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Relevant bibliographies by topics / HeLa cells; Osmotic cell swelling; Taurine

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Academic literature on the topic 'HeLa cells; Osmotic cell swelling; Taurine'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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Journal articles on the topic "HeLa cells; Osmotic cell swelling; Taurine"

1

Hall, J. A., J. Kirk, J. R. Potts, C. Rae, and K. Kirk. "Anion channel blockers inhibit swelling-activated anion, cation, and nonelectrolyte transport in HeLa cells." American Journal of Physiology-Cell Physiology 271, no. 2 (1996): C579—C588. http://dx.doi.org/10.1152/ajpcell.1996.271.2.c579.

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The effect of osmotic cell swelling on the permeability of HeLa cells to a range of structurally unrelated solutes including taurine, sorbitol, thymidine, choline, and K+ (96Rb+) was investigated. For each solute tested, reduction in the osmolality of the medium from 300 to 200 mosmol/kgH2O caused a significant increase in the unidirectional influx rate. In each case, the osmotically activated transport component was nonsaturable up to external substrate concentrations of 50 mM. Inhibitors of the swelling-activated anion channel of HeLa cells [quinine, 4,4'-diisothiocyanostilbene-2,2'-disulfon

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Stutzin, A., A. L. Eguiguren, L. P. Cid, and F. V. Sepulveda. "Modulation by extracellular Cl- of volume-activated organic osmolyte and halide permeabilities in HeLa cells." American Journal of Physiology-Cell Physiology 273, no. 3 (1997): C999—C1007. http://dx.doi.org/10.1152/ajpcell.1997.273.3.c999.

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Organic osmolyte and halide permeability pathways activated in epithelial HeLa cells by osmotically induced cell swelling were studied using electrophysiological and radiotracer efflux techniques. On hypotonic challenge, HeLa cells responded by activating an efflux pathway for [3H]taurine and a swelling-induced outwardly rectifying Cl- channel. Removal of extracellular Cl-, or its replacement by a less permeable anion, enhanced taurine efflux and decreased the inward current (Cl- efflux). The effect of Cl- removal on taurine efflux was not a consequence of changes in membrane potential. The de

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Numata, Tomohiro, Takahiro Shimizu, and Yasunobu Okada. "TRPM7 is a stretch- and swelling-activated cation channel involved in volume regulation in human epithelial cells." American Journal of Physiology-Cell Physiology 292, no. 1 (2007): C460—C467. http://dx.doi.org/10.1152/ajpcell.00367.2006.

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Stretch- and swelling-activated cation (SSAC) channels play essential roles not only in sensing and transducing external mechanical stresses but also in regulating cell volume in living cells. However, the molecular nature of the SSAC channel has not been clarified. In human epithelial HeLa cells, single-channel recordings in cell-attached and inside-out patches revealed expression of a Mg2+- and Gd3+-sensitive nonselective cation channel that is exquisitely sensitive to membrane stretch. Whole cell recordings revealed that the macroscopic cationic currents exhibit transient receptor potential

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Tomassen, Sebastian F. B., Durk Fekkes, Hugo R. de Jonge, and Ben C. Tilly. "Osmotic swelling-provoked release of organic osmolytes in human intestinal epithelial cells." American Journal of Physiology-Cell Physiology 286, no. 6 (2004): C1417—C1422. http://dx.doi.org/10.1152/ajpcell.00468.2003.

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Human Intestine 407 cells respond to osmotic cell swelling by the activation of Cl−- and K+-selective ionic channels, as well as by stimulating an organic osmolyte release pathway readily permeable to taurine and phosphocholine. Unlike the activation of volume-regulated anion channels (VRAC), activation of the organic osmolyte release pathway shows a lag time of ∼30–60 s, and its activity persists for at least 8–12 min. In contrast to VRAC activation, stimulation of organic osmolyte release did not require protein tyrosine phosphorylation, active p21rho, or phosphatidylinositol 3-kinase activi

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Stutzin, Andrés, Rubén Torres, Macarena Oporto, et al. "Separate taurine and chloride efflux pathways activated during regulatory volume decrease." American Journal of Physiology-Cell Physiology 277, no. 3 (1999): C392—C402. http://dx.doi.org/10.1152/ajpcell.1999.277.3.c392.

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Organic osmolyte and halide permeability pathways activated in epithelial HeLa cells by cell swelling were studied by radiotracer efflux techniques and single-cell volume measurements. The replacement of extracellular Cl− by anions that are more permeant through the volume-activated Cl− channel, as indicated by electrophysiological measurements, significantly decreased taurine efflux. In the presence of less-permeant anions, an increase in taurine efflux was observed. Simultaneous measurement of the125I, used as a tracer for Cl−, and [3H]taurine efflux showed that the time courses for the two

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Basavappa, Srisaila, Stine F. Pedersen, Nanna K. Jørgensen, J. Clive Ellory, and Else K. Hoffmann. "Swelling-Induced Arachidonic Acid Release via the 85-kDa cPLA2 in Human Neuroblastoma Cells." Journal of Neurophysiology 79, no. 3 (1998): 1441–49. http://dx.doi.org/10.1152/jn.1998.79.3.1441.

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Basavappa, Srisaila, Stine F. Pedersen, Nanna K. Jørgensen, J. Clive Ellory, and Else K. Hoffmann. Swelling-induced arachidonic acid release via the 85-kDa cPLA2 in human neuroblastoma cells. J. Neurophysiol. 79: 1441–1449, 1998. Arachidonic acid or its metabolites have been implicated in the regulatory volume decrease (RVD) response after hypotonic cell swelling in some mammalian cells. The present study investigated the role of arachidonic acid (AA) during RVD in the human neuroblastoma cell line CHP-100. During the first nine minutes of hypo-osmotic exposure the rate of 3H-arachidonic acid

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Avella, Martine, Olivier Ducoudret, Didier F. Pisani, and Philippe Poujeol. "Swelling-activated transport of taurine in cultured gill cells of sea bass: physiological adaptation and pavement cell plasticity." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 296, no. 4 (2009): R1149—R1160. http://dx.doi.org/10.1152/ajpregu.90615.2008.

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We have investigated volume-activated taurine transport and ultrastructural swelling response of sea bass gill cells in culture, assuming that euryhaline fish may have developed particularly efficient mechanisms of salinity adaptation. In vivo, when sea basses were progressively transferred from seawater to freshwater, we noticed a decrease in blood osmotic pressure. When gill cells in culture were subjected to 30% hypotonic shock, we observed a five-fold stimulation of [3H]taurine efflux. This transport was reduced by various anion channel inhibitors with the following efficiency: 5-nitro-2-(

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Lambert, Ian Henry, Jane Vendelbo Jensen, and Per Amstrup Pedersen. "mTOR ensures increased release and reduced uptake of the organic osmolyte taurine under hypoosmotic conditions in mouse fibroblasts." American Journal of Physiology-Cell Physiology 306, no. 11 (2014): C1028—C1040. http://dx.doi.org/10.1152/ajpcell.00005.2014.

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Mammalian target of rapamycin (mTOR) is a serine/threonine kinase that modulates translation in response to growth factors and alterations in nutrient availability following hypoxia and DNA damage. Here we demonstrate that mTOR activity in Ehrlich Lettré ascites (ELA) cells is transiently increased within minutes following osmotic cell swelling and that inhibition of phosphatidylinositol-3-phosphatase (PTEN) counteracts the upstream phosphatidylinositol kinase and potentiates mTOR activity. PTEN inhibition concomitantly potentiates swelling-induced taurine release via the volume-sensitive tran

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Thoroed, S., M. Soergaard, E. Cragoe, and K. Fugelli. "The osmolality-sensitive taurine channel in flounder erythrocytes is strongly stimulated by noradrenaline under hypo-osmotic conditions." Journal of Experimental Biology 198, no. 2 (1995): 311–24. http://dx.doi.org/10.1242/jeb.198.2.311.

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Stimulation of flounder erythrocytes by noradrenaline under isosmotic conditions (330 mosmol kg-1) and physiological Na+ concentration (113 mmol l-1) caused swelling of the cells. The EC50 of this cell swelling was 0.65 &micro;mol l-1 noradrenaline. The effect of the noradrenaline-induced cell swelling on the taurine channel under isosmotic conditions was negligible. However, when the cells were stimulated by noradrenaline (1.0 &micro;mol l-1) before, simultaneously with or after reduction of osmolality (255 mosmol kg-1), the volume regulatory efflux of taurine mediated by the taurine

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Pedersen, Stine F., Kristian A. Poulsen, and Ian H. Lambert. "Roles of phospholipase A2 isoforms in swelling- and melittin-induced arachidonic acid release and taurine efflux in NIH3T3 fibroblasts." American Journal of Physiology-Cell Physiology 291, no. 6 (2006): C1286—C1296. http://dx.doi.org/10.1152/ajpcell.00325.2005.

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Osmotic swelling of NIH3T3 mouse fibroblasts activates a bromoenol lactone (BEL)-sensitive taurine efflux, pointing to the involvement of a Ca2+-independent phospholipase A2 (iPLA2) (Lambert IH. J Membr Biol 192: 19–32, 2003). We report that taurine efflux from NIH3T3 cells was not only increased by cell swelling but also decreased by cell shrinkage. Arachidonic acid release to the cell exterior was similarly decreased by shrinkage yet not detectably increased by swelling. NIH3T3 cells were found to express cytosolic calcium-dependent cPLA2-IVA, cPLA2-IVB, cPLA2-IVC, iPLA2-VIA, iPLA2-VIB, and

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Dissertations / Theses on the topic "HeLa cells; Osmotic cell swelling; Taurine"

1

Hall, James Anthony. "Swelling-activated transport of diverse solutes in mammalian cells." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.320647.

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Book chapters on the topic "HeLa cells; Osmotic cell swelling; Taurine"

1

Lambert, Ian H., and Francisco V. Sepúlveda. "Swelling-Induced Taurine Efflus from Hela Cells: Cell Volume Regulation." In Advances in Experimental Medicine and Biology. Springer US, 2002. http://dx.doi.org/10.1007/0-306-46838-7_54.

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