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Journal articles on the topic 'Heart Rupture'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2024

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1

Guo, Jun, Yun-dai Chen, Feng Tian, Hong-bin Liu, Lian Chen, Zhi-jun Sun, Yi-hong Ren, et al. "Thrombosis and morphology of plaque rupture using optical coherence tomography." Chinese Medical Journal 126, no. 6 (March 20, 2013): 1092–95. http://dx.doi.org/10.3760/cma.j.issn.0366-6999.20122642.

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Background Thrombosis following plaque rupture is the main cause of acute coronary syndrome, but not all plaque ruptures lead to thrombosis. There are limited in vivo data on the relationship between the morphology of ruptured plaque and thrombosis. Methods We used optical coherence tomography (OCT) to investigate the morphology of plaque rupture and its relation to coronary artery thrombosis in patients with coronary heart disease. Forty-two patients with coronary artery plaque rupture detected by OCT were divided into two groups (with or without thrombus) and the morphological characteristics of ruptured plaque, including fibrous cap thickness and broken cap site, were recorded. Results The fibrous cap of ruptured plaque with thrombus was significantly thinner compared to caps without thrombus ((57.00±17.00)μm vs. (96.00±48.00)μm; P=0.0076). Conclusions Plaque rupture associated with thrombosis occurs primarily in plaque covered by a thin fibrous cap. Thick fibrous caps are associated with greater stability of ruptured plaque.

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2

Ly, Q. H. "Myocardial infarction with myocardial rupture." Heart 89, no. 9 (September 1, 2003): 1077. http://dx.doi.org/10.1136/heart.89.9.1077.

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3

Arıkan, Erdinç, Arif Karagöz, Serdar Bayata, Levent Yilik, and Erden Erol Ünlüer. "A Rare Cause of Dyspnea: Sudden Rupture of Aortic Valsalva Sinus Aneurysm." Case Reports in Medicine 2013 (2013): 1–3. http://dx.doi.org/10.1155/2013/909302.

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Aneurysm of the sinus of Valsalva is an uncommon cardiac abnormality; however, the most common complication is rupture into the right heart chambers or rarely towards the left chambers. A ruptured aneurysm typically leads to an aortocardiac shunt and progressively worsening heart failure. We report a case of a 21-year-old male who suffered an aneurysm of the sinus of Valsalva rupture into the right atrium who underwent successful surgical repair.

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4

BANERJEE, S. "Coronary artery rupture treated with microcoil occlusion." Heart 86, no. 2 (August 1, 2001): 187. http://dx.doi.org/10.1136/heart.86.2.187.

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5

Murday, A. "Optimal management of acute ventricular septal rupture." Heart 89, no. 12 (December 1, 2003): 1462–66. http://dx.doi.org/10.1136/heart.89.12.1462.

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6

Dimopoulos, K. "Multiple coronary rupture after blunt chest trauma." Heart 89, no. 6 (June 1, 2003): 594. http://dx.doi.org/10.1136/heart.89.6.594.

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7

Wu, Tongbin, Yujun Xu, Lunfeng Zhang, Zhengyu Liang, Xiaohai Zhou, Sylvia M. Evans, and Ju Chen. "Filamin C is Essential for mammalian myocardial integrity." PLOS Genetics 19, no. 1 (January 27, 2023): e1010630. http://dx.doi.org/10.1371/journal.pgen.1010630.

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FLNC, encoding filamin C, is one of the most mutated genes in dilated and hypertrophic cardiomyopathy. However, the precise role of filamin C in mammalian heart remains unclear. In this study, we demonstrated Flnc global (FlncgKO) and cardiomyocyte-specific knockout (FlnccKO) mice died in utero from severely ruptured ventricular myocardium, indicating filamin C is required to maintain the structural integrity of myocardium in the mammalian heart. Contrary to the common belief that filamin C acts as an integrin inactivator, we observed attenuated activation of β1 integrin specifically in the myocardium of FlncgKO mice. Although deleting β1 integrin from cardiomyocytes did not recapitulate the heart rupture phenotype in Flnc knockout mice, deleting both β1 integrin and filamin C from cardiomyocytes resulted in much more severe heart ruptures than deleting filamin C alone. Our results demonstrated that filamin C works in concert with β1 integrin to maintain the structural integrity of myocardium during mammalian heart development.

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8

MENICONI, A. "How to survive myocardial rupture after myocardial infarction." Heart 84, no. 5 (November 1, 2000): 552. http://dx.doi.org/10.1136/heart.84.5.552.

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9

Kotecha, Monika Kantilal, Khurshid Merchant, Charmaine Jiahui Chan, Jonathan Tze Liang Choo, Krishna Revanna Gopagondanahalli, Dyan Zhewei Zhang, Teng Hong Tan, and Sreekanthan Sundararaghavan. "Endocardial Fibroelastosis as an Independent Predictor of Atrioventricular Valve Rupture in Maternal Autoimmune Antibody Exposed Fetus: A Systematic Review with Clinicopathologic Analysis." Diagnostics 13, no. 8 (April 20, 2023): 1481. http://dx.doi.org/10.3390/diagnostics13081481.

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Background: Neonatal lupus (NL) is a clinical syndrome that develops in the fetus as a result of maternal autoimmune antibodies. Congenital complete heart block (CHB) is the most common manifestation, while extranodal cardiac manifestations of NL, such as endocardial fibroelastosis (EFE) and myocarditis, are rare but more serious. Less is known about this atrioventricular valve rupture due to valvulitis as a consequence of maternal autoantibodies. We have described a case of cardiac neonatal lupus with an antenatally detected CHB patient who developed mitral and tricuspid valve chordal rupture at 45 days of age. We compared the cardiac histopathology and the fetal cardiac echocardiographic findings of this case with another fetus that was aborted after being antenatally diagnosed with CHB but without valvar rupture. A narrative analysis after a systematic review of the literature regarding atrioventricular valve apparatus rupture due to autoimmune etiology along with maternal characteristics, presentation, treatment, and outcome have been discussed in this article. Objectives: To describe published data on atrioventricular valve rupture in neonatal lupus, including clinical presentation, diagnostic evaluation, management, and outcomes. Methods: We conducted a PRISMA-compliant descriptive systematic examination of case reports that included accounts of lupus during pregnancy or in the newborn period that resulted in an atrioventricular valve rupture. We gathered information on the patient’s demographics, the details of the valve rupture and other comorbidities, the maternal therapy, the clinical course, and the results. We also used a standardized method to evaluate the cases’ quality. A total of 12 cases were investigated, with 11 cases drawn from 10 case reports or case series and 1 from our own experience. Results: Tricuspid valve rupture (50%) is more common than mitral valve rupture (17%). Unlike mitral valve rupture, which occurs postnatally, the timing of tricuspid valve rupture is perinatal. A total of 33% of the patients had concomitant complete heart block, while 75% of the patients had endocardial fibroelastosis on an antenatal ultrasound. Antenatal changes pertaining to endocardial fibroelastosis can be seen as early as 19 weeks of gestation. Patients with both valve ruptures generally have a poor prognosis, especially if they occur at close intervals. Conclusion: Atrioventricular valve rupture in neonatal lupus is rare. A majority of patients with valve rupture had antenatally detected endocardial fibroelastosis in the valvar apparatus. Appropriate and expedited surgical repair of ruptured atrioventricular valves is feasible and has a low mortality risk. Rupture of both atrioventricular valves occurring at close intervals carries a high mortality risk.

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10

Attasiriluk, Samrit. "Traumatic Heart Rupture: Case Series." Heart, Lung and Circulation 27 (2018): S547. http://dx.doi.org/10.1016/j.hlc.2018.04.126.

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11

Ivashchenko, Volodymyr S., Kostiantyn P. Chyzh, and Serhii A. Sokur. "Alternative Method of Surgical Treatment of Post-Infarction Left Ventricular Free Wall Rupture. Case Report." Ukrainian Journal of Cardiovascular Surgery 31, no. 3 (September 28, 2023): 134–38. http://dx.doi.org/10.30702/ujcvs/23.31(03)/ic016-134138.

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Acute myocardial infarction (AMI) is one of the causes of death in developed countries, despite the latest medical technologies. AMI is usually accompanied by numerous fatal complications. One of these complications is left ventricular free wall rupture (LVFWR). Myocardial rupture after AMI can occur from 1 day to 3 weeks after the infarction. Most ruptures occur 3-5 days after a heart attack. Left ventricular free wall rupture requires only emergency surgical intervention, which in most cases consists in applying U-shaped sutures or wrapping sutures using Teflon patch. The aim. To demonstrate an example of alternative surgical tactics and intraoperative management of a patient with myocardial infarction complicated by rupture of the free wall of the heart with the transition to cardiogenic shock. Case report. We presented the clinical case of patient V., 72 years old, who was delivered by ambulance on January 10, 2023 to the intensive care department of the National Amosov Institute of Cardiovascular Surgery (Kyiv, Ukraine). Main diagnosis: ischemic heart disease, acute coronary syndrome with ST-elevation myocardial infarction, cardiogenic shock. Conclusion. The only effective method of treatment of rupture of the free wall of the heart is surgical intervention. Using a sandwich patch with resection of necrotic areas of the myocardium and U-shaped sutures with Teflon patch passed from the side of the left ventricular cavity through the myocardium in the peri-infarct zone and through a patch from a vascular prosthesis allows hermetically sew up the LVFWR.

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12

Figueras, J. "Left ventricular free wall rupture: clinical presentation and management." Heart 83, no. 5 (May 1, 2000): 499–504. http://dx.doi.org/10.1136/heart.83.5.499.

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13

SINHA, M. "Sinus of Valsalva aneurysm rupture into the left atrium." Heart 85, no. 5 (May 1, 2001): 483. http://dx.doi.org/10.1136/heart.85.5.483.

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14

Katz, N. M. "Radiation induced valvulitis with late leaflet rupture." Heart 86, no. 6 (December 1, 2001): 20e—20. http://dx.doi.org/10.1136/heart.86.6.e20.

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15

Sanchez, P. L. "Acute left ventricular rupture after myocardial infarction." Heart 89, no. 2 (February 1, 2003): 138—a—138. http://dx.doi.org/10.1136/heart.89.2.138-a.

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16

Carvalho, Maria do Socorro Fernandes de. "“Toda a minha vida escrevi”: leitura de três livros de Melo e Castro / All My Life I Wrote”: Reading Three Books by Melo e Castro." Revista do Centro de Estudos Portugueses 40, no. 63 (April 8, 2020): 51. http://dx.doi.org/10.17851/2359-0076.40.63.51-58.

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Resumo: Este pequeno texto parte da constatação de recorrências literárias entre a Poesia Experimental de E. M. Melo e Castro e a Poesia Concreta brasileira, que tem origem no diálogo intertextual ocorrido no cerne das rupturas literárias das vanguardas nas décadas de 1950 e 1960 no Brasil e em Portugal. Trato dessas relações a partir da leitura de três livros de poesia escritos pelo poeta português: Queda livre, de 1961, Ideogramas, de 1962 e Poligonia do soneto, de 1963.Palavras-chave: experimentação; poesia concreta; vanguarda; ruptura; modernidade.Abstract: This text is based on the observation of literary recurrences between E. M. Melo e Castro’s Experimental Poetry and Brazilian Concrete Poetry, which originates from the intertextual dialogue that took place at the heart of the literary ruptures of the avant-garde in the 1950s and 1960s in Brazil and Portugal. I deal with these relations from the reading of three poetry books written by the Portuguese poet: Queda Livre, 1961, Ideogramas, 1962 and Poligonia do soneto, 1963.Keywords: experimentation; concrete poetry; avant-garde; rupture; modernity.

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17

Nikolic, Slobodan. "Forensic expertise of thoracic aorta, heart and pericardial injuries in car-occupant fatalities." Srpski arhiv za celokupno lekarstvo 137, no. 11-12 (2009): 627–31. http://dx.doi.org/10.2298/sarh0912627n.

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Introduction. Forensic expertise has not specified with certainty any specific injury among fatally injured frontal car-occupants in frontal car collisions. Objective. To establish if blunt injuries of thoracic aorta, heart, and pericardium could be used as predictors where the fatally injured car-occupant was at the moment of car-collision. Methods. A retrospective autopsy study was performed. The subjects were fatally injured car-drivers, front-seat and rearseat passengers in head-on car collisions. In each of them we analyzed the injuries of thoracic aorta, heart and pericardium. Results. 492 subjects were analyzed (378 male and 104 female): 239 car-drivers, 194 front-seat and 49 rear-seat passengers. The isthmus of aorta was the commonest site of simple blunt rupture among car-drivers and front-seat passengers. Among more than half of the observed subject, there was aortic blunt rupture as concomitant injury with heart and pericardium injuries. Heart and pericardium ruptures were most common among fatally injured car-drivers. Most frequently injured part of the heart was the right atrium. Injuries of thoracic aorta, heart and pericardium indicated a higher probability that the fatally injured would be the car-driver (?=0.818; df=2; p=0.011, ?=0.906; df=2; p=0.000, and ?=0.951; df=2; p=0.000); this was also pointed out by the rupture of the right atrium and multiple ruptures of the thoracic aorta (?=0.966; df=2; p=0.000 and ?=0.918; df=2; p=0.009). The concomitant injuries of the thoracic aorta with thoracic spine, sternum and heart pointed out that the injured person was the car-driver (?=0.971; df=4; p=0.007, ?=0.974; df=4; p=0.013 and ?=0.958; df=4; p=0.000), as well as the concomitant injuries of heart and sternal fracture (?=0.960; df=4; p=0.001). The probability of about 80% that the fatally injured person in head-on collisions was a car-driver was pointed out by concomitant blunt thoracic aorta rupture with fractured sternum and ribs (Wald. coeff.=8.611; df=1; p=0.003, and Wald. coeff.=3.875; df=1; p=0.049). Conclusion. The basic mechanism of the injury of thoracic organs is deceleration, as well as anteroposterior compression with caudorostral hyperextension. That is why thoracic organ injuries are mostly concomitant. Fatally injured car-drivers suffered more often of these injuries than other car-occupants. The probability that the fatally injured was the car-driver rises with the number of concomitant injuries of thoracic aorta, heart and pericardium, as well as with the fractured thoracic-cage bones.

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18

Ivanov, Igor, Aleksandra Lovrenski, Jadranka Dejanovic, Milovan Petrovic, Robert Jung, and Violetta Raffay. "Double heart rupture after acute myocardial infarction: A case report." Vojnosanitetski pregled 71, no. 12 (2014): 1151–54. http://dx.doi.org/10.2298/vsp1412151i.

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Introduction. Double heart rupture is a rare complication of acute myocardial infarction with high mortality. Case report. We presented a 67-year-old female patient with symptoms and signs of myocardial infarction, diagnosed with echocardiography, rupture of the septum, the presence of a thrombus and a small pericardial effusion. Soon after admission the patient died. Autopsy revealed tamponade and double myocardial rupture, free wall rupture and ventricular septal rupture, as a cause of death. Conclusion. This case highlights the need to evaluate patients with myocardial infarction, recurrent chest pain, echocardiographic signs of effusion and the presence of thrombus in the pericardium in terms of double rupture of the heart.

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19

Kato, Kazuyoshi, Kunihiro Mashiko, Shigeki Kushimoto, and Toshilumi Olsuka. "BLUNT TRAUMATIC RUPTURE OF THE HEART." Journal of Trauma: Injury, Infection, and Critical Care 35, no. 1 (July 1993): 164. http://dx.doi.org/10.1097/00005373-199307000-00052.

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20

Kato, Kazuyoshi, Shigeki Kushimoto, Kunihiro Mashiko, Hiroshi Henmi, Yasuhiro Yamamoto, and Toshibumi Otsuka. "BLUNT TRAUMATIC RUPTURE OF THE HEART." Journal of Trauma: Injury, Infection, and Critical Care 36, no. 6 (June 1994): 859–64. http://dx.doi.org/10.1097/00005373-199406000-00017.

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21

Beranek, Jiri T. "Preventive treatment of postinfarction heart rupture." Cardiovascular Drugs and Therapy 9, no. 1 (February 1995): 169–70. http://dx.doi.org/10.1007/bf00877758.

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22

Coisne, D. "Obstructive intramural coronary amyloidosis and papillary muscle rupture." Heart 89, no. 2 (February 1, 2003): 8e—8. http://dx.doi.org/10.1136/heart.89.2.e8.

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23

Verma, Divya, Sanjeev Kumar, and Premveetrag Sharma. "Intrapartum posterior wall rupture in unscarred uterus during labour augmentation with oxytocin; a case report." International Journal of Reproduction, Contraception, Obstetrics and Gynecology 11, no. 2 (January 28, 2022): 625. http://dx.doi.org/10.18203/2320-1770.ijrcog20220202.

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Intrapartum rupture of unscarred uterus during labour augmentation is an exceedingly rare occurrence with very few case reports of this entity in the literature. We intend to report such case to highlight the importance of constant vigilance during oxytocin infusion even in patients with unscarred uterus. The 27 years old lady who had a previous normal vaginal delivery, came in early labour at term gestation. Augmentation of labour was done with oxytocin. After 30 mins of starting oxytocin patient complained of severe pain abdomen and on CTG fetal heart dropped to 70 bpm. Despite all measures fetal heart rate did not pick up and patient was taken up for cesarean section. Intraoperatively, posterior uterine wall ruptured was found with baby and placenta lying in the abdominal cavity. Baby had a low Apgar score and died on third day. Uterine rupture repair was done and patient was discharged on 5th postoperative day. Although oxytocin has excellent safety record in unscarred uterus, the possibility of rupture uterus should be kept in mind in relevant clinical scenario.

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24

Ghawi, Hani, Kevin Engelhardt, Keith Dixon, Poonum Thankaval, Claudio Ramaciotti, Matthew S. Lemler, and Kristine J. Guleserian. "Sinus of Valsalva Aneurysm in a Patient With Mosaic Trisomy 13: Case Report and Brief Review of the Literature." World Journal for Pediatric and Congenital Heart Surgery 11, no. 4 (December 30, 2016): NP1—NP6. http://dx.doi.org/10.1177/2150135116682465.

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This report describes a unique case involving an obese 16-year-old boy with a mosaic form of trisomy 13 and no previous cardiac history who presented with a new murmur, hypertension, pleural effusions, and congestive heart failure in the context of sore throat and fever. Evaluation revealed a diagnosis of ruptured noncoronary sinus of Valsalva (SOV) aneurysm. The diagnosis and surgical management of a ruptured noncoronary SOV aneurysm in a pediatric patient are briefly outlined. An SOV aneurysm is an anatomic dilation of one of the sinuses of the aortic root. Aneurysmal dilation occurs more commonly in the right aortic sinus (70%-80%), compared to the noncoronary sinus (23%-25%), and more rarely the left coronary sinus (5%). Rupture of these aneurysms has been reported to be both spontaneous and secondary to physical exertion, hypertension, or trauma. Signs of rupture include a continuous murmur, patients may present with chest pain or with symptoms of acute congestive heart failure. Diagnosis, in this case, was made by transthoracic echocardiography with careful interpretation of color Doppler images.

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25

Kanna, M. "Giant left ventricular pseudoaneurysm following left ventricular rupture caused by myocardial infarction." Heart 88, no. 5 (December 1, 2002): 446. http://dx.doi.org/10.1136/heart.88.5.446.

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26

Kiziukevich, O., S. Spiridonov, A. Zhyhalkovich, and D. Isachkin. "SURGICAL TREATMENT OF PSEUDOANEURYSM OF THE FREE LEFT VENTRICLE WALL: A CLINICAL CASE." Emergency Cardiology and Cardiovascular Risks 5, no. 1 (2021): 1195–201. http://dx.doi.org/10.51922/2616-633x.2021.5.2.1195.

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Today diseases of the cardiovascular system are the leading cause of death in many countries. The key role in this pathology is played by ischemic heart disease. An extreme manifestation of ischemic heart disease - myocardial infarction is one of the main causes of complications and mortality in patients with ischemic heart disease. One of the most formidable complications of acute myocardial infarction is heart rupture, which most often leads to death, more than 60% of cases occurring in the prehospital stage. Many studies of similar groups of patients show a wide spread in assessing the incidence and mortality of this pathology. The development of myocardial ruptures has two frequency peaks: the first day and 5-7 days from the onset of AMI. The most common case is acute rupture of the free wall of the left ventricle with extensive hemorrhage in the pericardium leading to a fulminant death. The development of a pseudoaneurysm of the left ventricle is a very rare outcome of the myocardial rupture. There are no convincing data on the incidence of pseudoaneurysms as a result of myocardial infarction (according to many authors, it is less than 0.5% of all cases of myocardial infarction). This type of rupture is most favorable in terms of the possibility of providing assistance. The complexity of providing care to patients with pseudoaneurysms of the left ventricle lies in their low frequency of occurrence and often asymptomatic nature of the course, which complicates the diagnosis of this pathology. Timely diagnosis plays a key role in avoiding a fatal outcome, since the vast majority of pseudoaneurysms are extremely unstable and, except occasional cases, require urgent surgical intervention. This article describes a clinical case of a patient who underwent surgical treatment for pseudoaneurysm of the free wall of the left ventricle as a result of myocardial infarction. The article also presents a brief literature review of the available isolated data on risk factors for myocardial rupture, methods of diagnosis and treatment of this pathology.

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27

Peng, Hongmei, Jiang Xu, Xiao-Ping Yang, Kamal M. Kassem, Imane A. Rhaleb, Ed Peterson, and Nour-Eddine Rhaleb. "N-acetyl-seryl-aspartyl-lysyl-proline treatment protects heart against excessive myocardial injury and heart failure in mice." Canadian Journal of Physiology and Pharmacology 97, no. 8 (August 2019): 753–65. http://dx.doi.org/10.1139/cjpp-2019-0047.

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Myocardial infarction (MI) in mice results in cardiac rupture at 4–7 days after MI, whereas cardiac fibrosis and dysfunction occur later. N-acetyl-seryl-aspartyl-lysyl-proline (Ac-SDKP) has anti-inflammatory, anti-fibrotic, and pro-angiogenic properties. We hypothesized that Ac-SDKP reduces cardiac rupture and adverse cardiac remodeling, and improves function by promoting angiogenesis and inhibiting detrimental reactive fibrosis and inflammation after MI. C57BL/6J mice were subjected to MI and treated with Ac-SDKP (1.6 mg/kg per day) for 1 or 5 weeks. We analyzed (1) intercellular adhesion molecule-1 (ICAM-1) expression; (2) inflammatory cell infiltration and angiogenesis; (3) gelatinolytic activity; (4) incidence of cardiac rupture; (5) p53, the endoplasmic reticulum stress marker CCAAT/enhancer binding protein homology protein (CHOP), and cardiomyocyte apoptosis; (6) sarcoplasmic reticulum Ca2+ ATPase (SERCA2) expression; (7) interstitial collagen fraction and capillary density; and (8) cardiac remodeling and function. Acutely, Ac-SDKP reduced cardiac rupture, decreased ICAM-1 expression and the number of infiltrating macrophages, decreased gelatinolytic activity, p53 expression, and myocyte apoptosis, but increased capillary density in the infarction border. Chronically, Ac-SDKP improved cardiac structures and function, reduced CHOP expression and interstitial collagen fraction, and preserved myocardium SERCA2 expression. Thus, Ac-SDKP decreased cardiac rupture, ameliorated adverse cardiac remodeling, and improved cardiac function after MI, likely through preserved SERCA2 expression and inhibition of endoplasmic reticulum stress.

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Kojima, S. "Is preinfarction angina related to the presence or absence of coronary plaque rupture?" Heart 83, no. 1 (January 1, 2000): 64–68. http://dx.doi.org/10.1136/heart.83.1.64.

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Basso, C. "Fatal cardiac tamponade in a child due to rupture of a mycotic aneurysm." Heart 88, no. 1 (July 1, 2002): 89. http://dx.doi.org/10.1136/heart.88.1.89.

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30

Haq, Tawfiq Shahriar, Naharuma Aive Hyder Chowdhury, Abdul Mazid Khan, Jesmin Hossain, and Fazila Tun Nesa Malik. "Percutaneous Closure of Acquired Hole- First Case Report from Bangladesh." Bangladesh Heart Journal 35, no. 2 (April 7, 2021): 155–58. http://dx.doi.org/10.3329/bhj.v35i2.52903.

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Rupture sinus of Valsalva (RSOV) is an uncommon condition with a wide spectrum of presentation, ranging from an asymptomatic murmur to cardiogenic shock or even sudden cardiac death. Our case presented at 62 yrs with progressive onset of dyspnea, palpitation with deterioration of exercise capacity. Diagnosis of ruptured sinus of Valsalva was made by echocardiography, it was aneurysmal and opened into right ventricular outflow tract. Coronary artery disease was excluded by coronary angiogram. Probable cause of rupture was atherosclerosis.We closed percutaneously with ADO I device. The procedure was completed uneventfully. Patient discharged with duel antiplatelet and is on follow up. Bangladesh Heart Journal 2020; 35(2) : 155-158

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31

Pidello, Stefano, Erika Simonato, Fulvio Orzan, Simone Frea, Antonella Barreca, Mauro Rinaldi, and Massimo Boffini. "Interventricular Septal Rupture in a 62-Year-Old Man With Familial Amyloid Polyneuropathy." Texas Heart Institute Journal 47, no. 4 (January 20, 2020): 302–5. http://dx.doi.org/10.14503/thij-18-6799.

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Cardiac involvement in familial amyloid polyneuropathy consists of arrhythmias, conduction disturbances, and heart failure. To our knowledge, heart rupture has never been described in association with this condition. We report the case of a 62-year-old man with a 6-year history of refractory familial amyloid polyneuropathy who underwent liver transplantation. The operation was complicated by severe hypotension because the neuropathy involved the autonomic system. Perioperatively, the patient had a myocardial infarction, and during the next 10 days, a complete interventricular septal rupture developed, resulting in a systemic-to-pulmonary shunt. Coronary angiographic findings were normal. However, the shunt caused unstable hemodynamics, resulting in cardiogenic shock. An attempt to close the rupture percutaneously failed. The patient underwent successful heart transplantation 50 days later. Macroscopic examination of the explanted heart showed thickening of both ventricles, septal rupture, and a gray scar in the interventricular septum around the cavity. Histopathologic examination revealed intramural amyloid angiopathy. Our case shows that heart rupture can occur in patients with familial amyloid polyneuropathy who have no history of obstructive coronary artery disease, perhaps as a result of tissue fragility caused by amyloid angiopathy. Therefore, autonomic disturbances should be regarded with concern and promptly treated in the perioperative period.

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Kimura, Naoyuki, Atsushi Yamaguchi, Masashi Tanaka, Homare Okamura, Hideo Adachi, and Takashi Ino. "Postinfarction Heart Rupture of Posterior Wall Repaired by Covering Patch." Asian Cardiovascular and Thoracic Annals 16, no. 5 (October 2008): 407–9. http://dx.doi.org/10.1177/021849230801600513.

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A 46-year-old man underwent emergency surgery for heart rupture after acute infarction of the posterior wall. Echocardiography revealed limited myocardial thinning, so rather than sutureless repair, a covering patch was used in view of the risk of recurrent rupture. Postoperative echocardiography showed the myocardial thinning had progressed to a wide defect, and computed tomography demonstrated that the covering patch had prevented a repeat rupture.

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33

Tyagi, Shashank, Girish Tasgaonkar, and Rajesh Sukhadeve. "Rupture Of Heart: A Dilemma In Diagnosis." European Journal of Forensic Sciences 3, no. 1 (2016): 38. http://dx.doi.org/10.5455/ejfs.187844.

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34

Smith, Ann. "The Postop Heart: Case Example: Myocardial Rupture." American Journal of Nursing 91, no. 10 (October 1991): 25. http://dx.doi.org/10.2307/3426744.

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35

De Torres Alba, F., E. Refoyo Salicio, G. Guzman Martinez, M. Fernandez Velilla, A. Iniesta Manjavacas, N. Montoro Lopez, M. Torres Sanchez, E. Lopez De Sa, M. Moreno Yanguela, and J. L. Lopez Sendon. "Long-term outcomes of incomplete heart rupture." European Heart Journal 34, suppl 1 (August 2, 2013): P1330. http://dx.doi.org/10.1093/eurheartj/eht308.p1330.

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36

RUMISEK, JOHN D., MAX ROBINOWITZ, RENU VIRMANI, MICHAEL J. BARRY, and W. THEODORE STEUDEL. "Bioprosthetic Heart Valve Rupture Associated with Trauma." Journal of Trauma: Injury, Infection, and Critical Care 26, no. 3 (March 1986): 276–79. http://dx.doi.org/10.1097/00005373-198603000-00012.

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37

CALHOON, JOHN H., THOMAS H. HOFFMANN, J. KENT TRINKLE, P. KENT HARMAN, and FREDERICK L. GROVER. "Management of Blunt Rupture of the Heart." Journal of Trauma: Injury, Infection, and Critical Care 26, no. 6 (June 1986): 495–502. http://dx.doi.org/10.1097/00005373-198606000-00001.

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38

Beranek, Jiri T. "C-Reactive protein in postinfarction heart rupture." American Heart Journal 136, no. 3 (September 1998): 563–64. http://dx.doi.org/10.1016/s0002-8703(98)70237-6.

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39

Goldner, Alan. "Management of blunt rupture of the heart." Annals of Emergency Medicine 16, no. 2 (February 1987): 231. http://dx.doi.org/10.1016/s0196-0644(87)80027-6.

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40

Sundermann, T. "Acute heart insufficiency due to aortic rupture." Pferdeheilkunde Equine Medicine 25, no. 2 (2009): 119–23. http://dx.doi.org/10.21836/pem20090205.

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41

Weyrauch, Deland, Walter L. Kemp, and Mark Koponen. "Right Ventricle Rupture After Open Heart Surgery." American Journal of Forensic Medicine and Pathology 41, no. 1 (March 2020): 35–39. http://dx.doi.org/10.1097/paf.0000000000000523.

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42

Kurusz, Mark, Mark K. Girouard, and Paul S. Brown. "Coronary sinus rupture with retrograde cardioplegia." Perfusion 17, no. 1 (January 2002): 77–80. http://dx.doi.org/10.1191/0267659102pf534cr.

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Abstract:

Coronary sinus (CS) rupture occurring during retrograde cardioplegia (RCP) is a rare complication. Patients with left ventricular hypertrophy are at higher risk for injury to the CS. The patient was a 66-year-old female with hypertension, ischemic cardiomyopathy and dysrhythmias, who had evidence of an anterior wall myocardial infarction, congestive heart failure and angina. During coronary artery bypass surgery, antegrade cardioplegia was initially administered, but aortic insufficiency prevented adequate myocardial cooling. RCP was then administered and the heart cooled appropriately. After approximately 300 ml of blood cardioplegic solution had been given, the CS pressure suddenly dropped from 30 mmHg to zero. RCP administration was stopped, and the surgeon palpated a hematoma over the area of the CS, which later ruptured upon rotation of the heart. A primary repair could not be performed, so a pericardial patch was placed over the area of disruption, which appeared to provide adequate hemostasis. The patient was weaned from cardiopulmonary bypass (CPB), but began to bleed freely from the CS distal to the pericardial patch. The patient was placed back on CPB to allow further repair of the CS, but the tissues were thin and friable and the ventricle disassociated from the ventricular septum. The situation was deemed not salvageable and further attempts at repair were stopped. The perfusionist should monitor infusion pressures and the CS waveform during RCP delivery. Changes in the waveform may indicate cannula malposition, loss of balloon seal, or, more rarely, CS rupture; such changes should prompt immediate cessation of RCP delivery.

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43

Golestaneh, Safieh, Mohammad Amin Kashef, William L. Hiser, Amir S. Lotfi, and Timothy G. Egan. "Ruptured Sinus of Valsalva Aneurysm Associated with Situs Ambiguus, Isolated Levocardia, and Polysplenia." Texas Heart Institute Journal 44, no. 6 (December 1, 2017): 416–19. http://dx.doi.org/10.14503/thij-16-6106.

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Aneurysm of the sinus of Valsalva, a rare cardiac condition, results from dilation of an aortic sinus. Sudden aneurysm rupture can trigger rapidly progressive heart failure. We discuss the case of a 57-year-old woman with situs ambiguus, isolated levocardia, and polysplenia who presented with acute-onset heart failure. Transesophageal echocardiograms revealed an aneurysm of the right coronary sinus of Valsalva that had ruptured into the right atrial cavity. The patient underwent successful surgical repair. To our knowledge, this is the first report of a sinus of Valsalva aneurysm in a patient with this combination of congenital abnormalities. We briefly review the association between congenital heart disease, situs ambiguus, and ciliary dysfunction.

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Bigg, Hugh, Elijah Bolin, Dala Zakaria, and Renee Bornemeier. "Ruptured Sinus of Valsalva Aneurysm with Resultant Myocardial Pouch Formation in the Fetal Heart—A Diagnostic Challenge." Journal of Cardiovascular Development and Disease 11, no. 1 (January 14, 2024): 23. http://dx.doi.org/10.3390/jcdd11010023.

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Sinus of Valsalva aneurysms (SVAs) are infrequently seen in the pediatric population. When these aneurysms rupture, a significant hemodynamic burden is placed on the heart and increases the likelihood of cardiac failure. Here, we report a case of a ruptured SVA into the ventricular myocardium in a fetus with a form of double-inlet left ventricle. To the best of our knowledge, this has not previously been described.

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Tsui, K.-L. "Coronary rupture as a rare cause of acute myocardial ischaemia two months after angioplasty and stenting." Heart 89, no. 11 (November 1, 2003): 1315. http://dx.doi.org/10.1136/heart.89.11.1315.

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46

Osula, S. "Cardiac rupture caused by Staphylococcus aureus septicaemia and pericarditis: an incidental finding." Heart 85, no. 3 (March 1, 2001): 4e—4. http://dx.doi.org/10.1136/heart.85.3.e4.

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47

FULDA, G., C. M. E. BRATHWAITE, A. RODRIGUEZ, S. Z. TURNEY, C. M. DUNHAM, and R. A. COWLEY. "Blunt Traumatic Rupture of the Heart and Pericardium:." Journal of Trauma: Injury, Infection, and Critical Care 31, no. 2 (February 1991): 167–73. http://dx.doi.org/10.1097/00005373-199131020-00003.

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48

Ridgeway, Jeffrey J., Darin L. Weyrich, and Thomas J. Benedetti. "Fetal Heart-Rate Changes Associated With Uterine Rupture." Obstetrics & Gynecology 101, Supplement (April 2003): 61S—62S. http://dx.doi.org/10.1097/00006250-200304001-00144.

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Fulda, C., C. E. M. Brathvalle, A. Rodriguez, S. I. Turnay, and R. A. Cowley. "BLUNT TRAUMATIC RUPTURE OF THE HEART AND PERICARDIUM." Journal of Trauma: Injury, Infection, and Critical Care 29, no. 7 (July 1989): 1029. http://dx.doi.org/10.1097/00005373-198907000-00037.

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50

FULDA, G., C. M. E. BRATHWAITE, A. RODRIGUEZ, S. Z. TURNEY, C. M. DUNHAM, and R. A. COWLEY. "Blunt Traumatic Rupture of the Heart and Pericardium:." Journal of Trauma: Injury, Infection, and Critical Care 31, no. 2 (February 1991): 167–73. http://dx.doi.org/10.1097/00005373-199102000-00003.

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