Relevant bibliographies by topics / Hepatic Encephalopathy Caused By NAcetylcysteine

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Academic literature on the topic 'Hepatic Encephalopathy Caused By NAcetylcysteine'

Author: Grafiati
Published: 5 June 2025
Last updated: 16 July 2025

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Journal articles on the topic "Hepatic Encephalopathy Caused By NAcetylcysteine"

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Manas, Ranjan Mallick, Ranjan Behera Jyoti, Kumar Meher Alok, Behera Rupashree, and Kumar Giri Rupak. "Continuous Intravenous N- Acetylcysteine in iNon Acetaminophen Acute Liver Failure In Children." International Journal of Current Pharmaceutical Review and Research 15, no. 11 (2023): 448–53. https://doi.org/10.5281/zenodo.11580587.

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AbstractIntroduction: Assessing How Long-Term Intravenous N-Acetylcysteine Infusions Can Improve a Child's NonAcetaminophen Acute Liver Failure (Nai-Alf) Outcomes.Material and Methods: A predesigned proforma is used to conduct this observational research. Nacetylcysteine at a dosage of 150 mg/kg/day in 3 split doses by continuous intravenous infusion for up to 7consecutive days was given to 41 instances with non-acetaminophen caused acute liver failure in the age groupof 3 months to 14 years who came to VIMSAR's OPD & IPD, Burla. From the hospital registry, 37 patientswere chosen that did
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Salama, Fady, Anna Christina Leyson, Malay Shah, and Roberto Galuppo Monticelli. "A Challenging Case of Refractory Hepatic Encephalopathy in a Postliver Retransplant Patient with Thrombosed Portal Vein: A Shunt for a Shunt." Case Reports in Hepatology 2023 (February 25, 2023): 1–3. http://dx.doi.org/10.1155/2023/6765788.

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Hepatic encephalopathy (HE) is a frequent and serious complication of chronic liver disease. The mechanism of hepatic encephalopathy is not entirely clear. Hepatic encephalopathy is defined as brain dysfunction caused by liver insufficiency and/or portal-systemic blood shunting. It manifests as a wide spectrum of neurological or psychiatric abnormalities, ranging from subclinical alterations, detectable only by neuropsychological or neurophysiological assessment, to coma. Liver transplant (LT) is the definitive treatment for refractory hepatic encephalopathy. In this case, we present a challen
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Pisarek, Wojciech. "S100 Beta Protein as a Marker of Hepatic Encephalopathy: A Breakthrough in Diagnostics or a False Trail? Review of the Literature." Postępy Higieny i Medycyny Doświadczalnej 76, no. 1 (2022): 128–31. http://dx.doi.org/10.2478/ahem-2022-0018.

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Abstract Hepatic encephalopathy is a dysfunction of the central nervous system caused by chronic and acute liver disease. The dysfunction presents a wide spectrum of symptoms—from the undetectable in a standard clinical examination to hepatic coma—and could be caused by both chronic and acute liver diseases. For many years research has been conducted to find a marker that would allow for the accurate, quick, and possibly inexpensive detection of hepatic encephalopathy. Due to the pathogenesis of hepatic encephalopathy, researchers’ attention is focused on markers of damage to the central nervo
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Komiljonovich, Ibragimov Nematjon, Murotov Temur Malik Nizomovich, and Igamkulov Behzod Zafar O’g’li. "HEPATIC ENCEPHALOPATHY CAUSED BY ENDOGENOUS INTOXICATION IN PATIENTS WITH DIABETES MELLITUS COMPLICATED BY PURULENT-SEPTIC LESIONS OF SOFT TISSUES, WAYS OF THEIR CORRECTION." American Journal of Medical Sciences and Pharmaceutical Research 5, no. 11 (2023): 20–29. http://dx.doi.org/10.37547/tajmspr/volume05issue11-04.

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Objective:to study hepatic encephalopathy caused by endogenous intoxication in patients with diabetes mellitus (DM) complicated by necrotic soft tissue lesion (PNSTL) and ways of their correction. Materials and methods:58 patients with diabetes mellitus complicated by purulent-necrotic soft tissue lesion and the effect of endotoxicosis on the functional state of the liver were examined in the intensive care unit, with optimization of correction of hepatic encephalopathy. In addition to traditional therapy, hepatoprotectionsand drugs that stop transaminase activity are included in patients. Con
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Jarmołowicz, Jakub, Agata Boczar, Patryk Dryja, Sven Solisch, Izabela Stawicka, and Izabela Orzołek. "Minimal hepatic encephalopathy - from cirrhosis to neurological disorders." Journal of Education, Health and Sport 76 (December 8, 2024): 56382. https://doi.org/10.12775/jehs.2024.76.56382.

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Introduction and aim Minimal hepatic encephalopathy (MHE) is a subtle but significant brain dysfunction resulting from chronic liver damage, with symptoms that can be difficult to detect. It affects patients' quality of life by causing problems with concentration, memory, and motor coordination. Untreated MHE may progress to more severe neurological disorders, such as overt hepatic encephalopathy. The importance of this condition lies in its impact on patients' daily functioning and the increased risk of accidents, particularly in situations requiring quick responses, such as driving.Neurologi
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Ridola, Lorenzo, Jessica Faccioli, Silvia Nardelli, Stefania Gioia, and Oliviero Riggio. "Hepatic encephalopathy: Diagnosis and management." Journal of Translational Internal Medicine 8, no. 4 (2020): 210–19. http://dx.doi.org/10.2478/jtim-2020-0034.

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Abstract Type C hepatic encephalopathy (HE) is a brain dysfunction caused by severe hepatocellular failure or presence of portal-systemic shunts in patients with liver cirrhosis. In its subclinical form, called “minimal hepatic encephalopathy (MHE), only psychometric tests or electrophysiological evaluation can reveal alterations in attention, working memory, psychomotor speed and visuospatial ability, while clinical neurological signs are lacking. The term “covert” (CHE) has been recently used to unify MHE and Grade I HE in order to refer to a condition that is not unapparent but also non ove
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Ishii, Yuji, Yoshinori Inagaki, Katuya Hirai, and Teruaki Aoki. "Hepatic encephalopathy caused by congenital extrahepatic portosystemic venous shunt." Journal of Hepato-Biliary-Pancreatic Surgery 7, no. 5 (2000): 524–28. http://dx.doi.org/10.1007/s005340070026.

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Shah, Ruchit N., Michael Makar, Nasir Akhtar, and Erin Forster. "Lactulose to the Rescue: A Case of Toxic Hepatic Encephalopathy Caused by Portosystemic Shunting and Epistaxis in a Patient with Hereditary Hemorrhagic Telangiectasia." Case Reports in Hepatology 2019 (March 26, 2019): 1–4. http://dx.doi.org/10.1155/2019/7573408.

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Hereditary hemorrhagic telangiectasia (HHT) is an uncommon autosomal dominant disorder characterized by telangiectasias and arteriovenous malformations. Multiple organ systems are involved including the skin, lungs, gastrointestinal tract, and brain. Hepatic encephalopathy is an extremely rare complication of HHT and early diagnosis and treatment can be life-saving. We present a rare case of hepatic encephalopathy caused by HHT-induced portosystemic shunting treated with lactulose.
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Amodio, Piero, and Sara Montagnese. "Lights and Shadows in Hepatic Encephalopathy Diagnosis." Journal of Clinical Medicine 10, no. 2 (2021): 341. http://dx.doi.org/10.3390/jcm10020341.

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Hepatic encephalopathy (HE) is a form of brain dysfunction that is caused by liver insufficiency and/or portal-systemic shunting. The exact nature of HE is debated; as such, conflicting uses of the term “HE” may cause inconsistencies in its detection and management. This review highlights the meaning of the term “HE” on the basis of its historical origins and current consensus. It also provides criteria for the diagnosis of the condition based on its phenotypes and risk factors for its occurrence. The procedure for differential diagnosis from other conditions which result in similar phenotypes
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Setiorizaldi, Ilham, Amandianti Arimbi Tedjaningrum, Cindy Grace Panggabean, Enjelina Nangin, Jeffrey Christian Mahardhika, and Chandni P. Daryanani. "Pediatric Dengue Encephalopathy: A Review." Medical Clinical Update 1, no. 1 (2022): 5–7. http://dx.doi.org/10.58376/mcu.v1i1.5.

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Dengue encephalopathy is a very common neurological complication of dengue fever. Dengue encephalopathy or dengue hemorrhagic fever (DHF) with Central Nervous System (CNS) involvement used to be considered a relatively rare condition. However, the number of cases reported in human studies were increasing every year. Many factors caused the encephalopathy dengue. Possible mechanisms are hepatic failure (hepatic encephalopathy), cerebral hypoperfusion (shock), cerebral edema (vascular leakage) electrolyte disturbances, and intracranial hemorrhage due to thrombocytopenia or coagulopathy, which ar
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Book chapters on the topic "Hepatic Encephalopathy Caused By NAcetylcysteine"

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El-Mansoury, Bilal, Omar El Hiba, Abdelaati El Khiat, El Baz Soraia, and Arumugam Radhakrishnan Jayakumar. "Hepatic Encephalopathy as a Gliopathy." In Physiology and Function of Glial Cells in Health and Disease. IGI Global, 2023. http://dx.doi.org/10.4018/978-1-6684-9675-6.ch012.

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Hepatic encephalopathy (HE) is defined as a wide spectrum of neuropsychiatric abnormalities caused by liver dysfunction (acute or chronic) and/or portal-systemic shunting. The histopathologic hallmark of HE is astrocyte swelling following acute liver failure (ALF) or the presence of the so-called Alzheimer type 2 astrocytosis under chronic liver disease. HE can be classified according to the undelaying cause into three types: type A as an essential component of acute liver failure, type B as a consequence of porto-systemic shunts in the absence of liver dysfunction, and type C in patients with liver cirrhosis and porto-systemic bypass. While ammonia, manganese, proinflammatory cytokines, and other precipitating factors play a role in the pathogenesis of HE, the exact mechanisms leading to the development of HE are not fully elucidated. This chapter provides a brief overview of HE with a focus on the mechanisms of AT2A.
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Conference papers on the topic "Hepatic Encephalopathy Caused By NAcetylcysteine"

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Lu, K., B. Görg, M. Zimmermann, HJ Bidmon, D. Häussinger, and AS Reichert. "Hepatic encephalopathy is linked to alterations of autophagic flux in astrocytes caused by hyperammonemia." In 35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0038-1677089.

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Reports on the topic "Hepatic Encephalopathy Caused By NAcetylcysteine"

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Hu, Yang Yang, Xing Zhang, Yue Luo, and Yadong Wang. Systematic review and Meta analysis of the efficacy and safety of rifaximin in the prevention and treatment of hepatic encephalopathy. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.2.0061.

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Review question / Objective: P：Liver cirrhosis patients with risk factors associated with HE attack;HE patients caused by chronic liver diseases represented by cirrhosis. I: Rifaximin treatment. C: Other drugs or placebo. O：HE incidence; HE improvement; All-cause mortality; Blood ammonia level; PSE index; mental state; NCT-A; NCT-B; Adverse events. Condition being studied: Hepatic encephalopathy(HE) is a neuropsychiatric disorder syndrome based on metabolic disorders, which is caused by severe acute and chronic liver dysfunction or various abnormalities of portosystemic shunt (hereinafter refe
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