Journal articles: 'Hashimoto thyroiditis'

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Ahmed, Rania, Safa Al-Shaikh, and Mohammed Akhtar. "Hashimoto Thyroiditis." Advances In Anatomic Pathology 19, no. 3 (May 2012): 181–86. http://dx.doi.org/10.1097/pap.0b013e3182534868.

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Lichtenstein, A. O., and A. A. Agafonov. "Hashimoto type thyroiditis." Kazan medical journal 43, no. 5 (November 16, 2021): 55–56. http://dx.doi.org/10.17816/kazmj87888.

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Thyroiditis (struma limfomatosa - Hashimoto's disease) is a two-sided diffuse dense goiter, intimately fused with the trachea. Fusion with other organs and tissues is not observed. At the same time, the thyroid gland is often slightly enlarged and can cause compression of the trachea, which leads to hoarseness. The disease usually affects older women over 40 years of age (Babcock). On the cut, the tissue of the thyroid gland is dense, whitish, with significant lymphoid infiltration, proliferation of connective tissue, atrophy of the glandular tissue and degeneration of the epithelium. Struma Hashimoto was considered a disease of the Anglo-Saxon countries, where about 200 observations are described. However, in recent years, the domestic literature has been significantly enriched with reports of chronic lymphoid thyroiditis.

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Thompson, Lester D. R. "Chronic Lymphocytic Thyroiditis (Hashimoto Thyroiditis)." Ear, Nose & Throat Journal 93, no. 4-5 (April 2014): 152–53. http://dx.doi.org/10.1177/0145561314093004-508.

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Pakdaman, Michael Navid, Michael P. Hier, Martin J. Black, Michael Tamilia, and Richard J. Payne. "Micropapillary Thyroid Carcinoma and Hashimoto's Thyroiditis." Otolaryngology–Head and Neck Surgery 139, no. 2_suppl (August 2008): P40. http://dx.doi.org/10.1016/j.otohns.2008.05.132.

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Objective An increase in papillary thyroid carcinoma in patients with Hashimoto's thyroiditis has been described. We have yet to find literature discussing a relationship with micropapillary thyroid carcinoma(MPC). Our objective is (1) to assess the relationship between Papillary Microcarcinoma and Hashimoto's Thyroiditis in thyroids without malignant disease, (2) compare effect of the presence of Hashimoto's on prognostic factors in patients. Methods Pathology reports were reviewed for consecutive patients undergoing total thyroidectomy over a 6-year period from 2002 to 2007. Patients with benign pathologies were included. Patients with MPC and no other malignancy were also included. This lead to a cohort of 314 patients. Statistical significance was calculated using Fisher's exact test and a chi-squared analysis. Results Out of the 314 patients, 98 were positive for Hashimoto's thyroiditis on biopsy of the removed thyroid. 58% of the patients with Hashimoto's thyroiditis had an incidental MPC, as compared to 42% of patients where Hashimoto's was not found. Additionally, in Hashimoto patients, 61% of the MPCs were multifocal versus 43% multifocal in the non-Hashimoto group (p-value <0.05 in each comparison). Bilaterality of MPC was also increased in patients with Hashimoto's (22% vs 39%). Conclusions Patients with Hashimoto's thyroiditis in this study had an incidence of MPC of 58%, which is significantly greater than the 42% in those without. Hashimoto's was also associated with an increased risk of bilaterality and multifocality. Studies are needed to further corroborate these findings.

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Hapsari, Siti Nurul, and Sidarti Soehita. "HASHIMOTO’S THYROIDITIS HYPERTHYROID STAGE." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 26, no. 1 (November 22, 2019): 123. http://dx.doi.org/10.24293/ijcpml.v26i1.1423.

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Preliminary: Hashimoto thyroiditis (chronic autoimmune thyroiditis) is the most common cause of hypothyroidism in iodine- sufficient areas of the world, but can sometimes show hyperthyroidism. Case: A 39-year-old female was referred due to shortness of breath and tremor, four hours before hospitalization. There was nausea, chest pain, cold chills and palpitation. She was diagnosed with Hashimoto’s thyroiditis and routinely received tyrosol, propanolol and dexamethasone. Physical examination: cervical mass, afebrile, blood pressure 130/70 mmHg, pulse rate 110 x/minute and respiratory rate 20 x /minute. Laboratory examinations showed WBC 7.53 x 109/L, Hb 11.0 g/dL and platelet count 168 x 109/L. Chest X-Ray: negative for infiltrates. Several laboratory tests were performed, abnormal results were as follows: FT4 2.96 ng/dL (increased), TSH 0.003 uIU/mL (decreased), anti-TPO (antithyroid microsomal antibody) 306 IU/ml (increased), Ig E 213.6 IU/mL (increased). Peripheral blood smear, coagulation test, serum electrolytes, liver function tests, renal function tests, urinalysis, CEA and Ca 125 were within normal limits. A thyroid ultrasound resulted in a benign lesion. Fine Needle Aspiration Biopsy concluded in lymphocytic Hashimoto’s thyroiditis. Echocardiography showed hyperthyroid heart disease. Discussion: Due to an increase in anti-TPO and FT4, a decrease in TSH and lymphocytic thyroiditis from FNAB, this patient was diagnosed with Hashimoto’s Thyroiditis Hyperthyroid Stage. Conclusion: Thyroid function tests and thyroid antibody tests must be monitored to decide whether it is hyperthyroid or hypothyroid stage of Hashimoto thyroiditis.

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Sobolevskaia, Polina A., Boris V. Andreev, Anton N. Gvozdetckii, Anastasia A. Dolina, Anna M. Stepochkina, Yurii I. Stroev, Vladimir J. Utekhin, Tamara V. Fedotkina, and Leonid P. Churilov. "The association of neuropsychiatric disorders and endocrine parameters in hashimoto thyroiditis." Pediatrician (St. Petersburg) 11, no. 4 (December 8, 2020): 55–68. http://dx.doi.org/10.17816/ped11455-68.

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Hashimoto thyroiditis is the most common thyroid disease. This form of pathology has a diverse clinical picture, including neuropsychiatric disorders. There are frequent cases of comorbidity of autoimmune thyroiditis and psychiatric forms of pathology, along with such a nosological entity as Hashimotos encephalopathy (aka: Steroid-responsive encephalopathy of autoimmune thyroiditis), characterized by an increased level of antithyroid autoantibodies and various mental disorders, with still unclear pathogenesis. The question arises, how to regard patients with psychiatric disorders and Hashimoto thyroiditis either as patients having autoimmune thyroiditis, comorbid with psychiatric forms of pathology, or as patients with Hashimotos encephalopathy? We studied groups of patients with autoimmune thyroiditis free from any psychiatric disorders, autoimmune thyroiditis comorbid with psychiatric forms of pathology, and a group of healthy donors similar as regards to their age and sex. We also studied medical history, clinical manifestations of the disease, instrumental data and the serum levels of thyrotropin, thyroid hormones, various antithyroid autoantibodies, and prolactin. We analyzed the correlation of laboratory and instrumental parameters and clinical data in all groups of patients. Therewas a significant relationship (p 0,05) between various psychiatric symptoms and a decreased level of free thyroxine, an increased level of thyroid stimulating hormone (TSH), an increased level of prolactin and an increased volume of a thyroid gland. Asignificant relationship (p 0,05) was also found between various symptoms of hypothyroidism and a decreased level of free triiodothyronine (FT3), an increased level of antibodies to thyroglobulin (anti-TG Ab), and an increased level of antibodies to thyroid peroxidase (anti-TPO Ab).

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Jabeen, Sumerah, and Muhammad Qamar Masood. "SUBACUTE THYROIDITIS EVOLVING INTO HASHIMOTO THYROIDITIS." AACE Clinical Case Reports 4, no. 4 (July 2018): e297-e299. http://dx.doi.org/10.4158/accr-2017-0118.

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Hapsari, Siti Nurul, and Sidarti Soehita. "Hyperthyroid Phase of Hashimoto's Thyroiditis." INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY 26, no. 1 (November 22, 2019): 123. http://dx.doi.org/10.24293/ijcpml.v26i1.1779.

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Hashimoto thyroiditis (chronic autoimmune thyroiditis) is the most common cause of hypothyroidism in iodine-sufficient areas of the world. This condition, however, can sometimes show hyperthyroidism. A 39-year-old femalewas admitted to hospital due to shortness of breath and tremor four hours before hospitalization. There were nausea, chestpain, cold chills, and palpitation. She was diagnosed with Hashimoto's thyroiditis and routinely received tyrosol,propranolol, and dexamethasone. Physical examination showed cervical mass, afebrile, blood pressure of 130/70 mmHg,pulse rate of 110 beats/minute and respiratory rate of 20 breaths/minute. Laboratory examinations showed WBC 7.53 x 109/L, Hb 11.0 g/dL and platelet count of 168 x 109/L. Chest X-Ray: negative for infiltrates. Several laboratory testswere performed, abnormal results were as follows: FT4 level of 2.96 ng/dL (increased), TSH level of 0.003 µIU/mL(decreased), anti-TPO (antithyroid microsomal antibody) level of 306 IU/ml (increased), and IgE level of 213.6 IU/mL(increased). Peripheral blood smear, coagulation test, serum electrolytes, liver function tests, renal function tests, urinalysis,CEA and Ca 125 were within normal limits. Thyroid ultrasound was performed and showed a benign lesion. Fine needleaspiration biopsy showed lymphocytic Hashimoto's thyroiditis. Echocardiography showed hyperthyroid heart disease. Dueto an increase of anti-TPO and FT4 levels, a decrease of TSH levels and lymphocytic thyroiditis from FNAB, this patient wasdiagnosed with a hyperthyroid phase of Hashimoto's thyroiditis. Thyroid function tests and thyroid antibody tests must bemonitored to distinguish between the hyperthyroid and hypothyroid phase of Hashimoto thyroiditis.

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Atia, Ahmed, Rihan Alathream, and Abdulwahab Al-Deib. "Incidence of Hashimoto Thyroiditis Among Libyans: A Retrospective Epidemiological Study." Journal of Medical Research and Innovation 5, no. 1 (March 6, 2021): e000251. http://dx.doi.org/10.32892/jmri.251.

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Background and aims. Hashimoto's disease is an autoimmune disorder in which the body produces antibodies that attack the thyroid gland, leading to chronic inflammation, destruction of the gland, and hypothyroidism. This study aimed to assess the epidemiology of this disease among Libyan patients. Methods. A cross-sectional retrospective study conducted from June 2012 to April 2020 in order to examine the anti TPO level among Libyan population. Data was collected from eastern and western part of Libya, and were analyzed from available sample for 244 apparently patients with thyroid disorders collected from different private clinic’s laboratories. The analysis for serum anti-TPO was done by electrochemiluminescence protein binding assay (ECLIA) using Roche diagnostics and Cobas e411 analyzer. Results. The current results showed that females predominate the study, and most of them were in the age group of (>40) years old. About 49.18% of these cases were suffering from Hashimoto's disease (High ATPO level). The mean value of anti-TPO status among females was (0.5±2) nmol/L, while among males it was (0.45±3) nmol/L. Significantly, more women (81.66%) had Anti- TPO Above (34 IU/ml), compared to (18.33%) of male participants. Conclusion. Hashimoto disease is common among patients with thyroid dysfunction especially females. Our findings suggest that different interventional strategies are needed to reduce the chances of developing Hashimoto’s and its associated negative health outcomes in Libya.

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Pyzik, Aleksandra, Ewelina Grywalska, Beata Matyjaszek-Matuszek, and Jacek Roliński. "Immune Disorders in Hashimoto’s Thyroiditis: What Do We Know So Far?" Journal of Immunology Research 2015 (2015): 1–8. http://dx.doi.org/10.1155/2015/979167.

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This review of literature attempts to identify the factors that are involved in the pathogenesis of Hashimoto thyroiditis, an immune defect in an individual with genetic susceptibility accompanied with environmental factors. The frequency of Hashimoto’s disease is a growing trend and among Caucasians it is estimated at approximately 5%. The dysfunction of the gland may be clinically evident (0.1–2% of the population) or subclinical (10–15%). The pathology is diagnosed five to ten times more often in women than men and its incidence increases with the age (the peak of the number of cases is between 45 and 65); however, it can also be diagnosed in children. The pathogenesis of Hashimoto’s thyroiditis is still not fully comprehended. In the etiology of Hashimoto thyroiditis excessively stimulated T CD4+ cells are known to play the most important role. Recent research has demonstrated an increasing role of newly discovered cells such as Th17 (CD4+IL-17+) or T regulatory cells (CD4+CD25+highFoxP3+) in the induction of autoimmune disorders. The process of programmed cell death also plays an equally important role in the pathogenesis and the development of hypothyroidism.

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Espírito Santo, Raquel, Teresa Sabino, and Ana Agapito. "Oftalmopatia Tiroideia Severa e Tiroidite de Hashimoto, em Doente Eutiroideu." Acta Médica Portuguesa 29, no. 9 (September 30, 2016): 572. http://dx.doi.org/10.20344/amp.7015.

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Anderson, Lauren, William D. Middleton, Sharlene A. Teefey, Carl C. Reading, Jill E. Langer, Terry Desser, Margaret M. Szabunio, Charles F. Hildebolt, Susan J. Mandel, and John J. Cronan. "Hashimoto Thyroiditis: Part 1, Sonographic Analysis of the Nodular Form of Hashimoto Thyroiditis." American Journal of Roentgenology 195, no. 1 (July 2010): 208–15. http://dx.doi.org/10.2214/ajr.09.2459.

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Lygina, E. A., and V. V. Latyi. "Life and Heritage of Hakaru Hashimoto." Clinical and experimental thyroidology 17, no. 2 (September 8, 2021): 20–23. http://dx.doi.org/10.14341/ket12562.

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In May 2021, the 140th anniversary of his birth is celebrated by Japanese doctor Hakaru Hashimoto. Did not receive recognition during his lifetime, this amazing scientist discovered a new disease of the thyroid gland, which later became the eponym. For a long time, his name was forgotten, but the main work of his life was decades ahead of the development of science. Without it, the study of autoimmune pathologies, the incidence of which currently continues to increase, would be impossible. It was on patients with Hashimoto’s thyroiditis that a study was conducted for the first time, demonstrating not only the uniqueness of the pathogenesis of thyroiditis, but also the existence of autoimmune reactivity of the body as a whole.

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Jagodzinska, Julia, Renata Polaniak, Ewa Birkner, and Alicja Kasperska-Zajac. "Analysis of Circulating Vascular Endothelial Growth Factor and Its Soluble Receptors in Patients with Different Forms of Chronic Urticaria." BioMed Research International 2015 (2015): 1–6. http://dx.doi.org/10.1155/2015/578383.

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Background. Vascular endothelial growth factor (VEGF) is a powerful enhancer of vascular permeability and inflammatory response; however its significance in chronic urticaria is poorly recognised.Aim. To compare free circulating levels of VEGF and its soluble receptors (sVEGFR1 and VEGFR2) in patients with different forms of chronic urticaria.Methods. The concentrations of VEGF and its receptors in plateletpoor plasma (PPP)/plasma were measured using enzyme-linked immunosorbent assay in chronic urticaria: (1) chronic spontaneous urticaria (CSU) with positive autologous serum skin test (ASST), (2) CSU with negative response to ASST, (3) CSU with concomitant euthyroid Hashimoto’s thyroiditis (CSU/Hashimoto), (4) delayed pressure urticaria (DPU), and the healthy subjects.Results. There were no significant differences in VEGF concentration in PPP between CSU groups and the healthy subjects. Contrary, VEGF concentration was significantly higher in DPU and CSU/Hashimoto patients as compared with the healthy subjects and CSU groups. Furthermore, VEGF value in CSU/Hashimoto patients during the remission was similar to that of the active period and significantly higher than the healthy subjects; VEGF concentration was significantly correlated with TSH. Plasma concentrations of sVEGF1 and sVEGF2 were similar in chronic urticaria patients and the healthy subjects.Conclusions. Increased free circulating VEGF concentration may result from the urticarial process itself as well as concomitant Hashimoto’s thyroiditis.

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Arsovska, Blagica, Jihe Zhu, and Kristina Kozovska. "CASE REPORT - ACUPUNCTURE TREATMENT IN MALE PATIENT WITH HASHIMOTO THYROIDITIS." International Journal of Research -GRANTHAALAYAH 7, no. 9 (September 30, 2019): 288–91. http://dx.doi.org/10.29121/granthaalayah.v7.i9.2019.612.

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Hashimoto's thyroiditis is associated with the presence of anti-thyroid autoantibodies (anti-TPO). High serum antibodies are found in the active phases of chronic autoimmune thyroiditis. In many medical cases with Hashimoto’s thyroiditis it is possible to have high levels of anti-TPO antibodies and fT4, fT3 and TSH levels to be within the normal range. Most doctors believe that Hashimoto's thyroiditis is an incurable form of thyroid deficiency. With the TCM treatment the patient’s strength and vitality can be improved, the Qi flow can be harmonized, nourished and strengthened and the body’s system can be repaired and recharged so the patient may go into remission. The treated patient is 44 year old man, diagnosed with Hashimoto’s thyroiditis caused by stress. The patient has done 40 acupuncture treatments within 21 months, once a week. The patient wasn’t taking any additional hormonal medications. The parameters for fT4, fT3 and TSH were showing normal levels and the anti-TPO levels were increased. Before the treatment the anti-TPO antibodies amount was 252.4 IU/ml and after the treatment 4.07 IU/ml (normal <80 IU/ml). Treated points are: BL15 (XinShu), BL20 (PiShu), BL23 (ShenShu), DU4 (MingMen), DU14 (DaZhui), DU20 (BaiHui), ST9 (RenYing), LI4 (HeGu), KI3 (TaiXi), GB20 (FengChi), RN4 (QuanYuan), RN6 (QiHai), LR2 (TaiChong), SP9 (YinLingQuan), SP6 (SanYinJiao), ST36 (ZuSanLi) and Ashi points located on the neck (front and back).

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Chiarella, Giuseppe, Diego Russo, Fabio Monzani, Claudio Petrolo, Bruno Fattori, Giuseppe Pasqualetti, Ettore Cassandro, and Giuseppe Costante. "HASHIMOTO THYROIDITIS AND VESTIBULAR DYSFUNCTION." Endocrine Practice 23, no. 7 (July 2017): 863–68. http://dx.doi.org/10.4158/ep161635.ra.

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Catalán, R., R. Rossell, J. Treserra, R. Sorio, M. J. Chinchilla, and C. Udina. "Mood disorders in hashimoto thyroiditis." Biological Psychiatry 42, no. 1 (July 1997): 111S. http://dx.doi.org/10.1016/s0006-3223(97)87351-9.

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Luboshitzky, Rafael, Leonid Chervinsky, Ghali Qupti, and Muralee Dharan. "Cytodiagnostic Accuracy of Hashimoto Thyroiditis." Endocrinologist 19, no. 2 (March 2009): 55–57. http://dx.doi.org/10.1097/ten.0b013e318198b766.

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Roychowdhury, Monika, and Stefan E. Pambuccian. "Hashimoto thyroiditis with crystalloid formation." Diagnostic Cytopathology 39, no. 12 (October 1, 2010): 909–11. http://dx.doi.org/10.1002/dc.21536.

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Shih, George, Wei-Jen Shih, Wen-Sheng Huang, and Primo P. Milan. "Hashimoto Thyroiditis and Takayasu Aortitis." Clinical Nuclear Medicine 33, no. 5 (May 2008): 377–79. http://dx.doi.org/10.1097/rlu.0b013e31816a796d.

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Ginaldi, Sergio. "Malignant Lymphoma in Hashimoto Thyroiditis." Journal of Computer Assisted Tomography 10, no. 6 (November 1986): 1071–72. http://dx.doi.org/10.1097/00004728-198611000-00041.

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Petek-Balci, Belgin, Vildan Yayla, and Feriha ??zer. "Multiple Sclerosis and Hashimoto Thyroiditis." Neurologist 11, no. 5 (September 2005): 301–4. http://dx.doi.org/10.1097/01.nrl.0000162956.40653.38.

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Rishi, Rahul, Sudip Ringwala, James Tracy, and Shahnaz Fatteh. "Prurigo nodularis and Hashimoto thyroiditis." Annals of Allergy, Asthma & Immunology 113, no. 6 (December 2014): 673–74. http://dx.doi.org/10.1016/j.anai.2014.09.010.

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Ucan, Bekir, Mustafa Sahin, Muyesser Sayki Arslan, Nujen Colak Bozkurt, Muhammed Kizilgul, Askin Güngünes, Erman Cakal, and Mustafa Ozbek. "Vitamin D Treatment in Patients with Hashimoto’s Thyroiditis may Decrease the Development of Hypothyroidism." International Journal for Vitamin and Nutrition Research 86, no. 1-2 (February 2016): 9–17. http://dx.doi.org/10.1024/0300-9831/a000269.

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Abstract.The relationship between Hashimoto’s thyroiditis and vitamin D has been demonstrated in several studies. The aim of the present study was to evaluate vitamin D concentrations in patients with Hashimoto’s thyroiditis, the effect of vitamin D therapy on the course of disease, and to determine changes in thyroid autoantibody status and cardiovascular risk after vitamin D therapy. We included 75 patients with Hashimoto’s thyroiditis and 43 healthy individuals. Vitamin D deficiency is defined as a 25-hydroxy vitamin D (25(OH)D3) concentration less than 20ng/mL. Vitamin D deficient patients were given 50.000 units of 25(OH)D3 weekly for eight weeks in accordance with the Endocrine Society guidelines. All evaluations were repeated after 2 months of treatment. Patients with Hashimoto’s thyroiditis had significantly lower vitamin D concentrations compared with the controls (9.37±0.69 ng/mL vs 11.95±1.01 ng/mL, p < 0.05, respectively). Thyroid autoantibodies were significantly decreased by vitamin D replacement treatment in patients with euthyroid Hashimoto’s thyroiditis. Also, HDL cholesterol concentrations improved in the euthyroid Hashimoto group after treatment. The mean free thyroxine (fT4) concentrations were 0.89±0.02 ng/dL in patients with Hashimoto’s thyroiditis and 1.07±0.03 ng/dL in the healthy control group (p < 0.001). The mean thyroid volumes were 7.71±0.44 mL in patients with Hashimoto’s thyroiditis and 5.46±0.63 mL in the healthy control group (p < 0.01). Vitamin D deficiency is frequent in Hashimoto’s thyroiditis and treatment of patients with this condition with Vitamin D may slow down the course of development of hypothyroidism and also decrease cardiovascular risks in these patients. Vitamin D measurement and replacement may be critical in these patients.

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Deutsch, Melanie, John Koskinas, Konstatinos Tzannos, Dimitrios Vassilopoulos, Antonis Mailis, George Tolis, and Stephanos Hadziyannis. "Hashimoto Encephalopathy with Pegylated Interferon Alfa-2b and Ribavirin." Annals of Pharmacotherapy 39, no. 10 (October 2005): 1745–48. http://dx.doi.org/10.1345/aph.1g144.

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OBJECTIVE: To report an instance of Hashimoto encephalopathy probably resulting from pegylated interferon alfa-2b and ribavirin. CASE SUMMARY: A 36-year-old woman with a 10-year history of autoimmune thyroiditis presented with symptoms and signs consistent with Hashimoto encephalopathy during therapy with pegylated interferon alfa-2b and ribavirin for chronic hepatitis C. DISCUSSION: Hashimoto encephalopathy is a rare autoimmune condition that occurs in patients with Hashimoto thyroiditis and high titers of antithyroid antibodies. It is characterized by a variety of nonspecific neuropsychiatric symptoms, increased cerebrospinal fluid protein level, and abnormal brain imaging and electroencephalogram. Prompt response to corticosteroids is observed in most cases. As of August 29, 2005, this is the first report of such an association. An objective causality assessment revealed that the Hashimoto encephalopathy was probably caused by the patient's medications. CONCLUSIONS: Hashimoto encephalopathy may rarely be triggered by interferon alfa therapy in susceptible patients.

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Karki, S., and A. Shrestha. "Fine needle aspiration cytology of thyroid and its correlation with serological findings." Journal of Pathology of Nepal 7, no. 1 (March 30, 2017): 1054–58. http://dx.doi.org/10.3126/jpn.v7i1.16777.

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Background: Thyroid autoimmunity can cause several forms of thyroid disorders i.e. Grave’s disease, Hashimoto’s thyroiditis, atrophic autoimmune thyroiditis, post-partum thyroidits etc. Cytological diagnosis may be sometimes difficult is some cases. In such conditions, cytology along with serological tests aid at reaching a correct diagnosis.Hence, this study was undertaken to evaluate the serum level of anti-TPO antibody with respect to serum concentrations of thyroid hormones and its importance in diagnosing autoimmune thyroiditis.Material and Methods: This study was carried out in the department of pathology from June 2013-May 2014. Patients coming to the department of pathology TUTH, Maharajgunj for FNA of thyroid were included. TFT level was noted and anti TPO antibody level was evaluated by CLIA.Results: Ninety-five thyroid FNAC was included in the study, which comprises of 16.8 % males and 83.2% females with a M: F 1: 4.9. Maximum number of cases was seen in the age range 21-30 years (25.3%), mean age being 40.4years. The cytological diagnosis comprised colloid goiter(43.2%), lymphocytic thyroiditis (25.3%), Hashimoto thyroiditis(18.9%). Out of 42 cases of autoimmune thyroiditis diagnosed cytologically, 16 (38%) were hypothyroid, 4 (9.5%) hyperthyroid, 8 (19%) sub-clinical hypothyroid and 14 (33.5%) were euthyroid. The sensitivity and specificity of positive anti TPO in correctly identifying autoimmune thyroiditis was 85.7% and 79.2% respectively. The positive and negative predictive value for the test was 76.5% and 87.5% respectively.Conclusion: Nodular goitre can harbour a certain per cent of autoimmune thyroiditis and in such cases anti TPO antibody level along with cytodiagnosis appears to be helpful.

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Morais, Ana, Marta Resende, and José Pereira. "Tireoidite de Hashimoto e Doença Periodontal: Uma Revisão Narrativa." Acta Médica Portuguesa 29, no. 10 (October 31, 2016): 651. http://dx.doi.org/10.20344/amp.6704.

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Introduction: Currently there is a growing interest in studying systemic conditions with impact on the periodontium. The aim of this article is to determinate if there is a relation between Hashimoto’s thyroiditis and periodontal disease.Material and Methods: Founded on periodontology based on evidence and in the combination of the keywords: ‘Hashimoto disease’; ‘Hypothyroidism’; ‘Periodontal disease’; ‘Systemic Diseases’; a search and evaluation of articles was conducted in Medline, Scopus and Thomson Reuters databases, selecting 30 articles for integral analysis.Results: There have been developed several studies, searching for a better comprehension about the complexity and pathogenesis of periodontal diseases, associated them to multiple systemic conditions. Actually, the relationship that is best described in the literature is the one with rheumatoid arthritis; however, other relations have been pointed, such as Hashimoto’s thyroiditis.Discussion: The identification of multiple etiopathogenic mechanisms common to Hashimoto’s thyroiditis and periodontal disease allow to suspect of a relation between them. Some of these mechanisms include the proliferation of lymphocytes T helper 1 and T helper 17 and their impact on the periodontium, the dysfunction of vascular endothelium in gingival microcirculation and the influence of hypothyroidism on bone metabolism, namely on the alveolar bone.Conclusion: There is biological plausibility to support the establishment of an association between Hashimoto’s thyroiditis and periodontal disease. However, there are not enough studies to support the existence of a causal nexus between these two pathologies, so, in the future, more studies should be conducted to determinate there relation and interaction.

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Subhi, Ohoud, Hans-Juergen Schulten, Nadia Bagatian, Roa'a Al-Dayini, Sajjad Karim, Sherin Bakhashab, Reem Alotibi, et al. "Genetic relationship between Hashimoto`s thyroiditis and papillary thyroid carcinoma with coexisting Hashimoto`s thyroiditis." PLOS ONE 15, no. 6 (June 30, 2020): e0234566. http://dx.doi.org/10.1371/journal.pone.0234566.

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Semiz, H., M. Yalcin, and S. Kobak. "Coexistence of sarcoidosis and Hashimoto thyroiditis." Reumatismo 70, no. 2 (July 6, 2018): 106. http://dx.doi.org/10.4081/reumatismo.2018.1017.

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Sarcoidosis is a chronic, inflammatory disease with unknown cause characterized by non-caseating granuloma formations. It can present with bilateral hilar lymphadenopathy, skin lesions, eye involvement and locomotor system findings. Hashimoto thyroiditis is an organ-specific autoimmune disease characterized by increased autoantibody synthesis. Sarcoidosis can involve different endocrine glands. Thyroid gland involvement may lead to increased thyroid function disorders and autoantibodies. Herein, we report an 80-year-old female patient with sarcoidosis and Hashimoto coexistence.

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Dunne, Christopher, and Francesco De Luca. "Long-Term Follow-Up of a Child with Autoimmune Thyroiditis and Recurrent Hyperthyroidism in the Absence of TSH Receptor Antibodies." Case Reports in Endocrinology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/749576.

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Hashitoxicosis is an initial, transient, hyperthyroid phase that rarely affects patients with Hashimoto thyroiditis. We present here an unusual case of a child with Hashimoto thyroiditis and recurrent hyperthyroidism. A 4 yr 6/12 old male was diagnosed by us with autoimmune subclinical hypothyroidism (normal free T4, slightly elevated TSH, and elevated TG antibody titer). Two years and 6/12 later he experienced increased appetite and poor weight gain; a laboratory evaluation revealed suppressed TSH, elevated free T4, and normal TSI titer. In addition, an I123thyroid uptake was borderline-low. A month later, the free T4 had normalized. After remaining asymptomatic for 3 years, the patient presented again with increased appetite, and he was found with low TSH and high free T4. Within the following 3 months, his free T4 and TSH normalized. At his most recent evaluation, his TSH was normal and the free T4 was borderline-high; the TG antibody titer was still elevated and the TSI titer was negative. To our knowledge, this is the first patient reported with Hashimoto thyroiditis and recurrent hyperthyroidism. This case exemplifies the variability of the manifestations and natural history of Hashimoto thyroiditis and supports the need for a long-term evaluation of patients with autoimmune thyroid disease.

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Włochal, Małgorzata, Marcin A. Kucharski, and Marian Grzymisławski. "The effects of vitamins and trace minerals on chronic autoimmune thyroiditis." Journal of Medical Science 83, no. 2 (June 30, 2014): 167–72. http://dx.doi.org/10.20883/medical.e63.

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Hashimoto’s thyroiditis (HT), also known as chronic lymphocytic thyroiditis is one of the most frequent types of inflammation of the thyroid gland. The prevalence of the overt HT is about 2% but it is believed that Hashimoto thyroiditis is more frequent than expected. Hashimoto’s thyroiditis is characterized by dysfunction of the immune system, which leads to impaired tolerance of own tissues and increased production of autoantibodies against the thyroid cells. Thyroid peroxidase antibodies (anti-TPO), thyroglobulin antibodies (anti-Tg) and/or TSH receptors antibodies are the principal markers of the disease. The essential element of the treatment of HT is the supplementation of L-thyroxine. In Hashimoto’s disease, like in many other autoimmune diseases, researchers attempted to support pharmacological treatment by adequate nutrition. The aim of this paper was to review the existing literature on the levels of antioxidants (vitamin A, C, E, selenium, zinc) and vitamin D in patients with HT, as well as the influence of the nutritional supplementation of the above mentioned elements on the metabolism of the thyroid gland hormones and the level of anti-thyroid peroxidase (anti-TPO) antibodies.

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Hu, Yanjin, Zhi Yao, and Guang Wang. "The Relationship Between the Impairment of Endothelial Function and Thyroid Antibodies in Hashimoto’s Thyroiditis Patients with Euthyroidism." Hormone and Metabolic Research 52, no. 09 (June 15, 2020): 642–46. http://dx.doi.org/10.1055/a-1178-5882.

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AbstractEndothelial dysfunction is the important early step in the development of atherosclerosis. Hypothyroidism caused by Hashimoto’s thyroiditis and other thyroid disease is one of the risk factors of endothelial dysfunction. The present study tried to investigate the endothelial function and its associated factors in Hashimoto thyroiditis with euthyroidism. A total of 95 newly diagnosed Hashimoto’s thyroiditis patients with euthyroidism and 45 healthy controls were studied. Hashimoto’s patients were divided into 3 subgroups namely, single thyroglobulin antibody (TGAb) positive subgroup, single thyroid peroxidase antibody (TPOAb) positive subgroup, and both TGAb and TPOAb positive subgroup. Endothelial function was tested by the reactive hyperemia index (RHI). Hashimoto’s thyroiditis patients had lower RHI than healthy controls (1.73±0.42 vs 1.96±0.51, p<0.05). Hashimoto’s thyroiditis with single TGAb positive patients had higher RHI than single TPOAb positive (1.98±0.57 vs. 1.69±0.33, p<0.05) and TGAB + TPOAb positive patients (1.98±0.57 vs. 1.68±0.42, p<0.05). RHI were negatively associated with total cholesterol (TC, r=−0.215, p<0.05), low density lipoprotein cholesterol (LDL-C, r=−0.268, p<0.05), triglyceride (TG, r=−0.192, p<0.05), and TPOAb (r=−0.288, p<0.05). In the regression analysis, LDL-C (β=−0.146, p<0.05), TG (β=−0.034, p<0.05) and TPOAb (β=−0.001, p<0.05) were independently associated with RHI. Hashimoto’s patients had poor endothelial function. TPOAb levels were negatively associated with endothelial function.

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Diklic, Aleksandar, Vladan Zivaljevic, Ivan Paunovic, Nevena Kalezic, and Svetislav Tatic. "Surgical procedures in patients with thyroid autoimmune disease." Srpski arhiv za celokupno lekarstvo 133, Suppl. 1 (2005): 77–83. http://dx.doi.org/10.2298/sarh05s1077d.

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Autoimmune thyroid disease is a serious medical problem in which various operative procedures are performed. The objective of the study is to explore the type of applied surgical procedures in autoimmune thyroid disease, advantages and disadvantages of various procedures, and criteria they have to meet. This is retrospective clinical study on 1478 patients, operated for Graves? toxic goiter (117 males and 795 females mean age 37.7) and Hashimoto thyroiditis (27 males and 539 females mean age 50.6) from 1995 to April 2005. Cancer in Graves? disease was found in 61 patients (6.7%), papillary in 60 (occult in 53 or 6.6%) and metastatic in 1, Hashimoto thyroiditis and thyroid cancer was found in 141 patients (24.9%), papillary in 116 or 20.5% (occult in 55 or 9.7%), follicular in 2 (0.3%), Hurthle in 11 (1.9%), medullary in 8 (1.4%), anaplastic in 2 (0.3%) and lymphoma in 3 (0.5%). We performed subtotal bilateral lobectomy in 344 (312 in Graves and 32 in Hashimoto), total lobectomy on one side with subtotal on the oposite in 307 (228 in Graves and 79 in Hashimoto); out of them, in 59 patients, the remnant was left in the region of the upper pole which we called atypic lobectomy. The most common procedure, total or near by total thyroidectomy, performed in 719 (371 in Graves and 349 in Hashimoto). One side lobectomy was performed in 103 patients with Hashimoto thyroiditis. Lymph node dissection was performed in 21 ( 1 in Graves and 20 in Hashimoto), in all central, in 10 lateral functional and in 6 mediastinal, in 15 patients with cancer and in 6 patients with benign disease. There was no operative mortality. In Graves? disease, there was postoperative bleeding in 4 (0.4%), wound infection in 2 (0.2%) recurrent pulsy in 18 (2%) and permanent hypoparathyroidism in 13 (1.4%). In Hashimoto thyroiditis, there was postoperative bleeding in 2 (0.4%), recurrent nerve pulsy in 11 (1.9%) and permanent hypoparathyroidism in 6 (1.1%). The most common surgical procedure in autoimmune thyroid disease is total thyroidectomy which is followed by low complication rate in specialised centers. Cancer is more frequent in Hashimoto than in Graves? disease.

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TAGOE, CLEMENT E., ANNA ZEZON, and SAAKSHI KHATTRI. "Rheumatic Manifestations of Autoimmune Thyroid Disease: The Other Autoimmune Disease." Journal of Rheumatology 39, no. 6 (April 15, 2012): 1125–29. http://dx.doi.org/10.3899/jrheum.120022.

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Autoimmune thyroid disease (AITD) is an inflammatory thyroiditis that in some cases is characterized by lymphocytic infiltration of the thyroid gland, also referred to as chronic lymphocytic thyroiditis or Hashimoto thyroiditis. Hashimoto thyroiditis is one of the commonest causes of hypothyroidism. Hypothyroidism has been associated with osteoarthritis (OA) and inflammatory forms of arthritis and with several well defined connective tissue diseases, which in turn can cause arthritis. The presence of arthritis in patients with AITD with normal thyroid function is now being increasingly recognized. There is also considerable evidence to suggest that AITD is highly associated with fibromyalgia syndrome. We review the current literature on the rheumatologic manifestations of AITD and describe the features in its presentation that set it apart from other forms of autoimmune arthritis.

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Fiore, Alixandria A., Wilson B. Pfeiffer, Syed A. A. Rizvi, Anais Cortes, Conrad Ziembinski, Ronald Pham, Stephanie Graves, and Urvesh Patel. "Hashimoto Encephalopathy as a Complication of Autoimmune Thyroiditis." Medical Principles and Practice 28, no. 1 (October 24, 2018): 91–95. http://dx.doi.org/10.1159/000494800.

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Objective: To present a case of Hashimoto encephalopathy as a complication of autoimmune thyroiditis. Clinical Presentation and Intervention: A previously healthy 56-year-old female presented with rapidly progressive cognitive decline and visual hallucinations. Being a diagnosis of exclusion, Hashimoto encephalopathy required an extensive laboratory and diagnostic workup, which was done over the course of a 15-day hospitalization. The patient recovered after initial treatment with intravenous methylprednisolone and was then switched to prednisone p.o. Conclusion: This case report illustrates the importance of awareness for Hashimoto encephalopathy, as it remains one of the few easily treatable and reversible causes of rapid cognitive decline.

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Alp, Hayriye. "Homeopathic Approach in the Case of Secondary Infertility and Anosmia." Obstetrics Gynecology and Reproductive Sciences 5, no. 04 (May 15, 2021): 01–04. http://dx.doi.org/10.31579/2578-8965/073.

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A 38-year-old female patient diagnosed as secondary infertility, anosmia, myoma uteri, Hashimoto thyroiditis with the history of many unsuccessful IVF attempts regained her ability of smell, her thyroid antibody levels normalised, her basal hormones, AMH levels, ovarian follicular development and ovulation improved after 6 months of treatment with homeopathy and nutritional supports. In this case report, it is observed that many gynecological problems accompanied with infertility, anosmia and autoimmune problems like Hashimoto’s thyroiditis can be improved with a holistic approach to individual supportive therapies and homeopathy. It is hoped that this case report can be a reference for future studies in this field.

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Yeh, H. C., W. Futterweit, and P. Gilbert. "Micronodulation: ultrasonographic sign of Hashimoto thyroiditis." Journal of Ultrasound in Medicine 15, no. 12 (December 1996): 813–19. http://dx.doi.org/10.7863/jum.1996.15.12.813.

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Horvatíc Herceg, G., H. TomićBrzac, I. Bračić, D. Herceg, S. Kusačić-Kuna, and D. Dodig. "Thyroid cancer associated with Hashimoto thyroiditis." European Journal of Cancer 37 (April 2001): S109. http://dx.doi.org/10.1016/s0959-8049(01)80889-3.

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39

Uslu, Adam, Hüsnü Gürsu, Mehmet Hamdi Sakarya, Ahmet Nart, and Mustafa Tireli. "Hashimoto Thyroiditis: Results in 22 Patients." Journal of Tepecik Education and Research Hospital 2, no. 3 (1992): 311–14. http://dx.doi.org/10.5222/terh.1992.15163.

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Kandalaft, Osama, and Saima Shikari-Dossaji. "HASHIMOTO THYROIDITIS ASSOCIATED LARGE PERICARDIAL EFFUSION." Journal of the American College of Cardiology 73, no. 9 (March 2019): 2754. http://dx.doi.org/10.1016/s0735-1097(19)33360-1.

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Iyengar, KR, P. Sthaneshwar, JN Hayati, and G. Jayaram. "Hashimoto′s thyroiditis- A Malaysian perspective." Journal of Cytology 24, no. 3 (2007): 119. http://dx.doi.org/10.4103/0970-9371.41898.

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Oppenheimer, Daniel Corey, Ellen Giampoli, Simone Montoya, Swapnil Patel, and Vikram Dogra. "Sonographic Features of Nodular Hashimoto Thyroiditis." Ultrasound Quarterly 32, no. 3 (September 2016): 271–76. http://dx.doi.org/10.1097/ruq.0000000000000228.

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43

Babademez, Mehmet Ali, Kenan Selçuk Tuncay, Murat Zaim, Baran Acar, and Rza Murat Karaşen. "Hashimoto Thyroiditis and Thyroid Gland Anomalies." Journal of Craniofacial Surgery 21, no. 6 (November 2010): 1807–9. http://dx.doi.org/10.1097/scs.0b013e3181f43e32.

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Erden, Sacide, Suna Buyukozturk, Pervin Vural, and Sevgin Değirmencioğlu. "Acute-phase reactans in Hashimoto thyroiditis." International Immunopharmacology 8, no. 13-14 (December 2008): 1863–65. http://dx.doi.org/10.1016/j.intimp.2008.09.007.

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Möst, Johannes, Walter Knapp, and Georg Wick. "Class II antigens in Hashimoto thyroiditis." Clinical Immunology and Immunopathology 41, no. 2 (November 1986): 165–74. http://dx.doi.org/10.1016/0090-1229(86)90100-5.

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Möst, Johannes, and Georg Wick. "Class II antigens in Hashimoto thyroiditis." Clinical Immunology and Immunopathology 41, no. 2 (November 1986): 175–83. http://dx.doi.org/10.1016/0090-1229(86)90101-7.

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Caturegli, P., A. De Remigis, and N. R. Rose. "Hashimoto thyroiditis: Clinical and diagnostic criteria." Autoimmunity Reviews 13, no. 4-5 (April 2014): 391–97. http://dx.doi.org/10.1016/j.autrev.2014.01.007.

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Baumann‑Antczak, Aleksandra, Jerzy Kosowicz, Hanna Zamysłowska, and Marek Ruchała. "Anti‑livin antibodies in Hashimoto thyroiditis." Polish Archives of Internal Medicine 122, no. 11 (October 30, 2012): 527–30. http://dx.doi.org/10.20452/pamw.1454.

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Yoshida, Tomohiko, Akitoshi Nakayama, Ai Tamura, Seiichiro Higuchi, Ikki Sakuma, Hidekazu Nagano, Saulo Ja Felizola, et al. "A Case of Hashimoto’s Thyroiditis with Multiple Drug Resistance and High Expression of Efflux Transporters." Journal of Clinical Endocrinology & Metabolism 105, no. 2 (October 16, 2019): 399–406. http://dx.doi.org/10.1210/clinem/dgz073.

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Abstract Context Hashimoto’s thyroiditis is the most common cause of hypothyroidism. Patients usually respond well to oral synthetic thyroxine (levothyroxine); however, for unknown reasons some individuals present with treatment-resistant Hashimoto thyroiditis. In cases of cancer and certain infectious diseases, the ATP binding cassette (ABC) transporters have been implicated in multidrug resistance, and we hypothesized and investigated a role of ABC transporters in drug-resistant Hashimoto’s thyroiditis. Case Description The patient whose case we report had a history of Hashimoto’s thyroiditis, immune thrombocytopenia, and refractory hypertension, with varying treatment resistance to the oral medications prescribed for each condition. In order to establish or exclude a genetic basis for her illness, we examined the patient’s gene expression profiles using peripheral blood leukocytes, and found that ABCG2/BCRPexpression was significantly high compared with healthy volunteers. Also, the increased daunomycin efflux capacity of our patient’s lymphocytes was successfully inhibited by fumitremorgin C, a specific ABCG2/BCRP inhibitor, and the patient’s level of thyroid-stimulating hormone increased by 248.6% after administration of intact levothyroxine tablets but decreased by 45.1% when tablets were crushed. Her average blood pressure decreased from 166.3/108.5 mmHg to 125.9/78.8 mmHg when switching from intact to crushed losartan tablets. Conclusions High expression and accelerated efflux transporter activity of ABCG2/BCRP in the small intestine are expected to contribute to the ineffectiveness of orally administered intact tablets in cases with treatment-resistant Hashimoto’s thyroiditis, and crushed tablets can be more effective for some of these patients.

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Seo, Hye Mi, Miyeon Kim, Jaeseok Bae, Jo-Heon Kim, Jeong Won Lee, Sang Ah Lee, Gwanpyo Koh, and Dae Ho Lee. "A Case of Painful Hashimoto Thyroiditis that Mimicked Subacute Thyroiditis." Chonnam Medical Journal 48, no. 1 (2012): 69. http://dx.doi.org/10.4068/cmj.2012.48.1.69.

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Journal articles on the topic 'Hashimoto thyroiditis'

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