Relevant bibliographies by topics / Hemolytic Disease

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Hemolytic Disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Hemolytic Disease"

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Dorrah, Moataza, Chaima Bensaoud, Amr A. Mohamed, Daniel Sojka, Taha T. M. Bassal, and Michail Kotsyfakis. "Comparison of the hemolysis machinery in two evolutionarily distant blood-feeding arthropod vectors of human diseases." PLOS Neglected Tropical Diseases 15, no. 2 (2021): e0009151. http://dx.doi.org/10.1371/journal.pntd.0009151.

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Host blood protein digestion plays a pivotal role in the ontogeny and reproduction of hematophagous vectors. The gut of hematophagous arthropods stores and slowly digests host blood and represents the primary gateway for transmitted pathogens. The initial step in blood degradation is induced lysis of host red blood cells (hemolysis), which releases hemoglobin for subsequent processing by digestive proteolytic enzymes. The activity cycles and characteristics of hemolysis in vectors are poorly understood. Hence, we investigated hemolysis in two evolutionarily distant blood-feeding arthropods: Th
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Udou, Takezo. "Extracellular hemolytic activity in rapidly growing mycobacteria." Canadian Journal of Microbiology 40, no. 4 (1994): 318–21. http://dx.doi.org/10.1139/m94-052.

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Little is known about virulence factors associated with rapidly growing mycobacteria. We evaluated 42 clinical isolates of Mycobacterium fortuitum and Mycobacterium chelonae and 4 reference strains of Mycobacterium smegmatis for the production of hemolysin (or hemolytic substance) as a possible contributor to the pathogenesis of disease caused by these organisms. All the strains tested possessed extracellular hemolytic activity that was stable after heating and proteinase treatment, and the active substance had a molecular weight less than 10 000. The activity accumulated in culture medium dur
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Gallagher, Patrick G. "Diagnosis and management of rare congenital nonimmune hemolytic disease." Hematology 2015, no. 1 (2015): 392–99. http://dx.doi.org/10.1182/asheducation-2015.1.392.

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AbstractRare, congenital nonimmune hemolytic disorders of the erythrocyte, although uncommon, are important causes of anemia in the child and adult. These are a heterogeneous group of diseases that disrupt normal erythrocyte structure and function in varying ways. Predominant are abnormalities of hemoglobin stability, defects of erythrocyte metabolism, and disorders of erythrocyte hydration. Unstable hemoglobinopathies may lead to chronic or episodic hemolysis. Perturbation of critical enzymes of the Embden–Meyerhof pathway lead to altered erythrocyte metabolism and chronic hemolysis. Disorder
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Merle, Nicolas S., Romain Paule, Juliette Leon, et al. "P-selectin drives complement attack on endothelium during intravascular hemolysis in TLR-4/heme-dependent manner." Proceedings of the National Academy of Sciences 116, no. 13 (2019): 6280–85. http://dx.doi.org/10.1073/pnas.1814797116.

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Hemolytic diseases are frequently linked to multiorgan failure subsequent to vascular damage. Deciphering the mechanisms leading to organ injury upon hemolytic event could bring out therapeutic approaches. Complement system activation occurs in hemolytic disorders, such as sickle cell disease, but the pathological relevance and the acquisition of a complement-activating phenotype during hemolysis remain unclear. Here we found that intravascular hemolysis, induced by injection of phenylhydrazine, resulted in increased alanine aminotransferase plasma levels and NGAL expression. This liver damage
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Brodsky, Robert A. "Complement in hemolytic anemia." Hematology 2015, no. 1 (2015): 385–91. http://dx.doi.org/10.1182/asheducation.v2015.1.385.3917876.

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Complement is increasingly being recognized as an important driver of human disease, including many hemolytic anemias. Paroxysmal nocturnal hemoglobinuria (PNH) cells are susceptible to hemolysis because of a loss of the complement regulatory proteins CD59 and CD55. Patients with atypical hemolytic uremic syndrome (aHUS) develop a thrombotic microangiopathy (TMA) that in most cases is attributable to mutations that lead to activation of the alternative pathway of complement. For optimal therapy, it is critical, but often difficult, to distinguish aHUS from other TMAs, such as thrombotic thromb
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Louachama, Ouidad, Aicha Bourrahouat, Ibtissam Khattou, Imane Ait Sab, and Mohamed Sbihi. "Recurrent Hemolytic Anemia as an Inaugural Manifestation of Wilson Disease in Children: A Case Report." Open Pediatric Medicine Journal 9, no. 1 (2019): 5–6. http://dx.doi.org/10.2174/1874309901909010005.

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Wilson disease (WD) is a disorder of copper metabolism. Liver and brain disorders are the main presentations, hemolytic anemia in WD is a rare inaugural symptom. We report a case of a child who developed recurrent hemolytic anemia associated with liver failure in the second hemolysis episode as the first manifestation of WD. Wilson's disease is not exceptional in children with hemolytic anemia, but another differential diagnosis must be excluded.
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Brodsky, Robert A. "Complement in hemolytic anemia." Blood 126, no. 22 (2015): 2459–65. http://dx.doi.org/10.1182/blood-2015-06-640995.

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Abstract Complement is increasingly being recognized as an important driver of human disease, including many hemolytic anemias. Paroxysmal nocturnal hemoglobinuria (PNH) cells are susceptible to hemolysis because of a loss of the complement regulatory proteins CD59 and CD55. Patients with atypical hemolytic uremic syndrome (aHUS) develop a thrombotic microangiopathy (TMA) that in most cases is attributable to mutations that lead to activation of the alternative pathway of complement. For optimal therapy, it is critical, but often difficult, to distinguish aHUS from other TMAs, such as thrombot
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Brodsky, Robert A. "Complement in hemolytic anemia." Hematology 2015, no. 1 (2015): 385–91. http://dx.doi.org/10.1182/asheducation-2015.1.385.

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Abstract Complement is increasingly being recognized as an important driver of human disease, including many hemolytic anemias. Paroxysmal nocturnal hemoglobinuria (PNH) cells are susceptible to hemolysis because of a loss of the complement regulatory proteins CD59 and CD55. Patients with atypical hemolytic uremic syndrome (aHUS) develop a thrombotic microangiopathy (TMA) that in most cases is attributable to mutations that lead to activation of the alternative pathway of complement. For optimal therapy, it is critical, but often difficult, to distinguish aHUS from other TMAs, such as thrombot
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Nouraie, Mehdi, Caterina Minniti, Craig Sable, et al. "Association of Hemolysis with Clinical Manifestations of Sickle Cell Disease." Blood 112, no. 11 (2008): 2482. http://dx.doi.org/10.1182/blood.v112.11.2482.2482.

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Abstract Background: Sickle cell disease shares common complications such as vasculopathy and organ dysfunction involving the heart, the lungs, the liver and the kidneys with other hemolytic conditions. We hypothesized that a hemolytic vasculopathy may underlie some of these complications. Distinguishing whether a complication is due to hemolysis or to the degree of anemia has been a challenge. Methods: A prospective, multicenter study of 310 children and adolescents with sickle cell disease in steady state was conducted. The associations of measures of hemolysis and of hemoglobin concentratio
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Hsu, Tsungda, David L. Hutto, F. Chris Minion, Richard L. Zuerner, and Michael J. Wannemuehler. "Cloning of a Beta-Hemolysin Gene ofBrachyspira (Serpulina) hyodysenteriaeand Its Expression in Escherichia coli." Infection and Immunity 69, no. 2 (2001): 706–11. http://dx.doi.org/10.1128/iai.69.2.706-711.2001.

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ABSTRACT Brachyspira (Serpulina)hyodysenteriae induces a mucohemorrhagic diarrheal disease in pigs. The production of a beta-hemolysin has been considered a major virulence attribute of this organism. Previous reports have failed to correlate a specific cloned gene sequence with a purified beta-hemolytic protein sequence. Thus, questions still remain concerning the structural gene sequence of the hemolysin. To answer this question unequivocally, the beta-hemolytic toxin was purified from extracts of log-phase spirochetes, and the N-terminal amino acid sequence was determined (K-D-V-V-A-N-Q-L-N
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Dissertations / Theses on the topic "Hemolytic Disease"

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Fashola, Bola. "The Effect of Sodium Chloride on Beta-Hemolytic Streptococci." TopSCHOLAR®, 1987. https://digitalcommons.wku.edu/theses/2321.

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The drug of choice for the treatment of Stieptococcal pharyngitis is penicillin G. However, a common home remedy prescribes the use of salt-water solutions for gargling. Members of Beta -hemolytic streptococcal groups A, B, and C were isolated from the upper -respiratory tracts of patients diagnosed as having streptococcal pharyngitis. These cultures we:e obtained from HCA Greenview Hospital (Bowling Green, Kentucky) and used to study the effects of sodium chloride on the isolates. The minimum inhibitory concentration of sodium chloride was determined for each of eight hospital isolates. Croup
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Maga, Tara Kristen. "Unraveling the complex genetics of atypical hemolytic uremic syndrome." Diss., University of Iowa, 2012. https://ir.uiowa.edu/etd/2935.

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Atypical hemolytic uremic syndrome (aHUS) is characterized by acute renal failure, thrombocytopenia, and microangiopathic hemolytic anemia. aHUS is far less common and more severe than typical HUS, which is caused by E. coli infection and manifests as diarrheal illness. The pathogenesis of the disease is linked to dysregulation of the alternative pathway of the complement cascade. Mutations in the complement regulators factor H (CFH), membrane cofactor protein (MCP), factor B (CFB), and factor I (CFI) have been implicated
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Bu, Fengxiao. "Exploring the genetics of a complex disease - atypical hemolytic uremic syndrome." Diss., University of Iowa, 2016. https://ir.uiowa.edu/etd/3055.

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Atypical hemolytic uremic syndrome (aHUS) is a rare renal disorder characterized by thrombotic microangiopathy, thrombocytopenia, and acute kidney injury. Its pathogenesis has been attributed to a ‘triggering' event that leads to dysregulation of the complement cascade at the level of the endothelial cell surface. Consistent with this understanding of the disease, mutations in complement genes have been definitively implicated in aHUS. However, the existence of other genetic contributors is supported by two observations. First, in ~50% of cases, disease-causing variants are not identified in c
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Kuruthukulangare, Joseph Sebastian. "Prevention of Rh-hemolytic disease of the newborn : an evaluation of competing strategies." Thesis, McGill University, 1995. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=28806.

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Rh hemolytic disease of the newborn, once a major cause of perinatal mortality and long-term disability, is rarely seen in developed countries today. This drastic reduction in the frequency of disease occurrence has followed the widespread postpartum use of Rh immunoglobulin. However, more than half the world's population does not have access to this health care technology.<br>The objective of this thesis was to study the epidemiology of Rh disease in developed country settings and specifically to quantify the magnitude of Rh disease reduction that occurred secondary to Rh prophylaxis, and to
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Maranho, Caroline Klein. "Prevalência de anticorpos irregulares em gestantes atendidas em serviços públicos da hemorrede de Santa Catarina." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17155/tde-07062017-145049/.

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A doença hemolítica perinatal (DHPN) é caracterizada pela destruição das hemácias fetais por anticorpos da classe IgG presentes na circulação materna. Esses anticorpos, dirigidos contra antígenos eritrocitários presentes nas hemácias do feto, atravessam a barreira placentária e promovem a hemólise prematura dos eritrócitos, podendo levar à anemia fetal. O presente trabalho teve como objetivo principal determinar a prevalência dos anticorpos irregulares em gestantes atendidas em maternidades públicas da hemorrede de Santa Catarina por meio de um estudo observacional, com coorte retrospectiva, e
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Miller, Rachel MD, Alex Yu, and Demetrio Rebano MD Macariola. "Virulent Bacteria in Appalachian Tennessee Waters." Digital Commons @ East Tennessee State University, 2018. https://dc.etsu.edu/asrf/2018/schedule/133.

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BACKGROUND: Over the past 5 years, 634 cases of Shigatoxin E. coli (STEC) infection were reported to Tennessee Health Department 1. At our local children’s hospital, 4-5 children are hospitalized with STEC infection each year. Some of these children had no history of ingesting food items that could have placed them at risk to develop STEC infection; however, there are other ways that humans could get infected, such as exposure to contaminated water from cattle farms 2. GOALS: To determine if bodies of water in the city are contaminated with STEC. METHODS: Fifty (50) ml of water samples were co
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Sá, Cynthia Amaral Moura. "Doença hemolítica perinatal pelo fator Rh: experiência de 10 anos do Instituto Fernandes Figueira." Instituto Fernandes Figueira, 2006. https://www.arca.fiocruz.br/handle/icict/7361.

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Made available in DSpace on 2014-02-26T19:13:30Z (GMT). No. of bitstreams: 2 license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) 56122.pdf: 943164 bytes, checksum: 31ab7a412fa0e24ec2743d0b364e47f6 (MD5) Previous issue date: 2013-07-22<br>Fundação Oswaldo Cruz. Instituto Fernandes Figueira. Departamento de Ensino. Programa de Pós-Graduação em Saúde da Criança e da Mulher. Rio de Janeiro, RJ, Brasil.<br>Introdução: a Doença Hemolítica Perinatal pelo fator Rh é causada pela incompatibilidade entre o sangue da mãe e do recém-nascido, levando a destruição de hemácias feta
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Cavalcante, Francimary de Oliveira. "Presença de Aloanticorpos Eritrocitários em gestantes Rh negativo, atendidas na Fundação de Hematologia e Hemoterapia do Amazonas (Hemoam)." Universidade Federal do Amazonas, 2005. http://tede.ufam.edu.br/handle/tede/2236.

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Made available in DSpace on 2015-04-11T13:38:35Z (GMT). No. of bitstreams: 1 Dissertacao Francimary.pdf: 640225 bytes, checksum: 1eebacf1e67b120750607e8d1a1c46d2 (MD5) Previous issue date: 2005-10-26<br>Conselho Nacional de Desenvolvimento Científico e Tecnológico<br>The presence of G class irregular erythrocyte alloantibodies in the blood circulation during pregnancy may cause a series of consequences to the newborn. The alloimmunization can occur under different situations, such as: blood incompatibility during delivery, miscarriage, amniocentese, blood transfusion, among others, all
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Mohamed, Bashir. "NGS-baserad metod för fetal blodgruppstypning." Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-106208.

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Hemolytic disease of fetus or newborn (HDFN) är en komplikation där foster eller nyföddas erytrocyter förstörs för tidigt. HDFN uppstår när det föreligger blodgruppsinkompatibilitet mellan moder och barnet. Komplikationerna/ symptomen kan variera allt från mildare symptom till fosterdöd. HDFN orsakas framförallt av antikropp D (RhD-immunisering) och på grund av detta utförs det typning av fetalt RhD i maternell plasma. Utöver RhD-immuniseringar kan svåra fall av HDFN ibland orsakas av andra blodgruppssystem som c (Rh) och K (Kell). Fetal RhD-typning görs idag som screening på alla RhD-negativa
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Mallick, Emily M. "A New Murine Model For Enterohemorrhagic Escherichia coli Infection Reveals That Actin Pedestal Formation Facilitates Mucosal Colonization and Lethal Disease: A Dissertation." eScholarship@UMMS, 2012. https://escholarship.umassmed.edu/gsbs_diss/601.

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Enterohemorrhagic Escherichia coli (EHEC) colonizes the intestine and produces the phage-encoded Shiga toxin (Stx) which is absorbed systemically and can lead to hemolytic uremic syndrome (HUS) characterized by hemolytic anemia, thrombocytopenia, and renal failure. EHEC, and two related pathogens, Enteropathogenic E. coli (EPEC), and the murine pathogen, Citrobacter rodentium, are attaching and effacing (AE) pathogens that intimately adhere to enterocytes and form actin “pedestals” beneath bound bacteria. The actin pedestal, because it is a unique characteristic of AE pathogens, has been the s
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Books on the topic "Hemolytic Disease"

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F, Zipfel Peter, ed. Complement and kidney disease. Birkhäuser, 2005.

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Zaki, Maysaa El Sayed. Parvovirus B19 and hematological disordersin [sic] children. Nova Science Publishers, 2010.

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1957-, Hadley Andrew, and Soothill Peter 1957-, eds. Alloimmune disorders of pregnancy: Anaemia, thrombocytopenia, and neutropenia in the fetus and newborn. Cambridge University Press, 2002.

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Geha, Raif, and FRED Rosen. Case Studies in Immunology: Hemolytic Disease of the Newborn. W.W. Norton & Company, 2010. http://dx.doi.org/10.4324/9780203853290.

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Shahcheraghi, Ali. The pathophysiology of anti-Kell "hemolytic disease of newborn". 2005.

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Shahcheraghi, Ali. The pathophysiology of anti-Kell "hemolytic disease of newborn". 2005.

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Geha, Raif, and Fred Rosen. Case Studies in Immunology : Hemolytic Disease of the Newborn: A Clinical Companion. Taylor & Francis Group, 2010.

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Geha, Raif, and Fred Rosen. Case Studies in Immunology : Hemolytic Disease of the Newborn: A Clinical Companion. Norton & Company, Incorporated, W. W., 2010.

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Geha, Raif, and Fred Rosen. Case Studies in Immunology : Hemolytic Disease of the Newborn: A Clinical Companion. Norton & Company, Incorporated, W. W., 2010.

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Geha, Raif, and Fred Rosen. Case Studies in Immunology : Hemolytic Disease of the Newborn: A Clinical Companion. Norton & Company, Incorporated, W. W., 2010.

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Book chapters on the topic "Hemolytic Disease"

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Dias Corrêa Júnior, Mário, Gabriel Martins Cruz Campos, and Priscila Chaves Pita. "Perinatal Hemolytic Disease." In Perinatology. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-83434-0_38.

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Nomdedeu, Meritxell, Joan Cid, Gabriela Simona Ene, Alexandra Pedraza, Arturo Pereira, and Cristina Sanz. "Autoimmune Hemolytic Anemia." In Autoimmune Disease Diagnosis. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-69895-8_103.

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, et al. "Hemolytic Anemia." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_9203.

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Loirat, Chantal, and Véronique Frémeaux-Bacchi. "Atypical Hemolytic Uremic Syndrome." In Pediatric Kidney Disease. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-52972-0_24.

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Bitzan, Martin, and Anne-Laure Lapeyraque. "Postinfectious Hemolytic Uremic Syndrome." In Pediatric Kidney Disease. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-52972-0_26.

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Malina, Michal, Veronique Fremeaux-Bacchi, and Sally Johnson. "Atypical Hemolytic Uremic Syndrome." In Pediatric Kidney Disease. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-11665-0_22.

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Bitzan, Martin, and Anne-Laure Lapeyraque. "Postinfectious Hemolytic Uremic Syndrome." In Pediatric Kidney Disease. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-11665-0_24.

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Pelicano, Miquel Blasco. "Atypical Hemolytic Uremic Syndrome." In Autoimmune Disease Diagnosis. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-69895-8_102.

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Scharnagl, Hubert, Winfried März, Markus Böhm, et al. "Anemia, Hemolytic Autoimmune." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_180.

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, et al. "Hemolytic Uremic Syndrome." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_763.

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Conference papers on the topic "Hemolytic Disease"

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Coz, Julian, and Kishan Patel. "Chilling Complications: A Case of COVID-Associated Cold Autoimmune Hemolytic Anemia (AIHA)." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.44_2024.

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Cold Agglutinin disease (CAD) also known as Cold Autoimmune Hemolytic Anemia (AIHA) is a form of autoimmune hemolytic anemia wherein cold agglutinins (IgM autoantibodies against red blood cell (RBC) antigens) bind during cold temperatures causing clinical symptoms related to RBC agglutination resulting to hemolytic anemia. Clinicians should recognize that Cold Agglutinin disease can be secondary to an underlying pathology such as COVID-19. Here we describe an unusual case of Cold Agglutinin Autoimmune Hemolytic Anemia which was diagnosed in the Emergency Department with the presence of COVID-1
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Souto, Andreza Karine de Barros Almeida, Cristiano Augusto Andrade de Resende, Rafael Brito Foureaux Ribeiro, Andrea Arredondo Farias, and Ana Carolina Silba Barbosa. "Non-immune hemolytic anemia in a patient with advanced breast cancer on capecitabine: A rare adverse event." In Brazilian Breast Cancer Symposium 2023. Mastology, 2023. http://dx.doi.org/10.29289/259453942023v33s1046.

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Background: Stage IV triple-negative breast cancer has a high mortality rate, and the treatment strategy will be based on the presence of biomarkers, disease burden, need for a response rate, and treatment tolerability. Among the various management modalities and effective treatments, capecitabine is a frequently used option due to its known benefits and relatively good tolerance. However, there are several commonly known adverse effects when using capecitabine, including non-immune hemolytic anemia, a very rare and unexpected side effect. Capecitabine is a form of fluoropyrimidine that is hyp
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Ree, I., R. Middelburg, D. Oepkes, J. van der Bom, M. de Haas, and E. Lopriore. "Neonatal Top-Up Transfusions in Alloimmune Hemolytic Disease of the Newborn: Incidence and Risk Factors." In 7th International Conference on Clinical Neonatology—Selected Abstracts. Thieme Medical Publishers, 2018. http://dx.doi.org/10.1055/s-0038-1647096.

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Jin, Yan. "Clinical Case Study of ABO Hemolytic Disease in Full-term Newborns Complicated with Neonatal Pneumonia and Intracranial Hemorrhage." In International Conference on Health Big Data and Intelligent Healthcare. SCITEPRESS - Science and Technology Publications, 2022. http://dx.doi.org/10.5220/0011370300003438.

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Jin, Yan. "Clinical Case Study of ABO Hemolytic Disease in Full-term Newborns with Positive Free Test and Direct Antiglobulin Test." In Conference on Artificial Intelligence and Healthcare. SCITEPRESS - Science and Technology Publications, 2021. http://dx.doi.org/10.5220/0011163000003444.

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Bini, A., V. D"Agati, C. Pirani, B. Kudryk, and K. L. Kaplan. "MONOCLONAL ANTIBODY IDENTIFICATION OF FIBRIN DEPOSITS IN RENAL DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643319.

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Glomerular and vascular "fibrin" deposition has frequently been reported in human and experimental renal diseases. The biochemical form of this "fibrin" has not been well defined. We studied 16 renal biopsies (Bouin's fixed paraffin embedded) with the ABC-immunoperoxidase technique using monoclonal antibodies (MAbs); MAb I8C6 (Bβ1-42) to fibrinogen and fibrin I; MAb T2G1 (β15-42) to fibrin II; and MAb GC4 to fragment D or D-D. Polyclonal antisera to fibrinogen, albumin and IgG were used as controls. Renal biopsy specimens included 9 cases of microangiopathy (Group I: 6 hemolytic uremic syndrom
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Santos, João Vitor Ribeiro dos, Mariana Spitz, and Ana Carolina Andorinho. "Stroke secondary to thrombotic microangiopathy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.300.

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Introduction: Thrombotic thrombocytopenic purpura (TTP) is a hematological disease resulting from the ADAMTS 13 plasmatic protein deficit. It can be congenital or sporadic, and is usually autoimmune. Pathological platelet adhesion occurs, leading to microthrombi in capillary and arterial circulation, microangiopathic anemia and ischemia. The clinical picture includes thrombocytopenia, renal dysfunction, fluctuating neurological symptoms, microangiopathic hemolytic anemia, and fever. Methods: Case report of a 51-year-old male hypertensive patient, diagnosed with idiopathic thrombocytopenic purp
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Taskin, M. Ertan, Tao Zhang, Berry Gellman, Kurt A. Dasse, Bartley P. Griffith, and Zhongjun J. Wu. "3D Flow Modeling and Blood Damage Characterization of the UltraMag™ Blood Pump." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192105.

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Cardiovascular and lung diseases are the leading causes of death and disability worldwide. There are many therapies available for their treatment such as simple lifestyle changes, medicines, and surgical intervention or corrections. For the cases where the above treatments do not alleviate the patients’ conditions, the use of artificial devices may be the only option to treat or replace the diseased organ(s). However, the consideration of blood containing biomedical devices may introduce a prolonged contact between the blood and inorganic surfaces which induces variety of blood damages includi
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9

SCHLEGEL, N., J. MOAKE, C. LOIRAT, M. F. HURTAUD, S. LEVY-TOLEDANO, and H. MATHIEU. "CHILDHOOD HEMOLYTIC UREMIC SYNDROME (HUS) : VON WILLEBRAND FACTOR (vWF) AND PLATELET AGGREGATING ACTIVITY (PAA) STUDIES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643475.

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Abstract:
It has been suggested that a vWF High Molecular Weight Multi-mers (HMWM) decrease or a PAA were involved in the pathogenesis of HUS. We have studied 8 children (6 girls,_2 boys; 7 months-8_1/2 years old) with HUS : plasma creatinine /μmol/l; mean(range)/=306 (105-524), hemoglobin (g/100ml)-7(6.3-7.8), schistocytes (%)=8(1-18), platelets (x103/mm3)-57(10-115). The vWF was studied quantitatively (antigen ; vWF RAg assay) and qualitatively (multimeric pattern : immunoblotting and autoradiography). PAA studied by incubating the patient's platelet poor plasma (RPR) with washed normal platelets (agg
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10

González, Luis A., Graciela S. Alarcón, Guillermina B. Harvey, et al. "LP-061 Male sex and disease activity at diagnosis are predictors of severe hemolytic anemia in patients with systemic lupus erythematosus: data from a multiethnic Latin American cohort." In The 15th International Congress on Systemic Lupus Erythematosus and The 43rd KCR Annual Scientific Meeting & 17th International Symposium (LUPUS & KCR 2023). Lupus Foundation of America, 2023. http://dx.doi.org/10.1136/lupus-2023-kcr.170.

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