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Journal articles on the topic 'Hendra'

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Published: 4 June 2021
Last updated: 7 February 2022

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1

Young, Jeannette R., Christine E. Selvey, and Rick Symons. "Hendra virus." Medical Journal of Australia 195, no. 5 (September 2011): 250–51. http://dx.doi.org/10.5694/mja11.10967.

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2

Middleton, Deborah. "Hendra Virus." Veterinary Clinics of North America: Equine Practice 30, no. 3 (December 2014): 579–89. http://dx.doi.org/10.1016/j.cveq.2014.08.004.

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3

BARCLAY, A. J., and D. J. PATON. "Hendra (Equine Morbillivirus)." Veterinary Journal 160, no. 3 (November 2000): 169–76. http://dx.doi.org/10.1053/tvjl.2000.0508.

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4

Meulendyke, Kelly Ann, Mark Allen Wurth, Richard O. McCann, and Rebecca Ellis Dutch. "Endocytosis Plays a Critical Role in Proteolytic Processing of the Hendra Virus Fusion Protein." Journal of Virology 79, no. 20 (October 15, 2005): 12643–49. http://dx.doi.org/10.1128/jvi.79.20.12643-12649.2005.

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ABSTRACT The Hendra virus fusion (F) protein is synthesized as a precursor protein, F0, which is proteolytically processed to the mature form, F1+F2. Unlike the case for the majority of paramyxovirus F proteins, the processing event is furin independent, does not require the addition of exogenous proteases, is not affected by reductions in intracellular Ca2+, and is strongly affected by conditions that raise the intracellular pH (C. T. Pager, M. A. Wurth, and R. E. Dutch, J. Virol. 78:9154-9163, 2004). The Hendra virus F protein cytoplasmic tail contains a consensus motif for endocytosis, YXXΦ. To analyze the potential role of endocytosis in the processing and membrane fusion promotion of the Hendra virus F protein, mutation of tyrosine 525 to alanine (Hendra virus F Y525A) or phenylalanine (Hendra virus F Y525F) was performed. The rate of endocytosis of Hendra virus F Y525A was significantly reduced compared to that of the wild-type (wt) F protein, confirming the functional importance of the endocytosis motif. An intermediate level of endocytosis was observed for Hendra virus F Y525F. Surprisingly, dramatic reductions in the rate of proteolytic processing were observed for Hendra virus F Y525A, although initial transport to the cell surface was not affected. The levels of surface expression for both Hendra virus F Y525A and Hendra virus F Y525F were higher than that of the wt protein, and these mutants displayed enhanced syncytium formation. These results suggest that endocytosis is critically important for Hendra virus F protein cleavage, representing a new paradigm for proteolytic processing of paramyxovirus F proteins.
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5

Pager, Cara Theresia, Mark Allen Wurth, and Rebecca Ellis Dutch. "Subcellular Localization and Calcium and pH Requirements for Proteolytic Processing of the Hendra Virus Fusion Protein." Journal of Virology 78, no. 17 (September 1, 2004): 9154–63. http://dx.doi.org/10.1128/jvi.78.17.9154-9163.2004.

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ABSTRACT Proteolytic cleavage of the Hendra virus fusion (F) protein results in the formation of disulfide-linked F1 and F2 subunits, with cleavage occurring after residue K109 in the sequence GDVK↓L. This unusual cleavage site and efficient propagation of Hendra virus in a furin-deficient cell line indicate that the Hendra F protein is not cleaved by furin, the protease responsible for proteolytic activation of many viral fusion proteins. To identify the subcellular site of Hendra F processing, Vero cells transfected with pCAGGS-Hendra F or pCAGGS-SV5 F were metabolically labeled and chased in the absence and presence of inhibitors of exocytosis. The addition of carbonyl-cyanide-3-chlorophenylhydrazone, monensin, brefeldin A, or NaF-AlCl3 or incubation of cells at 20°C all inhibited processing of the Hendra F protein, suggesting that cleavage of Hendra F occurs either in secretory vesicles budding from the trans-Golgi network or at the cell surface. In contrast to proteolytic cleavage of the simian virus 5 (SV5) F protein by the Ca2+-dependent protease furin, proteolytic cleavage of the Hendra F protein was not significantly inhibited by decreases in Ca2+ levels following incubation with EGTA or A23187. However, in the presence of weak amines and H+ V-ATPase inhibitors, known to raise intracellular pH, cleavage of Hendra F protein was inhibited while processing of the SV5 F protein was not significantly affected. The subcellular location, sensitivity to pH changes, and decreased Ca2+ requirement suggest that the protease responsible for cleavage of Hendra F protein differs from proteases previously shown to be involved in the processing of other viral glycoproteins.
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6

Field, Hume. "Hendra virus re-visited." Virologica Sinica 24, no. 2 (April 2009): 105–9. http://dx.doi.org/10.1007/s12250-009-3034-3.

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7

Quealy-Gainer, Kate. "Cake by Sue Hendra." Bulletin of the Center for Children's Books 72, no. 6 (2019): 256–57. http://dx.doi.org/10.1353/bcc.2019.0101.

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8

Berglind, Natalie. "Supertato by Sue Hendra." Bulletin of the Center for Children's Books 73, no. 11 (2020): 477. http://dx.doi.org/10.1353/bcc.2020.0463.

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9

PÁEZ, D. J., J. GILES, H. MCCALLUM, H. FIELD, D. JORDAN, A. J. PEEL, and R. K. PLOWRIGHT. "Conditions affecting the timing and magnitude of Hendra virus shedding across pteropodid bat populations in Australia." Epidemiology and Infection 145, no. 15 (September 25, 2017): 3143–53. http://dx.doi.org/10.1017/s0950268817002138.

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SUMMARYUnderstanding infection dynamics in animal hosts is fundamental to managing spillover and emergence of zoonotic infections. Hendra virus is endemic in Australian pteropodid bat populations and can be lethal to horses and humans. However, we know little about the factors driving Hendra virus prevalence in resevoir bat populations, making spillover difficult to predict. We use Hendra virus prevalence data collected from 13 000 pooled bat urine samples across space and time to determine if pulses of prevalence are periodic and synchronized across sites. We also test whether site-specific precipitation and temperature affect the amplitude of the largest annual prevalence pulses. We found little evidence for a periodic signal in Hendra virus prevalence. Although the largest amplitude pulses tended to occur over winter, pulses could also occur in other seasons. We found that Hendra virus prevalence was weakly synchronized across sites over short distances, suggesting that prevalence is driven by local-scale effects. Finally, we found that drier conditions in previous seasons and the abundance of Pteropus alecto were positively correlated with the peak annual values of Hendra virus prevalence. Our results suggest that in addition to seasonal effects, bat density and local climatic conditions interact to drive Hendra virus infection dynamics.
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10

M. Rafiek, M. Rafiek. "PEMASYARAKATAN BAHASA INDONESIA MELALUI MADIHIN BANJAR JHON TRALALA DAN HENDRA SEBAGAI UPAYA MEMPERERAT PERSATUAN BANGSA INDONESIA (THE SOCIALIZATION OF INDONESIAN LANGUAGE THROUGH MADIHIN BANJAR BY JHON TRALALA AND HENDRA AS EFFORTS TO STRENGTHEN NATIONAL UNITY OF INDONESIA)." JURNAL BAHASA, SASTRA DAN PEMBELAJARANNYA 3, no. 2 (March 1, 2018): 184. http://dx.doi.org/10.20527/jbsp.v3i2.4552.

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AbstractThe Socialization of Indonesian Language through Madihin Banjar by Jhon Tralalaand Hendra as Efforts to Strengthen National Unity of Indonesia.Madihin Banjar hostedby Jhon Tralala quite known in Indonesia. Jhon Tralala and Hendra, his son is able toadjust the language of poetry or rhyme madihin that brought the audience in attendancein front of him. When many people Banjar, of language poetry or rhymes that broughtmadihin Banjar language. However, when appearing before the lot is not the Banjar,Jhon Tralala and Hendra, Indonesian children will use in its presentation. Things likethis that make madihin Banjar Jhon Tralala and Hendra, his son is able to be acceptedby the people of Indonesia. Through poems or rhymes that speak Indonesian madihinJhon Tralala and Hendra, his son is able to disseminate ideas, ideas, and messages tostrengthen national unity of Indonesia.Keywords: madihin, indonesian, unifyingAbstrakPemasyarakatan Bahasa Indonesia melalui Madihin Banjar Jhon Tralala dan Hendrasebagai Upaya Mempererat Persatuan Bangsa Indonesia. Madihin Banjar yangdibawakan oleh Jhon Tralala cukup dikenal di Indonesia. Jhon Tralala dan Hendra,anaknya mampu menyesuaikan bahasa syair atau pantun madihin yang dibawakannyadengan khalayak yang hadir di hadapannya. Bila banyak orang Banjar, tentu bahasasyair atau pantun madihin yang dibawakannya menggunakan bahasa Banjar. Akantetapi bila yang hadir di hadapan lebih banyak bukan orang Banjar, Jhon Tralala danHendra, anaknya akan menggunakan bahasa Indonesia dalam penyajiannya. Halseperti inilah yang membuat madihin Banjar Jhon Tralala dan Hendra, anaknya mampuditerima oleh masyarakat Indonesia. Melalui syair atau pantun madihin berbahasaIndonesia itulah Jhon Tralala dan Hendra, anaknya mampu menyebarluaskan ide,gagasan, dan pesan-pesannya untuk mempererat persatuan dan kesatuan bangsaIndonesia.Kata-kata kunci: madihin, bahasa indonesia, pemersatu bangsa
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11

Pager, Cara Theresia, and Rebecca Ellis Dutch. "Cathepsin L Is Involved in Proteolytic Processing of the Hendra Virus Fusion Protein." Journal of Virology 79, no. 20 (October 15, 2005): 12714–20. http://dx.doi.org/10.1128/jvi.79.20.12714-12720.2005.

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ABSTRACT Proteolytic processing of paramyxovirus fusion (F) proteins is essential for the generation of a mature and fusogenic form of the F protein. Although many paramyxovirus F proteins are proteolytically processed by the cellular protease furin at a multibasic cleavage motif, cleavage of the newly emerged Hendra virus F protein occurs by a previously unidentified cellular protease following a single lysine at residue 109. We demonstrate here that the cellular protease cathepsin L is involved in converting the Hendra virus precursor F protein (F0) to the active F1 + F2 disulfide-linked heterodimer. To initially identify the class of protease involved in Hendra virus F protein cleavage, Vero cells transfected with pCAGGS-Hendra F or pCAGGS-SV5 F (known to be proteolytically processed by furin) were metabolically labeled and chased in the absence or presence of serine, cysteine, aspartyl, and metalloprotease inhibitors. Nonspecific and specific protease inhibitors known to decrease cathepsin activity inhibited proteolytic processing of Hendra virus F but had no effect on simian virus 5 F processing. We next designed shRNA oligonucleotides to cathepsin L which dramatically reduced cathepsin L protein expression and enzyme activity. Cathepsin L shRNA-expressing Vero cells transfected with pCAGGS-Hendra F demonstrated a nondetectable amount of cleavage of the Hendra virus F protein and significantly decreased membrane fusion activity. Additionally, we found that purified human cathepsin L processed immunopurified Hendra virus F0 into F1 and F2 fragments. These studies introduce a novel mechanism for primary proteolytic processing of viral glycoproteins and also suggest a previously unreported biological role for cathepsin L.
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12

Halpin, K., P. L. Young, H. E. Field, and J. S. Mackenzie. "Isolation of Hendra virus from pteropid bats: a natural reservoir of Hendra virus." Journal of General Virology 81, no. 8 (August 1, 2000): 1927–32. http://dx.doi.org/10.1099/0022-1317-81-8-1927.

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Since it was first described in Australia in 1994, Hendra virus (HeV) has caused two outbreaks of fatal disease in horses and humans, and an isolated fatal horse case. Our preliminary studies revealed a high prevalence of neutralizing antibodies to HeV in bats of the genus Pteropus, but it was unclear whether this was due to infection with HeV or a related virus. We developed the hypothesis that HeV excretion from bats might be related to the birthing process and we targeted the reproductive tract for virus isolation. Three virus isolates were obtained from the uterine fluid and a pool of foetal lung and liver from one grey-headed flying-fox (Pteropus poliocephalus), and from the foetal lung of one black flying-fox (P. alecto). Antigenically, these isolates appeared to be closely related to HeV, returning positive results on immunofluorescent antibody staining and constant-serum varying-virus neutralization tests. Using an HeV-specific oligonucleotide primer pair, genomic sequences of the isolates were amplified. Sequencing of 200 nucleotides in the matrix gene identified that these three isolates were identical to HeV. Isolations were confirmed after RNA extracted from original material was positive for HeV RNA when screened on an HeV Taqman assay. The isolation of HeV from pteropid bats corroborates our earlier serological and epidemiological evidence that they are a natural reservoir host of the virus.
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13

Irawan, Yuda, Uci Rahmalisa, Refni Wahyuni, and Yesica Devis. "Sistem Informasi Penjualan Furniture Berbasis Web Pada CV. Satria Hendra Jaya Pekanbaru." JTIM : Jurnal Teknologi Informasi dan Multimedia 1, no. 2 (August 30, 2019): 150–59. http://dx.doi.org/10.35746/jtim.v1i2.4.

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CV Satria Hendra jaya is a trading company engaged in the sale of furniture fixtures household appliances such as tables, chairs, cabinets, beds etc., where the promotion and sale is still conventional. This means that customers have to go to the CV Satria Hendra jaya to be able to purchase the product. Development of the e-commerce sales in the CV Satria Hendra jaya is an effort to increase sales and promotion of products that can benefit the company. The purpose of building an ecommerce sales of furniture, customers can order the products without having to come into the CV Satria Hendra jaya, the company was able to overcome the problem of product processing, booking processing thus providing convenience to consumers to get information about products CV Satria Hendra jaya. The system is built using PHP and MySQL database program and use the notepad ++ editor. The results of this research is to implement an e-commerce CV Satria Hendra jaya can be used as a sales promotion tool that can be accessed anywhere and anytime, the buying process can be directly without having to come to the store
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14

Whitman, Shannon D., Everett Clinton Smith, and Rebecca Ellis Dutch. "Differential Rates of Protein Folding and Cellular Trafficking for the Hendra Virus F and G Proteins: Implications for F-G Complex Formation." Journal of Virology 83, no. 17 (June 24, 2009): 8998–9001. http://dx.doi.org/10.1128/jvi.00414-09.

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ABSTRACT Hendra virus F protein-promoted membrane fusion requires the presence of the viral attachment protein, G. However, events leading to the association of these glycoproteins remain unclear. Results presented here demonstrate that Hendra virus G undergoes slower secretory pathway trafficking than is observed for Hendra virus F. This slowed trafficking is not dependent on the G protein cytoplasmic tail, the presence of the G receptor ephrin B2, or interaction with other viral proteins. Instead, Hendra virus G was found to undergo intrinsically slow oligomerization within the endoplasmic reticulum. These results suggest that the critical F-G interactions occur only after the initial steps of synthesis and cellular transport.
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15

Whitman, Shannon D., and Rebecca Ellis Dutch. "Surface density of the Hendra G protein modulates Hendra F protein-promoted membrane fusion: Role for Hendra G protein trafficking and degradation." Virology 363, no. 2 (July 2007): 419–29. http://dx.doi.org/10.1016/j.virol.2007.01.029.

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16

Rafiek, M. "Madihin of John Tralala and Hendra: A Study of Presentation, Structure, Form, Value, and Function." Celt: A Journal of Culture, English Language Teaching & Literature 19, no. 2 (May 27, 2021): 257. http://dx.doi.org/10.24167/celt.v19i2.1844.

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Abstract: This study aims to describe and explain about (1) the structure of the presentation of madihin, (2) the forms of madihin, (3) the values in madihin, and (4) the functions in madihin that were performed by pamadihinan John Tralala and Hendra. This study used Parry and Lord and oral literary approach and it was qualitative research type. The oral literary approach was used to study Madihin's oral literature in terms of presentation structure, form, value, and function. As an oral literature, Madihin can be described qualitatively because the data is submitted verbally by the pamadihinan (someone who presents madihin). After that, the madihin data is discussed in accordance with its presentation structure, form, value, and function. The results of this study are (a) the presentation structure consists of opening, installing tabi, presenting the contents (manguran), and closing; (b) the forms of madihin of John Tralala and Hendra are (1) sampiran, (2) question and answer or question and answer sentence, (3) poem, (4) poem of more than 4 lines and broken rhymes, (5) broken rhyming poem, (6) poem of less than 4 lines, and (7) Answers. So, there are 7 forms of Madihin of John Tralala and Hendra; (c) the values in madihin of John Tralala and Hendra are (1) the value of loving art, (2) the value of preservation, (3) the value of working together, (4) the value of art appreciation, (5) the value of hard work, (6) the value of honesty, (7) the value of self-confidence or self-flattering, (8) the value of "dropping", (9) the value of reminding (moral), (10) the value of courage to self-defend or not want to lose, (11) the value of self-defending or not willing to budge, and (12) the value of education. So, there are 12 values in Madihin of John Tralala and Hendra; (d) functions in madihin John Tralala and Hendra are functions of education, advice, and entertainment (humor).Key words: presentation, structure, form, value, function, madihin Abstrak: Penelitian ini bertujuan untuk mendeskripsikan dan menjelaskan tentang (1) struktur penyajian madihin yang dibawakan oleh pamadihinan John Tralala dan Hendra, (2) bentuk-bentuk madihin yang dibawakan oleh pamadihinan John Tralala dan Hendra, (3) nilai-nilai dalam madihin yang dibawakan oleh pamadihinan John Tralala dan Hendra, dan (4) fungsi-fungsi dalam madihin yang dibawakan oleh pamadihinan John Tralala dan Hendra. Penelitian ini menggunakan pendekatan sastra lisan Parry dan Lord dan berjenis penelitian kualitatif. Pendekatan sastra lisan digunakan untuk mengkaji sastra lisan madihin dari segi struktur penyajian, bentuk, nilai, dan fungsi. Sebagai sebuah sastra lisan, madihin dapat diuraikan secara kualitatif karena data disampaikan oleh pamadihinan secara lisan. Data madihin tersebut kemudian dibahas sesuai dengan struktur penyajian, bentuk, nilai, dan fungsinya. Hasil penelitian ini adalah (a) struktur penyajian terdiri atas pembukaan, memasang tabi, menyampaikan isi (manguran), dan penutup; (b) bentuk-bentuk madihin John Tralala dan Hendra adalah (1) sampiran, (2) tanya jawab atau kalimat tanya jawab, (3) syair, (4) syair lebih dari 4 larik dan berima patah, (5) syair berima patah, (6) syair kurang dari 4 larik, dan (7) Jawaban. Jadi, ada 7 bentuk madihin John Tralala dan Hendra; (c) nilai-nilai dalam madihin John Tralala dan Hendra adalah (1) nilai cinta seni, (2) nilai pelestarian, (3) nilai bekerja sama, (4) nilai apresiasi seni, (5) nilai kerja keras, (6) nilai keterusterangan, (7) nilai kepercayaan diri atau menyanjung diri, (8) nilai “menjatuhkan”, (9) nilai mengingatkan (moral), (10) nilai keberanian membela diri atau tidak mau kalah, (11) nilai membela diri atau tidak mau mengalah, dan (12) nilai pendidikan. Jadi, ada 12 nilai dalam madihin John Tralala dan Hendra; (d) fungsi-fungsi dalam madihin John Tralala dan Hendra adalah fungsi pendidikan, nasihat, dan hiburan (humor).Kata kunci: struktur penyajian; bentuk; nilai; fungsi; madihin
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17

WESTBURY, H. A. "Hendra virus disease in horses." Revue Scientifique et Technique de l'OIE 19, no. 1 (April 1, 2000): 151–59. http://dx.doi.org/10.20506/rst.19.1.1203.

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18

Williamson, K. M., S. Wheeler, J. Kerr, J. Bennett, P. Freeman, J. Kohlhagen, A. J. Peel, et al. "Hendra in the Hunter Valley." One Health 10 (December 2020): 100162. http://dx.doi.org/10.1016/j.onehlt.2020.100162.

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19

Hooper, Peter T., and Mark M. Williamson. "Hendra and Nipah Virus Infections." Veterinary Clinics of North America: Equine Practice 16, no. 3 (December 2000): 597–603. http://dx.doi.org/10.1016/s0749-0739(17)30098-6.

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20

Field, Hume. "Horses, humans and Hendra virus." Microbiology Australia 30, no. 4 (2009): 133. http://dx.doi.org/10.1071/ma09133.

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Hendra virus again demonstrated its zoonotic capacity with the infection of two veterinary clinic staff (one fatally) in an outbreak in a Brisbane equine referral veterinary practice in 2008. Bats are recognised as the natural host of the virus.
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21

Porotto, M., L. Doctor, P. Carta, M. Fornabaio, O. Greengard, G. E. Kellogg, and A. Moscona. "Inhibition of Hendra Virus Fusion." Journal of Virology 80, no. 19 (October 1, 2006): 9837–49. http://dx.doi.org/10.1128/jvi.00736-06.

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ABSTRACT Hendra virus (HeV) is a recently identified paramyxovirus that is fatal in humans and could be used as an agent of bioterrorism. The HeV receptor-binding protein (G) is required in order for the fusion protein (F) to mediate fusion, and analysis of the triggering/activation of HeV F by G should lead to strategies for interfering with this key step in viral entry. HeV F, once triggered by the receptor-bound G, by analogy with other paramyxovirus F proteins, undergoes multistep conformational changes leading to a six-helix bundle (6HB) structure that accomplishes fusion of the viral and cellular membranes. The ectodomain of paramyxovirus F proteins contains two conserved heptad repeat regions (HRN and HRC) near the fusion peptide and the transmembrane domains, respectively. Peptides derived from the HRN and HRC regions of F are proposed to inhibit fusion by preventing F, after the initial triggering step, from forming the 6HB structure that is required for fusion. HeV peptides have previously been found to be effective at inhibiting HeV fusion. However, we found that a human parainfluenza virus 3 F-peptide is more effective at inhibiting HeV fusion than the comparable HeV-derived peptide.
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McCORMACK, J. G. "Hendra, Menangle and Nipah viruses." Australian and New Zealand Journal of Medicine 30, no. 1 (February 2000): 9–10. http://dx.doi.org/10.1111/j.1445-5994.2000.tb01047.x.

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23

Thornley, Mark. "Hendra Virus under the microscope." Australian Veterinary Journal 83, no. 1-2 (January 2005): 2. http://dx.doi.org/10.1111/j.1751-0813.2005.tb12169.x.

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24

Field, Hume E. "Hendra virus ecology and transmission." Current Opinion in Virology 16 (February 2016): 120–25. http://dx.doi.org/10.1016/j.coviro.2016.02.004.

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Mire, C. E., J. B. Geisbert, K. N. Agans, Y. R. Feng, K. A. Fenton, K. N. Bossart, L. Yan, Y. P. Chan, C. C. Broder, and T. W. Geisbert. "A Recombinant Hendra Virus G Glycoprotein Subunit Vaccine Protects Nonhuman Primates against Hendra Virus Challenge." Journal of Virology 88, no. 9 (February 12, 2014): 4624–31. http://dx.doi.org/10.1128/jvi.00005-14.

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26

Hanna, Jeffrey N., William J. McBride, Dianne L. Brookes, Jack Shield, Carmel T. Taylor, Ina L. Smith, Scott B. Craig, and Greg A. Smith. "Hendra virus infection in a veterinarian." Medical Journal of Australia 185, no. 10 (November 2006): 562–64. http://dx.doi.org/10.5694/j.1326-5377.2006.tb00692.x.

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Prociv, Paul. "Hendra virus infection in a veterinarian." Medical Journal of Australia 186, no. 6 (March 2007): 325–26. http://dx.doi.org/10.5694/j.1326-5377.2007.tb00921.x.

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Aljofan, Mohamad. "Hendra and Nipah infection: Emerging paramyxoviruses." Virus Research 177, no. 2 (November 2013): 119–26. http://dx.doi.org/10.1016/j.virusres.2013.08.002.

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King, C. "Call for Hendra virus antibody testing." Australian Veterinary Journal 98, no. 12 (December 2020): 622–23. http://dx.doi.org/10.1111/avj.13025.

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Hess, Isabel M. R., Peter D. Massey, Belinda Walker, Deborah J. Middleton, and Therese M. Wright. "Hendra virus: what do we know?" New South Wales Public Health Bulletin 22, no. 6 (2011): 118. http://dx.doi.org/10.1071/nb10077.

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Hughes, K. "Focus on: Hendra virus in Australia." Veterinary Record 175, no. 21 (November 27, 2014): 533–34. http://dx.doi.org/10.1136/vr.g6836.

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Pallister, Jackie, Deborah Middleton, Lin-Fa Wang, Reuben Klein, Jessica Haining, Rachel Robinson, Manabu Yamada, et al. "A recombinant Hendra virus G glycoprotein-based subunit vaccine protects ferrets from lethal Hendra virus challenge." Vaccine 29, no. 34 (August 2011): 5623–30. http://dx.doi.org/10.1016/j.vaccine.2011.06.015.

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33

Kirkland, Peter D., Melinda Gabor, Ian Poe, Kristie Neale, Kim Chaffey, Deborah S. Finlaison, Xingnian Gu, et al. "Hendra Virus Infection in Dog, Australia, 2013." Emerging Infectious Diseases 21, no. 12 (December 2015): 2182–85. http://dx.doi.org/10.3201/eid2112.151324.

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Eaton, Bryan, Christopher Broder, and Lin-Fa Wang. "Hendra and Nipah Viruses: Pathogenesis and Therapeutics." Current Molecular Medicine 5, no. 8 (December 1, 2005): 805–16. http://dx.doi.org/10.2174/156652405774962308.

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Mahalingam, Suresh, Lara J. Herrero, E. Geoffrey Playford, Kirsten Spann, Belinda Herring, Michael S. Rolph, Deborah Middleton, Bradley McCall, Hume Field, and Lin-Fa Wang. "Hendra virus: an emerging paramyxovirus in Australia." Lancet Infectious Diseases 12, no. 10 (October 2012): 799–807. http://dx.doi.org/10.1016/s1473-3099(12)70158-5.

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Wang, Lin-Fa, Brian H. Harcourt, Meng Yu, Azaibi Tamin, Paul A. Rota, William J. Bellini, and Bryan T. Eaton. "Molecular biology of Hendra and Nipah viruses." Microbes and Infection 3, no. 4 (April 2001): 279–87. http://dx.doi.org/10.1016/s1286-4579(01)01381-8.

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37

Daniels, Peter, Thomas Ksiazek, and Bryan T. Eaton. "Laboratory diagnosis of Nipahand Hendra virus infections." Microbes and Infection 3, no. 4 (April 2001): 289–95. http://dx.doi.org/10.1016/s1286-4579(01)01382-x.

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38

Field, Hume, and Brad McCall. "Hendra virus – a One Health success story." Microbiology Australia 33, no. 4 (2012): 167. http://dx.doi.org/10.1071/ma12167.

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Zoonoses account for 60% of emerging diseases threatening humans. Wildlife are the origin of an increasing proportion of zoonoses over recent decades to a point where they now account for 75% of all zoonoses1. Concurrently and/or consequentially, there has been an increasing recognition of the inter-connectedness of wildlife, livestock and human health, and increasing momentum of an ecosystem-level approach (most commonly termed ‘One Health’) to complex emerging disease scenarios2. This paper describes the evolution and application of such an approach to periodic Hendra virus incidents in horses and humans in Australia.
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Broder, Christopher C., Dawn L. Weir, and Peter A. Reid. "Hendra virus and Nipah virus animal vaccines." Vaccine 34, no. 30 (June 2016): 3525–34. http://dx.doi.org/10.1016/j.vaccine.2016.03.075.

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40

WESTBURY, HARVEY. "Hendra Virus: A Highly Lethal Zoonotic Agent." Veterinary Journal 160, no. 3 (November 2000): 165–66. http://dx.doi.org/10.1053/tvjl.2000.0512.

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41

Amaya, Moushimi, and Christopher C. Broder. "Vaccines to Emerging Viruses: Nipah and Hendra." Annual Review of Virology 7, no. 1 (September 29, 2020): 447–73. http://dx.doi.org/10.1146/annurev-virology-021920-113833.

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Hendra virus (HeV) and Nipah virus (NiV) are bat-borne zoonotic para-myxoviruses identified in the mid- to late 1990s in outbreaks of severe disease in livestock and people in Australia and Malaysia, respectively. HeV repeatedly re-emerges in Australia while NiV continues to cause outbreaks in South Asia (Bangladesh and India), and these viruses have remained transboundary threats. In people and several mammalian species, HeV and NiV infections present as a severe systemic and often fatal neurologic and/or respiratory disease. NiV stands out as a potential pandemic threat because of its associated high case-fatality rates and capacity for human-to-human transmission. The development of effective vaccines, suitable for people and livestock, against HeV and NiV has been a research focus. Here, we review the progress made in NiV and HeV vaccine development, with an emphasis on those approaches that have been tested in established animal challenge models of NiV and HeV infection and disease.
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Smith, Ina, Alice Broos, Carol de Jong, Anne Zeddeman, Craig Smith, Greg Smith, Fred Moore, et al. "Identifying Hendra Virus Diversity in Pteropid Bats." PLoS ONE 6, no. 9 (September 28, 2011): e25275. http://dx.doi.org/10.1371/journal.pone.0025275.

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43

Eaton, Bryan T., Christopher C. Broder, Deborah Middleton, and Lin-Fa Wang. "Hendra and Nipah viruses: different and dangerous." Nature Reviews Microbiology 4, no. 1 (January 2006): 23–35. http://dx.doi.org/10.1038/nrmicro1323.

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Nakka, P., G. J. Amos, N. Saad, and S. Jeavons. "MRI findings in acute Hendra virus meningoencephalitis." Clinical Radiology 67, no. 5 (May 2012): 420–28. http://dx.doi.org/10.1016/j.crad.2011.10.008.

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45

Syukur, Apolonaris Servant, Yunita Anas Sriwulandari, and Artifa Sorraya. "Kepedulian Tokoh pada Novel Mimpi Anak Papua Karya Jackie Ambadar dan Hendra G. Lukito, Kajian Sosiologi Sastra." ALFABETA: Jurnal Bahasa, Sastra, dan Pembelajarannya 4, no. 1 (April 27, 2021): 77–85. http://dx.doi.org/10.33503/alfabeta.v4i1.1285.

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Penelitian ini merupakan penelitian deskriptif dengan menggunakan pendekatan sosiologi sastra. Subjek penelitian ini adalah novel Mimpi Anak Papua karya Jackie Ambadar dan Hendra G Lukito. Penelitian ini difokuskan pada permasalahan yang berkaitan dengan nilai kepedulian tokoh dengan kajian sosiologi sastra. Data dianalisis dengan teknik deskripsi kualitatif.Teknik pengumpulan data yang digunakan dalam ini yaitu teknik baca dan catat. Rumusan masalah dalam penelitian ini adalah bagaimana nilai kepedulian tokoh dalam Novel Mimpi Anak Papua karya Jackie Ambadar dan Hendra G.Lukito, kajian sosiologi sastra?. Tujuan dalam penelitian ini berkorelasi dari rumusan masalah tersebut, yaitu mendeskripsikan nilai kepedulian tokoh dalam novel Mimpi Anak Papua Papua karya Jackie Ambadar dan Hendra G.Lukito, kajian sosiologi sastra. Berdasarkan hasil penelitian dapat ditarik beberapa kesimpulan, nilai kepedulian memiliki empat jenis: 1) nilai kepedulian sosial budaya dalam hubungan manusia, 2) nilai kepedulian diri sendiri dalam hubungan manusia, 3) nilai kepedulian orang lain dalam hubungan sesama manusia, 4) nilai kepedulian terhadap sesama manusia dalam memberi perhatian dan kasih sayang.
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Hendra, Patrick. "THE DISCOVERY OF SERS: an idiosyncratic account from a vibrational spectroscopist." Analyst 141, no. 17 (2016): 4996–99. http://dx.doi.org/10.1039/c6an90055k.

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Petkovic, K., G. Metcalfe, H. Chen, Y. Gao, M. Best, D. Lester, and Y. Zhu. "Rapid detection of Hendra virus antibodies: an integrated device with nanoparticle assay and chaotic micromixing." Lab on a Chip 17, no. 1 (2017): 169–77. http://dx.doi.org/10.1039/c6lc01263a.

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Field, Hume, Kylie Schaaf, Nina Kung, Craig Simon, David Waltisbuhl, Heather Hobert, Frederick Moore, et al. "Hendra Virus Outbreak with Novel Clinical Features, Australia." Emerging Infectious Diseases 16, no. 2 (February 2010): 338–40. http://dx.doi.org/10.3201/eid1602.090780.

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Wong, KT. "Nipah and Hendra viruses: recent advances in pathogenesis." Future Virology 5, no. 2 (March 2010): 129–31. http://dx.doi.org/10.2217/fvl.10.7.

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Foord, Adam J., Deborah Middleton, and Hans G. Heine. "Hendra virus detection using Loop-Mediated Isothermal Amplification." Journal of Virological Methods 181, no. 1 (April 2012): 93–96. http://dx.doi.org/10.1016/j.jviromet.2012.01.020.

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