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1

Codd, Mary B. Heart failure in Ireland: Epidemiology and economic implications. University College Dublin, Centre for Health Economics, 1994.

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2

S, Parfrey Patrick, and Harnett John D, eds. Cardiac dysfunction in chronic uremia. Kluwer Academic Publishers, 1992.

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3

M, Gotto Antonio, Fondazione Giovanni Lorenzini, and International Symposium on Multiple Risk Factors in Cardiovascular Disease, eds. Multiple risk factors in cardiovascular disease: Strategies of prevention of coronary heart disease, cardiac failure, and stroke. Kluwer Academic Publishers, 1998.

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4

Heart Failure: Epidemiology and Research Methods. Elsevier, 2018. http://dx.doi.org/10.1016/c2016-0-01350-5.

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5

Heart Failure: Epidemiology and Research Methods. Elsevier - Health Sciences Division, 2017.

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6

Farmakis, Dimitrios, John Parissis, and Gerasimos Filippatos. Acute heart failure: epidemiology, classification, and pathophysiology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0051.

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Acute heart failure is defined as the rapid development or change of symptoms and signs of heart failure that requires urgent medical attention and usually hospitalization. Acute heart failure is the first reason for hospital admission in individuals aged 65 or more and accounts for nearly 70% of the total health care expenditure for heart failure. It is characterized by an adverse prognosis, with an in-hospital mortality rate of 4-7%, a 2-3-month post-discharge mortality of 7-11%, and a 2-3-month readmission rate of 25-30%. The majority of patients have a previous history of heart failure and
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7

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Classification, epidemiology, and pathophysiology of heart failure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0744_update_003.

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8

Young, James B., and Roger M. Mills. Clinical Management of Heart Failure. Professional Communications, 2001.

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9

Hypertension and Heart Failure: Epidemiology, Mechanisms and Treatment. Springer International Publishing AG, 2024.

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10

Voicu, Victor, Giuseppe Mancia, Guido Grassi, and Maria Dorobantu. Hypertension and Heart Failure: Epidemiology, Mechanisms and Treatment. Springer, 2019.

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11

Iung, Bernard. Epidemiology and physiopathology. Edited by Gilbert Habib. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0389.

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The incidence of infective endocarditis (IE) is estimated at between 15 and 60 cases per million inhabitants per year from population-based studies in industrialized countries. The presentation of IE has changed since patients are getting older and Staphylococcus is now becoming the microorganism most frequently responsible, which is partly attributable to healthcare-associated infections. The incidence of IE is higher in patients with heart valve prosthesis, previous endocarditis, and complex congenital heart disease. In developing countries, IE occurs in younger patients with a majority of r
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12

Parfrey, Patrick S. Cardiac Dysfunction in Chronic Uremia. Springer, 2012.

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13

Cardiac Dysfunction in Chronic Uremia (Topics in Renal Medicine). Springer, 1991.

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14

Arntz, Hans-Richard. Sudden cardiac death: epidemiology and prevention. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0005.

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Even if sudden cardiac death is considered to be the most frequent cause of death in adults in industrialized countries, its incidence varies widely, depending on the definition and the source and quality of underlying data. It is estimated that about 70-80% of cases are due to coronary heart disease. The remaining 20% are attributable to a wide variety of inborn, genetically determined or acquired diseases, including a small group with hitherto undefined background. Prevention primarily encompasses the treatment of cardiovascular risk factors to avoid manifestations of coronary heart disease.
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15

Cardinale, Daniela, and Carlo Maria Cipolla. Anthracycline-related cardiotoxicity: epidemiology, surveillance, prophylaxis, management, and prognosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0290.

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Anthracycline-induced cardiotoxicity is of considerable concern, as it may compromise the clinical effectiveness of treatment, affecting both quality of life and overall survival in cancer patients, independently of the oncological prognosis. It is probable that anthracycline-induced cardiotoxicity is a unique and continuous phenomenon starting with myocardial cell injury, followed by progressive left ventricular ejection fraction (LVEF) decline that, if disregarded and not treated progressively leads to overt heart failure. The main strategy for minimizing anthracycline-induced cardiotoxicity
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16

Antonio M. Gotto Jr. (Editor), Claude Lenfant (Editor), Rodolfo Paoletti (Editor), Alberico L. Catapano (Editor), and Ann S. Jackson (Editor), eds. Multiple Risk Factors in Cardiovascular Disease: Strategies of Prevention of Coronoary Heart Disease, Cardiac Failure (Medical Science Symposia Series). Springer, 1998.

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17

Plebani, Mario, Monica Maria Mion, and Martina Zaninotto. Biomarkers of renal and hepatic failure. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0039.

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In the last few years, major advances have been achieved in the understanding of the molecular and pathophysiological mechanisms which underlie the complex interactions between the heart and the kidney, as well as between the heart and the liver. According to these new insights, new biomarkers have been proposed for better evaluating and monitoring patients affected by cardiovascular diseases. In addition, some biomarkers should be used as risk factors and for an early identification and treatment of these severe diseases. This chapter reviews the most important biomarkers for evaluating the ‘
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18

Jivanji, Salim, and Michael Rigby, eds. Challenging Concepts in Congenital and Acquired Heart Disease in the Young. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198759447.001.0001.

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Challenging Concepts in Congenital and Acquired Heart Disease in the Young contains a series of challenging concepts in paediatric cardiology covering all subspecialty areas, including general paediatric cardiology, intervention, cardiac imaging, electrophysiology, heart failure and cardiomyopathies, cardiac devices, transplant medicine, epidemiology, and fetal cardiology. Each case provides an in-depth review of current practice, the application of national and international guidelines, and a summary of evidence from the medical literature. Data sets, investigation results, and cardiac imagin
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19

Fox, Grenville, Nicholas Hoque, and Timothy Watts. Cardiovascular problems. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198703952.003.0009.

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This chapter describes the aetiology, presentation, investigation, and management of congenital heart disease (CHD) and acquired cardiovascular problems. It begins by describing the fetal circulation, transition to the neonatal circulation at birth, and the epidemiology of congenital heart disease. There is a guide to the diagnosis of congenital heart disease using a problem-based approach (heart failure, cyanosis, collapse and shock, heart murmurs), together with general principles of management including the use of inotropes, prostaglandin, and surgery. Types of structural heart disease are
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20

Jennings, Catriona, Felicity Astin, Donna Fitzsimons, Ekaterini Lambrinou, Lis Neubeck, and David R. Thompson, eds. ESC Textbook of Cardiovascular Nursing. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198849315.001.0001.

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The ESC Textbook of Cardiovascular Nursing is the official textbook of the European Society of Cardiology (ESC) Association of Cardiovascular Nursing and Allied Professions. It aims to provide in-depth learning for nurses specializing in caring for patients with coronary heart disease, heart failure, valvular disease, arrhythmias, congenital heart disease, and inherited cardiovascular conditions. The textbook builds on the ESC Core Curriculum for the Continuing Professional Development of Nurses Working in Cardiovascular Care. The pathology of these conditions is described as well as the norma
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21

Goldsmith, David J. Cardiovascular disease and chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0098.

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Even after as full a statistical adjustment as can be made for traditional cardiovascular risk factors has been undertaken, impaired kidney function and raised concentrations of albumin in urine each increase the risk of cardiovascular disease (CVD) by two- to fourfold, the degree increasing with severity. If the patient is also suffering from diabetes (as either the cause of CKD or a complication of it), the risks of CVD increase two- to fourfold again. CKD patients should, therefore, be acknowledged as having perhaps the highest cardiovascular risk of any patient cohort. CVD is underdiagnose
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22

Lameire, Norbert. Renal outcomes of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0238_update_001.

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This chapter summarizes the accumulating evidence that incomplete or even apparent complete recovery of renal function after acute kidney injury (AKI) may be an important contributor to a growing number of incident chronic kidney disease (CKD) and end-stage renal disease (ESRD) cases, largely in excess of the global growth in CKD prevalence. Evidence based on epidemiologic studies supports the notion that even after adjustment for several important covariates AKI is independently associated with an increased risk for both CKD and ESRD. Several risk factors for the subsequent development of CKD
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23

Wald, Ron, and Ziv Harel. The Long-Term Outcomes of Acute Kidney Injury. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0015.

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Recent research has provided important insights on the long-term outcomes of patients who develop acute kidney injury (AKI) in the setting of critical illness. Large epidemiologic studies have demonstrated compelling associations between episodes of AKI and progressive kidney disease and death, respectively, although such studies do not establish causality due to the potential for confounding. Whether AKI is intrinsically toxic or a mere by-product of serious comorbidities (e.g. prior chronic kidney disease, heart failure, diabetes), there is no doubt that AKI survivors are a high-risk group w
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