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Dissertations / Theses on the topic 'Herpes simplex virus'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Nahum, Joseph. "Thérapeutique des infections à Herpes simplex virus." Paris 5, 1999. http://www.theses.fr/1999PA05P200.

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2

Ingemarson, Rolf. "Herpes simplex ribonucleotide reductase." Doctoral thesis, Umeå universitet, Institutionen för medicinsk kemi och biofysik, 1989. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-101352.

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In all bacterial, plant and animal cells, as well as in many viruses, genetic information resides in DNA (deoxyribonucleic acid). Replication of DNA is essential for proliferation, and DNA-containing viruses (such as herpesviruses) must carry out this process within the mammalian cells they infect. The enzyme ribonucleotide reductase catalyzes the first unique step leading to the production of the four deoxy-ribonucleotides used to make DNA. Each deoxyribonucleotide is produced by reduction of the corresponding ribonucleotide. After infection of a mammalian cell with herpes simplex virus (HSV)

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3

Turner, Angelina. "Herpes simplex virus induced membrane fusion." Thesis, University of Cambridge, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.625097.

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4

Bajric, Amina. "Validering av Varicella Zoster virus och Herpes Simplex virus." Thesis, Malmö universitet, Fakulteten för hälsa och samhälle (HS), 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-24474.

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Syftet med denna valideringsstudie är att värdera lämpligheten att överföra den manuella analysen av aktuell infektion av Varicella Zoster Virus (aVZV IgM) och Herpes Simplex Virus (aHSV IgM) med SIEMENS Enzygnost® till en av de automatiserade analysinstrumenten EUROIMMUN Analyzer I (ELISA) eller DiaSorin LIAISON® XL. Arbetet utfördes på Klinisk Mikrobiologi i Lund. Konsekutiva serumprover för VZV IgM (n=108) och för HSV IgM (n=116) från det vardagliga flödet analyserades, tillsammans med 10 PCR- eller serokonversion-konfirmerade positiva serumprover av primär infektion VZV och HSV samt 10 pos

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5

Atfield, Rachel Sarah. "Herpes simplex virus glycoprotein-mediated membrane fusion." Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.615860.

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6

Barr, Beverly Bell Brown. "Molecular epidemiology of herpes simplex virus infections." Thesis, University of Edinburgh, 1987. http://hdl.handle.net/1842/19152.

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7

Graham, Richard Peter. "Purification of glycoproteins from herpes simplex virus." Master's thesis, University of Cape Town, 1985. http://hdl.handle.net/11427/25694.

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The aim of this work was to purify type-specific glycoproteins from herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) for diagnostic use. The most likely candidate for a type-specific glycoprotein of HSV-1 is glycoprotein C (gC), although it has recently been shown to contain some type-common antigenic determinants. HSV-1 and HSV-2 were produced in BHK-21 cells and labelled with either (³H)-glucosamine ((³H)-gln) or a mixture of (¹⁴C)-amino acids ((¹⁴C)-aa). Analysis of the radiolabelled products by analytical sodium dodecyl sulphate polyacrylamide gel electrophoresis (SOS-PAGE) and autor

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8

Delboy, Mark. "PROTEASOME-DEPENDENT ENTRY OF HERPES SIMPLEX VIRUS." VCU Scholars Compass, 2010. http://scholarscompass.vcu.edu/etd/47.

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Herpes simplex virus entry into cells is a multistep process that engages the host cell machinery. The proteasome is a large, ATP-dependent, multisubunit protease that plays a critical role in the maintenance of cell homeostasis. A battery of assays were used to demonstrate that proteasome inhibitors blocked an early step in herpes simplex virus entry that occurred after capsid penetration into the cytosol but prior to capsid arrival at the nuclear periphery. Proteasome-dependent viral entry was not reliant on host or viral protein synthesis. MG132, a peptide aldehyde that competitively inhibi

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9

Dambrosi, Sarah. "Neurovirulence et latence des virus Herpes simplex mutants." Thesis, Université Laval, 2009. http://www.theses.ulaval.ca/2009/26368/26368.pdf.

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10

Whiteley, Alison. "Studies of herpes simplex virus type-1 envelopment." Thesis, University of Cambridge, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621703.

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11

Wu, Zetang. "Silencing Suppression by Herpes Simplex Virus Type 1." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1213287215.

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12

Kogishi, Junichi. "Mutant herpes simplex virus-mediated suppression of retinoblastoma." Kyoto University, 1999. http://hdl.handle.net/2433/181258.

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13

Harman, Laura Emily Rose. "Immunological control of herpes simplex virus type 1 latency." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708822.

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14

Svobodová, Stanislava. "Study of herpes simplex virus type 1 tegument assembly." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607957.

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15

Teepe, Annette. "Relationship of Estrous Cycle to Herpes Simplex Virus Type 2 Susceptibility in Female Mice." Thesis, North Texas State University, 1987. https://digital.library.unt.edu/ark:/67531/metadc500408/.

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In CBA/NJ mice, splenic natural killer (NK) cell activity varies with stages of estrous. Susceptibility of ICR mice to intravaginal inoculation of herpes simplex virus type 2 (HSV-2) decreases with age. Susceptibility of female ICR and CBA/NJ mice to HSV-2 inoculated intravaginally and intraperitoneally was examined during the estrous cycle. In cycling ICR mice, greatest susceptibility to intravaginal inoculation was observed during diestrous and the least during metestrous. CBA/NJ mice were most susceptible to intravaginal inoculation of HSV-2 during diestrous. ICR mice were ovariectomized to

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16

Ewing, Stephen Michael George. "Herpes simplex virus type 1 infection of dendritic leucocytes." Thesis, University of Oxford, 1992. http://ora.ox.ac.uk/objects/uuid:e83750b1-3aa7-452e-a876-be51690252be.

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17

Hodge, Paul Daniel. "The encapsidation of herpes simplex virus type 1 DNA." Thesis, University of Glasgow, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301951.

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18

Johnson, Paul Andrew. "The control of herpes simplex virus late gene transcription." Thesis, University of Glasgow, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.481097.

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19

McBride, Brian William. "Mucosal immune responses induced by herpes simplex virus infection." Thesis, University of Southampton, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254770.

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20

Stumpf, Thomas Hugo. "Immunopathology of herpes simplex virus infections in the eye." Thesis, University of Bristol, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.250985.

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21

Atkinson, Helen Ruth. "Molecular studies of herpes simplex virus type 1 latency." Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241568.

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22

Stoner, Terri Dorene. "Indole-3-Carbinol Inhibition of Herpes Simplex Virus Replication." Kent State University / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=kent1228328838.

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23

Hedner, Ewa. "Herpes Simplex virus type 1 and intraoral wound healing." [Göteborg] : Departments of Clinical Virology, Faculty of Medicine, Oral Microbiology and Oral Surgery, Faculty of Odontology, University of Göteborg, 1993. http://catalog.hathitrust.org/api/volumes/oclc/30761199.html.

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24

Zhou, Amy Yuan. "Exploring the functional role of herpes simplex virus type-1 glycoprotein H in virus entry." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648658.

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25

Naldinho, Souto Raquel Cristina. "Studies of Herpes Simplex Virus type-1 tegument proteins during virus egress." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.613035.

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26

Denes, Christopher. "Virus-host interactions of herpes simplex virus type-1 envelope glycoprotein E." Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/23098.

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Herpes simplex virus type-1 (HSV-1) lays dormant in approximately 67% of individuals worldwide. The typical orolabial or genital lesions are relatively innocuous, but infection can cause encephalitis and systemic infections in susceptible populations. With no vaccine available, research into new antivirals is critical for managing the increasing incidence of antiviral resistance as well as the impact of virus shedding events. One approach to the discovery of new antivirals is the identification of host-pathogen interfaces necessary for viral replication and spread. Glycoprotein E (gE) is an HS

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27

Görander, Staffan. "Functions of glycoprotein G of herpes simplex virus type 2." Göteborg : Dept. of Infectious Medicine, Section for Virology, Sahlgrenska Academy, 2010. http://hdl.handle.net/2077/21861.

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28

Williams, N. A. "The role of Langerhans cells in infection with herpes simplex virus." Thesis, University of Bristol, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.235240.

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29

Liu, Lei. "Immunology of herpes simplex keratitis and its treatment by corneal transplantation." Thesis, Available from the University of Aberdeen Library and Historic Collections Digital Resources. Online version available for University member only until Sep. 7, 2010, 2009. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=33585.

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30

Rajaguru, Suesha Chandani. "Inhibition of herpes simplex virus replication using small interfering rna that target icp4 gene of herpes simplex type 2." [Gainesville, Fla.] : University of Florida, 2004. http://purl.fcla.edu/fcla/etd/UFE0007066.

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Thesis (M.S.)--University of Florida, 2004.<br>Typescript. Title from title page of source document. Document formatted into pages; contains 65 pages. Includes Vita. Includes bibliographical references.

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31

Liu, Mingyu. "Gene regulation and function of ICP0 in herpes simplex virus infected cells." OpenSIUC, 2010. https://opensiuc.lib.siu.edu/dissertations/143.

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Herpes simplex virus (HSV) is a clinically important virus, whose life cycle alternates between productive replication and latency. Infected cell protein 0 (ICP0) is generally believed to play a key role in determining the outcome of HSV infections. Synthesis of ICP0 promotes the productive replication of HSV, whereas absence of ICP0 renders HSV prone to establish latent infections. In the first part of the dissertation, I attempt to address the question how is ICP0 gene regulated. To tackle this question, we constructed recombinant HSV that encodes GFP-tagged ICP0 so that the reg

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32

Cheung, Peter. "Brefeldin A arrests the maturation and egress of herpes simplex virus particles during infection." Thesis, University of British Columbia, 1991. http://hdl.handle.net/2429/29798.

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Herpes Simplex Virus (HSV) requires the host cell secretory apparatus for the maturation and egress of newly synthesized viral particles. Not only do viral glycoproteins rely on the host ER and Golgi compartments for their proper processing, it is believed that enveloped particles are transported through these same organelles for their export out of the cells. Brefeldin A (BFA) is a compound that induces retrograde movement of material from the Golgi apparatus to the ER and causes the disassembly of the Golgi complex. In this study, the effects of BFA on the propagation of HSV-1 in infected ce

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33

Grapes, Matthew Giles Robert. "Analysis of transcriptional activation by the HSV-1 protein VP16 and its EHV-1 homologue." Thesis, Imperial College London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325048.

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34

Kew, Chun, and 喬駿. "Inhibition of PACT mediated type 1 interferon production by herpes simplex virus type 1 Us11 protein." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206481.

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Mammals have complicated antiviral innate immunity to combat viral infection and this poses a strong selection pressure on the viruses. As a result, many viruses have evolved different strategies to disrupt the function of hosts’ antiviral innate immunity. Herpes simplex virus type 1 (HSV-1) is one of the examples. HSV-1 is a common and important human pathogen. HSV-1 infection induces type I interferons (IFNs) which restrict viral replication potently. To ensure persistent infection and successful replication, HSV-1 encodes several IFN-suppressing proteins. One example is Us11. Interaction be

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35

Targett-Adams, Paul. "Characterisation of the HSV-1 DNA packaging protein encoded by the UL25 gene." Thesis, University of Glasgow, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368523.

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36

Leslie, Jenny. "An investigation into factors that influence the incorporation of proteins into the HSV-1 tegument." Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297029.

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37

Manickasingham, Shrivanthi Prithiva. "The effects on Langerhans cells and dermal leukocyte populations of agents which trigger herpes simplex reactivation." Thesis, University of Bristol, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337696.

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38

Pawliczek, Tobias. "Studies on the role of the cellular ESCRT machinery in herpes simplex virus type 1 replication." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609315.

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39

Nicoll, Michael Peter. "The role of the Herpes simplex virus type 1 latency-associated transcripts during the establishment and maintenance of latency." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607713.

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40

Brown, Elizabeth L. "Consequences of genital herpes simplex virus infection among vulnerable populations /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/10885.

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41

Striebinger, Hannah. "Membran-assoziierte Protein-Protein-Interaktionen des Herpes simplex-Virus 1." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-146882.

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42

Littlejohn, Emma Sophie Vout. "The Sensitivity of Adenovirus and Herpes simplex virus to Honey." The University of Waikato, 2009. http://hdl.handle.net/10289/2804.

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Honey has been used for centuries as a medicine to treat various ailments and infections. A large amount of research has established that honey has potent antibacterial activity. However, the sensitivity to honey of viral species that cause infections has been studied in only a small number of cases. The aim of this study was to obtain data to clarify and extend knowledge obtained from these previous studies of honey's antiviral activity, and especially study those viruses that cause localised infections which have limited or no therapy available, which are suitable to treatment with topically

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43

Preetha, Balan K. V. "Analysis of dispensable glycoproteins of herpes simplex virus type 1." Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.320012.

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44

McGeehan, John Edward. "Molecular characterisation of Herpes simplex virus type 1 deoxyuridine triphosphatase." Thesis, University of Glasgow, 1998. http://theses.gla.ac.uk/6104/.

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Analysis of primary sequence data revealed a subset of open reading frames that were predicted to encode HSV-I dUTPases based on five areas of local primary sequence conservation. The differences in the primary sequence organisation of these motif regions allowed the description of two distinct dUTPase classes. The class I dUTPases are encoded by a diverse range of organisms and are characterised by a trimeric arrangement with subunit protein lengths approximating 150 amino acids. The class II dUTPases are specific to the herpesviruses and are characterised by a monomeric arrangement with a pr

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45

Orzalli, Megan Jenkins. "Inhibition of Nuclear DNA Sensing by Herpes Simplex Virus 1." Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:10779.

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The detection of immunostimulatory DNA is well documented to occur at several cellular sites, but there is limited evidence of nuclear innate DNA sensing. Prior to this study, the detection of herpesviral DNA was thought to be restricted to the cytosol so as to limit the sensing of host DNA in the nucleus. However, given the nuclear lifecycle of these viruses, we hypothesized that viral DNA could be sensed in the nucleus of infected cells. To test this hypothesis we examined the activation of interferon regulatory factor 3 (IRF-3) in response to herpes simplex virus 1 (HSV-1) infection of pr

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46

Porter, Iain Malcolm. "Characterisation of the herpes simplex virus type 1 mutant, ambUL12." Thesis, University of Glasgow, 2002. http://theses.gla.ac.uk/3959/.

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The herpes simplex virus type 1 (HSV-1) UL12 gene encodes an alkaline nuclease. Although the UL12 gene is not absolutely essential for replication, UL12 null mutants produce 100-1000 fold less viable virus than wt HSV-1. It has been suggested that the alkaline nuclease functions to remove branched structures from high molecular weight concatemeric DNA prior to its cleavage into monomeric genomes that are packaged into viral capsids. Failure to remove the branches results in unstable packaging of DNA into capsids which fail to egress from the nucleus. This thesis describes detailed characterisa

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47

Dalrymple, M. A. "The regulation of herpes simplex virus immediate early gene expression." Thesis, University of Glasgow, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.378173.

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48

Galdiero, Massimiliano. "Mutagenesis of glycoprotein H of herpes simplex virus type 1." Thesis, University of Cambridge, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624784.

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49

Jenkins, Mary K. "Metabolism of Herpes Simplex Virus 1 infected RAW 264.7 macrophages." Wright State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=wright1472203113.

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50

Doll, Jessica R. "Insights into Herpes Simplex Virus Pathogenesis: Neuronal Fate Post-Reactivation." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1535458248721271.

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