Academic literature on the topic 'Heart disease in the newborn'

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Journal articles on the topic "Heart disease in the newborn"

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Taksande, Amar, and Sachin Dhamke. "Critical Congenital Heart Disease in Newborns." Pediatric Education and Research 5, no. 2 (2017): 87–95. http://dx.doi.org/10.21088/per.2321.1644.5217.17.

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&NA;. "Missed Heart Disease in the Newborn." Advances in Neonatal Care 9, no. 1 (February 2009): 4. http://dx.doi.org/10.1097/01.anc.0000346085.74744.13.

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El Idrissi Slitine, Nadia, Fatiha Bennaoui, Craig A. Sable, Gerard R. Martin, Lisa A. Hom, Amal Fadel, Soufiane Moussaoui, et al. "Pulse Oximetry and Congenital Heart Disease Screening: Results of the First Pilot Study in Morocco." International Journal of Neonatal Screening 6, no. 3 (June 30, 2020): 53. http://dx.doi.org/10.3390/ijns6030053.

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Congenital heart disease (CHD) is the most common congenital malformation. Diagnosis of critical congenital heart disease (CCHD), the most severe type of congenital heart disease, in a newborn may be difficult. The addition of CCHD screening, using pulse oximetry, to clinical assessment significantly improves the rate of detection. We conducted a pilot study in Morocco on screening neonates for critical congenital heart disease. This study was conducted in the maternity ward of Mohammed VI University Hospital of Marrakesh, Morocco, and included asymptomatic newborns delivered between March 2019 and January 2020. The screening of CCHD was performed by pulse oximetry measuring the pre- and post-ductal saturation. Screening was performed on 8013/10,451 (76.7%) asymptomatic newborns. According to the algorithm, 7998 cases passed the screening test (99.82%), including one inconclusive test that was repeated an hour later and was normal. Fifteen newborns failed the screening test (0.18%): five CCHD, five false positives, and five CHD but non-critical. One false negative case was diagnosed at 2 months of age. Our results encourage us to strengthen screening for CCHD by adding pulse oximetry to the routine newborn screening panel.
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WENDLING, PATRICE. "HHS Calls for Newborn Heart Disease Screening." Pediatric News 45, no. 11 (November 2011): 25. http://dx.doi.org/10.1016/s0031-398x(11)70299-5.

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Al-Ammouri, Iyad, Fares Ayoub, and Rima Dababneh. "Is pre-discharge echocardiography indicated for asymptomatic neonates with a heart murmur? A retrospective analysis." Cardiology in the Young 26, no. 6 (September 10, 2015): 1056–59. http://dx.doi.org/10.1017/s1047951115001705.

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AbstractObjectiveThe purpose of this study was to determine whether a murmur detected on routine pre-discharge examination of asymptomatic newborn children in the first 48 hours of life warrants further investigation with echocardiography.MethodsWe conducted a retrospective review of all echocardiography studies of neonates born at Jordan University Hospital between August, 2007 and June, 2014. Findings on physical examination as well as the indication of the echocardiographic studies were reviewed. We included asymptomatic neonates for whom echocardiographic studies were carried out due to the sole indication of a heart murmur on routine pre-discharge neonatal physical examination.ResultsOf 309 asymptomatic newborns with murmurs on pre-discharge examination, echocardiography revealed 68 (22%) cases of CHD, with 18 (6%) designated as significant heart disease with anticipated intervention during infancy or childhood. The most common abnormality was ventricular septal defect occurring in 36 cases. Critical heart diseases detected included hypoplastic left heart syndrome in two and aortic valve stenosis in four newborns.ConclusionsAlthough most asymptomatic neonates with heart murmurs have normal hearts, a small percentage may have significant heart disease. The decision to refer an asymptomatic newborn with a murmur for echocardiography before discharge from the hospital remains controversial and must be supported by other evidence such as murmur characteristics and local trends in parental compliance with follow-up well-baby visits.
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Seignior, Helen Louise. "What is the significance of hearing a heart murmur during the newborn physical examination?" British Journal of Midwifery 29, no. 3 (March 2, 2021): 158–70. http://dx.doi.org/10.12968/bjom.2021.29.3.158.

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Background Newborn screening for congenital heart disease (CHD) forms part of the newborn physical examination (NPE) in the UK. However, research has shown that up to 50% of cases of CHD can be missed by this examination. The purpose of this literature review is to investigate the significance of hearing an asymptomatic heart murmur at the NPE in relation to the presence of CHD. Methodology A critical literature review was conducted to answer the research question. The findings were discussed using a narrative synthesis approach. Findings There was considerable heterogeneity of results. The prevalence of murmur in newborns ranged between 0.6%–10.7%. Between 13%–67% of those newborns had some form of CHD, although many had clinically insignificant lesions. Between 2%–9% of newborns with murmurs had a form of critical CHD (CCHD). The ability to discern whether a heart murmur relates to pathology or not improves with experience. No single optimal timing for the NPE was identified. Pulse oximetry was shown to increase sensitivity of screening for CCHD. Conclusion There is a significant increase in the prevalence of CHD when a newborn has a heart murmur, hence referral for diagnostic echocardiography is required when a heart murmur is identified. Pulse oximetry is an important adjunct to newborn screening for CCHD. Some newborns affected by CCHD will not present with murmur at the NPE. Therefore, those caring for newborns during the early days must know the signs and symptoms of CHD so that appropriate medical assistance can be sought.
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Sahota, Ravi, Navpreet Kaur, Gurpal Singh, Veena Joshi, Bharti Gahtori, Divya Mehrotra, and Nisha Upadhyay. "Study the universal critical congenital heart disease screening in a peripheral area of Uttarakhand, India." International Journal of Contemporary Pediatrics 7, no. 9 (August 25, 2020): 1849. http://dx.doi.org/10.18203/2349-3291.ijcp20203641.

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Background: Congenital heart disease (CHD) is the most frequently occurring congenital disorder, responsible for 28% of all congenital birth defects. The birth prevalence of CHD is reported to be 8-12/1000 live births. Considering a rate of 9/1000, about 1.35 million babies are born with CHD each year globally. Objective of research work to study the prevalence of CHD among newborn and its types.Methods: This cross-sectional study was carried among 34 cases of CHD/5126 newborn screened at 4 birthing places in Kashipur a small town in Uttrakhand at pediatrics department of Sahota Super-specialty hospital, Kashipur, Uttarakhand. Screening program between 22 August 2014, and March 30, 2019. All newborns, including preterm babies, delivered in these facilities were eligible for inclusion in this study.Results: Present study found the prevalence of CHD was 0.7 per 1000 children (34/5126). Around 26.5% participants have cyanotic CHD and 73.5% have acynotic CHD. Almost 44%, 28%, 20%, 4% and 4% participants of acynotic congenital heart diseases have VSD, ASD, PDA, AVSD and valvular PS respectively and 55.6%, 22.2%, 11.1% and 11.1% participants of cynotic congenital heart diseases have TOF, DORV/VSD, dTGA/VSD and tricuspid atresia respectively.Conclusions: Screening for congenital heart disease should be included as a part of newborn assessment as it is a common congenital problem. Early identification influences outcome. Barriers in implementation of the screening programmes in resource limited setting is a challenging feature. This study can provide observed data that can help in policy making in the health sector.
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Nartsissova, G. P., O. A. Lenko, Yu S. Sinelnikov, and I. I. Volkova. "Staged and consistent perinatal diagnostics of congenital heart disease." Patologiya krovoobrashcheniya i kardiokhirurgiya 17, no. 2 (October 10, 2015): 75. http://dx.doi.org/10.21688/1681-3472-2013-4-75-80.

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The principles of organization of perinatal diagnostics of congenital heart disease (CHD) carried out by the advisory group of the cardiac center are presented. 1 601 pregnant women at the age from 18 to 40 years and 20 to 38-39 weeks of gestation were examined. 657 (41%) of them had risk factors of CHD development, while 336 (21%) were diagnosed with congenital heart disease. Only 19.1% of pregnant women with CHD fetus had risk factors, with no such relation observed in other women. Surgical repair was performed in 56.4% of cases during a newborn period. A three-stage system of perinatal diagnostics of congenital heart disease performed by the cardiac center's experts over prenatal and neonatal periods allows providing consistency and efficiency of rendering assistance to newborns with congenital heart disease.
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Belanger, Michael, Luke Tan, and Carin Wittnich. "Does young age really put the heart at risk?" Canadian Journal of Physiology and Pharmacology 95, no. 10 (October 2017): 1177–82. http://dx.doi.org/10.1139/cjpp-2017-0072.

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Despite significant advances in the management and treatment of heart disease in children, there continue to be patients who have worse outcomes than might be expected. A number of risk factors that could be responsible have been identified. Evidence-based findings will be reviewed, including whether young age and (or) reduced body weight exacerbate these responses. For example, newborn children undergoing congenital cardiac surgery are known to have worse outcomes than older children. Evidence exists that newborn hearts do not tolerate ischemia as well as adult hearts, developing irreversible injury sooner and exhibiting at-risk metabolic profiles. As well, in response to the administration of heparin, elevations in free fatty acids occur during congenital heart surgery in children, which can have detrimental effects on the heart. Furthermore, myocardial energetic state has also been suggested to impact outcomes. Unfavourable energetic profiles were correlated to lower body weights in the same age healthy newborn piglet model. Newborn children suffering from congenital heart disease, with lower body weights, also had lower myocardial energetic state and this correlated with longer postoperative ventilatory support as well as a trend to longer intensive care unit stay. These findings imply that unfavourable myocardial metabolic profiles could contribute to postoperative complications.
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Badawi, Deborah, Johnna Watson, Steven Maschke, and Lawrence Reid. "First-Year Outcomes of Critical Congenital Heart Disease Screening in Maryland." Global Pediatric Health 6 (January 2019): 2333794X1986822. http://dx.doi.org/10.1177/2333794x19868226.

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Objectives. Newborn screening for critical congenital heart disease (CCHD) was added to the Recommended Uniform Screening Panel in 2011, and states have been gradually adding pulse oximetry as point-of-care screening to panels. Few data are available on the effectiveness of pulse oximetry as a mandated screening. This study describes outcomes of the first year of screening in Maryland. Methods. A web-based data collection tool for screening results and outcomes, eScreener Plus, was utilized. Data collected from the start of screening from September 1, 2012, to December 31, 2013, were analyzed. Well-baby nursery data were evaluated separately from neonatal intensive care unit (NICU) data to determine whether setting influenced effectiveness. Results. In the first 15 months of newborn screening for CCHD in Maryland, 4 asymptomatic infants were diagnosed with a critical cardiac condition by newborn screening. Eleven infants passed but were later identified with a primary or secondary target condition. Seventy-one percent of infants with CCHD were identified prenatally or by clinical signs and symptoms. Pulse oximetry screening for CCHD had a specificity of more than 99% in both the well-baby nursery and the NICU. Sensitivity in the well-baby nursery was 10% and 60% in the NICU. Conclusion. Further investigation and interpretation of specific protocols that were used and outcomes of screening is needed for continued refinement of the well-baby algorithm and NICU protocol development. Pulse oximetry screening in newborns provides valuable clinical information, but many infants with CCHD are still not identified with current protocols.
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Dissertations / Theses on the topic "Heart disease in the newborn"

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Coe, J. Y. "Alteration of neonatal pulmonary vascular tone." Thesis, University of Newcastle Upon Tyne, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381381.

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Tseng, Stephanie Y. "Altered Erythropoiesis in Newborns with Congenital Heart Disease." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1592170832331138.

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Bueno, Mariana. "Dor e analgesia em recém-nascidos submetidos a cirurgias cardíacas." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/7/7132/tde-03102006-111524/.

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A dor ocasiona uma série de alterações sistêmicas que afetam o desenvolvimento do recém-nascido (RN), em particular, os submetidos à cirurgia cardíaca, além de acarretar alterações hemodinâmicas que podem comprometer sua sobrevivência. O objetivo do estudo foi caracterizar a população de neonatos submetidos a cirurgias cardíacas e descrever as práticas de controle da dor implementadas no primeiro pós-operatório. Estudo descritivo, transversal realizado na Sociedade Hospital Samaritano de São Paulo. Os dados foram obtidos de prontuários médicos de RN submetidos à cirurgia cardíaca, internados entre julho de 2001 e dezembro de 2005. Os registros sobre a avaliação da dor e intervenção medicamentosa para o alívio da dor no primeiro pós-operatório foram analisados. Dentre quarenta e dois RN submetidos a cirurgias cardíacas no período estudado, 30 atenderam aos critérios de inclusão e constituíram a casuística do estudo. A maioria dos RN nasceu a termo, pesava 2.500g ou mais na data do procedimento cirúrgico e foi submetida à cirurgia na primeira semana de vida. Foram identificados 24 diferentes diagnósticos de cardiopatias congênitas e 14 tipos de intervenções cirúrgicas, corretivas ou paliativas, no grupo estudado. Dentre 30 prontuários, foram identificados registros de avaliação de dor em 24 (80,0%). A avaliação por meio da escala NIPS foi anotada em 17 (56,7%) prontuários e em 15 (50,0%) verificou-se registro de comportamentos e alterações nos parâmetros fisiológicos indicativos de dor. Dos RN avaliados, 17 (56,7%) apresentaram registro de ocorrência de dor no primeiro pós-operatório. A maioria, 29 (96,7%), recebeu analgesia farmacológica: 24 (80,0%) RN receberam citrato de fentanil contínuo, 5 (16,7%) RN, dipirona intermitente, 5 (16,7%) RN, citrato de fentanil contínuo e dipirona intermitente, 1 (3,3%) RN recebeu citrato de fentanil contínuo e morfina intermitente. As doses administradas variaram amplamente. Foram também utilizados sedativos associados aos analgésicos: 12 (40,0%) RN receberam midazolan contínuo, 5 (16,7%) RN, midazolan intermitente, 1 (3,3%), besilato de cisatracúrio contínuo, 1 (3,3%) cloridrato de clorpromazina contínuo e 1 (3,3%) propofol intermitente Os resultados evidenciam não haver uniformidade e padronização na avaliação da dor pós-operatória e nos tipos e doses de fármacos utilizados para o controle da dor pós-operatória dos RN submetidos a cirurgias cardíacas no serviço estudado.
Pain causes several systemic alterations that affect the newborn’s development, specially the ones who underwent cardiac surgery. Besides, hemodynamic alterations related to pain can put their lives into risk. The aim of the study was to characterize pain control practices in newborns who underwent cardiac surgery. This is a cross-sectional and descriptive study performed in Sociedade Hospital Samaritano of São Paulo. The data were collected from the medical reports of the newborns who underwent cardiac surgery from July 2001 to December 2005. The records related to pain assessment and pharmacological treatment in the first post-operative day were analyzed. From 42 newborns who underwent cardiac surgery during the period studied, 30 of them were in accordance with the criteria of inclusion. Most of the neonates were term and their weights were 2.500 grams or more on the date of the surgery that was mostly performed in the first week of life. In the studied group, it was identified 24 different diagnoses of congenital heart deffects and 14 types of corrective or palliative surgical interventions. From the 30 medical reports, records of pain assessment were found in 24 of them (80.0%). The evaluations taken by using the NIPS were written in 17 medical reports (56.7%) and in 15 of them (50.0%) it was verified records of behavior and physiological parameters alterations that indicate pain. From the newborns evaluated, 17 (56.7%) presented records of pain in the first day post-surgery. 29 neonates (96.7%) received analgesics, as it follows: continuous fentanyl citrate, 24 newborns (80.0%); intermittent dipyrone, 5 (16.7%); continuous fentanyl citrate and intermittent dipyrone, 5 (16.7%) and fentanyl citrate and intermittent morphine, 1 (3.3%). The doses given to the neonates were very different. 12 (40.0%) newborns received the sedative midazolam by continuous infusion with analgesics. 5 (16.7%) neonates received intermittent midazolam, 1 (3.3%) received continuous cisatracurium besylate, 1 (3.3%) received continuous chlorpromazine hydrochloride and 1 (3.3%) received intermittent propofol. The results showed that there is no pattern and uniformity of pain assessment after surgery, neither of the types and doses of drugs used for pain control of the newborns who underwent cardiac surgery in the service that was studied.
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Ceballos, Andres. "A multiscale model of the neonatal circulatory system following Hybrid Norwood palliation." Master's thesis, University of Central Florida, 2011. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/4866.

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A system of 32 first-order differential equations is formulated and solved for the LP model using a fourth-order adaptive Runge-Kutta solver. The output pressure and flow waveforms obtained from the LP model are imposed as boundary conditions on the CFD model. Coupling of the two models is done through an iterative process where the parameters in the LP model are adjusted to match the CFD solution. The CFD model domain is a representative HLHS anatomy of an infant after undergoing the Hybrid Norwood procedure and is comprised of the neo-aorta, pulmonary roots, aortic arch with branching arteries, and pulmonary arteries. The flow field is solved over several cardiac cycles using an implicit-unsteady RANS equation solver with the k-epsilon turbulence model.; Hypoplastic left heart syndrome (HLHS) is a complex cardiac malformation in neonates suffering from congenital heart disease and occurs in nearly 1 per 5000 births. HLHS is uniformly fatal within the first hours or days after birth as the severely malformed anatomies of the left ventricle, mitral and aortic valves, and ascending aorta are not compatible with life. The regularly implemented treatment, the Norwood operation, is a complex open heart procedure that attempts to establish univentricular circulation by removing the atrial septum (communicating the right and left ventricle), reconstructing the malformed aortic arch, and connecting the main pulmonary artery into the reconstructed arch to allow direct perfusion from the right ventricle into the systemic circulation. A relatively new treatment being utilized, the Hybrid Norwood procedure, involves a less invasive strategy to establish univentricular circulation that avoids a cardiopulmonary bypass (heart-lung machine), deliberate cardiac arrest, and circulatory arrest of the patient during the procedure. The resulting systemic-pulmonary circulation is unconventional; blood is pumped simultaneously and in parallel to the systemic and pulmonary arteries after the procedure. Cardiac surgeons are deeply interested in understanding the global and local hemodynamics of this anatomical configuration. To this end, a multiscale model of the entire circulatory system was developed utilizing an electrical lumped parameter model for the peripheral or distal circulation coupled with a 3D Computational Fluid Dynamics (CFD) model to understand the local hemodynamics. The lumped parameter (LP) model is mainly a closed loop circuit comprised of RLC compartments that model cardiac function as well as the viscous drag, flow inertia, and compliance of the different arterial and venous beds in the body.
ID: 030423155; System requirements: World Wide Web browser and PDF reader.; Mode of access: World Wide Web.; Thesis (M.S.)--University of Central Florida, 2011.; Includes bibliographical references (p. 59-61).
M.S.
Masters
Mechanical, Materials, and Aerospace Engineering
Engineering and Computer Science
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Holt, Jim. "Heart Disease." Digital Commons @ East Tennessee State University, 2003. https://dc.etsu.edu/etsu-works/6509.

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Quigley, Gillian Margaret. "Inflammation of the heart in heart disease." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/inflammation-of-the-heart-in-heart-disease(eae19e58-aeb4-4673-924e-1dbd1c831fec).html.

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Heart failure patients have dysfunction of the cardiac conduction system that contributes to a high burden of arrhythmias including atrial fibrillation and sudden cardiac death. Heart failure has been associated with the inflammatory response, but it is unknown if inflammation is playing a role in the remodelling of the cardiac conduction system in heart failure. Inflammation has been shown to be present in the myocardium from failing hearts and it is known to have detrimental effects on cardiac function, inducing fibrosis, remodelling of ion channels and even arrhythmias. However, the effect of inflammation on the cardiac conduction system has not been investigated. The aims of this study were to determine if there is an increase of pro-inflammatory cytokines and inflammatory cells in the cardiac conduction system in heart failure. In addition, to identify if there is possible inflammation-associated fibrosis and apoptosis in the cardiac conduction system in heart failure. To test these aims, three models of heart failure were used: a rat model of pulmonary arterial hypertension, a rabbit model of congestive heart failure and a rat model of myocardial infarction. In the rat model of pulmonary arterial hypertension there was a bradycardia, a prolongation of the QT interval, and an increase in the atrioventricular and ventricular refractory periods, suggesting electrical remodelling in these animals. The rats with pulmonary arterial hypertension displayed an increase in pro-inflammatory cytokines such as interleukins 1β and TGFβ in the right side of the heart, including the sinoatrial node and right Purkinje fibres of the cardiac conduction system. In addition, in these areas, there was an increase in components of the extracellular matrix, including fibronectin, collagen I and vimentin. Histology revealed regions of non-myocyte nuclei, only in the right ventricle of the rats with pulmonary arterial hypertension. Immunohistochemistry demonstrated patches of CD68 and vimentin expression (markers for macrophages and fibroblasts, respectively) in the right side of the heart in these animals. TUNEL staining also revealed an increase in apoptosis in the right side of the heart. In the rabbit model of congestive heart failure, the region most affected by inflammation was the right atrium, while few changes were measured in the ventricles or cardiac conduction system. Although these results are surprising, it is suggested that the atria could be more sensitive to the physical stretch produced in this model. In the rat model of myocardial infarction, there were regions of non-myocyte nuclei in the border zone. This region also had increases in pro-inflammatory and fibrosis markers. In conclusion, this work has presented the novel finding that there can be inflammation in the cardiac conduction system in heart failure. This could be contributing to the arrhythmias seen in heart failure patients. This could possibly lead the way to anti-inflammatories as a possible novel therapeutic for heart failure patients.
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Misner, Scottie. "Heart Disease in Women." College of Agriculture and Life Sciences, University of Arizona (Tucson, AZ), 1999. http://hdl.handle.net/10150/146457.

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Heart disease, or cardiovascular disease (CVD), includes stroke and coronary heart disease, which are serious disorders of the heart and blood vessel system. Heart disease is the leading cause of illness and death in North American women. This publication addresses what kind of heart diseases affect women and how to live the heart-healthy way.
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Rix, Louise Katharine. "Biochemistry of heart disease." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334889.

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Makinde, Abiola Olufemi. "Maturation of fatty acid oxidation in the newborn rabbit heart." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape9/PQDD_0004/NQ39561.pdf.

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Brown, Jessie Ann. "RUNX2 in Embryonic Heart Development and Heart Disease." Thesis, The University of Arizona, 2011. http://hdl.handle.net/10150/144250.

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Books on the topic "Heart disease in the newborn"

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N, Benson Leland, and Smallhorn Jeffrey F, eds. Neonatal heart disease. London: Springer-Verlag, 1992.

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Manual of neonatal and paediatric heart disease. London: Whurr, 2002.

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Lynn, Mahony, and Teitel David F, eds. Neonatal cardiology. 2nd ed. New York: McGraw-Hill, 2010.

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Summer of the heart: Saving baby Alexandre. Toronto: Macfarlane, Walter & Ross, 2003.

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Sharykin, A. S. Perinatalʹnai͡a kardiologii͡a: Rukovodstvo dli͡a pediatrov, akusherov, neonatologov. Moskva: Volshebnyĭ fonarʹ, 2007.

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Mothers, babies and disease in later life. London: BMJ Pub. Group, 1994.

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M, Monset-Couchard, and Institut national de la santé et de la recherche médicale (France), eds. Cardiovascular and respiratory physiology in the fetus and neonate =: Physiologie cardiovasculaire et respiratoire du foetus et du nouveau-ńe : proceedings of the international conference held in Paris (France) 20-29 January 1985. London: Libbey, 1986.

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Special heart: A journey of faith, hope, courage and love. New York: Center Street, 2014.

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Central Health Services Council. Standing Medical Advisory Committee. Haemolytic disease of the newborn. London: Department of health and Social Security, 1996.

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Heart disease. Santa Barbara, Calif: Greenwood, an imprint of ABC-CLIO, 2012.

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Book chapters on the topic "Heart disease in the newborn"

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Ralphe, J. Carter, and Thomas D. Scholz. "Cardiac Metabolic Protection for the Newborn Heart." In Cardiac Energy Metabolism in Health and Disease, 265–76. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1227-8_17.

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Teitel, David, Daniel Sidi, Dan Bernstein, Michael A. Heymann, and Abraham M. Rudolph. "Experimental Cyanotic Heart Disease in the Newborn Lamb." In Pediatric Cardiology, 842–46. New York, NY: Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4613-8598-1_227.

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Makinde, A.-Olufemi, Paul F. Kantor, and Gary D. Lopaschuk. "Maturation of fatty acid and carbohydrate metabolism in the newborn heart." In Molecular and Cellular Effects of Nutrition on Disease Processes, 49–56. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5763-0_6.

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Nakanishi, Hidehiko, Shunichi Morikawa, Shuji Kitahara, Asuka Yoshii, Atsushi Uchiyama, Satoshi Kusuda, and Taichi Ezaki. "Morphological Characterization of Pulmonary Microvascular Disease in Bronchopulmonary Dysplasia Caused by Hyperoxia in Newborn Mice." In Molecular Mechanism of Congenital Heart Disease and Pulmonary Hypertension, 91–93. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-1185-1_11.

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Strife, Janet L., and Veronica Donoghue. "Imaging of the Chest in the Newborn and Young Infant." In Diseases of the Heart and Chest, Including Breast 2011–2014, 47–54. Milano: Springer Milan, 2011. http://dx.doi.org/10.1007/978-88-470-1938-6_7.

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Harinck, Eric, Gertjan van Mill, and André J. Moulaert. "Ultrasonic Assessment of Cyanotic Heart Disease in Newborns: The Current State of the Art." In Pediatric Cardiology, 76–79. New York, NY: Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4613-8598-1_18.

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Suri, Vanita, and Pooja Sikka. "Heart Disease." In Labour Room Emergencies, 77–83. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-10-4953-8_8.

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Wallace, Rodrick, and Deborah Wallace. "Heart Disease." In Gene Expression and Its Discontents, 87–105. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-48078-7_8.

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Harr, Jeffrey N., Philip F. Stahel, Phillip D. Levy, Antoine Vieillard-Baron, Yang Xue, Muhammad N. Iqbal, Jeffrey Chan, et al. "Heart Disease." In Encyclopedia of Intensive Care Medicine, 1013. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1669.

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Orbell, Sheina, Havah Schneider, Sabrina Esbitt, Jeffrey S. Gonzalez, Jeffrey S. Gonzalez, Erica Shreck, Abigail Batchelder, et al. "Heart Disease." In Encyclopedia of Behavioral Medicine, 939. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_1266.

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Conference papers on the topic "Heart disease in the newborn"

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Rugonyi, Sandra, and Kent Thornburg. "Modeling the Effect of Hemodynamics on Cardiac Growth During Embryonic Development." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13171.

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Congenital heart disease (CHD) affects about 1% of newborn babies in the US, and is the leading cause of non-infectious death in children. Abnormal blood flow dynamics during early development can lead to CHD. Although the effect of hemodynamic conditions on cardiac development — even under normal conditions — has been widely accepted, the mechanisms by which blood flow influences cardiac cell responses are only starting to emerge. Mathematical models of cardiac growth could then help elucidate key aspects of cardiac development.
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Kaya, Ersin, Bulent Oran, and Ahmet Arslan. "A rough sets approach for diagnostic M-mode evaluation in newborn with congenital heart diseases." In 2010 3rd International Conference on Human System Interactions (HSI). IEEE, 2010. http://dx.doi.org/10.1109/hsi.2010.5514578.

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Kennington, Jeffrey R., Steven Frankel, Jun Chen, Mark D. Rodefeld, and Guruprasad A. Giridharan. "Design of a Novel Cavopulmonary Assist Device for Fontan Procedures: CFD, PIV, and Hydraulic Testing." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19558.

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Single ventricle heart disease is the leading cause of death for birth defects in children under one years of age [1]. The current surgical procedure requires the use of a shunt for the first stage of the surgery. The following surgeries remove the shunt but cannot be performed on a newborn due to higher lung resistance during the first weeks of life. The overall surgical process, known as the Fontan procedure, results in a reconstructed anatomy where the left and right pulmonary arteries are sutured to the superior and inferior vena cavae (SVC/IVC), hence bypassing the right heart. This anatomy is called a total cavopulmonary connection or TCPC.
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"The prevalence of congenital heart disease in newborns hospitalized in neonatal department." In International Conference on Medicine, Public Health and Biological Sciences. CASRP Publishing Company, Ltd. Uk, 2016. http://dx.doi.org/10.18869/mphbs.2016.208.

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Davis, Natalie L., Elena Donald, and Maura Heffernan. "Can We Use Critical Congenital Heart Disease (cchd) Screening to Replace Car Seat Testing in Newborns?" In Selection of Abstracts From NCE 2016. American Academy of Pediatrics, 2018. http://dx.doi.org/10.1542/peds.141.1_meetingabstract.505.

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Jakab, A., E. Meuwli, M. von Rhein, R. Tuura, I. Scheer, B. Latal, and W. Knirsch. "Longitudinal MRI Portrays Impaired Cerebral Growth and Its Association to Neurodevelopmental Outcome in Newborns with Congenital Heart Disease Undergoing Cardiopulmonary Bypass Surgery." In 50th Annual Meeting of the German Society for Pediatric Cardiology (DGPK). Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1628123.

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Qian, Y., J. L. Liu, M. Umezu, K. Itatani, and K. Miyaji. "A Study of Surgical Optimization for Congenital Heart Diseases Based on Computational Hemodynamic Analysis." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206443.

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Hypoplastic left heart syndrome (HLHS) is one kind of serious congenital heart diseases (CHD). With a small, underdeveloped left ventricle, a heart suffering from HLHS cannot effectively supply enough blood flow to provide for the needs of the body. In order to improve blood circulation, surgery for HLHS has to be carried out at a very early stage. In general, three-stage palliative surgical management for newborns is now widely accepted [1, 2]; Norwood, Glenn, and Fontan.
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De Jonckheere, J., T. Rakza, R. Logier, M. Jeanne, R. Jounwaz, and L. Storme. "Heart rate variability analysis for newborn infants prolonged pain assessment." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6091909.

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Rahman, Mafizur, Maryam Mehzabin Zahin, and Linta Islam. "Effective Prediction On Heart Disease: Anticipating Heart Disease Using Data Mining Techniques." In 2019 International Conference on Smart Systems and Inventive Technology (ICSSIT). IEEE, 2019. http://dx.doi.org/10.1109/icssit46314.2019.8987776.

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Jalali, Ali, C. Nataraj, Gerard F. Jones, and Daniel J. Licht. "Computational Modeling of Hypoplastic Left Heart Syndrome (HLHS) in Newborn Babies." In ASME 2011 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2011. http://dx.doi.org/10.1115/detc2011-48825.

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Hypoplastic left heart syndrome (HLHS) is a congenital heart defect (CHD) in which left side of the heart is severely underdeveloped. To better understand this unique physiology, a computational model of the hypoplastic heart was constructed on the basis of compartmental analysis. Lumped parameter model of HLHS is developed based on the electrical circuit analogy. Model is made up of three parts: hypoplastic heart, pulmonary circulation and systemic circulation. Plots of blood pressure and flow for various parts of body show great match between predicted values and what we expected for the case of HLHS babies. Influence of patent ductus arteriosus (PDA) and ASD resistances on cardiac output and pulmonary to systemic flow was also studied. Results show that by increasing the PDA resistance causes more flow to pulmonary compartments and so the ratio increases. Blood flow increases by decreasing of pulmonary artery resistant. Increasing the PDA resistance causes decrease the cardiac output because of more resistance against blood occurs. Saturation increases by decreasing of pulmonary artery resistant.
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Reports on the topic "Heart disease in the newborn"

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De Jong, Marla J., and Debra K. Moser. Anxiety and Heart Disease. Fort Belvoir, VA: Defense Technical Information Center, January 2003. http://dx.doi.org/10.21236/ada420275.

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Corey Goldman, Corey Goldman. What's Calcium's role in heart and kidney disease? Experiment, December 2013. http://dx.doi.org/10.18258/1719.

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Krauss, Ronald. CRADA Final Report: Genetic Testing for Evaluation of Heart Disease Risk. Office of Scientific and Technical Information (OSTI), January 2002. http://dx.doi.org/10.2172/1157021.

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Pleszkoch, Mark G., Hilda B. Klasky, Aneel Advani, Edmon Begoli, Aileen Boone, and Stephan D. Fihn. Game Theoretic Approach for Understanding and Modeling Clinical Pathways (Stable Ischemic Heart Disease). Office of Scientific and Technical Information (OSTI), October 2018. http://dx.doi.org/10.2172/1513403.

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Dong, Guoqi, Mengye Lu, Xiaoliang Wu, Hao Chen, Hongru Zhang, and Yihuang Gu. Network meta-analysis of Traditional Chinese medicines for depression in coronary heart disease patients. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2020. http://dx.doi.org/10.37766/inplasy2020.5.0036.

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Wienke, Andreas, Anne M. Herskind, Kaare Christensen, Axel Skytthe, and Anatoli I. Yashin. The influence of smoking and BMI on heritability in susceptibility to coronary heart disease. Rostock: Max Planck Institute for Demographic Research, January 2002. http://dx.doi.org/10.4054/mpidr-wp-2002-003.

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Rich-Edwards, Janet, and Ellen Seely. Can an Online Program Help Women with a History of Preeclampsia Reduce Their Risk of Heart Disease? Patient-Centered Outcomes Research Institute® (PCORI), January 2020. http://dx.doi.org/10.25302/1.2020.cer.130601603.

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Lei, Yuping, Meili Wang, Guiqiang Sun, Yong Liu, Yapei Yang, and Dong Hao. Chinese herbal medicine injections (CHMIs) for chronic pulmonary heart disease: protocol for a Bayesian network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2020. http://dx.doi.org/10.37766/inplasy2020.12.0004.

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Yoshinobu, Blain H. Use of Tele-Technology for Heart Disease Management: Improving Clinical and Economic Outcomes in a Managed Care Population. Fort Belvoir, VA: Defense Technical Information Center, April 2003. http://dx.doi.org/10.21236/ada417066.

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Akzhigitova, D. Z., and S. S. Spitsina. Prevalence of rhythm disturbances and conduction of the heart in the structure of ischemic disease in rheumatoid arthritis. Actual problems of experimental and clinical medicine: Materials 77th International Scientific and Practical Conference of Young Scientists and students, 2019. http://dx.doi.org/10.18411/akzhigitova-d-z.

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