Relevant bibliographies by topics / Hepatic disease

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Hepatic disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Hepatic disease"

1

KAMATAKI, TETSUYA. "Hepatic disease." Rinsho yakuri/Japanese Journal of Clinical Pharmacology and Therapeutics 22, no. 1 (1991): 383–86. http://dx.doi.org/10.3999/jscpt.22.383.

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Sauerbruch, Tilman, Wolfgang H. Caselmann, and Ulrich Spengler. "Hepatic disease." European Journal of Gastroenterology & Hepatology 11, no. 1 (1999): 41–46. http://dx.doi.org/10.1097/00042737-199901000-00009.

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Taylor, George A. "ACUTE HEPATIC DISEASE." Radiologic Clinics of North America 35, no. 4 (1997): 799–813. http://dx.doi.org/10.1016/s0033-8389(22)00433-x.

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Cataño, Juan Carlos, and Diana Karina Agredo. "Hepatic-pulmonary disease." European Journal of Internal Medicine 64 (June 2019): e5-e6. http://dx.doi.org/10.1016/j.ejim.2018.12.013.

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Al-Quorain, AbdulazizA. "Chronic Hepatic Disease." Saudi Journal of Medicine and Medical Sciences 2, no. 1 (2014): 1. http://dx.doi.org/10.4103/1658-631x.128393.

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Gad, Khaled Hussein, and Nevine Nabil Moustafa. "Hepatic hydatid disease." Egyptian Liver Journal 1, no. 1 (2011): 25–32. http://dx.doi.org/10.1097/01.elx.0000397023.10424.78.

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Newell, Susan M., Barbara A. Selcer, Elisabeth Girard, Gregory D. Roberts, James P. Thompson, and Jay M. Harrison. "Correlations between ultrasonographic findings and specific hepatic diseases in cats: 72 cases (1985-1997)." Journal of the American Veterinary Medical Association 213, no. 1 (1998): 94–98. http://dx.doi.org/10.2460/javma.1998.213.01.94.

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Objective To identify correlations between ultrasonographic findings and specific hepatic diseases in cats. Design Retrospective study. Sample Population Medical records of 72 cats with a histopathologic diagnosis of hepatic disease and diagnostic-quality abdominal ultrasonograms between 1985 and 1997. Procedure Abdominal ultrasonographic findings in 72 cats with histologically confirmed hepatic disease (hepatic lipidosis excluded) were reviewed. Rather than attempt to combine individual ultrasonographic findings with specific hepatic diseases, 2 classification trees were created as models to
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Alemayehu, Brook, Zekewos Demissie, and Eyob Beyene. "Human fascioliasis: A diagnostic challenge in resource limited setting." Ethiopian Medical Journal 61, no. 4 (2023): 391–94. http://dx.doi.org/10.4314/emj.v61i4.11.

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Fascioliasis, a disease caused by liver flukes. It is one of the neglected zoonotic diseases. There are only a few reports of fascioliasis from Ethiopia and no case with predominant extrahepatic presentation has been reported so far. We report a case of young woman, who presented with a two weeks of dry cough and generalized pruritus. Her workup revealed marked eosinophilia and multiple, hypodense, and hypovascular hepatic lesions. An enzymelinked assay became positive for Fasciola hepatica. Fasciola hepatica can present with predominant extra hepatic presentation and should be considered in p
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Goel, Varun, and Premashis Kar. "Hepatic Osteodystrophy." Tropical Gastroenterology 31, no. 2 (2010): 82–86. https://doi.org/10.4103/trog_20103102_82.

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Hepatic Osteodystrophy (HO) is a generic definition for the metabolic bone disease that may occur in individuals with chronic liver disease. Hepatic Osteodystrophy is an important but frequently overlooked complication, seen in chronic liver disease patients. This review article illustrates its significance, various causes and methods to diagnose this complication and recent advances and recommendations to treat Hepatic Osteodystrophy. Two distinct bone metabolic processes, osteoporosis (OP) and osteomalacia (OM) are combined together in various proportions in HO syndromes. It has been describ
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Maeso-Díaz, Raquel, and Jordi Gracia-Sancho. "Aging and Chronic Liver Disease." Seminars in Liver Disease 40, no. 04 (2020): 373–84. http://dx.doi.org/10.1055/s-0040-1715446.

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AbstractAging increases the incidence of chronic liver disease (CLD), worsens its prognosis, and represents the predominant risk factor for its development at all different stages. The hepatic sinusoid, which is fundamental for maintaining liver homeostasis, is composed by hepatocytes, liver sinusoidal endothelial cells, hepatic stellate cells, and hepatic macrophages. During CLD progression, hepatic cells suffer deregulations in their phenotype, which ultimately lead to disease development. The effects of aging on the hepatic sinusoid phenotype and function are not well understood, neverthele
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Dissertations / Theses on the topic "Hepatic disease"

1

Carlisle, K. M. "Hepatic haemodynamics in health and disease." Thesis, University of Bristol, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261289.

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Kawser, Choudhury Ali. "Regulation of secretion of #alpha#-2 macroglobulin by hepatic lipocytes in relation to hepatic fibrosis." Thesis, University of Southampton, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240644.

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Mackinnon, Kathryn Linsey. "Hepatic inactivation of Leu-enkephalin." Thesis, Liverpool John Moores University, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337884.

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Menon, David Krishna. "Hepatic '3'1P magnetic resonance spectroscopy in parenchymal liver disease." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243481.

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Akie, Thomas E. "Regulation of Metabolism by Hepatic OXPHOS: A Dissertation." eScholarship@UMMS, 2015. https://escholarship.umassmed.edu/gsbs_diss/857.

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Non-alcoholic fatty liver disease (NAFLD) is an increasingly prevalent issue in the modern world, predisposing patients to serious pathology such as cirrhosis and hepatocellular carcinoma. Mitochondrial dysfunction, and in particular, diminished hepatic oxidative phosphorylation (OXPHOS) capacity, have been observed in NAFLD livers, which may participate in NAFLD pathogenesis. To examine the role of OXPHOS in NAFLD, we generated a model of enhanced hepatic OXPHOS using mice with liver-specific transgenic expression of LRPPRC, a protein which activates mitochondrial transcription and augments O
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Akie, Thomas E. "Regulation of Metabolism by Hepatic OXPHOS: A Dissertation." eScholarship@UMMS, 2010. http://escholarship.umassmed.edu/gsbs_diss/857.

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Non-alcoholic fatty liver disease (NAFLD) is an increasingly prevalent issue in the modern world, predisposing patients to serious pathology such as cirrhosis and hepatocellular carcinoma. Mitochondrial dysfunction, and in particular, diminished hepatic oxidative phosphorylation (OXPHOS) capacity, have been observed in NAFLD livers, which may participate in NAFLD pathogenesis. To examine the role of OXPHOS in NAFLD, we generated a model of enhanced hepatic OXPHOS using mice with liver-specific transgenic expression of LRPPRC, a protein which activates mitochondrial transcription and augments O
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Johnston, Richard David. "Nutritional modulation of hepatic lipid metabolism in health and disease." Thesis, University of Nottingham, 2012. http://eprints.nottingham.ac.uk/12655/.

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The objective of this thesis was to assess the impact of altering macronutrient intakes on hepatic lipid metabolism. Two separate studies were performed, with liver triglyceride content being the principal outcome of both. In the first study 32 healthy and centrally overweight males were randomised to 2 periods, each of 2 weeks, of either a high fructose or glucose intake in a non-crossover fashion. Isoenergetic status was maintained by providing foodstuffs during the first period, followed by a 6 week washout and then a second period of ad libitum overfeeding. In the second study 55 patients
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Ren, Hui. "REGULATION OF HEPATIC GENE EXPRESSION DURING LIVER DEVELOPMENT AND DISEASE." UKnowledge, 2012. http://uknowledge.uky.edu/microbio_etds/6.

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My first project was to investigate the role of Hepatocyte Nuclear Factor 1 (HNF1) and Nuclear Factor I (NFI) on alpha-fetoprotein (AFP) promoter activity during liver development. AFP is highly expressed in the fetal liver, silenced at birth, and remains at very low levels in the adult liver. A GA substitution located at -119 of the human AFP promoter is associated with hereditary persistence of AFP (HPAFP) expression in the adult liver (Hum Molec Genet, 1993, 2:379). The -120 region harbors overlapping binding sites for HNF1 and NFI. While it has been shown that the GA substitution increas
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Purswani, Sudha. "Characterising hepatic B cell subsets in human chronic liver disease." Thesis, University of Birmingham, 2017. http://etheses.bham.ac.uk//id/eprint/7590/.

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B cells have been proven to have a significant role in liver fibrosis. We postulate that enrichment of B cell subsets in hepatic diseases may implicate this population in liver pathogenesis. When comparing total B cells from human immune and non-immune-mediated liver disease explants, we found an enrichment of CD20+ B cells in PBC. Furthermore, phenotypic characterization of 11 B cell subsets in matched liver and blood highlighted an enriched naïve peripheral population, and activated B cell subsets in livers. Newly identified CD19+CD24-CD38- and CD19+CD24-CD38int B cells were also augmented i
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Ghadieh, Hilda E. "Impaired Hepatic Insulin Clearance Links Fatty Liver Disease to Atherosclerosis." University of Toledo Health Science Campus / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=mco1533216686796754.

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Books on the topic "Hepatic disease"

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Council on Community Health, Hospital, Institutional, and Medical Affairs. Ad Hoc Committee on Protocol Development. Patients with hepatic disease. American Dental Association, 1990.

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Yurdaydın, Cihan, and Hakan Bozkaya. Advances in hepatic encephalopathy & metabolism in liver disease: ISHE 1999. Ankara Univeristy Press, 2000.

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Aguila, Marcia Barbosa. Hepatic insulin resistance and nonalcoholic fatty liver disease. Nova Science, 2011.

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Armstrong, P. Jane. Feline hepatic disease and topical issues in feline nutrition. Veterinary Continuing Education, Texas A&M University, 1990.

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Hanan, Al-Mardini, Record Christopher, University of Newcastle upon Tyne. School of Medicine., and International Symposium on Ammonia (9th : 1996 : Newcastle upon Tyne, England), eds. Advances in hepatic encephalopathy & metabolism in liver disease: ISA 96. Medical Faculty, University of Newcastle upon Tyne, 1997.

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6

K, Rustgi Vinod, ed. Hepatic diseases. W. B. Saunders Co., 1989.

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7

1961-, Berry Elizabeth, and National Co-ordinating Centre for HTA (Great Britain), eds. A Systematic literature review of spiral and electron beam computed tomography: With particular reference to clinical applicatiomns in hepatic lesions, pulmonary embolus and coronary artery disease. Core Research on behalf of the NCCHTA, 1999.

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Tanikawa, Kyuichi, and Takato Ueno, eds. Liver Diseases and Hepatic Sinusoidal Cells. Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-67935-6.

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1932-, Tanikawa Kyuichi, and Ueno T. 1951-, eds. Liver diseases and hepatic sinusoidal cells. Springer, 1999.

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Richard, Heatley, ed. Gastrointestinal and hepatic immunology. Cambridge University Press, 1994.

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Book chapters on the topic "Hepatic disease"

1

Yepes, Juan F. "Hepatic Disease." In The ADA Practical Guide to Patients with Medical Conditions. John Wiley & Sons, Inc., 2015. http://dx.doi.org/10.1002/9781119121039.ch6.

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Kallwitz, Eric, and Zurabi Lominadze. "Hepatic Encephalopathy." In Liver Disease. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-98506-0_14.

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Brunelle, Francis, Danièle Pariente, and Pierre Chaumont. "Hepatic Tumours." In Liver Disease in Children. Springer London, 1994. http://dx.doi.org/10.1007/978-1-4471-3822-8_4.

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Balemans, Wendy, Wim Van Hul, Marian Valko, et al. "Venoocclusive Hepatic Disease." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7150.

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Stravitz, R. Todd. "Hematological Issues in Liver Disease." In Hepatic Critical Care. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66432-3_13.

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Heintz, Kathleen, and Steven M. Hollenberg. "Circulatory Physiology in Liver Disease." In Hepatic Critical Care. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66432-3_2.

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Bergl, Paul, and Jonathon D. Truwit. "Respiratory Physiology in Liver Disease." In Hepatic Critical Care. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66432-3_3.

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Singbartl, Kai. "Renal Physiology in Liver Disease." In Hepatic Critical Care. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66432-3_5.

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DellaVolpe, Jeffrey, Minjee Kim, Thomas P. Bleck, and Ali Al-Khafaji. "Cerebrovascular Physiology in Liver Disease." In Hepatic Critical Care. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66432-3_6.

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Schoeman, Tanya. "Hepatic Lipidosis." In Chronic Disease Management for Small Animals. John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781119201076.ch25.

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Conference papers on the topic "Hepatic disease"

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Al Athamneh, Ahmad, Mahmoud E. Farfoura, Jamal Hashem, and Hakam Alqabani. "A Neural Network-based Predictive Model for Hepatic Disease Detection Against Syrian Refugees in Jordan." In 2025 1st International Conference on Computational Intelligence Approaches and Applications (ICCIAA). IEEE, 2025. https://doi.org/10.1109/icciaa65327.2025.11013229.

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Rapoport, Eliot Alexander, and Karin Drooff. "Improvement in hepatic steatosis but worse lipid profile after alcohol cessation." In Digestive Disease Week 2022, edited by Rachel Giles. Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/1655ac74.

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Yang, Xinxiang, Jinyang Li, Rongrong Zhong, et al. "IDDF2023-ABS-0305 CXCR3+ hepatic natural killer cells promote hepatic ischemia-reperfusion injury in liver transplantation." In Abstracts of the International Digestive Disease Forum (IDDF), Hong Kong, 10–11 June 2023. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2023. http://dx.doi.org/10.1136/gutjnl-2023-iddf.44.

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Shah, Apurva. "IDDF2018-ABS-0042 Overt hepatic encephalopathy in extra hepatic portal venous obstruction with lupus nephritis in young female." In International Digestive Disease Forum (IDDF) 2018, Hong Kong, 9–10 June 2018. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2018. http://dx.doi.org/10.1136/gutjnl-2018-iddfabstracts.196.

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Abdukeyim, Nadiya, Zhihua Liu, and Chuanbo Yan. "CT Images Classification of Hepatic Hydatid Disease by Different Methods." In 2019 6th International Conference on Systems and Informatics (ICSAI). IEEE, 2019. http://dx.doi.org/10.1109/icsai48974.2019.9010119.

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Nalbach, Karsten, Revathi Sekar, AnaJimena Alfaro, et al. "Novel function for endosomal trafficking adaptors in hepatic metabolic disease." In 38. Jahrestagung der Deutsche Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0041-1740752.

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Patel, Vishal Rajendrabhai, Payal Dhanesha, and Parthi Dharmeshkumar Soni. "Machine Learning Applications in Early Detection of Hepatic Steatosis Disease." In 2024 3rd International Conference on Applied Artificial Intelligence and Computing (ICAAIC). IEEE, 2024. http://dx.doi.org/10.1109/icaaic60222.2024.10575821.

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Mostardeiro, Thomaz, and Pilar Bayona-Molano. "Portal Vein Stenosis Causing Recurrent Ascites in a Post Hepatic Transplantation Patient." In Abstracts of 5th Annual Meeting of the American Society of Digestive Disease Interventions. Thieme Medical Publishers, 2019. http://dx.doi.org/10.1055/s-0039-1689024.

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Partovi, Sasan, Alice Yu, Ziang Lu, et al. "Technical Success Rates of Contrast-Enhanced Ultrasound-Guided Biopsy of Hepatic Lesions." In Abstracts of 3rd Annual Meeting of the American Society of Digestive Disease Interventions. Thieme Medical Publishers, 2017. http://dx.doi.org/10.1055/s-0037-1603718.

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Calles, F., R. de Vos, R. del Bono, et al. "HEREDITARY HYPOFIBRINOGENEMIA WITH HEPATIC STORAGE OF FIBRINOGEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642884.

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An Italian family suffering from a novel hereditary disease, i.e. hypofihrinogenemia connected with hepatic storage of fibrinogen, has been identified. A similar disorder has previously been described in two German families. The discovery originated from the finding of a massive hepatic storage of fibrinogen in a liver biopsy specimen from a 65 years old female who during a laparotomy unexpectedly turned out to display cirrhosis. Identification of the stored material as fibrinogen was carried out by specific immunostaining applied on both light and electron-microscopic level. The stored fibrin
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Reports on the topic "Hepatic disease"

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Peters, Allison. Vitamin D Status in Gastrointestinal Disease and Hepatic Disorders. Iowa State University, 2019. http://dx.doi.org/10.31274/cc-20240624-1592.

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Ismaiel, Abdulrahman, Ayman Jaaouani, Daniel-Corneliu Leucuta, Stefan-Lucian Popa, and Dan-Lucian Dumitrascu. The Visceral Adiposity Index in Non-Alcoholic Fatty Liver Disease and Liver Fibrosis — Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.12.0056.

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Review question / Objective: The objective of the study was to compare the mean difference and AUROC of Visceral Adiposity Index (VAI) in NAFLD/NASH/liver fibrosis patients and controls in observational studies. Condition being studied: Nonalcoholic fatty liver disease (NAFLD) is a multi-system disease, being mainly a liver pathology involving excessive hepatic fat accumulation unrelated to alcohol consumption or other secondary causes of hepatic steatosis. It is an emerging cause of concern and increasing clinical burden, imposing a public health challenge. NAFLD is the most common chronic li
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Ismaiel, Abdulrahman, Oana Ciobanu, Mohamed Ismaiel, et al. Atherogenic Index of Plasma in Non-Alcoholic Fatty Liver Disease: Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0043.

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Review question / Objective: P - Non-alcoholic fatty liver disease (NAFLD) I - Atherogenic index of plasma (AIP) C - Imaging and histopathology O - Mean difference and Area Under the Curve S - Observational studies. Condition being studied: Non-alcoholic fatty liver disease (NAFLD), is a common liver disease characterized by the presence of excessive fat build up within hepatocytes, in the absence of other conditions that result in hepatic steatosis and with little to no alcohol consumption. It refers to a broad range of conditions including steatosis, non-alcoholic steatohepatitis (NASH) and
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Guo, menglan, Qianren Tan, and jingjing Li. Meta-analysis of the value of transient elastography in the diagnosis of hepatic fibrosis staging in fatty liver disease. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.10.0030.

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Sowjanya, Dr Kaniti, Dr Bongu Srinivas, and Dr Metta Lakshmana Rao. A STUDY ON FIBROSCAN COMPARED TO AST TO PLATELET RATIO INDEX(APRI) FOR ASSESSMENT OF LIVER FIBROSIS WITH NONALCOHOLIC FATTY LIVER DISEASE(NAFLD). World Wide Journals, 2023. http://dx.doi.org/10.36106/ijar/1606016.

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Background : Nonalcoholic fatty liver disease (NAFLD) is increasingly recognized as a cause of chronic liver disease, and often results in the devastating outcomes of decompensated liver cirrhosis and hepatocellular carcinoma and is an important feature of metabolic syndromes and insulin resistance. The assessment of liver brosis is essential for predicting the prognosis and outcome of all forms of chronic liver disease. A liver biopsy is the gold standard for the assessment of liver brosis, but it has its limitations, which include life-threatening complications. Alternative methods of non-
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Hu, Yang Yang, Xing Zhang, Yue Luo, and Yadong Wang. Systematic review and Meta analysis of the efficacy and safety of rifaximin in the prevention and treatment of hepatic encephalopathy. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.2.0061.

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Review question / Objective: P：Liver cirrhosis patients with risk factors associated with HE attack;HE patients caused by chronic liver diseases represented by cirrhosis. I: Rifaximin treatment. C: Other drugs or placebo. O：HE incidence; HE improvement; All-cause mortality; Blood ammonia level; PSE index; mental state; NCT-A; NCT-B; Adverse events. Condition being studied: Hepatic encephalopathy(HE) is a neuropsychiatric disorder syndrome based on metabolic disorders, which is caused by severe acute and chronic liver dysfunction or various abnormalities of portosystemic shunt (hereinafter refe
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