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Dissertations / Theses on the topic 'Heart Muscles Diseases'

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Published: 4 June 2021

Last updated: 31 January 2023

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1

Gieseking, Elizabeth Robinson. "Control mechanism for the papillary muscles of the mitral valve : an In Vitro study." Thesis, Georgia Institute of Technology, 1989. http://hdl.handle.net/1853/10912.

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2

Warner, Anke Sigrid. "The expression, regulation and effects of inducible nitric oxide synthase in hibernating myocardium." Title page, contents and summary only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09phw279.pdf.

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Amendments inserted at back. "May 2002" Includes bibliographical references (leaves 237-290) Experiments described in this thesis address the potential role of inducible nitric oxide synthase (iNOS) in hibernating myocardium. Specifically it was sought to establish a cellular model of hibernating myocardium and investigate the expression, regulation and effects of iNOS in this model. Experiments were performed using primary cultures of neonatal rat ventricular myocytes.

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3

Messaggi-Sartor, Monique 1984. "Respiratory muscle dysfunction in respiratory and non-respiratory diseases : clinical and therapeutic approaches." Doctoral thesis, Universitat Pompeu Fabra, 2017. http://hdl.handle.net/10803/565809.

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Respiratory muscle dysfunction is a clinical condition that may be present in both respiratory and non-respiratory diseases. This impairment of muscle function can have a negative effect on clinical outcomes, contributing to a further worsening of the patient’s clinical condition. This doctoral thesis has been directed by the ‘Rehabilitation Research Group’ (RERG) in collaboration with the Muscle Wasting and Cachexia in Chronic Respiratory Diseases and Lung Cancer Group (Lung Cancer and Muscle Research Group) of the Institut Hospital del Mar d’Investigacions Mèdiques (IMIM) in Barcelona. Muscl

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4

Spinner, Erin M. "Tricuspid valve mechanics: understanding the effect of annular dilatation and papillary muscle displacement." Diss., Georgia Institute of Technology, 2011. http://hdl.handle.net/1853/45754.

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Tricuspid regurgitation (TR), back flow of blood from the right ventricle to the right atrium, has been reported in approximately 85% of the population, with 16% having mild or severe TR. Patients with untreated moderate to severe TR are likely to experience decreased exercise capacity and have increased morbidity and mortality, thus affecting the patient's quality of life. Current methods of repair offer limited rates of success, and many patients require further operations to correct returning levels of TR. Incomplete repair may be due to incomplete understanding of the functional anatomy a

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5

Yentrapalli, Venkata Ramesh. "Novel radiation targets in the endothelium and heart muscle." Doctoral thesis, Stockholms universitet, Institutionen för molekylär biovetenskap, Wenner-Grens institut, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-90429.

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Worldwide, people are being exposed to natural and man-made sources of radiation. Epidemiological studies have shown an increased risk of vascular diseases in populations that have been exposed to ionizing radiation. Vascular endothelium is implicated as one of the targets for radiation leading to the development of cardiovascular diseases. However, the molecular mechanisms behind the development of radiation-induced cardiovascular disease in acute or chronic exposed people are not fully elucidated. The hypothesis that chronic low dose rate ionizing radiation accelerates the onset of senescenc

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6

Pandofino, Alexandra. "A molecular analysis of the basis of cardiovirulence of Coxsackievirus B3." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.267166.

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7

Colegrave, Melanie. "Expression of #beta#-cardiac myosin in a myogenic cell line." Thesis, King's College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342254.

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8

Vesier, Carol Cockerham. "The role of papillary muscle-mitral valve geometry in systolic anterior motion of the mitral valve." Diss., Georgia Institute of Technology, 1991. http://hdl.handle.net/1853/10279.

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9

Chung, Jae-Hoon. "Regulation of Human Cardiac Muscle Contraction and Relaxation in Health and Disease." The Ohio State University, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=osu1522851185767187.

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10

Johnson, Andrew William. "Metabolic control of energetics in human heart and skeletal muscle." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:82c0dce6-a162-4c08-b061-3ea7f2e35134.

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Myocardial and skeletal muscle high energy phosphate metabolism is abnormal in heart failure, but the pathophysiology is not understood. Plasma non-esterified fatty acids (NEFA) increase in heart failure due to increased sympathetic drive, and regulate the transcription of mitochondrial uncoupling protein-3 (UCP3), through peroxisome proliferator-activated receptor-α. The aim of the work in this thesis was to determine whether cardiac PCr/ATP ratios and skeletal muscle PCr kinetics during exercise were related to cardiac and skeletal muscle UCP3 levels respectively, thus providing a mechanism

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11

Andersen, Kasper. "Physical Activity and Cardiovascular Disease." Doctoral thesis, Uppsala universitet, Institutionen för medicinska vetenskaper, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-217309.

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The aim was to investigate associations of fitness and types and levels of physical activity with subsequent risk of cardiovascular disease. Four large-scale longitudinal cohort studies were used. The exposures were different measures related to physical activity and the outcomes were obtained through linkage to the Swedish In-Patient Register. In a cohort of 466 elderly men without pre-existing cardiovascular disease, we found that skeletal muscle morphology was associated with risk of cardiovascular events. A high amount of type I (slow-twitch, oxidative) skeletal muscle fibres was associate

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12

Gonçalves, Ana Clara Campagnolo Real [UNESP]. "Exercício resistido em cardiopatas: revisão sistemática da literatura e análise do comportamento autonômico em recuperação pós-exercício resistido aplicado de forma aguda." Universidade Estadual Paulista (UNESP), 2010. http://hdl.handle.net/11449/87292.

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Made available in DSpace on 2014-06-11T19:22:48Z (GMT). No. of bitstreams: 0 Previous issue date: 2010-12-10Bitstream added on 2014-06-13T19:47:20Z : No. of bitstreams: 1 goncalves_accr_me_prud.pdf: 684147 bytes, checksum: a81c4f89900e68d55d13d636f49523a1 (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>A perda de massa muscular secundária à idade e à inatividade física é clinicamente relevante na população cardíaca, contudo a prescrição do exercício resistido dinâmico para esses pacientes apresenta-se inconclusiva na literatura. Objetivo: Reunir informações so

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13

Morris, Robert Tyler. "Maladaptation of cardiac and skeletal muscle in chronic disease effects of exercise /." Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://edt.missouri.edu/Summer2007/Dissertation/MorrisR-080307-D8225/.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2007.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. "August 2007" Includes bibliographical references.

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14

Bundgaard, Henning. "Potassium regulation in heart and skeletal muscles : relation to level of K intake, disease mechanisms and pharmacotherapy /." København : Lægeforeningen, 2005. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=013175180&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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15

Natarajan, Aruna Ramachandran. "Mechanism of dopamine-mediated activation of BK channels in human coronary artery smooth muscle cells." Connect to Electronic Thesis (ProQuest) Connect to Electronic Thesis (CONTENTdm), 2008. http://worldcat.org/oclc/457179694/viewonline.

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16

Gonçalves, Ana Clara Campagnolo Real. "Exercício resistido em cardiopatas : revisão sistemática da literatura e análise do comportamento autonômico em recuperação pós-exercício resistido aplicado de forma aguda /." Presidente Prudente : [s.n.], 2010. http://hdl.handle.net/11449/87292.

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Orientador: Luiz Carlos Marques Vanderlei<br>Banca: Carlos Marcelo Pastre<br>Banca: Francis da Silva Lopes<br>Resumo: A perda de massa muscular secundária à idade e à inatividade física é clinicamente relevante na população cardíaca, contudo a prescrição do exercício resistido dinâmico para esses pacientes apresenta-se inconclusiva na literatura. Objetivo: Reunir informações sobre a especificidade dos componentes do treinamento resistido prescrito à população cardiopata. Metodologia: Foi realizada busca sistemática de literatura, a partir das bases de dados LILACS, SciELO e PubMed, utilizando

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17

Jacob, Ashok J. "A study of the prevalence, pathogenesis and natural history of heart muscle disease associated with HIV infection." Thesis, University of Edinburgh, 1995. http://hdl.handle.net/1842/20587.

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Heart muscle disease was found in 14.2% of HIV patients and took three principal forms - dilated cardiomyopathy, borderline left ventricular dysfunction and isolated right ventricular dilation. Dilated cardiomyopathy was associated significantly with a very low CD4 count indicative of late stage HIV disease. It was invariably irreversible. In contrast, some patients with borderline left ventricular dysfunction and isolated right ventricular dilation subsequently reverted to normal. The latter was usually related to pressure or volume overload of the right ventricle rather than to a primary myo

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18

Chappell, Joel. "Vascular smooth muscle cell heterogeneity and plasticity in models of cardiovascular disease." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/274543.

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Vascular smooth muscle cell (VSMC) accumulation is a hallmark of atherosclerosis and vascular injury. However, fundamental aspects of proliferation and the phenotypic changes within individual VSMCs, which underlie vascular disease remain unresolved. In particular, it is not known if all VSMCs proliferate and display plasticity, or whether individual cells can switch to multiple phenotypes. To assess whether proliferation and plasticity in disease is a general characteristic of VSMCs or a feature of a subset of cells, multi-colour lineage labelling is used to demonstrate that VSMCs in injury-i

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19

Maddali, Kamala Kalyani. "A mandatory requirement of PKC-[delta] in testosterone regulated coronary smooth muscle cell differentiation, proliferation and apoptosis /." Free to MU Campus, others may purchase, 2005. http://proquest.umi.com/pqdweb?did=1232392431&sid=1&Fmt=2&clientId=45247&RQT=309&VName=PQD.

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20

Ohnaka, Motoaki. "Effect of microRNA-145 to prevent vein graft disease in rabbits by regulation of smooth muscle cell phenotype." Kyoto University, 2014. http://hdl.handle.net/2433/192144.

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The final publication is available at http://dx.doi.org/10.1016/j.jtcvs.2013.11.054. Motoaki Ohnaka, Akira Marui, Kenichi Yamahara, Kenji Minakata, Kazuhiro Yamazaki, Motoyuki Kumagai, Hidetoshi Masumoto, Shiro Tanaka, Tadashi Ikeda, Ryuzo Sakata, Effect of microRNA-145 to prevent vein graft disease in rabbits by regulation of smooth muscle cell phenotype, The Journal of Thoracic and Cardiovascular Surgery, Volume 148, Issue 2, August 2014, Pages 676-682.e2, ISSN 0022-5223.<br>Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(医学)<br>甲第18544号<br>医博第3937号<br>新制||医||1006(附属図書館)<br>31444<br>京都大学大学院

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21

Zakariyah, Abeer. "The Characterization of a Human Disease-Associated Mutation Nkx2.5 R142C Using In vitro and In vivo Models." Thesis, Université d'Ottawa / University of Ottawa, 2017. http://hdl.handle.net/10393/35817.

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Nkx2.5 is a cardiac transcription factor that plays a critical role in heart development. In humans, heterozygous mutations in the NKX2.5 gene result in congenital heart defects (CHDs), but the molecular mechanisms by which these mutations cause the defects are still unknown. NKX2.5 R142C is a mutation that is found to be associated with atrial septal defect and atrioventricular block in 13 patients from one family. The R142C mutation is located within both the DNA-binding domain and the nuclear localization sequence of NKX2.5 protein. The pathogenesis of CHDs in humans with R142C point mutati

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22

Jenkins, William Stephen Arthur. "Novel applications of positron emission tomography in the non-invasive assessment of cardiovascular disease." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31229.

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Introduction. Fused Positron Emission Tomography and Computed Tomography (PET/CT) is an emerging investigative tool in cardiovascular disease that provides an imaging-based quantification of pathophysiological processes of interest. The purpose of this thesis was to study the application of PET to identify fundamental pathophysiological processes driving 3 forms of cardiovascular disease: aortic stenosis, myocardial infarction, and atherosclerosis. Methods. Aortic Stenosis. Patients with a spectrum of calcific aortic valve disease (n=121) who underwent PET-CT imaging for the identification of

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23

Braitsch, Caitlin M. "The role of Pod1/Tcf21 in epicardium-derived cells in cardiac development and disease." University of Cincinnati / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1367925962.

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24

Tikkanen, Heikki O. "The influence of skeletal muscle properties, physical activity and physical fitness on serum lipids and the risk of coronary heart disease." Helsinki : University of Helsinki, 2001. http://ethesis.helsinki.fi/julkaisut/laa/kliin/vk/tikkanen/.

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25

Raad, Farah [Verfasser], Wolfram-Hubertus [Akademischer Betreuer] [Gutachter] Zimmermann, and Lutz [Gutachter] Tietze. "Characterization and Application of Bioengineered Heart Muscle as a New Tool to Study Human Heart Development and Disease / Farah Raad ; Gutachter: Wolfram-Hubertus Zimmermann, Lutz Tietze ; Betreuer: Wolfram-Hubertus Zimmermann." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2016. http://d-nb.info/1135487642/34.

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26

Wamhoff, Brian R. "Calcium regulation in coronary smooth muscle : mechanisms of cardioprotection /." free to MU campus, to others for purchase, 2001. http://wwwlib.umi.com/cr/mo/fullcit?p3013038.

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27

Hill, Brent J. F. "Myoplasmic calcium regulation and the function of nucleotide and endothelin receptors in models of coronary artery disease." free to MU campus, to others for purchase, 2000. http://wwwlib.umi.com/cr/mo/fullcit?p9988669.

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28

Seifert, Elena. "Metabolic Changes in Pulmonary Arterial Smooth Muscle Cells Exposed to Increased Mechanical Forces from an Ovine Model of Congenital Heart Disease with Increased Pulmonary Blood Flow." Scholarship @ Claremont, 2019. https://scholarship.claremont.edu/cmc_theses/2094.

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An important cause of pulmonary arterial hypertension (PAH) in children with congenital heart disease (CHD) is increased pulmonary blood flow (PBF). To gain a better understanding of the disease process, the changes in biochemical pathways and metabolism of pulmonary arterial smooth muscle cells (PASMCs) were studied using a unique surgical ovine model of increased pulmonary blood flow. PASMCs isolated from 4-week-old lambs with increased PBF (shunt) showed lower oxygen consumption rates and lower extracellular acidification rates linked to glutamine metabolism when compared to controls. Shunt

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29

Intaitė, Gintarė. "Relaksacijos taikymas arterinio kraujo spaudimo reguliavimui sergančiųjų išemine širdies liga stacionarinės reabilitacijos etape." Master's thesis, Lithuanian Academic Libraries Network (LABT), 2008. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2008~D_20080828_135113-12189.

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Darbo problema – vis dar lieka neaiškus PRR efektyvumas ir jos sąsajos su amžiumi, lytimi, išsilavinimu, IŠL forma, subjektyvia savijauta, subjektyviu sveikatos vertinimu bei organizmo raumenų įtampa, sergantiems IŠL. Todėl šio tyrimo tikslas - nustatyti progresuojančios raumenų relaksacijos taikymo efektyvumą AKS reguliavimui, atsižvelgiant į lytį, amžių, ligos formą, subjektyvų sveikatos vertinimą, išsilavinimą, raumenų įtampą ir subjektyvią savijautos įtampą, sergantiems IŠL, stacionarinės reabilitacijos etape. Tyrime buvo pakviesti dalyvauti 204 Abromiškių reabilitacinės ligoninės, kardiol

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30

Hiemann, Nicola. "Histomorphometrische Untersuchungen myokardialer Blutgefäßveränderungen nach Herztransplantation." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 1998. http://dx.doi.org/10.18452/14461.

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Anhand von immunhistochemischen Färbemethoden wurde die Expression von CD 31 (immunhistochemischer Marker für Endothelzellen) und -Aktin (immunhistochemischer Marker für glatte Muskelzellen) auf Zellen der intramyokardialen Blutgefäße herztransplantierter Patienten histomorphometrisch ausgewertet. In die Bewertung der myokardialen Strukturen ging auch die Untersuchung der jeweiligen zugehörigen HE-Färbungen mit ein. Ziel dieser Untersuchungen war die Beurteilung von qualitativen und quantitativen Unterschieden dieser Marker während eines Untersuchungszeitraumes von 14 Monaten nach Herztra

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31

Fernandes, Adriane Sayuri Nakashima. "Avaliação da efetividade e segurança do treinamento da manobra de empilhamento de ar nas distrofinopatias." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-01042015-101822/.

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INTRODUÇÃO: As distrofias musculares (DM) constituem um grupo de doenças genéticas caracterizadas por fraqueza muscular progressiva decorrente da degeneração irreversível do tecido muscular esquelético. O comprometimento da função respiratória é um sinal precoce da progressão da doença. A fraqueza progressiva dos músculos respiratórios torna o paciente com distrofia muscular incapaz de realizar inspirações profundas de forma independente para promover uma tosse eficaz. Portanto, torna-se necessário fornecer insuflações regulares com volumes que o paciente aprende a empilhar com o fechamento da

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32

Lamotte, Michel. "Contribution à l'étude de la réponse hémodynamique lors d'exercices de renforcement musculaire: sujets sains et patients de réadaptation cardio-vasculaire." Doctoral thesis, Universite Libre de Bruxelles, 2011. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209825.

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33

Picichè, Marco. "L’augmentation de la circulation collatérale non coronarienne : l’hypothèse d’une méthode alternative de revascularisation myocardique." Thesis, Paris 11, 2011. http://www.theses.fr/2011PA11T044/document.

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La circulation collatérale non coronarienne (CCNC) perfuse le cœur en provenant d’artères bronchiques, médiastinales et péricardiques. L’éventualité que la CCNC puisse être artificiellement augmentée pour fournir un traitement alternatif pour les patients ischémiques représente une hypothèse intrigante sans réponse en raison de la nature difficile de ce champ de recherche. En se basant sur plusieurs aspects, tel que (1) l’existence des collatérales naturelles entre les artères coronaires et les artères thoraciques internes (ATIs), (2) les effets potentiels de la ligature des ATIs; (3) la capac

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34

Ara?jo, Thaise Lucena. "For?a muscular respirat?ria, qualidade de vida e modula??o auton?mica da frequ?ncia card?aca na distrofia miot?nica." Universidade Federal do Rio Grande do Norte, 2009. http://repositorio.ufrn.br:8080/jspui/handle/123456789/16675.

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Made available in DSpace on 2014-12-17T15:16:06Z (GMT). No. of bitstreams: 1 ThaiseLA.pdf: 779453 bytes, checksum: fe87c118f2c753bc8ffd4a32cb6a1146 (MD5) Previous issue date: 2009-07-22<br>Background: The myotonic dystrophy (MD) is a multisystem neuromuscular disease that can affect the respiratory muscles and heart function, and cause impairment in quality of life. Objectives: Investigate the changes in respiratory muscle strength, health-related quality of life (HRQoL) and autonomic modulation heart rate (HR) in patients with MD. Methods: Twenty-three patients performed assessment of pulmo

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35

Clark, Amanda. "MEF2-regulated Gtl2-Dio3 noncoding RNAs in cardiac muscle and disease." Thesis, 2016. https://hdl.handle.net/2144/14521.

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The MEF2 transcription factor is a central regulator of skeletal and cardiac muscle development. Recently, we showed that MEF2A regulates skeletal muscle regeneration through direct regulation of the Gtl2-Dio3 microRNA mega-cluster. In addition to their expression in skeletal muscle, temporal expression analysis of selected Gtl2-Dio3 microRNAs revealed high enrichment in cardiac muscle. Therefore, I investigated the role of selected microRNAs from the Gtl2-Dio3 noncoding RNA locus in the heart. First, I found that Gtl2-Dio3 microRNAs are expressed at higher levels in perinatal hearts compared

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36

Raad, Farah. "Characterization and Application of Bioengineered Heart Muscle as a New Tool to Study Human Heart Development and Disease." Doctoral thesis, 2016. http://hdl.handle.net/11858/00-1735-0000-002B-7C05-A.

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Frisk, Emelie. "Lower limb muscle function in children and adolescents with Fontan circulation : A cross-sectional study." Thesis, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-160552.

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Introduction: Impaired isometric muscle strength and muscle endurance in adults with Fontan circulation has previously been reported. However, the knowledge if corresponding impairment is present in children and adolescents with Fontan circulation is scarce. Aim: The aim was to examine the isometric muscle strength and muscle endurance of the lower limbs in children and adolescents with Fontan circulation in comparison to age and sex matched controls. Method: In this cross-sectional study 43 children and adolescents (6-18 years) with Fontan circulation and 43 controls were included. Isometric

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"Effect of hyperkalemia and ischemia on large conductance calcium-activated potassium channels in porcine coronary arterial smooth muscle: relevance to cardioplegic arrest." 2008. http://library.cuhk.edu.hk/record=b5893576.

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Han, Jianguo.<br>Thesis (M.Phil.)--Chinese University of Hong Kong, 2008.<br>Includes bibliographical references (leaves 66-76).<br>Abstracts in English and Chinese.<br>Declaration --- p.i<br>Acknowledgement --- p.□<br>Publication --- p.□<br>Abstract (English) --- p.□xi<br>Abstract (Chinese) --- p.□<br>Abbreviations --- p.ix<br>List of figures / tables --- p.x<br>Chapter Chapter 1. --- General Introduction<br>Chapter 1.1 --- Role of vascular smooth muscle cells in the control of coronary circulation --- p.1<br>Chapter 1.1.1 --- Potassium channels in the coronary smooth muscle cells ---

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39

McKenney, Mikaela Lee. "Coronary artery disease progression and calcification in metabolic syndrome." Thesis, 2014. http://hdl.handle.net/1805/6460.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>For years, the leading killer of Americans has been coronary artery disease (CAD), which has a strong correlation to the U.S. obesity epidemic. Obesity, along with the presence of other risk factors including hyperglycemia, hypercholesterolemia, dyslipidemia, and high blood pressure, comprise of the diagnosis of metabolic syndrome (MetS). The presentation of multiple MetS risk factors increases a patients risk for adverse cardiovascular events. CAD is a complex progressive disease. We utilized the superb model of CAD and MetS, the Os

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Rodenbeck, Stacey Dineen. "Coronary artery disease in metabolic syndrome: a role for the sarcoplasmic reticulum Ca2+ ATPase." Diss., 2016. http://hdl.handle.net/1805/10649.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Coronary artery disease (CAD) is a leading cause of death among Americans and is fueled by underlying metabolic syndrome (MetS). The prevalence and lethality of CAD necessitates rigorous investigations into its underlying mechanisms and to facilitate the development of effective treatment options. Coronary smooth muscle (CSM) phenotypic modulation from quiescent to synthetic, proliferative, and osteogenic phenotypes is a key area of investigation, with underlying mechanisms that remain poorly understood. Using a well-established pre-

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41

Noblet, Jillian Nicole. "Coronary perivascular adipose tissue and vascular smooth muscle function: influence of obesity." Diss., 2016. http://hdl.handle.net/1805/9815.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Factors released from coronary perivascular adipose tissue (PVAT), which surrounds large coronary arteries, have been implicated in the development of coronary disease. However, the precise contribution of coronary PVAT-derived factors to the initiation and progression of coronary vascular dysfunction remains ill defined. Accordingly, this investigation was designed to delineate the mechanisms by which PVAT-derived factors influence obesity-induced coronary smooth muscle dysfunction. Isometric tension studies of coronary arteries fro

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42

Owen, Meredith Kohr. "Effect of coronary perivascular adipose tissue on vascular smooth muscle function in metabolic syndrome." Thesis, 2013. http://hdl.handle.net/1805/3789.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Obesity increases cardiovascular disease risk and is associated with factors of the “metabolic syndrome” (MetS), a disorder including hypertension, hypercholesterolemia and/or impaired glucose tolerance. Expanding adipose and subsequent inflammation is implicated in vascular dysfunction in MetS. Perivascular adipose tissue (PVAT) surrounds virtually every artery and is capable of releasing factors that influence vascular reactivity, but the effects of PVAT in the coronary circulation are unknown. Accordingly, the goal of this inves

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43

Silva, João Gabriel Branco. "The Effects of High Fat Feeding on the Metabolomics of Heart, Kidney and Skeletal Muscle in C57BL6J Mice as Analysed by 1H NMR." Master's thesis, 2021. http://hdl.handle.net/10316/96156.

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Dissertação de Mestrado em Biologia Celular e Molecular apresentada à Faculdade de Ciências e Tecnologia<br>A doença do fígado gordo não alcoólico (NAFLD) é uma manifestação hepática associada à síndrome metabólica, com uma prevalência mundial documentada de 25% em 2020 e com tendência para um aumento da sua incidência no futuro. A NAFLD adquiriu um interesse e preocupação global devido ao rápido crescimento da sua prevalência ao longo dos últimos anos, enquanto os métodos de diagnóstico e tratamento atuais continuam incomparáveis à dimensão do problema. Atualmente nenhuma despistagem de rotin

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