Relevant bibliographies by topics / Hemodynamic responses

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Hemodynamic responses'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 February 2022

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Journal articles on the topic "Hemodynamic responses"

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Martindale, John, John Mayhew, Jason Berwick, Myles Jones, Chris Martin, Dave Johnston, Peter Redgrave, and Ying Zheng. "The Hemodynamic Impulse Response to a Single Neural Event." Journal of Cerebral Blood Flow & Metabolism 23, no. 5 (May 2003): 546–55. http://dx.doi.org/10.1097/01.wcb.0000058871.46954.2b.

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This article investigates the relation between stimulus-evoked neural activity and cerebral hemodynamics. Specifically, the hypothesis is tested that hemodynamic responses can be modeled as a linear convolution of experimentally obtained measures of neural activity with a suitable hemodynamic impulse response function. To obtain a range of neural and hemodynamic responses, rat whisker pad was stimulated using brief (≤2 seconds) electrical stimuli consisting of single pulses (0.3 millisecond, 1.2 mA) combined both at different frequencies and in a paired-pulse design. Hemodynamic responses were measured using concurrent optical imaging spectroscopy and laser Doppler flowmetry, whereas neural responses were assessed through current source density analysis of multielectrode recordings from a single barrel. General linear modeling was used to deconvolve the hemodynamic impulse response to a single “neural event” from the hemodynamic and neural responses to stimulation. The model provided an excellent fit to the empirical data. The implications of these results for modeling schemes and for physiologic systems coupling neural and hemodynamic activity are discussed.
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Premen, A. J. "Splanchnic and renal hemodynamic responses to intraportal infusion of glucagon." American Journal of Physiology-Renal Physiology 253, no. 6 (December 1, 1987): F1105—F1112. http://dx.doi.org/10.1152/ajprenal.1987.253.6.f1105.

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We compared the 60-min splanchnic and renal hemodynamic responses to intraportal (IPV) and intravenous infusion of glucagon (5 ng.kg-1.min-1) in anesthetized dogs and quantitated the importance of glucose in mediating the renal hemodynamic responses to intraportal infusion of glucagon. Intraportal glucagon elevated superior mesenteric (SMA) and renal (RA) artery blood flows by 8 and 16%, respectively, by minute 15. By minute 30, RA flow had increased by 23%. Thereafter, SMA flow returned to control, while RA flow remained elevated by 24%. Glomerular filtration rate (GFR) followed the same pattern as RA flow over 60 min. Intravenous glucagon elicited smaller hemodynamic responses. During intraportal and intravenous glucagon infusion, plasma glucose rose by 20-25%. Renal hemodynamics were not affected by incremental changes in blood glucose of up to 6.25 mmol/l. At an incremental change in glucose of 10.06 mmol/l, RA flow and GFR were elevated by 12 and 9%, respectively. We conclude that intraportal glucagon infusion increases splanchnic and renal hemodynamics, although the splanchnic response is evanescent. Importantly, hepatic release of glucose into the circulation during intraportal glucagon infusion does not have a significant effect on renal hemodynamics. Thus, similar to intravenous infusion of the hormone, renal hemodynamic responses to intraportal glucagon are independent of and dissociated from elevations in blood glucose produced during hormone infusion.
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D'souza, Olvyna, and Suman Sahu. "Effect of Dexmedetomidine in Attenuating Hemodynamic Responses During Extubation." Indian Journal of Anesthesia and Analgesia 6, no. 2 (2019): 619–25. http://dx.doi.org/10.21088/ijaa.2349.8471.6219.38.

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Jahr, Jonathan S., Bin Kang, Carlos G. Paxtor, and Chang Jian Feng. "HEMODYNAMIC RESPONSES TO PAPAVERINE." American Journal of Therapeutics 2, no. 4 (April 1995): 258–64. http://dx.doi.org/10.1097/00045391-199504000-00007.

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Kennerley, Aneurin J., Sam Harris, Michael Bruyns-Haylett, Luke Boorman, Ying Zheng, Myles Jones, and Jason Berwick. "Early and Late Stimulus-Evoked Cortical Hemodynamic Responses Provide Insight into the Neurogenic Nature of Neurovascular Coupling." Journal of Cerebral Blood Flow & Metabolism 32, no. 3 (November 30, 2011): 468–80. http://dx.doi.org/10.1038/jcbfm.2011.163.

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Understanding neurovascular coupling is a prerequisite for the interpretation of results obtained from modern neuroimaging techniques. This study investigated the hemodynamic and neural responses in rat somatosensory cortex elicited by 16 seconds electrical whisker stimuli. Hemodynamics were measured by optical imaging spectroscopy and neural activity by multichannel electrophysiology. Previous studies have suggested that the whisker-evoked hemodynamic response contains two mechanisms, a transient ‘backwards’ dilation of the middle cerebral artery, followed by an increase in blood volume localized to the site of neural activity. To distinguish between the mechanisms responsible for these aspects of the response, we presented whisker stimuli during normocapnia (‘control’), and during a high level of hypercapnia. Hypercapnia was used to ‘predilate’ arteries and thus possibly ‘inhibit’ aspects of the response related to the ‘early’ mechanism. Indeed, hemodynamic data suggested that the transient stimulus-evoked response was absent under hypercapnia. However, evoked neural responses were also altered during hypercapnia and convolution of the neural responses from both the normocapnic and hypercapnic conditions with a canonical impulse response function, suggested that neurovascular coupling was similar in both conditions. Although data did not clearly dissociate early and late vascular responses, they suggest that the neurovascular coupling relationship is neurogenic in origin.
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Lefferts, Elizabeth C., Alexander J. Rosenberg, Georgios Grigoriadis, Sang Ouk Wee, Stephen Kerber, Kenneth W. Fent, Gavin P. Horn, Denise L. Smith, and Bo Fernhall. "Firefighter hemodynamic responses to different fire training environments." Vascular Medicine 26, no. 3 (February 19, 2021): 240–46. http://dx.doi.org/10.1177/1358863x20987608.

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Firefighting is associated with an increased risk for a cardiovascular (CV) event, likely due to increased CV strain. The increase in CV strain during firefighting can be attributed to the interaction of several factors such as the strenuous physical demand, sympathetic nervous system activation, increased thermal burden, and the environmental exposure to smoke pollutants. Characterizing the impact of varying thermal burden and pollutant exposure on hemodynamics may help understand the CV burden experienced during firefighting. The purpose of this study was to examine the hemodynamic response of firefighters to training environments created by pallets and straw; oriented strand board (OSB); or simulated fire/smoke (fog). Twenty-three firefighters had brachial blood pressure measured and central blood pressure and hemodynamics estimated from the pressure waveform at baseline, and immediately and 30 minutes after each scenario. The training environment did not influence the hemodynamic response over time (interaction, p > 0.05); however, OSB scenarios resulted in higher pulse wave velocity and blood pressure (environment, p < 0.05). In conclusion, conducting OSB training scenarios appears to create the largest arterial burden in firefighters compared to other scenarios in this study. Environmental thermal burden in combination with the strenuous exercise, and psychological and environmental stress placed on firefighters should be considered when designing fire training scenarios and evaluating CV risk.
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Liu, Spencer S., and Randall L. Carpenter. "Hemodynamic Responses to Intravascular Injection of Epinephrine-containing Epidural Test Doses in Adults during General Anesthesia." Anesthesiology 84, no. 1 (January 1, 1996): 81–87. http://dx.doi.org/10.1097/00000542-199601000-00010.

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Background Epidural anesthesia is sometimes initiated during general anesthesia, yet few data exist concerning efficacy of epinephrine-containing test doses. Methods Thirty-six patients were randomized to receive either 0.5 MAC isoflurane, 1 MAC isoflurane, or 0.5 MAC each (1 MAC total) of isoflurane and nitrous oxide. Each subject received intravenous saline followed by three test doses containing 45 mg lidocaine with 7.5, 15, and 30 micrograms epinephrine in a randomized, double-blind fashion. Heart rate and systolic, diastolic, and mean blood pressures were measured for 5 min after injection. Positive hemodynamic criteria identifying intravascular injection were determined from peak increases in hemodynamics during administration of saline. Dose-effect relationships between epinephrine and peak increases in hemodynamics were assessed with linear regression. Minimum required doses of epinephrine to produce peak positive hemodynamic increases on average were determined from linear regression. Results Positive hemodynamic criteria were identified as increases in heart rate &gt; or = 8 beats/min, systolic blood pressure &gt; or = 13 mmHg, diastolic blood pressure &gt; or = 7 mmHg, and mean blood pressure &gt; or = 9 mmHg. Significant dose-effect relationships were observed for epinephrine and peak increases in hemodynamics (correlation coefficients ranged from 0.61-0.91). Minimum required doses of epinephrine ranged from 6 to 19 micrograms depending on hemodynamic measurement and anesthetic group. Conclusions Hemodynamic responses to intravascular injection of test doses vary with dose of epinephrine and depth and type of general anesthetic used. Thus, the 15 micrograms epinephrine contained in the standard test dose may not be sufficient during all anesthetic conditions.
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Ohsumi, H., M. Sakamoto, T. Yamazaki, and F. Okumura. "Effects of fentanyl on carotid sinus baroreflex control of circulation in rabbits." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 256, no. 3 (March 1, 1989): R625—R631. http://dx.doi.org/10.1152/ajpregu.1989.256.3.r625.

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The effects of intravenous administration of fentanyl on carotid sinus baroreflex control of hemodynamics were investigated in chronically instrumented rabbits. Carotid sinus baroreflex was assessed by bilateral carotid occlusion (BCO), and the responses of mean arterial pressure (MAP), heart rate (HR), mean ascending aortic flow (MAF), and total peripheral resistance (TPR) were obtained. Hemodynamic responses to BCO were examined with cumulative doses of 5, 10, and 15 micrograms/kg of fentanyl. Fentanyl did not affect MAP and TPR but reduced HR and MAF dose dependently. Fentanyl did not attenuate the MAP response to BCO significantly. In contrast, fentanyl significantly attenuated the TPR response from 0.126 +/- 0.003 to 0.104 +/- 0.005 mmHg.min-1.ml-1 and augmented the HR response from 31 +/- 2 to 47 +/- 3 beats/min in the conscious state and at 15 micrograms/kg of fentanyl, respectively. The administration of atropine after the fentanyl attenuated MAP and HR responses to 79.9 and 27.7% of those of 10 micrograms/kg of fentanyl, respectively. We suggest that these dissociated hemodynamic responses reflect the vagotonic and sympatholytic effects of fentanyl on the baroreflex pathways.
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Ford, Judith M., Matthew B. Johnson, Susan L. Whitfield, William O. Faustman, and Daniel H. Mathalon. "Delayed hemodynamic responses in schizophrenia." NeuroImage 26, no. 3 (July 2005): 922–31. http://dx.doi.org/10.1016/j.neuroimage.2005.03.001.

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Behnia, Rahim, Colin A. Shanks, Andranik Ovassapian, and Lawrence A. Wilson. "Hemodynamic Responses Associated with Lithotripsy." Anesthesia & Analgesia 66, no. 4 (April 1987): 354???356. http://dx.doi.org/10.1213/00000539-198704000-00014.

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Dissertations / Theses on the topic "Hemodynamic responses"

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Youra, Sean. "Investigating Hemodynamic Responses to Electrical Neurostimulation." DigitalCommons@CalPoly, 2014. https://digitalcommons.calpoly.edu/theses/1285.

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Since the 1900s, the number of deaths attributable to cardiovascular disease has steadily risen. With the advent of antihypertensive drugs and non-invasive surgical procedures, such as intravascular stenting, these numbers have begun to level off. Despite this trend, the number of patients diagnosed with some form of cardiovascular disease has only increased. By 2030, prevalence of coronary heart disease is expected to increase approximately by 18% in the United States. By 2050, prevalence of peripheral arterial occlusive disease is expected to increase approximately by 98% in the U.S. No single drug or surgical intervention offers a complete solution to these problems. Thus, a multi-faceted regimen of lifestyle changes, medication, and device or surgical interventions is usually necessary. A potential adjunct therapy and cost-effective solution for treating cardiovascular disease that has been overlooked is neurostimulation. Recent studies show that using neurostimulation techniques, such as transcutaneous electrical nerve stimulation (TENS), can help to reduce ischemic pain, lower blood pressure, increase blood flow to the periphery, and decrease systemic vascular resistance. The mechanisms by which these hemodynamic changes occur is still under investigation. The primary aim of this thesis is to elucidate these mechanisms through a thorough synthesis of the existing literature on this subject. Neurostimulation, specifically TENS, is thought to modulate both the metaboreflex and norepinephrine release from sympathetic nerve terminals. To test the hypothesis that TENS increases local blood flow, decreases mean arterial pressure, and decreases cutaneous vascular resistance compared to placebo, in which the electrodes are attached but no electrical stimulation is applied, a protocol was developed to test the effect of neurostimulation on healthy subjects. Implementation of this protocol in a pilot study will determine if neurostimulation causes significant changes in blood flow using the most relevant perfusion measurement instrumentation. Before conducting this study, pre-pilot comparison studies of interferential current therapy (IFC) versus TENS, low frequency (4 Hz) TENS versus high frequency (100 Hz) TENS, and electrode placement on the back versus the forearm were conducted. The only statistically significant difference found was that the application of IFC on the back decreased the reperfusion time, meaning that the time required to reach the average baseline perfusion unit value after occlusion decreased. Further pre-pilot work investigating these different modalities and parameters is necessary to ensure that favorable hemodynamic changes can be detected in the pilot study.
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Paul, Erin E. "Central hemodynamic responses to an acute sodium load." Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file 0.33 Mb., 63p, 2005. http://wwwlib.umi.com/dissertations/fullcit/1428183.

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Scott, Nadia Aleyna. "Optical probing of hemodynamic responses in vivo with channelrhodopsin-2." Thesis, University of British Columbia, 2011. http://hdl.handle.net/2429/36449.

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Maintenance of neuronal function depends on the timely delivery of oxygen and glucose through changes in blood flow that are linked to the level of ongoing neuronal and glial activity, yet the mechanisms underlying this stimulus-dependent control of blood flow remain unclear. Here, using transgenic mice expressing channelrhodopsin-2 in a subset of layer 5b pyramidal neurons, we report that changes in intrinsic optical signals and blood flow can be evoked by activation of channelrhodopsin-2 neurons without direct involvement of other cell types. We have used a combination of imaging and pharmacology to examine the importance of glutamatergic synaptic signaling in neurovascular coupling. In contrast to sensory-evoked responses, we observed that glutamate-dependent neuronal signalling is not essential for the production of channelrhodopsin-evoked hemodynamic responses. Our results rather suggest that ChR2-activated neurons are coupled to the surrounding vasculature through a glutamate-dependent astrocytic pathway mediated by the Group I metabotropic glutamate receptor mGluR5.
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Moore, Stephanie M. "EFFECT OF BODY MASS INDEX ON POST-EXERCISE HEMODYNAMIC RESPONSES." UKnowledge, 2014. http://uknowledge.uky.edu/khp_etds/14.

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To assess the relationships of body mass index (BMI) on arterial stiffness at rest and post-maximal treadmill graded exercise testing (GXT). Forty-four apparently healthy, young adult males (22.1 ± 0.48 years) were recruited and divided into either a healthy weight (H, ≤24.9 kg/m2), overweight (OV, 24.9-29.9 kg/m2) or obese (OB, ≥29.9 kg/m2) group based on BMI. All subjects underwent arterial stiffness (carotid-femoral pulse wave velocity, cfPWV), blood pressure (BP), pulse pressure (PP), mean arterial pressure (MAP) and body composition (bioelectrical impedance analysis, BIA) measurements at rest. Following the GXT, measures of arterial stiffness (cfPWV) and BP were acquired. Resting measures of cfPWV, BMI, systolic BP, diastolic BP, MAP, and PP were significantly (p <0.05) greater in OV and OB compared with H. Compared with OV, OB had a greater BMI. Relative peak oxygen consumption (VP2peak) was greater in H compared with OV and OB (p<0.05). systolic BP was positively associated, whereas VO2peak was inversely related to cfPWV (p<0.05). No significant inter-group interactions were observed with cfPWV after the GXT. However, interactions were observed for SBP, DBP and PP (p<0.05). In young men with varying BMI, SBP and VO2peak were associated with resting cfPWV. However, similar cardiovascular responses were observed between groups after a maximal GXT.
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Kelsey, Kira Q. "Familial history of hypertension : hemodynamic responses to exercise in children." Virtual Press, 1996. http://liblink.bsu.edu/uhtbin/catkey/1020150.

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Hypertension afflicts one in four American adults and is a major risk factor for cardiovascular disease. Studies have shown that a family history of hypertension is an important predictor of future hypertension. Two hemodynamic factors control blood pressure (BP); cardiac output (CO) and total peripheral resistance (TPR). Although children of hypertensive parents may exhibit normal levels of these hemodynamic variables at rest, the response of these variables during exercise stress may differ. Therefore, the present study was designed to investigate whether children with a positive family history of hypertension exhibit an exaggerated BP response due to either an increased CO or an attenuated decrease in TPR during dynamic submaximal exercise compared to children of normotensive parents. Eleven children 12.2 ± 1.8 yr (M ± SE) of normotensive parents and 11 children 12.0 ± 2.4 yr of at least one hypertensive parent completed an orientation session, graded maximal cycle ergometer test, and a submaximal exercise bout consisting of 6 minutes of steady state cycling at 50 and 80% of maximal heart rate reserve. Blood pressure, CO and TPR were measured during the last 3 minutes of each submaximal exercise stage. An independent t-test was used to determine differences in the resting measures. The changes in TPR, BP and CO from rest through 80% intensity stage were examined using a twoway (group x intensity) ANOVA. The groups were evenly matched for age, weight, height, and body fatness. The children with a positive family history of hypertension had significantly higher resting systolic BP, diastolic BP (DBP), and mean arterial pressure (MAP) (p<0.05) compared to those children with a negative family history. Although there were no significant interactions among any of the variables studied, there was a tendency for TPR to be higher (p>0.05) at rest and throughout exercise in the positive history group. MAP and DBP were significantly higher in the positive family history group at rest and remained higher throughout exercise. In conclusion children of hypertensive parents exhibit a modest but significantly higher MAP and DBP at rest and during submaximal exercise. This subtle difference in BP control reflects an early trend toward increased TPR.
School of Physical Education
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Herzog, Chad D. "Hemodynamic responses per MET during the BSU/Bruce Ramp protocol." Virtual Press, 2000. http://liblink.bsu.edu/uhtbin/catkey/1179130.

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The purpose of this study was to determine the association of age, gender, and cardiorespiratory fitness level upon normative heart rate and systolic blood pressure (SBP) responses per MET during the BSU/Bruce Ramp protocol. This research was delimited to 451 subjects, 201 men (mean age 46.5 ± 11.9 yrs) and 250 women (mean age 42.9 ± 11.4 yrs), low to moderate risk subjects. The majority of subjects were tested to enter the Ball State University Adult Physical Fitness Program. These subjects were tested using the BSU/Bruce Ramp protocol between 1992 and 1998.Multiple regression showed gender had a positive association upon submaximal SBP values. Gender's association with heart rate was negative between minute 3-6 and positive between minute 6-9. Age only had an association upon submaximal heart rate, which was negative. Cardiorespiratory fitness had a negative association upon SBP between minute 6-9 and a negative association with heart rate between minute 3-6.SBP increased 6.6 ± 4.4 and 6.0 ± 4.2 mmHg/MET between minute 3-6 for men and women, respectively. Analysis of variance demonstrated gender was not statistically significant between minute 3-6. SBP increased 4.7 ± 3.1 and 3.8 + 2.7 mmHg/MET between minute 6-9 for men and women, respectively. Gender was statisticallysignificant between minute 6-9 (p<.05). Heart rate increased 8.5 + 2.3 and 10.7 + 3.3 bpm/MET between minute 3-6 for men and women, respectively. Analysis of variance demonstrated gender was statistically significant between minute 3-6 (p<.05). The increase was 9.5 + 2.3 and 9.2 + 2.7 bpm/MET between minute 6-9 for men and women, respectively. Gender was not statistically significant between minute 6-9.In conclusion, this study demonstrated that the normative hemodynamic responses during the BSU/Bruce Ramp protocol are similar to submaximal normative data previously reported in the literature for incremental type protocols.
School of Physical Education
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Wang, De, and 王得. "A comparison of hemodynamic responses in losartan- and enalapril- treated normotensive rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B31969653.

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Borzage, Matthew Thomas. "Regional cerebral hemodynamic responses to hypoxia in humans using MRI and NIRS." Thesis, University of Southern California, 2014. http://pqdtopen.proquest.com/#viewpdf?dispub=3628123.

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The primary goal of my thesis was to address my hypothesis that: there is preferential perfusion of the hindbrain regions, controlling autonomic function. To test this hypothesis I developed a system for delivering hypoxic challenges to volunteers while they were in the MRI. I developed NIRS protocols that allowed monitoring of the cerebellum. And I developed MRI methods that allowed for PC MRI to be used to monitor flow to the forebrain and hindbrain. Finally I combined these elements to investigate how the brain would react to hypoxia. Ultimately neither NIRS nor MRI detected systematic differences between the forebrain and hindbrain response to hypoxia but the developed methods are available for future studies that aim to explore the hemodynamic response in the developing brain or in adults with pathological conditions.

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Wang, De. "A comparison of hemodynamic responses in losartan- and enalapril- treated normotensive rats." Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21106447.

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LeSage, Susan. "Central and peripheral hemodynamic responses to a tilt table simulation of -/+ Gz transitions." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ39205.pdf.

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Books on the topic "Hemodynamic responses"

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Livingstone, Kristina. The cardiovascular hemodynamic responses to various levels of orthostatic stress in children. St. Catharines, Ont: Brock University, Faculty of Applied Health Sciences, 2007.

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Partanen, Juhani. Cardiovascular responses induced by haemodynamic interventions and inotropics: A series of noninvasive studies. Helsinki: University Central Hospital, 1989.

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Seth, Runjan. Inotropic and lusitropic response to gbs-adrenergic stimulation, hemodynamics, and metabolic parameters in early experimental heart failure. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Cardiovascular dynamics: A psychophysiological study : behavioral control, type A, task performance, test anxiety, and cardiovascular responses. Berwyn [Pa.]: Swets North America, 1986.

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Hemodynamic responses of cardiovascular diseased patients during submaximal cycling in water. 1985.

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Hemodynamic and ADH responses to central blood volume shifts in cardiac-denervated humans. [Washington, D.C: National Aeronautics and Space Administration, 1990.

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Hemodynamic responses to postural change in habitually physically active and inactive older men. 1987.

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Hemodynamic responses to postural change in habitually physically active and inactive older men. 1988.

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Cardiovascular and hemodynamic responses to combinations of exercise before and after a whirlpool bath. 1988.

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Cardiovascular and hemodynamic responses to combinations of exercise before and after a whirlpool bath. 1989.

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Book chapters on the topic "Hemodynamic responses"

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Orbell, Sheina, Havah Schneider, Sabrina Esbitt, Jeffrey S. Gonzalez, Jeffrey S. Gonzalez, Erica Shreck, Abigail Batchelder, et al. "Hemodynamic Stress Responses." In Encyclopedia of Behavioral Medicine, 957. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_100789.

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Perrey, Stéphane. "NIRS for Measuring Cerebral Hemodynamic Responses During Exercise." In Functional Neuroimaging in Exercise and Sport Sciences, 335–49. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3293-7_14.

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Langille, B. L., A. I. Gotlieb, and D. W. Kim. "In vivo Responses of Endothelial Cells to Hemodynamic Stress." In Role of Blood Flow in Atherogenesis, 157–61. Tokyo: Springer Japan, 1988. http://dx.doi.org/10.1007/978-4-431-68399-5_23.

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Rhee, H. M., J. L. Valentine, D. Hendrix, M. Schweisthal, and M. Soria. "Hemodynamic Responses to Vasoactive Compounds in Chronically Alcohol Treated Rats." In Oxygen Transport to Tissue XI, 629–39. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5643-1_70.

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Tuan, Vo Nhut, Nguyen Duc Thang, Vo Van Toi, Tran Le Giang, Nguyen Huynh Minh Tam, and Dinh Dong Luong. "Differentiation of Hemodynamic Responses of the Brain with Typing and Writing." In IFMBE Proceedings, 395–98. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-11776-8_96.

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Shao, Wan-Chen, Chun-Wei Wu, Chun-Man Yuan, and Jia-Jin Jason Chen. "Assessment of Hemodynamic Responses in Cerebral Ischemic Rats Using Near Infrared Spectroscopy." In IFMBE Proceedings, 154–57. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-12262-5_43.

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Engel, B. T. "Nocturnal Hemodynamic Responses to Chronic, Mild Atrial Demand Pacing in Nonhuman Primates." In Temporal Variations of the Cardiovascular System, 164–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-662-02748-6_10.

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Parer, J. T. "Fetal Hemodynamic Responses to Reduced Uterine Blood Flow in the Sheep Fetus." In Fetal Heart Rate Monitoring, 82–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70358-4_10.

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Alpert, Bruce S., Darlene M. Moes, Robert H. DuRant, and William B. Strong. "Hemodynamic Responses to Exercise in Children with Cyanotic Forms of Heart Disease." In Pediatric Cardiology, 241–45. New York, NY: Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4613-8598-1_64.

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Le Giang, Tran, Nguyen Duc Thang, Vo Van Toi, Nguyen Huynh Minh Tam, Dinh Dong Luong, and Truong Quang Dang Khoa. "Evaluation of Hemodynamic Responses to Visual Tasks Using Functional Near Infrared Spectroscopy." In IFMBE Proceedings, 486–90. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-11776-8_120.

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Conference papers on the topic "Hemodynamic responses"

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abdi, Rabah Al, Bin Deng, Aiza Kabeer, and Stefan Carp. "Mechanical and Hemodynamic Responses of Breast Tissue During Compression." In Clinical and Translational Biophotonics. Washington, D.C.: OSA, 2020. http://dx.doi.org/10.1364/translational.2020.jtu3a.4.

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Ni, Songlin, Pengcheng Li, Yuanyuan Yang, Xiaohua Lv, and Qingming Luo. "Hemodynamic responses to functional activation accessed by optical imaging." In ICO20:Biomedical Optics, edited by Gert von Bally and Qingming Luo. SPIE, 2006. http://dx.doi.org/10.1117/12.667100.

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Bighamian, Ramin, Sadaf Soleymani, Andrew T. Reisner, Istvan Seri, and Jin-Oh Hahn. "Modeling and System Identification of Hemodynamic Responses to Vasopressor-Inotropes." In ASME 2013 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/dscc2013-3726.

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In an effort to establish an initial step towards the ultimate goal of developing an analytic tool to optimize the vasopressor-inotrope therapy through individualized dose-response relationships, we propose a phenomenological model intended to reproduce the hemodynamic response to vasopressor-inotropes. The proposed model consists of a cardiovascular model relating blood pressure to cardinal cardiovascular parameters (stroke volume and total peripheral resistance) and the phenomenological relationships between the cardinal cardiovascular parameters and the vasopressor-inotrope dose, in such a way that the model can be adapted to individual patient solely based upon blood pressure and heart rate responses to medication dosing. In this paper, the preliminary validity of the proposed model is shown using the experimental epinephrine dose versus blood pressure and heart rate response data collected from five newborn piglets. Its performance and potential usefulness are discussed. It is anticipated that, potentially, the proposed phenomenological model may offer a meaningful first step towards the automated control of vasopressor-inotrope therapy.
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Nunes, Catarina S., and Pedro Amorim. "A neuro-fuzzy approach for predicting hemodynamic responses during anesthesia." In 2008 30th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2008. http://dx.doi.org/10.1109/iembs.2008.4650536.

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Yamakoshi, T., K. Yamakoshi, S. Tanaka, M. Nogawa, Y. Sawada, and P. Rolfe. "Hemodynamic Responses during Simulated Automobile Driving in a Monotonous Situation." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.259279.

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Yamakoshi, T., K. Yamakoshi, S. Tanaka, M. Nogawa, Y. Sawada, and P. Rolfe. "Hemodynamic Responses during Simulated Automobile Driving in a Monotonous Situation." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.4398608.

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Lu, Feng-Mei, Yi-Feng Wang, and Zhen Yuan. "Hemodynamic responses can modulate the brain oscillations in low frequency." In SPIE BiOS, edited by Steen J. Madsen, Victor X. D. Yang, E. Duco Jansen, Qingming Luo, Samarendra K. Mohanty, and Nitish V. Thakor. SPIE, 2016. http://dx.doi.org/10.1117/12.2211237.

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Dolan, Jennifer, Sukhjinder Singh, Hui Meng, and John Kolega. "Differential Responses of Endothelial Cells to Positive and Negative Wall Shear Stress Gradients." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19535.

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Cerebral aneurysms tend to develop at bifurcation apices or the outer side of curved vessels where the blood vessel wall experiences complex hemodynamics. In vivo studies have recently revealed that the initiation of cerebral aneurysms is confined to a well-defined hemodynamic microenvironment. Specifically aneurysms form where the vessel wall experiences high fluid shear stress (wall shear stress, WSS) and flow is accelerating, so that the wall is exposed to a positive spatial gradient in the fluid shear stress (wall shear stress gradient, WSSG)[1,2]. Closer examination of such in vivo studies reveals that exposure of the vessel wall to equally high WSS in the presence of decelerating flow, that is, negative WSSG, does not result in aneurysm-like remodeling.
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Cao, Rui, Jun Li, Bo Ning, Naidi Sun, Tianxiong Wang, Zhiyi Zuo, and Song Hu. "Photoacoustic microscopy of cerebral hemodynamic and oxygen-metabolic responses to anesthetics." In SPIE BiOS, edited by Qingming Luo and Jun Ding. SPIE, 2017. http://dx.doi.org/10.1117/12.2251684.

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Abston, Eric D., Nicole Sborz, Robert Weiss, Hunter Champion, and Clarke G. Tankersley. "Differential Ventricular Hemodynamic Responses To Particulate Matter (PM) Or Ozone (O3)." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1722.

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Reports on the topic "Hemodynamic responses"

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Ghambaryan, Anna. Heart Rate Variability, Catecholamine and Hemodynamic Responses During Rest and Stress in Coronary Artery Disease Patients: The PIMI Study. Fort Belvoir, VA: Defense Technical Information Center, January 2007. http://dx.doi.org/10.21236/ad1013978.

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Thompson, A., C. R. Valeri, and W. Lieberthal. Endothelial Receptor a Blockade Alters the Hemodynamic Response to Nitric Oxide Inhibition in the Rat. Fort Belvoir, VA: Defense Technical Information Center, June 1994. http://dx.doi.org/10.21236/ada360327.

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Schwieger, Alexandra, Kaelee Shrewsbury, and Paul Shaver. Dexmedetomidine vs Fentanyl in Attenuating the Sympathetic Surge During Endotracheal Intubation: A Scoping Review. University of Tennessee Health Science Center, July 2021. http://dx.doi.org/10.21007/con.dnp.2021.0007.

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Purpose/Background Direct laryngoscopy and endotracheal intubation after induction of anesthesia can cause a reflex sympathetic surge of catecholamines caused by airway stimulation. This may cause hypertension, tachycardia, and arrhythmias. This reflex can be detrimental in patients with poor cardiac reserve and can be poorly tolerated and lead to adverse events such as myocardial ischemia. Fentanyl, a potent opioid, with a rapid onset and short duration of action is given during induction to block the sympathetic response. With a rise in the opioid crisis and finding ways to change the practice in medicine to use less opioids, dexmedetomidine, an alpha 2 adrenergic agonist, can decrease the release of norepinephrine, has analgesic properties, and can lower the heart rate. Methods In this scoping review, studies published between 2009 and 2021 that compared fentanyl and dexmedetomidine during general anesthesia induction and endotracheal intubation of surgical patients over the age of 18 were included. Full text, peer-reviewed studies in English were included with no limit on country of study. The outcomes included post-operative reviews of decrease in pain medication usage and hemodynamic stability. Studies that were included focused on hemodynamic variables such as systolic blood pressure, diastolic blood pressure, mean arterial pressure, heart rate, and use of opioids post-surgery. Result Of 2,114 results from our search, 10 articles were selected based on multiple eligibility criteria of age greater than 18, patients undergoing endotracheal intubation after induction of general anesthesia, and required either a dose of dexmedetomidine or fentanyl to be given prior to intubation. Dexmedetomidine was shown to effectively attenuate the sympathetic surge during intubation over fentanyl. Dexmedetomidine showed a greater reduction in heart rate, systolic blood pressure, diastolic blood pressure, mean arterial pressure than fentanyl, causing better hemodynamic stability in patients undergoing elective surgery.Implications for Nursing Practice Findings during this scoping review indicate that dexmedetomidine is a safe and effective alternative to fentanyl during induction of general anesthesia and endotracheal intubation in attenuating the hemodynamic response. It is also a safe choice for opioid-free anesthesia.
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