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Journal articles on the topic 'Herpes simplex virus type 2'

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Published: 4 June 2021
Last updated: 1 February 2022

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1

Oh, Jang O. "Type 2 Herpes Simplex Virus Infections." Yonsei Medical Journal 27, no. 1 (1986): 1. http://dx.doi.org/10.3349/ymj.1986.27.1.1.

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2

Millichap, J. Gordon. "Herpes Simplex Virus Type 2 Neurologic Complications." Pediatric Neurology Briefs 22, no. 6 (June 1, 2008): 46. http://dx.doi.org/10.15844/pedneurbriefs-22-6-8.

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3

Mills, John. "Testing for Herpes Simplex Virus Type 2." Sexually Transmitted Diseases 27, no. 5 (May 2000): 270–71. http://dx.doi.org/10.1097/00007435-200005000-00006.

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4

Mark, Hayley D., Ashley P. Hanahan, and Stacie C. Stender. "Herpes Simplex Virus Type 2: An Update." Nurse Practitioner 28, no. 11 (November 2003): 34–37. http://dx.doi.org/10.1097/00006205-200311000-00012.

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5

Tobian, Aaron A. R., Godfrey Kigozi, Maria J. Wawer, David Serwadda, Thomas C. Quinn, and Ronald H. Gray. "Herpes simplex virus type-2 assay specificity and male circumcision to reduce herpes simplex virus type-2 acquisition." AIDS 27, no. 1 (January 2013): 147–49. http://dx.doi.org/10.1097/qad.0b013e32835aa181.

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6

Lachmann, Robin. "Herpes simplex virus latency." Expert Reviews in Molecular Medicine 5, no. 29 (December 5, 2003): 1–14. http://dx.doi.org/10.1017/s1462399403006975.

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Herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) are ubiquitous human pathogens. They share with other herpesviruses the ability to establish lifelong latent infection of the host. Periodic reactivation from latency is responsible for most of the clinical disease burden of HSV infection. This review focuses on what we have learned from molecular studies in model systems of HSV latency, and the implications these findings have for treating recurrent HSV disease.
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7

Docherty, John J., Anthony T. Dobson, John J. Trimble, and Beth Ann Jennings. "Herpes Simplex Virus Type 1 That Exhibits Herpes Simplex Virus Type 2 Sensitivity to (E)-5-(2-Bromovinyl)-2′-Deoxyuridine." Intervirology 32, no. 5 (1991): 308–15. http://dx.doi.org/10.1159/000150213.

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8

González Rodríguez, Antonio Javier, Encarnación Montesinos Villaescusa, and Esperanza Jordá Cuevas. "Pityriasis Lichenoides Chronica Associated with Herpes Simplex Virus Type 2." Case Reports in Dermatological Medicine 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/737428.

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Introduction. Pityriasis lichenoides is a rare, acquired spectrum of skin conditions of an unknown etiology.Case Report. A 28-year-old man presented with recurrent outbreaks of herpes simplex virus associated with the onset of red-to-brown maculopapules located predominantly in trunk in each recurrence. Positive serologies to herpes simplex virus type 2 were detected. Histopathological examination of one of the lesions was consistent with a diagnosis of pityriasis lichenoides chronica.Discussion. Pityriasis lichenoides is a rare cutaneous entity of an unknown cause which includes different clinical presentations. A number of infectious agents have been implicated based on the clustering of multiple outbreaks and elevated serum titers to specific pathogens (human immunodeficiency virus, cytomegalovirus, Epstein-Barr virus,Toxoplasma gondii, and herpes simplex virus). In our patient, resolution of cutaneous lesions coincided with the administration of antiviral drugs and clinical improvement in each genital herpes recurrence. In conclusion, we report a case in which cutaneous lesions of pityriasis lichenoides chronica and a herpes simplex virus-type 2-mediated disease have evolved concomitantly.
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9

Mirakhur, B., and M. McKenna. "Recurrent Herpes Simplex Type 2 Virus (Mollaret) Meningitis." Journal of the American Board of Family Medicine 17, no. 4 (July 1, 2004): 303–5. http://dx.doi.org/10.3122/jabfm.17.4.303.

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10

Vinikoor, Michael J., Yu-Tung Wong, and David M. Margolis. "Herpes Simplex Virus Type 2 Mimicking Necrotizing Fasciitis." Annals of Internal Medicine 155, no. 8 (October 18, 2011): 567. http://dx.doi.org/10.7326/0003-4819-155-8-201110180-00030.

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11

Arvin, Ann M., and Charles G. Prober. "Herpes Simplex Virus Type 2 — A Persistent Problem." New England Journal of Medicine 337, no. 16 (October 16, 1997): 1158–59. http://dx.doi.org/10.1056/nejm199710163371609.

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12

VARELA, JOS?? A., PILAR GARC??A-CORBEIRA, M. VICTORIA AG??ANELL, REYES BOCETA, JUAN BALLESTEROS, LORENZO AGUILAR, FERNANDO V??ZQUEZ-VALD??S, and RAFAEL DAL-R?? "Herpes Simplex Virus Type 2 Seroepidemiology in Spain." Sexually Transmitted Diseases 28, no. 1 (January 2001): 47–50. http://dx.doi.org/10.1097/00007435-200101000-00011.

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13

Davies, Nicholas, Julian Tang, and Katherine Nora Ward. "Herpes simplex virus type 2 and recurrent meningitis." Lancet 364, no. 9433 (August 2004): 501–2. http://dx.doi.org/10.1016/s0140-6736(04)16804-9.

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14

Duerst, Rebecca J., and Lynda A. Morrison. "Innate Immunity to Herpes Simplex Virus Type 2." Viral Immunology 16, no. 4 (December 2003): 475–90. http://dx.doi.org/10.1089/088282403771926300.

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15

Wald, A. "Oral shedding of herpes simplex virus type 2." Sexually Transmitted Infections 80, no. 4 (August 1, 2004): 272–76. http://dx.doi.org/10.1136/sti.2003.007823.

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16

Silke Heilingloh, Christiane, Christopher Lull, Elissa Kleiser, Mira Alt, Leonie Schipper, Oliver Witzke, Mirko Trilling, Anna-Maria Eis-Hübinger, Ulf Dittmer, and Adalbert Krawczyk. "Herpes Simplex Virus Type 2 Is More Difficult to Neutralize by Antibodies Than Herpes Simplex Virus Type 1." Vaccines 8, no. 3 (August 27, 2020): 478. http://dx.doi.org/10.3390/vaccines8030478.

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Infections with herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) are a global health burden. Besides painful oral or genital lesions in otherwise healthy subjects, both viruses can cause devastating morbidity and mortality in immune-compromised and immune-immature individuals. The latter are particularly susceptible to a disseminated, life-threatening disease. Neutralizing antibodies (NAb) constitute a correlate of protection from disease, and are promising candidates for the prophylactic or therapeutic treatment of severe HSV infections. However, a clinical vaccine trial suggested that HSV-2 might be more resistant to NAbs than HSV-1. In the present study, we investigated the antiviral efficacy of the well-characterized humanized monoclonal antibody (mAb) hu2c against HSV-2, in a NOD/SCID immunodeficiency mouse model. Despite the fact that hu2c recognizes a fully conserved epitope and binds HSV-1 and HSV-2 glycoprotein B with equal affinity, it was much less effective against HSV-2 in vitro and in NOD/SCID mice. Although intravenous antibody treatment prolonged the survival of HSV-2-infected mice, complete protection from death was not achieved. Our data demonstrate that HSV-2 is more resistant to NAbs than HSV-1, even if the same antibody and antigen are concerned, making the development of a vaccine or therapeutic antibodies more challenging.
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17

Ramachandran, Lintu, Vibhav K Bansal, Muhammad Janjua, Taha Mohamed Djirdeh, and Janine Borja. "Herpes Simplex Virus Type 2 Encephalitis with Herpes Simplex Virus Type 1 MRI Findings in an Immunocompetent Adult." Acta Scientific Neurology 4, no. 6 (May 27, 2021): 125–27. http://dx.doi.org/10.31080/asne.2021.04.0373.

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18

Cunningham, Anthony L., Cheryl A. Jones, and Min Kim. "Herpes simplex virus vaccines." Microbiology Australia 32, no. 3 (2011): 123. http://dx.doi.org/10.1071/ma11123.

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Herpes simplex virus (HSV) types 1 and 2 cause herpes labialis and genital herpes respectively, although genital herpes caused by HSV-1 is increasing in adolescence. Adult HSV-1 seroprevalence in western countries is 55% to 80% (80% in Australia) and acquired in two peaks, in infancy and adolescence. HSV-2 seroprevalence is highly variable geographically, reaching 12% in Australian adults but up to 90% in African countries. After initial HSV-1 or 2 infection, asymptomatic shedding occurs in the mouth and genital tract respectively in nearly all infected subjects. Complications of HSV-1 include keratitis and blindness and life-threatening encephalitis. Severe complications of HSV-2 include acute urinary retention, meningitis and neonatal herpes (25% fatality). In addition, prior infection with HSV-2 consistently enhances HIV acquisition three- to fourfold. In immunosuppressed persons, HSV-1 and 2 may cause indolent ulcers, oesophagitis and pneumonia. Ultimately, a childhood vaccine effective against both HSV-1 and 2 disease is needed.
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19

Rathore, Shagufta, Aditi Jamwal, and Vipin Gupta. "Herpes simplex virus type 2: Seroprevalence in antenatal women." Indian Journal of Sexually Transmitted Diseases and AIDS 31, no. 1 (2010): 11. http://dx.doi.org/10.4103/0253-7184.68994.

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20

Kim, Dae Eun, Ramee Pae, E. Young Bae, Ji Yoon Han, Seung Beom Han, Dae Chul Jeong, In Goo Lee, and Jin Han Kang. "Neonatal Meningoencephalitis caused by Herpes Simplex Virus Type 2." Korean Journal of Pediatric Infectious Diseases 21, no. 2 (2014): 150. http://dx.doi.org/10.14776/kjpid.2014.21.2.150.

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21

TSURUMI, Tatsuya, Koichio MAENO, and Yukihiro NISHIYAMA. "Molecular Cloning of Herpes Simplex Virus Type 2 DNA." Journal of Biochemistry 99, no. 3 (1986): 981–84. http://dx.doi.org/10.1093/oxfordjournals.jbchem.a135561.

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22

Belec, L., and M. Vidailhet. "Neuralgia preceding recurrent herpes simplex virus type 2 whitlow." Neurology 42, no. 11 (November 1, 1992): 2224. http://dx.doi.org/10.1212/wnl.42.11.2224.

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23

&NA;. "CE Test: Herpes Simplex Virus Type 2: An Update." Nurse Practitioner 28, no. 11 (November 2003): 41. http://dx.doi.org/10.1097/00006205-200311000-00013.

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24

ROSA-SANTOS, OMAR LUPI, ANGELA GONÇALVES SILVA, and ANTORNIO CARLOS PEREIRA. "HERPES SIMPLEX VIRUS TYPE 2 IN BRAZIL: SEROEPIDEMIOLOGIC SURVEY." International Journal of Dermatology 35, no. 11 (November 1996): 794–96. http://dx.doi.org/10.1111/j.1365-4362.1996.tb02976.x.

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25

Farazmand, P., P. D. Woolley, and G. R. Kinghorn. "Mollaret's meningitis and herpes simplex virus type 2 infections." International Journal of STD & AIDS 22, no. 6 (June 2011): 306–7. http://dx.doi.org/10.1258/ijsa.2010.010405.

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26

Rodríguez et al. "Herpes simplex virus type 2 infection increases HIV incidence." AIDS 17, Supplement 4 (2003): S128—S130. http://dx.doi.org/10.1097/00002030-200317004-00028.

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27

Gobbi, Claudio, Carlo Tosi, Claudio Städler, Claude Merenda, and Enos Bernasconi. "Recurrent Myelitis Associated with Herpes Simplex Virus Type 2." European Neurology 46, no. 4 (2001): 215–18. http://dx.doi.org/10.1159/000050808.

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28

De Clercq, E., and M. J. Robins. "Xylotubercidin against herpes simplex virus type 2 in mice." Antimicrobial Agents and Chemotherapy 30, no. 5 (November 1, 1986): 719–24. http://dx.doi.org/10.1128/aac.30.5.719.

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29

Dolan, Aidan, Fiona E. Jamieson, Charles Cunningham, Barbara C. Barnett, and Duncan J. McGeoch. "The Genome Sequence of Herpes Simplex Virus Type 2." Journal of Virology 72, no. 3 (March 1, 1998): 2010–21. http://dx.doi.org/10.1128/jvi.72.3.2010-2021.1998.

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ABSTRACT The genomic DNA sequence of herpes simplex virus type 2 (HSV-2) strain HG52 was determined as 154,746 bp with a G+C content of 70.4%. A total of 74 genes encoding distinct proteins was identified; three of these were each present in two copies, within major repeat elements of the genome. The HSV-2 gene set corresponds closely with that of HSV-1, and the HSV-2 sequence prompted several local revisions to the published HSV-1 sequence (D. J. McGeoch, M. A. Dalrymple, A. J. Davison, A. Dolan, M. C. Frame, D. McNab, L. J. Perry, J. E. Scott, and P. Taylor, J. Gen. Virol. 69:1531–1574, 1988). No compelling evidence for the existence of any additional protein-coding genes in HSV-2 was identified.
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30

el Sayed, F., M. C. Marguery, B. Periole, P. Bayle, and J. Bazex. "Urticarial manifestations associated with herpes simplex virus type 2." Sexually Transmitted Infections 71, no. 3 (June 1, 1995): 196. http://dx.doi.org/10.1136/sti.71.3.196.

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31

Softer, D., and J. R. Martin. "REMYELINATION IN EXPERIMENTAL HERPES SIMPLEX VIRUS TYPE 2 INFECTION." Journal of Neuropathology and Experimental Neurology 45, no. 3 (May 1986): 336. http://dx.doi.org/10.1097/00005072-198605000-00070.

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32

Li, Yiu-Tai, and Peng-Hui Wang. "The Lactobacillus and herpes simplex virus type 2 infection." Journal of the Chinese Medical Association 81, no. 9 (September 2018): 757–58. http://dx.doi.org/10.1016/j.jcma.2018.01.004.

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33

Leone, Peter. "Type-specific serologic testing for herpes simplex virus-2." Current Infectious Disease Reports 5, no. 2 (March 2003): 159–65. http://dx.doi.org/10.1007/s11908-003-0053-3.

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34

Freilinger, Tobias M., Martin Lieb, Christoph Schankin, and Soheyl Noachtar. "Herpes simplex virus type 2 meningitis and symptomatic migraine." Journal of Neurology 258, no. 4 (October 19, 2010): 689–90. http://dx.doi.org/10.1007/s00415-010-5790-2.

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35

Hodgman, T. C., and A. C. Minson. "The herpes simplex virus type 2 equivalent of the herpes simplex virus type 1 US7 gene and its flanking sequences." Virology 153, no. 1 (August 1986): 1–11. http://dx.doi.org/10.1016/0042-6822(86)90002-4.

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36

OKAZAKI, Katsuichiro, and Susumu KIMURA. "Immunoblotting analysis for typification of herpes simplex virus isolates using antiserum to herpes simplex virus type 2 glycoproteins." Agricultural and Biological Chemistry 53, no. 1 (1989): 255–57. http://dx.doi.org/10.1271/bbb1961.53.255.

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37

Yoshitake, Hidenori, Teruo Iwamasa, Yoshidai Makino, and Masatoshi Fukuda. "Seroepidemiology of Herpes Simplex Virus and Restriction Endonuclease Cleavage Analysis of Herpes Simplex Virus Type 2 in Okinawa." Pathology International 41, no. 1 (January 1991): 24–30. http://dx.doi.org/10.1111/j.1440-1827.1991.tb03268.x.

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38

Okazaki, Katsuichiro, and Susumu Kimura. "Immunoblotting Analysis for Typification of Herpes Simplex Virus Isolates Using Antiserum to Herpes Simplex Virus Type 2 Glycoproteins." Agricultural and Biological Chemistry 53, no. 1 (January 1989): 225–57. http://dx.doi.org/10.1080/00021369.1989.10869275.

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39

Shintaku, Masayuki, Yasushi Umehara, Keiko Iwaisako, Masao Tahara, and Yasushi Adachi. "Herpes Simplex Pancreatitis." Archives of Pathology & Laboratory Medicine 127, no. 2 (February 1, 2003): 231–34. http://dx.doi.org/10.5858/2003-127-231-hs.

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Abstract Lesions of the pancreas induced by viral infection have drawn relatively little attention because of their low incidence, and the histopathologic features of viral pancreatitis have not been fully elucidated. We report the autopsy findings of 2 patients, a 59-year-old woman with allergic granulomatous angiitis and a 73-year-old man with invasive pulmonary aspergillosis who had a disseminated visceral herpes simplex virus (HSV) infection. In both cases, the liver was the organ most severely affected by the viral infection. The pancreas showed multiple small foci of hemorrhagic necrosis, which were not accompanied by fat necrosis of the surrounding adipose tissue. Histopathologically, Cowdry type A intranuclear inclusions and a ground-glass appearance of the nuclei were found in many degenerated acinar cells around the necrotic foci. The gross appearance and histopathologic features of HSV pancreatitis were characteristic and, in particular, distinct from those of the more common acute hemorrhagic pancreatitis. Immunohistochemistry using an anti-HSV antibody revealed immunoreactivity in the intranuclear inclusions and ground-glass nuclei, and polymerase chain reaction analysis disclosed that the causative virus in these 2 cases was HSV-1. Herpes simplex virus pancreatitis constitutes a rare, but distinct pathologic entity among a group of acute pancreatitis diseases with diverse etiopathogenesis.
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40

Serafini-Cessi, Franca, Nadia Malagolini, Fabio Dall'olio, Lenore Pereira, and Gabriella Campadelli-Fiume. "Oligosaccharide chains of herpes simplex virus type 2 glycoprotein gG.2." Archives of Biochemistry and Biophysics 240, no. 2 (August 1985): 866–76. http://dx.doi.org/10.1016/0003-9861(85)90097-9.

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41

Aurelian, L. "Herpes Simplex Virus Type 2 Vaccines: New Ground for Optimism?" Clinical Diagnostic Laboratory Immunology 11, no. 3 (May 2004): 437–45. http://dx.doi.org/10.1128/cdli.11.3.437-445.2004.

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ABSTRACT The development of effective prophylactic and therapeutic vaccines against genital herpes has proven problematic. Difficulties are associated with the complexity of the virus life cycle (latency) and our relatively poor understanding of the mechanism of immune control of primary and recurrent disease. The types of effector cells and the mechanisms responsible for their activation and regulation are particularly important. Studies from my and other laboratories have shown that recurrent disease is prevented by virus-specific T helper 1 (Th1) cytokines (viz., gamma interferon) and activated innate immunity. Th2 cytokines (viz., interleukin-10 [IL-10]) and regulatory (suppressor) T cells downregulate this immune profile, thereby allowing unimpeded replication of reactivated virus and recurrent disease. Accordingly, an effective therapeutic vaccine must induce Th1 immunity and be defective in Th2 cytokine production, at least IL-10. These concepts are consistent with the findings of the most recent clinical trials, which indicate that (i) a herpes simplex virus type 2 (HSV-2) glycoprotein D (gD-2) vaccine formulated with a Th1-inducing adjuvant has prophylactic activity in HSV-2- and HSV-1-seronegative females, an activity attributed to the adjuvant function, and (ii) a growth-defective HSV-2 mutant (ICP10ΔPK), which is deleted in the Th2-polarizing gene ICP10PK, induces Th1 immunity and has therapeutic activity in both genders. The ICP10ΔPK vaccine prevents recurrent disease in 44% of treated subjects and reduces the frequency and severity of recurrences in the subjects that are not fully protected. Additional studies to evaluate these vaccines are warranted.
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42

Shields, Lisa B. E., and Mohammad S. Alsorogi. "Herpes Simplex Virus Type 2 Radiculomyelitis Disguised as Conversion Disorder." Case Reports in Neurology 11, no. 1 (April 16, 2019): 117–23. http://dx.doi.org/10.1159/000499701.

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Herpes simplex virus type 2 (HSV-2) is the most common cause of genital herpes with a seroprevalence of 20–30% in developed countries and 80% worldwide. In addition to neonatal encephalitis and meningitis, HSV-2 is associated with radiculomyelitis marked by pain, paresis, sphincter disturbances, sensory loss, or ascending necrotizing myelitis. We report the case of a patient with a lengthy psychiatric history who presented with lower extremity pain and weakness. Cervical, thoracic, and lumbar MRI scans with and without gadolinium contrast revealed no significant stenosis, neural compression, or other abnormal findings, and the brain MRI with and without gadolinium contrast was normal. The initial diagnosis was conversion disorder due to myriad psychological stressors. Polymerase chain reaction (PCR) of CSF detected HSV-2 and a lymphocytic pleocytosis, and the diagnosis of radiculomyelitis was confirmed. She was treated with i.v. acyclovir for 3 weeks followed by valacyclovir. The patient attained no improvement of her symptoms within 8 months; however, she reported decreased pain and improved strength of the lower extremities by 17 months. Neurologists should be aware of the association between HSV-2 and radiculomyelitis, particularly in the setting of a patient with psychiatric comorbidities. Recognition of HSV-2 through PCR of CSF and prompt treatment with acyclovir may prevent devastating neurological sequelae.
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43

Millichap, J. Gordon. "Brainstem Involvement in Neonatal Herpes Simplex Virus Type 2 Encephalitis." Pediatric Neurology Briefs 21, no. 9 (September 1, 2007): 66. http://dx.doi.org/10.15844/pedneurbriefs-21-9-2.

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44

Kallio-Laine, Katariina, Mikko Seppänen, Hannu Kautiainen, Marja-Liisa Lokki, Maija Lappalainen, Ville Valtonen, Markus Färkkilä, and Eija Kalso. "Recurrent Lymphocytic Meningitis Positive for Herpes Simplex Virus Type 2." Emerging Infectious Diseases 15, no. 7 (July 2009): 1119–22. http://dx.doi.org/10.3201/eid1507.080716.

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45

Lai, Vivien Wai Yun, Zaal Meher-Homji, Faye Liu, and Joe Sasadeusz. "Primary herpes simplex virus type 2 hepatitis diagnosed during laparoscopy." Lancet 396, no. 10265 (December 2020): e90. http://dx.doi.org/10.1016/s0140-6736(20)32388-6.

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46

Godet, C., A. Beby-Defaux, G. Agius, O. Pourrat, and R. Robert. "Maternal Herpes simplex virus type 2 encephalitis following Cesarean section." Journal of Infection 47, no. 2 (August 2003): 174–75. http://dx.doi.org/10.1016/s0163-4453(03)00017-3.

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47

Tirabassi, Rebecca S., Christopher I. Ace, Tatyana Levchenko, Vladimir P. Torchilin, Liisa K. Selin, Siwei Nie, Dennis L. Guberski, and Kejian Yang. "A mucosal vaccination approach for herpes simplex virus type 2." Vaccine 29, no. 5 (January 2011): 1090–98. http://dx.doi.org/10.1016/j.vaccine.2010.11.076.

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48

Hawkes, C. H., G. Giovannoni, G. Keir, M. Cunnington, and E. J. Thompson. "Seroprevalence of herpes simplex virus type 2 in multiple sclerosis." Acta Neurologica Scandinavica 114, no. 6 (December 2006): 363–67. http://dx.doi.org/10.1111/j.1600-0404.2006.00677.x.

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49

Bergstrom, T., A. Vahlne, K. lestig, S. Jeansson, M. Forsgren, and E. Lycke. "Primary and Recurrent Herpes Simplex Virus Type 2-Induced Meningitis." Journal of Infectious Diseases 162, no. 2 (August 1, 1990): 322–30. http://dx.doi.org/10.1093/infdis/162.2.322.

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50

EIS-HÜBINGER, ANNA MARIA, EMMANUEL NYANKIYE, DIDIER MBOUA BITOUNGUI, and JEAN NDJOMOU. "Prevalence of Herpes Simplex Virus Type 2 Antibody in Cameroon." Sexually Transmitted Diseases 29, no. 11 (November 2002): 637–42. http://dx.doi.org/10.1097/00007435-200211000-00004.

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