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Journal articles on the topic 'Hemodynamics. Striated muscle Muscles'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Kuo, L., and R. N. Pittman. "Influence of hemoconcentration on arteriolar oxygen transport in hamster striated muscle." American Journal of Physiology-Heart and Circulatory Physiology 259, no. 6 (1990): H1694—H1702. http://dx.doi.org/10.1152/ajpheart.1990.259.6.h1694.

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We investigated the influence of isovolemic hemoconcentration on microcirculatory hemodynamics and oxygen transport in the hamster cheek pouch retractor muscle. In 17 hamsters, measurements of red blood cell velocity, hematocrit, vessel diameter, segment length (L), hemoglobin oxygen saturation (SO2), and longitudinal SO2 gradient (delta SO2/L) were made in four branching orders of arterioles before and after isovolemic exchange with packed red blood cells. Hemoconcentration increased systemic hematocrit from 50 to 65%; systemic blood gases were unchanged, but mean arterial blood pressure incr

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Thorlacius, Henrik, Brigitte Vollmar, Simone Westermann, Leif Torkvist, and Michael D. Menger. "Effects of Local Cooling on Microvascular Hemodynamics and Leukocyte Adhesion in the Striated Muscle of Hamsters." Journal of Trauma: Injury, Infection, and Critical Care 45, no. 4 (1998): 715–19. http://dx.doi.org/10.1097/00005373-199810000-00016.

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3

Kuo, L., and R. N. Pittman. "Effect of hemodilution on oxygen transport in arteriolar networks of hamster striated muscle." American Journal of Physiology-Heart and Circulatory Physiology 254, no. 2 (1988): H331—H339. http://dx.doi.org/10.1152/ajpheart.1988.254.2.h331.

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Experiments were performed on the hamster cheek pouch retractor muscle to investigate the influence of isovolemic hemodilution on microcirculatory hemodynamics and the rate of oxygen transport to striated muscle. In 23 hamsters, measurements of red blood cell velocity, hematocrit, vessel diameter, segment length (L), hemoglobin oxygen saturation (SO2), and longitudinal SO2 gradient (delta SO2/L) were made in four branching orders of arterioles before and after isovolemic exchange with plasma. Hemodilution decreased systemic hematocrit from 52 to 33%. In first- through fourth-order arterioles,

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4

Sulbarán, Guidenn, Lorenzo Alamo, Antonio Pinto, et al. "An invertebrate smooth muscle with striated muscle myosin filaments." Proceedings of the National Academy of Sciences 112, no. 42 (2015): E5660—E5668. http://dx.doi.org/10.1073/pnas.1513439112.

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Muscle tissues are classically divided into two major types, depending on the presence or absence of striations. In striated muscles, the actin filaments are anchored at Z-lines and the myosin and actin filaments are in register, whereas in smooth muscles, the actin filaments are attached to dense bodies and the myosin and actin filaments are out of register. The structure of the filaments in smooth muscles is also different from that in striated muscles. Here we have studied the structure of myosin filaments from the smooth muscles of the human parasite Schistosoma mansoni. We find, surprisin

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5

Mironov, S. P., N. A. Es'kin, A. K. Orletskiy, L. L. Laylin, D. R. Bogdashevskiy, and L. S. Arhzakova. "Ultrasound Diagnosis of Striated Muscles Pathology." N.N. Priorov Journal of Traumatology and Orthopedics 12, no. 1 (2005): 24. http://dx.doi.org/10.17816/vto20050124.

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Ultrasound examination of 897 patients with injuries and pathology of striated muscles was performed. Ultrasound criteria of muscle diseases and injuries were detected. The evaluation of diagnostic efficacy of ultrasound data relative to data of invasive methods (surgical interventions and diagnostic puncture) was performed. Ultrasound examination of striated muscles is showed to be a highly specific and sensitive method for diagnosis of traumatic muscle tissue lesions. Ultrasound allows to assess the hematoma volume, injury degree of muscle and diastasis length between muscle fibers; regenera

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6

Krier, J., and T. Adams. "Properties of Sphincteric Striated Muscle." Physiology 5, no. 6 (1990): 263–67. http://dx.doi.org/10.1152/physiologyonline.1990.5.6.263.

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External anal and urethral sphincters have several common features. Each has striated muscle fibers encircling those of smooth muscle that reflexly contract to guard an orifice or relax to allow evacuation. These functions require special contractile and morphological properties and reflex control not demanded of striated skeletal muscles.

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Harfe, B. D., C. S. Branda, M. Krause, M. J. Stern, and A. Fire. "MyoD and the specification of muscle and non-muscle fates during postembryonic development of the C. elegans mesoderm." Development 125, no. 13 (1998): 2479–88. http://dx.doi.org/10.1242/dev.125.13.2479.

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Basic-helix-loop helix factors of the myoD/myf5/ myogenin/MRF4 family have been implicated in acquisition and elaboration of muscle cell fates. Here we describe both myogenic and non-myogenic roles for the Caenorhabditis elegans member of this family (CeMyoD) in postembryonic mesodermal patterning. The postembryonic mesodermal lineage in C. elegans provides a paradigm for many of the issues in mesodermal fate specification: a single mesoblast ('M') divides to generate 14 striated muscles, 16 non-striated muscles, and two non-muscle cells. To study CeMyoD function in the M lineage, we needed to

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Blondelle, Jordan, Andrea Biju, and Stephan Lange. "The Role of Cullin-RING Ligases in Striated Muscle Development, Function, and Disease." International Journal of Molecular Sciences 21, no. 21 (2020): 7936. http://dx.doi.org/10.3390/ijms21217936.

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The well-orchestrated turnover of proteins in cross-striated muscles is one of the fundamental processes required for muscle cell function and survival. Dysfunction of the intricate protein degradation machinery is often associated with development of cardiac and skeletal muscle myopathies. Most muscle proteins are degraded by the ubiquitin–proteasome system (UPS). The UPS involves a number of enzymes, including E3-ligases, which tightly control which protein substrates are marked for degradation by the proteasome. Recent data reveal that E3-ligases of the cullin family play more diverse and c

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9

Manders, Emmy, Silvia Rain, Harm-Jan Bogaard, et al. "The striated muscles in pulmonary arterial hypertension: adaptations beyond the right ventricle." European Respiratory Journal 46, no. 3 (2015): 832–42. http://dx.doi.org/10.1183/13993003.02052-2014.

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Pulmonary arterial hypertension (PAH) is a fatal lung disease characterised by progressive remodelling of the small pulmonary vessels. The daily-life activities of patients with PAH are severely limited by exertional fatigue and dyspnoea. Typically, these symptoms have been explained by right heart failure. However, an increasing number of studies reveal that the impact of the PAH reaches further than the pulmonary circulation. Striated muscles other than the right ventricle are affected in PAH, such as the left ventricle, the diaphragm and peripheral skeletal muscles. Alterations in these str

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10

Hood, David A., Atila Balaban, Michael K. Connor, et al. "Mitochondrial Biogenesis in Striated Muscle." Canadian Journal of Applied Physiology 19, no. 1 (1994): 12–48. http://dx.doi.org/10.1139/h94-002.

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Mitochondrial biogenesis (synthesis) has been observed to occur in skeletal muscle in response to chronic use. It also occurs in cardiac muscle during growth and hypertrophy, and it may be impaired during the aging process. This review summarizes the literature on the processes of mitochondrial biogenesis at the biochemical and molecular levels, with particular reference to striated muscles. Mitochondrial biogenesis involves the expression of nuclear and mitochondrial genes and the coordination of these two genomes, the synthesis of proteins and phospholipids and their import into the organell

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11

Jakubiec-Puka, A., C. Catani, and U. Carraro. "Myosin heavy-chain composition in striated muscle after tenotomy." Biochemical Journal 282, no. 1 (1992): 237–42. http://dx.doi.org/10.1042/bj2820237.

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The myosin heavy-chain (MHC) isoform pattern was studied by biochemical methods in the slow-twitch (soleus) and fast-twitch (gastrocnemius) muscles of adult rats during atrophy after tenotomy and recovery after tendon regeneration. The tenotomized slow muscle atrophied more than the tenotomized fast muscle. During the 12 days after tenotomy the total MHC content decreased by about 85% in the slow muscle, and only by about 35% in the fast muscle. In the slow muscle the ratio of MHC-1 to MHC-2A(2S) remained almost unchanged, showing that similar diminution of both isoforms occurs. In the fast mu

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Savolainen, J., and M. Vornanen. "Parvalbumin content in striated muscles of the common shrew (Sorex araneus)." Canadian Journal of Zoology 76, no. 12 (1998): 2194–99. http://dx.doi.org/10.1139/z98-160.

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The parvalbumin content of mammalian muscles correlates positively with isometric relaxation rate and fiber type IIB frequency of the muscles but negatively with animal size. Since shrews are small-bodied animals with a relatively low number of type IIB fibers, it is of some interest to know how the parvalbumin content of shrew muscle correlates with the above factors. Parvalbumin content in heart, diaphragm, and gastrocnemius muscle of the common shrew, mouse, and rat was determined electrophoretically. Parvalbumin was not found in heart muscle of any species. Shrew diaphragm (0.29 ± 0.04 g/k

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13

Khurana, T. S., R. A. Prendergast, H. S. Alameddine, et al. "Absence of extraocular muscle pathology in Duchenne's muscular dystrophy: role for calcium homeostasis in extraocular muscle sparing." Journal of Experimental Medicine 182, no. 2 (1995): 467–75. http://dx.doi.org/10.1084/jem.182.2.467.

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Duchenne muscular dystrophy (DMD) is characterized by clinical weakness and progressive necrosis of striated muscle as a consequence of dystrophin deficiency. While all skeletal muscle groups are thought to be affected, enigmatically, the extraocular muscles (EOM) appear clinically unaffected. Here we show that dystrophin deficiency does not result in myonecrosis or pathologically elevated levels of intracellular calcium ([Ca2+]i) in EOM. At variance with a previous report, we find no evidence for dystrophin-related protein/utrophin up-regulation in EOM. In vitro experiments demonstrate that e

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14

Board, Editorial. "Session 18 / ІѴ". Kazan medical journal 20, № 3 (2021): 556–57. http://dx.doi.org/10.17816/kazmj76631.

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15

Johnston, I. A. "Muscle action during locomotion: a comparative perspective." Journal of Experimental Biology 160, no. 1 (1991): 167–85. http://dx.doi.org/10.1242/jeb.160.1.167.

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This essay explores how the properties of striated muscles are matched to the tasks they perform during running, swimming and flying. During exercise the major locomotory muscles undergo alternate cycles of lengthening and shortening. Force development is greatly influenced by the timing of stimulation in relation to the length-change cycle and by the nature of elastic structures connecting the muscle fibres to the skeleton. The storage and recovery of elastic strain energy by the tendons (apodema in insects) results in a considerable saving of metabolic energy. Strain is independent of locomo

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Cruz, Yolanda, and John W. Downie. "Sexually dimorphic micturition in rats: relationship of perineal muscle activity to voiding pattern." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 289, no. 5 (2005): R1307—R1318. http://dx.doi.org/10.1152/ajpregu.00088.2005.

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In the present study we examined the possibility that striated muscle activity may underlie sexually dimorphic micturition in rats. Micturition dynamics, the gross anatomy of the external urethral sphincter, and the participation of the striated perineal muscles in micturition were compared in urethane-anesthetized adult male and female rats. Bladder contraction characteristics, particularly the magnitude of bladder high-frequency pressure waves during voiding, differed between sexes. Dissections indicated that the sphincter was more extensive and thicker in males than in females. Electromyogr

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17

Gayan-Ramirez, Ghislaine, and Marc Decramer. "Mechanisms of striated muscle dysfunction during acute exacerbations of COPD." Journal of Applied Physiology 114, no. 9 (2013): 1291–99. http://dx.doi.org/10.1152/japplphysiol.00847.2012.

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During acute exacerbations of chronic obstructive pulmonary disease (COPD), limb and respiratory muscle dysfunction develops rapidly and functional recovery is partial and slow. The mechanisms leading to this muscle dysfunction are not yet fully established. However, recent evidence has shown that several pathways involved in muscle catabolism, apoptosis, and oxidative stress are activated in the vastus lateralis muscle of patients during acute exacerbations of COPD, while those implicated in mitochondrial function are downregulated. These pathways may be targeted in different ways by factors

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18

Shiina, Takahiko, Takeshi Shima, Kazuaki Masuda, et al. "Contractile Properties of Esophageal Striated Muscle: Comparison with Cardiac and Skeletal Muscles in Rats." Journal of Biomedicine and Biotechnology 2010 (2010): 1–7. http://dx.doi.org/10.1155/2010/459789.

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The external muscle layer of the mammalian esophagus consists of striated muscles. We investigated the contractile properties of esophageal striated muscle by comparison with those of skeletal and cardiac muscles. Electrical field stimulation with single pulses evoked twitch-like contractile responses in esophageal muscle, similar to those in skeletal muscle in duration and similar to those in cardiac muscle in amplitude. The contractions of esophageal muscle were not affected by an inhibitor of gap junctions. Contractile responses induced by high potassium or caffeine in esophageal muscle wer

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19

Wang, Seu-Mei, Seu-Hwa Wang, Jenny Li-Chun Lin, and Jim Jung-Ching Lin. "Striated muscle tropomyosin-enriched microfilaments of developing muscles of chicken embryos." Journal of Muscle Research and Cell Motility 11, no. 3 (1990): 191–202. http://dx.doi.org/10.1007/bf01843573.

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BRELLOU (Γ.Δ. ΜΠΡΕΛΛΟΥ), G. D., V. PSYCHAS (Β. ΨΥΧΑΣ), and I. VLEMMAS (Ι. ΒΛΕΜΜΑΣ). "Rhabdomyosarcomas arising from striated muscles in elderly dogs: pathological features of 4 cases." Journal of the Hellenic Veterinary Medical Society 64, no. 2 (2017): 105. http://dx.doi.org/10.12681/jhvms.15483.

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Primary rhabdomyosarcomas are rare in dogs. Based on their classification, embryonal rhabdomyosarcoma is the most common, while alveolar and especially pleomorphic types occur less often. Four cases diagnosed as primary canine rhabdomyosarcomas of striated muscles were retrieved from our files. All the animals were cross-breeds, aged over 8 years. Two of them had died after developing disseminated intravascular coagulation and gastric ulcer, respectively, and two others were euthanized. Of those two, one had been admitted with neurological and cardiovascular symptoms and one with disseminated

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Shield, M. A., H. S. Haugen, C. H. Clegg, and S. D. Hauschka. "E-box sites and a proximal regulatory region of the muscle creatine kinase gene differentially regulate expression in diverse skeletal muscles and cardiac muscle of transgenic mice." Molecular and Cellular Biology 16, no. 9 (1996): 5058–68. http://dx.doi.org/10.1128/mcb.16.9.5058.

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Previous analysis of the muscle creatine kinase (MCK) gene indicated that control elements required for transcription in adult mouse muscle differed from those required in cell culture, suggesting that distinct modes of muscle gene regulation occur in vivo. To examine this further, we measured the activity of MCK transgenes containing E-box and promoter deletions in a variety of striated muscles. Simultaneous mutation of three E boxes in the 1,256-bp MCK 5' region, which abolished transcription in muscle cultures, had strikingly different effects in mice. The mutations abolished transgene expr

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Grove, B. K., L. Cerny, J. C. Perriard, H. M. Eppenberger, and L. E. Thornell. "Fiber type-specific distribution of M-band proteins in chicken muscle." Journal of Histochemistry & Cytochemistry 37, no. 4 (1989): 447–54. http://dx.doi.org/10.1177/37.4.2926123.

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The functions of two myofibrillar proteins, myomesin (Mr 185,000) and M-protein (Mr 165,000), associated with the M-band are as yet unknown. To extend our knowledge of these proteins, we have examined chicken striated muscles with fast and slow contractile properties, e.g., pectoralis major, PLD, ALD, medial adductor, and lateral adductor, to determine the expression and isoform composition of myomesin and M-protein in various muscles and fiber types. The high molecular weight M-band proteins were characterized and quantitated using monoclonal antibodies in immunoblotting and double-antibody s

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Butz, C. Eric, Grant B. McClelland, and George A. Brooks. "MCT1 confirmed in rat striated muscle mitochondria." Journal of Applied Physiology 97, no. 3 (2004): 1059–66. http://dx.doi.org/10.1152/japplphysiol.00009.2004.

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We sought to test the hypothesis that monocarboxylate transporter isoform 1 (MCT1) is the inner mitochondrial membrane lactate/pyruvate transporter, and, as such, contributes to functioning of the intracellular lactate shuttle. However, presence of a mammalian mitochondrially localized MCT1 (mMCT1) has been contested. We sought to confirm by Western blotting the mitochondrial localization of MCT1 in rat cardiac, soleus, and extensor digitorum longus muscles utilizing three different cell fractionation methods and three different antibodies. We performed Western blotting using antibodies to cel

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Sulbarán, Guidenn, Lorenzo Alamo, Antonio Pinto, et al. "Schistosome Muscles Contain Striated Muscle-Like Myosin Filaments in a Smooth Muscle-Like Architecture." Biophysical Journal 106, no. 2 (2014): 159a. http://dx.doi.org/10.1016/j.bpj.2013.11.911.

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Freyssenet, Damien, Martino DiCarlo, Patricia Escobar, Janice Grey, Jeremy Schneider, and David A. Hood. "Zidovudine (AZT) induced alterations in mitochondrial biogenesis in rat striated muscles." Canadian Journal of Physiology and Pharmacology 77, no. 1 (1999): 29–35. http://dx.doi.org/10.1139/y98-151.

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Zidovudine (AZT) and didanosine (ddI), two drugs used in the treatment of AIDS, are also known to cause mitochondrial abnormalities. We investigated the physiological relevance of the mitochondrial defects by measuring in situ skeletal muscle performance and cytochrome c oxidase (CYTOX) enzyme activity in heart muscle, red high-oxidative (RG) and white low-oxidative (WG) portions of the gastrocnemius muscle of control (n = 17), AZT- (n = 14), or ddI-treated (n = 11) rats for 28 days. We also evaluated the hypothesis that AZT treatment could alter the expression of the mitochondrial transcripti

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Takano-Ohmuro, H., T. Obinata, M. Kawashima, T. Masaki, and T. Tanaka. "Embryonic chicken skeletal, cardiac, and smooth muscles express a common embryo-specific myosin light chain." Journal of Cell Biology 100, no. 6 (1985): 2025–30. http://dx.doi.org/10.1083/jcb.100.6.2025.

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It has been demonstrated that embryonic chicken gizzard smooth muscle contains a unique embryonic myosin light chain of 23,000 mol wt, called L23 (Katoh, N., and S. Kubo, 1978, Biochem. Biophys. Acta, 535:401-411; Takano-Ohmuro, H., T. Obinata, T. Mikawa, and T. Masaki, 1983, J. Biochem. (Tokyo), 93:903-908). When we examined myosins in developing chicken ventricular and pectoralis muscles by two-dimensional gel electrophoresis, the myosin light chain (Le) that completely comigrates with L23 was detected in both striated muscles at early developmental stages. Two monoclonal antibodies, MT-53f

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Tovbina, M. G., V. G. Pishchik, S. V. Lapin, and S. M. Nuraliev. "Antibodies to striated muscle and acetylcholine receptors in evaluation of surgical treatment outcomes in patients with autoimmune myasthenia gravis." Grekov's Bulletin of Surgery 176, no. 3 (2017): 21–27. http://dx.doi.org/10.24884/0042-4625-2017-176-3-21-27.

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OBJECTIVE. The authors evaluated the role of antibodies to striated muscle and acetylcholine receptors in diagnostics of myasthenia gravis and thymoma, as well as outcomes of thymectomy and prognosis of myasthenia course. MATERIAL AND METHODS. The study investigated correlations of antibody content to striated muscles and acetylcholine receptors from the presence and size of thymoma, myasthenia in 157 patients with various pathologies of the thymus. The dynamics of antibody concentrations was followed up after thymectomy. RESULTS. Antibody titer to striated muscle depended on the presence and

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Testa, Marco, Bianca Rocca, Lucia Spath, et al. "Expression and activity of cyclooxygenase isoforms in skeletal muscles and myocardium of humans and rodents." Journal of Applied Physiology 103, no. 4 (2007): 1412–18. http://dx.doi.org/10.1152/japplphysiol.00288.2007.

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Conflicting data have been reported on cyclooxygenase (COX)-1 and COX-2 expression and activity in striated muscles, including skeletal muscles and myocardium, in particular it is still unclear whether muscle cells are able to produce prostaglandins (PGs). We characterized the expression and enzymatic activity of COX-1 and COX-2 in the skeletal muscles and in the myocardium of mice, rats and humans. By RT-PCR, COX-1 and COX-2 mRNAs were observed in homogenates of mouse and rat hearts, and in different types of skeletal muscles from all different species. By Western blotting, COX-1 and -2 prote

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Nicolas, Hannah A., Marie-Andrée Akimenko, and Frédérique Tesson. "Cellular and Animal Models of Striated Muscle Laminopathies." Cells 8, no. 4 (2019): 291. http://dx.doi.org/10.3390/cells8040291.

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The lamin A/C (LMNA) gene codes for nuclear intermediate filaments constitutive of the nuclear lamina. LMNA has 12 exons and alternative splicing of exon 10 results in two major isoforms—lamins A and C. Mutations found throughout the LMNA gene cause a group of diseases collectively known as laminopathies, of which the type, diversity, penetrance and severity of phenotypes can vary from one individual to the other, even between individuals carrying the same mutation. The majority of the laminopathies affect cardiac and/or skeletal muscles. The underlying molecular mechanisms contributing to suc

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Hidaka, O., M. Yanagi, and K. Takada. "Mental Stress-induced Physiological Changes in the Human Masseter Muscle." Journal of Dental Research 83, no. 3 (2004): 227–31. http://dx.doi.org/10.1177/154405910408300308.

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The effect of a long mental stress on the hemodynamics of masticatory muscles has not been investigated to date. We hypothesized some hemodynamic and electromyographic changes in jaw-closure muscles related to sympathetic nervous system activity. While healthy adult female volunteers performed a two-hour mental stress task, electromyographic activity of the temporal and masseteric muscles was recorded, and hemodynamic changes of the masseter muscle were measured non-invasively. Autonomic function was assessed by heart rate spectral analysis. Integrated electromyographic activity of the tempora

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Lutz, Anne-Kathrin, Stefanie Pfaender, Berra Incearap, et al. "Autism-associated SHANK3 mutations impair maturation of neuromuscular junctions and striated muscles." Science Translational Medicine 12, no. 547 (2020): eaaz3267. http://dx.doi.org/10.1126/scitranslmed.aaz3267.

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Heterozygous mutations of the gene encoding the postsynaptic protein SHANK3 are associated with syndromic forms of autism spectrum disorders (ASDs). One of the earliest clinical symptoms in SHANK3-associated ASD is neonatal skeletal muscle hypotonia. This symptom can be critical for the early diagnosis of affected children; however, the mechanism mediating hypotonia in ASD is not completely understood. Here, we used a combination of patient-derived human induced pluripotent stem cells (hiPSCs), Shank3Δ11(−/−) mice, and Phelan-McDermid syndrome (PMDS) muscle biopsies from patients of different

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Ackermann, Maegen A., and Aikaterini Kontrogianni-Konstantopoulos. "Myosin Binding Protein-C: A Regulator of Actomyosin Interaction in Striated Muscle." Journal of Biomedicine and Biotechnology 2011 (2011): 1–9. http://dx.doi.org/10.1155/2011/636403.

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Myosin-Binding protein-C (MyBP-C) is a family of accessory proteins of striated muscles that contributes to the assembly and stabilization of thick filaments, and regulates the formation of actomyosin cross-bridges, via direct interactions with both thick myosin and thin actin filaments. Three distinct MyBP-C isoforms have been characterized; cardiac, slow skeletal, and fast skeletal. Numerous mutations in the gene for cardiac MyBP-C (cMyBP-C) have been associated with familial hypertrophic cardiomyopathy (FHC) and have led to increased interest in the regulation and roles of the cardiac isofo

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Sweeney, H. L., B. F. Bowman, and J. T. Stull. "Myosin light chain phosphorylation in vertebrate striated muscle: regulation and function." American Journal of Physiology-Cell Physiology 264, no. 5 (1993): C1085—C1095. http://dx.doi.org/10.1152/ajpcell.1993.264.5.c1085.

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The regulatory light chain of myosin (RLC) is phosphorylated in striated muscles by Ca2+/calmodulin-dependent myosin light chain kinase. Unique biochemical and cellular properties of this phosphorylation system in fast-twitch skeletal muscle maintain RLC in the phosphorylated form for a prolonged period after a brief tetanus or during low-frequency repetitive stimulation. This phosphorylation correlates with potentiation of the rate of development and maximal extent of isometric twitch tension. In skinned fibers, RLC phosphorylation increases force production at low levels of Ca2+ activation,

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Collins, Teresa, Josephine E. Joya, Ruth M. Arkell, Vicki Ferguson та Edna C. Hardeman. "Reappearance of the minor α-sarcomeric actins in postnatal muscle". American Journal of Physiology-Cell Physiology 273, № 6 (1997): C1801—C1810. http://dx.doi.org/10.1152/ajpcell.1997.273.6.c1801.

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The postnatal expression profiles of α-sarcomeric actin transcripts and protein are quantified in mouse striated muscles from birth to postnatal day 56 by Northern and Western blot analyses. α-Cardiac actin (α-CA) transcripts transiently increase between 12 and 21 days after birth in the quadriceps muscle, reaching ∼90% that found in the adult mouse heart. Although α-CA is the α-sarcomeric actin isoform expressed in the immature fiber, the expression profiles of other contractile protein isoforms indicate that this postnatal period is not reflective of an immature phenotype. α-Skeletal actin (

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Bennett, R. A., R. N. Pittman, and S. M. Sullivan. "Capillary spatial pattern and muscle fiber geometry in three hamster striated muscles." American Journal of Physiology-Heart and Circulatory Physiology 260, no. 2 (1991): H579—H585. http://dx.doi.org/10.1152/ajpheart.1991.260.2.h579.

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Since most oxygen exchange in muscle is thought to occur by diffusion across the walls of capillaries, it is important to determine the spatial relationship between capillaries and muscle fibers. We have extended the work of Kayar et al. (Microvasc. Res 24: 326-341, 1982) to include other statistical tests that allow one to make stronger statements regarding the spatial pattern. Data for hamsters were obtained from two sartorius, three retractor, and five soleus muscle sections. Distances between all pairs of capillaries, distances between a capillary and its first nearest neighbor for all cap

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Vainshtein, Anna, Lawrence Kazak, and David A. Hood. "Effects of endurance training on apoptotic susceptibility in striated muscle." Journal of Applied Physiology 110, no. 6 (2011): 1638–45. http://dx.doi.org/10.1152/japplphysiol.00020.2011.

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An increase in the production of reactive oxygen species occurs with muscle disuse, ischemic cardiomyopathy, and conditions that arise with senescence. The resulting oxidative stress is associated with apoptosis-related myopathies. Recent research has suggested that chronic exercise is protective against mitochondrially mediated programmed cell death. To further investigate this, we compared soleus (Sol) and cardiac muscles of voluntary wheel-trained (T; 10 wk) and untrained (C) animals. Training produced a 52% increase in muscle cytochrome c oxidase (COX) activity. Sol and left ventricle (LV)

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Price, M. G. "Skelemins: cytoskeletal proteins located at the periphery of M-discs in mammalian striated muscle." Journal of Cell Biology 104, no. 5 (1987): 1325–36. http://dx.doi.org/10.1083/jcb.104.5.1325.

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The cytoskeletons of mammalian striated and smooth muscles contain a pair of high molecular weight (HMW) polypeptides of 220,000 and 200,000 mol wt, each with isoelectric points of about 5 (Price, M. G., 1984, Am. J. Physiol., 246:H566-572) in a molar ratio of 1:1:20 with desmin. The HMW polypeptides of mammalian muscle have been named "skelemins," because they are in the insoluble cytoskeletons of striated muscle and are at the M-discs. I have used two-dimensional peptide mapping to show that the two skelemin polypeptides are closely related to each another. Polyclonal antibodies directed aga

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Fleming, Jennifer R., Alankrita Rani, Jamie Kraft, Sanja Zenker, Emma Börgeson, and Stephan Lange. "Exploring Obscurin and SPEG Kinase Biology." Journal of Clinical Medicine 10, no. 5 (2021): 984. http://dx.doi.org/10.3390/jcm10050984.

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Three members of the obscurin protein family that contain tandem kinase domains with important signaling functions for cardiac and striated muscles are the giant protein obscurin, its obscurin-associated kinase splice isoform, and the striated muscle enriched protein kinase (SPEG). While there is increasing evidence for the specific roles that each individual kinase domain plays in cross-striated muscles, their biology and regulation remains enigmatic. Our present study focuses on kinase domain 1 and the adjacent low sequence complexity inter-kinase domain linker in obscurin and SPEG. Using Ph

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Andrews, M. A., R. E. Godt, and T. M. Nosek. "Influence of physiological L(+)-lactate concentrations on contractility of skinned striated muscle fibers of rabbit." Journal of Applied Physiology 80, no. 6 (1996): 2060–65. http://dx.doi.org/10.1152/jappl.1996.80.6.2060.

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These experiments investigated the effects of physiological concentrations of L(+)-lactate on the contractility of chemically skinned rabbit fast-twitch psoas, slow-twitch soleus, and cardiac muscles at pH 7.L(+)-Lactate depressed maximal calcium-activated force (Fmax) of all muscles studied within the range of 5-20 (slow-twitch muscle) or 5-25 mM (fast-twitch and cardiac muscles). Fmax of fast-twitch fibers was inhibited to the greatest degree (9% in K2 creatine phosphate solutions). In all of these muscle types, Fmax returned to control levels as L(+)-lactate was increased to 30-50 mM. Subst

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Marini, Gabriela, Angélica M. Pascon Barbosa, Débora C. Damasceno, et al. "Morphological changes in the fast vs slow fiber profiles of the urethras of diabetic pregnant rats." Urogynaecologia 25, no. 1 (2011): 9. http://dx.doi.org/10.4081/uij.2011.e9.

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<em>Background</em>. This study was undertaken to test the hypothesis that diabetes and pregnancy detrimentally affect the normal function of urethral striated muscles in rats, providing a model for additional studies related to urinary incontinence. The aim of this study was to evaluate morphological alterations in the urethral striated muscles of diabetic pregnant rats. <em>Design and methods. </em>Twenty female Wistar rats were distributed into four experimental groups of five rats as follows: virgin, pregnant, diabetic virgin, and diabetic pregnant. Diabetes was ind

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Elhanany-Tamir, Hadas, Yanxun V. Yu, Miri Shnayder, Ankit Jain, Michael Welte, and Talila Volk. "Organelle positioning in muscles requires cooperation between two KASH proteins and microtubules." Journal of Cell Biology 198, no. 5 (2012): 833–46. http://dx.doi.org/10.1083/jcb.201204102.

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Striated muscle fibers are characterized by their tightly organized cytoplasm. Here, we show that the Drosophila melanogaster KASH proteins Klarsicht (Klar) and MSP-300 cooperate in promoting even myonuclear spacing by mediating a tight link between a newly discovered MSP-300 nuclear ring and a polarized network of astral microtubules (aMTs). In either klar or msp-300ΔKASH, or in klar and msp-300 double heterozygous mutants, the MSP-300 nuclear ring and the aMTs retracted from the nuclear envelope, abrogating this even nuclear spacing. Anchoring of the myonuclei to the core acto-myosin fibrill

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42

D'souza, Donna, Ruanne Y. J. Lai, Michael Shuen, and David A. Hood. "mRNA stability as a function of striated muscle oxidative capacity." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 303, no. 4 (2012): R408—R417. http://dx.doi.org/10.1152/ajpregu.00085.2012.

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A change in mRNA stability alters the abundance of mRNA available for translation and is emerging as a critical pathway influencing gene expression. Variations in the stability of functional and regulatory mitochondrial proteins may contribute to the divergent mitochondrial densities observed in striated muscle. Thus we hypothesized that the stability of mRNAs encoding for regulatory nuclear and mitochondrial transcription factors would be inversely proportional to muscle oxidative capacity and would be facilitated by the activity of RNA binding proteins (RBPs). The stability of mitochondrial

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Rassier, Dilson E. "Sarcomere mechanics in striated muscles: from molecules to sarcomeres to cells." American Journal of Physiology-Cell Physiology 313, no. 2 (2017): C134—C145. http://dx.doi.org/10.1152/ajpcell.00050.2017.

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Muscle contraction is commonly associated with the cross-bridge and sliding filament theories, which have received strong support from experiments conducted over the years in different laboratories. However, there are studies that cannot be readily explained by the theories, showing 1) a plateau of the force-length relation extended beyond optimal filament overlap, and forces produced at long sarcomere lengths that are higher than those predicted by the sliding filament theory; 2) passive forces at long sarcomere lengths that can be modulated by activation and Ca2+, which changes the force-len

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Franzini-Armstrong, C., and F. Protasi. "Ryanodine receptors of striated muscles: a complex channel capable of multiple interactions." Physiological Reviews 77, no. 3 (1997): 699–729. http://dx.doi.org/10.1152/physrev.1997.77.3.699.

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The ryanodine receptor (RyR) is a high-conductance Ca2+ channel of the sarcoplasmic reticulum in muscle and of the endoplasmic reticulum in other cells. In striated muscle fibers, RyRs are responsible for the rapid release of Ca2+ that activates contraction. Ryanodine receptors are complex molecules, with unusually large cytoplasmic domains containing numerous binding sites for agents that control the state of activity of the channel-forming domain of the molecule. Structural considerations indicate that long-range interactions between cytoplasmic and intramembrane domains control channel func

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Wray, S. "Smooth muscle intracellular pH: measurement, regulation, and function." American Journal of Physiology-Cell Physiology 254, no. 2 (1988): C213—C225. http://dx.doi.org/10.1152/ajpcell.1988.254.2.c213.

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Smooth muscle performs many functions that are essential for the normal working of the human body. Changes in pH are thought to affect many aspects of smooth muscle. Despite this, until recently little was known about either intracellular pH (pHi) values or pHi regulation in smooth muscle. Recent work measuring pHi with either microelectrodes or nuclear magnetic resonance spectroscopy is now providing some of this much needed information for smooth muscles. From these studies, it can be concluded tentatively that pHi is the same in different smooth muscles, approximately 7.06 (37 degrees C). T

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Bosso, F. J., S. A. Lang, and M. B. Maron. "Role of hemodynamics and vagus nerves in development of fibrin-induced pulmonary edema." Journal of Applied Physiology 69, no. 6 (1990): 2227–32. http://dx.doi.org/10.1152/jappl.1990.69.6.2227.

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The interosseous external intercostal (EI) muscles of the upper rib cage are electrically active during inspiration, but the mechanical consequence of their activation is unclear. In 16 anesthetized dogs, we simultaneously measured EI (3rd and 4th interspaces) and parasternal intercostal (PA) (3rd interspace) electromyogram and length. Muscle length was measured by sonomicrometry and expressed as a percentage of resting length (%LR). During resting breathing, each muscle was electrically active and shortened to a similar extent. Sequential EI muscle denervation (3rd and 4th interspaces) follow

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Ordway, G. A., K. Li, G. A. Hand, and R. S. Williams. "RNA subunit of mitochondrial RNA-processing enzyme is induced by contractile activity in striated muscle." American Journal of Physiology-Cell Physiology 265, no. 6 (1993): C1511—C1516. http://dx.doi.org/10.1152/ajpcell.1993.265.6.c1511.

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A small RNA encoded within the nucleus of yeast and mammalian cells is an essential subunit of a mitochondrial RNA-processing endonuclease (RNase MRP) that generates primers for mitochondrial DNA (mtDNA) replication. We examined expression of MRP-RNA in specialized subtypes of mammalian striated muscles that differ markedly in respiratory activity and in muscles subjected to chronic stimulation via the motor nerve, a potent stimulus to mitochondrial biogenesis. MRP-RNA was more abundant in mitochondria-rich cardiac and slow-twitch skeletal muscles than in glycolytic fast-twitch skeletal muscle

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Mi-Mi, Lei, SarahBeth Votra, Kenneth Kemphues, Anthony Bretscher, and David Pruyne. "Z-line formins promote contractile lattice growth and maintenance in striated muscles of C. elegans." Journal of Cell Biology 198, no. 1 (2012): 87–102. http://dx.doi.org/10.1083/jcb.201202053.

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Muscle contraction depends on interactions between actin and myosin filaments organized into sarcomeres, but the mechanism by which actin filaments incorporate into sarcomeres remains unclear. We have found that, during larval development in Caenorhabditis elegans, two members of the actin-assembling formin family, CYK-1 and FHOD-1, are present in striated body wall muscles near or on sarcomere Z lines, where barbed ends of actin filaments are anchored. Depletion of either formin during this period stunted growth of the striated contractile lattice, whereas their simultaneous reduction profoun

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Hidaka, O., M. Yanagi, and K. Takada. "Changes in Masseteric Hemodynamics Time-related to Mental Stress." Journal of Dental Research 83, no. 2 (2004): 185–90. http://dx.doi.org/10.1177/154405910408300220.

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Mental stress may cause a dissociation of sympathetic outflow to different regions. However, it remains unclear how the sympathetic outflow to jaw muscles is related to other sympathetic outflow under mental stress. The objective of this study was to clarify the temporal relationship between the finger sweat expulsion elicited by mental stress and the hemodynamic and electromyographic changes in the masseter muscle. Healthy adult female volunteers participated in this study. Masseteric hemodynamic changes were closely time-related to mental stress, showing a decrease in oxygen saturation of mu

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Schachat, Fred, and Margaret M. Briggs. "Phylogenetic implications of the superfast myosin in extraocular muscles." Journal of Experimental Biology 205, no. 15 (2002): 2189–201. http://dx.doi.org/10.1242/jeb.205.15.2189.

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SUMMARY Extraocular muscle exhibits higher-velocity and lower-tension contractions than other vertebrate striated muscles. These distinctive physiological properties are associated with the expression of a novel extraocular myosin heavy chain (MYH). Encoded by the MYH13 gene, the extraocular myosin heavy chain is a member of the fast/developmental MYH gene cluster on human chromosome 17 and the syntenic MYH cluster on mouse chromosome 11. Comparison of cDNA sequences reveals that MYH13 also encodes the atypical MYH identified in laryngeal muscles, which have similar fast contractile properties

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