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Relevant bibliographies by topics / Heart disease; Cardiac metabolism

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Dissertations / Theses

Academic literature on the topic 'Heart disease; Cardiac metabolism'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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Dissertations / Theses on the topic "Heart disease; Cardiac metabolism"

1

Hopkins, James Charles Alex. "Myocardial glycogen, glucose uptake and insulin sensitivity : interrelations and changes with disease." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363766.

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2

Luongo, Timothy Scott. "The Role of Mitochondrial Calcium Exchange in Cardiac Physiology and Disease." Diss., Temple University Libraries, 2017. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/437718.

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Biomedical Sciences<br>Ph.D.<br>The high metabolic demand of the heart makes it essential that an efficient and tightly controlled system be in place to regulate energy production. Contractility is mediated by a variable flux in intracellular calcium (iCa2+), which is proposed to be integrated into mitochondria to regulate cardiac energetics. Moreover, mitochondrial Ca2+ (mCa2+)-overload is known to activate the mitochondrial permeability transition pore (MPTP) and induce cell death. However, the true function of cardiac mCa2+ in physiology remains unknown. Recent studies have reported that th

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3

Steeples, Violetta Rae. "Metabolic modulation through deletion of hypoxia-inducible factor-1α and fumarate hydratase in the heart". Thesis, University of Oxford, 2015. http://ora.ox.ac.uk/objects/uuid:f546ca24-6226-4846-b492-30de26836e94.

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Hypoxia inducible factor-1α (HIF-1α) plays a critical role in the oxygen homeostasis of all metazoans. HIF-1α is a master transcriptional regulator which coordinates the adaptive response to low oxygen tension. Through activation of a plethora of downstream target genes, HIF-1α facilitates oxygenation by promoting angiogenesis and blood vessel dilation, in addition to modulating metabolic pathways to inhibit oxidative phosphorylation and promote glycolytic energy production. Given the critical roles of hypoxia, insufficient blood supply and perturbed energetics in the p

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4

Mahmod, Masliza. "Multiparametric cardiovascular magnetic resonance for the assessment of cardiac function and metabolism in hypertrophy and heart failure." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:ff24c167-e00d-4c6d-9809-82203979ba7a.

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Both hypertrophied and failing hearts are characterised by pathological left ventricular (LV) remodelling, impaired myocardial energy status and alteration in substrate metabolism. Cardiac magnetic resonance imaging (CMR) and magnetic resonance spectroscopy (MRS) are powerful tools in the characterisation of these disease conditions. More recent techniques have allowed assessment of myocardial steatosis using <sup>1</sup>H-MRS and tissue oxygenation using blood oxygen level dependent (BOLD) CMR. In hypertrophy and heart failure, studies on steatosis and the relationship with other parameters s

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5

Dewan, Aaraf. "A Unique Role for Sarcolemmal Membrane Associated Protein Isoform 1 (SLMAP1) as a Regulator of Cardiac Metabolism and Endosomal Recycling." Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/35088.

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Altered glucose metabolism is the underlying factor in many metabolic disorders, including diabetes. A novel protein recently linked to diabetes through animal and clinical studies is Sarcolemmal Membrane Associated Protein (SLMAP) but its role in metabolism remains undefined. The data here reveals a novel role for SLMAP isoform1 in glucose metabolism within the myocardium. Neonatal cardiomyocytes (NCMs) harvested from hearts of transgenic mice expressing SLMAP1, presented with increased glucose uptake, glycolytic rate, as well as glucose transporter 4 (GLUT4) expressions with minimal impact

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6

Wachowiak, Paul Stephen. "Relationships among Cynical Hostility, Metabolic Syndrome, and Cardiac Structure and Function in Multi-Ethnic Post-Myocardial Infarction Patients: A Structural Modeling Approach." Scholarly Repository, 2009. http://scholarlyrepository.miami.edu/oa_dissertations/291.

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BACKGROUND: Risk factors associated with Metabolic Syndrome (MetS) have been implicated in cardiovascular disease (CVD) development and outcomes. Few studies have investigated relationships between psychological variables, MetS factors, and indices of cardiac structure and function (CSF) among healthy individuals in a single conceptual model. No studies to date have analyzed such relationships in patients with CVD. METHODS: The present study examined associations between cynical hostility (CynHo), MetS factors, and CSF in 186 multi-ethnic post-myocardial infarction (MI) patients. Structura

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7

Cutter, Zachary S. "EFFECTS OF THE NA-CL CO-TRANSPORTER (NCC) IN WESTERN DIET INDUCED METABOLIC AND CARDIAC DYSFUNCTION." VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5431.

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Interleukin-18 (IL-18) is a pro-inflammatory cytokine known to be involved in maintaining metabolic homeostasis; however, also capable of inducing cardiac dysfunction. Additionally, IL-18, has been shown to bind to a novel receptor, the Na-Cl Co-transporter (NCC). We hypothesized that NCC mediates IL-18 metabolic and cardiac signaling in mice. Using male C57BL/6J mice, we compared the metabolic and cardiac function changes after at least 8 weeks of high-saturated fat high sugar diet (Western Diet) in NCC knockout (NCCKO), IL-18 knockout (IL-18KO), and wild-type mice. We show that NCCKO mice ha

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8

Dumaresq, Danielle Maia Holanda. "AvaliaÃÃo dos efeitos metabÃlico e oxidativo em cirurgia cardÃaca pediÃtrica: influÃncia da tÃcnica anestÃsica." Universidade Federal do CearÃ, 2006. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=409.

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Instituto Dr. JosÃ Frota<br>A cirurgia cardÃaca pediÃtrica freqÃentemente necessita de circulaÃÃo extracorpÃrea (CEC) durante a intervenÃÃo cirÃrgica. A combinaÃÃo de estresse cirÃrgico e CEC evoca uma resposta inflamatÃria sistÃmica multifatorial, com ativaÃÃo das cascatas humoral e celular. Somado Ã isto, a CEC proporciona perÃodos de isquemia-reperfusÃo, levando Ã condiÃÃes favorÃveis para formaÃÃo de radicais livres e criando uma situaÃÃo de desequilÃbrio que Ã denominada de estresse oxidativo. As espÃcies reativas do oxigÃnio (ERO) formadas durante o perÃodo de isquemia-reperfusÃo, estÃo

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9

Murray, Andrew James. "Control of cardiac metabolism and efficiency." Thesis, University of Oxford, 2003. http://ora.ox.ac.uk/objects/uuid:858cc1f9-7ba0-4999-a1c8-614a950888c2.

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10

Quigley, Gillian Margaret. "Inflammation of the heart in heart disease." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/inflammation-of-the-heart-in-heart-disease(eae19e58-aeb4-4673-924e-1dbd1c831fec).html.

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Heart failure patients have dysfunction of the cardiac conduction system that contributes to a high burden of arrhythmias including atrial fibrillation and sudden cardiac death. Heart failure has been associated with the inflammatory response, but it is unknown if inflammation is playing a role in the remodelling of the cardiac conduction system in heart failure. Inflammation has been shown to be present in the myocardium from failing hearts and it is known to have detrimental effects on cardiac function, inducing fibrosis, remodelling of ion channels and even arrhythmias. However, the effect

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