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Journal articles on the topic 'Hexachlorobenzene – Toxicology'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Sopena, Yolanda E., Ana M. Ferramola de Sancovich, and Horacio A. Sancovich. "Hexachlorobenzene Treatment on Hepatic Mitochondrial Function Parameters and Intracellular Coproporphyrinogen Oxidase Location." International Journal of Toxicology 27, no. 6 (2008): 455–65. http://dx.doi.org/10.1080/10915810802657002.

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These studies try to elucidate why isocoproporphyrin appears in hexachlorobenzene-poisoned rats’ feces. Chronic exposure of hexachlorobenzene to rats produces an experimental model for human porphyria cutanea tarda. After 8 weeks of treatment, rats showed high porphyrin excreta and 50% inhibition of liver uroporphyrinogen decarboxylase activity. Uroporphyrin plus heptacarboxylic porphyrin exceeded coproporphyrin in urine, whereas in feces, isocoproporphyrin, from abnormal pentacarboxylic porphyrinogen III oxidative decarboxylation by liver coproporphyrinogen oxidase, became the main porphyrin.

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2

van Birgelen, Angelique P. J. M. "Response: Hexachlorobenzene." Environmental Health Perspectives 107, no. 4 (1999): A184. http://dx.doi.org/10.2307/3434579.

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3

Aylward, Lesa L., Sean M. Hays, Michelle Gagné, Andy Nong, and Kannan Krishnan. "Biomonitoring equivalents for hexachlorobenzene." Regulatory Toxicology and Pharmacology 58, no. 1 (2010): 25–32. http://dx.doi.org/10.1016/j.yrtph.2010.06.003.

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4

Llambías, Elena B. C., Marta B. Mazzetti, Sandra M. Lelli, Carmen Aldonatti, and Leonor C. San Martín de Viale. "Melatonin Formation in Pineal Gland from Rats with Hexachlorobenzene Experimental Porphyria." International Journal of Toxicology 26, no. 6 (2007): 545–51. http://dx.doi.org/10.1080/10915810701707643.

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Hexachlorobenzene produces an experimental hepatic por-phyria in rats, which is similar to human porphyria cutanea tarda, with hyperpigmentation as one of its characteristic features. Alterations in tryptophan metabolism have been previously observed in this chronic porphyria. Melatonin formation from tryptophan via serotonin shows diurnal rhythmicity in the pineal gland, and higher values are observed during the dark phase of an imposed light-dark cycle. The purpose of this study was to determine the contents of tryptophan and its metabolites in pineal gland of normal and hexachlorobenzene-tr

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5

Egeler, Philipp, Michael Meller, Joerg Roembke, and Peter Spoerlein. "Biomagnification of hexachlorobenzene." Environmental Science and Pollution Research 8, no. 4 (2001): 230. http://dx.doi.org/10.1007/bf02987398.

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6

ANDREWS, JAMES E., K. DIANE COURTNEY, ANDREW G. STEAD, and W. E. DONALDSON. "Hexachlorobenzene-Induced Hyperparathyroidism and Osteosclerosis in Rats." Toxicological Sciences 12, no. 2 (1989): 242–51. http://dx.doi.org/10.1093/toxsci/12.2.242.

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7

Alvarez, Laura, Andrea Randi, Paula Alvarez, et al. "Reproductive effects of hexachlorobenzene in female rats." Journal of Applied Toxicology 20, no. 1 (2000): 81–87. http://dx.doi.org/10.1002/(sici)1099-1263(200001/02)20:1<81::aid-jat629>3.0.co;2-z.

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8

Renner, Gerhard. "Hexachlorobenzene and its metabolism." Toxicological & Environmental Chemistry 18, no. 1 (1988): 51–78. http://dx.doi.org/10.1080/02772248809357308.

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9

Nurislamova, Tatyana V., T. S. Ulanova, N. A. Popova, and O. A. Maltseva. "METHODICAL SUPPORT FOR CONTROL OVER THE CONCENTRATION OF PERSISTENT ORGANIC POLLUTANT HEXACHLOROBENZENE IN BLOOD." Hygiene and sanitation 98, no. 3 (2019): 343–48. http://dx.doi.org/10.18821/0016-9900-2019-98-3-343-348.

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Introduction. Persistent organic pollutants (POP) are extremely dangerous compounds contaminating the environment and exerting the negative impact on human health. Organochlorine pesticides, including hexachlorobenzene, are a specific group among POP. Our research goal is methodological support for the hexachlorobenzene content control in human biological environments. Material and methods. We developed highly both sensitive and selective procedure for determining hexachlorobenzene in blood. Hexachlorobenzene determination in blood is based on the application of capillary gas chromatography wi

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10

Carthew, P., R. E. Edwards, and A. G. Smith. "Immunotoxic Effects of Hexachlorobenzene on the Pathogenesis of Systemic, Pneumonic and Hepatic Virus Infections in the Mouse." Human & Experimental Toxicology 9, no. 6 (1990): 403–11. http://dx.doi.org/10.1177/096032719000900608.

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A quantitative histopathological method has been developed for the evaluation of the effects of hexachlorobenzene (HCB) on the pathogenesis of three virus infections in the mouse. Hexachlorobenzene was selected because a substantial amount of immunotoxicological data already exists with which we could compare our results. To establish the validity of the method a systemic virus infection (mouse cytomegalovirus, MCMV), a pneumonia causing virus (pneumonia virus of mice, PVM) and a hepatitis virus (mouse hepatitis virus, MHV) were used. We have compared the existing data with the actual patholog

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11

Arnold, D. L., and D. Krewski. "Long-term toxicity of hexachlorobenzene." Food and Chemical Toxicology 26, no. 2 (1988): 169–74. http://dx.doi.org/10.1016/0278-6915(88)90113-5.

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12

Schielen, P., W. Schoo, J. Tekstra, H. H. A. Oostermeijer, W. Seinen, and N. Bloksma. "Autoimmune Effects of Hexachlorobenzene in the Rat." Toxicology and Applied Pharmacology 122, no. 2 (1993): 233–43. http://dx.doi.org/10.1006/taap.1993.1192.

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13

Chiappini, Florencia, Laura Alvarez, Victoria Lux-Lantos, Andrea S. Randi, and Diana L. Kleiman de Pisarev. "Hexachlorobenzene Triggers Apoptosis in Rat Thyroid Follicular Cells." Toxicological Sciences 108, no. 2 (2009): 301–10. http://dx.doi.org/10.1093/toxsci/kfp016.

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14

Queiroz, M. LS, C. Bincoletto, R. CR Perlingeiro, M. R. Quadros, and C. A. Souza. "Immunoglobulin levels in workers exposed to hexachlorobenzene." Human & Experimental Toxicology 17, no. 3 (1998): 172–75. http://dx.doi.org/10.1177/096032719801700308.

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The serum immunoglobulin (IgG, IgM and IgA) concentrations of 52 chlorinated-exposed workers were examined and compared with those of non-exposed, age- and sex-matched individuals. At the time of testing, the exposed population had mean hexachlorobenzene (HCB) blood levels of 3.84 mg/dl with a range of 0.1 to 16 mg/dl. Increased IgG and IgM levels were found in the HCB- exposed workers (P50.05 and P50.01, respectively). Hepatic function was evaluated by serum aspartate (AST) and alanine aminotransferase (ALT) activities, as well as by bilirubin levels. IgM concentrations were positively correl

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15

Queiroz, M. L. S., C. Bincoletto, R. C. R. Perlingeiro, M. R. Quadros, and C. A. Souza. "Immunoglobulin levels in workers exposed to hexachlorobenzene." Human & Experimental Toxicology 17, no. 3 (1998): 172–75. http://dx.doi.org/10.1191/096032798678908477.

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16

To-Figueras, J., J. Gómez-Catalán, M. Rodamilans, and J. Corbella. "Sulphur Derivative of Hexachlorobenzene in Human Urine." Human & Experimental Toxicology 11, no. 4 (1992): 271–73. http://dx.doi.org/10.1177/096032719201100406.

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Pentachlorothiophenol, a sulphur derivative of the widespread environmental pollutant hexachlorobenzene (HCB) has been detected and quantified in the urine of a human general population with high body burden of HCB. The sulphur derivative was analysed by GLC-MS as pentachlorothioanisole (PCTA) after hydrolysis and methylation of the respective conjugate and was found in 100% of the samples ( n = 40) with a mean concentration of 1.85 ± 0.98 ng ml-1 (X ± s.d., range 0.58-4.50 ng ml-1). No correlation with urinary pentachlorophenol (PCP) and no sex-related differences were found. The derivative m

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17

Salmon, M. L., S. G. Madanagopal, R. Blando, J. Berner, and K. Williams. "Effects of hexachlorobenzene on embryonic mammalian cells." Toxicology in Vitro 16, no. 5 (2002): 539–48. http://dx.doi.org/10.1016/s0887-2333(02)00054-1.

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18

Purchase, Rupert. "Hexachlorobenzene: Proceedings of an international symposium." Food and Chemical Toxicology 25, no. 11 (1987): 877. http://dx.doi.org/10.1016/0278-6915(87)90268-7.

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19

Giribaldi, Laura, Florencia Chiappini, Carolina Pontillo, Andrea S. Randi, Diana L. Kleiman de Pisarev, and Laura Alvarez. "Hexachlorobenzene induces deregulation of cellular growth in rat liver." Toxicology 289, no. 1 (2011): 19–27. http://dx.doi.org/10.1016/j.tox.2011.07.004.

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20

ANDREWS, J. "Hexachlorobenzene-induced hyperparathyroidism and osteosclerosis in rats*1, *2." Fundamental and Applied Toxicology 12, no. 2 (1989): 242–51. http://dx.doi.org/10.1016/0272-0590(89)90041-9.

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21

Dybing, Erik, and Tore Aune. "Hexachlorobenzene Induction of 2,4-Diaminoanisole Mutagenicity in Vitro." Acta Pharmacologica et Toxicologica 40, no. 5 (2009): 575–83. http://dx.doi.org/10.1111/j.1600-0773.1977.tb02113.x.

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22

Dybing, Erik, and Tore Aune. "Hexachlorobenzene Induction of 2,4-Diaminoanisole Mutagenicity in Vitro." Acta Pharmacologica et Toxicologica 40 (March 13, 2009): 575–83. http://dx.doi.org/10.1111/j.1600-0773.1977.tb03560.x.

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23

Siekel, Peter, Ivan Chalupa, Jozef Beňo, Milan Blaško, Jozef Novotný, and Ján Burian. "A genotoxicological study of hexachlorobenzene and pentachloroanisole." Teratogenesis, Carcinogenesis, and Mutagenesis 11, no. 1 (1991): 55–60. http://dx.doi.org/10.1002/tcm.1770110107.

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24

Smith, Andrew G., David Dinsdale, Jos� R. P. Cabral, and Anne L. Wright. "Goitre and wasting induced in hamsters by hexachlorobenzene." Archives of Toxicology 60, no. 5 (1987): 343–49. http://dx.doi.org/10.1007/bf00295753.

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25

Schwab, Bradley W. "The TEF Approach for Hexachlorobenzene." Environmental Health Perspectives 107, no. 4 (1999): A183. http://dx.doi.org/10.2307/3434578.

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26

Billi de Catabbi, Silvia C., Alicia Faletti, Federico Fuentes, Leonor C. San Martín de Viale, and Adriana C. Cochón. "Hepatic arachidonic acid metabolism is disrupted after hexachlorobenzene treatment." Toxicology and Applied Pharmacology 204, no. 2 (2005): 187–95. http://dx.doi.org/10.1016/j.taap.2004.09.001.

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27

Ezendam, Janine, Kevin Kosterman, Henneke Spijkerboer, et al. "Macrophages are involved in hexachlorobenzene-induced adverse immune effects." Toxicology and Applied Pharmacology 209, no. 1 (2005): 19–27. http://dx.doi.org/10.1016/j.taap.2005.03.010.

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28

Andrews, J. E., L. D. Jackson, A. G. Stead, and W. E. Donaldson. "Morphometric analysis of osteosclerotic bone resulting from hexachlorobenzene exposure." Journal of Toxicology and Environmental Health 31, no. 3 (1990): 193–201. http://dx.doi.org/10.1080/15287399009531448.

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29

Queiroz, Mls, C. Bincoletto, Rcr Perlingeiro, CA Souza, and H. Toledo. "Defective neutrophil function in workers occupationally exposed to hexachlorobenzene." Human & Experimental Toxicology 16, no. 6 (1997): 322–26. http://dx.doi.org/10.1177/096032719701600605.

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In this work we have studied the respiratory burst and chemotaxis of polymorphonuclear leukocytes from 51 workers exposed to chlorinated compounds, which were compared with those of non-exposed, age- and sex- matched invididuals. These two neutrophil functions were significantly reduced as compared to controls. No correlation was observed between the length of exposure, hexachlorobenzene (HCB) blood concentrations and neu trophil chemotaxis or the extent of nitroblue tetrazolium reduction.

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30

To-Figueras, J., C. Barrot, M. Rodamilans, et al. "Accumulation of hexachlorobenzene in humans: a long standing risk." Human & Experimental Toxicology 14, no. 1 (1995): 20–23. http://dx.doi.org/10.1177/096032719501400105.

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1 Hexachlorobenzene (HCB) internal dose in the general population of Barcelona (Spain) was estimated after new indications of the carcinogenicity of this chemical in humans were recently reported. Hospital blood bank facilities and randomly selected volunteers were used for HCB analyses in serum ( n=100) and cerumen ( n=25). Other main organochlorine residues often found in human tissues and blood (pp'DDE, β-HCH,) were also determined. 2 HCB serum levels currently found (Range 0.7-19.7 ng ml-1; X ± s.d.: 4.13 ± 3.61; GM: 3.05) were compared to those found in a similar survey made in 1986 on th

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31

Jarrell, John F., Avril McMahon, David Villeneuve, et al. "Hexachlorobenzene toxicity in the monkey primordial germ cell without induced porphyria." Reproductive Toxicology 7, no. 1 (1993): 41–47. http://dx.doi.org/10.1016/0890-6238(93)90008-u.

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32

Ezendam, Janine, Ischa Vissers, Rob Bleumink, Joseph G. Vos, and Raymond Pieters. "Immunomodulatory Effects of Tetrachlorobenzoquinone, a Reactive Metabolite of Hexachlorobenzene." Chemical Research in Toxicology 16, no. 6 (2003): 688–94. http://dx.doi.org/10.1021/tx034016p.

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33

Rozman, K., J. R. Gorski, P. Rozman, and A. Parkinson. "Reduced serum thyroid hormone levels in hexachlorobenzene-induced porphyria." Toxicology Letters 30, no. 1 (1986): 71–78. http://dx.doi.org/10.1016/0378-4274(86)90181-5.

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34

Nair, Amit, and M. K. K. Pillai. "Monitoring of hexachlorobenzene residues in Delhi and Faridabad, India." Bulletin of Environmental Contamination and Toxicology 42, no. 5 (1989): 682–86. http://dx.doi.org/10.1007/bf01700388.

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35

Palkovičová Murínová, Ľubica, Soňa Wimmerová, Kinga Lancz, et al. "Partitioning of hexachlorobenzene between human milk and blood lipid." Environmental Pollution 229 (October 2017): 994–99. http://dx.doi.org/10.1016/j.envpol.2017.07.087.

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36

Gómez-Catalán, J., J. To-Figueras, J. Planas, M. Rodamilans, and J. Corbella. "Pentachlorophenol and Hexachlorobenzene in Serum and Urine of the Population of Barcelona." Human Toxicology 6, no. 5 (1987): 397–400. http://dx.doi.org/10.1177/096032718700600509.

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1 Urinary chlorophenols of the general population of Barcelona, Spain were determined. Pentachlorophenol (PCP: 25.0 ± 3.9 ng/ml; x ± s.e.m., n = 50) and tetrachlorophenol (TCP: 6.2 ± 1.6 ng/ml; x ± s.e.m., n = 25) were found in all samples. 2 Pentachlorophenol and hexachlorobenzene were also determined in serum. Both were present in all samples (PCP: 21.9 ± 1.9 ng/ml; HCB: 11.1 ± 1.1 ng/ml; x ± s.e.m., n = 100). Their concentrations do not show any correlation, suggesting no metabolic relation between them.

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37

Bouthillier, Léo, Erminio Greselin, Jules Brodeur, Claude Viau, and Michel Charbonneau. "Male rat specific nephrotoxicity resulting from subchronic administration of hexachlorobenzene." Toxicology and Applied Pharmacology 110, no. 2 (1991): 315–26. http://dx.doi.org/10.1016/s0041-008x(05)80014-6.

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38

Carpenter, Hillary M., David E. Williams, and Donald R. Buhler. "Hexachlorobenzene‐induced porphyria in Japanese quail: Changes in microsomal enzymes." Journal of Toxicology and Environmental Health 15, no. 3-4 (1985): 431–44. http://dx.doi.org/10.1080/15287398509530670.

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39

Stonard, Michael D., Giuseppe Poli, and Francesco De Matteis. "Stimulation of liver heme oxygenase in hexachlorobenzene-induced hepatic porphyria." Archives of Toxicology 72, no. 6 (1998): 355–61. http://dx.doi.org/10.1007/s002040050514.

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40

Jarrell, J. F., A. Gocmen, D. Akyol, and R. Brant. "Hexachlorobenzene exposure and the proportion of male births in Turkey 1935–1990." Reproductive Toxicology 16, no. 1 (2002): 65–70. http://dx.doi.org/10.1016/s0890-6238(01)00196-4.

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41

Foster, Warren G., Julie A. Pentick, Avril McMahon, and Pierre R. Lecavalier. "Body distribution and endocrine toxicity of hexachlorobenzene (HCB) in the female rat." Journal of Applied Toxicology 13, no. 2 (1993): 79–83. http://dx.doi.org/10.1002/jat.2550130203.

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42

Chen, I.-Ming, Wanit Wanitchapichat, Teeranuch Jirakittayakorn, et al. "Hexachlorobenzene dechlorination by indigenous sediment microorganisms." Journal of Hazardous Materials 177, no. 1-3 (2010): 244–50. http://dx.doi.org/10.1016/j.jhazmat.2009.12.024.

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43

Harleman, J. H., F. Staedtler, J. Ezendam, et al. "2 Toxicogenomics of subchronic hexachlorobenzene exposure of brown Norway rats." Toxicology Letters 144 (September 2003): s1. http://dx.doi.org/10.1016/s0378-4274(03)90001-4.

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44

Zhan, Y. Xu, A. H. Li, J. Zhang, K., W. "Endocrine Disruption by Hexachlorobenzene in Crucian Carp ( Carassius auratus gibelio )." Bulletin of Environmental Contamination and Toxicology 65, no. 5 (2000): 560–66. http://dx.doi.org/10.1007/s001280000160.

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45

Hamdy, M. K. "Effect of hexachlorobenzene on growth and survival of various microorganisms." Bulletin of Environmental Contamination and Toxicology 41, no. 4-6 (1988): 936–42. http://dx.doi.org/10.1007/bf02021058.

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46

Gorman, Nadia, Heidi S. Trask, Susan W. Robinson, et al. "Hexachlorobenzene stimulates uroporphyria in low affinity AHR mice without increasing CYP1A2." Toxicology and Applied Pharmacology 221, no. 2 (2007): 235–42. http://dx.doi.org/10.1016/j.taap.2007.03.007.

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47

Sinclair, Peter R., Nadia Gorman, Heidi S. Walton, et al. "CYP1A2 Is Essential in Murine Uroporphyria Caused by Hexachlorobenzene and Iron." Toxicology and Applied Pharmacology 162, no. 1 (2000): 60–67. http://dx.doi.org/10.1006/taap.1999.8832.

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48

Carpenter, Hillary M., Michael J. Harvey, and Donald R. Buhler. "The effect of tetrachlorohydroquinone on hexachlorobenzene‐induced porphyria in Japanese quail." Journal of Toxicology and Environmental Health 15, no. 1 (1985): 81–92. http://dx.doi.org/10.1080/15287398509530637.

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49

Ezendam, J. "Hexachlorobenzene-induced Immunopathology in Brown Norway Rats is Partly Mediated by T Cells." Toxicological Sciences 78, no. 1 (2004): 88–95. http://dx.doi.org/10.1093/toxsci/kfh034.

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50

Plante, I., D. G. Cyr, and M. Charbonneau. "Sexual Dimorphism in the Regulation of Liver Connexin32 Transcription in Hexachlorobenzene-Treated Rats." Toxicological Sciences 96, no. 1 (2006): 47–57. http://dx.doi.org/10.1093/toxsci/kfl181.

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