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Journal articles on the topic 'Hemosiderose pulmonaire'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 February 2022

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1

Zinoune, B., H. En-Nouali, B. Radouane, M. Jidal, S. Chaouir, T. Amil, A. Hanine, and M. Benameur. "RP38 Hemosiderose pulmonaire idiopathique. A propos d’une observation." Journal de Radiologie 87, no. 10 (October 2006): 1548. http://dx.doi.org/10.1016/s0221-0363(06)87944-8.

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2

Vaysman, Dmitriy, and Haesoon Lee. "Idiopathic Pulmonary Hemosiderosi." Chest 124, no. 4 (January 2003): 276S. http://dx.doi.org/10.1378/chest.124.4_meetingabstracts.276s-a.

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3

Levy, Jacov, and Robert W. Wilmott. "Pulmonary hemosiderosis." Pediatric Pulmonology 2, no. 6 (November 1986): 384–91. http://dx.doi.org/10.1002/ppul.1950020612.

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4

Asano, Takeshi, Miho Maeda, and Masao Yamamoto. "Pulmonary hemosiderosis." Journal of Nippon Medical School 64, no. 4 (1997): 356–58. http://dx.doi.org/10.1272/jnms1923.64.356.

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5

Barut, Kenan, Sezgin Sahin, Amra Adrovic, Velat Sen, and Ozgur Kasapcopur. "Idiopathic Pulmonary Hemosiderosis in a Child with Recurrent Macrophage Activation Syndrome Secondary to Systemic Juvenile Idiopathic Arthritis." Case Reports in Pediatrics 2017 (2017): 1–4. http://dx.doi.org/10.1155/2017/5693501.

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Macrophage activation syndrome, a severe complication of systemic juvenile idiopathic arthritis and other inflammatory diseases, represents one of the most important rheumatological emergencies. Delayed diagnosis could lead to life-threatening complications. Pulmonary hemosiderosis has been classically characterized by a triad of anemia, hemoptysis, and lung infiltrates on chest radiogram. Although the majority of patients of pulmonary hemosiderosis are considered idiopathic, secondary hemosiderosis associated with known diseases could be seen. In this case report, we aimed to present gradually increased pulmonary manifestations due to pulmonary hemosiderosis with recurrent macrophage activation syndrome attacks in a child with systemic juvenile idiopathic arthritis.

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6

Muñoz Durán, Julián Andrés, and Ana Fernanda Muñoz Durán. "Hallazgos imagenológicos de la hemosiderosis pulmonar idiopática en el adulto: una revisión de la literatura de los últimos 20 años." Revista Médicas UIS 33, no. 2 (August 31, 2020): 55–64. http://dx.doi.org/10.18273/revmed.v33n2-2020010.

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La hemosiderosis pulmonar idiopática es una entidad rara caracterizada por hemorragia alveolar capilar. Su tríada clásica es hemoptisis crónica o recurrente, anemia por deficiencia de hierro y opacidades en las imágenes pulmonares. El objetivo de esta revisión fue determinar la frecuencia en radiografía y tomografía de tórax, de los hallazgos de hemosiderosis pulmonar idiopática en adultos, reportados en la literatura durante los últimos 20 años, de acuerdo con los hallazgos semiológicos en imagen, localización y distribución. Se hizo una búsqueda de publicaciones en bases de datos. Se seleccionaron 42 estudios, se estratificaron variables y se recopilaron los hallazgos. La consolidación y las opacidades reticulonodulares fueron el hallazgo más frecuente en radiografía. En tomografía el hallazgo principal fue el vidrio deslustrado en la región basal. No hubo diferencias en los hallazgos semiológicos de imagen en cuanto al compromiso de acuerdo con el género, aunque las mujeres tendieron a presentar una distribución difusa. MÉD.UIS.2020;33(2):55-64

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7

Cohen, Schlomo. "Idiopathic Pulmonary Hemosiderosis." American Journal of the Medical Sciences 317, no. 1 (January 1999): 67–74. http://dx.doi.org/10.1016/s0002-9629(15)40474-4.

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8

Limme, Boris, Ramona Nicolescu, and Jean-Paul Misson. "Neonatal Pulmonary Hemosiderosis." Case Reports in Pediatrics 2014 (2014): 1–3. http://dx.doi.org/10.1155/2014/463973.

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Idiopathic pulmonary hemosiderosis (IPH) is a rare complex entity characterized clinically by acute or recurrent episodes of hemoptysis secondary to diffuse alveolar hemorrhage. The radiographic features are variable, including diffuse alveolar-type infiltrates, and interstitial reticular and micronodular patterns. We describe a 3-week-old infant presenting with hemoptysis and moderate respiratory distress. Idiopathic pulmonary hemosiderosis was the first working diagnosis at the Emergency Department and was confirmed, 2 weeks later, by histological studies (bronchoalveolar lavage). The immunosuppressive therapy by 1 mg/kg/d prednisone was immediately started, the baby returned home on steroid therapy at a dose of 0,5 mg/kg/d. The diagnosis of idiopathic pulmonary hemosiderosis should be evocated at any age, even in the neonate, when the clinical presentation (hemoptysis and abnormal radiological chest images) is strongly suggestive.

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9

Vaysman, Dmitriy, Wilfredo G. Veloira, Allan L. Schuss, and Haesoon Lee. "Idiopathic Pulmonary Hemosiderosis." Pediatric Asthma, Allergy & Immunology 17, no. 2 (July 2004): 151–55. http://dx.doi.org/10.1089/0883187041269904.

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10

DELAGE, JEAN-MARIE. "Essential Pulmonary Hemosiderosis." Acta Medica Scandinavica 145, no. 5 (April 24, 2009): 382–85. http://dx.doi.org/10.1111/j.0954-6820.1953.tb07033.x.

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11

WIESMANN, W., D. WOLVIUS, and M. C. VERLOOP. "Idiopathic Pulmonary Hemosiderosis." Acta Medica Scandinavica 146, no. 5 (April 24, 2009): 341–45. http://dx.doi.org/10.1111/j.0954-6820.1953.tb10248.x.

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12

Poggi, Vincenzo, Andrea Lo Vecchio, Francesco Menna, and Giuseppe Menna. "Idiopathic Pulmonary Hemosiderosis." Journal of Pediatric Hematology/Oncology 33, no. 4 (May 2011): e160-e162. http://dx.doi.org/10.1097/mph.0b013e318212a6df.

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13

COHEN, SCHLOMO. "Idiopathic Pulmonary Hemosiderosis." American Journal of the Medical Sciences 317, no. 1 (January 1999): 67–74. http://dx.doi.org/10.1097/00000441-199901000-00012.

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14

Rego, S. J. "Idiopathic pulmonary hemosiderosis." Indian Journal of Pediatrics 68, no. 7 (July 2001): 689–90. http://dx.doi.org/10.1007/bf02752290.

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15

Dua, Tarun, Jagdish Chandra, Manjula Jain, Susan Mary Passah, and Ashok Kumar Dutta. "Idiopathic pulmonary hemosiderosis." Indian Journal of Pediatrics 67, no. 9 (September 2000): 693–94. http://dx.doi.org/10.1007/bf02762188.

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16

Sánchez Duran, M., D. Gómez Pastrana, and A. Andrés Martín. "Hemosiderosis pulmonar idiopática." Archivos de Bronconeumología 32, no. 5 (May 1996): 260–61. http://dx.doi.org/10.1016/s0300-2896(15)30777-8.

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17

Kasilingam, Sangeeth Kumar, Shylaja Prashanth, Manjunath B. Govindagoudar, Bheemaraya Devaramani, and Chaitra Channarayapatna Swamygowda. "Idiopathic Pulmonary Hemosiderosis in an Adult with Unusual Radiologic Features." Journal of Postgraduate Medicine, Education and Research 48, no. 4 (2014): 199–200. http://dx.doi.org/10.5005/jp-journals-10028-1131.

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ABSTRACT Idiopathic pulmonary hemosiderosis is an exceptionally rare cause of diffuse alveolar hemorrhage that occurs primarily in infants and children. Few cases are reported in adults. Patients usually present with recurrent episodes of hemoptysis, fatigue breathlessness and severe anemia with asymptomatic periods in between. Our patient, who was diagnosed with idiopathic pulmonary hemosiderosis demonstrated diffuse ground glass opacity, focal centrilobular emphysema and multiple sub pleural cysts and few parenchymal cysts during acute episode of hemoptysis. Unusual findings in our case were sub pleural cysts, parenchymal cysts and focal centrilobular emphysema which was not documented in any case reports of idiopathic pulmonary hemosiderosis cases to that of our knowledge. How to cite this article Kasilingam SK, Prashanth S, Govindagoudar MB, Devaramani B, Swamygowda CC. Idiopathic Pulmonary Hemosiderosis in an Adult with Unusual Radiologic Features. J Postgrad Med Edu Res 2014;48(4):199-200.

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18

Keller, Charles, and Claire Langston. "Childhood Idiopathic Pulmonary Hemosiderosis." New England Journal of Medicine 343, no. 11 (September 14, 2000): 781. http://dx.doi.org/10.1056/nejm200009143431105.

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19

Barrera, Ana Madeleine, and Leslie Vargas. "Idiopathic pulmonary hemosiderosis with dendriform pulmonary ossification." Biomédica 36, no. 4 (December 1, 2016): 504. http://dx.doi.org/10.7705/biomedica.v36i4.2968.

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La osificación pulmonar es un hallazgo poco usual, generalmente asintomático, que se reporta como incidental en biopsias de pulmón. Asimismo, la hemosiderosis pulmonar idiopática es una causa poco frecuente de infiltración pulmonar. Se presenta el caso de un hombre de 64 años con síntomas respiratorios crónicos, en quien se detectaron estas dos condiciones en el estudio histopatológico.

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20

Ferrari, Giesela F., José R. Fioretto, Adriano F. R. Alves, and Gicélia S. Brandão. "Idiopathic pulmonary hemosiderosis: case report." Jornal de Pediatria 76, no. 2 (March 15, 2000): 149–52. http://dx.doi.org/10.2223/jped.57.

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21

Abbdallah Fatma, Chermiti Ben, Chtourou Amel, Mahouchi Ridha, Smaïl Olfa, Taktak Sophia, Mezni Faouzi, and Ben Kheder Ali. "Idiopathic pulmonary hemosiderosis in adult." Respiratory Medicine CME 3, no. 4 (2010): 238–40. http://dx.doi.org/10.1016/j.rmedc.2009.10.004.

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22

Ioachimescu, Octavian C., Arthur Kotch, and James K. Stoller. "Idiopathic Pulmonary Hemosiderosis in Adults." Clinical Pulmonary Medicine 12, no. 1 (January 2005): 16–25. http://dx.doi.org/10.1097/01.cpm.0000151455.26383.84.

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23

Alsukhon, J., A. Leonov, A. Elisa, and G. Koon. "P327 Food-induced pulmonary hemosiderosis." Annals of Allergy, Asthma & Immunology 119, no. 5 (November 2017): S77. http://dx.doi.org/10.1016/j.anai.2017.08.218.

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24

Pacheco Galván, A., and L. Fogué Calvo. "Diagnóstico de hemosiderosis pulmonar idiopática." Archivos de Bronconeumología 36, no. 8 (September 2000): 489–90. http://dx.doi.org/10.1016/s0300-2896(15)30132-0.

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25

Tsymbalista, O. L. "Intra-Alveolar Hemorrhage in Certain Orphan Lung Diseases in Children (Lecture)." Modern pediatrics. Ukraine, no. 7(111) (November 29, 2020): 72–80. http://dx.doi.org/10.15574/sp.2020.111.72.

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The theme is relevant due to the diagnostic difficulties, severe clinical course and prognosis of idiopathic pulmonary hemosiderosis and Goodpasture syndrome. Idiopathic pulmonary hemosiderosis and Goodpasture syndrome are severe, life-threatening immunopathologic diseases due to alveolar hemorrhage and a hundred percent mortality within a short period of time after the onset of clinical manifestations. Idiopathic pulmonary hemosiderosis generally occurs in children at the age of 3–8 years as a separate condition, or as a stage of Goodpasture syndrome. It manifests itself as shortness of breath, pneumonia, prune juice sputum, hemoptysis, hemorrhage. During exacerbation, the patients' condition is determined by the degree of pulmonary hemorrhage, pulmonary heart disease, acute posthemorrhagic anemia. The exacerbation lasts from a few hours to 1–2 weeks. The duration of each episode and remission varies among patients being unpredictable. Each new exacerbation is more severe. In Goodpasture syndrome, predominant pulmonary and renal vascular lesions of autoimmune nature are observed. It affects young males more frequently; is rare in children. Hemorrhagic alveolitis as a form of lung damage develops first; then, the kidneys are involved, and anemia occurs. Glomerulonephritis (GN) manifests itself as nephrotic syndrome with rapid progression of kidney failure. In case of the predominant pulmonary pathology, recurrent hemoptysis and pulmonary hemorrhage are observed; in end-stage disease with cardiopulmonary failure manifestations, rapidly progressive GN and kidney failure develop. The second variant of Goodpasture syndrome is characterized by relatively slow progression of pulmonary changes and renal lesions. Goodpasture syndrome is rarely accompanied by GN from the onset to the end of the disease and pulmonary pathology manifests itself at the terminal phase of the disease. The treatment of both diseases includes lifetime therapy with glucocorticoids, cytostatics. Pulse therapy using these preparations, discrete plasma exchange and intravenous immunoglobulin administration, syndromic treatment are carried out. No conflict of interest was declared by the authors. Keywords: children, idiopathic pulmonary hemosiderosis, Goodpasture syndrome, clinical course, therapy.

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26

Shiba, T., Y. Nakamura, H. Tanaka, S. Yoshimura, M. Okada, N. Mild, T. Yamanaka, M. Matsumura, and M. Nambu. "Idiopathic pulmonary hemosiderosis is susceptible to pulmonary zygomycosis." Paediatric Respiratory Reviews 12 (June 2011): S74. http://dx.doi.org/10.1016/s1526-0542(11)70082-3.

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27

Takemura, Masao, Hiroatsu Agata, Naomi Kondo, Osamu Fukutomi, Hideaki Tashita, Yuki Kobayashi, Shinji Shinoda, Takashi Nishida, Mitsuyoshi Shinbara, and Tadao Orii. "Pulmonary Hemosiderosis with Hypersensitivity to Buckwheat." Annals of Allergy, Asthma & Immunology 78, no. 2 (February 1997): 233–37. http://dx.doi.org/10.1016/s1081-1206(10)63394-7.

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28

Yaprak, Işın, Sezin Aşık, Berrak Sarıoğlu, Savaş Kansoy, Cengiz Sert, and Hürriyet Turgut. "A Child With Idiopathic Pulmonary Hemosiderosis." Journal of Tepecik Education and Research Hospital 3, no. 1 (1993): 73–76. http://dx.doi.org/10.5222/terh.1993.25488.

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29

Fujino, Hisanori, Yoshihiko Takano, Ako Tatsumi, Shinya Hida, and Shinichi Sumimoto. "Helicobacter pylori Associated With Pulmonary Hemosiderosis." Pediatric Infectious Disease Journal 35, no. 1 (January 2016): 121. http://dx.doi.org/10.1097/inf.0000000000000943.

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30

Saeed, Muhammad M., Marlyn S. Woo, Eithne F. Mac Laughlin, Monique F. Margetis, and Thomas G. Keens. "Prognosis in Pediatric Idiopathic Pulmonary Hemosiderosis." Chest 116, no. 3 (September 1999): 721–25. http://dx.doi.org/10.1378/chest.116.3.721.

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31

Pichardo, Caren, William Muinos, Carole Brathwaite, and Erick Hernandez. "Pulmonary Hemosiderosis Associated With Celiac Disease." Journal of Pediatric Gastroenterology and Nutrition 64, no. 5 (May 2017): e133. http://dx.doi.org/10.1097/mpg.0000000000000833.

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32

Gencer, Mehmet, Erkan Ceylan, Muharrem Bitiren, and Ahmet Koc. "Two Sisters with Idiopathic Pulmonary Hemosiderosis." Canadian Respiratory Journal 14, no. 8 (2007): 490–93. http://dx.doi.org/10.1155/2007/150926.

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Idiopathic pulmonary hemosiderosis (IPH) is a rare cause of diffuse alveolar hemorrhage with unknown etiology. In the present report, the presentations of two sisters are described: one sister had IPH, eosinophilia and a high serum immunoglobulin E (IgE) level; and the other had IPH, pneumothorax, eosinophilia and a high serum IgE level. Both cases had quite unusual presentations. The first patient was 23 years of age, and had suffered from dry cough and progressive dyspnea for four years. Her hemoglobin level was 60 g/L, total serum IgE level was 900 U/mL and eosinophilia was 9%. Her chest radiography revealed diffuse infiltration. She died due to respiratory failure. The second patient was 18 years of age. She had also suffered from dry cough and gradually increasing dyspnea for two years. She had partial pneumothorax in the right lung and diffuse infiltration in other pulmonary fields on chest radiography. Her hemoglobin level was 99 g/L, total serum IgE level was 1200 U/mL and eosinophilia was 8%. IPH was diagnosed by open lung biopsy. All these findings suggested that familial or allergic factors, as well as immunological factors, might have contributed to the etiology of IPH.

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33

Popp, Alina. "Severe Alveolar Hemorrhage – What’s in it for the Gastroenterologist?" Journal of Gastrointestinal and Liver Diseases 25, no. 4 (December 1, 2016): 555–58. http://dx.doi.org/10.15403/jgld.2014.1121.254.cut.

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Background: Alveolar hemorrhage is a potentially life-threatening condition which is usually managed by the pulmonologist. When considering its etiology, there is a rare association that sets the disease into the hands of the gastroenterologist. Case presentation: We report the case of a 48 year-old female who was admitted to the intensive care unit for severe anemia and hemoptysis. On imaging, diffuse pulmonary infiltrates suggestive of alveolar hemorrhage were detected and a diagnosis of pulmonary hemosiderosis was made. She received cortisone therapy and hematologic correction of anemia, with slow recovery. In search of an etiology for the pulmonary hemosiderosis, an extensive workup was done, and celiac disease specific serology was found positive. After confirmation of celiac disease by biopsy, a diagnosis of Lane-Hamilton syndrome was established. The patient was recommended a gluten-free diet and at 6 months follow-up, resolution of anemia and pulmonary infiltrates were observed. Conclusion: Although the association is rare, celiac disease should be considered in a patient with idiopathic pulmonary hemosiderosis. In our case, severe anemia and alveolar infiltrates markedly improved with glucocorticoids and gluten-free diet. Abbreviations: APTT: activated partial thromboplastin time; BAL: bronchoalveolar lavage; CD: celiac disease; Cd: crypt depth; GFD: gluten-free diet; GI: gastrointestinal; IEL: intraepithelial lymphocyte; INR: international normalized ratio; IPH: idiopathic pu

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34

Saha, Biplab K. "Is It Time to Call Idiopathic Pulmonary Hemosiderosis by the Correct Name: Immune-Mediated Pulmonary Hemosiderosis?" American Journal of the Medical Sciences 361, no. 6 (June 2021): 809–11. http://dx.doi.org/10.1016/j.amjms.2021.01.006.

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35

Karavelikova, Ivanka P. "Place of bronchoscopy in the diagnostics and follow-up of patients with idiopathic pulmonary hemosiderosis." Folia Medica 63, no. 4 (August 31, 2021): 582–85. http://dx.doi.org/10.3897/folmed.63.e56088.

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The idiopathic pulmonary hemosiderosis is a rare, life-threatening condition observed mainly in children and characterized by recurrent episodes of diffuse alveolar hemorrhages. The disease is characterized by the triad of hemoptysis, alveolar infiltrates in chest radiography, and iron-deficiency anemia. The recurrent episodes of alveolar hemorrhage can lead to chronic iron-deficiency anemia and irreversible pulmonary fibrosis; therefore, early diagnosis and treatment are crucial to the outcome of the disease.The idiopathic pulmonary hemosiderosis is a rare, life-threatening condition observed mainly in children and characterized by recurrent episodes of diffuse alveolar hemorrhages. The disease is characterized by the triad of hemoptysis, alveolar infiltrates in chest radiography, and iron-deficiency anemia. The recurrent episodes of alveolar hemorrhage can lead to chronic iron-deficiency anemia and irreversible pulmonary fibrosis; therefore, early diagnosis and treatment are crucial to the outcome of the disease.

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36

Salih, Zeynep N., Afreen Akhter, and Javeed Akhter. "Specificity and Sensitivity of Hemosiderin-Laden Macrophages in Routine Bronchoalveolar Lavage in Children." Archives of Pathology & Laboratory Medicine 130, no. 11 (November 1, 2006): 1684–86. http://dx.doi.org/10.5858/2006-130-1684-sasohm.

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Abstract Context.—The presence of iron or hemosiderin in macrophages obtained in routine bronchoalveolar lavage is considered crucial in the diagnosis of the clinical syndrome of hemosiderosis. However, there do not appear to be any data on the sensitivity and specificity of the finding of hemosiderin-laden macrophages (HLMs) in bronchoalveolar lavage in children. Objective.—To review data from bronchoalveolar lavage studies done in children to correlate the presence of HLMs with pneumonia and hemosiderosis and to determine what proportion of HLMs has the optimal sensitivity and specificity for the diagnosis of hemosiderosis. Design.—One hundred ten bronchoalveolar lavage specimens obtained via flexible bronchoscopy were reviewed retrospectively. The data collected for demographics, indication for the bronchoscopy, diagnosis of pneumonia, anemia, and bronchoscopy and bronchoalveolar lavage findings were compared between patients diagnosed with hemosiderosis and those diagnosed with other diseases. Results.—Six patients were diagnosed with hemosiderosis by clinical findings, lung biopsy, or autopsy. There were no statistical differences in pneumonia (P > .99), anemia (P > .99), or coughing (P = .08) between patients with hemosiderosis and other patients. Hemoptysis was the only symptom that was significantly different between the 2 groups (P = .04). The mean HLM index for patients with hemosiderosis was 56% ± 16.17% and for other patients, 7.5% ± 10.74% (P < .001). A HLM index of 35% gave a sensitivity of 1% and a specificity of .96%. Conclusions.—These results confirm a strong association between HLM index and diagnosis of hemosiderosis in a pediatric population. Availability of this HLM index will result in accurate and timely diagnosis of pulmonary hemosiderosis, which may influence treatment and long-term prognosis.

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37

Gordon, Ilyssa O., Nicole Cipriani, Qudsia Arif, A. Craig Mackinnon, and Aliya N. Husain. "Update in Nonneoplastic Lung Diseases." Archives of Pathology & Laboratory Medicine 133, no. 7 (July 1, 2009): 1096–105. http://dx.doi.org/10.5858/133.7.1096.

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Abstract Context.—Nonneoplastic lung diseases include a wide range of pathologic disorders from asthma to interstitial lung disease to pulmonary hypertension. Recent advances in our understanding of the pathophysiology of many of these disorders may ultimately impact diagnosis, therapy, and prognosis. It is important for the practicing pathologist to be aware of this new information and to understand how it impacts the diagnosis, treatment, and outcome of these diseases. Objective.—To update current progress toward elucidating the pathophysiology of pulmonary alveolar proteinosis, idiopathic pulmonary hemosiderosis, and pulmonary arterial hypertension, as well as to present classification systems for pulmonary hypertension, asthma, and interstitial lung disease and describe how these advances relate to the current practice of pulmonary pathology. Data Sources.—Published literature from PubMed (National Library of Medicine) and primary material from the authors' institution. Conclusions.—Improved understanding of the pathophysiology of pulmonary alveolar proteinosis, pulmonary hypertension, and idiopathic hemosiderosis may impact the role of the surgical pathologist. New markers of disease may need to be assessed by immunohistochemistry or molecular techniques. The classification systems for interstitial lung disease, asthma, and pulmonary hypertension are evolving, and surgical pathologists should consider the clinicopathologic context of their diagnoses of these entities.

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38

Ogo, Naruhiko, Toyoshi Yanagihara, Ryota Nishimura, Hiroshi Mannoji, Reiko Yoneda, Masayasu Hayashi, Ayaka Egashira, Tatsuma Asoh, and Takashige Maeyama. "Pulmonary amyloidosis complicated with pulmonary hemosiderosis, diagnosed with bronchoscopy." Respiratory Medicine Case Reports 33 (2021): 101400. http://dx.doi.org/10.1016/j.rmcr.2021.101400.

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39

Abdul Rasheed Qureshi, Huma Bilal, and Muhammad Sajid. "Giant Pulmonary Chondroid Hamartoma in Welder’s Lung: A Unique Case Report." Journal of Islamabad Medical & Dental College 9, no. 3 (September 29, 2020): 219–24. http://dx.doi.org/10.35787/jimdc.v9i3.584.

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Pulmonary Hamartomas are benign lung tumors, containing cartilage, connective tissue, fat and usually appear as peripheral lesions. They are 2-4 cm in size, frequently asymptomatic, comprising of 5–8% of all solitary pulmonary nodules. The peak incidence occurs in 40-70 years of age with a male preponderance. We describe a unique case of centrally located, giant chondroid hamartoma, in the background of pulmonary hemosiderosis. Patient presented with cough, shortness of breath and left sided chest pain, masquerading as pleural effusion, lung cancer and left sided chest pain mimicking myocardial disease. Chest x-ray raised the suspicion of pleural effusion but ultrasound and CT-scan indicated a benign calcified central mass. A presumptive diagnosis of cartilage-containing benign mass was made on ultrasound-guided percutaneous biopsy, while surgical resection followed by histopathology revealed a chondroid hamartoma. No recurrence was noted on follow-up. Chondroid hamartoma can be large, symptomatic and requires modern modalities for diagnosis while its association with pulmonary hemosiderosis, still requires to be investigated.

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40

Rubin, GD, DK Edwards, MA Reicher, JM Doemeny, and SH Carson. "Diagnosis of pulmonary hemosiderosis by MR imaging." American Journal of Roentgenology 152, no. 3 (March 1989): 573–74. http://dx.doi.org/10.2214/ajr.152.3.573.

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41

Alshehri, M. "Pulmonary Hemosiderosis in An Infant with Psoriasis." Annals of Saudi Medicine 18, no. 6 (November 1998): 539–41. http://dx.doi.org/10.5144/0256-4947.1998.539.

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42

Foglia, Lisa M., Shad H. Deering, Lisa M. Foglia, and Shad H. Deering. "Post-partum exacerbation of idiopathic pulmonary hemosiderosis." Journal of Maternal-Fetal & Neonatal Medicine 21, no. 12 (January 2008): 895–97. http://dx.doi.org/10.1080/14767050802326263.

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43

Chen, Chung-Hua, Hsiao-Bai Yang, Shyh-Ren Chiang, and Po-Chien Wang. "Idiopathic Pulmonary Hemosiderosis: Favorable Response to Corticosteroids." Journal of the Chinese Medical Association 71, no. 8 (August 2008): 421–24. http://dx.doi.org/10.1016/s1726-4901(08)70094-7.

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44

Kayser, Klaus, Monika Plodziszewska, Elzbieta Waitr, Janina Slodkowska, Mithat Altiner, and Hans-Joachim Gabius. "Diffuse Pulmonary Hemosiderosis after Exposure to Pesticides." Respiration 65, no. 3 (1998): 214–18. http://dx.doi.org/10.1159/000029265.

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45

Kurahara, David, Marina Morie, Maya Yamane, Sarah Lam, Wallace Matthews, Keolamau Yee, and Kara Yamamoto. "Pulmonary Hemosiderosis in Children with Bronchopulmonary Dysplasia." Case Reports in Pediatrics 2014 (2014): 1–3. http://dx.doi.org/10.1155/2014/876195.

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We describe a possible association between pulmonary hemosiderosis (PH) and a history of bronchopulmonary dysplasia (BPD). Both patients were born at 28-week gestation and presented with PH at ages 22 months and 6 years, respectively. Both initially presented with cough and tachypnea, and bronchoalveolar lavage showed evidence of hemosiderin-laden macrophages. Initial hemoglobin levels were < 4 g/dL and chest radiographs showed diffuse infiltrates that cleared dramatically within days after initiation of intravenous corticosteroids. In the first case, frank pulmonary blood was observed upon initial intubation, prompting the need for high frequency ventilation, immediate corticosteroids, and antibiotics. The mechanical ventilation wean was made possible by the addition of mycophenolate mofetil (MMF) and hydroxychloroquine. Slow tapering off of medications was accomplished over 6 years. These cases represent a possible correlation between prematurity-associated BPD and PH. We present a review of the literature regarding this possible association. In addition, MMF proved to be life-saving in one of the PH cases, as it has been in pulmonary hemorrhage related to systemic lupus erythematosus. Further studies are warranted to investigate the possible association between PH and prematurity-related BPD, as well as the use of MMF in the treatment of PH.

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46

Watanabe, Hirofumi, Mamoru Ayusawa, Masataka Kato, Ami Chou, Akiko Komori, Yuriko Abe, Masaharu Matsumura, Hiroshi Kamiyama, Hiroyuki Izumi, and Shori Takahashi. "Idiopathic pulmonary hemosiderosis complicated by Down syndrome." Pediatrics International 57, no. 5 (October 2015): 1009–12. http://dx.doi.org/10.1111/ped.12690.

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47

Akyar, Serdar, and Süreyya Özbek. "Computed Tomography Findings in Idiopathic Pulmonary Hemosiderosis." Respiration 60, no. 1 (1993): 63–64. http://dx.doi.org/10.1159/000196175.

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48

Soto, Roy G., and Margarida M. Soares. "Idiopathic pulmonary hemosiderosis in pregnancy: anesthetic implications." Journal of Clinical Anesthesia 17, no. 6 (September 2005): 482–84. http://dx.doi.org/10.1016/j.jclinane.2005.06.002.

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49

Michaeli, J., A. Kornberg, M. Menashe, G. Lugassy, and P. Mogle. "Exacerbation of idiopathic pulmonary hemosiderosis in pregnancy." European Journal of Obstetrics & Gynecology and Reproductive Biology 25, no. 2 (June 1987): 153–58. http://dx.doi.org/10.1016/0028-2243(87)90119-5.

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50

Seay, Brandon, Robert Smith, Silvia Delgado-Villalta, and Mutasim Abu-Hasan. "Pulmonary Hemosiderosis and Fibrosis in Trisomy 21." Chest 150, no. 4 (October 2016): 956A. http://dx.doi.org/10.1016/j.chest.2016.08.1059.

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