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Journal articles on the topic 'Heart Cardiovascular system Heart'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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1

Bennett, M. R. "APOPTOSIS IN THE CARDIOVASCULAR SYSTEM." Heart 87, no. 5 (2002): 480–87. http://dx.doi.org/10.1136/heart.87.5.480.

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2

BROOMHEAD, C. J. "Glucose, insulin, and the cardiovascular system." Heart 85, no. 5 (2001): 495–96. http://dx.doi.org/10.1136/heart.85.5.495.

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3

Curtis, M. J. "The Heart and Cardiovascular System." Cardiovascular Research 26, no. 7 (1992): 720b. http://dx.doi.org/10.1093/cvr/26.7.720b.

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4

Shudo, Yasuhiro, Rhodalene Benjamin-Addy, Tiffany K. Koyano, William Hiesinger, John W. MacArthur, and Y. Joseph Woo. "Donors after circulatory death heart trial." Future Cardiology 17, no. 1 (2021): 11–17. http://dx.doi.org/10.2217/fca-2020-0070.

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Orthotopic heart transplantation is the gold standard treatment for end-stage heart failure. However, the persistent shortage of available donor organs has resulted in an ever-increasing waitlist and longer waiting periods for transplantation. On the contrary, increasing the number of heart transplants by preserving extended criteria donors and donation after circulatory death hearts with the Organ Care System™ (OCS) Heart System has the potential to provide the gold standard, life-saving treatment to patients with end-stage heart failure. The objective of the Donation After Circulatory Death

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5

Ferrari, Alberto U., Alberto Radaelli, and Marco Centola. "Invited Review: Aging and the cardiovascular system." Journal of Applied Physiology 95, no. 6 (2003): 2591–97. http://dx.doi.org/10.1152/japplphysiol.00601.2003.

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Aging is associated with complex and diversified changes of cardiovascular structure and function. The heart becomes slightly hypertrophic and hyporesponsive to sympathetic (but not parasympathetic) stimuli, so that the exercise-induced increases in heart rate and myocardial contractility are blunted in older hearts. The aorta and major elastic arteries become elongated and stiffer, with increased pulse wave velocity, evidence of endothelial dysfunction, and biochemical patterns resembling early atherosclerosis. The arterial baroreflex is sizably altered in aging, but different components are

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6

van den Berg, M. P. "Role of the autonomic nervous system in vagal atrial fibrillation." Heart 89, no. 3 (2003): 333–35. http://dx.doi.org/10.1136/heart.89.3.333.

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7

APPLEBY, M. A. "Angiographic assessment of myocardial perfusion: TIMI myocardial perfusion (TMP) grading system." Heart 86, no. 5 (2001): 485–86. http://dx.doi.org/10.1136/heart.86.5.485.

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8

HAMZA CHERIF, L., S. M. DEBBAL, and F. BEREKSI-REGUIG. "SEGMENTATION OF HEART SOUNDS AND HEART MURMURS." Journal of Mechanics in Medicine and Biology 08, no. 04 (2008): 549–59. http://dx.doi.org/10.1142/s0219519408002759.

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Heart murmurs are often the first signs of pathological changes of the heart valves, and are usually found during auscultation in primary health care. Many pathological conditions of the cardiovascular system cause murmurs and aberrations in heart sounds. Phonocardiography provides the clinician with a complementary tool to record the heart sounds heard during auscultation. The advancement of intracardiac phonocardiography, combined with modern digital processing techniques, has strongly renewed researchers' interest in studying heart sounds and murmurs. This paper presents an algorithm for th

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9

Cilliers, Louise, and François Retief. "The cardiovascular system, as understood in antiquity." Suid-Afrikaanse Tydskrif vir Natuurwetenskap en Tegnologie 26, no. 3 (2007): 205–15. http://dx.doi.org/10.4102/satnt.v26i3.134.

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Cardiovascular concepts in antiquity were primitive up to the early 5th century BC, when Greek philosopher-physicians like Empedocles and Diogenes divorced human physiology from its previous magico-religious base in order to find answers in the natural sciences. The heart was not initially seen as central to the cardiovascular system – blood (containing life-giving pneuma) moved through the body in blood vessels (phlebes) by way of a spontaneous “ebb and flow” motion. Their perceived anatomical vascular models were quite fanciful, but nevertheless accepted by the Hippocratic doctors, who, exce

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10

Zakov, Z. N., and D. Underwood. "The Heart and Cardiovascular System--Scientific Foundations." Cleveland Clinic Journal of Medicine 54, no. 6 (1987): 537. http://dx.doi.org/10.3949/ccjm.54.6.537-a.

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11

Mock, Michael B. "The Heart and Cardiovascular System: Scientific Foundations." Mayo Clinic Proceedings 68, no. 2 (1993): 204–5. http://dx.doi.org/10.1016/s0025-6196(12)60179-7.

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12

Freiman, Alvin. "The Heart and Cardiovascular System: Scientific Foundations." JAMA: The Journal of the American Medical Association 267, no. 20 (1992): 2817. http://dx.doi.org/10.1001/jama.1992.03480200125038.

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13

BERRY, C. "Renin angiotensin system inhibition is associated with reduced free radical concentrations in arteries of patients with coronary heart disease." Heart 86, no. 2 (2001): 217–20. http://dx.doi.org/10.1136/heart.86.2.217.

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14

Tseluyko, V. Y. "COVID-19 and heart." Medicine of Ukraine, no. 6(252) (July 27, 2021): 39–44. http://dx.doi.org/10.37987/1997-9894.2021.6(252).239654.

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The coronavirus pandemic has significantly affected the risk of developing and the clinical course of cardiovascular disease. The article considers in detail the main provisions of the effect of coronavirus on the cardiovascular system and the peculiarities of the management of patients with certain diseases of the cardiovascular system. The injuries can be divided into several variants: myocarditis, myocardial ischemia associated with impaired blood supply, against the background of both atherosclerotic and intact arteries, myocardial changes as a result of endothelium, and, finally, a combin

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15

Arduini, Maurizio. "Central Nervous System and Cardiovascular Diseases." Donald School Journal of Ultrasound in Obstetrics and Gynecology 11, no. 4 (2017): 362–66. http://dx.doi.org/10.5005/jp-journals-10009-1544.

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ABSTRACT Children with congenital heart diseases (CHD) are at increased risk of neurodevelopmental delay (NDD). In fetuses with CHD, blood streaming in the brain may be partially deoxygenated. Signs of cerebrovascular autoregulation comparable to the “brain-sparing effect” in growth-restricted fetuses may be shown in fetuses with CHD depending on the type of heart disease. This effect appears to be predominantly present in fetuses with left-side obstructive lesions (LSOL). During prenatal counseling, it is recommended to mention the possibility of an increased risk of NDD. The severity of this

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16

Fairweather, DS. "Aging of the heart and the cardiovascular system." Reviews in Clinical Gerontology 2, no. 2 (1992): 83–103. http://dx.doi.org/10.1017/s095925980000304x.

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17

McPherson, David D., and Anne M. Gillis. "Book ReviewThe Heart and Cardiovascular System: Scientific foundations." New England Journal of Medicine 316, no. 22 (1987): 1417–18. http://dx.doi.org/10.1056/nejm198705283162219.

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18

Boudoulas, Konstantinos Dean, Jeffrey S. Borer, and Harisios Boudoulas. "Heart Rate, Life Expectancy and the Cardiovascular System: Therapeutic Considerations." Cardiology 132, no. 4 (2015): 199–212. http://dx.doi.org/10.1159/000435947.

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It has long been known that life span is inversely related to resting heart rate in most organisms. This association between heart rate and survival has been attributed to the metabolic rate, which is greater in smaller animals and is directly associated with heart rate. Studies have shown that heart rate is related to survival in apparently healthy individuals and in patients with different underlying cardiovascular diseases. A decrease in heart rate due to therapeutic interventions may result in an increase in survival. However, there are many factors regulating heart rate, and it is quite p

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19

RYDEN, L. "Renin-angiotensin system modulating treatments in the management of cardiovascular disease." Heart 84, no. 90001 (2000): 1i—1. http://dx.doi.org/10.1136/heart.84.suppl_1.i1.

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20

Tian, Rong, Wilson S. Colucci, Zoltan Arany, et al. "Unlocking the Secrets of Mitochondria in the Cardiovascular System." Circulation 140, no. 14 (2019): 1205–16. http://dx.doi.org/10.1161/circulationaha.119.040551.

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Mitochondria have emerged as a central factor in the pathogenesis and progression of heart failure, and other cardiovascular diseases, as well, but no therapies are available to treat mitochondrial dysfunction. The National Heart, Lung, and Blood Institute convened a group of leading experts in heart failure, cardiovascular diseases, and mitochondria research in August 2018. These experts reviewed the current state of science and identified key gaps and opportunities in basic, translational, and clinical research focusing on the potential of mitochondria-based therapeutic strategies in heart f

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21

Chen, J. N., P. Haffter, J. Odenthal, et al. "Mutations affecting the cardiovascular system and other internal organs in zebrafish." Development 123, no. 1 (1996): 293–302. http://dx.doi.org/10.1242/dev.123.1.293.

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In a screen for early developmental mutants of the zebrafish, we have identified mutations specifically affecting the internal organs. We identified 53 mutations affecting the cardiovascular system. Nine of them affect specific landmarks of heart morphogenesis. Mutations in four genes cause a failure in the fusion of the bilateral heart primordia, resulting in cardia bifida. In lonely atrium, no heart venticle is visible and the atrium is directly fused to the outflow tract. In the overlooped mutant, the relative position of the two heart chambers is distorted. The heart is enormously enlarged

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22

Pritzwald-Stegmann, P., J. Passage, T. Müller, et al. "Beating Heart Preservation of Donor Hearts for Transplantation—A Working Heart Perfusion System for Pig Hearts to Extend Organ Preservation Time." Heart, Lung and Circulation 20, no. 1 (2011): 50–51. http://dx.doi.org/10.1016/j.hlc.2010.10.036.

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23

Grad, Cosmin, and Dumitru Zdrenghea. "Heart rate recovery in patients with ischemic heart disease - risk factors." Medicine and Pharmacy Reports 87, no. 4 (2014): 220–25. http://dx.doi.org/10.15386/cjmed-385.

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Background and aim. Central nervous system dysfunction is associated with mortality and morbidity in patients with cardiovascular disease, post-workout recovery and faster heart rate being mediated by the dynamic interaction between the sympathetic nervous system (SNS) and the parasympathetic nervous system (PNS), as components of the autonomic nervous system. Heart rate recovery is the decline in heart rate after exercise. The aim of the study was to determine the influence of certain clinical and paraclinical parameters on heart rate recovery after exercise in patients with ischemic heart di

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24

Habib, Shaista, Wardat us Salam, M. Arif Butt, M. Akram, and F. Smarandache. "A neutrosophic clinical decision-making system for cardiovascular diseases risk analysis." Journal of Intelligent & Fuzzy Systems 39, no. 5 (2020): 7807–29. http://dx.doi.org/10.3233/jifs-201163.

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Cardiovascular diseases are the leading cause of death worldwide. Early diagnosis of heart disease can reduce this large number of deaths so that treatment can be carried out. Many decision-making systems have been developed, but they are too complex for medical professionals. To target these objectives, we develop an explainable neutrosophic clinical decision-making system for the timely diagnose of cardiovascular disease risk. We make our system transparent and easy to understand with the help of explainable artificial intelligence techniques so that medical professionals can easily adopt th

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25

Ortlepp, J. R. "Genetic polymorphisms in the renin-angiotensin-aldosterone system associated with expression of left ventricular hypertrophy in hypertrophic cardiomyopathy: a study of five polymorphic genes in a family with a disease causing mutation in the myosin binding protein C gene." Heart 87, no. 3 (2002): 270–75. http://dx.doi.org/10.1136/heart.87.3.270.

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26

Peiser, F. E., and C. M. L. Steimann. "An artificial heart for calibration of the working rat heart system." Journal of Molecular and Cellular Cardiology 22 (May 1990): S111. http://dx.doi.org/10.1016/0022-2828(90)91857-4.

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27

Qodirova, Gulchekhrakhon Ibrakhimovna. "Gas Analytic Method For Diagnostics Of Ischemic Heart Disease." American Journal of Medical Sciences and Pharmaceutical Research 03, no. 01 (2021): 41–45. http://dx.doi.org/10.37547/tajmspr/volume03issue01-07.

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The enormous damage to human health from cardiovascular disease (CVD) poses significant challenges to both clinicians and public health workers. In most cases, CVDs develop latently; their clinical signs appear at a late stage. Often, patients are unaware of the presence of the disease and die suddenly; it is extremely rare for a doctor to have the opportunity to examine a patient before his cardiovascular system is seriously affected. It follows from this that therapeutic measures alone cannot solve the problem of death from CVD. There is a need to make significant changes in research methods

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28

Redi, Carlo Alberto. "Regenerating the heart - Stem cells and the cardiovascular system." European Journal of Histochemistry 56, no. 2 (2012): 6. http://dx.doi.org/10.4081/ejh.2012.br6.

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29

Olejnickova, Veronika, Marie Novakova, and Ivo Provaznik. "Isolated heart models: cardiovascular system studies and technological advances." Medical & Biological Engineering & Computing 53, no. 7 (2015): 669–78. http://dx.doi.org/10.1007/s11517-015-1270-2.

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30

Fortrat, Jacques-Olivier, Charlotte Baum, Christian Jeanguillaume, and Marc-Antoine Custaud. "Noisy fluctuation of heart rate indicates cardiovascular system instability." European Journal of Applied Physiology 113, no. 9 (2013): 2253–61. http://dx.doi.org/10.1007/s00421-013-2655-2.

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31

Zarins, Christopher K. "The heart and cardiovascular system: Scientific foundations, 2nd edition." Journal of Vascular Surgery 18, no. 3 (1993): 548. http://dx.doi.org/10.1016/0741-5214(93)90313-b.

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32

Raleigh, Juan M. Valle, Stefano Toldo, Anindita Das, Antonio Abbate, and Fadi N. Salloum. "Relaxin’ the Heart." Journal of Cardiovascular Pharmacology and Therapeutics 21, no. 4 (2015): 353–62. http://dx.doi.org/10.1177/1074248415617851.

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The peptide hormone relaxin has traditionally been linked to the maternal adaptation of the cardiovascular system during the first trimester of pregnancy. By promoting nitric oxide formation through different molecular signaling events, relaxin has been proposed as a pleiotropic and cardioprotective hormone in the setting of many cardiovascular diseases. In fact, preclinical studies were able to demonstrate that relaxin promotes vasodilatation and angiogenesis, ameliorates ischemia/reperfusion injury, and regulates extracellular matrix turnover and remodeling. In the RELAX-AHF phase 3 clinical

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33

Esler, Murray, David Kaye, Gavin Lambert, Danielle Esler, and Garry Jennings. "Adrenergic Nervous System in Heart Failure." American Journal of Cardiology 80, no. 11 (1997): 7L—14L. http://dx.doi.org/10.1016/s0002-9149(97)00844-8.

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34

Akki, Ashwin, Ashish Gupta, and Robert G. Weiss. "Magnetic resonance imaging and spectroscopy of the murine cardiovascular system." American Journal of Physiology-Heart and Circulatory Physiology 304, no. 5 (2013): H633—H648. http://dx.doi.org/10.1152/ajpheart.00771.2011.

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Magnetic resonance imaging (MRI) has emerged as a powerful and reliable tool to noninvasively study the cardiovascular system in clinical practice. Because transgenic mouse models have assumed a critical role in cardiovascular research, technological advances in MRI have been extended to mice over the last decade. These have provided critical insights into cardiac and vascular morphology, function, and physiology/pathophysiology in many murine models of heart disease. Furthermore, magnetic resonance spectroscopy (MRS) has allowed the nondestructive study of myocardial metabolism in both isolat

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35

Lin, Li, S. C. Kim, Yin Wang, et al. "HSP60 in heart failure: abnormal distribution and role in cardiac myocyte apoptosis." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 4 (2007): H2238—H2247. http://dx.doi.org/10.1152/ajpheart.00740.2007.

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Heat shock protein (HSP) 60 is a mitochondrial and cytosolic protein. Previously, we reported that HSP60 doubled in end-stage heart failure, even though levels of the protective HSP72 were unchanged. Furthermore, we observed that acute injury in adult cardiac myocytes resulted in movement of HSP60 to the plasma membrane. We hypothesized that the inflammatory state of heart failure would cause translocation of HSP60 to the plasma membrane and that this would provide a pathway for cardiac injury. Two models were used to test this hypothesis: 1) a rat model of heart failure and 2) human explanted

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36

Dawson, T. "Similitude in the cardiovascular system of mammals." Journal of Experimental Biology 204, no. 3 (2001): 395–407. http://dx.doi.org/10.1242/jeb.204.3.395.

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Scaling laws governing the cardiovascular system of mammals are discussed in the present review in a manner emphasizing their experimental basis. Specific attention is given to the well-known experimental laws requiring the rate of oxygen consumption and the heart rate of mammals to vary with body mass raised to the powers 3/4 and −1/4, respectively. This review involves reconsideration and further discussion of the previous work of the writer in which these and other scaling relationships were developed from fundamental considerations. The predicted scaling laws remain unchanged from the earl

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37

Lymperopoulos, Anastasios, Giuseppe Rengo, and Walter J. Koch. "Adrenergic Nervous System in Heart Failure." Circulation Research 113, no. 6 (2013): 739–53. http://dx.doi.org/10.1161/circresaha.113.300308.

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38

Bashir, Muhammad Usman, Muhammad Imran, and Amjad Ali. "ENERGY DRINKS ON CARDIOVASCULAR SYSTEM." Professional Medical Journal 25, no. 11 (2018): 1717–22. http://dx.doi.org/10.29309/tpmj/18.4496.

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Objectives: The objective of this study was to determine the short term effectsof energy drinks on the cardiovascular system and physical performance of healthy adult malemedical students. Study Design: Experimental study. Place and Duration of Study: This studywas conducted at the Physiology departments of CMH Multan Institute of Medical Sciences,Multan and Continental Medical College, Lahore from February 2017 to April 2017. Subjectsand Methods: Sixty eight healthy, adult, male medical students participated in this study. Eachday, heart rates and blood pressures of 3 to 4 students were measu

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39

Kandhal Yazhini P, Gayatri Devi R, and Yuvaraj Babu K. "Knowledge and awareness about high temperature increase the cardiovascular mortality rate." International Journal of Research in Pharmaceutical Sciences 11, SPL3 (2020): 485–91. http://dx.doi.org/10.26452/ijrps.v11ispl3.2971.

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The relation between climatic changes and health is the major concern in the healthcare system; cardiovascular diseases are the leading causes of death worldwide. Both extreme hot and cold temperature can cause deaths associated with cardiovascular diseases. The aim of this study is to analyse the awareness and knowledge on the effect of extreme or high temperature in increased heart-related death rate. A survey with a set of 10 questions was prepared. The questions were administered to 100 participants through google docs by sharing the link. The questionnaire consists of questions related to

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40

Gardner, David G., Songcang Chen, and Denis J. Glenn. "Vitamin D and the heart." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 305, no. 9 (2013): R969—R977. http://dx.doi.org/10.1152/ajpregu.00322.2013.

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Vitamin D receptors (VDR) are found in cells throughout the cardiovascular system. A variety of experimental studies indicate that the liganded VDR may play an important role in controlling cardiac hypertrophy and fibrosis, regulating blood pressure, and suppressing the development of atherosclerosis. Some, but not all, observational studies in humans provide support for these experimental findings, raising the possibility that vitamin D or its analogs might prove useful therapeutically in the prevention or treatment of cardiovascular disease.

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41

Csige, Imre, Dóra Ujvárosy, Zoltán Szabó, et al. "The Impact of Obesity on the Cardiovascular System." Journal of Diabetes Research 2018 (November 4, 2018): 1–12. http://dx.doi.org/10.1155/2018/3407306.

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Obesity is a growing health problem worldwide. It is associated with an increased cardiovascular risk on the one hand of obesity itself and on the other hand of associated medical conditions (hypertension, diabetes, insulin resistance, and sleep apnoea syndrome). Obesity has an important role in atherosclerosis and coronary artery disease. Obesity leads to structural and functional changes of the heart, which causes heart failure. The altered myocardial structure increases the risk of atrial fibrillation and sudden cardiac death. However, obesity also has a protective effect on the clinical ou

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42

Tuka, Vladimír, and Kryštof Slabý. "(Cardiovascular adaptations to competitive sports - athlete's heart)." Cor et Vasa 62, no. 4 (2020): 354–56. http://dx.doi.org/10.33678/cor.2020.039.

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43

Zhumaeva, Zuhra, and Izimkul Manasova. "CHARACTERISTICS OF THE CARDIOVASCULAR SYSTEM DISEASES IN CHILDREN." Doctor's Herald/ Доктор ахборотномаси /Вестник врача 93, no. 1 (2020): 49–51. http://dx.doi.org/10.38095/2181-466x-2020931-49-51.

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The authors conducted a study on the influence of risk factors on the frequency and structure of congenital heart disease in children in the Bukhara region. It was found that the incidence of disease of the cardiovascular system is 6,42 cases for every 1000 births, 6 times more common in children living in rural conditions. Analysis of cities and districts of Bukhara region showed that congenital heart disease is more common in industrialized regions of the region.

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44

Toffoli, Barbara, Bruno Fabris, Giacomo Bartelloni, Fleur Bossi, and Stella Bernardi. "Dyslipidemia and Diabetes Increase the OPG/TRAIL Ratio in the Cardiovascular System." Mediators of Inflammation 2016 (2016): 1–7. http://dx.doi.org/10.1155/2016/6529728.

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Background. Dyslipidemia and diabetes are two of the most well established risk factors for the development of cardiovascular disease (CVD). Both of them usually activate a complex range of pathogenic pathways leading to organ damage. Here we hypothesized that dyslipidemia and diabetes could affect osteoprotegerin (OPG) and TNF-related apoptosis-inducing ligand (TRAIL) expression in the vessels and the heart. Materials and Methods. Gene and protein expression of OPG, TRAIL, and OPG/TRAIL ratio were quantified in the aorta and the hearts of control mice, dyslipidemic mice, and diabetic mice. Re

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45

Popovic, Dejana, Predrag Brkic, Dejan Nesic, Stanimir Stojiljkovic, Ljiljana Scepanovic, and Miodrag Ostojic. "Electrophysiological characteristics of the athlete's heart." Medical review 60, no. 3-4 (2007): 156–59. http://dx.doi.org/10.2298/mpns0704156p.

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Introduction. The athletic heart syndrome is characterized by morphological, functional and electrophysiological alterations as an adaptive response to vigorous physical activity. Athletes heart is predominantly associated with a programmed, intensive training. But as there are different kinds of physical activities, the degree of these changes is highly variable. Electrophysiological characteristics of the athlete's heart. The response of the body to vigorous physical activity is a multiorgan system phenomenon. The integrated functioning of each of these organ systems is very important, but t

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46

Gutkowska, Jolanta, Malgorzata Miszkurka, Bogdan Danalache, Natig Gassanov, Donghao Wang, and Marek Jankowski. "Functional arginine vasopressin system in early heart maturation." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 4 (2007): H2262—H2270. http://dx.doi.org/10.1152/ajpheart.01320.2006.

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Since the neurohypophyseal hormone 8-arginine vasopressin (AVP) is involved in cardiovascular tissue hypertrophy and myocyte differentiation, it is possible that local AVP plays a role in heart maturation. AVP-specific RIA, RT-PCR, and immunoblot measurement of AVP receptors (VR) were used to investigate heart tissues from newborn and adult rats. To test AVP's role in differentiation and specialization into ventricle-like cardiomyocytes, we studied GFP-P19Cl6 stem cells, which express green fluorescence protein (GFP) reporter under transcriptional control of the myosin light chain-2v promoter.

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47

Brilla, C. G. "Renin-angiotensin system mediated mechanisms: cardioreparation and cardioprotection." Heart 84, no. 90001 (2000): 18i—19. http://dx.doi.org/10.1136/heart.84.suppl_1.i18.

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48

Chaturvedi, N. "Modulation of the renin-angiotensin system and retinopathy." Heart 84, no. 90001 (2000): 29i—31. http://dx.doi.org/10.1136/heart.84.suppl_1.i29.

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49

KAWAGUCHI, H., and A. KITABATAKE. "Renin-angiotensin system in failing heart." Journal of Molecular and Cellular Cardiology 27, no. 1 (1995): 201–9. http://dx.doi.org/10.1016/s0022-2828(08)80019-3.

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50

Olshansky, Brian, Hani N. Sabbah, Paul J. Hauptman, and Wilson S. Colucci. "Parasympathetic Nervous System and Heart Failure." Circulation 118, no. 8 (2008): 863–71. http://dx.doi.org/10.1161/circulationaha.107.760405.

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