Relevant bibliographies by topics / Heart Heart Heart Septal Defects, Ventricular

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Heart Heart Heart Septal Defects, Ventricular'

Author: Grafiati
Published: 4 June 2021
Last updated: 2 February 2022

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Journal articles on the topic "Heart Heart Heart Septal Defects, Ventricular"

1

Thanopoulos, B. D. "Transcatheter closure of perimembranous ventricular septal defects with the Amplatzer asymmetric ventricular septal defect occluder: preliminary experience in children." Heart 89, no. 8 (2003): 918–22. http://dx.doi.org/10.1136/heart.89.8.918.

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Anderson, Robert H., Edward J. Baker, Siew Yen Ho, Michael L. Rigby, and Tjark Ebels. "The morphology and diagnosis of atrioventricular septal defects." Cardiology in the Young 1, no. 4 (1991): 290–305. http://dx.doi.org/10.1017/s1047951100010362.

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SummaryIn this review, we discuss and describe those features which distinguish hearts with abnormal atrioventricular septation from the normal heart. The hearts, best described as atrioventricular septal defects, are unified by having a common atrioventricular junction guarded by a valve having five leaflets. The left component has three leaflets and cannot be interpreted in terms of a cleft in a normal mitral valve. The papillary muscles supporting this valve are also markedly dissimilar from the arrangement seen in normal hearts. The subaortic outflow tract is displaced superiorly compared
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Kumar, Rupesh, Subhendu Sekhar Mahapatra, Monalisa Datta, et al. "Holt-Oram Syndrome in Adult Presenting with Heart Failure: A Rare Presentation." Case Reports in Cardiology 2014 (2014): 1–3. http://dx.doi.org/10.1155/2014/130617.

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Holt-Oram syndrome is a rare inherited disorder involving the hands, arms, and the heart. The defects involve carpal bones of the wrist and the thumb and the associated cardiac anomalies like atrial or ventricular septal defects. Congenital cardiac and upper-limb malformations frequently occur together and are classified as heart-hand syndromes. The most common amongst the heart-hand disorders is the Holt-Oram syndrome, which is characterized by septal defects of the heart and preaxial radial ray abnormalities. Its incidence is one in 100,000 live births. Approximately three out of four patien
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Nygren, A. "Preoperative evaluation and surgery in isolated ventricular septal defects: a 21 year perspective." Heart 83, no. 2 (2000): 198–204. http://dx.doi.org/10.1136/heart.83.2.198.

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Marques, Laís Costa, Gabriel Romero Liguori, Ana Carolina Amarante Amarante Souza, and Vera Demarchi Aiello. "Left Ventricular Noncompaction Is More Prevalent in Ventricular Septal Defect Than Other Congenital Heart Defects: A Morphological Study." Journal of Cardiovascular Development and Disease 7, no. 4 (2020): 39. http://dx.doi.org/10.3390/jcdd7040039.

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Left ventricular noncompaction (LVNC) is a condition characterized by prominent ventricular trabeculae and deep intertrabecular recesses and has been described as a possible substrate for arrhythmias, thromboembolism, and heart failure. Herein, we explored the prevalence of LVNC morphology among hearts with congenital heart defects (CHD). We examined 259 postnatal hearts with one of the following CHD: isolated ventricular septal defect (VSD); isolated atrial septal defect (ASD); atrioventricular septal defect (AVSD); transposition of the great arteries (TGA); isomerism of the atrial appendages
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Bourdial, Hélène, Karim Jamal-Bey, Abdelhafid Edmar, et al. "Congenital heart defects in La Réunion Island: a 6-year survey within a EUROCAT-affiliated congenital anomalies registry." Cardiology in the Young 22, no. 5 (2012): 547–57. http://dx.doi.org/10.1017/s1047951112000054.

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AbstractObjectivesThis study compares the prevalence and perinatal mortality of congenital heart defects on La Réunion with European (EUROCAT) standards.Methods and resultsData were extracted from a EUROCAT-affiliated congenital malformations registry, covering 88,025 births during the period 2002–2007, on the whole island territory. A total of 512 congenital heart defects were registered, including 424 live births, 18 foetal deaths from 16 weeks of gestation, and 70 terminations of pregnancy. The total prevalence of congenital heart defects was 5.8 per 1000 births and live birth prevalence wa
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Chaiyakulsil, Chanapai, and Boonchu Sirichongkolthong. "Adams-Stokes attack with delayed heart block after ventricular septal repair." Asian Cardiovascular and Thoracic Annals 27, no. 9 (2019): 760–63. http://dx.doi.org/10.1177/0218492319851380.

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A child with Down syndrome, who had undergone uneventful ventricular septal defect repair one year earlier with a normal heart rate during routine follow-up, presented with recurrent seizure-like episodes that did not respond to antiepileptics. She was subsequently found to have complete heart block with significant bradycardia requiring permanent pacemaker insertion. This late and unusual presentation of complete heart block after ventricular septal defect repair is discussed. Prudence must be applied in the evaluation of children who have undergone surgical correction of congenital heart def
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Leung, Carmen, Anish Engineer, Mella Y. Kim, Xiangru Lu, and Qingping Feng. "Myocardium-Specific Deletion of Rac1 Causes Ventricular Noncompaction and Outflow Tract Defects." Journal of Cardiovascular Development and Disease 8, no. 3 (2021): 29. http://dx.doi.org/10.3390/jcdd8030029.

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Background: Left ventricular noncompaction (LVNC) is a cardiomyopathy that can lead to arrhythmias, embolic events and heart failure. Despite our current knowledge of cardiac development, the mechanisms underlying noncompaction of the ventricular myocardium are still poorly understood. The small GTPase Rac1 acts as a crucial regulator of numerous developmental events. The present study aimed to investigate the cardiomyocyte specific role of Rac1 in embryonic heart development. Methods and Results: The Nkx2.5-Cre transgenic mice were crossed with Rac1f/f mice to generate mice with a cardiomyocy
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Dhillon, R. "Transcatheter closure of atrial septal defect preserves right ventricular function." Heart 87, no. 5 (2002): 461–65. http://dx.doi.org/10.1136/heart.87.5.461.

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Siwik, E. S. "Crossed pulmonary arteries, ventricular septal defect, and chromosome 22q11 deletion." Heart 88, no. 1 (2002): 88. http://dx.doi.org/10.1136/heart.88.1.88.

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Dissertations / Theses on the topic "Heart Heart Heart Septal Defects, Ventricular"

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洪克賢 and Newman Hung. "Recovery kinetics in Chinese children with simple repaired congenital heart disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31257227.

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Hung, Newman. "Recovery kinetics in Chinese children with simple repaired congenital heart disease /." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk:8888/cgi-bin/hkuto%5Ftoc%5Fpdf?B23425386.

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Amano, Masashi. "Progression of aortic regurgitation after subpulmonic infundibular ventricular septal defect repair." Kyoto University, 2020. http://hdl.handle.net/2433/252982.

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Mostefa, Kara Meriem. "Communication interventriculaire : de l'anatomie sur spécimens à la numérisation tridimensionnelle Anatomy of the ventricular septal defect in outflow tract defects: similarities and differences Channels between the ventricles: geometry and geography are both important “A new anatomic approach of the ventricular septal defect in the Interruption of the aortic arch Anatomy of the ventricular septal defect in congenital heart defects: a random association?" Thesis, Sorbonne Paris Cité, 2018. http://www.theses.fr/2018USPCB170.

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Partie1 : la communication interventriculaire (CIV) est la cardiopathie congénitale la plus courante et fait partie intégrante de la plupart des malformations cardiaques congénitales complexes. Dans ce travail, nous avons essayé de déterminer la distribution des types anatomiques de CIV dans diverses cardiopathies congénitales (CC). Nous avons étudié 1178 spécimens atteints de cardiopathie congénitale provenant de la collection anatomique du Centre français de référence pour malformations cardiaques congénitales complexes. Au cours de l'étude morphologique, une attention particulière a été por
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Ching, Yung-Hao. "Molecular genetics of human atrial septal defects." Thesis, University of Nottingham, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246413.

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Kirk, Edwin Philip Enfield Women's &amp Children's Health Faculty of Medicine UNSW. "The genetics of atrial septal defect and patent foramen ovale." Awarded by:University of New South Wales, 2007. http://handle.unsw.edu.au/1959.4/34759.

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Congenital heart disease is the most common form of birth defect, affecting approximately 1% of liveborn babies. Secundum atrial septal defect (ASD) is the second most common form of congenital heart disease (CHD). Most cases have no known cause. Chromosomal, syndromal and teratogenic causes account for a minority of cases. The hypothesis that mutations in the ASD genes NKX2-5 and GATA4 may cause apparently sporadic ASD was tested by sequencing them in unrelated probands with ASD. In this study, 1/102 individuals with ASD had an NKX2-5 mutation, and 1/129 had a deletion of the GATA4 gene. The
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Chang, Sheng-Wei. "Deficits in Cardiomyocyte Proliferation: Contributors to Congenital Heart Defects." The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1397643835.

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Duong, Tiffany. "Mechanisms of NR2Fs in Heart Valve Development." University of Cincinnati / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505149242216719.

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Linstrum, Kelsey. "Pbx4 is Required to Restrict Second Heart Field and Ventricular Outflow Tract Size." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1447689641.

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Frid, Christina. "Children with Down syndrome - : an epidemiological study with special focus on congenital heart defects." Doctoral thesis, Uppsala University, Department of Women's and Children's Health, 2002. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3142.

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<p>To assess the impact of congenital malformations in Down syndrome (DS) on morbidity, mortality and outcome at birth, information on all children with DS born in the northern part of Sweden in 1973-80 (n=211) and 1995-98 (n=88) was collected. Most common were congenital heart defects (CHD), dominated by atrioventricular septal defects (AVSD). Up to age 10 years, morbidity and mortality were more than 10 times higher in DS children with CHD than in healthy DS children. The DS children seemed more vulnerable at birth than Swedish children in general: they had increased frequencies of Cesarean
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Books on the topic "Heart Heart Heart Septal Defects, Ventricular"

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1931-, Lue Hung-Chi, Takao Atsuyoshi, and Ando M, eds. Subpulmonic ventricular septal defect: Proceedings of the Third Asian Congress of Pediatric Cardiology. Springer-Verlag, 1986.

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Hras̆ka, Viktor. Surgical management of congenital heart disease I: Complex transposition of great arteries right and left, ventricular outflow, tract obstruction, Ebsteins anomaly : a video manual. Springer, 2012.

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H, Anderson Robert, Van Arsdell Glen S, and SpringerLink (Online service), eds. Congenital Diseases in the Right Heart. Springer London, 2009.

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Lewis, Wendy A. Sabrina: The girl with a hole in her heart : based on a true story of hope and big hearts. Two Dolphins Pub. Group, 2011.

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Lancellotti, Patrizio, and Bernard Cosyns. Adult Congenital Heart Disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0013.

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Echocardiography has a fundamental role in patients with adult congenital heart disease. This chapter identifies the role of echocardiography in atrial septal defects, ventricular septal defects, atrioventricular septal defects, patent ductus arteriosus, and persistent left superior vena cava. For each condition, the role of transthoracic and transoesophagael echocardiogram are shown alongside examples of main types and features and haemodynamic effect. Echocardiographic findings of LV outflow tract obstruction, supravalvular aortic stenosis, aortic stenosis, and aortic coarction are covered,
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Atrial and Ventricular Septal Defects: Molecular Determinants, Impact of Environmental Factors and Non-Surgical Interventions. Nova Science Pub Inc, 2013.

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Serfass, Evan R., and Justin D. Ramos. Ventricular Septal Defect. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0007.

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Ventricular septal defect (VSD) is the most common congenital cardiac lesion, and VSDs are found as isolated lesions in up to 20% of children with congenital heart disease. The natural history and pathophysiology of VSD varies by patient age, patient size, anatomic location, and size of the defect. Patients who have large lesions and significant left-to-right shunt resulting in heart failure symptoms, failure to thrive, pulmonary hypertension, or recurrent respiratory infections may be indicated for early surgical repair during infancy. This chapter presents a clinical scenario of a symptomati
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Orchard, Liz. Congenital heart disease in adults. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0088.

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Congenital heart disease is the most common congenital abnormality, affecting 0.8% of births. There have been major advances in both the surgical and interventional treatment of congenital heart disease, with about 85% of patients now surviving into adulthood. Congenital heart disease can be divided into simple and complex lesions. Simple lesions include atrial septal defect, ventricular septal defect, patent ductus arteriosus, coarctation of the aorta, and left ventricular outflow tract lesions. Complex lesions include tetralogy of Fallot, complete transposition of great arteries, single vent
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Basso, Cristina, José Maria Perèz-Pomares, Gaetano Thiene, and Lucile Houyel. Coronary anomalies. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0025.

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Coronary artery anomalies occur either in isolation or in the context of congenital heart defects (CHD). Isolated coronary artery anomalies include anomalies of connection to the pulmonary artery or to the aorta, anomalies of the intrinsic coronary arterial anatomy including anomalous orifices, and anomalies of myocardial/coronary arterial interaction including myocardial bridges and fistulae. Such defects are of major significance in clinical cardiology and cardiac surgery because of their association with myocardial ischaemia and sudden death. Coronary anomalies associated with CHD can resul
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Thorne, Sara, and Sarah Bowater. Functionally univentricular hearts and Fontan circulation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759959.003.0015.

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This chapter describes tetralogy of Fallot and pulmonary atresia with ventricular septal defect (VSD), including natural (unoperated) or shunt-palliated history, operated history, follow-up after radical repair, and pulmonary atresia with VSD.
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Book chapters on the topic "Heart Heart Heart Septal Defects, Ventricular"

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Corno, Antonio F. "Ventricular septal defect." In Congenital Heart Defects. Steinkopff, 2003. http://dx.doi.org/10.1007/978-3-642-57358-3_6.

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McConnell, Michael E., and Alan Branigan. "Ventricular Septal Defects." In Pediatric Heart Sounds. Springer London, 2008. http://dx.doi.org/10.1007/978-1-84628-684-1_4.

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van Doorn, C., and M. R. de Leval. "Ventricular Septal Defects." In Surgery for Congenital Heart Defects. John Wiley & Sons, Ltd, 2006. http://dx.doi.org/10.1002/0470093188.ch26.

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Corno, Antonio F. "Pulmonary atresia with ventricular septal defect." In Congenital Heart Defects. Steinkopff, 2004. http://dx.doi.org/10.1007/978-3-7985-1934-3_4.

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Kung, Grace C., and Pierre C. Wong. "Ventricular Septal Defects." In Transesophageal Echocardiography for Congenital Heart Disease. Springer London, 2013. http://dx.doi.org/10.1007/978-1-84800-064-3_9.

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Chessa, Massimo, and Gianfranco Butera. "Ventricular Septal Defects." In Cardiac Catheterization for Congenital Heart Disease. Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5681-7_28.

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Chessa, Massimo, Gianfranco Butera, and Angelo Fabio d’Aiello. "Ventricular Septal Defects." In Cardiac Catheterization for Congenital Heart Disease. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-69856-0_33.

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Freedom, R. M., and L. N. Benson. "Ventricular Septal Defect." In Neonatal Heart Disease. Springer London, 1992. http://dx.doi.org/10.1007/978-1-4471-1814-5_34.

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Pacifico, A. D. "Atrio-Ventricular Septal Defects." In Surgery for Congenital Heart Defects. John Wiley & Sons, Ltd, 2006. http://dx.doi.org/10.1002/0470093188.ch27.

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Moguillansky, Diego, Traci M. Kazmerski, Ricardo Muñoz, and Victor O. Morell. "Ventricular Septal Defects." In Critical Care of Children with Heart Disease. Springer London, 2009. http://dx.doi.org/10.1007/978-1-84882-262-7_16.

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Conference papers on the topic "Heart Heart Heart Septal Defects, Ventricular"

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Jhun, Choon-Sik, Mark B. Ratcliffe, and Julius M. Guccione. "Ventricular Wall Stress and Pump Function of Ventricular Septal Defect of Congenital Heart Defects." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206320.

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About 36,000 infants are born each year with a congenital heart defect (CHD) and charges for treatment surpass $2.2 billion for inpatient surgery alone. Of many different types of CHDs, ventricular septal defect (VSD) is the most common class (∼1/3 of CHDs) of heart deformity present at birth. Though many close spontaneously and rarely require treatment, VSD still accounts for ∼15% of defects requiring an invasive procedure within the first year of life [1]. Generally, the ventricular performance is indexed by geometry, shape, diastolic and systolic function, and myocardial contractility [2].
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Sun, Shuping, Haibin Wang, Chi Cheng, Zhenhui Chang, and Dayong Huang. "PCA-based heart sound feature generation for a ventricular septal defect discrimination." In 2017 14th International Computer Conference on Wavelet Active Media Technology and Information Processing (ICCWAMTIP). IEEE, 2017. http://dx.doi.org/10.1109/iccwamtip.2017.8301464.

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Hart, Keir D., Max B. Mitchell, and Mark E. Rentschler. "Initial Design and Evaluation of an Intra-Cardiac Camera System for Ventricular Septal Defects." In ASME 2010 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/detc2010-28759.

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The primary objective of this project is to design, fabricate, and test a small, integrated camera system for aiding in the visualization and surgical repair of ventricular septal defects (VSDs), or holes in the heart wall, in pediatric patients. Currently, a less invasive device to view VSDs from the left ventricle of the heart does not exist. This left perspective is ideal for obtaining an unobstructed view of the VSD. The proposed VSD camera device would also provide a platform for passing a suture through the hole in the septal wall, with future work implementing additional tools capable o
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Das, Ashish, William Gottliebson, and Rupak K. Banerjee. "Comparison of Right Ventricular Stroke Work for Tetralogy Patient and Normal Subject." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193145.

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Tetralogy of Fallot (TOF), also called blue-baby syndrome is one of the most common congenital heart defects in children after infancy and is estimated to account for 10% of all congenital heart defects [3]. TOF consists of four interrelated lesions: i) ventricular septal defect ii) Pulmonary stenosis iii) Right ventricular (RV) hypertrophy and (iv) Overriding Aorta [3]. TOF has been successfully repaired for several decades (Fig. 1). There are now an estimated 100,000 adult “repaired TOF” patients in the United States alone. As a result, long-term sequelae of the disease and repair have becom
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Tang, Dalin, Chun Yang, Tal Geva, Glenn Gaudette, and Pedro J. del Nido. "Regenerated Contracting Myocardium May Improve Post-Surgery Right Ventricle Function: Patch Comparison Using MRI-Based Two-Layer Anisotropic Models of Human Right and Left Ventricles." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19067.

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Patients with repaired Tetralogy of Fallot (ToF), a congenital heart defect which includes a ventricular septal defect and severe right ventricular outflow obstruction, account for the majority of cases with late onset RV failure. The current surgical approach, which includes pulmonary valve replacement/insertion (PVR), has yielded mixed results. One reason for the unpredictable results is that the PVR surgery only addresses pulmonary regurgitation. New surgical options including scar tissue reduction and RV remodeling have been proposed in order to improve RV function recovery [1]. Various RV
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Alser, Maha W., Huseyin Enes Salman, and Huseyin Cagatay Yalcin. "Altered Inflow Hemodynamics affects Heart Development in a Side Specific Manner in the Embryonic Heart." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0172.

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Background: Hemodynamics, forces from the flowing blood in the heart, is a major epigenetic factor for heart development. Disturbed hemodynamics were shown to induce cardiac malformations in the embryonic heart. Clinically relevant congenital heart defects (CHDs) can be introduced surgically in the lab by disturbing the hemodynamics, like Hypoplastic left heart syndrome (HLHS), characterized by underdeveloped left ventricle is underdeveloped. Left atrial ligation (LAL) on chick embryo is an experimental technique to produce a HLHS-like phenotype. Aims: To reveal mechanobiological mechanisms as
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Li, Yiming, Kun Sun, and Chengli Song. "Finite Element Modeling and Analysis of Ventricular Septal Defect Occluders." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6837.

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Transcatheter closure of ventricular septal defect (VSD) has become an alternative therapy to open-chest surgery because of simple procedure, less invasion, and high safety [1–2]. The most important occluder device of the therapy is double-disc structure which occludes the VSD, with the discs of the occluder clamp the margin of VSD while the waist of the occluder supports the VSD hole (Figure 1(a)). Commercially available occluders are woven by 72 nitinol wires (Figure1(b)) and then formed by heat treatment. However, the implantation of metal occluders in perimembranous part will result in a s
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Temel, F. Zeynep, Hayley McClintock, Christopher J. Payne, et al. "Pop-Up-Inspired Design of a Septal Anchor for a Ventricular Assist Device." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3458.

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Heart failure (HF) is a serious condition in which the heart cannot pump sufficient blood to sustain the metabolic needs of the body. A common indication of failure is a low ejection fraction, or the volumetric proportion of blood ejected when the ventricle contracts. In end-stage HF, support from a ventricular assist device (VAD) can assume some or all of the heart’s pumping work, improving the ejection fraction and restoring normal circulation. VAD therapy options for end-stage right heart failure (RHF) are limited, with only a few FDA-approved devices available for mechanical circulatory su
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Valenti, Isabella E., Breigh N. Roszelle, Michael V. Perone, Steven Deutsch, and Keefe B. Manning. "Impact of Outlet Valve Orientation on Fluid Dynamics of the 12 cc Penn State Pediatric Ventricular Assist Device." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192744.

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Congenital cardiovascular defects are the leading cause of death among live births [1]. These defects involve the interior walls of the heart, valves, arteries, and veins and change the normal flow of blood through the heart and into the systemic system. Fortunately, several options exist for the more than 35,000 children born with congenital heart disease. Ventricular assist devices (VADs) currently hold the most promise for bridge-to-transplant treatment; however, a major problem for these devices is thrombus formation and deposition.
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Taylor, Joshua O., Kory P. Witmer, Thomas Neuberger, et al. "Experimental and Computational Studies of a Formed Thrombus Within a Backward-Facing Step Geometry." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80571.

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Heart disease is one of the leading causes of death in the United States. This condition affects roughly 5.7 million Americans, with approximately 670,000 new cases and 300,000 deaths each year [1]. Heart failure, resulting from heart disease, is primarily treated with the implantation of a ventricular assist device (VAD) [2]. Along with VADs, arterial stents (primarily for treatment of atherosclerosis) and prosthetic heart valves (for defects in or other failures of the native heart valves) are other devices that are regularly used by clinicians to treat conditions within the circulatory syst
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