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Relevant bibliographies by topics / Hemodynamics. Striated muscle Muscles

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Journal articles

Academic literature on the topic 'Hemodynamics. Striated muscle Muscles'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Hemodynamics. Striated muscle Muscles"

1

Kuo, L., and R. N. Pittman. "Influence of hemoconcentration on arteriolar oxygen transport in hamster striated muscle." American Journal of Physiology-Heart and Circulatory Physiology 259, no. 6 (1990): H1694—H1702. http://dx.doi.org/10.1152/ajpheart.1990.259.6.h1694.

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We investigated the influence of isovolemic hemoconcentration on microcirculatory hemodynamics and oxygen transport in the hamster cheek pouch retractor muscle. In 17 hamsters, measurements of red blood cell velocity, hematocrit, vessel diameter, segment length (L), hemoglobin oxygen saturation (SO2), and longitudinal SO2 gradient (delta SO2/L) were made in four branching orders of arterioles before and after isovolemic exchange with packed red blood cells. Hemoconcentration increased systemic hematocrit from 50 to 65%; systemic blood gases were unchanged, but mean arterial blood pressure incr

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2

Thorlacius, Henrik, Brigitte Vollmar, Simone Westermann, Leif Torkvist, and Michael D. Menger. "Effects of Local Cooling on Microvascular Hemodynamics and Leukocyte Adhesion in the Striated Muscle of Hamsters." Journal of Trauma: Injury, Infection, and Critical Care 45, no. 4 (1998): 715–19. http://dx.doi.org/10.1097/00005373-199810000-00016.

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3

Kuo, L., and R. N. Pittman. "Effect of hemodilution on oxygen transport in arteriolar networks of hamster striated muscle." American Journal of Physiology-Heart and Circulatory Physiology 254, no. 2 (1988): H331—H339. http://dx.doi.org/10.1152/ajpheart.1988.254.2.h331.

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Experiments were performed on the hamster cheek pouch retractor muscle to investigate the influence of isovolemic hemodilution on microcirculatory hemodynamics and the rate of oxygen transport to striated muscle. In 23 hamsters, measurements of red blood cell velocity, hematocrit, vessel diameter, segment length (L), hemoglobin oxygen saturation (SO2), and longitudinal SO2 gradient (delta SO2/L) were made in four branching orders of arterioles before and after isovolemic exchange with plasma. Hemodilution decreased systemic hematocrit from 52 to 33%. In first- through fourth-order arterioles,

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4

Sulbarán, Guidenn, Lorenzo Alamo, Antonio Pinto, et al. "An invertebrate smooth muscle with striated muscle myosin filaments." Proceedings of the National Academy of Sciences 112, no. 42 (2015): E5660—E5668. http://dx.doi.org/10.1073/pnas.1513439112.

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Muscle tissues are classically divided into two major types, depending on the presence or absence of striations. In striated muscles, the actin filaments are anchored at Z-lines and the myosin and actin filaments are in register, whereas in smooth muscles, the actin filaments are attached to dense bodies and the myosin and actin filaments are out of register. The structure of the filaments in smooth muscles is also different from that in striated muscles. Here we have studied the structure of myosin filaments from the smooth muscles of the human parasite Schistosoma mansoni. We find, surprisin

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5

Mironov, S. P., N. A. Es'kin, A. K. Orletskiy, L. L. Laylin, D. R. Bogdashevskiy, and L. S. Arhzakova. "Ultrasound Diagnosis of Striated Muscles Pathology." N.N. Priorov Journal of Traumatology and Orthopedics 12, no. 1 (2005): 24. http://dx.doi.org/10.17816/vto20050124.

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Ultrasound examination of 897 patients with injuries and pathology of striated muscles was performed. Ultrasound criteria of muscle diseases and injuries were detected. The evaluation of diagnostic efficacy of ultrasound data relative to data of invasive methods (surgical interventions and diagnostic puncture) was performed. Ultrasound examination of striated muscles is showed to be a highly specific and sensitive method for diagnosis of traumatic muscle tissue lesions. Ultrasound allows to assess the hematoma volume, injury degree of muscle and diastasis length between muscle fibers; regenera

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6

Krier, J., and T. Adams. "Properties of Sphincteric Striated Muscle." Physiology 5, no. 6 (1990): 263–67. http://dx.doi.org/10.1152/physiologyonline.1990.5.6.263.

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External anal and urethral sphincters have several common features. Each has striated muscle fibers encircling those of smooth muscle that reflexly contract to guard an orifice or relax to allow evacuation. These functions require special contractile and morphological properties and reflex control not demanded of striated skeletal muscles.

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7

Harfe, B. D., C. S. Branda, M. Krause, M. J. Stern, and A. Fire. "MyoD and the specification of muscle and non-muscle fates during postembryonic development of the C. elegans mesoderm." Development 125, no. 13 (1998): 2479–88. http://dx.doi.org/10.1242/dev.125.13.2479.

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Basic-helix-loop helix factors of the myoD/myf5/ myogenin/MRF4 family have been implicated in acquisition and elaboration of muscle cell fates. Here we describe both myogenic and non-myogenic roles for the Caenorhabditis elegans member of this family (CeMyoD) in postembryonic mesodermal patterning. The postembryonic mesodermal lineage in C. elegans provides a paradigm for many of the issues in mesodermal fate specification: a single mesoblast ('M') divides to generate 14 striated muscles, 16 non-striated muscles, and two non-muscle cells. To study CeMyoD function in the M lineage, we needed to

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8

Blondelle, Jordan, Andrea Biju, and Stephan Lange. "The Role of Cullin-RING Ligases in Striated Muscle Development, Function, and Disease." International Journal of Molecular Sciences 21, no. 21 (2020): 7936. http://dx.doi.org/10.3390/ijms21217936.

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The well-orchestrated turnover of proteins in cross-striated muscles is one of the fundamental processes required for muscle cell function and survival. Dysfunction of the intricate protein degradation machinery is often associated with development of cardiac and skeletal muscle myopathies. Most muscle proteins are degraded by the ubiquitin–proteasome system (UPS). The UPS involves a number of enzymes, including E3-ligases, which tightly control which protein substrates are marked for degradation by the proteasome. Recent data reveal that E3-ligases of the cullin family play more diverse and c

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9

Manders, Emmy, Silvia Rain, Harm-Jan Bogaard, et al. "The striated muscles in pulmonary arterial hypertension: adaptations beyond the right ventricle." European Respiratory Journal 46, no. 3 (2015): 832–42. http://dx.doi.org/10.1183/13993003.02052-2014.

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Pulmonary arterial hypertension (PAH) is a fatal lung disease characterised by progressive remodelling of the small pulmonary vessels. The daily-life activities of patients with PAH are severely limited by exertional fatigue and dyspnoea. Typically, these symptoms have been explained by right heart failure. However, an increasing number of studies reveal that the impact of the PAH reaches further than the pulmonary circulation. Striated muscles other than the right ventricle are affected in PAH, such as the left ventricle, the diaphragm and peripheral skeletal muscles. Alterations in these str

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10

Hood, David A., Atila Balaban, Michael K. Connor, et al. "Mitochondrial Biogenesis in Striated Muscle." Canadian Journal of Applied Physiology 19, no. 1 (1994): 12–48. http://dx.doi.org/10.1139/h94-002.

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Mitochondrial biogenesis (synthesis) has been observed to occur in skeletal muscle in response to chronic use. It also occurs in cardiac muscle during growth and hypertrophy, and it may be impaired during the aging process. This review summarizes the literature on the processes of mitochondrial biogenesis at the biochemical and molecular levels, with particular reference to striated muscles. Mitochondrial biogenesis involves the expression of nuclear and mitochondrial genes and the coordination of these two genomes, the synthesis of proteins and phospholipids and their import into the organell

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