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Journal articles on the topic 'Hemorrhagic colitis'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Lior, H. "Hemorrhagic Colitis." Canadian Institute of Food Science and Technology Journal 21, no. 4 (October 1988): 372. http://dx.doi.org/10.1016/s0315-5463(88)70969-4.

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2

Moon, Hee-Jung, Byung-Ik Jang, Sung-Bum Kim, Ho-Chan Lee, Jae-Hyun Park, Jong-Ryul Eun, and Tae-Nyeun Kim. "Ulcerative Colitis Mimicking Acute Hemorrhagic Colitis." Yeungnam University Journal of Medicine 25, no. 2 (2008): 182. http://dx.doi.org/10.12701/yujm.2008.25.2.182.

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3

Khaertynov, H. S., D. S. Semenova, and K. V. Sushnikov. "Clinical and epidemiological features of acute intestinal infections with hemorragic colitis in children." Kazan medical journal 94, no. 2 (April 15, 2013): 208–11. http://dx.doi.org/10.17816/kmj1590.

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Aim. To study the clinical and epidemiological features of acute intestinal infections associated with hemorrhagic colitis in children. Methods. The clinical and epidemiological features of hemorrhagic colitis were studied in 70 children with acute intestinal infections aged from 1 month to 14 years who were admitted to the Infectious Diseases Hospital in Kazan, Russia. The following stool tests were performed: single bacteriology test for pathogenic and conditionally pathogenic microbiota, rotavirus antigen detection by latex agglutination, campylobacter DNA detection by polymerase chain reaction and microscopy to detect protozoa. Results. Hemorrhagic colitis was present mainly in infants (56 children, 80%), the main reasons for hemorrhagic colitis were: Salmonella enteritidis - 12 (17.1%) children, Campylobacter - 6 (8.6%) children, Klebsiella pneumoniae - 6 (8.6%) children. There were single cases of hemorrhagic colitis associated with Shigella, Enterobacter, S. aureus, Ps. aeruguinosa and E. hystolitica. The diagnosis of Campylobacter infection was based on the detection of the DNA but not the bacteria itself like in other acute intestinal infections. The majority of hemorrhagic colitis cases (52 children, 74.3%) were registered in spring and summertime. Hemorrhagic colitis was moderately severe and manifested as traces of blood and mucus in stool. Hemorrhagic colitis usually lasted up to 3 days in majority of children. Conclusion. The main reasons for acute intestinal infections associated with hemorrhagic colitis were: Salmonella enteritidis, Campilobacter and Klebsiella pneumonia, hemorrhagic colitis was most common in infants in spring and summertime.
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4

Moulis, Harry, and Ronald J. Vender. "Antibiotic-Associated Hemorrhagic Colitis." Journal of Clinical Gastroenterology 18, no. 3 (April 1994): 227–31. http://dx.doi.org/10.1097/00004836-199404000-00012.

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5

Olayode, Adegboyega. "Antibiotic-Associated Hemorrhagic Colitis." American Journal of Gastroenterology 109 (October 2014): S430. http://dx.doi.org/10.14309/00000434-201410002-01452.

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6

MOSES, FRANK M. "Running-Associated Proximal Hemorrhagic Colitis." Annals of Internal Medicine 108, no. 3 (March 1, 1988): 385. http://dx.doi.org/10.7326/0003-4819-108-3-385.

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7

Ganesan, Rajeshkumar, and Rajalakshmi Ettiyan. "Abrus Precatorius Induced Hemorrhagic Colitis." American Journal of Pharmacology and Toxicology 10, no. 2 (February 1, 2015): 40–45. http://dx.doi.org/10.3844/ajptsp.2015.40.45.

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8

Kram, Michael T., Drew A. Olsen, Louis D. May, and Stephen Goodman. "Hemorrhagic colitis caused by amyloid." Gastrointestinal Endoscopy 56, no. 4 (October 2002): 564–65. http://dx.doi.org/10.1016/s0016-5107(02)70445-8.

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9

Brown, Christopher, Mark Metwally, Christopher Bach, Vikram Anand, Justin Provost, and William Hale. "Oseltamivir-Induced Acute Hemorrhagic Colitis." American Journal of Gastroenterology 109 (October 2014): S416. http://dx.doi.org/10.14309/00000434-201410002-01407.

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10

Riley, Lee W. "Hemorrhagic colitis—A “new” disease." Clinical Microbiology Newsletter 7, no. 7 (April 1985): 47–49. http://dx.doi.org/10.1016/s0196-4399(85)80061-1.

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11

Kram, Michael T., Drew A. Olsen, Louis D. May, and Stephen Goodman. "Hemorrhagic colitis caused by amyloid." Gastrointestinal Endoscopy 56, no. 4 (October 2002): 564–65. http://dx.doi.org/10.1067/mge.2002.127152.

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12

Jonides, Linda, Sally Walsh, and Carol Rudy. "Hemorrhagic colitis in a toddler." Journal of Pediatric Health Care 8, no. 4 (July 1994): 191. http://dx.doi.org/10.1016/0891-5245(94)90036-1.

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13

Nishiwaki, Satoshi, Matsuyoshi Maeda, Masahiro Yamada, Shingo Okuno, Yasuhiko Harada, Kotaro Suzuki, Shingo Kurahashi, Fumihiro Urano, Shozo Okamura, and Isamu Sugiura. "Significance of Fecal Occult Blood Test Followed By Colonoscopy for Asymptomatic Dasatinib-Induced Hemorrhagic Colitis (CML1501 Study)." Blood 128, no. 22 (December 2, 2016): 1917. http://dx.doi.org/10.1182/blood.v128.22.1917.1917.

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Abstract Introduction: Positive fecal occult blood test (FOBT) was occasionally observed in some chronic myeloid leukemia (CML) patients treated with a tyrosine kinase inhibitor (TKI). Although there are several case reports of hemorrhagic colitis in patients treated with dasatinib, the incidence of TKI-induced hemorrhagic colitis and the screening efficacy of FOBT followed by colonoscopy are unknown. Methods: We prospectively enrolled CML patients treated with a TKI. The first FOBT (FOBT1) was performed for all patients. The first CF (CF1) was performed for patients with positive FOBT1. When there were any lesions, tissue biopsy was performed for pathological analyses. After confirmation of TKI-induced colitis by pathological analyses, the TKI was interrupted for 2-4 weeks. And then, FOBTs were re-evaluated (FOBT2). Results: Between February 2015 and September 2015, 30 patients were enrolled in this study. All patients were in chronic phase. FOBT1 was positive in 10 of 30 patients. All patients with positive FOBT1 were treated with dasatinib and revealed no symptoms. CF1 was performed for 10 patients with positive FOBT1. All patients showed abnormal endoscopic findings: colitis in 6 patients, polyps in 3, and hemorrhoid in 1. Dasatinib-induced hemorrhagic colitis was confirmed in 6 of 18 patients treated with dasatinib (33%). Median duration of dasatinib before FOBT1 was 16 months (range 3-35 months). Median dose of dasatinib was 85mg (range 50-100mg). It had been reduced in 3 patients due to pleural effusion. Its endoscopic feature was read flare and/or erosion. Immunohistological analyses showed CD3+, CD8+, CD56+ and Granzyme B+ cytotoxic T lymphocyte infiltration. Dasatinib was discontinued at a median of 27 days after CF1 in patents with dasatinib-induced hemorrhagic colitis. Discontinuation period was 14 days in 4 patients, 27 days in 1, and 28 days in 1. FOBT2 was negative in all but one patient who had concurrent colorectal polyps. No CML progression due to dasatinib discontinuation was observed. Conclusion: Dasatinib-induced hemorrhagic colitis was observed in a third of asymptomatic patients treated with dasatinib. FOBT can be a useful non-invasive screening for the disease, and it can be confirmed by the following CF and pathological analyses. It was caused by cytotoxic T lymphocytes, and hemorrhage was resolved after dasatinib discontinuation. Disclosures No relevant conflicts of interest to declare.
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14

Yoon, Seok Ho, Il Joong Park, and Wee Gyo Lee. "A Case ofEscherichia coliO157 Hemorrhagic Colitis." Korean Journal of Clinical Microbiology 11, no. 1 (2008): 66. http://dx.doi.org/10.5145/kjcm.2008.11.1.66.

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15

Stavric, S., and J. I. Speirs. "Escherichia coli Associated with Hemorrhagic Colitis." Canadian Institute of Food Science and Technology Journal 22, no. 3 (June 1989): 205–8. http://dx.doi.org/10.1016/s0315-5463(89)70375-8.

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16

Elli, Luca, Maria Teresa Bardella, Paolo D. Pigatto, and Gianpaolo Guzzi. "Mercury Vapor Overexposure and Hemorrhagic Colitis." American Journal of Gastroenterology 104, no. 8 (August 2009): 2124. http://dx.doi.org/10.1038/ajg.2009.243.

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17

Yeo, Shen-Ann Eugene, Yon Kuei Lim, Kiat Hon Tony Lim, and Choong-Leong Tang. "Toxic Haemorrhagic Colitis: A Rare Presentation of Eosinophilic Colitis." Case Reports in Gastrointestinal Medicine 2012 (2012): 1–3. http://dx.doi.org/10.1155/2012/279813.

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Eosinophilic colitis is a rare condition that usually presents with non specific abdominal symptoms. Very uncommonly it presents with an acute surgical emergency such as peritonitis or haemorrhage. We present a rare presentation of eosinophilic colitis with toxic hemorrhagic colitis and ischaemic bowel requiring laparotomy and bowel resection.
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18

Parks, Paul T., and Alexander S. Easton. "Cerebral Vasculitis in Ulcerative Colitis Is Predominantly Venular: Case Report and Review of the Literature." Case Reports in Rheumatology 2019 (February 26, 2019): 1–6. http://dx.doi.org/10.1155/2019/9563874.

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Extraintestinal complications of ulcerative colitis include isolated case reports of cerebral vasculitis. In this case report, we describe autopsy findings in a 50-year-old female who died as a result of massive multifocal cerebral hemorrhage. Microscopic examination of the left colon showed findings typical for ulcerative colitis. Examination of the brain showed an extensive vasculitis. More affected vessels were noted in grey matter than in white matter. Many showed fibrinoid necrosis, invasion by neutrophils and thrombosis. There was extensive perivascular hemorrhage with associated infarction. Vessel analysis shows most of the vessels to have been venous rather than arterial. There were no perivascular sleeves of demyelination to suggest a primary demyelinating disorder, such as acute hemorrhagic leucoencephalitis. Our analysis shows that veins are the likely target of cerebral vasculitis in ulcerative colitis. This has clinical implications because venous occlusion generally causes massive intracerebral hemorrhage with a high mortality.
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19

Molochkova, O. V., O. B. Kovalev, O. V. Shamsheva, N. V. Sokolova, A. A. Sakharova, N. I. Krylatova, E. V. Galeeva, A. A. Korsunskiy, and O. A. Kashchenko. "Bacterial diarrhea in hospitalized children." CHILDREN INFECTIONS 18, no. 4 (December 6, 2019): 12–18. http://dx.doi.org/10.22627/2072-8107-2019-18-4-12-18.

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Objective: to study the clinical and laboratory manifestations of acute intestinal infections of bacterial etiology in hospitalized children, depending on the etiology, age, topic of the lesion, and complications.A cohort clinical study of 570 children hospitalized from January to October 2019 in the infectious wards of Children's Clinical Hospital No.9 in Moscow was conducted. Studies included routine laboratory methods, bacteriological analysis of feces, Latex test, ELISA, PCR to detect pathogens, serological reactions to detect specific antibodies in blood serum. Two groups of patients were specially formed to compare the features of the course of bacterial diarrhea without hemorrhagic colitis (n = 111) and with hemorrhagic colitis (n = 125).Among 33.2% of cases of deciphered etiology of bacterial diarrhea, Salmonella was isolated in 14.6%, Campylobacter — in 4.7%, Shigella — in 3%, in other cases — conditionally pathogenic flora. Salmonellosis and Сampylobacteriosis with the same frequency are recorded in children in the age groups of 1—3 and 3—7 years, in half of the patients proceed as enterocolitis (58 and 52%, respectively). Shigellosis is diagnosed in children older than 1 year of life with the same frequency in age groups 1—3, 3—7 and older than 7 years, a third of patients (35%) develop severe forms.In most cases (83.2%), a complicated course of bacterial diarrhea was detected, more often dehydration 1 and 2 degrees (36.5%), acute respiratory viral infections (19.6%), mesadenitis (11.2%), community-acquired pneumonia (4.6%).Enterocolitis is the topic of lesion in half of the children, and every 5th patient develops hemorrhagic colitis (21.9%). In 44% of cases of established etiology of hemorrhagic colitis, Salmonella was confirmed. Hemocolitis also develops more often with shigellosis, campylobacteriosis and clostridiosis. In the group of bacterial diarrhea with hemorrhagic colitis, significant differences were revealed compared with the group without hemocolitis: by the frequency of development in children under 3 years of age; by the duration of treatment in a hospital; by the frequency of mesadenitis; by the content of stab neutrophils in the hemogram, reflecting a more pronounced inflammation in hemorrhagic colitis.Thus, bacterial diarrhea in children remains relevant due to the severity of inflammation, the incidence of complicated course and hemorrhagic colitis, which requires hospitalization.
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20

Millichap, J. Gordon. "E Coli Hemorrhagic Colitis and Neurologic Symptoms." Pediatric Neurology Briefs 7, no. 6 (June 1, 1993): 42. http://dx.doi.org/10.15844/pedneurbriefs-7-6-2.

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21

Bennett, Michael R., Arthur P. Fine, and James T. Hanlon. "Cytomegalovirus hemorrhagic colitis in a nontransplant patient." Postgraduate Medicine 77, no. 8 (June 1985): 227–32. http://dx.doi.org/10.1080/00325481.1985.11699040.

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22

Gorgis, Sarah, Dorian Jones, Kajali Mishra, and Yousuf Siddiqui. "Lenalidomide Induced Hemorrhagic Colitis in Multiple Myeloma." American Journal of Gastroenterology 113, Supplement (October 2018): S950. http://dx.doi.org/10.14309/00000434-201810001-01652.

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23

Byrd, Richard L., Marshall W. Cunningham, and Leonard I. Goldman. "Nonocclusive ischemic colitis secondary to hemorrhagic shock." Diseases of the Colon & Rectum 30, no. 2 (February 1987): 116–18. http://dx.doi.org/10.1007/bf02554948.

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24

Abe, Koichiro, Yu Kawashima, Hitoshi Aoyagi, Satoshi Kimura, Akari Isono, Tadahisa Ebato, Takatsugu Yamamoto, Hiroto Kita, and Yasushi Kuyama. "Antibiotic-associated hemorrhagic colitis with ischemic change." Endoscopy 46, S 01 (December 11, 2014): E601—E602. http://dx.doi.org/10.1055/s-0034-1390717.

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25

Kulkarni, Ketan, Doug Weine, and Charles Maltz. "Klebsiella Oxytoca and Antibiotic-Associated Hemorrhagic Colitis." American Journal of Gastroenterology 103 (September 2008): S226. http://dx.doi.org/10.14309/00000434-200809001-00584.

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26

Hettiarachchi, Irasha Thulani, Manish Hegde, Andrew Charles Planner, and Lawrence John. "Escherichia coli O157:H7 causing hemorrhagic colitis." Gastrointestinal Endoscopy 75, no. 3 (March 2012): 674–75. http://dx.doi.org/10.1016/j.gie.2011.11.024.

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27

Eidus, LB, M. Guindi, J. Drouin, S. Gregoire, and JR Barr. "Colitis Caused by Escherichia Coli 01587:H7: A Study of Six Cases." Canadian Journal of Gastroenterology 4, no. 4 (1990): 141–46. http://dx.doi.org/10.1155/1990/125627.

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The clinical and pathologic features of six patients with proven verotoxin-producing Escherichia coli colitis are described. Clinical data, 25 biopsy specimens and two autopsies from these patients are reviewed. All presented with crampy abdominal pain and bloody diarrhea. Colonoscopic findings included edema, erythema, pseudomembranes and hemorrhage. On biopsy, two patients had ischemic colitis, one had pseudomembranous colitis and three had a combination of concurrent ischemic and pseudomembranous colitis. Four cases showed fibrin-platelet thrombi in mucosal capillaries and submucosal arterioles. The classical pattern of infectious colitis was not seen in these cases. Other nonspecific changes included patchy mucosal edema, congestion, focally prominent interstitial hemorrhage and mild, patchy increase of the lymphoplasmocytic component of the lamina. Ischemic necrosis was present in 10 of 25 biopsies (40%), pseudomembranes in seven of 25 biopsies (28%), and four of 25 biopsies (16%) showed both. Colon from one autopsy revealed edema, pseudo membranes and intramural infarction . Concurrent thrombotic thrombocytopenic purpura was clinically documented in three of six patients. It is concluded that, in the context of hemorrhagic colitis, the following observations are indicative of E coli 0157:H7: the combination of pseudomcmbranous and ischemic colitis; ischemic colitis in a young patient; or pseudomembranous colitis with Clostridium difficile-negative culture and toxin. Multiple biopsies are required to demonstrate the full-blown features. E coli 0157:H7 colitis should be added to the differential diagnosis of submucosal edema.
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28

Fisher, Aaron, and Alexandra Halalau. "A Case Report and Literature Review of Clostridium difficile Negative Antibiotic Associated Hemorrhagic Colitis Caused by Klebsiella oxytoca." Case Reports in Gastrointestinal Medicine 2018 (September 24, 2018): 1–4. http://dx.doi.org/10.1155/2018/7264613.

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Klebsiella oxytoca hemorrhagic colitis is a rare form of antibiotic associated hemorrhagic colitis that is Clostridium difficile negative. Klebsiella oxytoca colitis has been shown to be triggered by penicillin administration, yet other antibiotics have been implicated as well. It can mimic the appearance of ischemic colitis on endoscopy; however it will generally be found in young, otherwise healthy patients without risk factors. We present a case of a 33-year-old Caucasian female who presented to the emergency room with profuse, bloody diarrhea for 5 days, after a one-week course of ampicillin. Colonoscopy was notable for ulcerated mucosa with erythema and easy friability and the biopsy was suggestive of ischemic colitis. Stool culture was positive for many Klebsiella oxytoca. The patient was discharged home with resolution of symptoms after three days in the hospital. She was instructed to avoid penicillin antibiotics and minimize nonsteroidal anti-inflammatory drug use.
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29

M. Robson, Wm Lane, Alexander K. C. Leung, and Darlene J. Miller-Hughes. "RECURRENT HEMORRHAGIC COLITIS CAUSED BY ESCHERICHIA COLI 0157." Pediatric Infectious Disease Journal 12, no. 8 (August 1993): 699–700. http://dx.doi.org/10.1097/00006454-199308000-00018.

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30

Carvalho, Alexandre, Rana Alqusairi, Anna Adams, Michelle Paul, Neelay Kothari, Stevany Peters, and Anthony T. DeBenedet. "SARS-CoV-2 Gastrointestinal Infection Causing Hemorrhagic Colitis." American Journal of Gastroenterology 115, no. 6 (June 2020): 942–46. http://dx.doi.org/10.14309/ajg.0000000000000667.

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31

Kishida, Teruyuki, Jun Sato, Shunji Fujimori, Sadamu Minami, Susumu Yamakado, Yasuhito Tamagawa, Fumihiko Taguchi, Yutaka Yoshida, and Masafumi Kobayashi. "An endoscopic study of antibiotic-associated hemorrhagic colitis." Journal of Nippon Medical School 59, no. 6 (1992): 450–56. http://dx.doi.org/10.1272/jnms1923.59.450.

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32

Koga, Hideki, Kunihiko Aoyagi, Ryuji Yoshimura, Yutaka Kimura, Mitsuo Iida, and Masatoshi Fujishima. "Can quinolones cause hemorrhagic colitis of late onset?" Diseases of the Colon & Rectum 42, no. 11 (November 1999): 1502–4. http://dx.doi.org/10.1007/bf02235056.

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33

Morchón-Simón, David, and Juan Carlos Martín-Escudero. "Hemorrhagic colitis as the onset of Wegener’s granulomatosis." International Journal of Colorectal Disease 26, no. 2 (June 11, 2010): 259–60. http://dx.doi.org/10.1007/s00384-010-0977-7.

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34

Shanshal, Mohamed, Andrew Shakespeare, Seshadri Thirumala, Boyd Fenton, and Donald P. Quick. "Dasatinib-Induced T-Cell-Mediated Colitis: A Case Report and Review of the Literature." Acta Haematologica 136, no. 4 (2016): 219–28. http://dx.doi.org/10.1159/000448983.

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Dasatinib is a potent inhibitor of the altered tyrosine kinase activity in disease states associated with BCR/ABL1. This agent has been shown to exhibit broad off-target kinase inhibition and immunomodulating properties. These effects may be responsible for dasatinib's unique side effects including a distinctive form of hemorrhagic colitis. We report a case of hemorrhagic colitis associated with dasatinib use in a patient with chronic myelogenous leukemia. Colon biopsies at the time of symptomatic colitis confirmed CD3+CD8+ T cell infiltration. The process rapidly resolved following drug discontinuation, but relapsed when rechallenged with a reduced dose of dasatinib. Colitis did not recur when the patient was treated with an alternative agent. A literature review of prior cases involving dasatinib-induced T-cell mediated colitis provides insight into commonalities that may facilitate the recognition and management of this entity. Most incidences occurred after a 3-month drug exposure and may be accompanied by large granular lymphocytes. The process uniformly resolves within a few days following drug discontinuation and will generally recur in a shorter period of time if the drug is reintroduced. Most patients will require an alternative agent, although select patients could be continued on dasatinib if other options are limited.
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35

Kekilli, Murat. "Dasatinib-Induced Severe Hemorrhagic Colitis in Chronic Myeloid Leukemia." International Journal of Hematology and Oncology 26, no. 1 (March 1, 2016): 1–2. http://dx.doi.org/10.4999/uhod.740.

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36

Kasap, Burcu. "Ischemic Colitis after Hemorrhagic Shock Due to Obstetrical Reasons." Clinical Studies & Medical Case Reports 2, no. 1 (March 27, 2015): 1–3. http://dx.doi.org/10.24966/csmc-8801/100011.

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37

Okayama, Seiji, Shoichi Arakawa, Kenji Ogawa, and Tsunehisa Makino. "A case of hemorrhagic colitis after influenza A infection." Journal of Microbiology, Immunology and Infection 44, no. 6 (December 2011): 480–83. http://dx.doi.org/10.1016/j.jmii.2011.04.003.

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38

Philbrick, Ann M., and Michael E. Ernst. "Amoxicillin-Associated Hemorrhagic Colitis in the Presence ofKlebsiella oxytoca." Pharmacotherapy 27, no. 11 (November 2007): 1603–7. http://dx.doi.org/10.1592/phco.27.11.1603.

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39

Pérez-Castrillón, José L., Antonio Dueñas, Maria A. Goyeneche, Juan C. Martin-Escudero, and Vicente Herreros. "Hemorrhagic Colitis Due to Amoxicillin/Clavulanate and Nasal Decongestants?" Journal of Clinical Gastroenterology 25, no. 4 (December 1997): 701. http://dx.doi.org/10.1097/00004836-199712000-00035.

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40

Bopp, C. A., K. D. Greene, F. P. Downes, E. G. Sowers, J. G. Wells, and I. K. Wachsmuth. "Unusual verotoxin-producing Escherichia coli associated with hemorrhagic colitis." Journal of Clinical Microbiology 25, no. 8 (1987): 1486–89. http://dx.doi.org/10.1128/jcm.25.8.1486-1489.1987.

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41

Riley, L. W. "The Epidemiologic, Clinical, and Microbiologic Features of Hemorrhagic Colitis." Annual Review of Microbiology 41, no. 1 (October 1987): 383–405. http://dx.doi.org/10.1146/annurev.mi.41.100187.002123.

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42

Heise, Laura A., Brant M. Wagener, Jennifer R. Vigil, Mohamed Othman, and Parsa Shahinpoor. "Hemorrhagic Colitis Secondary to Acute Elemental Mercury Vapor Poisoning." American Journal of Gastroenterology 104, no. 2 (January 13, 2009): 530–31. http://dx.doi.org/10.1038/ajg.2008.54.

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43

Cho, S., S. Kim, W. Kim, L. Chul-Hyun, and B. Go. "Hemorrhagic bullous colitis in a patient with multiple myeloma." Endoscopy 45, S 02 (May 28, 2013): E157—E158. http://dx.doi.org/10.1055/s-0032-1326494.

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44

Kim, Hyun Soo, Min Wook Kim, Si Ho Kim, Geun Kim, and Jung Hyun Lee. "Acute Hemorrhagic Colitis Induced by Oral Administration of Oseltamivir." Ewha Medical Journal 39, no. 2 (2016): 56. http://dx.doi.org/10.12771/emj.2016.39.2.56.

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45

Corinaldesi, Giorgio, and Christian Corinaldesi. "Thrombocitopenic Purpura Following Hemorrhagic Colitis Associeted with Immunosoppressive Agents." American Journal of Gastroenterology 100 (September 2005): S288—S289. http://dx.doi.org/10.14309/00000434-200509001-00777.

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46

Saito, Motonobu, Atsushi Ishino, Taisuke Ito, Takeshi Sakuma, Masami Matsuzaki, Naoto Katagata, Fumiaki Watanabe, Seiichi Takenoshita, and Tadashi Nomizu. "Hemorrhagic Cytomegalovirus Colitis in a Postoperative Colon Cancer Patient." Case Reports in Oncology 6, no. 1 (2013): 109–13. http://dx.doi.org/10.1159/000348711.

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47

Parks, Lauren M., Shirley Cohen-Mekelburg, and David Wan. "Klebsiella Oxytoca: A Case of Antibiotic- Associated Hemorrhagic Colitis." American Journal of Gastroenterology 111 (October 2016): S611—S612. http://dx.doi.org/10.14309/00000434-201610001-01357.

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48

Fawley, Ryan K., and Joseph G. Cheatham. "Klebsiella Oxytoca: A Commonly Unrecognized Cause of Hemorrhagic Colitis." American Journal of Gastroenterology 111 (October 2016): S624. http://dx.doi.org/10.14309/00000434-201610001-01384.

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49

Moxley, Rodney A. "Special Issue: Shiga Toxin-Producing Escherichia coli." Microorganisms 9, no. 1 (December 22, 2020): 1. http://dx.doi.org/10.3390/microorganisms9010001.

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Globally, Shiga toxin-producing Escherichia coli (STEC) is an important cause of diarrheal disease, most notably hemorrhagic colitis, and post-diarrheal sequela, such as hemolytic-uremic syndrome (HUS) [...]
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Svetoch, E. A., I. A. Dyatlov, N. N. Kartsev, B. V. Eruslanov, M. E. Kanashenko, and N. K. Fursova. "Development of candidate vaccines against infection caused by shiga-toxin producing Escherichia coli. Part 1." Bacteriology 5, no. 2 (2020): 56–70. http://dx.doi.org/10.20953/2500-1027-2020-2-56-70.

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Abstract:
Shiga-toxin producing Escherichia coli (STEC) strains cause serious and life-threatening diseases, hemorrhagic colitis (HC) and associated hemolytic uremic syndrome (HUS). Antibacterial etiotropic therapy against these diseases are not recommended. There are no vaccines against human HA and HUS. The review provides materials on the design of various types of candidate vaccines against STEC strains and assessment of their immunogenic and protective properties in experiments on laboratory and farm animals. The prospects for the use of inactivated corpuscular and live (vector) vaccines, lipopolysaccharide vaccines, DNA vaccines and nanovaccines, vaccines based on bacterial cell membranes (ghost), as well as vaccines created by reverse vaccinology methods in practice are considered. Key words: STEC, hemorrhagic colitis, immunodominant antigens, shiga toxins, EspA, EspB, Tir, intimine, IgG, sIgA
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