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Books on the topic 'Helicobacter pylori - Molecular aspects'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 February 2022

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1

Luke, Catherine J. Molecular characterisation of Helicobacter pylori. University of Birmingham, 1992.

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2

Tegtmeyer, Nicole, and Steffen Backert, eds. Molecular Pathogenesis and Signal Transduction by Helicobacter pylori. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50520-6.

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3

Gasbarrini, G., and S. Pretolani, eds. Basic and Clinical Aspects of Helicobacter pylori Infection. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78231-2.

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4

Backert, Steffen, ed. Molecular Mechanisms of Inflammation: Induction, Resolution and Escape by Helicobacter pylori. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-15138-6.

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5

and, M. Achtman. Helicobacter Pylori: Molecular & Cellular Biology. Garland Science, 2001.

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6

Mark, Achtman, and Suerbaum Sebastian, eds. Helicobacter pylori: Molecular and cellular biology. Horizon Scientific Press, 2001.

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7

Yoshio, Yamaoka, ed. Helicobacter pylori: Molecular genetics and cellular biology. Caister Academic, 2008.

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8

Backert, Steffen, and Nicole Tegtmeyer. Molecular Pathogenesis and Signal Transduction by Helicobacter pylori. Springer, 2017.

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9

Backert, Steffen, and Nicole Tegtmeyer. Molecular Pathogenesis and Signal Transduction by Helicobacter pylori. Springer, 2018.

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10

Yoshimasa, Yamamoto, Friedman Herman 1931-, and Hoffman Paul, eds. Helicobacter pylori infection and immunity. Kluwer Academic/Plenum, 2002.

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11

Gasbarrini, G., and S. Pretolani. Basic and Clinical Aspects of Helicobacter pylori Infection. Springer, 2012.

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12

G, Gasbarrini, Pretolani S. 1956-, and European Helicobacter Pylori Study Group. Workshop, eds. Basic and clinical aspects of Helicobacter pylori infection. Springer-Verlag, 1994.

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13

Backert, Steffen. Molecular Mechanisms of Inflammation: Induction, Resolution and Escape by Helicobacter pylori. Springer, 2019.

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14

B, Ernst Peter, Michetti Pierre, and Smith, Phillip D., M.D., eds. The immunobiology of H. pylori: From pathogenesis to prevention. Lippincott-Raven, 1997.

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15

(Editor), Yoshimasa Yamamoto, Herman Friedman (Editor), and Paul S. Hoffman (Editor), eds. Helicobacter pylori Infection and Immunity (INFECTIOUS AGENTS AND PATHOGENESIS Volume 15) (Infectious Agents and Pathogenesis). Springer, 2001.

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16

Contreras, Mario Huesca. Molecular characterization of the mechanisms of adhesion of Helicobacter pylori: A model of environmental regulation of bacterial attachment. 1999.

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17

García Cuan, Aracely. Detección de Helicobacter pylori por PCR anidada en muestras no invasivas: guía práctica. Universidad Libre Seccional Barranquilla, 2019. http://dx.doi.org/10.18041/978-958-9145-77-7.

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Abstract:

Helicobacter pylori es un bacilo, microaerófilo y agente etiológico asociado con la enfermedad ácido-péptica en humanos. Actualmente se le conoce responsable del 85-95 % de las úlceras duodenales, 70- 80 % de las úlceras gástricas y tiene protagonismo relevante en el 60- 70 % de los casos de cáncer gástrico, neoplasias más frecuentes a nivel mundial (1,2). En zonas costeras de Colombia (Barranquilla, Cartagena y Santa Marta), un estudio realizado a partir de biopsias gástricas reportó una frecuencia de infección por H. pylori de 77,9% (3). El riesgo de desarrollo, distribución y la severidad d

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18

Martel, Catherine de, and Julie Parsonnet. Stomach Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0031.

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Stomach cancer is the fifth most common incident cancer worldwide and the third leading cause of cancer death. Almost half of the world’s cases occur in Asia, with 42% in China alone. Although the incidence and mortality rates from stomach cancer are decreasing, the global disease burden remains high. Moreover, the absolute number of cases continues to rise because of population aging. Adenocarcinomas comprise over 90% of gastric malignancies. The adenocarcinomas are further classified according to anatomic location (cardia vs. non-cardia), histology (e.g., intestinal or diffuse, signet ring o

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