Academic literature on the topic 'Herpes simplex virus (HSV)'

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Journal articles on the topic "Herpes simplex virus (HSV)"

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Cunningham, Anthony L., Cheryl A. Jones, and Min Kim. "Herpes simplex virus vaccines." Microbiology Australia 32, no. 3 (2011): 123. http://dx.doi.org/10.1071/ma11123.

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Herpes simplex virus (HSV) types 1 and 2 cause herpes labialis and genital herpes respectively, although genital herpes caused by HSV-1 is increasing in adolescence. Adult HSV-1 seroprevalence in western countries is 55% to 80% (80% in Australia) and acquired in two peaks, in infancy and adolescence. HSV-2 seroprevalence is highly variable geographically, reaching 12% in Australian adults but up to 90% in African countries. After initial HSV-1 or 2 infection, asymptomatic shedding occurs in the mouth and genital tract respectively in nearly all infected subjects. Complications of HSV-1 include keratitis and blindness and life-threatening encephalitis. Severe complications of HSV-2 include acute urinary retention, meningitis and neonatal herpes (25% fatality). In addition, prior infection with HSV-2 consistently enhances HIV acquisition three- to fourfold. In immunosuppressed persons, HSV-1 and 2 may cause indolent ulcers, oesophagitis and pneumonia. Ultimately, a childhood vaccine effective against both HSV-1 and 2 disease is needed.
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KAWAGUCHI, Yasushi. "Herpes simplex virus (HSV)." Uirusu 60, no. 2 (2010): 187–96. http://dx.doi.org/10.2222/jsv.60.187.

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Lachmann, Robin. "Herpes simplex virus latency." Expert Reviews in Molecular Medicine 5, no. 29 (December 5, 2003): 1–14. http://dx.doi.org/10.1017/s1462399403006975.

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Herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) are ubiquitous human pathogens. They share with other herpesviruses the ability to establish lifelong latent infection of the host. Periodic reactivation from latency is responsible for most of the clinical disease burden of HSV infection. This review focuses on what we have learned from molecular studies in model systems of HSV latency, and the implications these findings have for treating recurrent HSV disease.
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Whitley, Richard J., and Cecelia Hutto. "Neonatal Herpes Simplex Virus Infections." Pediatrics In Review 7, no. 4 (October 1, 1985): 119–26. http://dx.doi.org/10.1542/pir.7.4.119.

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Infections caused by herpes simplex viruses have been recognized since ancient Roman times, when Herodotus associated mouth ulcers and lip vesicles with fever. However, neonatal herpes simplex infection was not identified as a distinct disease until centuries later. Only 50 years ago, the first written descriptions of neonatal herpes were attributed nearly simultaneously to Hass, who described the histopathologic findings in a fatal case, and to Batingani who described a newborn child with herpes simplex virus (HSV) keratitis. For several decades our understanding of neonatal infections with herpes simplex virus was predicated upon histopathologic descriptions of the disease. These indicated a broad spectrum of involvement in infants. In the mid-1960s, Nahmias and Dowdle demonstrated two antigenic types of herpes simplex virus, HSV-1 and HSV-2. Recognition of these types prompted a rapid series of developments leading to a better characterization of the biochemical and molecular characteristics of the virus. One consequence of these advances has been the development of methods of typing of viruses which have been utilized to define the epidemiology of HSV infections. Herpes simplex viral infections "above-the-belt," primarily of the lip and oropharynx, have been found in most cases to be associated with HSV-1, whereas infections "below-the-belt" are usually caused by HSV-2.
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Singh, Shweta, Nikesh Sinha, and Manoj Kumar Shashank Nand Tiwari. "Disease Burden of Herpes Simplex Virus among Patients Attending Rajendra Institute of Medical Sciences, Ranchi, Jharkhand." International Journal of Current Microbiology and Applied Sciences 11, no. 1 (January 10, 2022): 219–23. http://dx.doi.org/10.20546/ijcmas.2022.1101.026.

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HSV-1 has been associated with oro-labial disease, with most infections occurring during childhood, and HSV-2 with genital disease. Additionally, HSV-2 likely contributes substantially to the spread of HIV infection. To estimate the disease burden of Herpes Simplex Virus among the patients attending Rajendra Institute of Medical Sciences. Samples of clinically suspected cases of herpes simplex virus received in the Department of Microbiology, RIMS, Ranchi, from January 2020 to December 2021 were included in the study. Results: During the study period, a total of 1737 samples from clinically suspected cases of herpes simplex virus were received for testing. Of these 1737 samples, 150 samples tested positive for herpes simplex virus. Of total suspected cases, 935 were males and 802 were females. The global burden of HSV-2 infection is large, causing increased risk of genital ulcer disease, HIV acquisition, and transmission of HSV-2 to partners or neonates. These estimates highlight the critical need for development of vaccines, and other new HSV prevention strategies.
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Kobty, Magidah. "Herpes Simplex Virus: Beyond the Basics." Neonatal Network 34, no. 5 (2015): 279–83. http://dx.doi.org/10.1891/0730-0832.34.5.279.

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AbstractOne of the most common sexually transmitted infections is the herpes simplex virus (HSV) Type 2. Although the incidence of newborn infection is not as common as in adults, approximately 1,500 neonates are diagnosed annually with HSV infection. HSV can be detrimental to the life of a newborn, with morbidity and mortality rates of up to 65 percent. This article addresses the maternal and fetal complications of HSV and the impact of HSV on the newborn along with diagnostic evaluation methods. In addition, treatment options and evidence-based practices regarding HSV are defined. Despite growing technology and medical treatment for early identification of HSV, this virus remains challenging and can deeply impact the life of an infant and his or her family. Early diagnosis, treatment, and intervention of an infant with HSV are crucial to ensure the livelihood of the newborn.
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Simmons, Bryan P., and Michael S. Gelfand. "Herpes Simplex Virus." Infection Control 7, no. 7 (July 1986): 380–83. http://dx.doi.org/10.1017/s0195941700064511.

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Herpes simplex virus (HSV) causes a variety of illnesses in th e community and hospital settings (Table). Nosocomial infections with this virus may result from: 1) reactivation of latent infection, especially in patients whose immune systems are compromised; 2) spread from mother to infant; 3) spread from patients to hospital personnel; 4) spread from hospital personnel to patients; and 5) cross-infection among patients. The latter two possibilities seem to occur infrequently but merit some discussion because of the serious implications of such infections.
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Jackson, Julia O., and Richard Longnecker. "Reevaluating Herpes Simplex Virus Hemifusion." Journal of Virology 84, no. 22 (September 15, 2010): 11814–21. http://dx.doi.org/10.1128/jvi.01615-10.

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ABSTRACT Membrane fusion induced by enveloped viruses proceeds through the actions of viral fusion proteins. Once activated, viral fusion proteins undergo large protein conformational changes to execute membrane fusion. Fusion is thought to proceed through a “hemifusion” intermediate in which the outer membrane leaflets of target and viral membranes mix (lipid mixing) prior to fusion pore formation, enlargement, and completion of fusion. Herpes simplex virus type 1 (HSV-1) requires four glycoproteins—glycoprotein D (gD), glycoprotein B (gB), and a heterodimer of glycoprotein H and L (gH/gL)—to accomplish fusion. gD is primarily thought of as a receptor-binding protein and gB as a fusion protein. The role of gH/gL in fusion has remained enigmatic. Despite experimental evidence that gH/gL may be a fusion protein capable of inducing hemifusion in the absence of gB, the recently solved crystal structure of HSV-2 gH/gL has no structural homology to any known viral fusion protein. We found that in our hands, all HSV entry proteins—gD, gB, and gH/gL—were required to observe lipid mixing in both cell-cell- and virus-cell-based hemifusion assays. To verify that our hemifusion assay was capable of detecting hemifusion, we used glycosylphosphatidylinositol (GPI)-linked hemagglutinin (HA), a variant of the influenza virus fusion protein, HA, known to stall the fusion process before productive fusion pores are formed. Additionally, we found that a mutant carrying an insertion within the short gH cytoplasmic tail, 824L gH, is incapable of executing hemifusion despite normal cell surface expression. Collectively, our findings suggest that HSV gH/gL may not function as a fusion protein and that all HSV entry glycoproteins are required for both hemifusion and fusion. The previously described gH 824L mutation blocks gH/gL function prior to HSV-induced lipid mixing.
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Weston, William L., Sylvia L. Brice, Joy D. Jester, Scott Stockert, J. Clark Huff, and Alfred T. Lane. "Herpes Simplex Virus in Childhood Erythema Multiforme." Pediatrics 89, no. 1 (January 1, 1992): 32–34. http://dx.doi.org/10.1542/peds.89.1.32.

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Although an association between herpes simplex virus (HSV) infection and erythema multiforme (EM) minor has been documented in adults, this has not been reported in the pediatric population. This study assessed the potential role of HSV infection in the pathogenesis of EM minor in children. Erythema multiforme skin lesions from 20 children, aged 1 to 16 years, were examined for the presence of HSV by using the polymerase chain reaction. The children included all fit strict clinical criteria for EM minor. Ten had a clinical history of an antecedent herpes infection ("herpes-associated EM"), and 10 did not ("idiopathic EM"). Herpes simplex virus DNA was detected in skin lesions of 8 of 10 children with herpes-associated EM and in 8 of 10 with idiopathic EM. Control skin biopsies from children with other bullous inflammatory diseases were negative. In addition, no HSV could be detected in a biopsy of normal uninvolved skin of a child in whom HSV was present in lesional skin. In situ hybridization on selected biopsies by means of an HSV-specific riboprobe confirmed the presence of HSV and localized it to the epidermis. It is concluded that HSV is a significant precipitating factor for EM minor in children, as it is in adults, and that clinicians should maintain a high index of suspicion of HSV even in the absence of a known history of herpes infection.
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Shintaku, Masayuki, Yasushi Umehara, Keiko Iwaisako, Masao Tahara, and Yasushi Adachi. "Herpes Simplex Pancreatitis." Archives of Pathology & Laboratory Medicine 127, no. 2 (February 1, 2003): 231–34. http://dx.doi.org/10.5858/2003-127-231-hs.

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Abstract Lesions of the pancreas induced by viral infection have drawn relatively little attention because of their low incidence, and the histopathologic features of viral pancreatitis have not been fully elucidated. We report the autopsy findings of 2 patients, a 59-year-old woman with allergic granulomatous angiitis and a 73-year-old man with invasive pulmonary aspergillosis who had a disseminated visceral herpes simplex virus (HSV) infection. In both cases, the liver was the organ most severely affected by the viral infection. The pancreas showed multiple small foci of hemorrhagic necrosis, which were not accompanied by fat necrosis of the surrounding adipose tissue. Histopathologically, Cowdry type A intranuclear inclusions and a ground-glass appearance of the nuclei were found in many degenerated acinar cells around the necrotic foci. The gross appearance and histopathologic features of HSV pancreatitis were characteristic and, in particular, distinct from those of the more common acute hemorrhagic pancreatitis. Immunohistochemistry using an anti-HSV antibody revealed immunoreactivity in the intranuclear inclusions and ground-glass nuclei, and polymerase chain reaction analysis disclosed that the causative virus in these 2 cases was HSV-1. Herpes simplex virus pancreatitis constitutes a rare, but distinct pathologic entity among a group of acute pancreatitis diseases with diverse etiopathogenesis.
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Dissertations / Theses on the topic "Herpes simplex virus (HSV)"

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Delboy, Mark. "PROTEASOME-DEPENDENT ENTRY OF HERPES SIMPLEX VIRUS." VCU Scholars Compass, 2010. http://scholarscompass.vcu.edu/etd/47.

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Herpes simplex virus entry into cells is a multistep process that engages the host cell machinery. The proteasome is a large, ATP-dependent, multisubunit protease that plays a critical role in the maintenance of cell homeostasis. A battery of assays were used to demonstrate that proteasome inhibitors blocked an early step in herpes simplex virus entry that occurred after capsid penetration into the cytosol but prior to capsid arrival at the nuclear periphery. Proteasome-dependent viral entry was not reliant on host or viral protein synthesis. MG132, a peptide aldehyde that competitively inhibits the degradative activity of the proteasome, had a reversible inhibitory effect on herpes simplex virus capsid transport. Herpes simplex virus can use endocytic or nonendocytic pathways to enter cells. These distinct entry routes were both dependent on proteasome-mediated proteolysis. In addition, herpes simplex virus successfully entered cells in the absence of a functional host ubiquitin-activating enzyme, suggesting that viral entry is ubiquitin independent. Herpes simplex virus immediate-early protein ICP0 is a multifunctional regulator of herpes simplex virus infection. Late in infection ICP0 interacts dynamically with cellular proteasomes. ICP0 has a RING finger domain with E3 ubiquitin ligase activity that is necessary for its IE functions. The fundamental and functional properties of ICP0 that is present in the virion tegument layer have not been well characterized. For these reasons, I sought to characterize tegument ICP0 and determine the role of tegument ICP0 during proteasome-dependent entry of herpes simplex virus. Protein compositions of wild-type and ICP0 null virions were similar, suggesting that the absence of ICP0 does not grossly impair virion assembly. Virions with mutations in the RING finger domain contained greatly reduced levels of tegument ICP0, suggesting that the domain influences the incorporation of ICP0. Virion ICP0 was resistant to removal by detergent and salt and was associated with capsids, features common to inner tegument proteins. ICP0 mutations that resulted in the absence of ICP0 in the tegument layer, allow herpes simplex virus to enter cells independently of the proteasome activity. I propose that proteasomal degradation of virion and/or host proteins is regulated by ICP0 to allow for efficient delivery of incoming herpes simplex virus capsids to the nucleus.
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Wu, Zetang. "Silencing Suppression by Herpes Simplex Virus Type 1." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1213287215.

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Simmonds, Peter. "Detection of antibody responses to infection with herpes simplex virus and human immunodeficiency virus." Thesis, University of Edinburgh, 1988. http://hdl.handle.net/1842/26933.

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Targett-Adams, Paul. "Characterisation of the HSV-1 DNA packaging protein encoded by the UL25 gene." Thesis, University of Glasgow, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368523.

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Yirrell, D. L. "Double infections with HSV in the mouse." Thesis, University of Bristol, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234887.

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Dalrymple, M. A. "The regulation of herpes simplex virus immediate early gene expression." Thesis, University of Glasgow, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.378173.

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Grapes, Matthew Giles Robert. "Analysis of transcriptional activation by the HSV-1 protein VP16 and its EHV-1 homologue." Thesis, Imperial College London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325048.

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Leslie, Jenny. "An investigation into factors that influence the incorporation of proteins into the HSV-1 tegument." Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297029.

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Leddon, Jennifer. "Oncolytic Herpes Simplex Virus Therapy for the Treatment of Pediatric Rhabdomyosarcoma." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1427980753.

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Collins, Terry Cordell. "The effect of HSV-2 infection on the expression of cellular mitochondrial aspartate aminotransferase." Thesis, University of Glasgow, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266539.

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Books on the topic "Herpes simplex virus (HSV)"

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Khodari, Yousif Abdulwahed Mohammad. Quantification of Herpes simplex virus type 1(HSV-1) DNA by the polymerase chain reaction. Manchester: University of Manchester, 1995.

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Herpes simplex virus. London: Springer-Verlag, 1989.

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Rouse, Barry T., ed. Herpes Simplex Virus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77247-4.

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Diefenbach, Russell J., and Cornel Fraefel, eds. Herpes Simplex Virus. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-9814-2.

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Diefenbach, Russell J., and Cornel Fraefel, eds. Herpes Simplex Virus. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0428-0.

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Mindel, Adrian. Herpes Simplex Virus. London: Springer London, 1989. http://dx.doi.org/10.1007/978-1-4471-1683-7.

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Brown, S. Moira, and Alasdair R. MacLean. Herpes Simplex Virus Protocols. New Jersey: Humana Press, 1997. http://dx.doi.org/10.1385/0896033473.

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Tabery, Helen. Herpes Simplex Virus Epithelial Keratitis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-01012-5.

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Herpes simplex. London: Routledge, 1998.

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Media, Springer Science+Business, ed. Herpes simplex virus: Methods and protocols. New York: Humana Press, 2014.

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Book chapters on the topic "Herpes simplex virus (HSV)"

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Jakob, Nurith J., and Michaela Handermann. "Herpes-simplex-Virus (HSV)." In Lexikon der Infektionskrankheiten des Menschen, 388–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_466.

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Orbell, Sheina, Havah Schneider, Sabrina Esbitt, Jeffrey S. Gonzalez, Jeffrey S. Gonzalez, Erica Shreck, Abigail Batchelder, et al. "Herpes Simplex Virus (HSV) Infection." In Encyclopedia of Behavioral Medicine, 961. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_100794.

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Syed, Zeba A., Beeran B. Meghpara, and Christopher J. Rapuano. "Herpes Simplex Virus (HSV) Keratitis." In Infections of the Cornea and Conjunctiva, 187–209. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-8811-2_13.

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Young, Hugh, and Marie Ogilvie. "Herpes simplex virus (HSV-1; HSV-2) (Genital herpes)." In Genitourinary Infections, 184–238. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-017-5080-6_7.

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Leone, Peter A. "Herpes Simplex Virus Infections." In Sexually Transmitted Infections in HIV-Infected Adults and Special Populations, 111–23. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56694-8_6.

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Pourchet, Aldo, Matias E. Melendez, Anna Greco, and Alberto L. Epstein. "Herpes Simplex Virus 1 (HSV-1)-Based Vectors." In Neuromethods, 51–93. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-610-8_4.

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Kefalides, Nicholas A., J. M. Brinker, and Z. Ziaie. "Response of Vascular Cells to Herpes Simplex Virus (HSV) Infection." In Vascular Endothelium, 260–61. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3736-6_38.

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Hadjipanayis, Costas G. "Herpes Simplex Virus 1 (HSV-1) for Glioblastoma Multiforme Therapy." In CNS Cancer, 1105–36. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-553-8_46.

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Melendez, Matias E., Cornel Fraefel, and Alberto L. Epstein. "Herpes Simplex Virus Type 1 (HSV-1)-Derived Amplicon Vectors." In Methods in Molecular Biology, 81–98. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0428-0_6.

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Fraefel, Cornel, and Alberto L. Epstein. "Preparation of Herpes Simplex Virus Type 1 (HSV-1)-Based Amplicon Vectors." In Methods in Molecular Biology, 91–109. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9814-2_5.

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Conference papers on the topic "Herpes simplex virus (HSV)"

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Silva, Bruno Vinícius Diniz e., Brunna Rodrigues de Oliveira, Larissa Silva Magalhães, Kamila Cardoso dos Santos, Livia Melo Vilar, Vanessa Salete de Paula, Karlla Antonieta Amorim Caetano, Sheila Araújo Teles, and Megmar Aparecida dos Santos Carneiro. "Seroepidemiological study of herpes simplex virus type 2 (HSV-2) infection in transgender women in Goiás." In XIII Congresso da Sociedade Brasileira de DST - IX Congresso Brasileiro de AIDS - IV Congresso Latino Americano de IST/HIV/AIDS. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/dst-2177-8264-202133p058.

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Introduction: Herpes simplex virus type 2 (HSV-2) causes lesions in the orolabial and anogenital region that last for a lifetime. Data show that about 491.5 million people live with HSV-2. Objective: The aim of this study was to evaluate the epidemiological profile of HSV-2 infection in a population of transgender women in Goiânia-GO and cities in the interior of the state. Methods: This is a cross-sectional study that estimates the prevalence of HSV-2 in transgender women residing or in transit in the metropolitan region of Goiânia and cities in the interior of the state. The Respondent-Driven Sampling (RDS) method was used for recruitment (sample size), the prevalence of HSV-2 was assessed by enzyme immunoassay. Statistical analyses were performed using the Statistical Package for the Social Science (SPSS). The database was analyzed to generate an adjusted prevalence of the characteristics of the study population. The study was approved by the Ethics Committee of the Universidade Federal de Goiás. Results: The prevalence was 8.2% (95% CI 5.0-12.2) for anti-HSV-2 IgM and 70.0% (95% CI 63.0-77.3) for anti-HSV-2 IgG; the bivariate analysis showed an association between positivity by IgG HSV-2 and age >30 years (p<0.0001), exchange of sex for money/drugs or consumer goods (p=0.002), more than 20 sexual partnerships in the past 7 days (p=0.001), and insertive anal sex (p=0.011); in the multivariate analysis, age ≥30 years (p=0.001) and more than 20 sexual partnerships in the past 7 days (p=0.008) were shown statistically related to HSV-2 infection. Conclusion: The data showed a high seroprevalence of HSV-2 among transgender women in the state of Goiás, indicating the need to develop public policies aimed at sexual education and improve this population’s health conditions.
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Pikman, P., A. C. Campagna, D. Fitelson, S. K. V. Guda, C. Paquette, and R. J. Webster. "Tracheoesophageal Fistula (TEF) as a Complication of Herpes Simplex Virus (HSV) Esophagitis in Immunocompetent Host." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a2225.

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Israel, J. R., and Q. N. Hoang. "Tracheo-Esophageal Fistula as a Complication of Herpes Simplex Virus (HSV) Tracheitis in an Immunocompetent Patient." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3973.

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Johnston, Christine, Amalia Magaret, Michael Stern, M. Huang, Stacy Selke, Keith Jerome, David Koelle, and Anna Wald. "O03.1 Natural history of genital and oral herpes simplex virus-1 (HSV-1) shedding after first episode genital HSV-1 infection." In Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.116.

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Omori, Ryosuke, and Laith J. Abu-Raddad. "P3.196 Sexual network drivers of hiv and herpes simplex virus type 2 (HSV-2) transmission: a comparative mathematical modelling analysis." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.431.

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Coudray, Makella, Abraham Degarege, Anisa Khan, Kavitha Ravi, Vijaya Srinivas, Jeffrey Klausner, Purnima Madhivanan, and Caitlyn Placek. "P095 Age disparity and sociodemographic correlates of herpes simplex virus type 2 (HSV-2) seropositivity in south India." In Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.290.

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Tahboub, M., A. Al-Shyoukh, L. Numan, O. Abughanimeh, M. Younis, and A. Gohar. "A Lethal Combination of Disseminated Herpes Simplex Virus-1 (HSV-1) and Varicella Zoster Virus (VZV) Co-Infection in an Immunosuppressed Adult." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6587.

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AL, Cunningham, M. Kim, Truong NR, Sandgren KJ, Harman AN, Bertram KM, L. Bosnjak, et al. "O06.2 Initial interactions of herpes simplex virus with human skin dendritic cells." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.31.

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Matthias, James, Sonya Du Bernard, Gayle Keller, Julia Schillinger, Thomas Peterman, and Craig Wilson. "P091 Estimating neonatal herpes simplex virus infections using chapman’s capture-recapture method, florida, 2011–2017." In Abstracts for the STI & HIV World Congress (Joint Meeting of the 23rd ISSTDR and 20th IUSTI), July 14–17, 2019, Vancouver, Canada. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/sextrans-2019-sti.286.

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Johnston, Christine, Hyunju Son, Amalia Magaret, Michael Stern, Meei-Li Huang, Stacy Selke, Keith R. Jerome, David M. Koelle, and Anna Wald. "O11.1 Decline in genital shedding in the year after first clinical episode genital herpes simplex virus type 1." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.61.

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Reports on the topic "Herpes simplex virus (HSV)"

1

Ye, Shanli. DNA Sequences Involved in the Regulation of Human c-myc Gene Expression by Herpes Simplex Virus Type 1 (HSV-1). Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.7097.

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Zhang, Xiaoliu. A Fusogenic Oncolytic Herpes Simplex Virus for Therapy of Advanced Ovarian Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2005. http://dx.doi.org/10.21236/ada444233.

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Zhang, Xiaoliu. A Potent Oncolytic Herpes Simplex Virus for Therapy of Advanced Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, July 2005. http://dx.doi.org/10.21236/ada442299.

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Zhang, Xiaoliu. A Fusogenic Oncolytic Herpes Simplex Virus for Therapy of Advanced Ovarian Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2004. http://dx.doi.org/10.21236/ada426841.

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Zhang, Xiaoliu. A Potent Oncolytic Herpes Simplex Virus for the Therapy of Advanced Prostate. Fort Belvoir, VA: Defense Technical Information Center, July 2006. http://dx.doi.org/10.21236/ada459160.

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Zhang, Xiaoliu. A Potent Oncolytic Herpes Simplex Virus for the Therapy of Advanced Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, July 2004. http://dx.doi.org/10.21236/ada429083.

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Bertke, Andrea S. Influence of Herpes Simplex Virus Latency-Associated Transcript (LAT) on the Distribution of Latently Infected Neurons. Fort Belvoir, VA: Defense Technical Information Center, February 2007. http://dx.doi.org/10.21236/ad1013850.

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Lown, Rosemary. Investigations of Factors Affecting the Transcriptional Regulation of Herpes Simplex Virus Type 1 βγ (Leaky-Late) Genes. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6731.

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Liu, Xuehui. Studies on the Role of Cellular Factor, YY1, in Herpes Simplex Virus Type 1 Late Gene Expression. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6732.

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Chen, Shin. The DNA Sequence Required for the Maximal Transactivation of the VP5 Gene of Herpes Simplex Virus Type 1. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.6600.

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