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Books on the topic 'Herpes simplex virus (HSV)'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 May 2022

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1

Khodari, Yousif Abdulwahed Mohammad. Quantification of Herpes simplex virus type 1(HSV-1) DNA by the polymerase chain reaction. Manchester: University of Manchester, 1995.

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2

Herpes simplex virus. London: Springer-Verlag, 1989.

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3

Rouse, Barry T., ed. Herpes Simplex Virus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77247-4.

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4

Diefenbach, Russell J., and Cornel Fraefel, eds. Herpes Simplex Virus. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-9814-2.

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5

Diefenbach, Russell J., and Cornel Fraefel, eds. Herpes Simplex Virus. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0428-0.

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6

Mindel, Adrian. Herpes Simplex Virus. London: Springer London, 1989. http://dx.doi.org/10.1007/978-1-4471-1683-7.

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7

Brown, S. Moira, and Alasdair R. MacLean. Herpes Simplex Virus Protocols. New Jersey: Humana Press, 1997. http://dx.doi.org/10.1385/0896033473.

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8

Tabery, Helen. Herpes Simplex Virus Epithelial Keratitis. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-01012-5.

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9

Herpes simplex. London: Routledge, 1998.

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10

Media, Springer Science+Business, ed. Herpes simplex virus: Methods and protocols. New York: Humana Press, 2014.

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11

Anna, Wald, ed. Managing herpes: Living and loving with HSV. Research Triangle Park, N.C: American Social Health Association, 2007.

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12

Stanberry, Lawrence R. Understanding herpes. Jackson, Miss: University Press of Mississippi, 2006.

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13

Understanding herpes. Jackson: University Press of Mississippi, 1998.

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14

New findings on HSV-induced retinitis in the von Szily model. Boston: Butterworth-Heinemann, 1994.

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15

Lin, Woan-Ru. Possible role of herpes simplex virus type 1 in alzheimer's disease. Manchester: UMIST, 1996.

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16

Akpulonu, Lawson Alozie. Herpes, 20th century epidemic: Authoritative, comprehensive discussion and analysis of herpes simplex virus, prevention, and treatment. Los Angeles, CA (3841 W. Washington Blvd., Los Angeles 90018): Herpes Hotline, Virus Research & Treatment Center, 1985.

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17

Billstrom, Marcella Anne. The interactions of concanavalin A and herpes simplex virus type 1: Application to virus purification. Birmingham: University of Birmingham, 1987.

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18

Tabery, Helena M. Herpes simplex virus epithelial keratitis: In vivo morphology in the human cornea. Heidelberg: Springer, 2010.

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19

Ba-Bair, Yasser Hassan Saleh. Development of in situ PCR to detect herpes simplex virus DNA in cornea. Manchester: University of Manchester, 1996.

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20

Skinner, Gordon R. B. The immunological relatedness, immunogenicity and oncogenicity of herpes simplex virus: Development of a vaccinetowards prevention of herpes genitalis in human subjects. Birmingham: University of Birmingham, 1989.

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21

West, A. Investigations by mass spectrometry of the interactions of novel serine protease inhibitors with Herpes Simplex Virus type 2 and Human Cytomegalovirus proteases. [s.l.]: typescript, 1999.

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22

Current issues in clinical neurovirology: Pathogenesis, diagnosis and treatment. Philadelphia, Pa: Saunders, 2008.

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23

Alyazidi, Raidan, and Soren Gantt. Herpes simplex Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0007.

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Herpes simplex virus (HSV) types 1 and 2 cause several important syndromes, including congenital and perinatal infections that can cause devastating consequences in newborns (i.e., neonatal HSV). Most neonatal HSV infections are acquired intrapartum in the infected maternal birth canal. Since genital HSV infections are common, neonatal HSV is an important complication in infected women, even if maternal symptoms are absent. As a result of the developmental status of the fetal and newborn immune system, neonatal HSV infection is associated with life-threatening disease. This chapter reviews the clinical presentations of neonatal HSV infection, as well as advances in diagnosis and therapy. Skin vesicles and fever are often absent, which contributes to a delay in initiating effective therapy. Early recognition is key. Despite significant advances in diagnostic testing and antiviral treatment for neonatal HSV, morbidity and mortality remain high and no vaccine is currently available for clinical use.

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24

Kness, Ron. Herpes Simplex Virus: Managing HSV Holistically. CreateSpace Independent Publishing Platform, 2018.

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25

Keshav, Satish, and Palak Trivedi. Viral hepatitis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0212.

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Hepatitis means ‘inflammation of the liver’ and is manifest with symptoms that include malaise, anorexia, fever, flu-like symptoms, and pain in the right upper quadrant of the abdomen, with the pain being caused by swelling of the liver and its capsule. Elevations in circulating hepatic enzymes, particularly aspartate transaminase and alanine transaminase, are common, with jaundice occurring some time after the onset of other symptoms and signs. There are five viruses that primarily cause viral hepatitis: hepatitis A, B, C, D, and E viruses, abbreviated HAV, HBV, HCV, HDV, and HEV, respectively. These viruses are all hepatotrophic, in that the liver is the primary site of infection. HAV, HBV, and HEV are usually acute, self-limiting infections that may, nonetheless, cause morbidity and, in the case of HEV, fatality. However, HBV and, more so, HCV can cause chronic carriage of the virus over many years, as well as the development of chronic hepatitis. HDV is only pathogenic in conjunction with HBV. After recovery from acute infection with HAV, individuals have long-lasting immunity against further infection. The same holds true for the majority of individuals with acute HBV infection. There seems to be little natural immunity to HCV infection, and a significant proportion of cases result in chronic hepatitis. Immunity to HEV is not long-lasting, and repeated infections are possible. Many other viruses can cause hepatitis, of which cytomegalovirus, herpes simplex virus, Epstein–Barr virus, and flaviviruses such as dengue and yellow fever are the most important. The liver, however, is not their primary site of replication or cellular damage.

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26

Biloshytsky, Vadym, and Roman Cregg. Pioneering use of gene therapy for pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0083.

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The landmark paper discussed in this chapter is ‘Gene therapy for pain: Results of a Phase I clinical trial’, published by Fink et al. in 2011. In this study, the first of its kind, researchers studied the efficacy and safety of a modified herpes simplex virus (HSV) vector used to deliver PENK, which encodes proenkephalin, which is cleaved into the enkephalin peptides Met-enkephalin and Leu-enkephalin, which induce analgesia by acting on opioid receptors. The development of the HSV vector was based in part on results studies in which adenovirus, adeno-associated virus, or non-viral vectors were used to overexpress genes. Overexpression of a variety of large molecules leads to a reduction in pain-related behaviour in animals. Gene therapy in the treatment of chronic pain seems to offer a promising alternative to systemic or highly invasive therapies. However, additional research is needed to determine the safety, effectiveness, and cost-efficiency of this approach.

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27

DeAugustinas, M., and A. Kiely. Infectious Keratitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0016.

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Keratitis is an inflammation of the cornea, which can lead to corneal opacification or ulceration. The most common cause of infectious keratitis is herpes simplex virus type 1 (HSV-1). Noninfectious corneal infiltrates related to trauma, collagen vascular disease, autoimmune inflammation, vasculitis, or atopy (which predisposes to HSV keratitis) must be considered. HSV-associated stromal keratitis is the most common cause of infectious corneal blindness in the United States, yet its presentation can be fairly subtle. For this reason, symptoms out of proportion to exam findings or a history concerning for viral infection is an indication for prompt referral to ophthalmology. Topical antibiotic drops achieve high tissue concentrations and are the treatment of choice. Empiric coverage should be prescribed and tailored later under the care of an ophthalmologist. Other keys to effective treatment include discontinuing contact lens use and protecting the eye with a rigid shield without a patch, as patching provides a reservoir for infection.

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28

Herpes Simplex Virus. Springer, 2011.

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29

Mindel, Adrian. Herpes Simplex Virus. Springer, 2011.

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30

Moira, Brown S., and MacLean Alasdair R, eds. Herpes simplex virus protocols. Totowa, N.J: Humana Press, 1998.

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31

Marie, Studahl, Cinque Paola, and Bergström T, eds. Herpes simplex viruses. Boca Raton: Taylor & Francis, 2006.

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32

Wald, Charles Ebel and Anna, and MPH. Managing Herpes: Living and Loving With HSV. American Social Health Association, 2007.

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33

Dence, Carlton W., and Barry T. Rouse. Herpes Simplex Virus: Pathogenesis, Immunobiology and Control. Brand: Springer, 2011.

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34

T, Rouse Barry, ed. Herpes simplex virus: Pathogenesis, immunobiology and control. Berlin: Springer-Verlag, 1992.

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35

Bunker, Professor Christopher, and Dr Arani Chandrakumar. Dermatological diseases and emergencies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.00017.

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Chapter 17 covers dermatological diseases and emergencies including a general introduction to the subject, followed by information on erythroderma, drug eruptions, angio-oedema, Kawasaki disease, staphylococcal toxic shock syndrome, Streptococcal toxic shock syndrome (streptococcal TSS), staphylococcal scalded skin syndrome, necrotizing fasciitis, psoriasis, eczema and dermatitis, cutaneous vasculitis, immunobullous disorders, pyoderma gangrenosum, scarring alopecia, herpes simplex viruses 1 and 2, varicella zoster virus infection, bacterial infections affecting the skin, fungal infections affecting the skin, ectoparasitic disease, HIV infection and the skin, malignant melanoma, non-melanoma skin cancer, and cutaneous T cell lymphoma.

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36

Rouse, Barry T. Herpes Simplex Virus (Current Topics in Microbiology & Immunology). Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1992.

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37

Rapp, F. Rapp Herpesvirus. John Wiley & Sons Inc, 1985.

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38

Rouse, Barry T. Herpes Simplex Virus: Pathogenesis, Immunobiology and Control (Current Topics in Microbiology and Immunology). Springer, 1992.

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39

Liu, Fenyong. The human herpes simplex virus I protease and its substrate, the capsid scaffolding protein. 1993.

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40

(Editor), Marie Studahl, Paola Cinque (Editor), and Tomas Bergstrom (Editor), eds. Herpes Simplex Viruses (Infectious Disease and Therapy). Informa Healthcare, 2005.

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41

Lum, Brian. The mechanism of encapsidation of Herpes simplex virus DNA. 1990.

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42

Lydyard, Peter, Michael Cole, John Holton, Will Irving, Nino Porakishvili, Pradhib Venkatesan, and Kate Ward. Case Studies in Infectious Disease: Herpes simplex virus 1. Garland Science, 2009. http://dx.doi.org/10.4324/9780203853863.

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43

Lydyard, Peter, Michael Cole, John Holton, Will Irving, Nino Porakishvili, Pradhib Venkatesan, and Kate Ward. Case Studies in Infectious Disease: Herpes simplex virus 2. Garland Science, 2009. http://dx.doi.org/10.4324/9780203853870.

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44

Hubenthal-Voss, Jeffrey. An analysis of the genes and DNA sequences encoded in short component terminal repeat of herpes simplex virus-1. 1988.

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45

Aspects of the regulation of herpes simplex virus-1 gene transcription by ICP4. 1993.

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46

(Editor), A. A. Gershon, C. H. Calisher (Editor), and A. M. Arvin (Editor), eds. Immunity to and Prevention of Herpes Zoster (Archives of Virology. Supplementa). Springer, 2001.

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47

Dick, Liane Carolyn *. Genetic characterization of a temperature-sensitive mutant of herpes simplex virus type I. 1988.

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48

Struyf, Frank. Genetic Analysis Of Herpesvirus Entry Receptors And Host Susceptibility To Herpes Simplex Virus Infection. Leuven Univ Pr, 2004.

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49

R, Stanberry Lawrence, ed. Genital and neonatal herpes. Chichester: John Wiley, 1996.

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50

K, Wagner Edward, ed. Herpesvirus transcription and its regulation. Boca Raton, Fla: CRC Press, 1991.

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