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Books on the topic 'Heart valve disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 19 February 2023

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1

Zamorano, Jose, Patrizio Lancellotti, Luc Pierard, and Philippe Pibarot, eds. Heart Valve Disease. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-23104-0.

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2

Valvular heart disease. Dordrecht: Humana Press, 2009.

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3

1951-, Al Zaibag Muayed, and Gomez-Duran Carlos, eds. Valvular heart disease. New York: M. Dekker, 1994.

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4

Otto, Catherine M. Valvular heart disease. 2nd ed. Philadelphia, PA: Saunders, 2002.

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5

Valvular heart disease. 2nd ed. Philadelphia, Pa: W.B. Saunders Co., 2004.

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6

Valvular heart disease. Philadelphia: W.B. Saunders, 1999.

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7

M, Dunn Jeffrey, ed. Cardiac valve disease in children. New York, N.Y: Elsevier, 1988.

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8

Acar, Jean. Textbook of acquired heart valve disease. London: ICR, 1995.

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9

Acar, Jean. Textbook of acquired heart valve disease. London: ICR, 1995.

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10

Giamberti, Alessandro, and Massimo Chessa, eds. The Tricuspid Valve in Congenital Heart Disease. Milano: Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5400-4.

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11

Droogmans, Steven. Drug-induced valvular heart disease. Hauppauge, N.Y: Nova Science Publishers, 2010.

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12

I, Ionescu Marian, and Cohn Lawrence H. 1937-, eds. Mitral valve disease: Diagnosis and treatment. London: Butterworths, 1985.

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13

Structural heart disease interventions. Philadelphia: Wolters Kluwer Health Lippincott Williams & Wilkins, 2012.

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14

Einthoven Meeting on Past and Present Cardiology (4th 1985 Leiden, Netherlands). Theme, valvular disease: Diagnostic, anatomical, and surgical aspects. Edited by Arntzenius Alexander C, Dunning A. J, and Snellen H. A. Assen, The Netherlands: Van Gorcum, 1986.

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15

The heart disease sourcebook. Los Angeles: Lowell House, 1997.

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16

Goldsmith, Ira Robert Anup. A clinical and pathophysiological study of thrombogenesis in patients with heart valve disease and prosthetic heart valves. Birmingham: University of Birmingham, 2000.

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17

Karen, Bellenir, ed. Heart diseases and disorders sourcebook: Basic consumer health information about heart attacks, angina, rhythm disorders, heart failure, valve disease, congenital heart disorders, and more ... 2nd ed. Detroit, MI: Omnigraphics, 2000.

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18

G, Julian Desmond, ed. Diseases of the Cardiac Valves. Edinburgh: Churchill Livingstone, 1989.

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19

Muraru, Denisa, Ashraf M. Anwar, and Jae-Kwan Song. Heart valve disease: tricuspid valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0037.

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The tricuspid valve is currently the subject of much interest from echocardiographers and surgeons. Functional tricuspid regurgitation is the most frequent aetiology of tricuspid valve pathology, is characterized by structurally normal leaflets, and is due to annular dilation and/or leaflet tethering. A primary cause of tricuspid regurgitation with/without stenosis can be identified only in a minority of cases. Echocardiography is the imaging modality of choice for assessing tricuspid valve diseases. It enables the cause to be identified, assesses the severity of valve dysfunction, monitors the right heart remodelling and haemodynamics, and helps decide the timing for surgery. The severity assessment requires the integration of multiple qualitative and quantitative parameters. The recent insights from three-dimensional echocardiography have greatly increased our understanding about the tricuspid valve and its peculiarities with respect to the mitral valve, showing promise to solve many of the current problems of conventional two-dimensional imaging. This chapter provides an overview of the current state-of-the-art assessment of tricuspid valve pathology by echocardiography, including the specific indications, strengths, and limitations of each method for diagnosis and therapeutic planning.
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20

Miller, Owen I., and Werner Budts. Heart valve disease: pulmonary valve disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0038.

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Congenital abnormalities of the pulmonary valve (PV) are common either as a single lesion or in the context of more complex congenital lesions where abnormalities of the PV play a major role in the cardiac physiology. Transthoracic echocardiographic (TTE) imaging of the PV is relatively straightforward in the normally connected heart due to its anterior position close to common sonographic windows. Imaging of the abnormally positioned PV requires modifications to standard projections and may be better demonstrated by a transoesophageal (TOE) or three-dimensional (3D) echocardiographic approach. Standard 3D TTE may offer advantages in surgical planning for an abnormally positioned pulmonary valve in complex congenital anatomy and 3D TOE may add value to the demonstration of abnormalities of the subpulmonary right ventricular outflow tract.
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21

Lancellotti, Patrizio, and Bernard Cosyns. Heart Valve Disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0007.

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Echocardiography plays a major role in the evaluation, monitoring and decision making of patients with valvular heart disease. This chapter examines the aetiologies, haemodynamic measurements, and various consequences in aortic, mitral and pulmonary valve stenosis. It also describes how to assess patients with valvular regurgitation (mitral, aortic and pulmonary), valvular prosthesis and definite or suspected infective endocarditis. For each condition, echocardiographic features of poor prognosis, including complications, embolic risk, and the timing for surgery are discussed. Indications for transoesophageal echocardiography and 3D echocardiography are highlighted, especially when a decision of valve repair is envisioned. The timing echocardiographic monitoring of patients with valvular heart disease is also described.
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22

Butchart, Eric G., Christa Gohllke-Barwolf, Manuel Antunes, and Roger J. C. Hall, eds. Heart Valve Disease. CRC Press, 2006. http://dx.doi.org/10.1201/b14629.

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23

H, Greenberg Barry, and Murphy Edward MD, eds. Valvular heart disease. Littleton, Mass: PSG Pub. Co., 1987.

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24

Unger, Philippe, and Gerald Maurer. Heart valve disease: mixed valve disease, multiple valve disease, and others. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0039.

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Multiple and mixed heart valve disease are highly prevalent. Echocardiography is the cornerstone technique for imaging these patients. As with patients with single-valve stenosis or regurgitation, one should aim to evaluate the aetiology, the mechanism(s) of dysfunction, as well as the consequences and the possibility of repair. There are, however, specific issues, which include the followings: (1) the lack of published data; (2) most indices of valvular regurgitation and of stenosis severity have been validated in patients with single-valve/single-lesion disease; and (3) the haemodynamic interactions that may affect the severity and the diagnosis of these lesions. A global assessment of the consequences of the lesions is of the utmost importance in the decision-making process: whereas only severe regurgitation or stenosis is usually considered for surgery by current guidelines in a single-valve lesion, the combination of two or more less-than-severe lesions causing symptoms, left ventricular dysfunction, and/or pulmonary pressure increase may warrant surgery. This chapter focuses on the echocardiographic assessment of these sometimes complex lesions, emphasizing some pitfalls and tips to take into account when managing these patients.
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25

Baumgartner, Helmut, Stefan Orwat, Elif Sade, and Javier Bermejo. Heart valve disease (aortic valve disease): aortic stenosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0032.

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Echocardiography has become the gold standard for the assessment of patients with aortic stenosis (AS). It allows morphological assessment of the aortic valve and provides information on the aetiology of the disease. The quantification of AS includes primarily the measurement of transaortic jet velocities and gradients as well as the calculation of the valve area, thus combining flow-dependent and relatively flow-independent variables. Awareness of potential pitfalls is fundamental when assessing these variables. Haemodynamic consequences of AS on left ventricular (LV) size, wall thickness, and function as well as associated valve lesions and estimates of pulmonary artery pressure are required for the comprehensive evaluation of the disease. In the setting of classical low-flow–low-gradient AS with reduced LV systolic function, low-dose dobutamine echocardiography is of particular diagnostic and prognostic importance. The entity of severe low-flow–low-gradient AS in the presence of preserved LV function remains a particular diagnostic challenge. For accurate differentiation from pseudo-severe AS or misclassified moderate AS, an integrated approach including additional variables such as the extent of valve calcification by computed tomography may be required. In addition to the assessment of AS aetiology and quantification of its severity, echocardiography can provide predictors of outcome that may have a major impact on the decision for intervention.
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26

Tribouilloy, Christophe, Patrizio Lancellotti, Ferande Peters, José Juan Gómez de Diego, and Luc A. Pierard. Heart valve disease (aortic valve disease): aortic regurgitation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0033.

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Echocardiography is the cornerstone examination for the assessment of aortic regurgitation (AR): it provides reliable evaluation of the aortic valve and allows diagnosis and identification of the mechanism of regurgitation. The specific aetiology of the disease can be identified in the majority of cases. A combination of quantitative and quantitative Doppler and two-dimensional (2D) echocardiographic parameters allows the evaluation of the severity of AR and determination of the haemodynamic and left ventricular function repercussions. Echocardiography allows the detection of associated lesions of the aortic root or other valves. In symptomatic patients, echocardiography is essential to confirm the severity of AR. In asymptomatic patients with moderate or severe AR, echocardiography is essential for regular follow-up, by providing precise and reproducible measurements of LV dimensions and function, and for identifying patients who should be considered for elective surgical intervention. In most cases, transthoracic echocardiography (TTE) provides all of the necessary information and transoesophageal echocardiography in usually not required. Real-time three-dimensional (3D) TTE can be complementary to 2D echocardiography for the assessment of the mechanism and quantification of AR by increasing the level of confidence, especially when 2D echocardiographic data are inconclusive or discordant with clinical findings. Tissue Doppler imaging and especially the speckle tracking method are promising approaches to detect early LV dysfunction in patients with asymptomatic severe AR. Echocardiography is therefore the key examination for the assessment of AR and at the centre of the strategic discussion concerning the indications and timing of surgery.
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27

Popescu, Bogdan A., Shantanu P. Sengupta, Niloufar Samiei, and Anca D. Mateescu. Heart valve disease (mitral valve disease): mitral stenosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0035.

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The most common cause of mitral stenosis (MS) is rheumatic fever followed by degenerative MS. Echocardiography is the key method to diagnose and evaluate MS. Echocardiographic findings are closely related to aetiology. In rheumatic disease echocardiography shows thickening of leaflet tips with restricted opening caused by commissural fusion resulting in ‘doming’ of the mitral valve in diastole. Quantitation of MS severity includes measuring mitral valve area (MVA) by planimetry (anatomical area, by two-/three-dimensional echo), or by the pressure half-time (PHT) method (functional area, by Doppler), and the mean pressure gradient. Planimetry is considered the reference method to determine MVA as it is relatively load independent. The PHT method is widely used due to its simplicity, but different factors influence the relationship between PHT and MVA. Other indices of MS severity are rarely used in clinical practice. Echocardiography also helps in the assessment of consequences of MS, and of associated valvular lesions. Exercise Doppler is recommended when there is discrepancy between the resting echocardiography findings and the clinical picture. Echocardiography is crucial in determining the timing and type of intervention in patients with MS. When considering percutaneous mitral commissurotomy (PMC) valve morphology should be comprehensively evaluated for mobility, thickness, calcifications, and subvalvular apparatus. The echo findings may determine the suitability for PMC, guide the procedure, and assess its results.
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28

Lancellotti, Patrizio, Raluca Dulgheru, Mani Vannan, and Kiyoshi Yoshida. Heart valve disease (mitral valve disease): mitral regurgitation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0036.

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Mitral regurgitation (MR) is increasingly prevalent in Europe. Echocardiography has a key role in the diagnosis and management of patients with MR. Each echocardiographic study in patients with MR should aim to characterize mitral valve morphology, identify the mechanism of valve dysfunction, quantify the severity of MR, and give hints regarding the aetiology of the disease affecting the valve. Assessment of MR severity should be based on a step-wise approach including two-dimensional-derived Doppler data and, when available, data derived from three-dimensional echocardiography. MR assessment by quantitative methods should be implemented in each patient when possible. It is imperative not only to quantify the MR severity, but also to assess its consequences on the left ventricle, left atrium, and pulmonary vascular bed and to put everything into the clinical context (presence of symptoms, individual risk assessment, etc.) before taking any decision to correct the valvular incompetence. A rigorous echocardiographic study and a correct interpretation in the individual clinical context are needed to decide if the patient should be operated on or followed up closely. Exercise stress echocardiography, when appropriate, should be part of the evaluation algorithm in patients with both primary and secondary MR, as it has proved to be useful in individual risk stratification.
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29

H, Rahimtoola Shahbudin, ed. Valvular heart disease. Philadelphia: Current Medicine, 1997.

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30

Valvular Heart Disease. 3rd ed. Lippincott Williams & Wilkins, 2000.

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31

Wang, Andrew, and Thomas M. Bashore. Valvular Heart Disease. Humana, 2012.

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32

Otto, Catherine M. Valvular Heart Disease. 2nd ed. Saunders, 2003.

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33

Obi, Dr Bennett Onyebuchukwu. Health Adviser: Heart Valve Disease. Createspace Independent Publishing Platform, 2016.

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34

Dx/Rx: Valvular Heart Disease (Dx/Rx Series). Jones & Bartlett Publishers, 2004.

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35

Basso, Cristina, Gaetano Thiene, and Siew Yen Ho. Heart valve disease (aortic valve disease): anatomy and pathology of the aortic valve. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0031.

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The gross features of the aortic valve apparatus, consisting of three semilunar leaflets, three interleaflet triangles, three commissures, and the aortic wall, are discussed both in terms of normal and pathological anatomy. The concept of aortic annulus and the relationship of the aortic valve with the coronary arteries, the membranous septum, and conduction system and the mitral valve are addressed. When dealing with pathology, the chapter focuses on the main distinctive features of aortic valve stenosis and aortic valve incompetence. Regarding the former, the abnormalities reside in the cusps, either two or three in number, with cusp thickening, and calcification with or without commissural fusion (thus distinguishing senile and chronic rheumatic valve disease); in the latter, the gross changes can affect either the cusps (infective endocarditis with tissue perforation/laceration and rheumatic valve disease with tissue retraction) or the aortic wall (ascending aorta aneurysm either inflammatory or degenerative). The distinctive gross abnormalities in the various conditions are illustrated.
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36

La Canna, Giovanni. Heart valve disease (mitral valve disease): anatomy and morphology of the mitral valve. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0034.

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The mitral valve is a complex anatomical structure that includes the valve tissue (leaflets), the left atrioventricular junction (annulus), and the valve suspension system (chordae tendineae, papillary muscles, and left ventricle). Its functional anatomy can be analysed using two- and three-dimensional transthoracic and transoesophageal echocardiography. Based on certain hallmarks (commissures, clefts), in vivo mitral valve tissue anatomy can be accurately categorized. In addition, three-dimensional reconstruction provides a quantitative model for comprehensive valve analysis. This chapter describes the anatomy and morphology of the mitral valve, including the subvalvular suspension system and functional anatomy and dynamics of the mitral annulus.
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37

Otto, Catherine M., and Robert O. Bonow. Valvular Heart Disease: A Companion to Braunwald's Heart Disease. Elsevier, 2020.

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38

M, Otto Catherine, Bonow Robert O, and Otto Catherine M, eds. Valvular heart disease: A companion to Braunwald's heart disease. 3rd ed. Philadelphia, PA: Saunders/Elsevier, 2009.

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39

C, Wells F., and Shapiro Leonard M, eds. Mitral valve disease. Oxford: Butterworth-Heinemann, 1996.

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40

Chessa, Massimo, and Alessandro Giamberti. Tricuspid Valve in Congenital Heart Disease. Springer Milan, 2016.

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41

Chessa, Massimo, and Alessandro Giamberti. Tricuspid Valve in Congenital Heart Disease. Springer London, Limited, 2014.

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42

Kovanen, Petri T., and Magnus Bäck. Valvular heart disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0015.

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The heart valves, which maintain a unidirectional cardiac blood flow, are covered by endothelial cells and structurally composed by valvular interstitial cells and extracellular matrix. Valvular heart disease can be either stenotic, causing obstruction of the valvular flow, or regurgitant, referring to a back-flow through the valve. The pathophysiological changes in valvular heart disease include, for example, lipid and inflammatory cell infiltration, calcification, neoangiogenesis, and extracellular matrix remodelling. The present chapter addresses the biology of the aortic and mitral valves, and the pathophysiology of aortic stenosis and mitral valve prolapse.
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43

Aikawa, Elena, Adrian Chester, Laura Iop, and Roney Orismar Sampaio, eds. Insights in Heart Valve Disease: 2021. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-658-1.

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44

Valvular Heart Disease : A Companion to Braunwald's Heart Disease: Expert Consult - Online and Print. Saunders, 2013.

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45

Otto, Catherine M., and Robert O. Bonow. Valvular Heart Disease: A Companion to Braunwald's Heart Disease E-Book. Elsevier - Health Sciences Division, 2013.

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46

Chessa, Massimo, and Alessandro Giamberti. The Tricuspid Valve in Congenital Heart Disease. Springer, 2014.

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47

(Editor), James T. Willerson, Jay N. Cohn (Editor), Hugh A. McAllister (Editor), Hisao Manabe (Editor), and Chikao Yutani (Editor), eds. Atlas of Valvular Heart Disease: Clinical and Pathologic Aspects. Churchill Livingstone, 1998.

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48

1939-, Willerson James T., Cohn Jay N, McAllister Hugh A, Manabe Hisao 1921-, and Yutani Chikao, eds. Atlas of valvular heart disease: Clinical and pathologic aspects. New York: Churchill Livingstone, 1998.

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49

Lancellotti, Patrizio, Jose Zamorano, Luc Pierard, and Philippe Pibarot. Heart Valve Disease: State of the Art. Springer International Publishing AG, 2020.

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50

Lancellotti, Patrizio, Jose Zamorano, Luc Pierard, and Philippe Pibarot. Heart Valve Disease: State of the Art. Springer, 2019.

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