Academic literature on the topic 'Hemolytic uremic syndrome Prevention'

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Journal articles on the topic "Hemolytic uremic syndrome Prevention"

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Yoshiya, Kunihiko, Kouichi Nakanishi, Seiko Itoh, Kazumoto Iijima, Norishige Yoshikawa, Hajime Nakamura, Tae Takeda, and Kazuhiro Kobayashi. "Etiology and prevention of hemolytic uremic syndrome." Nihon Shoni Jinzobyo Gakkai Zasshi 7, no. 2 (1994): 161–63. http://dx.doi.org/10.3165/jjpn.7.161.

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Afshar-Kharghan, Vahid. "Atypical hemolytic uremic syndrome." Hematology 2016, no. 1 (December 2, 2016): 217–25. http://dx.doi.org/10.1182/asheducation-2016.1.217.

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Abstract Atypical hemolytic uremic syndrome (aHUS) is a thrombotic microangiopathy (TMA) that affects multiple organs and the kidneys in particular. aHUS can be sporadic or familial and is most commonly caused by dysregulation of the alternative complement pathway. The initial attack of aHUS can occur at any age, and is associated with a high rate of progression to end stage renal disease. Many aHUS patients relapse in the native or transplanted kidneys, and require close monitoring and long-term management. Availability of anticomplement therapy has revolutionized the management of aHUS, and
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Goldwater, Paul N. "Treatment and prevention of enterohemorrhagicEscherichia coliinfection and hemolytic uremic syndrome." Expert Review of Anti-infective Therapy 5, no. 4 (August 2007): 653–63. http://dx.doi.org/10.1586/14787210.5.4.653.

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Haslam, David B. "Molecular Decoys: Novel Approaches to the Prevention of Hemolytic Uremic Syndrome." Pediatric Research 48, no. 3 (September 2000): 267–68. http://dx.doi.org/10.1203/00006450-200009000-00001.

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Joseph, Adrien, Aurélie Cointe, Patricia Mariani Kurkdjian, Cédric Rafat, and Alexandre Hertig. "Shiga Toxin-Associated Hemolytic Uremic Syndrome: A Narrative Review." Toxins 12, no. 2 (January 21, 2020): 67. http://dx.doi.org/10.3390/toxins12020067.

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The severity of human infection by one of the many Shiga toxin-producing Escherichia coli (STEC) is determined by a number of factors: the bacterial genome, the capacity of human societies to prevent foodborne epidemics, the medical condition of infected patients (in particular their hydration status, often compromised by severe diarrhea), and by our capacity to devise new therapeutic approaches, most specifically to combat the bacterial virulence factors, as opposed to our current strategies that essentially aim to palliate organ deficiencies. The last major outbreak in 2011 in Germany, which
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TARR, PHILLIP I. "Escherichia coli O157: H7: Overview of Clinical and Epidemiological Issues." Journal of Food Protection 57, no. 7 (July 1, 1994): 632–37. http://dx.doi.org/10.4315/0362-028x-57.7.632.

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Escherichia coli O157:H7 is an important and common human pathogen which causes diarrhea, bloody diarrhea (hemorrhagic colitis) and the life threatening post-diarrheal disorder, hemolytic uremic syndrome (HUS). Escherichia coli O157:H7 produces one or two potent cytotoxins, designated Shiga-like toxins (or verocytotoxins) I and II. While additional serotypes of cytotoxin-producing E. coli may cause human disease, E. coli O157:H7 is the most important such enteric pathogen in the United States. Epidemiologic data suggest that the incidence of hemolytic uremic syndrome is probably increasing. Un
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Davin, Jean-Claude, Charles Majoie, Jaap Groothoff, Valentina Gracchi, Antonia Bouts, Timothy H. J. Goodship, and Chantal Loirat. "Prevention of large-vessel stenoses in atypical hemolytic uremic syndrome associated with complement dysregulation." Pediatric Nephrology 26, no. 1 (July 24, 2010): 155–57. http://dx.doi.org/10.1007/s00467-010-1608-9.

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Nuernberger, Jens, Oliver Witzke, Russell P. Rother, Thomas Philipp, Udo Vester, Hideo Baba, Lothar Bernd Zimmerhackl, and Andreas Kribben. "Successful Treatment of Atypical Hemolytic Uremic Syndrome with the Complement Inhibitor Eculizumab." Blood 112, no. 11 (November 16, 2008): 2294. http://dx.doi.org/10.1182/blood.v112.11.2294.2294.

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Abstract Background: Atypical hemolytic uremic syndrome (aHUS) is a rare microangiopathic hemolytic anemia characterized by the uncontrolled progression of the alternative complement pathway due to genetic or acquired dysregulation of steady state alternative pathway activity leading to a proinflammatory and prothrombotic condition. Atypical HUS is characterized by intravascular hemolysis, consumptive thrombocytopenia, and microvascular glomerular thrombosis with the formation of thrombi in glomerular capillaries. As the thrombotic microangiopathy is particularly severe in the renal microvascu
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Noris, Marina, and Giuseppe Remuzzi. "Managing and preventing atypical hemolytic uremic syndrome recurrence after kidney transplantation." Current Opinion in Nephrology and Hypertension 22, no. 6 (November 2013): 704–12. http://dx.doi.org/10.1097/mnh.0b013e328365b3fe.

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Avila Bernabeu, Ana Isabel, Teresa Cavero Escribano, and Mercedes Cao Vilarino. "Atypical Hemolytic Uremic Syndrome: New Challenges in the Complement Blockage Era." Nephron 144, no. 11 (2020): 537–49. http://dx.doi.org/10.1159/000508920.

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Atypical hemolytic uremic syndrome (aHUS) is a rare cause of thrombotic microangiopathy (TMA), characterized by microangiopathic hemolytic anemia, consumptive thrombocytopenia, and multisystem end organ involvement, most commonly affecting the kidney. Diagnosis is clinical, after exclusion of other TMA causes. Primary aHUS arises from genetic abnormalities, resulting in uncontrolled complement activity, while a variety of clinical scenarios cause secondary aHUS, including infection, pregnancy, malignancy, autoimmune disease, and medications. They can also induce a temporary complement deregula
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Dissertations / Theses on the topic "Hemolytic uremic syndrome Prevention"

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Srimanote, Potjanee. "Analysis of putative virulence factors of a locus of enterocyte effacement-negative shiga-toxigenic Escherichia coli O113:H21 strain." Title page, contents and abstract only, 2003. http://web4.library.adelaide.edu.au/theses/09PH/09php863.pdf.

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"February 2003." Addendum and corrigenda inserted at back Includes bibliographical references (leaves 249-272) Aims to identify and characterise potential virulence-associated factors from the locus of enterocyte effacement-negative shiga-toxigenic Escherichia coli O113:H21 strain 98NK2 which was responsible for an outbreak of haemolytic uremic syndrome. Particular attention was focused on putative virulence genes encoded on the megaplasmid of this strain.
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Noris, Marina. "Genetics of hemolytic uremic syndrome." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 2006. http://arno.unimaas.nl/show.cgi?fid=7591.

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Maga, Tara Kristen. "Unraveling the complex genetics of atypical hemolytic uremic syndrome." Diss., University of Iowa, 2012. https://ir.uiowa.edu/etd/2935.

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Atypical hemolytic uremic syndrome (aHUS) is characterized by acute renal failure, thrombocytopenia, and microangiopathic hemolytic anemia. aHUS is far less common and more severe than typical HUS, which is caused by E. coli infection and manifests as diarrheal illness. The pathogenesis of the disease is linked to dysregulation of the alternative pathway of the complement cascade. Mutations in the complement regulators factor H (CFH), membrane cofactor protein (MCP), factor B (CFB), and factor I (CFI) have been implicated
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Edwards, Kelly Katherine. "Bacterial factors contributing to the pathogenesis of the hemolytic uremic syndrome." MU has:, 2002. http://wwwlib.umi.com/cr/mo/fullcit?p3060096.

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Bu, Fengxiao. "Exploring the genetics of a complex disease - atypical hemolytic uremic syndrome." Diss., University of Iowa, 2016. https://ir.uiowa.edu/etd/3055.

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Atypical hemolytic uremic syndrome (aHUS) is a rare renal disorder characterized by thrombotic microangiopathy, thrombocytopenia, and acute kidney injury. Its pathogenesis has been attributed to a ‘triggering' event that leads to dysregulation of the complement cascade at the level of the endothelial cell surface. Consistent with this understanding of the disease, mutations in complement genes have been definitively implicated in aHUS. However, the existence of other genetic contributors is supported by two observations. First, in ~50% of cases, disease-causing variants are not identified in c
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Karpman, Diana O. "Studies of the pathogenesis of hemolytic uremic syndrome and thrombotic thrombocytopenic purpura." Lund : Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/68945090.html.

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Valoti, Elisabetta. "Genetic factors associated with anti-factor H autoantibodies in atypical hemolytic uremic syndrome (aHUS)." Thesis, Open University, 2018. http://oro.open.ac.uk/55853/.

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Atypical hemolytic uremic syndrome (aHUS) is a rare form of thrombotic microangiopathy characterized by renal failure and determined by genetic and acquired defects of alternative pathway (AP) of the complement system. Autoantibodies against factor H (anti-FHs), a regulator of the AP, were reported in 10% of patients, and are associated with the deficiency of factor H related 1 (FHR1), a FH homologous protein. The aim of this thesis was to evaluate the contribution of genetics to the development of anti-FHs in aHUS. Thirty patients affected by aHUS resulted positive for anti-FHs (9.8%) and FHR
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Morigi, Marina. "Unravelling molecular and biochemical dysfunction by Shiga toxin: implication for thrombotic microangiopathy in Hemolytic Uremic Syndrome." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 2006. http://arno.unimaas.nl/show.cgi?fid=7590.

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Marinozzi, Maria Chiara. "Characterization of the complement hereditary and acquired abnormalities in atypical Hemolytic Uremic Syndrome and C3 Glomerulopathy." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB037/document.

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McGannon, Colleen M. "Antibiotic Therapy in the Treatment of E. coli O157:H7." University of Cincinnati / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1230919332.

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Books on the topic "Hemolytic uremic syndrome Prevention"

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on hemolytic uremic syndrome. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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Rotman, Tamara Avril. Role of verocytotoxin-1 in the pathogenesis of hemorrhagic colitis and hemolytic uremic syndrome. Ottawa: National Library of Canada, 1992.

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1941-, Kaplan Bernard S., Trompeter Richard S, and Moake Joel L. 1940-, eds. Hemolytic uremic syndrome and thrombotic thrombocytopenic purpura. New York: Marcel Dekker, Inc., 1992.

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Clayton, Glenna. Hemolytic Uremic Syndrome: Symptoms, Treatment Options and Prognosis. Nova Science Publishers, Incorporated, 2014.

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Globotriaosylceramide (Gb3) expression and membrane presentation: Implications for the hemolytic uremic syndrome and human immunodeficiency virus infection. Ottawa: National Library of Canada, 2003.

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Publications, ICON Health. The Official Patient's Sourcebook on Hemolytic Uremic Syndrome: A Revised and Updated Directory for the Internet Age. Icon Health Publications, 2002.

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Enterohemorrhagic Escherichia coli and Other Shiga Toxin-Producing E. coli. ASM Press, 2015.

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F, Zipfel Peter, ed. Complement and kidney disease. Basel: Birkhäuser, 2005.

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Escherichia coli O157:H7 and other shiga toxin-producing E. coli strains. Washington, DC: ASM Press, 1998.

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Book chapters on the topic "Hemolytic uremic syndrome Prevention"

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Sethi, Sidharth Kumar. "Hemolytic Uremic Syndrome." In Critical Care Pediatric Nephrology and Dialysis: A Practical Handbook, 187–94. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-2276-1_15.

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Ali, Uma, and Bradley P. Dixon. "Hemolytic Uremic Syndrome." In Critical Care Nephrology and Renal Replacement Therapy in Children, 129–44. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90281-4_9.

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Metze, Dieter, Vanessa F. Cury, Ricardo S. Gomez, Luiz Marco, Dror Robinson, Eitan Melamed, Alexander K. C. Leung, et al. "Hemolytic Uremic Syndrome." In Encyclopedia of Molecular Mechanisms of Disease, 801–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_763.

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Clark, Elaine, and Scott Orme. "Hemolytic-uremic syndrome." In Health-related disorders in children and adolescents: A guidebook for understanding and educating., 309–14. Washington: American Psychological Association, 1998. http://dx.doi.org/10.1037/10300-042.

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Eddins, Michelle McCown. "Hemolytic-Uremic Syndrome." In Consults in Obstetric Anesthesiology, 255–56. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-59680-8_73.

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Sethna, Christine B., and Shari Gurusinghe. "Hemolytic Uremic Syndrome." In Glomerulonephritis, 1–19. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-27334-1_42-1.

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Vercellone, Antonio, Piero Stratta, and Caterine Canavese. "Hemolytic Uremic Syndrome." In Advances in Experimental Medicine and Biology, 185–98. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-8240-9_24.

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Sethna, Christine B., and Shari Gurusinghe. "Hemolytic Uremic Syndrome." In Glomerulonephritis, 647–65. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-49379-4_42.

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Johnson, S., and C. Mark Taylor. "Hemolytic Uremic Syndrome." In Pediatric Nephrology, 1155–80. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-76341-3_48.

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Watkins, Sandra L. "Hemolytic Uremic Syndrome." In Textbook of Clinical Pediatrics, 2769–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_297.

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Conference papers on the topic "Hemolytic uremic syndrome Prevention"

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Bahlavouni, Alina, and Aleena Paul. "Hemolytic Uremic Syndrome Associated with Entamoeba Histolytica." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.866.

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Selvam, M. Masilamani, Divya Sasitharan, and W. Manohar Paul. "Clinical validation of vaccines against hemolytic uremic syndrome." In 2014 International Conference on Science Engineering and Management Research (ICSEMR). IEEE, 2014. http://dx.doi.org/10.1109/icsemr.2014.7043626.

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Pankratenko, T. E., O. V. Moskalec, and T. Yu Abaseeva T. "Cell adhesion molecules in childrenwith diarrhea-associated hemolytic uremic syndrome." In General question of world science. "Л-Журнал", 2018. http://dx.doi.org/10.18411/gq-31-03-2018-13.

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Moskalec, O. V., T. E. Pankratenko, and T. Yu Abaseeva. "Soluble adhesion molecules in children with typical hemolytic uremic syndrome." In Scientific achievements of the third millennium. SPC "LJournal", 2018. http://dx.doi.org/10.18411/scc-05-2018-06.

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Abdou, M., M. Alsharif, T. Naguib, and M. L. Patel. "Eculizumab: A Life Saver in Postpartum Atypical Hemolytic Uremic Syndrome." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6530.

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ROMERO LEGRO, IVAN, Sadia Shah, and Vaidehi Kaza. "Tacrolimus Induced Hemolytic Uremic Syndrome In A Lung Transplant Patient." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5834.

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Moskalets, O. V., T. E. Pankratenko, and T. Yu Abaseeva. "Biomarkers of endothelial disfunction in children with atypical hemolytic uremic syndrome." In Scientific achievements of the third millennium. SPC "LJournal", 2018. http://dx.doi.org/10.18411/scc-09-2018-08.

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Cruz, Pedro Costa e., Patrícia Mendes, Marisol Anselmo, and Luís Gonçalves. "P102 Typical vs atypical hemolytic-uremic syndrome: eculizumab, a difficult decision." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.190.

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Moskalets, O. V. "Prognostic value of sICAM-1 and sVCAM-1 in hemolytic uremic syndrome." In Global science. Development and novelty. НИЦ «Л-Журнал», 2018. http://dx.doi.org/10.18411/gdsn-25-12-2018-04.

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Pankratenko, T. E., and O. V. Moskalec. "sICAM-1 and sVCAM-1 in children with typical hemolytic uremic syndrome." In Global science. Development and novelty. НИЦ «Л-Журнал», 2018. http://dx.doi.org/10.18411/gdsn-28-02-2018-07.

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