Relevant bibliographies by topics / Hemodynamic recovery

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Hemodynamic recovery'

Author: Grafiati
Published: 28 May 2022
Last updated: 29 May 2022

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Journal articles on the topic "Hemodynamic recovery"

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Zheng, Hai, Yunlong Huo, Mark Svendsen, and Ghassan S. Kassab. "Effect of blood pressure on vascular hemodynamics in acute tachycardia." Journal of Applied Physiology 109, no. 6 (2010): 1619–27. http://dx.doi.org/10.1152/japplphysiol.01356.2009.

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Paroxysmal supraventricular tachycardia is accompanied by hypotension, which can affect vascular hemodynamics. Here, we hypothesized that a fall in blood flow as a result of hypotension has a larger effect on hemodynamics in medium-sized peripheral arteries compared with increased pulsatility in rapid pacing. To test this hypothesis, we experimentally and theoretically investigated hemodynamic changes in femoral, carotid, and subclavian arteries at heart rates of 95–170 beats/min after acute pacing. The arterial pressure, blood flow, and other hemodynamic parameters remained statistically unchanged for heart rates ≤135 beats/min. Systemic pressure and flow velocities, however, showed an abrupt decrease, resulting in larger alteration of hemodynamic parameters for heart rates ≥155 beats/min after pacing (initial period) and then recovered close to baseline after several minutes of pacing (recovery period). During the initial period, the pressure dropped from 88 mmHg (baseline) to 44 mmHg, and the flow velocity decreased to about one-third of baseline at heart rate of 170 beats/min. A hemodynamic analysis showed a velocity profile with a near-wall retrograde flow or a fully reversed flow during the initial period, which vanished at the recovery period. It was concluded that the initial fall of blood flow due to pressure drop led to transient flow reversal and negative wall shear stress because this phenomena was not observed at the recovery period. This study underscores the significant effects of hypotension on vascular hemodynamics, which may have relevance to physiology and chronic pathophysiology in paroxysmal supraventricular tachycardia.
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Sone, Yasuyuki, Anne Nicolaysen, and Norman C. Staub. "Effect of particles on sheep lung hemodynamics parallels depletion and recovery of intravascular macrophages." Journal of Applied Physiology 83, no. 5 (1997): 1499–507. http://dx.doi.org/10.1152/jappl.1997.83.5.1499.

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Sone, Yasuyuki, Anne Nicolaysen, and Norman C. Staub, Sr.Effect of particles on sheep lung hemodynamics parallels depletion and recovery of intravascular macrophages. J. Appl. Physiol. 83(5): 1499–1507, 1997.—We previously showed in newborn lambs that the pulmonary hemodynamic responses to foreign particulate matter (liposomes; Monastral blue) developed in parallel with the maturation of the pulmonary intravascular macrophage system. We now report our use of the liposome-encapsulated heavy-metal-chelating agent dichloromethylene diphosphonate to deplete the intravascular macrophages of small lambs. Functionally and by quantitative histology, we depleted the vast majority of the intravascular macrophages (71% by Monastral blue particle retention, n = 22; 77% by histology; n = 2). Depletion success increased to >90% as we optimized the liposome-depletion regime. Recovery of the lung hemodynamic response began within 3 days. By 2 wk, the functional responses had fully recovered ( n = 8), and, according to quantitative histology, the macrophage population ( n = 2) had recovered 65%. Macrophage depletion in lambs is relatively inexpensive and easy to achieve. It is a safe procedure and is followed by full recovery in ∼2 wk, provided that an aseptic technique is used to prevent bacteremia.
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Aggarwal, Akanksha, and Divya Mahajan. "Comparison of Dexmedetomidine with Fentanyl and Pentazocine – Promethazine in patients undergoing dilation and curettage in monitored anesthesia care." Indian Journal of Clinical Anaesthesia 8, no. 3 (2021): 396–400. http://dx.doi.org/10.18231/j.ijca.2021.076.

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Dilatation and curettage (D and C) is an essential and common minor surgery in obstetrics and gynecology. Sedation, hypnosis and analgesia are used in combination for such short procedures. These days conscious sedation is provided to patients for day care surgeries which includes analgesia, sedation and anxiolysis while rapid recovery is ensured without side effects. Dexmedetomidine is a highly selective alpha-2 agonist that provides anxiolysis and conscious sedation without respiratory depression. It was to study the effect of dexemedetomidine with fentanyl versus Pentazocine with promethazine on hemodynamic stability and recovery during sedation in dilatation and curettage procedure. The comparison included the hemodynamic data and recovery time. The effect of the drugs on hemodynamics and monitoring the occurrence of any complication were also done. In our study, 50 patients were randomly divided into 2 equal groups; group DF received dexmedetomidine loading dose 1 μg/kg over 10 min and followed by 0.5 μg/kg/hr infusion till completion of surgery and group PP received pentazocine 0.5 mg/kg (max 30mg) and Promethazine 12.5 mg slow intravenous Bolus. Dexmedetomidine is a safe drug which provides good hemodynamics and less recovery time. It also exerts sedative and analgesic effects without respiratory depression unlike most analgesic/sedative drugs, such as ketamine, pentazocine and benzodiazepines. This study demonstrates that dexmedetomidine is a safe drug with good hemodynamic and recovery profile. Dexmedetomidine administration showed better preservation of MAP and SpO2.
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Brown, S. P., H. Li, L. F. Chitwood, E. R. Anderson, and J. D. Boatwright. "489 RECOVERY THERMAL AND HEMODYNAMIC RESPONSES." Medicine & Science in Sports & Exercise 25, Supplement (1993): S86. http://dx.doi.org/10.1249/00005768-199305001-00491.

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Roberson, Kirk B., Joseph F. Signorile, Carlos Singer, et al. "Hemodynamic responses to an exercise stress test in Parkinson’s disease patients without orthostatic hypotension." Applied Physiology, Nutrition, and Metabolism 44, no. 7 (2019): 751–58. http://dx.doi.org/10.1139/apnm-2018-0638.

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The presence of postganglionic sympathetic denervation is well established in Parkinson’s disease (PD). Denervation at cardiac and blood vessel sites may lead to abnormal cardiovascular and hemodynamic responses to exercise. The aim of the present investigation was to examine how heart rate (HR) and hemodynamics are affected by an exercise test in PD patients without orthostatic hypotension. Thirty individuals without orthostatic hypotension, 14 individuals with PD, and 16 age-matched healthy controls performed an exercise test on a cycle ergometer. Heart rate, blood pressure, and other hemodynamic variables were measured in a fasted state during supine rest, active standing, exercise, and supine recovery. Peak HR and percent of age-predicted maximum HR (HRmax) achieved were significantly blunted in PD (p < 0.05, p < 0.01). HR remained significantly elevated in PD during recovery compared with controls (p = 0.03, p < 0.05). Systolic, diastolic, and mean arterial pressures were significantly lower at multiple time-points during active standing in PD compared with controls. Systemic vascular resistance index (SVRI) decreased significantly at the onset of exercise in PD, and remained significantly lower during exercise and the first minute of supine recovery. End diastolic volume index (EDVI) was significantly lower in PD during supine rest and recovery. Our results indicate for the first time that normal hemodynamics are disrupted during orthostatic stress and exercise in PD. Despite significant differences in EDVI at rest and during recovery, and SVRI during exercise, cardiac index was unaffected. Our finding of significantly blunted HRmax and HR recovery in PD patients has substantial implications for exercise prescription and recovery guidelines.
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Tan, Qitao, Yan Wang, Tony Lin-Wei Chen, et al. "Exercise-Induced Hemodynamic Changes in Muscle Tissue: Implication of Muscle Fatigue." Applied Sciences 10, no. 10 (2020): 3512. http://dx.doi.org/10.3390/app10103512.

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This research aims to investigate the development of muscle fatigue and the recovery process revealed by tissue oxygenation. The tissue hemodynamics were measured by near-infrared spectroscopy (NIRS) during a 30-min pre-exercise rest, a 40-cycle heel-lift exercise and a 30-min post-exercise recovery. Wavelet transform was used to obtain the normalized wavelet energy in six frequency intervals (I–VI) and inverse wavelet transform was applied to extract exercise-induced oscillations from the hemodynamic signals. During the exercise phase, the contraction-related oscillations in the total hemoglobin signal (ΔtHb) showed a decreasing trend while the fluctuations in the tissue oxygenation index (TOI) displayed an increasing tendency. The mean TOI value was significantly higher (p < 0.001) under recovery (65.04% ± 2.90%) than that under rest (62.35% ± 3.05%). The normalized wavelet energy of the ΔtHb signal in frequency intervals I (p < 0.001), II (p < 0.05), III (p < 0.05) and IV (p < 0.01) significantly increased by 43.4%, 23.6%, 18.4% and 21.6% during the recovery than that during the pre-exercise rest, while the value in interval VI (p < 0.05) significantly decreased by 16.6%. It could be concluded that NIRS-derived hemodynamic signals can provide valuable information related to muscle fatigue and recovery.
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Peter, Vigil, Sumesh Mathew, and Tom Thomas. "Thiopentone versus propofol-anaesthetic of choice in patients undergoing modified electroconvulsive therapy." International Journal of Research in Medical Sciences 5, no. 5 (2017): 1908. http://dx.doi.org/10.18203/2320-6012.ijrms20171816.

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Background: The use of electroconvulsive therapy (ECT) as a treatment modality has increased over the recent years. This is largely due to the use of general anaesthetics, which reduces the physical and psychological trauma associated with the procedure. We attempted to compare the hemodynamic variations and recovery characteristics, along with their effect on seizure quality in patients induced with Thiopentone /Propofol, for Modified ECT.Methods: This was a prospective, randomised controlled study, involving 80 patients. Patients in group 1 received Thiopentone 5 mg/kg, while patients in group 2 received Propofol 1 mg/kg. The hemodynamic status and recovery status were monitored in both the groups for the first thirty minutes. Seizural duration were also recorded. Data was analysed using Students t-test and Pearson Chi-square test.Results: The induction time as well as recovery time was found to be significantly lesser (p <0.05) in the propofol group. The hemodynamic response to was blunted and returned to baseline levels within 10-15 minutes after ECT in the propofol group, whereas it persisted even after 30 minutes in the thiopentone group. There was however, no significant difference in the duration of the seizural activity(p> 0.05).Conclusions: The quick and smooth induction, transient changes in hemodynamics, rapid recovery profile and minimal effects on the seizure quality altogether makes Propofol the preferred anaesthetic agent in Modified ECT.
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Villa, Gianluca, Faeq Husain-Syed, Thomas Saitta, et al. "Hemodynamic Instability during Acute Kidney Injury and Acute Renal Replacement Therapy: Pathophysiology and Clinical Implications." Blood Purification 50, no. 6 (2021): 729–39. http://dx.doi.org/10.1159/000513942.

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Hemodynamic instability associated with acute renal replacement therapy (aRRT, HIRRT) and/or with acute kidney injury (AKI) is frequently observed in the intensive care unit; it affects patients’ renal recovery, and negatively impacts short- and long-term mortality. A thorough understanding of mechanisms underlying HIRRT and AKI-related hemodynamic instability may allow the physician in adopting adequate strategies to prevent their occurrence and reduce their negative consequences. The aim of this review is to summarize the main alterations occurring in patients with AKI and/or requiring aRRT of those homeostatic mechanisms which regulate hemodynamics and oxygen delivery. In particular, a pathophysiological approach has been used to describe the maladaptive interactions between cardiac output and systemic vascular resistance occurring in these patients and leading to hemodynamic instability. Finally, the potential positive effects of aRRT on these pathophysiological mechanisms and on restoring hemodynamic stability have been described.
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Konecny, Filip. "Rodent General Anesthesia Suitable for Measurement of Experimental Invasive Hemodynamics." European Journal of Biology and Biotechnology 2, no. 4 (2021): 33–43. http://dx.doi.org/10.24018/ejbio.2021.2.4.259.

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In cases of experimentally performed invasive rodent cardiovascular measurements, selected general anesthesia for a non-recovery procedure and its proper pain control plays a fundamental role in obtaining good data recordings. Rodent anesthesia is challenging for several reasons including high metabolic rate with elevated possibility of hypothermia and hypoglycemia during the procedure, large body surface area to adjust drug medication and anticipate drug clearance. In this review article, suitable analgesia, and anesthesia to collect rodent hemodynamics is discussed with examples of commonly used methods and anesthetic combinations to assess rodent hemodynamics. In case of injectable anesthesia, hemodynamic parameters should be measured when HR and mean arterial pressure (MAP) becomes stable. If re-injection is necessary, re-evaluation of HR and MAP is crucial for data integrity. Likewise, to safeguard data quality, longitudinal collection of HRs, HR variability, MAP and body temperature should be provided. For this reason, creation of a rodent hemodynamic anesthesia protocol might be necessary. In many cases, to refine surgical anesthetic protocol suitable for hemodynamic study, pilot experiments might be required to find the correct dose, and to probe for adequacy and duration of anesthesia, anticipating technical and procedural problems. Additionally, ensuring repeatability of the hemodynamic exam, selected experimental anesthetics should not be extensively metabolized. If metabolized, the effects on central and peripheral hemodynamics (HR, pre, afterload and contractility) should be well-known and documented.
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Poitras, Veronica J., David J. Slattery, Brendon J. Gurd, and Kyra E. Pyke. "Evidence that meal fat content does not impact hemodynamic reactivity to or recovery from repeated mental stress tasks." Applied Physiology, Nutrition, and Metabolism 39, no. 11 (2014): 1314–21. http://dx.doi.org/10.1139/apnm-2014-0111.

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The magnitude (reactivity) and duration (recovery) of hemodynamic stress responses are predictive of cardiovascular risk, and fat intake has been shown to enhance hemodynamic reactivity to psychological stress tasks. The objective of this study was to determine the impact of a high-fat meal (HFM) on the magnitude and stability of hemodynamic stress reactivity and recovery. This was assessed by: (i) the peak changes from baseline to during stress for heart rate (HR); mean, systolic, and diastolic blood pressure; cardiac output; and total peripheral resistance; and (ii) the residual arousal in hemodynamic parameters at 2 points post-stress (“early” and “late” recovery). On different days, 10 healthy males (aged 23.2 ± 3.3 years) consumed either a HFM (54 g fat) or low-fat meal (LFM; 0 g fat) (∼1000 calories each), followed by 4 hourly 10-min stress tasks (mental arithmetic and speech tasks). Pre-stress (baseline) parameters did not differ between HFM and LFM conditions (all P > 0.05). Plasma triglycerides were greater following the HFM versus the LFM (P = 0.023). No reactivity or recovery parameters differed between meals (all P > 0.05). Stress reactivity and recovery parameters were stable over the 4 stress tasks (main effects of time, all P > 0.05), with the exception of HR (P < 0.05). Contrary to previous reports, meal fat content did not impact hemodynamic reactivity to laboratory stressors. These data also provide the first evidence that meal fat content does not impact hemodynamic recovery from repeated mental stress tasks.
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Dissertations / Theses on the topic "Hemodynamic recovery"

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Богма, Е. В., О. Н. Зацарная, О. Л. Медведь та Л. Ю. Свириденко. "Влияние малообъемного восстановления гемодинамики при политравме на выделительную функцию почек". Thesis, Сумский государственный университет, 2015. http://essuir.sumdu.edu.ua/handle/123456789/41575.

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Chuang, Chia-Jung, and 莊佳容. "Limb hemodynamic adaptation to a tennis training recovery." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/85718730608115394648.

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碩士<br>臺北巿立體育學院<br>運動技術研究所<br>97<br>Abstract 空格 Purpose: The objective of this study is the influence of training on asymmetric sport such as tennis on aerobic capacity and blood flow in the elbow region. Methods: Nine healthy male professional tennis players are recruited and performed once forehand tennis training for 10 min. Extremities muscle of oxy-hemoglobin, deoxy-hemoglobin, total-hemoglobin (TH) and O2 saturation were measured by NIRS before training, after training and following three days. Results: Higher circumflex of rocket-elbow were found compared to opposite-elbow. And present results showed that TH, oxy-hemoglobin and deoxy-hemoglobin of rocket-elbow were significant higher than those of opposite-elbow in the first and third days after training. O2 saturation of rocket-elbow was significant higher than that of opposite-elbow. Oxy-hemoglobin of right quadriceps was higher than that of left side in the third day after training. In third day after training, a significant increased TH, oxy-hemoglobin and deoxy-hemoglobin were found in right gastrocnemius muscle compared with left side. Conclusion: This study suggested that the blood flow change after acute training may provide more nutrition, oxygen and anabolic hormone to muscle in training muscle, and it is a possible reason for muscle hypertrophy of dominant elbow in tennis player.
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Books on the topic "Hemodynamic recovery"

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Comparison of total peripheral resistance and blood velocity as obtained from Doppler ultrasound waveforms during rest, exercise and recovery. 1991.

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Comparison of total peripheral resistance and blood velocity as obtained from Doppler ultrasound waveforms during rest, exercise and recovery. 1992.

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Comparison of total peripheral resistance and blood velocity as obtained from Doppler ultrasound waveforms during rest, exercise and recovery. 1991.

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Book chapters on the topic "Hemodynamic recovery"

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Duong, D. Jay, and Stewart J. Lustik. "Hemodynamic Instability in the Recovery Room." In Critical Care. Elsevier, 2005. http://dx.doi.org/10.1016/b978-0-323-02262-0.50044-0.

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Immonen, Riikka, and Nick Haywar. "MRI Characterization of Progressive Brain Alterations After Experimental Traumatic Brain Injury: Region Specific Tissue Damage, Hemodynamic Changes and Axonal Injury." In Brain Injury - Pathogenesis, Monitoring, Recovery and Management. InTech, 2012. http://dx.doi.org/10.5772/29829.

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Basile, David P., and Babu J. Padanilam. "Pathogenesis of Acute Kidney Injury." In Kidney Protection, edited by Vijay Lapsia, Bernard G. Jaar, and A. Ahsan Ejaz. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190611620.003.0002.

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Acute kidney injury represents a significant clinical disorder associated with a rapid loss of renal function following a variety of potential insults. This chapter reviews multiple issues related to the pathophysiology of AKI with an emphasis on studies from animal models. Early responses following kidney injury include impaired hemodynamic and bioenergetic responses. Reductions in renal ATP levels occur as a result of compromised fatty acid oxidation and impaired compensation by glycolysis. Sustained reductions in perfusion contribute to extension of AKI characterized by complex inflammatory and cellular injury responses, often leading to cell death. Concurrently, the kidney displays an elegant repair response, leading to successful recovery in most cases, characterized in part by epithelial cell growth, while maladaptive or incomplete recovery of tubules or capillaries can predispose the development of interstitial fibrosis and CKD progression.
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Prasad, Amit, and Kai Singbartl. "ECMO Configurations and Cannulation in Adult Patients." In Extracorporeal Membrane Oxygenation, edited by Marc O. Maybauer. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780197521304.003.0004.

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Extracorporeal membrane oxygenation (ECMO), a form of extracorporeal life support (ECLS), represents a life-saving intervention in patients with cardiac and/or respiratory failure not responding to medical therapy. Blood is drained from a central vein (V), pumped through a membrane oxygenator for oxygenation and CO<sub>2</sub> removal, and ultimately returned into a central vein or artery (A). Veno-arterial (V-A) ECMO is primarily used for patients in cardiogenic shock and provides both hemodynamic and respiratory support. V-A ECMO can limit or prevent secondary end-organ damage and allow time for myocardial recovery while a decision is made for the best definitive treatment. Conventional peripheral V-A ECMO, that is, return via femoral artery, carries the risks of differential oxygenation with upper body hypoxemia and extremity ischemia. Veno-venous (V-V) ECMO can rescue patients with the most severe respiratory failure but does not provide hemodynamic support. V-V ECMO allows for lung rest in patients who otherwise would receive potentially injurious ventilator support. Both V-A and V-V ECMO are invaluable, life-saving tools in patients who are failing maximum medical therapy.
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Rob, Daniel, and Jan Bělohlávek. "ECMO for Myocardial Infarction With Cardiogenic Shock." In Extracorporeal Membrane Oxygenation, edited by Marc O. Maybauer. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780197521304.003.0043.

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Cardiogenic shock (CS) is a life-threatening complication of cardiac disorders defined by reduced cardiac output with critical end-organ hypoperfusion. Its management is challenging. Standard CS therapy involving catecholamines, inotropes, and early revascularization is often insufficient, and mortality remains 50% despite current advanced therapeutic options. Veno-arterial (V-A) extracorporeal membrane oxygenation (ECMO) has been increasingly used in cases of CS as it allows rapid hemodynamic stabilization. ECMO support serves as a bridge to decision, recovery, cardiac transplantation, or long-term mechanical support device destination therapy. Early ECMO initiation, careful patient selection, and comprehensive intensive care by an experienced team is key to a successful outcome in patients with CS. This chapter reviews the clinical management of ECMO in CS.
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Gramegna, Mario, Giulia Nardi, and Federico Pappalardo. "Left Ventricular Venting Strategies During ECMO." In Extracorporeal Membrane Oxygenation, edited by Marc O. Maybauer. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780197521304.003.0045.

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Left ventricular (LV) unloading is a crucial point in the management of patients with severe LV dysfunction supported by extracorporeal membrane oxygenation (ECMO). Indeed, ECMO increases LV afterload, which may ultimately further impair or delay cardiac recovery, worsening prognosis. The choice of the proper strategy at the right time for each patient plays a fundamental role in managing these patients in the intensive care unit. Strict and accurate monitoring of the heart and hemodynamic parameters with echocardiography and pulmonary artery catheter is crucial for decision making about timing of the LV unloading and management of MCS devices, guiding either in upgrading or weaning from mechanical support. A case discussion followed by a critical review of the current literature and multiple-choice questions gives the opportunity to obtain an implementation of new concepts in the management of cardiogenic shock patients by learning more about systematic LV venting.
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Casaer, Michael P., and Greet Van den Berghe. "Nutrition support in acute cardiac care." In The ESC Textbook of Intensive and Acute Cardiovascular Care, edited by Marco Tubaro, Pascal Vranckx, Eric Bonnefoy-Cudraz, Susanna Price, and Christiaan Vrints. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198849346.003.0030.

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Malnutrition in cardiac and critical illness is associated with a compromised clinical outcome. The aim of nutrition therapy is to prevent these complications and particularly to attenuate lean tissue wasting and the loss of muscle force and of physical function. During the last decade, several well-powered randomized controlled nutrition trials have been performed. Their results challenge the existing nutrition practices in critically ill patients. Enhancing the nutritional intake and the administration of specialized formulations failed to evoke clinical benefit. Some interventions even provoked an increased mortality or a delayed recovery. These unexpected new findings might be, in part, caused by an important leap forward in the methodological quality in the recent trials. Perhaps reversing early catabolism in the critically ill patient by nutrition or anabolic interventions is impossible or even inappropriate. Nutrients effectively suppress the catabolic intracellular autophagy pathway. But autophagy is crucial for cellular integrity and function during metabolic stress, and consequently its inhibition early in critical illness might be deleterious. Evidence from large nutrition trials, particularly in acute cardiac illness, is scarce. Full enteral feeding in vasopressor dependent patients recovering from hemodynamic shock increases the risk for bowel ischemia. Nutrition therapy is therefore focused on avoiding iatrogenic harm. Some enteral nutrition is administered if possible and eventually temporary hypocaloric feeding is tolerated. Above all, the refeeding syndrome and other nutrition-related complications should be prevented. There is no indication for early parenteral nutrition, increased protein doses, specific amino acids, or modified lipids in critical illness.
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Linakis, Seth. "My Belly Hurts—And I Got Hit by the Hulk." In Pediatric Traumatic Emergencies. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190946623.003.0007.

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Abdominal trauma in pediatric patients can result in a variety of serious pathologies including solid organ lacerations, hollow viscus injuries, and others. Physical findings can provide insight to specific injuries. Management of children with intra-abdominal injuries should be guided by clinical picture and hemodynamic status rather than imaging, although imaging and laboratory testing are still frequently a useful part of the workup. Most patients may be managed nonoperatively in the case of solid organ injury, although a somewhat higher proportion receive packed red blood cells. Hollow viscus injuries can be more difficult to diagnose, as there are no reliable modalities for detecting them. Overall, however, most children with abdominal trauma recover and do very well long-term.
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Asleh, Rabea, and Sarah Schettle. "Device Selection for Short- and Long-Term Mechanical Circulatory Support." In Mechanical Circulatory Support. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190909291.003.0010.

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Mechanical circulatory support has expanded to meet the needs of a variety of patient populations requiring short-term and long-term applications and is utilized internationally. Short-term support devices offer treatment for cardiogenic shock and consist of varying support mechanisms, including univentricular and biventricular support options to improve hemodynamics and tissue perfusion. Investigational devices offer additional options to consider in device selection. Long-term support with ventricular assist devices or total artificial heart offers dischargeable options for lifelong support, recovery, or while patients await organ transplantation. Consideration of device availability, expenses and coverage, operator expertise, and technical challenges should be undertaken when determining patient support strategies. Reviewing patient comorbidities and time frame needed for support is imperative when considering device options to ensure appropriate device selection for each individual patient, thus mitigating risks and maximizing outcomes. Innovation will continue to drive progress in mechanical circulatory support with ongoing development of novel strategies to afford new options to optimize support of patients with heart failure.
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Daubenspeck, Danisa, and Mark A. Chaney. "Coronary Artery Bypass Grafting." In Cardiac Anesthesia: A Problem-Based Learning Approach, edited by Mohammed M. Minhaj. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190884512.003.0002.

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An estimated 92 million adults in the United States have cardiovascular disease, costing about $316 billion dollars in annual health care expenditures. Furthermore, it is estimated that about 16 million Americans greater than 20 years old have coronary artery disease (CAD), which results in a disruption of the oxygen supply-demand relationship in the myocardium and can have adverse effects on the function of the heart. Management of CAD involves both nonsurgical and surgical interventions, of which coronary artery bypass grafting (CABG) is the main surgical option. The majority of CABG surgery is done with the assistance of the cardiopulmonary bypass circuit (CPB), although in the last 30 years there has been a trend toward performing CABG without CPB, also known as off-pump CABG. Many cardiac surgical patients have other medical comorbidities that make significant contributions to their ability to recover. Management of the patient requiring CABG, both with and without CPB, poses several challenges for the cardiac anesthesiologist. These include planning for appropriate monitoring of hemodynamics and oxygenation, obtaining adequate intravascular access, and anticipating and reacting to changes in pathophysiology related to CPB.
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Conference papers on the topic "Hemodynamic recovery"

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Pandia, Keya, Sourabh Ravindran, Gregory T. A. Kovacs, Laurent Giovangrandi, and Randy Cole. "Chest-accelerometry for hemodynamic trending during valsalva-recovery." In 2010 3rd International Symposium on Applied Sciences in Biomedical and Communication Technologies (ISABEL 2010). IEEE, 2010. http://dx.doi.org/10.1109/isabel.2010.5702877.

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Oliveira, Rudolf, Manyoo Agarwal, Roza Badreslam, Alexander Opotowsky, Aaron Waxman, and David Systrom. "Central hemodynamic patterns during recovery from peak exercise." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2449.

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Agnoleto, Aline, Alessandro Heubel, Erika Kabbach, et al. "Arterial stiffness, hemodynamic and functional profile at exacerbation and recovery in COPD." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa1492.

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Sheriff, Jawaad, Jolyon Jesty, and Danny Bluestein. "Platelet Damage Accumulation and Recovery due to Hemodynamic Shear Stresses: An In Vitro Study." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192561.

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It is well established that shear stress exposure activates platelets, and it has been shown that this flow-induced activation contributes significantly to thromboembolic complications in mechanical heart valves (MHVs) [1]. In addition, the platelet activation state (PAS) assay has been demonstrated to be an efficient technique to measure procoagulant activity [2]. However, there is a lack of reliable models to predict platelet damage accumulation. Such a tool allows thrombogenicity optimization of implanted prosthetic devices. Prior to developing this tool, certain aspects of platelet behavior in response to shear stress must be elucidated. Of special importance for developing accountable damage accumulation models is the recovery potential of platelets during repeated passages through devices, when not exposed to the elevated stresses characterizing blood flow in these devices. To accomplish this, PAS measurements were conducted in a Hemodynamic Shearing Device (HSD), where platelets were exposed to prescribed waveforms with alternating periods of high and low shear stresses.
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Banerjee, Rupak K., Lloyd H. Back, and Martin R. Back. "Phasic Variations and Magnitude of Pressure Recovery Distal to Human Coronary Artery Stenoses During Angioplasty." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32581.

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Distal pressure recovery coefficients (cpr = kcpr∞) were determined from pulsatile hemodynamic computations for the 32 patient group of Wilson et al. 1988 during coronary angioplasty in conjunction with quantitative angiography and measurements of coronary flow reserve (CFR). Before angioplasty, values of the factor k(t) ranged 2–4 times high than a reference value, cpr∞ = 0.18, and varied during the flow acceleration and deceleration phases of the cardiac cycle. After angioplasty, values of k(t) ranged from 0.8–1.4, roughly the same magnitude as cpr∞ = 0.46, and also varied phasically.
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6

Jaggi, Tejinder Singh. "To compare the effects of dexmedetomidine versus propofol infusion on various parameters intraoperatively and their effects on the recovery profile postoperatively in patients undergoing laparoscopic assisted robotic pelvic surgeries." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685387.

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Methods: 80 ASA physical status I-II patients, 30-65 years, BMI ≤30 undergoing surgery for 120-180 minutes. Computer randomisation, 40 each, in dexmedetomidine group D and in propofol group P. Induction with fentanyl 1.5 mcg mkg−1 and propofol 2 mg kg−1. Maintained with desflurane 3-5% with air 50% and O2 50%. In D group (bolus 0.5 mcg mkg−1 for 10 minutes then maintenance 0.2-0.5 mcg mkg−1 hr−1) and in P group (propofol @ 50-150 mcg kg−1 min−1) started. At docking of robotic arms single dose morphine @ 0.075 mg kg−1 in both groups is given. Hemodynamic stability (MAP and HR) is adjusted within 20% of base line values. Results: Early and intermediate recovery was fast in D group and total fentanyl requirement intraoperatively was less in D group. Discussion: Dexmedetomidine is known to decrease sympathetic outflow and circulating catecholamine’s levels therefore has caused decrease in both MAP and HR similar to propofol. Dexmedetomidine has analgesia sparing effect hence less total fentanyl dose both intraoperatively. Patients with dexmedetomidine are early aroused, so early and intermediate recoveries were faster with dexmedetomidine than propofol. Thus dexmedetomidine may prove to be useful adjuvant for robotic surgeries. Conclusion: Dexmedetomidine more effective for both intraoperative and postoperative analgesia. Recoveries both early and intermediate are faster in dexmedetomidine group.
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7

Zhang, Ruihang, and Yan Zhang. "Pulsatile Flow Characteristics in a Stenotic Aortic Valve Model: An In Vitro Experimental Study." In ASME-JSME-KSME 2019 8th Joint Fluids Engineering Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/ajkfluids2019-4978.

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Abstract Aortic stenosis (AS) is one of the most common valvular heart diseases around the globe. The accurate assessment of AS severity is important and strongly associated with accurate interpretation of the hemodynamic parameters across the stenotic valve. In this study, we conducted in vitro fluid dynamic experiments to investigate the pulsatile flow characteristics of a stenotic aortic valve as a function of heart rate. An in vitro cardiovascular flow simulator was used to generate pulsatile flow with a prescribed waveform (40% systolic period and 4L/min cardiac output) under varied heart rates (50 bpm, 75 bpm and 100 bpm). The stenotic valve was constructed by molding silicone into three-leaflet aortic valve geometries wrapping around thin fabrics which increases its stiffness and tensile strength. Two-dimensional phase-locked particle image velocimetry (PIV) was employed to quantify the flow field characteristics of the stenotic valve. Pressure waveforms were recorded to evaluate the severity of the stenosis via the Gorlin and Hakki equations. Results suggest that as the heart rate increases, the peak pressure gradient across the stenotic aortic valve increases significantly under the same cardiac output. Analysis also shows the estimated aortic valve area (AVA) decreases as the heart rate increases under the same cardiac output using Gorlin equation estimation, while the trend is reversed using Hakki equation estimation. Under phase-locked conditions, quantitative flow characteristics, such as phase-averaged flow velocity, turbulence kinetic energy (TKE) for the stenotic aortic valve were analyzed based on the PIV data. Results suggest that the peak systolic jet velocity downstream of the valve increases as the heart rate increases, implying a longer pressure recovery distance as heart rate increases. While the turbulence at peak systole is higher under the slower heart rate, the faster heart rate contributes to a higher turbulence during the late systole and early diastole phases. Based on the comparison with no-valve cases, the differences in TKE was mainly related to the dynamics of leaflets under different heart rates. Overall, the results obtained in this study demonstrate that the hemodynamics of a stenotic aortic valve is complex and the assessment of AS could be significantly affected by the pulsating rate of the flow.
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8

Oshiro, Carlos André, Guilherme Diogo Silva, Cesar Castello Branco Lopes, and Marcia Rubia Rodrigues Gonçalves. "Man-in-the-barrel syndrome as a neurovascular manifestation after cardiac surgery: report of two cases." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.412.

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Context: The Man-in-the-barrel syndrome is characterized by brachial diparesis with preserved lower limb strength. This syndrome is an uncommon presentation of ischemic stroke. Cardiac surgery with or without cardiopulmonary bypass (CPB) favors hemodynamic and embolic mechanisms of stroke. Case reports: Case 1 - A 51-year-old hypertensive male presented acute bilateral upper limb weakness in the immediate postoperative period of surgical correction of atrial septal defect. CPB was not performed. Neurological examination showed grade III brachial diparesis with right-sided central facial palsy. Brain magnetic resonance imaging revealed diffusion restriction in the right pre-central gyrus, right occipitotemporal junction, and in the left perirolandic area. Case 2 – A previously healthy 53-year-old man presented grade III brachial diparesis with left-side central facial palsy in the immediate postoperative period for correction of Stanford type A aortic dissection. Surgical procedure included a synthetic tube, aortic valve repair, and 116 minutes of CPB. Brain magnetic resonance imaging showed diffusion restriction in the centrum semiovale bilaterally. None of the patients had significant stenosis of intracranial or extracranial vessels in the angiographic studies. Both patients had good recovery of upper limb function with rehabilitation. We believe that hypoperfusion associated with cardiac surgery led to watershed cerebral infarction. Conclusions: The Man-in-the-barrel syndrome is a rare complication of cardiac surgery. This clinical presentation is associated with watershed stroke.
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Qiao, Aike, and Zhanzhu Zhang. "Solid and Fluid Simulations of Vertebral Artery Stenosis Treated With Stents With Different Shapes of Link." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14062.

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Stenting technology has emerged as an effective alternative for treating arterial stenosis, which recovers blood fluency through mechanical scaffold using struts. Different kinds of endovascular stents cause varying degrees of injury on the artery wall, inducing different ratios of post-operative in-stent restenosis (ISR). Therefore, the design of stent structure has a significant influence on the therapeutic effect of stent intervention. From the viewpoint of solid mechanics, the artery is subjected to long-term press by stent strut after intervention, leading to external mechanical force acting on artery. Straightening, mechanical stress, and stress concentration occurred, causing in-stent intimal hyperplasia extremely, which consequently induce ISR. From the viewpoint of hemodynamics, hemodynamic environment is changed to some extent after stenting. As a result, local blood flow is changed greatly. Vortex and low WSS occur, promoting thrombosis and intimal hyperplasia, which induce ISR more easily. To investigate the effect of stents with different links on treating stenotic vertebral artery and the relation between the shape of link and ISR, as well as provide scientific guidelines for designing stent structure and selecting stent in clinical procedure, numerical simulations of solid mechanics and hemodynamics were performed in this paper, which coupled the boundaries of stent, plaque and blood in the stented vertebral arteries using three kinds of stent with different links.
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10

Kim, Byungchan, Zachary Rosenthal, Joseph P. Culver, Jin-Moo Lee, and Adam Q. Bauer. "Patterns of intrinsic neural and hemodynamic activity recover uniquely following stroke." In Optics and the Brain. OSA, 2019. http://dx.doi.org/10.1364/brain.2019.bm2a.6.

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