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Books on the topic 'Herpes simplex virus type 2'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

West, A. Investigations by mass spectrometry of the interactions of novel serine protease inhibitors with Herpes Simplex Virus type 2 and Human Cytomegalovirus proteases. [s.l.]: typescript, 1999.

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2

Diefenbach, Russell J., and Cornel Fraefel, eds. Herpes Simplex Virus. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-9814-2.

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3

Media, Springer Science+Business, ed. Herpes simplex virus: Methods and protocols. New York: Humana Press, 2014.

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4

Lin, Woan-Ru. Possible role of herpes simplex virus type 1 in alzheimer's disease. Manchester: UMIST, 1996.

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5

Billstrom, Marcella Anne. The interactions of concanavalin A and herpes simplex virus type 1: Application to virus purification. Birmingham: University of Birmingham, 1987.

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6

Khodari, Yousif Abdulwahed Mohammad. Quantification of Herpes simplex virus type 1(HSV-1) DNA by the polymerase chain reaction. Manchester: University of Manchester, 1995.

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7

Alyazidi, Raidan, and Soren Gantt. Herpes simplex Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0007.

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Herpes simplex virus (HSV) types 1 and 2 cause several important syndromes, including congenital and perinatal infections that can cause devastating consequences in newborns (i.e., neonatal HSV). Most neonatal HSV infections are acquired intrapartum in the infected maternal birth canal. Since genital HSV infections are common, neonatal HSV is an important complication in infected women, even if maternal symptoms are absent. As a result of the developmental status of the fetal and newborn immune system, neonatal HSV infection is associated with life-threatening disease. This chapter reviews the clinical presentations of neonatal HSV infection, as well as advances in diagnosis and therapy. Skin vesicles and fever are often absent, which contributes to a delay in initiating effective therapy. Early recognition is key. Despite significant advances in diagnostic testing and antiviral treatment for neonatal HSV, morbidity and mortality remain high and no vaccine is currently available for clinical use.

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8

Marie, Studahl, Cinque Paola, and Bergström T, eds. Herpes simplex viruses. Boca Raton: Taylor & Francis, 2006.

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9

Bale, James F. Congenital and Perinatal Viral Infections. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0160.

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Despite remarkable advancements in the treatment and prevention of infectious diseases, congenital (also known as intrauterine) and perinatal (also known as neonatal) infections remain major causes of permanent neurodevelopmental disabilities worldwide. Fortunately, relatively few viral pathogens can infect the developing fetus or the newborn postnatally and induce neurological disease. These pathogens include cytomegalovirus, rubella virus, herpes simplex virus types 1 and 2, varicella zoster virus, lymphocytic choriomeningitis virus, the nonpolio enteroviruses, parechovirus, and human immunodeficiency virus. This chapter describes the clinical manifestations, diagnosis, treatment, and outcome of these congenital and perinatal viral infections.

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10

Lydyard, Peter, Michael Cole, John Holton, Will Irving, Nino Porakishvili, Pradhib Venkatesan, and Kate Ward. Case Studies in Infectious Disease: Herpes simplex virus 2. Garland Science, 2009. http://dx.doi.org/10.4324/9780203853870.

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11

T, Rouse Barry, ed. Herpes simplex virus: Pathogenesis, immunobiology and control. Berlin: Springer-Verlag, 1992.

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12

(Editor), Marie Studahl, Paola Cinque (Editor), and Tomas Bergstrom (Editor), eds. Herpes Simplex Viruses (Infectious Disease and Therapy). Informa Healthcare, 2005.

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13

Rouse, Barry T. Herpes Simplex Virus (Current Topics in Microbiology & Immunology). Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1992.

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14

Dick, Liane Carolyn *. Genetic characterization of a temperature-sensitive mutant of herpes simplex virus type I. 1988.

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15

Arko, Edward Francis *. Genetic characterization of a delayed host shutoff temperature sensitive mutant of herpes simplex virus type I. 1989.

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16

Martin, Ana. Analysis of the interaction between the herpes simplex virus type-1 structural protein VP22 and cellular microtubules. 2001.

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17

Daksis, Jasmine Ilga *. Biochemical and genetic characterization of temperature-sensitive mutants of herpes simplex virus type 1 defective in the shutoff of cellular macromolecular synthesis. 1987.

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18

Bunker, Professor Christopher, and Dr Arani Chandrakumar. Dermatological diseases and emergencies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.00017.

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Chapter 17 covers dermatological diseases and emergencies including a general introduction to the subject, followed by information on erythroderma, drug eruptions, angio-oedema, Kawasaki disease, staphylococcal toxic shock syndrome, Streptococcal toxic shock syndrome (streptococcal TSS), staphylococcal scalded skin syndrome, necrotizing fasciitis, psoriasis, eczema and dermatitis, cutaneous vasculitis, immunobullous disorders, pyoderma gangrenosum, scarring alopecia, herpes simplex viruses 1 and 2, varicella zoster virus infection, bacterial infections affecting the skin, fungal infections affecting the skin, ectoparasitic disease, HIV infection and the skin, malignant melanoma, non-melanoma skin cancer, and cutaneous T cell lymphoma.

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19

DeAugustinas, M., and A. Kiely. Infectious Keratitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0016.

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Keratitis is an inflammation of the cornea, which can lead to corneal opacification or ulceration. The most common cause of infectious keratitis is herpes simplex virus type 1 (HSV-1). Noninfectious corneal infiltrates related to trauma, collagen vascular disease, autoimmune inflammation, vasculitis, or atopy (which predisposes to HSV keratitis) must be considered. HSV-associated stromal keratitis is the most common cause of infectious corneal blindness in the United States, yet its presentation can be fairly subtle. For this reason, symptoms out of proportion to exam findings or a history concerning for viral infection is an indication for prompt referral to ophthalmology. Topical antibiotic drops achieve high tissue concentrations and are the treatment of choice. Empiric coverage should be prescribed and tailored later under the care of an ophthalmologist. Other keys to effective treatment include discontinuing contact lens use and protecting the eye with a rigid shield without a patch, as patching provides a reservoir for infection.

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