Relevant bibliographies by topics / Helicobacter hepaticus

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Academic literature on the topic 'Helicobacter hepaticus'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Helicobacter hepaticus"

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Hynes, Sean O., Susann Teneberg, Niamh Roche, and Torkel Wadström. "Glycoconjugate Binding of Gastric and Enterohepatic Helicobacter spp." Infection and Immunity 71, no. 5 (May 2003): 2976–80. http://dx.doi.org/10.1128/iai.71.5.2976-2980.2003.

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ABSTRACT Helicobacter pylori is able to utilize several lectin-like, protein-carbohydrate interactions for binding to mucins, cell surfaces, and extracellular matrix proteins. As determined by hemagglutination assays and binding of radiolabeled bacteria to glycosphingolipids on thin-layer chromatograms, strains of gastric helicobacters and enterohepatic helicobacters, including Helicobacter canis, Helicobacter hepaticus, and Helicobacter bilis, also demonstrated evidence for the presence of lectin-hemagglutinin adhesins. In addition, in H. hepaticus and H. bilis, binding may be sialic acid dep
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Belzer, Clara, Jeroen Stoof, Catherine S. Beckwith, Ernst J. Kuipers, Johannes G. Kusters, and Arnoud H. M. van Vliet. "Differential regulation of urease activity in Helicobacter hepaticus and Helicobacter pylori." Microbiology 151, no. 12 (December 1, 2005): 3989–95. http://dx.doi.org/10.1099/mic.0.28188-0.

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Helicobacter hepaticus is a pathogen of rodents, which causes diverse enteric and hepatic inflammatory diseases and malignancies. The urease enzyme is an important colonization factor of gastric Helicobacter species like Helicobacter pylori, but little is known about the role and regulation of urease in enterohepatic Helicobacter species. Here it is reported that urease activity of H. hepaticus does not contribute to acid resistance, and that it is nickel-responsive at the post-translational level. H. hepaticus strain ATCC 51449 did not grow or survive at pH 3·0, and supplementation with urea
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Krishnan, Navasona, Alan R. Doster, Gerald E. Duhamel, and Donald F. Becker. "Characterization of a Helicobacter hepaticus putA Mutant Strain in Host Colonization and Oxidative Stress." Infection and Immunity 76, no. 7 (May 5, 2008): 3037–44. http://dx.doi.org/10.1128/iai.01737-07.

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ABSTRACT Helicobacter hepaticus is a gram-negative, spiral-shaped microaerophilic bacterium associated with chronic intestinal infection leading to hepatitis and colonic and hepatic carcinomas in susceptible strains of mice. In the closely related human pathogen Helicobacter pylori, l-proline is a preferred respiratory substrate and is found at significantly high levels in the gastric juice of infected patients. A previous study of the proline catabolic PutA flavoenzymes from H. pylori and H. hepaticus revealed that Helicobacter PutA generates reactive oxygen species during proline oxidation b
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Young, Vincent B., Kimberly A. Knox, and David B. Schauer. "Cytolethal Distending Toxin Sequence and Activity in the Enterohepatic Pathogen Helicobacter hepaticus." Infection and Immunity 68, no. 1 (January 1, 2000): 184–91. http://dx.doi.org/10.1128/iai.68.1.184-191.2000.

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ABSTRACT Little is known about the molecular pathogenesis of hepatitis and enterocolitis caused by enterohepatic Helicobacter species. Sonicates of the murine pathogen Helicobacter hepaticuswere found to cause progressive cell distension, accumulation of filamentous actin, and G2/M cell cycle arrest in HeLa cell monolayers. The genes encoding this cytotoxic activity were cloned fromH. hepaticus. Three open reading frames with closest homology to cdtA, cdtB, and cdtCfrom Campylobacter jejuni were identified. Sonicates of a laboratory strain of Escherichia coli carrying the clonedcdtABC gene clu
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Whary, M. T., T. J. Morgan, C. A. Dangler, K. J. Gaudes, N. S. Taylor, and J. G. Fox. "Chronic Active Hepatitis Induced by Helicobacter hepaticus in the A/JCr Mouse Is Associated with a Th1 Cell-Mediated Immune Response." Infection and Immunity 66, no. 7 (July 1, 1998): 3142–48. http://dx.doi.org/10.1128/iai.66.7.3142-3148.1998.

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ABSTRACT Helicobacter hepaticus infection in A/JCr mice results in chronic active hepatitis characterized by perivascular, periportal, and parenchymal infiltrates of mononuclear and polymorphonuclear cells. This study examined the development of hepatitis and the immune response of A/JCr mice to H. hepaticus infection. The humoral and cell-mediated T helper immune response was profiled by measuring the postinfection (p.i.) antibody response in serum, feces, and bile and by the production of cytokines and proliferative responses by splenic mononuclear cells to H. hepaticusantigens. Secretory im
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Shen, Zeli, David B. Schauer, Harry L. T. Mobley, and James G. Fox. "Development of a PCR-Restriction Fragment Length Polymorphism Assay Using the Nucleotide Sequence of the Helicobacter hepaticus Urease Structural Genes ureAB." Journal of Clinical Microbiology 36, no. 9 (1998): 2447–53. http://dx.doi.org/10.1128/jcm.36.9.2447-2453.1998.

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Infection with Helicobacter hepaticus causes chronic active hepatitis in certain strains of mice and is associated with hepatocellular carcinoma in A/JCr mice. Like the gastric helicobacters,H. pylori and H. mustelae, H. hepaticus possesses a high level of urease activity. However, theH. hepaticus urease structural gene sequences have not been previously determined, and the role of the urease enzyme in colonization and in pathogenesis is not known. PCR was used to amplify a portion of the urease structural genes from H. hepaticusgenomic DNA. Amplified DNA fragments were cloned, and the nucleot
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García, Alexis, Melanie M. Ihrig, Rebecca C. Fry, Yan Feng, Sandy Xu, Samuel R. Boutin, Arlin B. Rogers, Suresh Muthupalani, Leona D. Samson, and James G. Fox. "Genetic Susceptibility to Chronic Hepatitis Is Inherited Codominantly in Helicobacter hepaticus-Infected AB6F1 and B6AF1 Hybrid Male Mice, and Progression to Hepatocellular Carcinoma Is Linked to Hepatic Expression of Lipogenic Genes and Immune Function-Associated Networks." Infection and Immunity 76, no. 5 (February 19, 2008): 1866–76. http://dx.doi.org/10.1128/iai.01044-07.

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ABSTRACT Helicobacter hepaticus causes hepatitis in susceptible strains of mice. Previous studies indicated that A/JCr mice are susceptible and C57BL/6NCr mice are resistant to H. hepaticus-induced hepatitis. We used F1 hybrid mice derived from A/J and C57BL/6 matings to investigate their phenotype and determine their hepatic gene expression profile in response to H. hepaticus infection. F1 hybrid mice, as well as parental A/J and C57BL/6 mice, were divided equally into control and H. hepaticus-infected groups and euthanized at 18 months postinoculation. Hepatic lesions were evaluated histolog
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Péré-Védrenne, Christelle, Wencan He, Lamia Azzi-Martin, Valérie Prouzet-Mauléon, Alice Buissonnière, Bruno Cardinaud, Philippe Lehours, Francis Mégraud, Christophe F. Grosset, and Armelle Ménard. "The Nuclear Remodeling Induced by Helicobacter Cytolethal Distending Toxin Involves MAFB Oncoprotein." Toxins 12, no. 3 (March 12, 2020): 174. http://dx.doi.org/10.3390/toxins12030174.

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Enterohepatic Helicobacters, such as Helicobacter hepaticus and Helicobacter pullorum, are associated with several intestinal and hepatic diseases. Their main virulence factor is the cytolethal distending toxin (CDT). In the present study, whole genome microarray-based identification of differentially expressed genes was performed in vitro in HT-29 intestinal cells while following the ectopic expression of the active CdtB subunit of H. hepaticus CDT. A CdtB-dependent upregulation of the V-maf musculoaponeurotic fibrosarcoma oncogene homolog B (MAFB) gene encoding the MAFB oncoprotein was found
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Ananieva, Olga, Ingrid Nilsson, Tamara Vorobjova, Raivo Uibo, and Torkel Wadström. "Immune Responses to Bile-Tolerant Helicobacter Species in Patients with Chronic Liver Diseases, a Randomized Population Group, and Healthy Blood Donors." Clinical and Vaccine Immunology 9, no. 6 (November 2002): 1160–64. http://dx.doi.org/10.1128/cdli.9.6.1160-1164.2002.

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ABSTRACT Bile-tolerant Helicobacter species such as Helicobacter pullorum, Helicobacter bilis, and Helicobacter hepaticus are associated with hepatic disorders in animals and may be involved in the pathogenesis of chronic liver diseases (CLD) in humans. Antibody responses to cell surface proteins of H. pullorum, H. bilis, and H. hepaticus in serum samples from patients with CLD, a randomized population group, and healthy blood donors were evaluated by using enzyme linked immunosorbent assay (ELISA). The results were compared with the antibody responses to Helicobacter pylori. For analysis of a
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Livingston, Robert S., Lela K. Riley, Reuel R. Hook, Cynthia L. Besch-Williford, and Craig L. Franklin. "Cloning and Expression of an Immunogenic Membrane-Associated Protein of Helicobacter hepaticus for Use in an Enzyme-Linked Immunosorbent Assay." Clinical Diagnostic Laboratory Immunology 6, no. 5 (September 1, 1999): 745–50. http://dx.doi.org/10.1128/cdli.6.5.745-750.1999.

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ABSTRACT Helicobacter hepaticus is a bacterial pathogen that causes chronic active hepatitis and inflammatory bowel disease in mice. The purpose of this study was to develop a recombinant antigen-based enzyme-linked immunosorbent assay (ELISA) to detect H. hepaticus-infected mice. A genomic library of H. hepaticus was constructed and was screened with sera fromH. hepaticus-infected mice. A 459-bp open reading frame that coded for an 18-kDa immunoreactive protein, MAP18, was identified. The gene had high identity with genes coding for outer membrane proteins of other bacteria, and the predicted
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Dissertations / Theses on the topic "Helicobacter hepaticus"

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Myles, Matthew Howard. "Helicobacter hepaticus induced gene dysregulation in mice /." Free to MU Campus, others may purchase, 2004. http://wwwlib.umi.com/cr/mo/fullcit?p3137733.

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Lefevre, Marie. "Role of Helicobacter hepaticus in intestinal inflammation." Thesis, University of Oxford, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.504422.

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Thomas, Theodore Seth. "Development of a capillary based helicobacter hepaticus biosensor." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4572.

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Thesis (M.S.) University of Missouri-Columbia, 2006.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file viewed on (June 27, 2007) Includes bibliographical references.
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McBee, Megan Earley. "Immunomodulation by subclinical persistent infection with Helicobacter hepaticus." Thesis, Massachusetts Institute of Technology, 2007. http://hdl.handle.net/1721.1/39916.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Biological Engineering Division, 2007.<br>Includes bibliographical references (leaves 112-118).<br>Recognition of polymicrobial infections is becoming important for understanding differential host responses to environmental exposures, vaccines, as well as therapeutics. Citrobacter rodentium is a well-characterized model of infectious colitis with particular usefulness for modeling human diarrheal disease or inflammatory bowel disease. Infection with Helicobacter hepaticus is subclinical and persistent in C57BL/6 mice, but causes disease i
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Boutin, Samuel R. 1952. "Molecular pathogenesis of Helicobacter hepaticus induced liver disease." Thesis, Massachusetts Institute of Technology, 2005. http://hdl.handle.net/1721.1/35696.

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Thesis (Ph. D. in Molecular and Systems Bacterial Pathogenesis)--Massachusetts Institute of Technology, Biological Engineering Division, 2005.<br>This electronic version was submitted by the student author. The certified thesis is available in the Institute Archives and Special Collections.<br>Includes bibliographical references.<br>Helicobacter hepaticus infection of A/JCr mice is a model of liver cancer resulting from chronic active inflammation. We monitored hepatic global gene expression profiles and correlated them to histological liver lesions in H. hepaticus infected and control male A
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Avenaud, Philippe. "Hélicobacters et carcinomes hépatocelluaires : recherche de bactéries du genre Helicobacter dans le foie humain et de souris par méthode moléculaire." Bordeaux 2, 2003. http://www.theses.fr/2003BOR21067.

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Belzer, Clara. "Surviving the Enterohepatic Tract: Molecular Mechanisms of Stress Adaptation in Helicobacter hepaticus." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2007. http://hdl.handle.net/1765/10647.

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Okoli, Arinze Stanley Medical Sciences Faculty of Medicine UNSW. "Molecular studies of the response of Helicobacter hepaticus to bile, and the effect of Helicobacter bilis on human hepatoma cells." Publisher:University of New South Wales. Medical Sciences, 2009. http://handle.unsw.edu.au/1959.4/43379.

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Enterohepatic Helicobacter species (EHS) are emerging infectious disease agents. Infection of the enterohepatobiliary tract of several mammals by this group of bacteria results in various pathological disorders. The availability of the Helicobacter hepaticus sequenced and annotated genome, allowed molecular characterisation of the responses of H. hepaticus to host factors such as bile. The adaptation/responses of the bacterium to bovine, porcine and human bile were investigated using proteomics and transcriptomics. Ninety-one different proteins were identified in the responses of H. hepaticus
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Evans, Stephanie. "A computational approach to studying the processes active during Helicobacter hepaticus-induced intestinal inflammation." Thesis, University of York, 2016. http://etheses.whiterose.ac.uk/16125/.

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Inflammatory bowel disease (IBD) is a chronic inflammatory condition of the gastro-intestinal tract that usually manifests as either ulcerative colitis or Crohn's disease. Helicobacter hepaticus (Hh)-induced colitis is a mouse model of intestinal inflammation whereby IL-10 knock out mice are infected with Hh, resulting in pathology similar to that seen in Crohn's disease. Following the Complex Systems Modelling and Simulation (CoSMoS) process, a principled approach to the engineering of simulations of complex systems, we have developed IBDSim, a computational model of the processes active in t
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Kamdar, Shraddha Rashmi. "Profiling of microRNAs and IL-10 expression in intestinal CD4+ T cells following infection with Helicobacter hepaticus." Thesis, University of York, 2015. http://etheses.whiterose.ac.uk/12188/.

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In the Helicobacter hepaticus (Hh) colitis model, Hh infection of either wild-type mice treated with a blocking antibody to the IL-10 receptor (anti-IL-10R) or IL-10 KO mice results in intestinal inflammation associated with inflammatory Th1 and Th17 responses. Recent findings suggest that altered expression of the post-transcriptional gene regulators microRNAs contribute to pathogenic immune responses during intestinal inflammation. Here, examination of microRNA expression during Hh colitis showed that microRNAs are differentially expressed in the inflamed large intestine of Hh+ IL-10 KO mice
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Books on the topic "Helicobacter hepaticus"

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Keshav, Satish, and Alexandra Kent. Gastrointestinal infections. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0197.

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Gastrointestinal infections include diarrhoeal diseases that are major causes of morbidity and mortality worldwide; viral hepatitis; and infections such as Helicobacter pylori infection in the stomach, and intestinal tuberculosis. Here, only luminal gastrointestinal infections causing gastroenteritis, manifested by diarrhoea and/or vomiting, are considered.
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Bulterys, Marc, Julia Brotherton, and Ding-Shinn Chen. Prevention of Infection-Related Cancers. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0066.

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This chapter discusses primary prevention measures that disrupt transmission of oncogenic infections. It begins by discussing vaccination against hepatitis B virus (HBV) and human papillomavirus (HPV), two major causes of cancer for which safe and effective vaccines are currently available. It briefly discusses the importance of treatment and prophylaxis against human immunodeficiency virus type 1 (HIV-1), which potentiates the virulence of other viral infections as well as directly increasing the incidence of non-Hodgkin lymphoma. It does not discuss the treatment of HBV or hepatitis C virus
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Franceschi, Silvia, Hashem B. El-Serag, David Forman, Robert Newton, and Martyn Plummer. Infectious Agents. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0024.

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Eleven infectious agents (seven viruses, three parasites, and one bacterium) have been classified by the International Agency for Research on Cancer as carcinogenic to humans for one or more cancer sites: hepatitis B virus; hepatitis C virus; thirteen types of human papillomavirus (HPV); human immunodeficiency virus type 1 (HIV-1); human T-cell leukemia virus type 1; Epstein-Barr virus; Kaposi sarcoma herpesvirus; Helicobacter pylori; Opisthorchis viverrini; Clonorchis sinensis; and Schistosoma haematobium. Other infectious agents, such as Merkel cell polyomavirus, Plasmodium falciparum, and c
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Book chapters on the topic "Helicobacter hepaticus"

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Fox, J. G. "Helicobacter hepaticus and Helicobacter bilis: proinflammatory modulators of enterohepatic disease." In Helicobactor pylori, 15–29. Dordrecht: Springer Netherlands, 2003. http://dx.doi.org/10.1007/978-94-017-1763-2_2.

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Fox, J. G., K. A. Andrutis, and J. Yu. "Animal models for Helicobacter-induced gastric and hepatic cancer." In Helicobacter pylori, 504–22. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1418-9_49.

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Burnens, André P., John Stanley, and Jacques Nicolet. "Possible Association of Helicobacter pullorum with Lesions of Vibrionic Hepatitis in Poultry." In Campylobacters, Helicobacters, and Related Organisms, 291–93. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9558-5_51.

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Simon, Chantal, Hazel Everitt, Françoise van Dorp, Nazia Hussain, Emma Nash, and Danielle Peet. "Gastrointestinal medicine." In Oxford Handbook of General Practice, 343–406. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198808183.003.0012.

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This chapter in the Oxford Handbook of General Practice explores gastrointestinal medicine in general practice. It covers assessment of abdominal pain, vomiting and diarrhoea, gastroenteritis and food poisoning, constipation, and other abdominal symptoms and signs. It examines dyspepsia and Helicobacter pylori, oesophageal conditions, gastro-oesophageal reflux and gastritis, peptic ulceration, and gastro-oesophageal malignancy. It discusses hernias, appendicitis and small bowel disease, colorectal cancer, and other large bowel conditions. It describes anal and perianal conditions, patients with ostomies, malabsorption, faecal incontinence, coeliac disease, inflammatory bowel disease, and irritable bowel syndrome. It also explores jaundice and abnormal liver function, fatty liver disease, hepatitis, liver failure and portal hypertension, gallbladder disease, pancreatitis, and pancreatic tumours.
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Conference papers on the topic "Helicobacter hepaticus"

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Gong, Guanyu, Sureshkumar Muthupalani, Gerald N. Wogan, James G. Fox, and Steven R. Tannenbaum. "Abstract A17: Colitis-associated tumorigenesis in the Rag2 -/- Helicobacter hepaticus-infected mouse features DNA damage and Retinoblastoma-mediated cell proliferation via nitric oxide but not hypochlorous acid." In Abstracts: Third AACR International Conference on Frontiers in Basic Cancer Research - September 18-22, 2013; National Harbor, MD. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.fbcr13-a17.

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