suppl (August 2008): P40. http://dx.doi.org/10.1016/j.otohns.2008.05.132.

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Objective An increase in papillary thyroid carcinoma in patients with Hashimoto's thyroiditis has been described. We have yet to find literature discussing a relationship with micropapillary thyroid carcinoma(MPC). Our objective is (1) to assess the relationship between Papillary Microcarcinoma and Hashimoto's Thyroiditis in thyroids without malignant disease, (2) compare effect of the presence of Hashimoto's on prognostic factors in patients. Methods Pathology reports were reviewed for consecutive patients undergoing total thyroidectomy over a 6-year period from 2002 to 2007. Patients with benign pathologies were included. Patients with MPC and no other malignancy were also included. This lead to a cohort of 314 patients. Statistical significance was calculated using Fisher's exact test and a chi-squared analysis. Results Out of the 314 patients, 98 were positive for Hashimoto's thyroiditis on biopsy of the removed thyroid. 58% of the patients with Hashimoto's thyroiditis had an incidental MPC, as compared to 42% of patients where Hashimoto's was not found. Additionally, in Hashimoto patients, 61% of the MPCs were multifocal versus 43% multifocal in the non-Hashimoto group (p-value <0.05 in each comparison). Bilaterality of MPC was also increased in patients with Hashimoto's (22% vs 39%). Conclusions Patients with Hashimoto's thyroiditis in this study had an incidence of MPC of 58%, which is significantly greater than the 42% in those without. Hashimoto's was also associated with an increased risk of bilaterality and multifocality. Studies are needed to further corroborate these findings.
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Hapsari, Siti Nurul, and Sidarti Soehita. "HASHIMOTO’S THYROIDITIS HYPERTHYROID STAGE." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 26, no. 1 (November 22, 2019): 123. http://dx.doi.org/10.24293/ijcpml.v26i1.1423.

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Preliminary: Hashimoto thyroiditis (chronic autoimmune thyroiditis) is the most common cause of hypothyroidism in iodine- sufficient areas of the world, but can sometimes show hyperthyroidism. Case: A 39-year-old female was referred due to shortness of breath and tremor, four hours before hospitalization. There was nausea, chest pain, cold chills and palpitation. She was diagnosed with Hashimoto’s thyroiditis and routinely received tyrosol, propanolol and dexamethasone. Physical examination: cervical mass, afebrile, blood pressure 130/70 mmHg, pulse rate 110 x/minute and respiratory rate 20 x /minute. Laboratory examinations showed WBC 7.53 x 109/L, Hb 11.0 g/dL and platelet count 168 x 109/L. Chest X-Ray: negative for infiltrates. Several laboratory tests were performed, abnormal results were as follows: FT4 2.96 ng/dL (increased), TSH 0.003 uIU/mL (decreased), anti-TPO (antithyroid microsomal antibody) 306 IU/ml (increased), Ig E 213.6 IU/mL (increased). Peripheral blood smear, coagulation test, serum electrolytes, liver function tests, renal function tests, urinalysis, CEA and Ca 125 were within normal limits. A thyroid ultrasound resulted in a benign lesion. Fine Needle Aspiration Biopsy concluded in lymphocytic Hashimoto’s thyroiditis. Echocardiography showed hyperthyroid heart disease. Discussion: Due to an increase in anti-TPO and FT4, a decrease in TSH and lymphocytic thyroiditis from FNAB, this patient was diagnosed with Hashimoto’s Thyroiditis Hyperthyroid Stage. Conclusion: Thyroid function tests and thyroid antibody tests must be monitored to decide whether it is hyperthyroid or hypothyroid stage of Hashimoto thyroiditis.
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Sobolevskaia, Polina A., Boris V. Andreev, Anton N. Gvozdetckii, Anastasia A. Dolina, Anna M. Stepochkina, Yurii I. Stroev, Vladimir J. Utekhin, Tamara V. Fedotkina, and Leonid P. Churilov. "The association of neuropsychiatric disorders and endocrine parameters in hashimoto thyroiditis." Pediatrician (St. Petersburg) 11, no. 4 (December 8, 2020): 55–68. http://dx.doi.org/10.17816/ped11455-68.

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Hashimoto thyroiditis is the most common thyroid disease. This form of pathology has a diverse clinical picture, including neuropsychiatric disorders. There are frequent cases of comorbidity of autoimmune thyroiditis and psychiatric forms of pathology, along with such a nosological entity as Hashimotos encephalopathy (aka: Steroid-responsive encephalopathy of autoimmune thyroiditis), characterized by an increased level of antithyroid autoantibodies and various mental disorders, with still unclear pathogenesis. The question arises, how to regard patients with psychiatric disorders and Hashimoto thyroiditis either as patients having autoimmune thyroiditis, comorbid with psychiatric forms of pathology, or as patients with Hashimotos encephalopathy? We studied groups of patients with autoimmune thyroiditis free from any psychiatric disorders, autoimmune thyroiditis comorbid with psychiatric forms of pathology, and a group of healthy donors similar as regards to their age and sex. We also studied medical history, clinical manifestations of the disease, instrumental data and the serum levels of thyrotropin, thyroid hormones, various antithyroid autoantibodies, and prolactin. We analyzed the correlation of laboratory and instrumental parameters and clinical data in all groups of patients. Therewas a significant relationship (p 0,05) between various psychiatric symptoms and a decreased level of free thyroxine, an increased level of thyroid stimulating hormone (TSH), an increased level of prolactin and an increased volume of a thyroid gland. Asignificant relationship (p 0,05) was also found between various symptoms of hypothyroidism and a decreased level of free triiodothyronine (FT3), an increased level of antibodies to thyroglobulin (anti-TG Ab), and an increased level of antibodies to thyroid peroxidase (anti-TPO Ab).
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Jabeen, Sumerah, and Muhammad Qamar Masood. "SUBACUTE THYROIDITIS EVOLVING INTO HASHIMOTO THYROIDITIS." AACE Clinical Case Reports 4, no. 4 (July 2018): e297-e299. http://dx.doi.org/10.4158/accr-2017-0118.

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Hapsari, Siti Nurul, and Sidarti Soehita. "Hyperthyroid Phase of Hashimoto's Thyroiditis." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 26, no. 1 (November 22, 2019): 123. http://dx.doi.org/10.24293/ijcpml.v26i1.1779.

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Hashimoto thyroiditis (chronic autoimmune thyroiditis) is the most common cause of hypothyroidism in iodine-sufficient areas of the world. This condition, however, can sometimes show hyperthyroidism. A 39-year-old femalewas admitted to hospital due to shortness of breath and tremor four hours before hospitalization. There were nausea, chestpain, cold chills, and palpitation. She was diagnosed with Hashimoto's thyroiditis and routinely received tyrosol,propranolol, and dexamethasone. Physical examination showed cervical mass, afebrile, blood pressure of 130/70 mmHg,pulse rate of 110 beats/minute and respiratory rate of 20 breaths/minute. Laboratory examinations showed WBC 7.53 x 109/L, Hb 11.0 g/dL and platelet count of 168 x 109/L. Chest X-Ray: negative for infiltrates. Several laboratory testswere performed, abnormal results were as follows: FT4 level of 2.96 ng/dL (increased), TSH level of 0.003 µIU/mL(decreased), anti-TPO (antithyroid microsomal antibody) level of 306 IU/ml (increased), and IgE level of 213.6 IU/mL(increased). Peripheral blood smear, coagulation test, serum electrolytes, liver function tests, renal function tests, urinalysis,CEA and Ca 125 were within normal limits. Thyroid ultrasound was performed and showed a benign lesion. Fine needleaspiration biopsy showed lymphocytic Hashimoto's thyroiditis. Echocardiography showed hyperthyroid heart disease. Dueto an increase of anti-TPO and FT4 levels, a decrease of TSH levels and lymphocytic thyroiditis from FNAB, this patient wasdiagnosed with a hyperthyroid phase of Hashimoto's thyroiditis. Thyroid function tests and thyroid antibody tests must bemonitored to distinguish between the hyperthyroid and hypothyroid phase of Hashimoto thyroiditis.
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Atia, Ahmed, Rihan Alathream, and Abdulwahab Al-Deib. "Incidence of Hashimoto Thyroiditis Among Libyans: A Retrospective Epidemiological Study." Journal of Medical Research and Innovation 5, no. 1 (March 6, 2021): e000251. http://dx.doi.org/10.32892/jmri.251.

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Background and aims. Hashimoto's disease is an autoimmune disorder in which the body produces antibodies that attack the thyroid gland, leading to chronic inflammation, destruction of the gland, and hypothyroidism. This study aimed to assess the epidemiology of this disease among Libyan patients. Methods. A cross-sectional retrospective study conducted from June 2012 to April 2020 in order to examine the anti TPO level among Libyan population. Data was collected from eastern and western part of Libya, and were analyzed from available sample for 244 apparently patients with thyroid disorders collected from different private clinic’s laboratories. The analysis for serum anti-TPO was done by electrochemiluminescence protein binding assay (ECLIA) using Roche diagnostics and Cobas e411 analyzer. Results. The current results showed that females predominate the study, and most of them were in the age group of (>40) years old. About 49.18% of these cases were suffering from Hashimoto's disease (High ATPO level). The mean value of anti-TPO status among females was (0.5±2) nmol/L, while among males it was (0.45±3) nmol/L. Significantly, more women (81.66%) had Anti- TPO Above (34 IU/ml), compared to (18.33%) of male participants. Conclusion. Hashimoto disease is common among patients with thyroid dysfunction especially females. Our findings suggest that different interventional strategies are needed to reduce the chances of developing Hashimoto’s and its associated negative health outcomes in Libya.
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Pyzik, Aleksandra, Ewelina Grywalska, Beata Matyjaszek-Matuszek, and Jacek Roliński. "Immune Disorders in Hashimoto’s Thyroiditis: What Do We Know So Far?" Journal of Immunology Research 2015 (2015): 1–8. http://dx.doi.org/10.1155/2015/979167.

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This review of literature attempts to identify the factors that are involved in the pathogenesis of Hashimoto thyroiditis, an immune defect in an individual with genetic susceptibility accompanied with environmental factors. The frequency of Hashimoto’s disease is a growing trend and among Caucasians it is estimated at approximately 5%. The dysfunction of the gland may be clinically evident (0.1–2% of the population) or subclinical (10–15%). The pathology is diagnosed five to ten times more often in women than men and its incidence increases with the age (the peak of the number of cases is between 45 and 65); however, it can also be diagnosed in children. The pathogenesis of Hashimoto’s thyroiditis is still not fully comprehended. In the etiology of Hashimoto thyroiditis excessively stimulated T CD4+ cells are known to play the most important role. Recent research has demonstrated an increasing role of newly discovered cells such as Th17 (CD4+IL-17+) or T regulatory cells (CD4+CD25+highFoxP3+) in the induction of autoimmune disorders. The process of programmed cell death also plays an equally important role in the pathogenesis and the development of hypothyroidism.
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Dissertations / Theses on the topic "Hashimoto thyroiditis"

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陳結清 and Kit-ching Amanda Chan. "Detection of RET/PTC translocation in hashimoto thyroiditis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40738413.

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Chan, Kit-ching Amanda. "Detection of RET/PTC translocation in hashimoto thyroiditis." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B40738413.

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Quintino-Moro, Alessandra 1973. "Infertilidade em mulheres com Doença de Graves e Tireoidite de Hashimoto : Infertility among women with Graves's disease or Hashimoto's thyroiditis." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308429.

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Orientadores: Arlete Maria dos Santos Fernandes, Denise Engelbrecht Zantut Wittmann
Dissertação (Mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: A disfunção autoimune, representada pelo hipertireoidismo da Doença de Graves (DG) e pelo hipotireoidismo da Tireoidite de Hashimoto (TH), ocorre de forma mais frequente em mulheres e interfere no mecanismo da reprodução, em especial no processo ovulatório. Existe o senso comum de que as disfunções tireoideanas diminuem o potencial de fertilidade das mulheres; entretanto, não existem estudos sobre a prevalência da infertilidade nesses grupos. Objetivos: Determinar a prevalência de infertilidade em mulheres com TH e DG e as possíveis variáveis associadas. Sujeitos e métodos: Foi um estudo de corte transversal. Mulheres com TH (n=66) e idade entre 18 e 60 anos e DG (n=193) com idade entre 18 e 50 anos, em seguimento no Ambulatório de Tireoidopatias do HC/UNICAMP, no período de agosto de 2010 a dezembro de 2011, foram entrevistadas com respeito às variáveis ginecoobstétricas: história de períodos de infertilidade, alterações do ciclo menstrual, história obstétrica e antecedentes familiares. Infertilidade foi definida como ausência de gravidez após período de exposição ?12 meses. Após a entrevista, seus prontuários foram revisados para determinar as características da doença: idade ao diagnóstico, tempo do diagnóstico, antecedentes de outras doenças autoimunes associadas, exames laboratoriais e de imagem. O estudo obteve aprovação do Comitê de Ética em Pesquisa da FCM/UNICAMP. Os critérios de inclusão foram: ao menos um ano de convívio com parceiro masculino e concordância em participar da pesquisa. Os dados foram anotados em ficha de coleta e posteriormente digitados em banco de dados elaborado para o estudo. Após a consistência do banco, foi realizada análise descritiva das variáveis gineco-obstétricas e características da doença, e foram aplicados os testes quiquadrado ou exato de Fisher, teste de Mann-Whitney e teste de Wilcoxon. Posteriormente, foi realizada a análise univariada com o cálculo de Odds Ratio bruto e respectivo intervalo de confiança (IC) de 95%, seguida pela regressão logística múltipla incluindo todas as variáveis, buscando aquelas significativamente associadas à infertilidade. O grau de significância estatística foi de 5%. Resultados: A prevalência de infertilidade foi de 52,3% e 47,0% nas mulheres, respectivamente com DG e TH. Na DG, as perdas gestacionais tiveram prevalência de 18,8% e 21,7% em mulheres com e sem infertilidade, enquanto na TH as perdas gestacionais ocorreram em 22,6% e 20,6% nos mesmos grupos. A média do número de gestações foi menor após o diagnóstico de DG e TH nas mulheres com idade ?35 anos. No mesmo grupo de idade, a média do número de gestações antes do diagnóstico foi de 1,68 (DP ±1,41) para DG e 1,48 (DP ±1,31) para TH. As alterações de ciclo menstrual na DG ocorreram em 47,5% e 35,9% das mulheres, respectivamente com e sem infertilidade, e na TH as taxas foram de 64,5% e 34,3% nos mesmos grupos. A única variável associada à infertilidade na TH foi o tempo de doença menor que seis anos. Não houve variável associada nas mulheres com DG. Conclusões: a prevalência de infertilidade foi alta e, no grupo de mulheres com idade ?35 anos, houve diminuição na média de gestações, mostrando o comprometimento da fertilidade das mulheres com DG e TH. Mulheres com TH com menos tempo de doença foram as mais afetadas pela infertilidade
Abstract: The autoimmune dysfunction, hyperthyroidism represented by the Graves disease (GD) and by hypothyroidism of Hashimoto's thyroiditis (TH), occurs more often in women and interferes in the mechanism of reproduction, especially in the ovulatory process. There is common sense that thyroid dysfunction decreases the fertility potential of women, however, there are no studies on the prevalence of infertility in these groups. Objectives: To determine the prevalence of infertility in women with TH and DG and possible associated variables. Subjects and Methods: This was a cross-sectional study. Women with TH (n = 66) aged between 18 and 60 years and DG (n = 193) aged between 18 and 50 years was followed at the Endocrinology Division, Departament of Clinical Medicine, School of Medical Sciences, University of Campinas (UNICAMP), from August 2010 to December 2011 were interviewed with respect to gynecological and obstetric variables: history of periods of infertility, menstrual abnormalities, obstetric history and family history. Infertility was defined as 12 months of unprotected sexual intercourse without conception. After the interview, their medical records were reviewed to determine the characteristics of the disease: age at diagnosis, time since diagnosis, history of other autoimmune diseases associated, laboratory tests and imaging tests. The study was approved by the Research Ethics Committee of the FCM/UNICAMP. Inclusion criteria were: at least a year of living with a male partner, and agreed to participate. Data were recorded on collection and subsequently entered into a Summary - xvii database designed for the study. After the database consistency, descriptive analysis was performed gynecological and obstetric variables and disease characteristics, and applied the chi-square or Fisher's exact test, Mann-Whitney and Wilcoxon test. Subsequently, univariate analysis was performed to calculate crude odds ratios and confidence intervals (CI) of 95%, followed by multiple logistic regressions including all variables significantly associated with those seeking infertility. The level of statistical significance was 5%. Results: The prevalence of infertility was 52.3% and 47.0% in women, with DG and TH respectively. In DG, the miscarriages had a prevalence of 18.8% and 21.7% in women with and without infertility, while in the TH pregnancy loss occurred in 22.6% and 20.6% in the same groups. The mean number of pregnancies was lower after the diagnosis of GD and HT in women aged ? 35 years. In the same age group, the mean number of pregnancies before diagnosis was 1.68 (SD ± 1.41) for DG and 1.48 (SD ± 1.31) for TH. Changes in the menstrual cycle in DG occurred in 47.5% and 35.9% of women, respectively with and without infertility, and TH rates were 64.5% and 34.3% in the same groups. The only variable associated with infertility in TH disease duration was less than six years. There was no associated variable in women with GD. Conclusions: The prevalence of infertility was high and, in the group of women aged ? 35 years, there was a decrease in average pregnancies, showing impairment of fertility in women with GD and HT. Women with TH with shorter disease were most affected by infertility
Mestrado
Fisiopatologia Ginecológica
Mestra em Ciências da Saúde
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Noal, Cristiano Bicca. "ATIVIDADE DE ENZIMAS QUE DEGRADAM NUCLEOTÍDEOS E NUCLEOSÍDEO DE ADENINA EM PLAQUETAS DE PACIENTES COM TIREOIDITE DE HASHIMOTO EM TRATAMENTO COM LEVOTIROXINA SÓDICA." Universidade Federal de Santa Maria, 2012. http://repositorio.ufsm.br/handle/1/5976.

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Hashimoto Thyroiditis (HT) or chronic lymphocytic thyroiditis is an autoimmune disease that causes the destruction of the thyroid gland by inflammatory infiltrates and consequently loss of function. This disease is spread worldwide and predominantly affects females. The consequences of alterations in its activity range from cretinism to the vascular changes. It is known that the enzymes ectonucleoside diphosphohydrolase triphosphate (E-NTPDase, EC 3.6.1.5, CD39), ecto-5'nucleotidase (EC 3.1.3.5, CD73) and adenosine deaminase (ADA, EC 3.5.4.4) are involved in regulating the immune response and thrombotic events, since they regulate extracellular levels of adenine nucleotides and nucleoside. Since HT patients have an autoimmune response and present microvascular changes, the objective of this study was to evaluate the influence of purinergic signaling in the regulation of microvascular dysfunction triggered by the disease determining the activity of the ectoenzymes involved in the metabolism of ATP in platelets from patients with HT in treatment with levothyroxine. Samples were collected from patients with HT treated with levothyroxine and from a control group. In this study we determined the activity of the enzymes E-NTPDase, ecto-5'-nucleotidase and E-ADA; detected the expression of the enzyme in platelets and E-NTPDase; and measured hormone levels of TSH and fT4 in patients with HT treated with levothyroxine and control group. Results obtained in the enzyme activity showed that patients with HT in hormone replacement with levothyroxine presented no significant changes when compared with the control group. In conclusion, levothyroxine used in patients with HT may reverse the effects of hypothyroidism when used regularly in these patients, while maintaining the enzyme activity in basal levels.
A Tireoidite de Hashimoto (TH) ou tireoidite linfocítica crônica é uma doença autoimune que causa a destruição da glândula tireóide por meio de infiltrados inflamatórios e consequente perda da função. Esta patologia encontra-se difundida mundialmente e acomete prevalentemente o sexo feminino. As consequências das alterações de sua atividade vão desde cretinismo à alterações vasculares. Sabe-se que as enzimas ectonucleosídeo trifosfato difosfo-hidrolase (E-NTPDase; E.C. 3.6.1.5; CD39), ecto-5 nucleotidase (E.C.3.1.3.5; CD73) e adenosina desaminase (ADA; E.C.3.5.4.4) participam tanto da regulação da resposta imune quanto de eventos trombóticos, uma vez que regulam os níveis extracelulares dos nucleotídeos e nucleosídeo da adenina. Visto que, pacientes portadores da TH possuem uma resposta autoimune e também apresentam alterações microvasculares, o objetivo deste estudo foi avaliar a influência da sinalização purinérgica na regulação das disfunções microvasculares desencadeadas pela doença, através da determinação da atividade de ectoenzimas envolvidas no metabolismo do ATP em plaquetas de pacientes com TH em tratamento com levotiroxina. Foram coletadas amostras de pacientes com TH em tratamento com levotiroxina e um grupo controle. Neste estudo determinamos a atividade das enzimas E-NTPDase, ecto-5 -nucleotidase e E-ADA, verificamos a expressão da enzima E-NTPDase em plaquetas e dosamos os níveis hormonais de TSH e fT4 em pacientes com TH em tratamento com levotiroxina, bem como do grupo controle. Os resultados obtidos na atividade das enzimas e na concentração dos nucleotídeos e nucleosídeo da adenina, não demonstraram alterações significativas quando comparamos os pacientes com TH em reposição hormonal com levotiroxina, com o grupo controle. Em conclusão, sugere-se que a levotiroxina usada em pacientes com TH pode reverter os efeitos do hipotireoidismo quando usada regularmente por estes pacientes, mantendo as atividades das enzimas em níveis basais.
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Botelho, Ilka Mara Borges 1979. "Prevalência de insuficiência de vitamina D em pacientes com tireoidite de Hashimoto e sua relação com autoimunidade tireoideana = Prevalence of vitamin D insufficiency in patients with Hashimoto's thyroiditis and its relationship with thyroid autoimmunity." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308788.

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Orientadores: Denise Engelbrecht Zantut Wittmann, Sarah Monte Alegre
Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas
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Resumo: Introdução: Vitamina D tem sido apontada como importante regulador da resposta imune. Estudos tem demonstrado haver relação entre insuficiência de vitamina D e presença de doenças autoimunes como Tireoidite de Hashimoto (TH). É possível que o processo autoimune na TH seja inibido em diferentes estágios pela vitamina D em sua forma ativa. Nossos objetivos foram estudar a prevalência de insuficiência de vitamina D e a relação de suas concentrações séricas com marcadores de função e autoimunidade tireoideana. Material e Métodos: Amostras de sangue foram coletadas de 54 pacientes com TH e 54 indivíduos saudáveis sem diagnóstico de TH com idade entre 18 e 75 anos. Foram realizadas dosagens séricas de vitamina D (25OHD), TSH, T4 livre, cálcio, fósforo, paratormônio (PTH), anticorpos anti-tireoperoxidase (AcTPO), anti-tireoglobulina (AcTG) e anti-receptor de TSH (TRAb). Volume tireoideano foi estimado por ultrassonografia. Foram coletados dados demográficos, de peso, altura, índice de massa corporal (IMC) e tempo de diagnóstico. Pacientes e indivíduos do grupo de controle foram pareados por idade e sexo. O nível de significância estatística adotado foi 5%. Resultados: Prevalência de insuficiência de vitamina D foi encontrada em 68.5% dos pacientes e em 38.9% dos indivíduos do grupo de controle (p =0,002). Houve uma correlação positiva entre níveis de AcTPO e maior volumetireoideano nos pacientes (r = 0,319; p= 0.019). Não houve correlação entre concentração de vitamina D, TSH, T4livre,TRAb, AcTGe volume tireoideano. Conclusões: Demonstramosmaior prevalência deinsuficiência de vitamina Dem pacientescom tireoidite de Hashimotoem relaçãoa indivíduos de um grupo controlesaudável, não havendo correlaçãocom o estado hormnal tireoideanooumarcadores séricos deautoimunidadeda tireóide.Por sua vez, maior volume da tireóidese associou a maior grau de infiltração inflamatóriaautoimune,refletido pelacorrelaçãocom maiores concentrações AcTPO
Abstract: Introduction: Vitamin D has been pointed out as an important immune response regulator. Studies have shown a relationship between vitamin D insufficiency and the presence of autoimmune diseases such as Hashimoto's Thyroiditis (HT). It's possible that the autoimmune process in HT is inhibited in its different stages by vitamin D on its active form .Our aims were to study the prevalence of vitamin D insufficiency and relationship of the serum concentrations with thyroid function and autoimmunity markers. Material and Methods: Blood samples were collected from 54 patients with HT and 54 healthy individuals without a diagnosis of HT, aged 18 to 75 years. We conducted serum 25OH vitamin D, TSH, free T4, calcium, phosphorus, PTH, TPOAb, TgAb and TRAb. Thyroid volume was estimated by ultrasound. Data on demographic, weight, height, body mass index and time since diagnosis were collected. Patients and control subjects were matched by sexand age. The significance level for statistical analysis was 5%. Results: Prevalence of vitamin D insufficiency was found in 68.5% of patients and in 38.9% of subjects in the control group (p= 0.002). There was a positive correlation between TPOAb and volume in patients (p= 0.019). There was no correlation between vitamin D concentration and thyroid volume, TRAb, TgAb, TSH or free T4. Conclusions: We demonstrated a higher prevalence of vitamin D insufficiency in patients with Hashimoto's thyroiditis compared to individuals of a healthy control group, no correlation with thyroid state hormonal or serum markers of thyroid autoimmunity. In turn, greater thyroid volume was associated with a higher degree of autoimmune inflammatory infiltration, reflected by the correlation with higher concentrations AcTPO
Mestrado
Clinica Medica
Mestra em Ciências
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Alami, Harchali Asmae. "Détection par immunephelemetrie sur supports microparticulaires d'autoanticorps anti-thyroïde de spécificité épisodique définie : mise au point de la méthode et applications." Nancy 1, 1994. http://www.theses.fr/1994NAN10286.

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Ce travail a pour objectif d'adapter à la détection et à la caractérisation d'autoanticorps, la technique d'immunonephelemetrie sur supports microparticulaires. Le but est d'affiner le pouvoir discriminant de cette technique jusqu'à l'identification de la spécificité épitopique des anticorps décelés. Un premier groupe d'applications porte sur la détection des autoanticorps anti-thyroglobuline humaine. Un test de détection de ces autoanticorps anti-domaine antigénique deux de la molécule de thyroglobuline, a été développé. Il est basé sur l'aptitude des autoanticorps à inhiber les systèmes agglutinants thyroglobuline-anticorps monoclonaux anti-thyroglobuline ont ainsi été détectés et quantifiés chez tous les patients atteints de thyroïdite d'Hashimoto et dans plus du trois-quarts des patients présentant une maladie de basedow non traitée. Les résultats obtenus se comparent favorablement à ceux de l'immunofluorescence indirecte et de l'hemagglutination passive. Le système mis au point peut aussi se prêter au dosage de la thyroglobuline sérique par inhibition. La détection des autoanticorps anti-thyroperoxydase humaine est calquée dans son principe sur le protocole mis au point pour les anticorps anti-thyroglobuline. Ce test confirme la polyclonalité des autoanticorps anti-thyroperoxydase et leur réactivité préférentielle envers les épitopes localisés sur les domaines antigéniques a et b de la molécule de thyroperoxydase, épitopes impliques dans les maladies thyroïdiennes auto-immunes. Ces mêmes spécificités sont observées chez les patients atteints de thyroïdite d'Hashimoto et de maladie de basedow. Les données immunonephelemetriques sont en accord avec les résultats des autres moyens d'investigation et avec les autres éléments des dossiers cliniques
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MAERENS, BEATRICE. "Vitiligo et thyroidite lymphocytaire juvenile." Dijon, 1994. http://www.theses.fr/1994DIJOM012.

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Sebastianes, Fernando Moreno. "Alterações da PET-FDG na avaliação pré-operatória de pacientes com nódulos tireoidianos e correlação com marcadores imuno-histoquímicos." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-22072011-124925/.

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INTRODUÇÃO: Cerca de 80% dos nódulos de tireóide com citologia indeterminada são benignos. É possível que a tomografia por emissão de pósitrons (PET) com 2-[18F]- fluoro-2-desoxi-D-glicose (FDG) ajude a identificar quais dessas lesões são malignas. A captação de FDG depende da expressão de GLUTs (transportadores de glicose transmembrana) e hexoquinases. Captação difusa de FDG no leito tireoidiano tem ainda sido associada à tireoidite autoimune crônica (TAC), embora haja evidências de que pacientes com parênquima tireoidiano aparentemente sadio possam ter essa alteração. OBJETIVOS: (1) Determinar a sensibilidade e especificidade da PET-FDG no diagnóstico pré-operatório de malignidade dos nódulos tireoidianos, especialmente daqueles com resultados citológicos indeterminados, e avaliar esse desempenho no subgrupo de pacientes com TAC. (2) Determinar a correlação entre o achado de captação difusa tireoidiana da FDG à PET com o diagnóstico histopatológico de TAC e compará-la às concentrações plasmáticas dos anticorpos antitireoidianos. (3) Determinar a correlação entre a expressão dos marcadores imuno-histoquímicos GLUT 1, GLUT 3, GLUT 12, hexoquinase 2 e hexoquinase 3 com o diagnóstico histopatológico dos nódulos e com a captação de FDG apresentada pelos mesmos. MÉTODOS: 56 pacientes com nódulo de tireóide (42 com citologia indeterminada, 10 compatíveis com carcinoma papilífero e 4 com citologia benigna) realizaram PET-FDG e foram submetidos à tireoidectomia. Os resultados da PET-FDG foram comparados com o diagnóstico histopatológico do nódulo, com o infiltrado linfocitário no parênquima tireoidiano, com o diagnóstico de TAC e com os resultados do estudo imuno-histoquímico de micromatriz tecidual de tecido correspondente ao nódulo tireoidiano puncionado e ao tecido tireoidiano não nodular. RESULTADOS: 1) Todos os 21 pacientes com câncer de tireóide (11 com citologia indeterminada) apresentaram captação focal de FDG na tireóide. 2) Captação focal de FDG correlacionou-se com maior risco de malignidade (p<0,001). 3) Dos 31 pacientes com nódulos benignos com citologia indeterminada, 12 não tinham captação focal de FDG (especificidade de 39%). 4) Captação difusa no leito tireoidiano à PET-FDG foi associada à presença de TAC no exame histopatológico (p=0,019). Porém, 5 pacientes, sem sinais de infiltrado linfocitário, apresentaram captação difusa à PET-FDG. 5) Imunoexpressão dos anticorpos contra GLUTs 1, 3 e 12 e hexoquinases 2 e 3 nas células epiteliais dos nódulos não esteve positivamente associada com captação de FDG e com malignidade. 6) Não houve associação entre captação difusa de FDG no leito tireoidiano e imunoexpressão desses marcadores no parênquima tireoidiano não nodular. CONCLUSÕES: 1) A PET-FDG tem alta sensibilidade para diagnóstico de lesões malignas de tireóide, com especificidade de 39% para nódulos com citologia indeterminada. 2) Captação difusa de FDG no leito tireoidiano está associada à presença de TAC, mas pode ocorrer em pacientes sem infiltrado linfocitário. 3) A imuno-histoquímica para os anticorpos contra GLUTs 1, 3 e 12 e hexoquinases 2 e 3 não contribui para a diferenciação de nódulos malignos dos benignos e não se associa com a captação de FDG pelos nódulos
INTRODUCTION: Around 80% of thyroid nodules with indeterminate cytological result are benign. Positron emission tomography (PET) with 2-[18F]-fluoro-2-deoxy- D-glucose (FDG) might help to identify malignant thyroid lesions. FDG uptake depends on GLUTs expression (transmembranous glucose transporters) and on hexokinases. Although diffuse FDG thyroid uptake has been associated with chronic autoimmune thyroiditis (CAT), there is some evidence that patients with apparently normal thyroid parenchyma can display this pattern of FDG uptake. OBJECTIVES: (1) To assess the sensitivity and specificity of FDG PET in identifying thyroid malignancy in the preoperative evaluation of thyroid nodules and evaluate these results in the subgroup of patients with indeterminate cytological results and in the subgroup with CAT. (2) To compare the finding of diffuse FDG thyroid uptake with the histopathologic diagnosis of CAT and with the serum levels of antithyroid antibodies. (3) To evaluate the immunoexpression by thyroid nodules of GLUT 1, GLUT 3, GLUT 12, hexokinase 2 and hexokinase 3 and to compare it with the histopathologic diagnosis of these nodules and with FDG uptake. METHODS: 56 patients with thyroid nodules (42 with indeterminate cytological result, 10 with papillary carcinoma and 4 with benign cytological result) underwent FDG PET and, subsequently, thyroidectomy. FDG PET results were compared with the histopathologic diagnosis of the nodule, lymphocytic infiltrate of thyroid parenchyma, CAT diagnosis and immunohistochemical results of tissue microarray of the tissue from the thyroid nodule and the normal thyroid parenchyma. RESULTS: 1) All the 21 patients with thyroid cancer (11 with indeterminate cytological result) had focal thyroid FDG uptake. 2) Focal FDG uptake was associated with malignancy (p<0.001). 3) From 31 patients with benign nodules whose cytological result was indeterminate, 12 did not display focal FDG uptake (specificity of 39%). 4) Diffuse FDG uptake in thyroid bed was associated with CAT in the histopathologic exam (p=0.019). However, 5 patients without lymphocytic infiltrate had diffuse FDG uptake. 5) Immunoexpression of GLUTs 1, 3 and 12 and hexokinases 2 and 3 by epithelial cells of thyroid nodules was not positively associated with FDG uptake and malignancy. 6) There was no association between diffuse FDG uptake in thyroid bed and immunoexpression of these markers in the normal thyroid tissue. CONCLUSIONS: 1) FDG PET has high sensitivity to the diagnosis of malignancy in thyroid bed, with a specificity of 39% for thyroid nodules with indeterminate cytological result. 2) Diffuse FDG uptake in the thyroid bed is associated with CAT, but can be found in patients without lymphocytic infiltrate. 3) Immunohystochemistry against GLUTs 1, 3 and 12 and hexokinases 2 and 3 does not add in the differentiation of malignant and benign thyroid nodules and is not associated with FDG uptake by these nodules
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HIMBERT, PASCALE. "Exophtalmie unilaterale revelatrice d'une thyroidite de hashimoto : a propos d'un cas." Saint-Etienne, 1989. http://www.theses.fr/1989STET6021.

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DELIGNE, DI MARCANTONIO DI MARTINO AUTILIA. "Mecanismes de l'hypothyroidie au cours de la thyroidite de hashimoto." Lille 2, 1993. http://www.theses.fr/1993LIL2M170.

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Books on the topic "Hashimoto thyroiditis"

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International Hashimoto Symposium (1992 Fukuoka-shi, Japan). 80 years of Hashimoto disease: Proceedings of the International Hashimoto Symposium, 2-5 December, 1992, Fukuoka, Japan, International Satellite Meeting, 6-7 December, 1992, Kyoto, Japan : and memorial lecture on the International Hashimoto Symposium, 2 December, 1992, Fukuoka, Japan. Edited by Nagataki S, Mori Toru 1934-, Torizuka Kanji 1926-, and International Satellite Meeting on Thyrotropin Receptor Antibodies (1992 : Kyoto, Japan). Amsterdam: Excerpta Medica, 1993.

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International Hashimoto Symposium (1992 Fukuoka-shi, Japan). 80 years of Hashimoto disease: Proceedings of the International Hashimoto Symposium, 2-5 December, 1992, Fukuoka, Japan, International Satellite Meeting, 6-7 December, 1992, Kyoto, Japan, and Memorial Lecture on the International Hashimoto Symposium, 2 December, 1992, Fukuoka, Japan. Edited by Mori Toru 1934-, Nagataki S, Torizuka Kanji 1926-, and International Satellite Meeting on Thyrotropin Receptor Antibodies (1992 : Kyoto, Japan). Amsterdam: Elsevier Science Publishers, 1993.

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Kharrazian, Datis. Why do I still have thyroid symptoms?: When my lab tests are normal : a revolutionary breakthrough in understanding Hashimoto's disease and hypothyroidism. Carlsbad, CA, USA: Elephant Press LP, 2010.

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Nagataki, S., and Toru Mori. 80 Years of Hashimoto Disease: Proceedings of the : International Hashimoto Symposium, 2-5 December, 1992, Fukuoka, Japan; International Satellite M (International Congress). Elsevier Science & Technology, 1993.

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Why Do I Still Have Thyroid Symptoms When My Lab Tests Are Normal A Revolutionary Breakthrough In Understanding Hashimotos Disease And Hypothyroidism. Morgan James Publishing, 2009.

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Hashimoto's Thyroiditis: Lifestyle Interventions for Finding and Treating the Root Cause. Wentz LLC, 2013.

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(Compiler), Robert T. Dirgo, and Mary Dirgo (Editor), eds. How I Reversed My Hashimoto's Thyroiditis Hypothyroidism. Writers Club Press, 2001.

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Miller, Aaron E., and Teresa M. DeAngelis. Hashimoto’s Encephalopathy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0018.

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Hashimoto’s encephalopathy (HE) is a rare but potentially corticosteroid responsive disorder, which should be considered in cases of encephalopathy of unclear etiology. Hashimoto’s thyroiditis usually does not accompany HE and thyroid function is generally clinically normal. In this chapter, we review the differential diagnostic suspicions for a case of encephalopathy of unclear etiology and respective aspects that raise concern for HE. In addition, we discuss the utility of diagnostic testing and options for therapeutic management.
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Winter, Barbara, and Dr Shunzhong Shawn Bao. Hashimoto's Thyroiditis Hypothyroidism Fatigue: Questions From Real Patients Not Just Pills. ACE Health Publisher, 2018.

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The everything guide to Hashimoto's thyroiditis: A healing pl;an for managing symptons naturally. 2016.

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Book chapters on the topic "Hashimoto thyroiditis"

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Rocchi, Roberto, Noel R. Rose, and Patrizio Caturegli. "Hashimoto Thyroiditis." In Diagnostic Criteria in Autoimmune Diseases, 217–20. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_41.

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Adeniran, Adebowale J., and David Chhieng. "Hashimoto Thyroiditis." In Common Diagnostic Pitfalls in Thyroid Cytopathology, 59–72. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-31602-4_5.

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Basolo, Fulvio, Agnese Proietti, and Clara Ugolini. "Hashimoto Thyroiditis." In Encyclopedia of Pathology, 1–4. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-28845-1_5062-1.

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Cochand-Priollet, Beatrix. "Hashimoto Thyroiditis, Cytological Findings." In Encyclopedia of Pathology, 191–93. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-33286-4_891.

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Schmidt, K. P., Eva Deckart, R. Pilz, and E. Strehlau. "Combination of Hashimoto- and de Quervain thyroiditis." In New Aspects in Thyroid Diseases, edited by H. F. Deckart and E. Strehlau, 193–204. Berlin, Boston: De Gruyter, 1992. http://dx.doi.org/10.1515/9783110874051-027.

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Russell, Marika D., and Janet Chiang. "Diagnosis and Management of Thyroiditis: Hashimoto, de Quervain, Riedel." In Endocrine Surgery Comprehensive Board Exam Guide, 83–105. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-84737-1_4.

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Weiss, Marie-Luise, A. Blottner, and H. F. Deckart. "Hashimoto-thyroiditis vith long-term negative auto-antibody determination: a case report." In New Aspects in Thyroid Diseases, edited by H. F. Deckart and E. Strehlau, 184–92. Berlin, Boston: De Gruyter, 1992. http://dx.doi.org/10.1515/9783110874051-026.

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Kohila, S., and G. Sankara Malliga. "Texture Analysis on Thyroid Ultrasound Images for the Classification of Hashimoto Thyroiditis." In Computer Aided Intervention and Diagnostics in Clinical and Medical Images, 271–79. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-04061-1_28.

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Stern, John M., and Noriko Salamon. "Hashimoto Encephalopathy also Known as Steroid-Responsive Encephalopathy Associated with Autoimmune Thyroiditis (SREAT)." In Imaging of Epilepsy, 169–73. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-86672-3_40.

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Wiersinga, Wilmar M. "Hashimoto’s Thyroiditis." In Endocrinology, 205–47. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-45013-1_7.

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Conference papers on the topic "Hashimoto thyroiditis"

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Santhoshkumar, Sundar, Uma Ramasamy, Romany F. Mansuour, and E. Ramaraj. "A Review on Statistical Importance and Biomarkers Identification in Hashimoto Thyroiditis Disease." In 2021 11th International Conference on Cloud Computing, Data Science & Engineering (Confluence). IEEE, 2021. http://dx.doi.org/10.1109/confluence51648.2021.9377133.

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Chantziantoniou, Nikolaos. "Coexisting Papillary Thyroid Carcinoma and Hashimoto Thyroiditis in Thyroid Fna, with Associated Genomic Predisposition." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2016. http://dx.doi.org/10.5339/qfarc.2016.hbpp2392.

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Xue, Shuai, Peisong Wang, Zhe Han, Chen Guang, Jia Liu, and Li Zhang. "Analysis of Ultrasonic Characteristics of Hashimoto's Thyroiditis Benign Nodules and Its Relationship with Serum TSH." In 2015 7th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2015. http://dx.doi.org/10.1109/itme.2015.16.

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Moskva, K., O. Kikhtyak, and L. Lapovets. "CHANGES OF GUT MICROBIOTA UNDER THE INFLUENCE OF METFORMIN, PIOGLITAZONE, AND LEVOTHYROXINE IN OVERWEIGHT PATIENTS WITH TYPE 2 DIABETES MELLITUS AND HYPOTHYROIDISM CAUSED BY HASHIMOTO'S THYROIDITIS." In GRUNDLAGEN DER MODERNEN WISSENSCHAFTLICHEN FORSCHUNG. European Scientific Platform, 2021. http://dx.doi.org/10.36074/logos-10.09.2021.68.

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Reports on the topic "Hashimoto thyroiditis"

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Wen, Yi, Changda Li, Chunxue Zang, Chanyuan Zhou, and Tianshu Gao. A protocol for systematic review and bayesian network meta-analysis of Integrated traditional Chinese and Western medicine therapies for Hashimoto Thyroiditis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0106.

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