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Journal articles on the topic 'Helicobacter pylori - Molecular aspects'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 February 2022

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1

Zanotti, Giuseppe. "Molecular aspects of Helicobacter pylori cag-pathogenicity island." FEBS Journal 278, no. 8 (2011): 1189. http://dx.doi.org/10.1111/j.1742-4658.2011.08034.x.

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2

Torres Jiménez, Franklin, and Carlos Torres Bayona. "Molecular pathophysiology in infection by Helicobacter pylori." Salud Uninorte 32, no. 3 (2021): 500–512. http://dx.doi.org/10.14482/sun.32.3.9749.

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La infección por Helicobacter pylori es la infección bacteriana más común a nivel mundial, en 1994 la Agencia Internacional para la Investigación en Cáncer catalogó al Helicobacter Pylori como un carcinógeno tipo I. La infección por ésta bacteria incrementa el riesgo de padecer cáncer gástrico hasta 6 veces más que los individuos que no presentan la infección. Colombia tiene una prevalencia de infección por Helicobacter pylori del 86% en adultos mayores de 20 años y del 80% en niños mayores de 8 años. La infección persistente y recidivante de la mucosa gástrica puede generar una respuesta infl

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3

Owens, Scott R., and Lauren B. Smith. "Molecular Aspects of H. pylori-Related MALT Lymphoma." Pathology Research International 2011 (January 24, 2011): 1–8. http://dx.doi.org/10.4061/2011/193149.

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Helicobacter pylori-related extranodal marginal zone lymphoma of mucosa-associated lymphoid tissue is a paradigm for malignancy arising in an inflammatory background. While the diagnosis of H. pylori gastritis is often straightforward, distinction between severe gastritis and early lymphoma can be difficult and requires careful assessment of clinical findings in addition to histological features and immunohistochemical results. A number of cytogenetic abnormalities have been discovered in H. pylori-related lymphomas and several have clinical importance, related to the responsiveness of lymphom

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4

Buzás, György Miklós. "Helicobacter pylori – 2012." Orvosi Hetilap 153, no. 36 (2012): 1407–18. http://dx.doi.org/10.1556/oh.2012.29443.

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The author overviews some aspects of literature data of the past 2 years. Genetic research has dentified polymorphisms of Helicobacter pylori virulence factors and the host which could play a role in the clinical outcome of the infection (peptic ulcer or gastric cancer). So far they have been performed in research centers but with a decrease of costs, they will take their place in diagnosing the diseaes and tailoring the treatment. Antibiotic resistance is still growing in Southern European countries and is decreasing in Belgium and Scandinavia. Currently, the clarithromycin resistance rate is

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Bustamante Oviedo, Johana, Cecilia Sánchez Borja, Jovanny Santos Luna, Andrés Medina Preciado, and Marisela Segura Osorio. "Ozonoterapia y su acción en la bacteria helicobacter pylori. // Ozonetherapy and its action in the bacterium helicobacter pylori." CIENCIA UNEMI 10, no. 22 (2017): 98–104. http://dx.doi.org/10.29076/issn.2528-7737vol10iss22.2017pp98-104p.

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La ozonoterapia es la técnica que consiste en la aplicación en el organismo humano, con fines terapéuticos, de una mezcla compuesta de gas ozono con oxígeno medicinal, en la acción de la bacteria helicobacter pylori. Por lo tanto, se plantea como objetivo, el análisis de información recopilada, antecedentes, principales hallazgos que sustentan el uso científico y médico del ozono, específicamente enfocado en el tratamiento de helicobacter pylori, a través de la búsqueda de fuentes electrónicas disponibles y revisión de conceptos relacionados con la rama de las ciencias médicas, que se ocupa de

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6

Al-Khafaji, Zahra M., Aaisha B. Mahmood, and Marium B. Mahmood. "Food constituents for inhibition of BabA of Helicobacter pylori." Journal of Drug Delivery and Therapeutics 10, no. 5-s (2020): 152–63. http://dx.doi.org/10.22270/jddt.v10i5-s.4358.

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Helicobacter pylori causes several gastric pathogeneses to human, nowadays the bacterium developed incredible drug and antibiotic resistance. The bacterium starts its activities by attachment to gastric epithelia via BabA as the main player in the\is process. The study was carried out to find/discover food constituents as inhibitors. Five molecules were obtained from the screening process, 2_3_4_5_6_Penta_O_acetyl_D_glucose, N2_N2_Dimethylguanosine, 5__Methylthioadenosine, Glyceryl_5_hydroxydecanoate, Monoisopropyl_citrate , in addition to two drugs Rivoglitazone and Tiapirinol not used for He

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7

Kountouras, Jannis, Stergios A. Polyzos, Panagiotis Katsinelos, et al. "Potential molecular aspects of Helicobacter pylori-related hyperplastic polyp development and progression." European Journal of Gastroenterology & Hepatology 28, no. 7 (2016): 851–52. http://dx.doi.org/10.1097/meg.0000000000000625.

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8

Amaral, Odete, Isabel Fernandes, Nélio Veiga, et al. "Living Conditions and Helicobacter pylori in Adults." BioMed Research International 2017 (2017): 1–5. http://dx.doi.org/10.1155/2017/9082716.

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Introduction. Infection by the bacterium Helicobacter pylori (H. pylori) is transmissible and is considered a public health issue which affects people of all ages. The objective of this study was to identify factors (lifestyles, dietary factors, and hygiene conditions) related to the prevalence of H. pylori infection. Methods. We carried out an observational cross-sectional study with a community sample of adults from the municipalities of Viseu and Sátão, Portugal. The final sample resulted in 166 adults. The data were collected through a self-administered questionnaire with questions regardi

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9

Deptuła, Piotr, Łukasz Suprewicz, Tamara Daniluk, et al. "Nanomechanical Hallmarks of Helicobacter pylori Infection in Pediatric Patients." International Journal of Molecular Sciences 22, no. 11 (2021): 5624. http://dx.doi.org/10.3390/ijms22115624.

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background: the molecular mechanism of gastric cancer development related to Helicobacter pylori (H. pylori) infection has not been fully understood, and further studies are still needed. Information regarding nanomechanical aspects of pathophysiological events that occur during H. pylori infection can be crucial in the development of new prevention, treatment, and diagnostic measures against clinical consequences associated with H. pylori infection, including gastric ulcer, duodenal ulcer, and gastric cancer. Methods: in this study, we assessed mechanical properties of children's healthy and

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10

Sukhan, D. S., S. V. Vernygorodskyi, N. V. Haidukov, and H. P. Ludkevich. "Molecular and Genetic Aspects of Helicobacter pylori Interaction with Cells of Gastric Mucosa." Cytology and Genetics 54, no. 2 (2020): 147–53. http://dx.doi.org/10.3103/s0095452720020139.

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11

Baj, Jacek, Karolina Brzozowska, Alicja Forma, Amr Maani, Elżbieta Sitarz, and Piero Portincasa. "Immunological Aspects of the Tumor Microenvironment and Epithelial-Mesenchymal Transition in Gastric Carcinogenesis." International Journal of Molecular Sciences 21, no. 7 (2020): 2544. http://dx.doi.org/10.3390/ijms21072544.

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Infection with Helicobacter pylori, a Gram-negative, microaerophilic pathogen often results in gastric cancer in a subset of affected individuals. This explains why H. pylori is the only bacterium classified as a class I carcinogen by the World Health Organization. Several studies have pinpointed mechanisms by which H. pylori alters signaling pathways in the host cell to cause diseases. In this article, the authors have reviewed 234 studies conducted over a span of 18 years (2002–2020). The studies investigated the various mechanisms associated with gastric cancer induction. For the past 1.5 y

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12

Zanotti, Giuseppe, and Laura Cendron. "Functional and structural aspects of helicobacter pylori acidic stress response factors." IUBMB Life 62, no. 10 (2010): 715–23. http://dx.doi.org/10.1002/iub.382.

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13

Zanotti, Giuseppe, and Laura Cendron. "Functional and structural aspects of helicobacter pylori acidic stress response factors." IUBMB Life 62, no. 10 (2010): spcone. http://dx.doi.org/10.1002/iub.397.

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14

Clerici, Stefano Piatto, Patrícia Fernandes de Souza Oliveira, Erica Mie Akagi, et al. "A comprehensive review on the role of protein tyrosine phosphatases in gastric cancer development and progression." Biological Chemistry 402, no. 6 (2021): 663–74. http://dx.doi.org/10.1515/hsz-2020-0355.

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Abstract The main post-translational reversible modulation of proteins is phosphorylation and dephosphorylation, catalyzed by protein kinases (PKs) and protein phosphatases (PPs) which is crucial for homeostasis. Imbalance in this crosstalk can be related to diseases, including cancer. Plenty of evidence indicates that protein tyrosine phosphatases (PTPs) can act as tumor suppressors and tumor promoters. In gastric cancer (GC), there is a lack of understanding of the molecular aspects behind the tumoral onset and progression. Here we describe several members of the PTP family related to gastri

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15

Sticlaru, Liana, Florica Stăniceanu, Mirela Cioplea, et al. "Dangerous Liaison: Helicobacter pylori, Ganglionitis, and Myenteric Gastric Neurons: A Histopathological Study." Analytical Cellular Pathology 2019 (December 30, 2019): 1–9. http://dx.doi.org/10.1155/2019/3085181.

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Chronic inflammation induced by Helicobacter pylori (H. pylori) infection plays a major role in development of gastric cancer. However, recent findings suggested that progression of inflammation and neoplastic transformation in H. pylori infection are more complex than previously believed and could involve different factors that modulate gastric microenvironment and influence host-pathogen interaction. Among these factors, gastric myenteric plexus and its potential adaptive changes in H. pylori infection received little attention. This study is aimed at identifying the impact of H. pylori-asso

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16

Katsinelos, Taxiarchis, Michael Doulberis, Stergios A. Polyzos, Apostolis Papaefthymiou, Panagiotis Katsinelos, and Jannis Kountouras. "Molecular Links Between Alzheimer's Disease and Gastrointestinal Microbiota: Emphasis on Helicobacter pylori Infection Involvement." Current Molecular Medicine 20, no. 1 (2019): 3–12. http://dx.doi.org/10.2174/1566524019666190917125917.

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Alzheimer's disease (AD) is a neurodegenerative disease and the main form of dementia, characterized by progressive cognitive decline and detrimental consequences in both personal-family and global level. Within this narrative review, we provide recent molecular aspects of Tau, a microtubule AD-associated protein, as well as amyloid beta, involved in AD pathophysiology. Moreover, we provide additional emerging data from basic research as well as clinical studies indicating an implicating role of gastrointestinal microbiota (GI-M), including Helicobacter pylori infection (Hp-I), in AD pathophys

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17

Idris, Abeer Babiker, Hadeel Gassim Hassan, Maryam Atif Salaheldin Ali, et al. "Molecular Phylogenetic Analysis of 16S rRNA Sequences Identified Two Lineages of Helicobacter pylori Strains Detected from Different Regions in Sudan Suggestive of Differential Evolution." International Journal of Microbiology 2020 (October 27, 2020): 1–12. http://dx.doi.org/10.1155/2020/8825718.

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Background. Helicobacter pylori (H. pylori) is ubiquitous among humans and one of the best-studied examples of an intimate association between bacteria and humans. Phylogeny and Phylogeography of H. pylori strains are known to mirror human migration patterns and reflect significant demographic events in human prehistory. In this study, we analyzed the molecular evolution of H. pylori strains detected from different tribes and regions of Sudan using 16S rRNA gene and the phylogenetic approach. Materials and methods. A total of 75 gastric biopsies were taken from patients who had been referred f

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18

Amlashi, Fazel Isapanah, Zahra Norouzi, Ahmad Sohrabi, et al. "A systematic review and meta-analysis for association of Helicobacter pylori colonization and celiac disease." PLOS ONE 16, no. 3 (2021): e0241156. http://dx.doi.org/10.1371/journal.pone.0241156.

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Background and objectives Based on some previous observational studies, there is a theory that suggests a potential relationship between Helicobacter pylori (H. pylori) colonization and celiac disease (CeD); however, the type of this relationship is still controversial. Therefore, we aimed to conduct a systematic review and meta-analysis to explore all related primary studies to find any possible association between CeD and human H. pylori colonization. Data sources Studies were systematically searched and collected from four databases and different types of gray literature to cover all availa

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19

Donati, Manuela, Elisa Storni, Lucia D’Apote, et al. "PCR-Based Restriction Pattern Typing of the vacA Gene Provides Evidence for a Homogeneous Group among Helicobacter pylori Strains Associated with Peptic Ulcer Disease." Journal of Clinical Microbiology 37, no. 4 (1999): 912–15. http://dx.doi.org/10.1128/jcm.37.4.912-915.1999.

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The results of PCR-based molecular typing of Helicobacter pylori strains by restriction fragment length polymorphism analysis of a 1,161-bp nucleotide sequence of the midregion of thevacA gene are reported. A total of 48 H. pylori strains isolated from gastric biopsy specimens obtained from 18 patients with peptic ulcer dyspepsia, 15 patients with nonulcer dyspepsia, and 15 asymptomatic H. pylori-infected subjects were studied. Highly heterogeneous restriction patterns were obtained by digestion of PCR products with SauII,BglII, and HhaI, whereas HaeIII digestion resulted in a strictly homogen

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20

Taylor, Diane E. "Helicobacter pyloriand Its Genome: Lessons from the Treasure Map." Canadian Journal of Gastroenterology 13, no. 3 (1999): 218–23. http://dx.doi.org/10.1155/1999/964025.

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The availability of the complete genome sequence ofHelicobacter pylori26695 has opened new avenues for research in the molecular biology of this gastric pathogen. The present review gives a general overview ofH pyloriobtained from the complete genome sequence and compares this with data previously obtained from cloning and functional studies ofH pylori. The cagA pathogenicity island of 40 kilobases, which encodes a type IV secretion system, is discussed. The diversity ofH pylorigenomes is well known, yet new data indicate that some aspects of the genome, particularly outer membrane protein gen

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21

Cendron, Laura, and Giuseppe Zanotti. "Structural and functional aspects of unique type IV secretory components in the Helicobacter pylori cag-pathogenicity island." FEBS Journal 278, no. 8 (2011): 1223–31. http://dx.doi.org/10.1111/j.1742-4658.2011.08038.x.

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22

Sukhan, D., and S. Vernigorodskiy. "Prognostic aspects of immunohistochemical assessment of cellular renewal and transcription factor SOX2 with precancerous changes in the gastric mucosa depending on the genotype of Helicobacter pylori in patients with various types of chronic gastritis." Reports of Vinnytsia National Medical University 22, no. 4 (2018): 674–81. http://dx.doi.org/10.31393/reports-vnmedical-2018-22(4)-18.

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The precancerous potential of chronic gastritis (CG) associated with H. pylori is discussed in numerous writings, and today, CG is central to precancerous conditions of the stomach. A convincing theoretical basis for such an assessment of chronic gastritis is its characteristic feature — an interruption of cell renewal with the proliferation phase predominating over the differentiation phase. However, the determination of the degree of proliferative activity and impaired differentiation of the epithelium of the gastric mucosa (GM) remains not fully understood. The goal of our study was the imm

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23

Petryszyn, Pawel, Nicolas Chapelle, and Tamara Matysiak-Budnik. "Gastric Cancer: Where Are We Heading?" Digestive Diseases 38, no. 4 (2020): 280–85. http://dx.doi.org/10.1159/000506509.

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Background: Despite its decreasing incidence, gastric cancer (GC) remains one of the leading cancers in the world and an important global healthcare problem due to its overall high prevalence and high mortality rate. Summary: GC is a consequence of Helicobacter pylori infection in 90% of cases, while in 10% Epstein Barr Virus may be responsible. Moreover, some recent epidemiological data suggest an increasing incidence in some young patients groups possibly due to autoimmunity, and if this tendency is confirmed, it may change the epidemiology of GC in the future. The pathogenesis of GC is main

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Kuhns, Lisa G., Stéphane L. Benoit, Krishnareddy Bayyareddy, et al. "Carbon Fixation Driven by Molecular Hydrogen Results in Chemolithoautotrophically Enhanced Growth of Helicobacter pylori." Journal of Bacteriology 198, no. 9 (2016): 1423–28. http://dx.doi.org/10.1128/jb.00041-16.

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ABSTRACTA molecular hydrogen (H2)-stimulated, chemolithoautotrophic growth mode for the gastric pathogenHelicobacter pyloriis reported. In a culture medium containing peptides and amino acids, H2-supplied cells consistently achieved 40 to 60% greater growth yield in 16 h and accumulated 3-fold more carbon from [14C]bicarbonate (on a per cell basis) in a 10-h period than cells without H2. Global proteomic comparisons of cells supplied with different atmospheric conditions revealed that addition of H2led to increased amounts of hydrogenase and the biotin carboxylase subunit of acetyl coenzyme A

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25

Hudler, Petra. "Outlook on Epigenetic Therapeutic Approaches for Treatment of Gastric Cancer." Current Cancer Drug Targets 18, no. 1 (2017): 65–88. http://dx.doi.org/10.2174/1568009617666170203163745.

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The incidence of gastric cancer has been declining globally in the last decades. Despite the improvements in the diagnostic procedures, most cases are still detected at advanced stages due to lack of specific symptoms associated with early phases of tumour development. Consequently, gastric cancer poses a major health burden worldwide due to high mortality rates. Continuing advances in high-throughput technologies are revealing an intricate network of genetic and epigenetic changes associated with carcinogenesis. In addition, several risk factors, both environmental and genetic, have been reco

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Murata, Mariko, Raynoo Thanan, Ning Ma, and Shosuke Kawanishi. "Role of Nitrative and Oxidative DNA Damage in Inflammation-Related Carcinogenesis." Journal of Biomedicine and Biotechnology 2012 (2012): 1–11. http://dx.doi.org/10.1155/2012/623019.

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Chronic inflammation induced by biological, chemical, and physical factors has been found to be associated with the increased risk of cancer in various organs. We revealed that infectious agents including liver fluke,Helicobacter pylori, and human papilloma virus and noninfectious agents such as asbestos fiber induced iNOS-dependent formation of 8-nitroguanine and 8-oxo-7, 8-dihydro-2′-deoxyguanosine (8-oxodG) in cancer tissues and precancerous regions. Our results with the colocalization of phosphorylated ATM andγ-H2AX with 8-oxodG and 8-nitroguanine in inflammation-related cancer tissues sug

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27

Ondari, Edna, Esther Calvino-Sanles, Nicholas J. First, and Monica C. Gestal. "Eosinophils and Bacteria, the Beginning of a Story." International Journal of Molecular Sciences 22, no. 15 (2021): 8004. http://dx.doi.org/10.3390/ijms22158004.

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Eosinophils are granulocytes primarily associated with TH2 responses to parasites or immune hyper-reactive states, such as asthma, allergies, or eosinophilic esophagitis. However, it does not make sense from an evolutionary standpoint to maintain a cell type that is only specific for parasitic infections and that otherwise is somehow harmful to the host. In recent years, there has been a shift in the perception of these cells. Eosinophils have recently been recognized as regulators of immune homeostasis and suppressors of over-reactive pro-inflammatory responses by secreting specific molecules

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28

SHIMOYAMA, Takashi. "Clinical Aspects of Helicobacter Pylori." Journal of the Japanese Practical Surgeon Society 54, no. 5 (1993): 1119–43. http://dx.doi.org/10.3919/ringe1963.54.1119.

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29

Bartnik, Witold. "Clinical aspects of Helicobacter pylori infection." Polish Archives of Internal Medicine 118, no. 7-8 (2008): 426–30. http://dx.doi.org/10.20452/pamw.440.

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30

DIXON, MICHAEL F. "IV. Helicobacter pylori and peptic ulceration: Histopathological aspects." Journal of Gastroenterology and Hepatology 6, no. 2 (1991): 125–30. http://dx.doi.org/10.1111/j.1440-1746.1991.tb01451.x.

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31

Spechler, Stuart Jon. "Clinical Aspects of Genetic Variability in Helicobacter pylori." JAMA 283, no. 10 (2000): 1264. http://dx.doi.org/10.1001/jama.283.10.1264.

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32

Molina-Infante, Javier, and Akiko Shiotani. "Practical Aspects in Choosing a Helicobacter pylori Therapy." Gastroenterology Clinics of North America 44, no. 3 (2015): 519–35. http://dx.doi.org/10.1016/j.gtc.2015.05.004.

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33

Sorokman, T. V., P. M. Moldovan, N. O. Popelyuk, and I. V. Parfyonova. "Epidemiological aspects of Helicobacter pylori infection in children." ACTUAL INFECTOLOGY 9, no. 1 (2021): 28–32. http://dx.doi.org/10.22141/2312-413x.9.1.2021.228824.

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Background. Among bacterial infections those caused by Helicobacter pylori (H.pylori) have a special place. H.pylori infection is more common in childhood and is usually asymptomatic. The study of regional and subregional features of H.pylori infection in children is becoming increasingly important. Materials and methods. A retrospective analysis of statistical data of annual reports for 5 years (2015–2019) was conducted on the basis of gastroenterological hospitals in Chernivtsi. Results. For the last 5 years, gastroduodenal diseases were most common in the structure of digestive disorders. S

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34

Kalali, Behnam, Raquel Mejías-Luque, Anahita Javaheri, and Markus Gerhard. "H. pyloriVirulence Factors: Influence on Immune System and Pathology." Mediators of Inflammation 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/426309.

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Helicobacter pyloriis the most widespread chronic bacterial agent in humans and is well recognized for its association with ulcer disease and gastric cancer, with both representing major global health and socioeconomic issues. Given the high level of adaptation and the coevolution of this bacterium with its human host, a thorough and multidirectional view of the specific microbiological characteristics of this infection as well as the host physiology is needed in order to develop novel means of prevention of therapy. This review aims to pinpoint some of these potentially important angles, whic

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Westblom, T. Ulf. "Molecular Diagnosis of Helicobacter Pylori." Immunological Investigations 26, no. 1-2 (1997): 163–74. http://dx.doi.org/10.3109/08820139709048924.

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Tsukanov, V. V., A. V. Vasyutin, and Yu L. Tonkikh. "Modern Aspects of Stomach Cancer Prevention." Doctor.Ru 19, no. 7 (2020): 37–40. http://dx.doi.org/10.31550/1727-2378-2020-19-7-37-40.

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Objective of the Review: to analyse available data on stomach cancer prevention. Key Points. Key risk factors for stomach cancer are male gender, age of >50 years old, Helicobacter pylori and smoking. A number of international consensus opinions regulate screening, diagnostics and treatment of precancer changes in stomach; it is assumed to reduce the incidence of this disease. H. pylori eradication is the key method of primary stomach cancer prevention. Modern studies demonstrate that the optimal method of H. pylori eradication is the therapy with bismuth medication, proton pump inhibitor,

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Kusters, Johannes G., Arnoud H. M. van Vliet, and Ernst J. Kuipers. "Pathogenesis of Helicobacter pylori Infection." Clinical Microbiology Reviews 19, no. 3 (2006): 449–90. http://dx.doi.org/10.1128/cmr.00054-05.

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SUMMARY Helicobacter pylori is the first formally recognized bacterial carcinogen and is one of the most successful human pathogens, as over half of the world's population is colonized with this gram-negative bacterium. Unless treated, colonization usually persists lifelong. H. pylori infection represents a key factor in the etiology of various gastrointestinal diseases, ranging from chronic active gastritis without clinical symptoms to peptic ulceration, gastric adenocarcinoma, and gastric mucosa-associated lymphoid tissue lymphoma. Disease outcome is the result of the complex interplay betwe

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38

Leclerc, Henri. "Sur quelques aspects épidémiologiques de l’infection à Helicobacter pylori." Bulletin de l'Académie Nationale de Médecine 190, no. 4-5 (2006): 949–62. http://dx.doi.org/10.1016/s0001-4079(19)33277-7.

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Sadirovna, Shodieva Musharraf. "Immunogenetic Aspects Helicobacter Pylori Associated Gastroduodenal Pathology In Children." American Journal of Medical Sciences and Pharmaceutical Research 2, no. 08 (2020): 58–63. http://dx.doi.org/10.37547/tajmspr/volume02issue08-08.

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We investigated the associations between the host HLA-DQ variation and the prevalence of H ruli in the Indonesian population with an H. pylori-infected level of 11.2% in people living in the Bukhara region. It was noted that individuals carrying the DQB1 * 0401 genotypes are at significantly greater risk of developing H infection. .lori compared to individuals with the DQB1 0301 genotypes. The DQA1 or DQB1 haplotypes were not associated with H. pulori infection.

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Venerito, Marino, Michael Selgrad, and Peter Malfertheiner. "Helicobacter pylori: Gastric Cancer and Extragastric Malignancies - Clinical aspects." Helicobacter 18 (September 2013): 39–43. http://dx.doi.org/10.1111/hel.12078.

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41

Furuta, Takahisa, and David Y. Graham. "Pharmacologic Aspects of Eradication Therapy for Helicobacter pylori Infection." Gastroenterology Clinics of North America 39, no. 3 (2010): 465–80. http://dx.doi.org/10.1016/j.gtc.2010.08.007.

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42

Yang, Ji Yeong, Jong-Bae Kim, Pyeongjae Lee, and Sa-Hyun Kim. "Evodiamine Inhibits Helicobacter pylori Growth and Helicobacter pylori-Induced Inflammation." International Journal of Molecular Sciences 22, no. 7 (2021): 3385. http://dx.doi.org/10.3390/ijms22073385.

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Helicobacter pylori (H. pylori) classified as a class I carcinogen by the World Health Organization (WHO) plays an important role in the progression of chronic gastritis and the development of gastric cancer. A major bioactive component of Evodia rutaecarpa, evodiamine, has been known for its anti-bacterial effect and anti-cancer effects. However, the inhibitory effect of evodiamine against H. pylori is not yet known and the inhibitory mechanisms of evodiamine against gastric cancer cells are yet to be elucidated concretely. In this study, therefore, anti-bacterial effect of evodiamine on H. p

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Graham, David Y. "Helicobacter pylori: Molecular and cellular biology." Gastroenterology 121, no. 3 (2001): 740. http://dx.doi.org/10.1016/s0016-5085(01)70143-6.

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44

Maconi, G. "Helicobacter pylori: molecular and cellular biology." Digestive and Liver Disease 34, no. 8 (2002): 611. http://dx.doi.org/10.1016/s1590-8658(02)80100-1.

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Floch, Martin H. "Helicobacter pylori: Molecular and Cellular Biology." Journal of Clinical Gastroenterology 33, no. 4 (2001): 348. http://dx.doi.org/10.1097/00004836-200110000-00027.

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Krogfelt, Karen A. "Helicobacter pylori: molecular and cellular biology." International Journal of Food Microbiology 69, no. 3 (2001): 255–56. http://dx.doi.org/10.1016/s0168-1605(01)00557-8.

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Suarez, Giovanni, and Richard M. Peek. "Helicobacter pylori: expect the unexpected." Molecular Microbiology 91, no. 5 (2014): 858–61. http://dx.doi.org/10.1111/mmi.12532.

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Ljungh, Åsa. "Helicobacter pylori Interactions with Plasminogen." Methods 21, no. 2 (2000): 151–57. http://dx.doi.org/10.1006/meth.2000.0986.

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Mladenova, Irena. "Clinical Relevance of Helicobacter pylori Infection." Journal of Clinical Medicine 10, no. 16 (2021): 3473. http://dx.doi.org/10.3390/jcm10163473.

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Abstract:

Helicobacter pylori (H. pylori) is a Gram-negative helical, microaerophilic bacterium which colonizes the antrum and body of the stomach, surviving in its harsh environment through mechanisms of acid resistance and colonization factors. It infects approximately 50% of the world population. Although the prevalence of this infection varies from country to country, as well as between different ethnic, social or age groups, it is estimated that about 50% of the human population only carries this microorganism. While H. pylori has been found to play a major etiological and pathogenic role in chroni

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Kountouras, Jannis, Christos Zavos, Dimitrios Chatzopoulos, and Panagiotis Katsinelos. "New Aspects of Helicobacter pylori Infection Involvement in Gastric Oncogenesis." Journal of Surgical Research 146, no. 1 (2008): 149–58. http://dx.doi.org/10.1016/j.jss.2007.06.011.

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