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1

Livingstone, Kristina. The cardiovascular hemodynamic responses to various levels of orthostatic stress in children. St. Catharines, Ont: Brock University, Faculty of Applied Health Sciences, 2007.

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2

Partanen, Juhani. Cardiovascular responses induced by haemodynamic interventions and inotropics: A series of noninvasive studies. Helsinki: University Central Hospital, 1989.

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3

Seth, Runjan. Inotropic and lusitropic response to gbs-adrenergic stimulation, hemodynamics, and metabolic parameters in early experimental heart failure. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.

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4

Cardiovascular dynamics: A psychophysiological study : behavioral control, type A, task performance, test anxiety, and cardiovascular responses. Berwyn [Pa.]: Swets North America, 1986.

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5

Hemodynamic responses of cardiovascular diseased patients during submaximal cycling in water. 1985.

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6

Hemodynamic and ADH responses to central blood volume shifts in cardiac-denervated humans. [Washington, D.C: National Aeronautics and Space Administration, 1990.

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7

Hemodynamic responses to postural change in habitually physically active and inactive older men. 1987.

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8

Hemodynamic responses to postural change in habitually physically active and inactive older men. 1988.

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9

Cardiovascular and hemodynamic responses to combinations of exercise before and after a whirlpool bath. 1988.

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10

Cardiovascular and hemodynamic responses to combinations of exercise before and after a whirlpool bath. 1989.

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11

Nalini Naomi Dorothy Joy Jairath. Hemodynamic and subjective responses of 7th to 14th day acute myocardial infarction patients to stair climbing. 1985.

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12

The acute effects of miometric and pliometric muscle actions on delayed onset muscle soreness, hemodynamic responses, and exercise performance. 1989.

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13

The acute effects of miometric and pliometric muscle actions on delayed onset muscle soreness, hemodynamic responses, and exercise performance. 1989.

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14

Stamatakis, Emmanuel A., Eleni Orfanidou, and Andrew C. Papanicolaou. Functional Magnetic Resonance Imaging. Edited by Andrew C. Papanicolaou. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199764228.013.7.

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Functional magnetic resonance imaging (fMRI) is the most frequently used functional neuroimaging method and the one that accounts for most of the neuroimaging literature. It measures the blood oxygen level-dependent (BOLD) signal in different parts of the brain during rest and during task-induced activation of functional networks mediating basic and higher functions. A basic understanding of the various instruments and techniques of recording the hemodynamic responses of different brain regions and the manner in which we establish activation and connectivity patterns out of these responses is necessary for an appreciation of the contemporary functional neuroimaging literature. To facilitate such an understanding is the purpose of this chapter.
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15

Barrett, Betsy. The cardiovascular and hemodynamic responses to psychological stress in college-age females with a parental history of hypertension before and after a 12-week aerobic training program. 1989.

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16

Jackson, Zane Steven. Arterial remodeling in response to hemodynamic and axial forces. 2001.

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17

Effect of decreased gravity on circulation in the rat. [Washington, DC: National Aeronautics and Space Administration, 1987.

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18

United States. National Aeronautics and Space Administration., ed. Effect of decreased gravity on circulation in the rat. [Washington, DC: National Aeronautics and Space Administration, 1987.

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19

Uwe, Mehlhorn, and United States. National Aeronautics and Space Administration., eds. Body position does not affect the hemodynamic response to venous air embolism in dogs. [Washington, D.C: National Aeronautics and Space Administration, 1993.

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20

Uwe, Mehlhorn, and United States. National Aeronautics and Space Administration., eds. Body position does not affect the hemodynamic response to venous air embolism in dogs. [Washington, D.C: National Aeronautics and Space Administration, 1993.

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21

Hematological changes in response to moderate altitudes. 1985.

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22

Kaplan, Linda. The effect of wrist weight on the hemodynamic response to exercise in coronary artery disease. 1993.

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23

Hematological changes in response to exposure to moderate altitudes. 1985.

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24

Hemodynamic response to full (-90)̊ inversion before exercise and after exercise at 75% intensity on the treadmill. 1986.

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25

Hemodynamic response to full (-90)̊ inversion before exercise and after exercise at 75% intensity on the treadmill. 1987.

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26

Hemodynamic response to full (-90)̊ inversion before exercise and after exercise at 75% intensity on the treadmill. 1987.

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27

Hemodynamic response to full (-90ê) inversion before exercise and after exercise at 75% intensity on the treadmill. 1987.

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28

Cardiovascular responses to static and isodynamic exercise with similar muscle masses. 1986.

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29

Cardiovascular responses to static and isodynamic exercise with similar muscle masses. 1988.

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30

Bayer, Ilana. Regulation of matrix degrading enzymes in arterial remodeling during development and in response to experimental alterations in hemodynamic forces. 2002.

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31

Wang, Cynthia, and Michelle Y. Braunfeld. Acute Liver Failure. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0035.

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Acute liver failure produces widespread physiologic derangements including encephalopathy, coagulopathy, peripheral vasodilation, a systemic inflammatory response, and multiorgan failure. Morbidity is significant, and mortality is 50%. The classification of liver failure and the various etiologies, including viral hepatitis, drug-induced, toxins, and autoimmunity are reviewed here. The multisystem effects of acute liver failure influence all aspects of perioperative care and adequate supportive care during this time is crucial to providing the best possible outcome for the patient. Specific treatment objectives and recommendations are discussed, and the anesthetic management with regard to drug choices, hemodynamic goals, and intraoperative monitoring is reviewed.
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32

Myers, Sara P., and Matthew D. Neal. Management of Exsanguinating Hemorrhage: Hemostasis and Resuscitation (DRAFT). Edited by Raghavan Murugan and Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0022.

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This chapter describes the approach to massive bleeding in a patient within the setting of rapid response team (RRT) calls. Emphasis is placed on identifying peripheral sources of bleeding that can be controlled with compression. Military experience has aided in the understanding of how zeolite and clay can be used as chemical adjuncts. Resuscitative strategies and transfusion practices that promote hemodynamic stability are reviewed. Concurrent coagulopathy that can exacerbate hemorrhage is addressed and a brief overview is provided demonstrating how commonly prescribed anticoagulants can be monitored and reversed. To illustrate that multimodal therapy is often necessary to control hemorrhage, the chapter closes with surgical and minimally invasive techniques for definitive hemostasis.
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33

Brown, Stanley Paul. Left ventricular inotropic response during weight lifting in untrained, weight trained, and endurance trained men: Evaluation by echocardiography. 1990.

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34

Left ventricular inotropic response during weight lifting in untrained, weight trained, and endurance trained men: Evaluation by echocardiography. 1990.

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35

Brown, Stanley P. Left ventricular inotropic response during weight lifting in untrained, weight trained, and endurance trained men: Evaluation by echocardiography. 1989.

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36

Left ventricular inotropic response during weight lifting in untrained, weight trained, and endurance trained men: Evaluation by echocardiography. 1990.

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37

Chappell, Michael, Bradley MacIntosh, and Thomas Okell. Introduction to Perfusion Quantification using Arterial Spin Labelling. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198793816.001.0001.

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Arterial spin labeling (ASL) magnetic resonance imaging (MRI) is unique in being a completely non-invasive method for imaging perfusion in the brain. Relying upon a blood-borne tracer that is created by the MRI scanner itself, ASL is becoming a popular tool to study cerebral perfusion, as well as how this perfusion changes in response to neuronal activity or in disease. This primer provides an introduction to perfusion quantification using ASL MRI, focusing both on the methods needed to extract perfusion-weighted images and on how to quantify perfusion and other hemodynamic parameters. Starting with the simplest implementation of ASL, the primer details all the common acquisition methods, as well as the subsequent analysis steps required to quantify perfusion in an individual, detect changes in perfusion in response to neural activity or pharmacological intervention, and examine perfusion variations across groups of individuals. This is supported with examples from real data illustrating all the major steps in the analysis process, linked to online material where the reader can undertake the same analysis for themselves.
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38

Pfurtscheller, Gert, and Fernando Lopes da Silva. EEG Event-Related Desynchronization and Event-Related Synchronization. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0040.

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Event-related desynchronization (ERD) reflects a decrease of oscillatory activity related to internally or externally paced events. The increase of rhythmic activity is called event-related synchronization (ERS). They represent dynamical states of thalamocortical networks associated with cortical information-processing changes. This chapter discusses differences between ERD/ERS and evoked response potentials and methodologies for quantifying ERD/ERS and selecting frequency bands. It covers the interpretation of ERD/ERS in the alpha and beta bands and theta ERS and alpha ERD in behavioral tasks. ERD/ERS in scalp and subdural recordings, in various frequency bands, is discussed. Also presented is the modulation of alpha and beta rhythms by 0.1-Hz oscillations in the resting state and phase-coupling of the latter with slow changes of prefrontal hemodynamic signals (HbO2), blood pressure oscillations, and heart rate interval variations in the resting state and in relation to behavioral motor tasks. Potential uses of ERD-based strategies in stroke patients are discussed.
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39

The relationship of aerobic fitness, Type A behavior pattern, and hostility to baroreflex responses and cardiovascular reactivity to nonexertional stressors. 1990.

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40

A comparison of male and female runners, weight lifters, and sedentary controls in the R-amp response to exercise testing. 1989.

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41

A comparison of male and female runners, weight lifters, and sedentary controls in the R-amp response to exercise testing. 1990.

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42

A comparison of male and female runners, weight lifters, and sedentary controls in the R-amp response to exercise testing. 1991.

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43

A comparison of male and female runners, weight lifters, and sedentary controls in the R-amp response to exercise testing. 1991.

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