Relevant bibliographies by topics / Hematopoiesis. Hematopoietic Stem Cells Leukemia

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Hematopoiesis. Hematopoietic Stem Cells Leukemia'

Author: Grafiati
Published: 4 June 2021
Last updated: 3 February 2022

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Journal articles on the topic "Hematopoiesis. Hematopoietic Stem Cells Leukemia"

1

Abdel-Wahab, Omar, and Ross L. Levine. "Metabolism and the leukemic stem cell." Journal of Experimental Medicine 207, no. 4 (2010): 677–80. http://dx.doi.org/10.1084/jem.20100523.

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Acute leukemias are clonal disorders of hematopoiesis wherein a leukemic stem cell (LSC) acquires mutations that confer the capacity for unlimited self-renewal, impaired hematopoietic differentiation, and enhanced proliferation to the leukemic clone. Many recent advances in understanding the biology of leukemia have come from studies defining specific genetic and epigenetic abnormalities in leukemic cells. Three recent articles, however, further our understanding of leukemia biology by elucidating specific abnormalities in metabolic pathways in leukemic hematopoiesis. These studies potentially
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Kandarakov, Oleg, and Alexander Belyavsky. "Clonal Hematopoiesis, Cardiovascular Diseases and Hematopoietic Stem Cells." International Journal of Molecular Sciences 21, no. 21 (2020): 7902. http://dx.doi.org/10.3390/ijms21217902.

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Cardiovascular diseases and cancer, the leading causes of morbidity and mortality in the elderly, share some common mechanisms, in particular inflammation, contributing to their progression and pathogenesis. However, somatic mutagenesis, a driving force in cancer development, has not been generally considered as an important factor in cardiovascular disease pathology. Recent studies demonstrated that during normal aging, somatic mutagenesis occurs in blood cells, often resulting in expansion of mutant clones that dominate hematopoiesis at advanced age. This clonal hematopoiesis is primarily as
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Guitart, Amelie V., Theano I. Panagopoulou, Arnaud Villacreces, et al. "Fumarate hydratase is a critical metabolic regulator of hematopoietic stem cell functions." Journal of Experimental Medicine 214, no. 3 (2017): 719–35. http://dx.doi.org/10.1084/jem.20161087.

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Strict regulation of stem cell metabolism is essential for tissue functions and tumor suppression. In this study, we investigated the role of fumarate hydratase (Fh1), a key component of the mitochondrial tricarboxylic acid (TCA) cycle and cytosolic fumarate metabolism, in normal and leukemic hematopoiesis. Hematopoiesis-specific Fh1 deletion (resulting in endogenous fumarate accumulation and a genetic TCA cycle block reflected by decreased maximal mitochondrial respiration) caused lethal fetal liver hematopoietic defects and hematopoietic stem cell (HSC) failure. Reexpression of extramitochon
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Hu, Xiaoxia, Hongmei Shen, Chen Tian, et al. "Kinetics of normal hematopoietic stem and progenitor cells in a Notch1-induced leukemia model." Blood 114, no. 18 (2009): 3783–92. http://dx.doi.org/10.1182/blood-2009-06-227843.

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Abstract The predominant outgrowth of malignant cells over their normal counterparts in a given tissue is a shared feature for all types of cancer. However, the impact of a cancer environment on normal tissue stem and progenitor cells has not been thoroughly investigated. We began to address this important issue by studying the kinetics and functions of hematopoietic stem and progenitor cells in mice with Notch1-induced leukemia. Although hematopoiesis was progressively suppressed during leukemia development, the leukemic environment imposed distinct effects on hematopoietic stem and progenito
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Agarwal, Anupriya, William J. Bolosky, David B. Wilson, et al. "Differentiation of leukemic blasts is not completely blocked in acute myeloid leukemia." Proceedings of the National Academy of Sciences 116, no. 49 (2019): 24593–99. http://dx.doi.org/10.1073/pnas.1904091116.

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Hematopoiesis, the formation of blood cells, involves the hierarchical differentiation of immature blast cells into mature, functional cell types and lineages of the immune system. Hematopoietic stem cells precisely regulate self-renewal versus differentiation to balance the production of blood cells and maintenance of the stem cell pool. The canonical view of acute myeloid leukemia (AML) is that it results from a combination of molecular events in a hematopoietic stem cell that block differentiation and drive proliferation. These events result in the accumulation of primitive hematopoietic bl
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Alberta, Julia A., Gregory M. Springett, Helen Rayburn, et al. "Role of the WT1 tumor suppressor in murine hematopoiesis." Blood 101, no. 7 (2003): 2570–74. http://dx.doi.org/10.1182/blood-2002-06-1656.

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The WT1 tumor-suppressor gene is expressed by many forms of acute myeloid leukemia. Inhibition of this expression can lead to the differentiation and reduced growth of leukemia cells and cell lines, suggesting that WT1 participates in regulating the proliferation of leukemic cells. However, the role of WT1 in normal hematopoiesis is not well understood. To investigate this question, we have used murine cells in which the WT1 gene has been inactivated by homologous recombination. We have found that cells lacking WT1 show deficits in hematopoietic stem cell function. Embryonic stem cells lacking
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Ichikawa, Motoshi, Takashi Asai, Masahiro Nakagawa, et al. "AML1/Runx1 Negatively Regulates the Number of Quiescent Hematopoietic Stem Cells in Adult Hematopoiesis." Blood 108, no. 11 (2006): 4204. http://dx.doi.org/10.1182/blood.v108.11.4204.4204.

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Abstract Transcription factor AML1 (also called Runx1), which was initially isolated from the t(8;21) chromosomal translocation frequently found in the acute myelogenous leukemia FAB M2 subtype, is essential for the development of multilineage hematopoiesis in mouse embryos. By analyzing conditional AML1 knockout mice, we have previously shown that AML1 negatively regulates the number of immature hematopoietic cells defined as lineage-negative, CD34− Sca-1+ c-Kit+ (34KSL) cells in adult hematopoiesis, while it is required for megakaryocytic maturation and lymphocytic development. The former is
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Singh, Abhishek K., and Jose A. Cancelas. "Gap Junctions in the Bone Marrow Lympho-Hematopoietic Stem Cell Niche, Leukemia Progression, and Chemoresistance." International Journal of Molecular Sciences 21, no. 3 (2020): 796. http://dx.doi.org/10.3390/ijms21030796.

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The crosstalk between hematopoietic stem cells (HSC) and bone marrow (BM) microenvironment is critical for homeostasis and hematopoietic regeneration in response to blood formation emergencies after injury, and has been associated with leukemia transformation and progression. Intercellular signals by the BM stromal cells in the form of cell-bound or secreted factors, or by physical interaction, regulate HSC localization, maintenance, and differentiation within increasingly defined BM HSC niches. Gap junctions (GJ) are comprised of arrays of membrane embedded channels formed by connexin protein
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Zhang, Yiyun, and J. R. Joanna Yeh. "In VivoChemical Screening for Modulators of Hematopoiesis and Hematological Diseases." Advances in Hematology 2012 (2012): 1–12. http://dx.doi.org/10.1155/2012/851674.

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In vivochemical screening is a broadly applicable approach not only for dissecting genetic pathways governing hematopoiesis and hematological diseases, but also for finding critical components in those pathways that may be pharmacologically modulated. Both high-throughput chemical screening and facile detection of blood-cell-related phenotypes are feasible in embryonic/larval zebrafish. Two recent studies utilizing phenotypic chemical screens in zebrafish have identified several compounds that promote hematopoietic stem cell formation and reverse the hematopoietic phenotypes of a leukemia onco
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Kavalerchik, Edward, Daniel Goff, and Catriona H. M. Jamieson. "Chronic Myeloid Leukemia Stem Cells." Journal of Clinical Oncology 26, no. 17 (2008): 2911–15. http://dx.doi.org/10.1200/jco.2008.17.5745.

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Although rare, chronic myeloid leukemia (CML) represents an important paradigm for understanding the molecular events leading to malignant transformation of primitive hematopoietic progenitors. CML was the first cancer to be associated with a defined genetic abnormality, BCR-ABL, that is necessary and sufficient for initiating chronic phase disease as well as the first cancer to be treated with molecular targeted therapy. Malignant progenitors or leukemia stem cells (LSCs) evolve as a result of both epigenetic and genetic events that alter hematopoietic progenitor differentiation, proliferatio
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Dissertations / Theses on the topic "Hematopoiesis. Hematopoietic Stem Cells Leukemia"

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Watson, Alexander Scarth. "Autophagy in hematopoiesis and acute myeloid leukemia." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:2e66c5c3-4774-44d1-8345-d0dc827da16d.

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Acute myeloid leukemia (AML) develops following oncogenic alterations to hematopoietic stem (HSC) and progenitor cells (HSPCs) in the bone marrow, resulting in dysregulated proliferation of immature myeloid progenitors that interferes with normal hematopoiesis. Understanding the mechanisms of HSPC protection against damage and excessive division, and how these pathways are altered during leukemic progression, is vital for establishing effective therapies. Here, we show that autophagy, a lysosomal degradation pathway, is increased in HSPCs using a novel imaging flow cytometry autophagy assay. L
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Lewis, Ian D. "Characterisation of normal and leukaemic stem cells in chronic myeloid leukaemia /." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phl6745.pdf.

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Smith, Molly. "Alternative Splicing and Regulation of Innate Immune Mediators in Normal and Malignant Hematopoiesis." University of Cincinnati / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1563527303459942.

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Lin, Shan. "Modeling and Analysis of Acute Leukemia using Human Hematopoietic Stem and Progenitor Cells." University of Cincinnati / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1481032144780412.

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Feltenmark, Stina. "Studies on arachidonic acid metabolism in normal and malignant hematopoietic cells." Stockholm : Division of Physiological Chemistry II, Karolinska Institutet, 2010. http://diss.kib.ki.se/2010/978-91-7409-745-0/.

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Lam, Kar-yee. "Expression of EEN (endophilin II) : a fusion partner gene in leukemia, in haemopoietic cells /." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23765975.

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Verter, Erol. "TEL/ABL pathogenesis chronic myelogenous leukemia and small bowel syndrome /." Waltham, Mass. : Brandeis University, 2009. http://dcoll.brandeis.edu/handle/10192/23230.

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林嘉儀 and Kar-yee Lam. "Expression of EEN (endophilin II): a fusion partner gene in leukemia, in haemopoietic cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31226176.

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Fu, Jianing. "Targeting T-bet for Prevention of Graft-Versus-Host Disease and Leukemia Relapse after Allogeneic Hematopoietic Stem Cell Transplantation." Scholar Commons, 2015. http://scholarcommons.usf.edu/etd/5828.

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Allogeneic hematopoietic stem cell transplantation (allo-HSCT) is an effective therapeutic option for many malignant diseases. However, the efficacy of allo-HSCT is limited by the occurrence of destructive graft-versus-host disease (GVHD). Since allogeneic T cells are the driving force in the development of GVHD, their activation, proliferation, and differentiation are key factors to understanding GVHD pathogenesis. On the other hand, antigen-presenting cells (APCs) are essential for allogeneic T-cell priming and the development of GVHD. The T-box transcription factor T-bet is a master regulat
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Vo, Thanh-Trang. "Mitochondrial Priming Determines Chemotherapeutic Response in Acute Myeloid Leukemia." Thesis, Harvard University, 2012. http://dissertations.umi.com/gsas.harvard:10384.

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Gain- and loss-of-function studies of the BCL-2 family of proteins have shown that they can impact chemotherapeutic sensitivity. However, cells contain myriad anti-apoptotic and pro-apoptotic BCL-2 family members making it difficult to predict cell fate decisions based on the initial conditions of these proteins. BH3 profiling is a tool that measures mitochondrial priming, the readiness of a cell to die through the intrinsic (or mitochondrial) apoptotic pathway. Priming is due to the cumulative effect of the BCL-2 family of proteins that act as the gate keepers of the mitochondrial apoptoti
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Books on the topic "Hematopoiesis. Hematopoietic Stem Cells Leukemia"

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T, Miyazaki, Takaku Fumimaro, and Uchino Haruto 1926-, eds. Myelodysplastic syndrome and cytokines: Proceedings of the International Symposium on Myelodysplastic Syndrome and Cytokine, Sapporo, 28-29 September 1990. Excerpta Medica, 1991.

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Pálóczi, Katalin. Immunophenotypic analysis. 2nd ed. Landes Bioscience, 2005.

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Pálóczi, Katalin. Immunophenotypic analysis. 2nd ed. Landes Bioscience, 2005.

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Clinical applications of immunophenotypic analysis. R.G. Landes, 1994.

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Lothar, Kanz, and New York Academy of Sciences, eds. Hematopoietic stem cells VI. Blackwell Pub. on behalf of the New York Academy of Sciences, 2007.

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Nicholas, Dainiak, ed. The biology of hematopoiesis: Proceedings of the 15th Annual Frederick Stohlman, Jr., M.D., Memorial Symposium : an International Symposium on the Biology of Hematopoiesis, held in Cambridge, Massachusetts, October 15-20, 1989. Wiley-Liss, 1990.

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International Conference on the Occasion of the 60th Birthday of T.M. Fliedner (1989 Schloss Reisensburg, Germany). The hemopoietic stem cell: The origin and clinical usefulness of harvested peripheral blood stem cells : repair of stem cells and their kinetic properties in recovery from marrow injury by radiation and chemicals. Universitätsverlag Ulm, 1990.

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Brown, Geoffrey, and Rhodri Ceredig. Cell determination during hematopoiesis. Nova Biomedical Books, 2009.

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Symposium on Molecular Biology of Hemopoiesis (3rd 1987 Rye Brook, N.Y.). Molecular biology of hemopoiesis. Plenum Press, 1988.

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Macken, C. A. Stem cell proliferation and differentiation: A multitype branching process model. Springer-Verlag, 1988.

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Book chapters on the topic "Hematopoiesis. Hematopoietic Stem Cells Leukemia"

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Jager, Astraea, Jolanda Sarno, and Kara L. Davis. "Mass Cytometry of Hematopoietic Cells." In Leukemia Stem Cells. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0810-4_5.

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Strzelecka, Paulina M., Anna M. Ranzoni, and Ana Cvejic. "Single-Cell Transcriptomic Analysis of Hematopoietic Cells." In Leukemia Stem Cells. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0810-4_9.

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Venkatraman, Aparna, Sarah E. Smith, Sandra Pinho, Meng Zhao, Linheng Li, and Paul Frenette. "In Situ Hematopoietic Stem Cell Imaging." In Leukemia Stem Cells. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0810-4_23.

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Olender, Leonid, Klil Levy, and Roi Gazit. "Method for the Generation of Induced Hematopoietic Stem Cells." In Leukemia Stem Cells. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0810-4_25.

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Igarashi, Kyomi J., and Ryo Yamamoto. "In Vivo Clonal Analysis of Aged Hematopoietic Stem Cells: Single-Cell Transplantation." In Leukemia Stem Cells. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0810-4_11.

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Papa, Luena, Mansour Djedaini, Manisha Kintali, Christoph Schaniel, and Ronald Hoffman. "Ex Vivo Expansion of Adult Hematopoietic Stem and Progenitor Cells with Valproic Acid." In Leukemia Stem Cells. Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0810-4_16.

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Li, Zhixiong, Ute Modlich, and Mishra Anjali. "Leukemia Diagnosis in Murine Bone Marrow Transplantation Models." In Genetic Modification of Hematopoietic Stem Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-409-4_21.

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Sroczynska, Patrycja, Christophe Lancrin, Stella Pearson, Valerie Kouskoff, and Georges Lacaud. "In Vitro Differentiation of Embryonic Stem Cells as a Model of Early Hematopoietic Development." In Leukemia. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_16.

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Zhang, Wenjun, Guangming Wang, and Aibin Liang. "DNA Damage Response in Quiescent Hematopoietic Stem Cells and Leukemia Stem Cells." In Advances in Experimental Medicine and Biology. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-7342-8_7.

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Hoang, Van T., Isabel Hoffmann, Karina Borowski, et al. "Identification and Separation of Normal Hematopoietic Stem Cells and Leukemia Stem Cells from Patients with Acute Myeloid Leukemia." In Stem Cell Niche. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-508-8_19.

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Conference papers on the topic "Hematopoiesis. Hematopoietic Stem Cells Leukemia"

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Hemmati, Shayda, Taneisha Sinclair, Meng Tong, et al. "Abstract IA18: The PI3K isoforms in myeloid leukemia and hematopoietic stem cells." In Abstracts: AACR Special Conference on Targeting PI3K/mTOR Signaling; November 30-December 8, 2018; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.pi3k-mtor18-ia18.

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Jan, Max, Thomas M. Snyder, M. Ryan Corces-Zimmerman, Irving L. Weissman, Stephen R. Quake, and Ravindra Majeti. "Abstract 3303: Clonal evolution of pre-leukemic hematopoietic stem cells precedes human acute myeloid leukemia." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3303.

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Jan, Max, Thomas Snyder, M. Ryan Corces-Zimmerman, Stephen Quake, Irving Weissman, and Ravindra Majeti. "Abstract SY35-01: Clonal evolution of pre-leukemic hematopoietic stem cells precedes human acute myeloid leukemia." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-sy35-01.

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Yuan, Na, Qi Wang, Aihong Zhang, et al. "Abstract LB-40: Autophagy prevents leukemogenesis by protecting normal hematopoietic stem cells from transforming to leukemia stem cells." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-lb-40.

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Sugihara, Eiji, Takatsune Shimizu, Jo Ishizawa, Seiji Okada, Masanao Miwa, and Hideyuki Saya. "Abstract 3098: Myc preferentially transforms hematopoietic stem cells into Precursor B cell lymphoblastic leukemia/lymphoma." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3098.

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Jan Schuurhuis, Gerrit, Lisa Min, Monique Terwijn, et al. "Abstract A29: High aldehyde dehydrogenase activity is general marker for normal hematopoietic stem cells but not leukemic stem cells in acute myeloid leukemia." In Abstracts: AACR International Conference on Translational Cancer Medicine--; Mar 21–24, 2010; Amsterdam, The Netherlands. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1078-0432.tcme10-a29.

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Xie, Stephanie Z., Elisa Laurenti, and John E. Dick. "Abstract 4792: Elucidating stem cell-specific metabolic pathways in normal and malignant hematopoiesis to target human acute myeloid leukemia stem cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4792.

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Schuurhuis, Gerrit Jan, Lisa A. Min, Michael H. Meel, et al. "Abstract LB-45: High aldehyde dehydrogenase activity is a marker for normal hematopoietic stem cells but not leukemic stem cells in acute myeloid leukemia: novel therapeutic implications." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-lb-45.

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Merlo, ME Boggio, M. Mallardo, G. De Conti, et al. "PO-272 Leukemia-associated NPM mutations promote quiescence of hematopoietic stem cells and prevent their functional exhaustion upon oncogene-induced hyper-proliferation." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.303.

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leeuw, David C. de, Fedor Denkers, Peter Valk, et al. "Abstract 3890: MicroRNA-551b is highly expressed in hematopoietic stem cells and expression in acute myeloid leukemia is associated with relapse and poor survival." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3890.

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