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Journal articles on the topic 'Healthy siblings'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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1

Mourot, A., T. d'Amato, T. Rochet, et al. "Cerebral investigation of healthy siblings of schizophrenics." European Psychiatry 12, no. 6 (1997): 273–78. http://dx.doi.org/10.1016/s0924-9338(97)84785-2.

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SummaryComputed tomography (CT) studies have demonstrated that lateral ventricular size measured by ventricular brain ratio (VBR), as well as third ventricle width, is statistically enlarged in schizophrenics. Moreover, these cerebral abnormalities differ according to symptomatology evaluated with a positive and negative symptom scale. The aim of this study was to investigate, using CT scans, healthy siblings of schizophrenics, and relate the results to their ill siblings. Nineteen healthy siblings of 12 previously studied schizophrenics underwent CT scans, which were compared to those of thei

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Berge, Jerica M., Allan D. Tate, Amanda Trofholz, Katherine Conger, and Dianne Neumark-Sztainer. "Sibling eating behaviours and parental feeding practices with siblings: similar or different?" Public Health Nutrition 19, no. 13 (2016): 2415–23. http://dx.doi.org/10.1017/s1368980016000860.

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AbstractObjectiveLittle is known about whether siblings have similar or different eating behaviours or whether parents tailor their feeding practices to different siblings. The main objectives of the present study were to examine similarities and differences in child eating behaviours and parental feeding practices with siblings and to determine whether child eating behaviours and parental feeding practices differ depending on sibling concordant (i.e. both siblings overweight or healthy weight) or discordant (i.e. one sibling overweight and one sibling healthy weight) weight status.DesignCross

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Caliendo, Mariarosaria, Valentina Lanzara, Luigi Vetri, et al. "Emotional–Behavioral Disorders in Healthy Siblings of Children with Neurodevelopmental Disorders." Medicina 56, no. 10 (2020): 491. http://dx.doi.org/10.3390/medicina56100491.

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Background and Objectives: Siblings of disabled children are more at risk of developing mental illnesses. More than 50 international studies show that about 8% of children and adolescents suffer from a mental disorder, which is almost always a source of difficulties both at the interpersonal level (in the family and with peers) and at school. Healthy siblings of children with disabilities are one of the groups most at risk for consequences in psychological health and well-being. As some authors suggest, siblings build their idea of “being people”, in terms of character and personality, by cont

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Knytl, P., V. Vorackova, and P. Mohr. "Quality of Lfe in Healthy Siblings of Patients with First Episode of Psychotic Illness and its Predictors." European Psychiatry 41, S1 (2017): S269—S270. http://dx.doi.org/10.1016/j.eurpsy.2017.02.094.

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Families of patients with first episode of psychotic illness are exposed to numerous distress factors related to the care of their relative. It has been shown that these families experience higher levels of anxiety, depression, economic strain, and helplessness. According to the prior studies, long-term psychotic illness can also have negative impact on quality of life (QoL) in healthy siblings [1]. The aim of our study was to assess QoL in siblings of patients with first episode of psychosis and to examine effects of sibling-related and illness-related variables on QoL. Study sample consisted

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Καραμπέλα, Κ., Π. Δ. Χατήρα та Δ. Δαμίγος. "Υγιή αδέρφια παιδιών με καρκίνο". Psychology: the Journal of the Hellenic Psychological Society 18, № 2 (2020): 192. http://dx.doi.org/10.12681/psy_hps.23718.

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The basic purpose of this research is to understand how healthy siblings visualize and represent health and disease as conditions affecting them and others in their family. Furthermore, an attempt was made to evaluate the emotions about health and disease for healthy siblings, as well as to investigate the parameters that interfere with the physicaland emotional life of healthy siblings. Fourteen healthy children participated in the research, aged between 7 and 14, siblings to children diagnosed and receiving treatment for leukemia (acute lymphoblastic &myelogenous leukemia), nephroblastom

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Hazan, Sabine, Kimberly D. Spradling-Reeves, Andreas Papoutsis, and Stephen J. Walker. "Shotgun Metagenomic Sequencing Identifies Dysbiosis in Triplet Sibling with Gastrointestinal Symptoms and ASD." Children 7, no. 12 (2020): 255. http://dx.doi.org/10.3390/children7120255.

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The gut microbiome profile of a child with autism spectrum disorder (ASD) and co-occurring gastrointestinal (GI) symptoms was compared to that of her healthy triplet siblings to determine if she exhibited intestinal dysbiosis. Shotgun metagenomic sequencing was performed in individual fecal samples, and relative microbial abundance and diversity was determined. Microbial diversity was lower in sibling #3, coupled with a higher Bacteroidetes/Firmicutes ratio, a lower relative abundance of Actinobacteria, and an increased relative abundance of Proteobacteria. Our findings are suggestive of gut d

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Tacyildiz, Nurdan, Tugba Karakose, Emel Cabi Unal, Handan Dincaslan, Gulsah Tanyildiz, and Hatice Mine Çakmak. "Evaluation of the quality-of-life (QOL) and socio-demographic characteristics of patients with leukemia and lymphoma: Comparison with sibling and control group." Journal of Clinical Oncology 38, no. 15_suppl (2020): e22524-e22524. http://dx.doi.org/10.1200/jco.2020.38.15_suppl.e22524.

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e22524 Background: Pediatric cancer patients have increased survival rates with intensive and effective treatment methods which causing some chronic health problems and lower quality of life in long term follow up. In our study, patients in their active treatment period compared to patients who have completed their treatment in recent 5 years and more than 5 years groups, besides compared with their own siblings and healthy control group,evaluated in terms of quality of life ( QOL). Methods: This study has been daone in the Department of Pediatric Hematology-Oncology, Ankara University School

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Wandschneider, Britta, Seok-Jun Hong, Boris C. Bernhardt, et al. "Developmental MRI markers cosegregate juvenile patients with myoclonic epilepsy and their healthy siblings." Neurology 93, no. 13 (2019): e1272-e1280. http://dx.doi.org/10.1212/wnl.0000000000008173.

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ObjectiveMRI studies of genetic generalized epilepsies have mainly described group-level changes between patients and healthy controls. To determine the endophenotypic potential of structural MRI in juvenile myoclonic epilepsy (JME), we examined MRI-based cortical morphologic markers in patients and their healthy siblings.MethodsIn this prospective, cross-sectional study, we obtained 3T MRI in patients with JME, siblings, and controls. We mapped sulco-gyral complexity and surface area, morphologic markers of brain development, and cortical thickness. Furthermore, we calculated mean geodesic di

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Idowu, Modupe, Solomon Badejoko, Paul Rowan, and Harinder S. Juneja. "Academic Achievement for Adults with Sickle Cell Disease Compared with Healthy Siblings." Blood 124, no. 21 (2014): 4936. http://dx.doi.org/10.1182/blood.v124.21.4936.4936.

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Abstract Introduction: Sickle cell disease (SCD) is a disabling condition that affects about one out of every 500 African American births in the United States. Children and adolescents with SCD have high rates of school absenteeism and poor academic achievement, yet there are very few studies that have investigated the impact of SCD on adult patients’ academic achievement. Since SCD mostly affects individuals of African descent, the possible risk factors for poor academic achievement and school absenteeism are an intricate combination of disease, demographic, and socio-economic variables. Pote

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Shaddox, L., J. Wiedey, E. Bimstein, et al. "Hyper-responsive Phenotype in Localized Aggressive Periodontitis." Journal of Dental Research 89, no. 2 (2009): 143–48. http://dx.doi.org/10.1177/0022034509353397.

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The ‘hyper-responsive’ trait is an increased inflammatory response upon stimulation of innate immune receptors. Our objective was to determine if a hyper-reactive trait is present in a cohort diagnosed with aggressive periodontitis (LAgP). Peripheral blood was collected from 30 LAgP, 10 healthy unrelated, and 10 healthy sibling participants and stimulated with lipopolysaccharide (LPS) from E. coli and P. gingivalis. Cyto/chemokine response profiles were evaluated and analyzed by ANOVA. Elevated levels of pro-inflammatory cyto/chemokines were detected in E. coli and P. gingivalis LPS-stimulated

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Hyde, Thomas M., Terry E. Goldberg, Michael F. Egan, Marc C. Lener, and Daniel R. Weinberger. "Frontal release signs and cognition in people with schizophrenia, their siblings and healthy controls." British Journal of Psychiatry 191, no. 2 (2007): 120–25. http://dx.doi.org/10.1192/bjp.bp.106.026773.

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BackgroundFrontal release signs, a subset of neurological soft signs, are common in schizophrenia.AimsTo explore the relationship between frontal release signs and neuropsychological tests of frontal lobe function in people with schizophrenia, their siblings and healthy controls.MethodNeuropsychological tests and frontal release signs were measured in a cohort of index cases (n=302), their siblings (n=240) and healthy controls (n=346).ResultsThe mean total score of frontal release signs was 1.5 (s.d. = 1.58) in the schizophrenia group, 0.54 (s.d.=0.92) for siblings and 0.42 (s.d.=0.77) for con

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Marques, Goreti, Beatriz Araújo, and Luís Sá. "The impact of cancer on healthy siblings." Revista Brasileira de Enfermagem 71, no. 4 (2018): 1992–97. http://dx.doi.org/10.1590/0034-7167-2016-0449.

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ABSTRACT Objective: The objective is to identify the variables that influence the impact of cancer on healthy siblings. Method: This is a quantitative, descriptive and correlational study with 83 families of children with cancer. We used the “Questionnaire to Assess the Impact of the Children's Cancer on the Family”, “Social Support Satisfaction Scale”, “Graffar Scale” and “Apgar Scale”. Results: The results show a relation between social support (X²=5.031, gl=1, p=0.025), expenses (t=-2.009, gl=81, p=0.048), disease impact on family structure (t=-3.210, gl=81, p=0.002) and the impact of the d

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Markovic, Jasminka, Jelena Srdanovic-Maras, Valentina Sobot, Svetlana Ivanovic-Kovacevic, and Sladjana Martinovic-Mitrovic. "Some individual psychological characteristics as protective or risk factors for occurrence of conduct disorder." Psihologija 44, no. 2 (2011): 167–85. http://dx.doi.org/10.2298/psi1102167m.

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Our study included 30 pairs of siblings aged 12-18 years; one sibling with and one without conduct disorder in each pair. The aim of the study was to assess individual characteristics of those siblings, i.e. to determine differences in psychological characteristics of the siblings with regard to locus of control, stress coping strategies and frequency and structure of behavioral problems and emotions. The results suggested significant differences in individual characteristics of children with conduct disorder and their healthy siblings. These results mainly confirm previous results of foreign

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Hovi, Tapani, Noora Lindholm, Carita Savolainen, Mirja Stenvik, and Cara Burns. "Evolution of wild-type 1 poliovirus in two healthy siblings excreting the virus over a period of 6 months." Journal of General Virology 85, no. 2 (2004): 369–77. http://dx.doi.org/10.1099/vir.0.19518-0.

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Wild-type 1 poliovirus (wtPV1) strains were isolated from two young healthy brothers shortly after arrival in Finland from Somalia in 1993. Twelve (sibling A) and 18 (sibling B) specimens collected over a period of more than 6 months yielded wtPV1. Partial sequences obtained from the one and two earliest isolates from sibling A and B, respectively, were nearly identical, differing from each other by only one or two nucleotides. Subsequently, the virus evolved separately in both siblings so that maximal differences between strains derived from a given subject peaked at 2·2 % for sibling A, at 1

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Suhaib, Fatima, Asfia Saeed, Hashmat Gul, and Muhammad Kaleem. "Oral assessment of children with autism spectrum disorder in Rawalpindi, Pakistan." Autism 23, no. 1 (2017): 81–86. http://dx.doi.org/10.1177/1362361317730299.

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Comparison of oral health status, dental needs and dietary habits of children with autism and their healthy siblings. In this cross-sectional study, oral hygiene measures, sugar consumption and dental examination of 58 Pakistani children with autism spectrum disorder were assessed and compared to 27 of their siblings without autism spectrum disorder. Questionnaires were distributed to their parents regarding oral hygiene measures, dietary habits and past dental experiences. Clinical examination for the presence of dental caries, periodontal disease or parafunctional habits was carried out and

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Idowu, Modupe, Solomon Badejoko, Paul Rowan, and Harinder S. Juneja. "Occupational History for Forty Adults with Sickle Cell Disease Compared with Healthy Siblings." Blood 124, no. 21 (2014): 4937. http://dx.doi.org/10.1182/blood.v124.21.4937.4937.

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Abstract Introduction: Despite high rates of unemployment and disability in adults with sickle cell disease (SCD), these issues remain understudied. Since SCD mostly affect people of African descent, the potential risk factors for poor job performance and unemployment in adults with SCD are a complex combination of disease, demographic, and socio-economic variables. Recurrent acute vaso-occlusive crises and organ dysfunction related to SCD can limit employment options and interfere with job retention. Potential associates of unemployment and poor job performance in adults with SCD include heal

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van Os, Jim, Lotta-Katrin Pries, Philippe Delespaul, et al. "Replicated evidence that endophenotypic expression of schizophrenia polygenic risk is greater in healthy siblings of patients compared to controls, suggesting gene–environment interaction. The EUGEI study." Psychological Medicine 50, no. 11 (2019): 1884–97. http://dx.doi.org/10.1017/s003329171900196x.

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AbstractBackgroundFirst-degree relatives of patients with psychotic disorder have higher levels of polygenic risk (PRS) for schizophrenia and higher levels of intermediate phenotypes.MethodsWe conducted, using two different samples for discovery (n = 336 controls and 649 siblings of patients with psychotic disorder) and replication (n = 1208 controls and 1106 siblings), an analysis of association between PRS on the one hand and psychopathological and cognitive intermediate phenotypes of schizophrenia on the other in a sample at average genetic risk (healthy controls) and a sample at higher tha

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Eilertsen, Mary-Elizabeth Bradley, Malin Lövgren, Alexandra Eilegård Wallin, and Ulrika Kreicbergs. "Cancer-bereaved siblings' positive and negative memories and experiences of illness and death: A nationwide follow-up." Palliative and Supportive Care 16, no. 4 (2017): 406–13. http://dx.doi.org/10.1017/s1478951517000529.

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AbstractObjective:Our aim was to explore bereaved siblings' positive and negative memories and experiences of their brother's or sister's illness and death.Method:In our nationwide Swedish study, 174 of 240 (73%) bereaved siblings participated, and 70% responded to two open-ended statements, which focused on siblings' positive and negative memories and experiences of illness and death. The data were analyzed using systematic text condensation.Results:The bereaved siblings' responses were categorized into four different themes: (1) endurance versus vulnerability, (2) family cohesion versus fami

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de Wilde, O. M., L. Bour, P. Dingemans, T. Boerée, and D. Linszen. "Antisaccade deficit is present in young first-episode patients with schizophrenia but not in their healthy young siblings." Psychological Medicine 38, no. 6 (2007): 871–75. http://dx.doi.org/10.1017/s0033291707001894.

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BackgroundResults of studies on antisaccade (AS) deficit in relatives of patients with schizophrenia are inconclusive. We hypothesized that AS performance in siblings of patients with schizophrenia is worse than in healthy controls and better than in patients with schizophrenia.MethodWe included 55 first-episode patients with schizophrenia, 28 healthy siblings and 36 healthy controls to evaluate AS performance. Eye movements were measured electromagnetically by the double magnetic induction (DMI) method.ResultsPatients with schizophrenia had a significantly higher error rate than siblings (d=0

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Farmer, Anne, Tanya Harris, Kate Redman, Stephanie Sadler, Arshad Mahmood, and Peter McGuffin. "Cardiff Depression Study." British Journal of Psychiatry 176, no. 2 (2000): 150–55. http://dx.doi.org/10.1192/bjp.176.2.150.

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BackgroundAn excess of both depression and undesirable life events in first-degree relatives of probands with depression as compared with controls has been reported. This association may have reflected a familial factor in common.AimsTo examine the familiality of life events and depression and whether there may be a common familial factor influencing vulnerability to depression and the experiencing of life events.MethodIn a sib-pairdesign, 108 probands with depression and their siblings were compared with 105 healthy controls and their siblings for psychopathology and life events.ResultsThe li

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Knoll, Rebecca L., Kristoffer Forslund, Jens Roat Kultima, et al. "Gut microbiota differs between children with Inflammatory Bowel Disease and healthy siblings in taxonomic and functional composition: a metagenomic analysis." American Journal of Physiology-Gastrointestinal and Liver Physiology 312, no. 4 (2017): G327—G339. http://dx.doi.org/10.1152/ajpgi.00293.2016.

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Current treatment for pediatric inflammatory bowel disease (IBD) patients is often ineffective, with serious side effects. Manipulating the gut microbiota via fecal microbiota transplantation (FMT) is an emerging treatment approach but remains controversial. We aimed to assess the composition of the fecal microbiome through a comparison of pediatric IBD patients to their healthy siblings, evaluating risks and prospects for FMT in this setting. A case-control (sibling) study was conducted analyzing fecal samples of six children with Crohn’s disease (CD), six children with ulcerative colitis (UC

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Russell, K. Brooke, Erin L. Merz, Kathleen Reynolds, Fiona Schulte, and Lianne Tomfohr-Madsen. "Sleep Disturbances in Survivors of Pediatric Acute Lymphoblastic Leukemia and Their Siblings." Journal of Pediatric Psychology 45, no. 7 (2020): 707–16. http://dx.doi.org/10.1093/jpepsy/jsaa043.

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Abstract Objective Sleep disturbances have been identified by patients with cancer as common and distressing; however, conflicting evidence about the prevalence of these outcomes exists for survivors of childhood cancers. Additionally, little is known about how the experience of cancer might impact survivor siblings’ sleep. The current study compared the sleep of survivors of acute lymphoblastic leukemia who were 2–7 years off therapy and their siblings to healthy control/sibling dyads. Methods Participants (survivors, n = 45; survivor siblings, n = 27; controls, n = 45; control siblings, n =

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Callander, M., S. Haghighi, A. M. Landtblom, et al. "Multiple sclerosis immunopathic trait and HLA-DR(2)15 as independent risk factors in multiple sclerosis." Multiple Sclerosis Journal 13, no. 4 (2007): 441–45. http://dx.doi.org/10.1177/1352458506070264.

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We analysed HLA haplotypes in pairs of 78 sporadic multiple sclerosis (MS) patients and 78 healthy siblings. The presence of 2 oligoclonal IgG bands, detected by immunoblotting of the cerebrospinal fluid in healthy siblings, has previously been defined as MS immunopathic trait (MSIT), based on a cut-off derived from healthy unrelated volunteers. The frequency of MSIT was 17.9% (n=14/78 siblings). The HLA-DR(15)2 allelle was present in 21.4% (n=3/14) of the siblings with MSIT, in 40.6% (n =26/64) of the siblings without MSIT, and in 59% (n =46/78) of the patients with clinically-definite (CD) M

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Bediou, Benoit, Fatima Asri, Jerome Brunelin, et al. "Emotion recognition and genetic vulnerability to schizophrenia." British Journal of Psychiatry 191, no. 2 (2007): 126–30. http://dx.doi.org/10.1192/bjp.bp.106.028829.

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BackgroundEpidemiological studies of schizophrenia suggest that this disorder has a substantial genetic component. Cognitive and social abilities, as well as the volumes of brain regions involved in emotion processing, have been found to be distributed along a continuum when comparing patients, siblings and controls, with siblings showing intermediate scores.AimsTo establish whether facial expression recognition is impaired in unaffected siblings of patients.MethodEmotion and gender recognition were evaluated in a three-group pre–post study design in drugnaive patients with first-episode schiz

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Blair, J. C., A. Povall, P. Richardson, and M. Peak. "Parental attitudes to clinical studies in healthy siblings." Archives of Disease in Childhood 100, no. 11 (2015): 1096–97. http://dx.doi.org/10.1136/archdischild-2015-308938.

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Liu, Meijie, Ling-Li Zeng, Hui Shen, Zhening Liu, and Dewen Hu. "Potential risk for healthy siblings to develop schizophrenia." NeuroReport 23, no. 5 (2012): 265–69. http://dx.doi.org/10.1097/wnr.0b013e32834f60a5.

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Shats, Igor K. "Special child family: psychological problems of healthy siblings." Pushkin Leningrad State University Journal, no. 2 (2021): 383–97. http://dx.doi.org/10.35231/18186653_2021_2_383.

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Comabella, M., X. Montalban, A. Horga, et al. "Antiviral immune response in patients with multiple sclerosis and healthy siblings." Multiple Sclerosis Journal 16, no. 3 (2010): 355–58. http://dx.doi.org/10.1177/1352458509357066.

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The objective of this study was to determine the immune responses to candidate viral triggers of multiple sclerosis in patients and healthy siblings raised in the same family household. Virus antigen-specific IgG responses to Epstein—Barr virus-derived gene products as well as to human herpersvirus-6, human cytomegalovirus, and measles virus were evaluated in 25 multiple sclerosis patients and compared with 49 healthy full-siblings. IgG responses to the latent Epstein—Barr virus-encoded nuclear antigen-1 (EBNA1) were selectively increased in individuals with multiple sclerosis compared with th

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Duytschaever, Gwen, Geert Huys, Maarten Bekaert, Linda Boulanger, Kris De Boeck, and Peter Vandamme. "Cross-Sectional and Longitudinal Comparisons of the Predominant Fecal Microbiota Compositions of a Group of Pediatric Patients with Cystic Fibrosis and Their Healthy Siblings." Applied and Environmental Microbiology 77, no. 22 (2011): 8015–24. http://dx.doi.org/10.1128/aem.05933-11.

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ABSTRACTAlthough only poorly documented, it can be assumed that intensive antibiotic treatments of chronic lung infections in patients with cystic fibrosis (CF) also affect the diversity and metabolic functioning of the gastrointestinal microbiota and potentially lead to a state of dysbiosis. A better knowledge of the differences in gut microbiota composition and stability between patients with CF and healthy subjects could lead to optimization of current antibiotic therapies and/or development of add-on therapies. Using conventional culturing and population fingerprinting by denaturing gradie

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Klaassen, R. M. C., M. Heins, L. B. Luteijn, M. van der Gaag, and N. J. M. van Beveren. "Depressive symptoms are associated with (sub)clinical psychotic symptoms in patients with non-affective psychotic disorder, siblings and healthy controls." Psychological Medicine 43, no. 4 (2012): 747–56. http://dx.doi.org/10.1017/s0033291712001572.

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BackgroundDepression is a clinically relevant dimension, associated with both positive and negative symptoms, in patients with schizophrenia. However, in siblings it is unknown whether depression is associated with subclinical positive and negative symptoms.MethodDepressive symptoms and their association with positive and negative symptoms were examined in 813 healthy siblings of patients with a non-affective psychotic disorder, 822 patients and 527 healthy controls. Depressive episodes meeting DSM-IV-TR criteria (lifetime) and depressed mood (lifetime) were assessed with the Comprehensive Ass

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Houshmand, Kathrin, Peter Bräunig, Siegfried Gauggel, Katrin Kliesow, Rahul Sarkar, and Stephanie Krüger. "Emotional vulnerability and cognitive control in patients with bipolar disorder and their healthy siblings: a pilot study." Acta Neuropsychiatrica 22, no. 2 (2010): 54–62. http://dx.doi.org/10.1111/j.1601-5215.2010.00451.x.

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Scheuch K, Bräunig P, Gauggel S, Kliesow K, Sarkar R, Krüger S. Emotional vulnerability and cognitive control in patients with bipolar disorder and their healthy siblings: a pilot study.Objective:There is evidence that, even in remission, patients with bipolar disorder (BD) have deficits in cognitive function and emotional regulation. Siblings of patients with BD are also reported to exhibit minor dysfunction in neuropsychological domains. In this study, we examined the interference of acute mood state with reaction time (RT) and response inhibition in euthymic patients with BD, in their healt

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Bruno, Neuner, Sylvia von Mackensen, Susan Halimeh, et al. "Self- and Parent/Proxies-Reported Health-Related Quality Of Life In Children and Adolescents With Bleeding Disorders: Cross Sectional Investigation and Comparison With Healthy Siblings and Healthy Peers." Blood 122, no. 21 (2013): 4773. http://dx.doi.org/10.1182/blood.v122.21.4773.4773.

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Background Hereditary bleeding disorders (HBD) affect children from birth during their life course. Medical treatment and the overall prognosis of children with HBD have significantly progressed. But even in patients cared for in specialized treatment centers it remains unclear whether survival till adulthood goes along with health-related quality of life [Hr-QoL] comparable to other patients groups respectively comparable to population children. Therefore aim of the present study was to assess self- as well as parents/proxies reported Hr-QoL in children and adolescents with HBD and to compare

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Hurd, Russell C. "Sibling Support Systems in Childhood after a Parent Dies." OMEGA - Journal of Death and Dying 45, no. 4 (2002): 299–320. http://dx.doi.org/10.2190/b92t-lpqr-1rp9-9562.

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The research question addressed was: To what extent did the interactions of childhood sibling subsystems after a parent died facilitate healthy grieving in families where the surviving parent was emotionally unavailable to the children? Utilizing Q methodology, the researcher studied two quartets of adult sisters who, when 3–10 years old, had experienced the deaths of their fathers and the emotional absence of their mothers. Results included a variety of sibling interactions that demonstrated the potential for siblings to contribute to each other's healthy grieving, although the stable presenc

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Reid, H. F. M., D. C. J. Bassett, T. Poon-King, J. B. Zabriskie, and S. E. Read. "Group G streptococci in healthy school-children and in patients with glomerulonephritis in Trinidad." Journal of Hygiene 94, no. 1 (1985): 61–68. http://dx.doi.org/10.1017/s0022172400061131.

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SUMMARYThe group G streptococcus has generally not been considered a prominent pathogen. In a 1982 study of the colonization rate by β-haemoly tic streptococci in apparently healthy children, age 5–11 years, 25 of 69 isolates belonged to group G. This surprisingly high rate of group G colonization (14·3%) led to a retrospective study of school surveys in 1967 which showed that the colonization rate with this organism was 2·3% (range 1·3–3·5%). A review of bacitracin-sensitive streptococcal isolates from hospital admissions of patients with acute glomerulonephritis (AGN), rheumatic fever, and t

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Brunelin, Jerome, Thierry D'amato, Jim van Os, Jean Dalery, Marie-Françoise Suaud-Chagny, and Mohamed Saoud. "Serotonergic response to stress: A protective factor against abnormal dopaminergic reactivity in schizophrenia?" European Psychiatry 22, no. 6 (2007): 362–64. http://dx.doi.org/10.1016/j.eurpsy.2007.01.1219.

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AbstractIn a double-blind-crossover-study, 10 patients with schizophrenia, 10 of their unaffected siblings and 9 healthy controls randomly received metabolic stressor and placebo. A significant HVA plasma elevation in response to stress was found in siblings whose response was intermediate to that of patients and controls. Only siblings additionally displayed an exaggerated 5HIAA response.

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Chan, Carmen W. H., Kai Chow Choi, Wai Tong Chien, et al. "Health Behaviors of Chinese Childhood Cancer Survivors: A Comparison Study with Their Siblings." International Journal of Environmental Research and Public Health 17, no. 17 (2020): 6136. http://dx.doi.org/10.3390/ijerph17176136.

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Purpose: This study aimed to compare health behaviors between the childhood cancer survivors (CCS) and their sibling controls and to examine the pattern of health behaviors of the Hong Kong Chinese CCS and its associations with their health-related quality of life and psychological distress. Methods: A cross-sectional telephone survey was conducted. A total of 614 CCS and 208 sibling controls participated in this study. Patterns of health behaviors including lifestyle behaviors, cancer screening practices, and insurance coverage were compared. Multivariate regression analyses were performed fo

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Brans, Rachel G. H., Neeltje E. M. van Haren, G. Caroline M. van Baal, et al. "Longitudinal MRI study in schizophrenia patients and their healthy siblings." British Journal of Psychiatry 193, no. 5 (2008): 422–23. http://dx.doi.org/10.1192/bjp.bp.107.041467.

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SummaryTo investigate whether genetic and/or disease-related factors are involved in progressive structural brain changes in schizophrenia, magnetic resonance imaging scans with a 5-year scan interval were acquired in patients, their same-gender siblings and matched healthy controls. Structural equation modelling was applied to assess disease and familial effects. Whole brain and cerebral grey matter volumes decreased excessively in patients compared with their siblings and the controls, suggesting that the progressive brain loss in schizophrenia may be related to the disease process.

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Martinson, Ida M., Catherine Gilliss, Elizabeth Bossert, Mark Freeman, and Diane Coughlin Colaizzo. "Impact of childhood cancer on healthy school-age siblings." Cancer Nursing 13, no. 3 (1990): 183–90. http://dx.doi.org/10.1097/00002820-199006000-00008.

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Gulseren, Seref, Mustafa Gurcan, Leyla Gulseren, Fazil Gelal, and Almila Erol. "T2 Hyperintensities in Bipolar Patients and their Healthy Siblings." Archives of Medical Research 37, no. 1 (2006): 79–85. http://dx.doi.org/10.1016/j.arcmed.2005.04.009.

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Barak-Levy, Yael, Esther Goldstein, and Michael Weinstock. "Adjustment characteristics of healthy siblings of children with autism." Journal of Family Studies 16, no. 2 (2010): 155–64. http://dx.doi.org/10.5172/jfs.16.2.155.

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Koning, J. P., D. E. Tenback, R. S. Kahn, M. G. Vollema, W. Cahn, and P. N. van Harten. "Movement disorders are associated with schizotypy in unaffected siblings of patients with non-affective psychosis." Psychological Medicine 41, no. 10 (2011): 2141–47. http://dx.doi.org/10.1017/s0033291711000389.

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BackgroundMovement disorders and schizotypy are both prevalent in unaffected siblings of patients with schizophrenia and both are associated with the risk of developing psychosis or schizophrenia. However, to date there has been no research into the association between these two vulnerability factors in persons with an increased genetic risk profile. We hypothesized that unaffected siblings of patients with non-affective psychosis have more movement disorders and schizotypy than healthy controls and that these co-occur.MethodIn a cross-sectional design we assessed the prevalence and inter-rela

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Liddle, E. B., A. T. Bates, D. Das, et al. "Inefficient cerebral recruitment as a vulnerability marker for schizophrenia." Psychological Medicine 43, no. 1 (2012): 169–82. http://dx.doi.org/10.1017/s0033291712000992.

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BackgroundPatients with schizophrenia and their first-degree relatives exhibit both abnormally diminished and increased neural activation during cognitive tasks. In particular, excessive task-related activity is often observed when tasks are easy, suggesting that inefficient cerebral recruitment may be a marker of vulnerability for schizophrenia. This hypothesis might best be tested using a very easy task, thus avoiding confounding by individual differences in task difficulty.MethodEighteen people with schizophrenia, 18 unaffected full siblings of patients with schizophrenia and 26 healthy con

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Achaval, D. D., K. Buglioni, J. Lopez, et al. "Social functioning and cognition in patients with schizophrenia, their unaffected siblings and healthy controls: impact on quality of life." European Psychiatry 26, S2 (2011): 1398. http://dx.doi.org/10.1016/s0924-9338(11)73103-0.

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BackgroundPatients with schizophrenia show deficits in many cognitive domains and social functioning, social skills, and self management skills in their daily life activities.ObjectiveTo evaluate the relationship between general-social cognition, mental state and social functioning, and impact on quality of life.MethodsTwenty patients with chronic disorder of schizophrenia were evaluated and compared with 20 siblings and 20 healthy controls regarding performance in a series of tests: Cognitive Screening: Word Accentuation Test, MCCB (Matrics Consensus Cognitive Battery) Social Functioning: UPS

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Gromann, P. M., S. S. Shergill, L. de Haan, et al. "Reduced brain reward response during cooperation in first-degree relatives of patients with psychosis: an fMRI study." Psychological Medicine 44, no. 16 (2014): 3445–54. http://dx.doi.org/10.1017/s0033291714000737.

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Background.Psychosis is characterized by a profound lack of trust and disturbed social interactions. Investigating the neural basis of these deficits is difficult because of medication effects but first-degree relatives show qualitatively similar abnormalities to patients with psychosis on various tasks. This study aimed to investigate neural activation in siblings of patients in response to an interactive task. We hypothesized that, compared to controls, siblings would show (i) less basic trust at the beginning of the task and (ii) reduced activation of the brain reward and mentalizing system

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Deja, Grazyna, Przemyslawa Jarosz-Chobot, Joanna Polanska, Urszula Siekiera та Ewa Malecka-Tendera. "Is the Association Between TNF-α-308 A Allele and DMT1 Independent of HLA-DRB1, DQB1 Alleles?" Mediators of Inflammation 2006 (2006): 1–7. http://dx.doi.org/10.1155/mi/2006/19724.

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The aim of the study was to assess chosen factors of genetic susceptibility to DMT1: DRB1, DQB1, and TNF-αpolymorphisms-308 (G/A) in children with DMT1 and their up-to-now healthy siblings. Then we tested whether the association between TNF-αgenes and DMT1 is independent of HLA. 87 diabetic children, their 78 siblings, and 85 persons from healthy control group were followed up. The highest risk of DMT1 was connected with alleles: DRB1*0401 (OR = 3.39; CI: 1.55–7.41), DRB1*0301 (OR = 2.72; CI: 1.48–5.01), DQB1*0201 (OR = 4.04; CI: 2.17–7.52), DQB1*0302 (OR = 5.08; CI: 2.54–10.14), and TNF-α-308

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Landgraf, S., I. Amado, M. C. Bourdel, S. Leonardi, and M. O. Krebs. "Memory-guided saccade abnormalities in schizophrenic patients and their healthy, full biological siblings." Psychological Medicine 38, no. 6 (2007): 861–70. http://dx.doi.org/10.1017/s0033291707001912.

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BackgroundOcular-motor inhibition errors and saccadic hypometria occur at elevated rates in biological relatives of schizophrenic patients. The memory-guided saccade (MS) paradigm requires a subject to inhibit reflexive saccades (RSs) and to programme a delayed saccade towards a remembered target.MethodMS, RS, and central fixation (CF) tasks were administered to 16 patients who met the criteria for DSM-IV schizophrenia, 19 of their psychiatrically healthy siblings, and 18 controls.ResultsPatients and siblings showed elevated MS error rates reflecting a failure to inhibit RSs to a visible targe

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Lu, Chia-Feng, Yu-Te Wu, Shin Teng, et al. "Genetic Predisposition and Disease Expression of Bipolar Disorder Reflected in Shape Changes of the Anterior Limbic Network." Brain Sciences 9, no. 9 (2019): 240. http://dx.doi.org/10.3390/brainsci9090240.

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Bipolar disorder (BD) is a genetically and phenotypically complex psychiatric disease. Although previous studies have suggested that the relatives of BD patients have an increased risk of experiencing affective disturbances, most relatives who have similar genotypes may not manifest the disorder. We aim to identify the neuroimaging alterations—specifically, the cortical folding structures of the anterior limbic network (ALN)—in BD patients and their siblings, compared to healthy controls. The shared alterations in patients and their siblings may indicate the hereditary predisposition of BD, an

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Sarıçiçek, A., N. Zorlu, N. Yalın, et al. "Abnormal white matter integrity as a structural endophenotype for bipolar disorder." Psychological Medicine 46, no. 7 (2016): 1547–58. http://dx.doi.org/10.1017/s0033291716000180.

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BackgroundSeveral lines of evidence suggest that bipolar disorder (BD) is associated with white matter (WM) pathology. Investigation of unaffected first-degree relatives of BD patients may help to distinguish structural biomarkers of genetic risk without the confounding effects of burden of illness, medication or clinical state. In the present study, we applied tract-based spatial statistics to study WM changes in patients with BD, unaffected siblings and controls.MethodA total of 27 euthymic patients with BD type I, 20 unaffected siblings of bipolar patients and 29 healthy controls who did no

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Foley, Sonya F., Matthew Bracher-Smith, Katherine E. Tansey, Judith R. Harrison, Greg D. Parker, and Xavier Caseras. "Fractional anisotropy of the uncinate fasciculus and cingulum in bipolar disorder type I, type II, unaffected siblings and healthy controls." British Journal of Psychiatry 213, no. 3 (2018): 548–54. http://dx.doi.org/10.1192/bjp.2018.101.

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BackgroundFractional anisotropy in the uncinate fasciculus and the cingulum may be biomarkers for bipolar disorder and may even be distinctly affected in different subtypes of bipolar disorder, an area in need of further research.AimsThis study aims to establish if fractional anisotropy in the uncinate fasciculus and cingulum shows differences between healthy controls, patients with bipolar disorder type I (BD-I) and type II (BD-II), and their unaffected siblings.MethodFractional anisotropy measures from the uncinate fasciculus, cingulum body and parahippocampal cingulum were compared with tra

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Peng, Z. W., T. Xu, Q. H. He, et al. "Default network connectivity as a vulnerability marker for obsessive compulsive disorder." Psychological Medicine 44, no. 7 (2013): 1475–84. http://dx.doi.org/10.1017/s0033291713002250.

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BackgroundAberrant functional connectivity within the default network is generally assumed to be involved in the pathophysiology of obsessive compulsive disorder (OCD); however, the genetic risk of default network connectivity in OCD remains largely unknown.MethodHere, we systematically investigated default network connectivity in 15 OCD patients, 15 paired unaffected siblings and 28 healthy controls. We sought to examine the profiles of default network connectivity in OCD patients and their siblings, exploring the correlation between abnormal default network connectivity and genetic risk for

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